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Relevant bibliographies by topics / Antibiotic effects on cochlea

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Academic literature on the topic 'Antibiotic effects on cochlea'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Antibiotic effects on cochlea"

1

Moore, David R., Nina J. Rogers, and Stephen J. O'Leary. "Loss of Cochlear Nucleus Neurons following Aminoglycoside Antibiotics or Cochlear Removal." Annals of Otology, Rhinology & Laryngology 107, no. 4 (April 1998): 337–43. http://dx.doi.org/10.1177/000348949810700413.

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This study compared the effects of aminoglycoside ototoxicity and surgical ablation of the cochlea in infancy on the survival of neurons in the rat cochlear nucleus (CN). Ototoxicity was induced by a single, systemic dose of gentamicin sulfate and furosemide on postnatal day 6 (P6), P7, or P10, and assessed by the elevation of auditory brain stem response thresholds, as described in a companion paper. Unilateral cochlear removals were performed under Saffan anesthesia on P6, P9, and P12. Rats were painlessly sacrificed in adulthood, and the formalin-perfused brains and cochleas were embedded in wax, sectioned, and stained. Ototoxic treatment at P6 through P10 did not reduce neuron counts in the CN. Cochlear removal at P6 resulted in a 40% loss of CN neurons, but removal at P12 did not result in CN neuron loss. These data suggest that the critical period for the dependence of CN neurons on afferent input from the cochlea ends at the same time that susceptibility to aminoglycoside ototoxicity begins.

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Meli, Damian N., Roney S. Coimbra, Dominik G. Erhart, Gerard Loquet, Caroline L. Bellac, Martin G. Täuber, Ulf Neumann, and Stephen L. Leib. "Doxycycline Reduces Mortality and Injury to the Brain and Cochlea in Experimental Pneumococcal Meningitis." Infection and Immunity 74, no. 7 (July 2006): 3890–96. http://dx.doi.org/10.1128/iai.01949-05.

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ABSTRACT Bacterial meningitis is characterized by an inflammatory reaction to the invading pathogens that can ultimately lead to sensorineural hearing loss, permanent brain injury, or death. The matrix metalloproteinases (MMPs) and tumor necrosis factor alpha-converting enzyme (TACE) are key mediators that promote inflammation, blood-brain barrier disruption, and brain injury in bacterial meningitis. Doxycycline is a clinically used antibiotic with anti-inflammatory effects that lead to reduced cytokine release and the inhibition of MMPs. Here, doxycycline inhibited TACE with a 50% inhibitory dose of 74 μM in vitro and reduced the amount of tumor necrosis factor alpha released into the cerebrospinal fluid by 90% in vivo. In an infant rat model of pneumococcal meningitis, a single dose of doxycycline (30 mg/kg) given as adjuvant therapy in addition to ceftriaxone 18 h after infection significantly reduced the mortality, the blood-brain barrier disruption, and the extent of cortical brain injury. Adjuvant doxycycline (30 mg/kg given subcutaneously once daily for 4 days) also attenuated hearing loss, as assessed by auditory brainstem response audiometry, and neuronal death in the cochlear spiral ganglion at 3 weeks after infection. Thus, doxycycline, probably as a result of its anti-inflammatory properties, had broad beneficial effects in the brain and the cochlea and improved survival in this model of pneumococcal meningitis in infant rats.

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Xuan, Weijun, Mingmin Dong, and Minsheng Dong. "Effects of Compound Injection of Pyrola Rotundifolia L and Astragalus Membranaceus Bge on Experimental Guinea Pigs' Gentamicin Ototoxicity." Annals of Otology, Rhinology & Laryngology 104, no. 5 (May 1995): 374–80. http://dx.doi.org/10.1177/000348949510400507.

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In attempting to find drugs effective in preventing and remedying ototoxic injury caused by aminoglycoside antibiotics, we relied on the theory that the induction of ototoxic injury by aminoglycoside antibiotics is related to a decrease of cyclic adenosine monophosphate and RNA content in the cochlea or a dysfunction of the kidney. We selected Pyrola rotundifolia L and Astragalus membranaceus Bge from traditional Chinese herbal medicine, made a compound injection of them, and observed the effect on the pattern of gentamicin ototoxicity in guinea pigs. By electrocochleography and morphology by scanning electron microscopy, the experimental results indicated that the Chinese herbal compound possessed the definite effect of protecting the guinea pig cochlea. The determination of blood urea nitrogen, urinary N-acetyl-d-aminoglucosidase, and urinary protein and observation of renal morphology showed that it also protected the kidney against nephrotoxic nephritis of gentamicin. The conjecture that protection of the kidney by the Chinese herbs may be one of the important factors in preventing ototoxicity supports some explanations of ototoxic mechanisms induced by aminoglycoside antibiotics.

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Lee, Sun, Hyun Ju, Jin Choi, Yeji Ahn, Suhun Lee, and Young Seo. "Circulating Serum miRNA-205 as a Diagnostic Biomarker for Ototoxicity in Mice Treated with Aminoglycoside Antibiotics." International Journal of Molecular Sciences 19, no. 9 (September 19, 2018): 2836. http://dx.doi.org/10.3390/ijms19092836.

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Background: To confirm levels and detection timing of circulating microRNAs (miRNAs) in the serum of a mouse model for diagnosis of ototoxicity, circulating miR-205 in the serum was evaluated to reflect damages in the cochlear microstructure and compared to a kidney injury model. Method: A microarray for miRNAs in the serum was performed to assess the ototoxic effects of kanamycin-furosemide. Changes in the levels for the selected miRNAs (miR-205, miR-183, and miR-103) were compared in the serum and microstructures of the cochlea (stria vascularis, organ of Corti, and modiolus) between the ototoxicity and normal mouse groups. An acute kidney injury (AKI) mouse model was used to assess changes in miR-205 levels in the kidney by ototoxic drugs. Results: In the mouse model for ototoxicity, the serum levels of circulating miR-205 peaked on day 3 and were sustained from days 7–14. Furthermore, miR-205 expression was highly expressed in the organ of Corti at day 5, continued to be expressed in the modiolus at high levels until day 14, and was finally also in the stria vascularis. The serum miR-205 in the AKI mice did not change significantly compared to the normal group. Conclusions Circulating miR-205 from the cochlea, after ototoxic damage, migrates through the blood vessels to organs, which is then finally found in blood. In conditions of hearing impairment with ototoxic medications, detection of circulating miR-205 in the blood can be used to determine the extent of hearing loss. In the future, inner ear damage can be identified by simply performing a blood test before the hearing impairment due to ototoxic drugs.

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Huseynov, N. M., V. R. Fisenko, and P. R. Aslanov. "INFLUENCE OF OTOTOXIC PHARMACEUTICALS ON BIOELECTRIC RESPONSES IN CEREBRAL CORTEX AND COCHLEA." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 20, no. 2 (July 6, 2020): 124–28. http://dx.doi.org/10.31718/2077-1096.20.2.124.

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The article describes the results of the experiment aimed to find out the nature of changes in the excitability of the auditory cortex during prolonged taking of antibiotics, aminoglycosides, diuretics and salicylates. Methodology. The series of experiments was carried out on cats of both sexes weighing 3-3.5 kg, in natural behaviour. Rectangular threshold (1.5-3 V) and supra-threshold (5-7 V) electrical impulses lasting 0.1 ms were applied to the TCR fibres, using single and paired (interstimular intervals of 20-500ms) stimulation of the TCR fibres. As a source of irritating impulses, a multi-channel stimulator "SEN-3201" of the company "Nikon-Kohden" (Japan) was used. For each skeletal interval was carried out on 10 repetitions. The SOFTWARE was registered using the average Det-1100 average from Nikon-Kohden (Japan). The resulting visual control was performed from the screen of the "Disa-Indicator" oscilloscope (Denmark). The maximum level of the intensity value reached 110 dB. Sound signals were transmitted through special high-frequency phones (or speakers) of the GD-4 type, which were in a free state, at a distance of 10 cm from the ear of the test ear. All the studied pharmaceuticals (furosemide, streptomycin, gentamicin, kanamycin, monomycin, tobramycin and amikacin) except acetylsalicylic acid, which was injected intraperitoneally, were administered to cats intramuscularly 2 times a day at intervals of 10-12 hours. Bioelectric reactions of the cortex and cochlea were recorded daily 2-3 hours after the next administration of drugs. Streptomycin, kanamycin and monomycin were used at a dose of 40 mg / kg, which causes inhibition of the cochlea's MIC response (MOI) and auditory nerve PD only with long-term use. Gentamicin, tobramycin and amikacin were administered in a lower (15 mg/kg) dose. Results. Furosemide reduced by 50% the value of the microphone potential recorded during sound stimulation with a frequency of 10 kHz after 4 days, with a frequency of 1 kHz-5 days, with a frequency of 0.5 kHz-6 days from the beginning of use. The most sensitive to furosemide were auditory nerve PD, which occurred during sound stimulation at a frequency of 10 kHz (T50 – 5 days), and less – at a frequency of 0.5 kHz (T50 – 7 days). The cancellation of furosemide was accompanied by the restoration of the value of these bioelectric reactions to the initial level in 5 days. Thus, the data obtained indicate the possibility of a reversible depressing effect of furosemide on the responses of the CGM and the cochlea that occur on sound stimulation. All the studied antibiotics-aminoglycosides had the ability to reduce the value of the testing primary response when paired electrical stimulation of TCR fibres. These changes were observed 2-3 days after the start of the use of antibiotics. Initial changes in the value of MP and PD of the auditory nerve occurred after 12-24 times of drug administration. In this case, a decrease in these potentials by 20-25% was noted. Further use of antibiotics led to an increase in changes in responses registered in the cochlea, with the greatest sensitivity to the effects of drugs were reactions to high-frequency sound stimuli. When analyzing the ability of the studied tools to disrupt the activity of the peripheral fragment of the auditory analyzer, a pattern similar to that for the cerebral cortex was found. Withdrawal of drugs at the stage of initial changes in potentials registered in the cochlea is not accompanied by their recovery for a long (6 weeks) period after stopping the administration of antibiotics. Conclusions. Different rates of formation of damage to interneuronal transmissions were found during long-term use of different antibiotics.

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Kim, Ye-Ri, Tae-Jun Kwon, Un-Kyung Kim, In-Kyu Lee, Kyu-Yup Lee, and Jeong-In Baek. "Fursultiamine Prevents Drug-Induced Ototoxicity by Reducing Accumulation of Reactive Oxygen Species in Mouse Cochlea." Antioxidants 10, no. 10 (September 26, 2021): 1526. http://dx.doi.org/10.3390/antiox10101526.

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Drug-induced hearing loss is a major type of acquired sensorineural hearing loss. Cisplatin and aminoglycoside antibiotics have been known to cause ototoxicity, and excessive accumulation of intracellular reactive oxygen species (ROS) are suggested as the common major pathology of cisplatin- and aminoglycoside antibiotics-induced ototoxicity. Fursultiamine, also called thiamine tetrahydrofurfuryl disulfide, is a thiamine disulfide derivative that may have antioxidant effects. To evaluate whether fursultiamine can prevent cisplatin- and kanamycin-induced ototoxicity, we investigated their preventive potential using mouse cochlear explant culture system. Immunofluorescence staining of mouse cochlear hair cells showed that fursultiamine pretreatment reduced cisplatin- and kanamycin-induced damage to both inner and outer hair cells. Fursultiamine attenuated mitochondrial ROS accumulation as evidenced by MitoSOX Red staining and restored mitochondrial membrane potential in a JC-1 assay. In addition, fursultiamine pretreatment reduced active caspase-3 and TUNEL signals after cisplatin or kanamycin treatment, indicating that fursultiamine decreased apoptotic hair cell death. This study is the first to show a protective effect of fursultiamine against cisplatin- and aminoglycoside antibiotics-induced ototoxicity. Our results suggest that fursultiamine could act as an antioxidant and anti-apoptotic agent against mitochondrial oxidative stress.in cochlear hair cells

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Yoshida, Naohiro, M. Charles Liberman, M. Christian Brown, and William F. Sewell. "Gentamicin Blocks Both Fast and Slow Effects of Olivocochlear Activation in Anesthetized Guinea Pigs." Journal of Neurophysiology 82, no. 6 (December 1, 1999): 3168–74. http://dx.doi.org/10.1152/jn.1999.82.6.3168.

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The medial olivocochlear (MOC) efferent system, which innervates cochlear outer hair cells, suppresses cochlear responses. MOC-mediated suppression includes both slow and fast components, with time courses differing by three orders of magnitude. Pharmacological studies in anesthetized guinea pigs suggest that both slow and fast effects on cochlear responses require an initial acetylcholine activation of α-9 nicotinic receptors on outer hair cells and that slow effects require additional intracellular events downstream from those mediating fast effects. Gentamicin, an aminoglycoside antibiotic, has been reported to block fast effects of sound-evoked OC activation following intramuscular injection in unanesthetized guinea pigs, without changing slow effects. In the present study, we show that electrically evoked fast and slow effects in the anesthetized guinea pig are both blocked by either intramuscular or intracochlear gentamicin, with similar time courses and/or dose-response curves. We suggest that sound-evoked slow effects in unanesthetized animals are fundamentally different from electrically evoked slow effects in anesthetized animals, and that the former may arise from effects of the lateral OC system.

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Nakagawa, T., S. Kakehata, N. Akaike, S. Komune, T. Takasaka, and T. Uemura. "Effects of Ca2+ antagonists and aminoglycoside antibiotics on Ca2+ current in isolated outer hair cells of guinea pig cochlea." Brain Research 580, no. 1-2 (May 1992): 345–47. http://dx.doi.org/10.1016/0006-8993(92)90966-d.

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O’Sullivan, Mary E., Yohan Song, Robert Greenhouse, Randy Lin, Adela Perez, Patrick J. Atkinson, Jacob P. MacDonald, et al. "Dissociating antibacterial from ototoxic effects of gentamicin C-subtypes." Proceedings of the National Academy of Sciences 117, no. 51 (December 7, 2020): 32423–32. http://dx.doi.org/10.1073/pnas.2013065117.

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Gentamicin is a potent broad-spectrum aminoglycoside antibiotic whose use is hampered by ototoxic side-effects. Hospital gentamicin is a mixture of five gentamicin C-subtypes and several impurities of various ranges of nonexact concentrations. We developed a purification strategy enabling assaying of individual C-subtypes and impurities for ototoxicity and antimicrobial activity. We found that C-subtypes displayed broad and potent in vitro antimicrobial activities comparable to the hospital gentamicin mixture. In contrast, they showed different degrees of ototoxicity in cochlear explants, with gentamicin C2b being the least and gentamicin C2 the most ototoxic. Structure–activity relationships identified sites in the C4′-C6′ region on ring I that reduced ototoxicity while preserving antimicrobial activity, thus identifying targets for future drug design and mechanisms for hair cell toxicity. Structure–activity relationship data suggested and electrophysiological data showed that the C-subtypes both bind and permeate the hair cell mechanotransducer channel, with the stronger the binding the less ototoxic the compound. Finally, both individual and reformulated mixtures of C-subtypes demonstrated decreased ototoxicity while maintaining antimicrobial activity, thereby serving as a proof-of-concept of drug reformulation to minimizing ototoxicity of gentamicin in patients.

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Beitel, Ralph E., Russell L. Snyder, Christoph E. Schreiner, Marcia W. Raggio, and Patricia A. Leake. "Electrical Cochlear Stimulation in the Deaf Cat: Comparisons Between Psychophysical and Central Auditory Neuronal Thresholds." Journal of Neurophysiology 83, no. 4 (April 1, 2000): 2145–62. http://dx.doi.org/10.1152/jn.2000.83.4.2145.

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Cochlear prostheses for electrical stimulation of the auditory nerve (“electrical hearing”) can provide auditory capacity for profoundly deaf adults and children, including in many cases a restored ability to perceive speech without visual cues. A fundamental challenge in auditory neuroscience is to understand the neural and perceptual mechanisms that make rehabilitation of hearing possible in these deaf humans. We have developed a feline behavioral model that allows us to study behavioral and physiological variables in the same deaf animals. Cats deafened by injection of ototoxic antibiotics were implanted with either a monopolar round window electrode or a multichannel scala tympani electrode array. To evaluate the effects of perceptually significant electrical stimulation of the auditory nerve on the central auditory system, an animal was trained to avoid a mild electrocutaneous shock when biphasic current pulses (0.2 ms/phase) were delivered to its implanted cochlea. Psychophysical detection thresholds and electrical auditory brain stem response (EABR) thresholds were estimated in each cat. At the conclusion of behavioral testing, acute physiological experiments were conducted, and threshold responses were recorded for single neurons and multineuronal clusters in the central nucleus of the inferior colliculus (ICC) and the primary auditory cortex (A1). Behavioral and neurophysiological thresholds were evaluated with reference to cochlear histopathology in the same deaf cats. The results of the present study include: 1) in the cats implanted with a scala tympani electrode array, the lowest ICC and A1 neural thresholds were virtually identical to the behavioral thresholds for intracochlear bipolar stimulation; 2) behavioral thresholds were lower than ICC and A1 neural thresholds in each of the cats implanted with a monopolar round window electrode; 3) EABR thresholds were higher than behavioral thresholds in all of the cats (mean difference = 6.5 dB); and 4) the cumulative number of action potentials for a sample of ICC neurons increased monotonically as a function of the amplitude and the number of stimulating biphasic pulses. This physiological result suggests that the output from the ICC may be integrated spatially across neurons and temporally integrated across pulses when the auditory nerve array is stimulated with a train of biphasic current pulses. Because behavioral thresholds were lower and reaction times were faster at a pulse rate of 30 pps compared with a pulse rate of 2 pps, spatial-temporal integration in the central auditory system was presumably reflected in psychophysical performance.

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Dissertations / Theses on the topic "Antibiotic effects on cochlea"

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Wright, A. "Structural changes in the human cochlea during drug treatment." Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.371567.

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Berninger, Erik. "Quinine as a model for the study of cochlear hearing loss in humans /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4272-2/.

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Chin, Alex C. "Anti-inflammatory effects of the macrolide antibiotic tilmicosin." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ31336.pdf.

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El-Sabbagh, Nasser Mohamed. "Effects of dissolved carbon dioxide on antibiotic production." Thesis, University of Strathclyde, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415315.

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Khan, David D. "Pharmacokinetic-Pharmacodynamic modeling and prediction of antibiotic effects." Doctoral thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-282604.

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Problems of emerging antibiotic resistance are becoming a serious threat worldwide, and at the same time, the interest to develop new antimicrobials has declined. There is consequently a need for efficient methods to develop new treatments that minimize the risk of resistance development and that are effective on infections caused by resistant strains. Based on in silico mathematical models, describing the time course of exposure (Pharmacokinetics, PK) and effect (Pharmacodynamics, PD) of a drug, information can be collected and the outcome of various exposures may be predicted. A general model structure, that characterizes the most important features of the system, has advantages as it can be used for different situations. The aim of this thesis was to develop Pharmacokinetic-Pharmacodynamic (PKPD) models describing the bacterial growth and killing after mono- and combination exposures to antibiotics and to explore the predictive ability of PKPD-models across preclinical experimental systems. Models were evaluated on data from other experimental settings, including prediction into animals. A PKPD model characterizing the growth and killing for a range of E. coli bacteria strains, with different MICs, as well as emergence of resistance, was developed. The PKPD model was able to predict results from different experimental conditions including high start inoculum experiments, a range of laboratory and clinical strains as well as experiments where wild-type and mutant bacteria are competing at different drug concentrations. A PKPD model, developed based on in vitro data, was also illustrated to have the capability to replicate the data from an in vivo study. This thesis illustrates the potential of PKPD models to characterize in vitro data and their usage for predictions of different types of experiments. The thesis supports the use of PKPD models to facilitate development of new drugs and to improve the use of existing antibiotics.

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Webb, Jason Crispin John. "The Effects of Antibiotic Combinations on Bone Cement Properties." Thesis, University of Bristol, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.525435.

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Wikblom, Ida. "Antibiotic Use and Effects in the Terminally Ill : – A Retrospective Review." Thesis, Örebro universitet, Institutionen för medicinska vetenskaper, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-66978.

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Fisher, Morgane, (Dennison) Jaime Thomas, and Danielle Weimann. "Effects of an Educational Intervention on Parental Knowledge Regarding Antibiotic Resistance." The University of Arizona, 2008. http://hdl.handle.net/10150/624276.

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Class of 2008 Abstract
Objectives: To evaluate changes in parental knowledge regarding antibiotic use and antibiotic resistance with an educational intervention given at elementary school parent-teacher association (PTA) meetings. Methods: This was an analytical pre-test/post-test study of an educational intervention given at two elementary schools in the Phoenix metro area. The primary dependent variable was a knowledge measure, calculated as a total score. The changes between the pre- and post-test total score means were compared using a dependent t-test. The a-priori alpha level used was 0.05. Results: The study sample consisted of 25 participants. Study data were collected between September 2007 and December 2007. The mean (SD) pre- and post-test scores were 33.7 (4.4) and 40.7 (2.7), respectively (p < 0.05). Conclusions: The educational intervention presented at elementary school PTA meetings resulted in a significant knowledge increase regarding the appropriate use of antibiotics when pre- and post-test scores were compared.

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Gbejuade, Herbert Olukayode. "The effects of antibiotic loaded bone cement combinations on bacteria biofilms." Thesis, University of Bristol, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.705469.

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Gordon, C. A. "The contribution of alginate to the antibiotic susceptibility of Pseudomonas aeruginosa." Thesis, University of Brighton, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.384621.

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Books on the topic "Antibiotic effects on cochlea"

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Vekshin, N. L. Biophysics of DNA-antibiotic complexes. Hauppauge, N.Y: Nova Science Publishers, 2010.

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Enzybiotics: Antibiotic enzymes as drugs and therapeutics. Hoboken, N.J: John Wiley & Sons, 2010.

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Tackling antibiotic resistance from a food safety perspective in Europe. Copenhagen: World Health Organization, 2011.

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Offit, Paul A. Breaking the antibiotic habit: A parent's guide to coughs, colds, ear infections, and sore throats. New York: John Wiley, 1999.

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Audiovestibular toxicity of drugs. Boca Raton, Fla: CRC Press, 1989.

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Wright, Jennifer Gordon. Use of anthrax vaccine in the United States: Recommendations of the Advisory Committee on Immunization Practices (ACIP), 2009. Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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Amabile-Cuevas, Carlos F. Antibiotic Resistance. Eurekah.Com Inc, 2002.

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B, Levy Stuart, Novick Richard P. 1932-, Cold Spring Harbor Laboratory, United States. Environmental Protection Agency., and National Science Foundation (U.S.), eds. Antibiotic resistance genes: Ecology, transfer, and expression. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1986.

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S, Moore Wesley, and Gelabert Hugh A, eds. Antibiotic-impregnated vascular grafts. Austin: R.G. Landes, 1992.

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Enzybiotics: Antibiotic enzymes as drugs and therapeutics. Hoboken, N.J: John Wiley & Sons, 2010.

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Book chapters on the topic "Antibiotic effects on cochlea"

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Singer, Andrew C., and Heike Schmitt. "Antibiotic Use During an Influenza Pandemic: Downstream Ecological Effects and Antibiotic Resistance." In Antimicrobial Resistance in the Environment, 503–37. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118156247.ch26.

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Gnarpe, Håkan, and Judy Belsheim. "Indirect Antibiotic Effects on Granulocyte Migration and Phagocytosis." In The Influence of Antibiotics on the Host-Parasite Relationship II, 159–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70748-3_17.

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Frank, Uwe, and Evelina Tacconelli. "Antibiotics, Antimycotics: Spectrum – Dosage – Adverse Effects – Costs." In The Daschner Guide to In-Hospital Antibiotic Therapy, 77–175. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-18402-4_9.

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Forni, C., E. Tel-Or, E. Bar, and M. Grilli Caiola. "Effects of antibiotic treatments on Azolla-Anabaena and Arthrobacter." In Nitrogen Fixation, 457–61. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3486-6_95.

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D’Angelo, Salvo, Marcello Masili, and Riccardo Malvano. "Linear and Non-Linear Effects in a Physical Model of the Cochlea." In Lecture Notes in Biomathematics, 282–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-50038-1_35.

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Andersson, Dan I., and Diarmaid Hughes. "Effects of Antibiotic Resistance on Bacterial Fitness, Virulence, and Transmission." In Evolutionary Biology of Bacterial and Fungal Pathogens, 307–18. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815639.ch26.

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Bamigboye, Olutoyin Omolara, and Idowu Jesulayomi Adeosun. "Antimicrobial Compounds from Medicinal Plants: Effects on Antibiotic Resistance to Human Pathogens." In The Therapeutic Properties of Medicinal Plants, 123–34. Series statement: Innovations in plant science for better health: from soil to fork: Apple Academic Press, 2019. http://dx.doi.org/10.1201/9780429265204-6.

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Schultz, Markus J., and Peter E. Spronk. "The Effects of Hand-Washing, Restrictive Antibiotic Use and SDD on Morbidity." In Selective Digestive Tract Decontamination in Intensive Care Medicine: a Practical Guide to Controlling Infection, 99–110. Milano: Springer Milan, 2008. http://dx.doi.org/10.1007/978-88-470-0653-9_7.

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Hashmi, Muhammad Zaffar, Adeel Mahmood, Dambaru Ballab Kattel, Sohaib Khan, Ahmad Hasnain, and Zulkifl Ahmed. "Antibiotics and Antibiotic Resistance Genes (ARGs) in Soil: Occurrence, Fate, and Effects." In Soil Biology, 41–54. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47744-2_4.

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Szentivanyi, Andor, Herman Friedman, Günther Gillissen, and Judith Szentivanyi. "Immunomodulatory Effects of Some Antibiotic-Induced Changes in Bacterial and Host Determinants." In Antibiosis and Host Immunity, 3–11. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1901-6_1.

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Conference papers on the topic "Antibiotic effects on cochlea"

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Qiu, Fan, Li Meng, Hao Jin, and Jun Chen. "Effects of a Restrictive Antibiotic Policy on Antibiotic Usage and Staphylococcus Aureus Resistance." In International Conference on Electronics, Mechanics, Culture and Medicine. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/emcm-15.2016.97.

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Ramlachan, N., T. L. Poole, W. K. Kim, T. R. Callaway, S. C. Ricke, Robin C. Anderson, R. B. Harvey, and David J. Nisbet. "Effects of antibiotic-suppelmented media on recovery of enterobacteria." In Sixth International Symposium on the Epidemiology and Control of Foodborne Pathogens in Pork. Iowa State University, Digital Press, 2005. http://dx.doi.org/10.31274/safepork-180809-788.

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Hamao, Nobuyoshi, Isao Ito, Satoshi Konishi, Naoya Tanabe, Issei Oi, and Toyohiro Hirai. "Frequency of side effects in antibiotic treatment for pneumonia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4552.

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Karamova, Kamalya. "EFFECTS OF COMPOST CONTAINING ANTIBIOTIC RESISTANT GENES ON SOIL CHARACTERISTICS." In 19th SGEM International Multidisciplinary Scientific GeoConference EXPO Proceedings. STEF92 Technology, 2019. http://dx.doi.org/10.5593/sgem2019/5.2/s20.040.

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Arora, Shuchi, Chu Sing Lim, and Carl Baptista. "Screening of Antibiotic-Maggot Serum Combinations and Their Effects on Bacterial Physiology." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE 2010). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5516865.

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Skweres, Joyce A., Virginia J. Bassinger, and S. K. Mishra, and Duane L. Pierson. "Effects of Refrigerating Preinoculated Vitek Cards on Microbial Physiology and Antibiotic Susceptibility." In International Conference On Environmental Systems. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1992. http://dx.doi.org/10.4271/921214.

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Babic, Sandra, Davor Ljubas, Durdica Ivanec Sipusic, Lidija Curkovic, and Jasmina Beganovic. "Effects of pH and water matrix on photoinduced degradation of antibiotic cefdinir." In The 4th World Congress on Civil, Structural, and Environmental Engineering. Avestia Publishing, 2019. http://dx.doi.org/10.11159/iceptp19.118.

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"Effects of Scenedesmus microalgae and virginiamycin antibiotic on blood parameters of broiler chicks." In International Conference on Medicine, Public Health and Biological Sciences. CASRP Publishing Company, Ltd. Uk, 2016. http://dx.doi.org/10.18869/mphbs.2016.214.

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Sequeira, Damien, Christian Breuninger, Albrecht Eiber, and Alexander Huber. "THE EFFECTS OF COMPLEX STAPES MOTION ON THE RESPONSE OF THE COCHLEA IN GUINEA PIGS." In Proceedings of the 4th International Symposium. WORLD SCIENTIFIC, 2007. http://dx.doi.org/10.1142/9789812708694_0017.

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CILTAS, ABDULKADIR, and YUSUF ZEKERIYA. "Effects of Oxytetracycline Antibiotic on GST Enzyme Activity and Gene Expression Levels in Rainbow Trout." In Fifth International Conference On Advances in Applied Science and Environmental Technology- ASET 2016. Institute of Research Engineers and Doctors, 2016. http://dx.doi.org/10.15224/978-1-63248-106-1-32.

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Reports on the topic "Antibiotic effects on cochlea"

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Oliveira, Mariana, Vívian Souza, Guilherme Tavares, Rodrigo Fabri, and Ana Carolina Apolônio. Effects of antibiotic-loaded chitosan nanoparticles against resistant bacteria: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2021. http://dx.doi.org/10.37766/inplasy2021.6.0069.

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Teixeira, Jaclyn. Effects of Antibiotic Mixtures across Marine Intertidal Trophic Levels: Examining Environmentally-Relevant Contaminant Concentrations. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.5263.

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Block, Jason, Christopher Forrest, Douglas Lunsford, Juliane Reynolds, and Bridget Nolan. Looking at the Effects of Antibiotic Use on Childhood Obesity and Growth -- The PCORnet® Antibiotics Study. Patient-Centered Outcomes Research Institute (PCORI), October 2020. http://dx.doi.org/10.25302/10.2020.obs.150530699.

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Evidence Update for Clinicians: Narrow- versus Broad-Spectrum Antibiotics for Common Infections in Children. Patient-Centered Outcomes Research Institute (PCORI), October 2018. http://dx.doi.org/10.25302/eu5.2018.10.

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Comparing Narrow- vs. Broad-Spectrum Antibiotics for Common Infections in Children. The choice of antibiotic to treat acute bacterial upper respiratory tract infections in children can affect both symptom resolution and the risk of side effects such as diarrhea and vomiting. The findings of a PCORI-funded study published in JAMA can help clinicians treating children for acute respiratory tract infections (ARTIs)—including acute otitis media, Group A streptococcal pharyngitis, and acute sinusitis—make decisions with parents about the medicine that is best for the child. The study, led by Jeffrey Gerber, a pediatrician and researcher at the Children’s Hospital of Philadelphia, included 30,086 children ages 6 months to 12 years taking narrow- and broad-spectrum antibiotics to treat ARTIs.

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