Relevant bibliographies by topics / Anti-aging strategy

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Anti-aging strategy'

Author: Grafiati
Published: 25 May 2024

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Journal articles on the topic "Anti-aging strategy"

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Gonos, Efstathios S. "Proteasome activation as a novel anti-aging strategy." Free Radical Biology and Medicine 65 (September 2013): S5. http://dx.doi.org/10.1016/j.freeradbiomed.2013.08.114.

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Gonos, Efstathios. "Proteasome activation as a novel anti-aging strategy." Free Radical Biology and Medicine 75 (October 2014): S7. http://dx.doi.org/10.1016/j.freeradbiomed.2014.10.842.

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Gonos, S. "THE AGING PROTEASOME AND ITS ACTIVATION AS A NOVEL ANTI-AGING STRATEGY." Innovation in Aging 1, suppl_1 (June 30, 2017): 937. http://dx.doi.org/10.1093/geroni/igx004.3362.

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Choudhery, Mahmood S., and David T. Harris. "Cryopreservation can be used as an anti-aging strategy." Cytotherapy 16, no. 12 (December 2014): 1771–73. http://dx.doi.org/10.1016/j.jcyt.2014.08.012.

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Meiliana, Anna, Nurrani Mustika Dewi, and Andi Wijaya. "In Search for Anti-Aging Strategy: Can We Rejuvenate Our Aging Stem Cells?" Indonesian Biomedical Journal 7, no. 2 (August 1, 2015): 57. http://dx.doi.org/10.18585/inabj.v7i2.72.

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BACKGROUND: Recent evidence suggested that we grow old partly because of our stem cells grow old as a result of mechanisms that suppress the development of cancer over a lifetime. We believe that a further, more precise mechanistic understanding of this process will be required before this knowledge can be translated into human anti-aging therapies.CONTENT: A diminished capacity to maintain tissue homeostasis is a central physiological characteristic of aging. As stem cells regulate tissue homeostasis, depletion of stem cell reserves and/or diminished stem cell function have been postulated to contribute to aging. It has further been suggested that accumulated DNA damage could be a principal mechanism underlying age-dependent stem cell decline. It is interesting that many of the rejuvenating interventions act on the stem cell compartments, perhaps reflecting shared genetic and biochemical pathways controlling stem cell function and longevity. Strategy to slow down the aging processes is based on caloric restriction refers to a dietary regimen low in calories but without undernutrition. Sirtuin (SIRT)1 and 3, increases longevity by mimicking the beneficial effects of caloric restriction. SIRT3 regulates stress-responsive mitochondrial homeostasis, and more importantly, SIRT3 upregulation rejuvenates aged stem cells in tissues. Resveratrol (3,5,4’-trihydroxystilbene), a natural polyphenol found in grapes and wine, was the most powerful natural activator of SIRT1. In fact, resveratrol treatment has been demonstrated to rescue adult stem cell decline, slow down bodyweight loss, improve trabecular bone structure and mineral density, and significantly extend lifespan.SUMMARY: Tissue-specific stem cells persist throughout the entire lifespan to repair and maintain tissues, but their self-renewal and differentiation potential become dysregulated with aging. Given that adult stem cells are thought to be central to tissue maintenance and organismal survival, SIRT3 may promote organismal longevity by maintaining the integrity of tissue-speciic stem cells.KEYWORDS: rejuvenation, aging, stem cell, DNA damage, sirtuin activator
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Lee, Yun Haeng, Ji Yun Park, Haneur Lee, Eun Seon Song, Myeong Uk Kuk, Junghyun Joo, Sekyung Oh, Hyung Wook Kwon, Joon Tae Park, and Sang Chul Park. "Targeting Mitochondrial Metabolism as a Strategy to Treat Senescence." Cells 10, no. 11 (November 3, 2021): 3003. http://dx.doi.org/10.3390/cells10113003.

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Mitochondria are one of organelles that undergo significant changes associated with senescence. An increase in mitochondrial size is observed in senescent cells, and this increase is ascribed to the accumulation of dysfunctional mitochondria that generate excessive reactive oxygen species (ROS). Such dysfunctional mitochondria are prime targets for ROS-induced damage, which leads to the deterioration of oxidative phosphorylation and increased dependence on glycolysis as an energy source. Based on findings indicating that senescent cells exhibit mitochondrial metabolic alterations, a strategy to induce mitochondrial metabolic reprogramming has been proposed to treat aging and age-related diseases. In this review, we discuss senescence-related mitochondrial changes and consequent mitochondrial metabolic alterations. We assess the significance of mitochondrial metabolic reprogramming for senescence regulation and propose the appropriate control of mitochondrial metabolism to ameliorate senescence. Learning how to regulate mitochondrial metabolism will provide knowledge for the control of aging and age-related pathologies. Further research focusing on mitochondrial metabolic reprogramming will be an important guide for the development of anti-aging therapies, and will provide novel strategies for anti-aging interventions.
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Berezina, T. N., A. V. Litvinova, and A. A. Zinatullina. "Interrelation of Individual-Personal Anti-Aging Strategies with Biological Age." Современная зарубежная психология 11, no. 4 (2022): 73–89. http://dx.doi.org/10.17759/jmfp.2022110407.

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<p>The definition of anti-aging is given as a condition that reduces the biological age, improves health or increases life expectancy. Based on the meta-analysis, 13 anti-aging strategies were identified: sports, control, creative, intellectual, subject, altruistic, humor, self-improvement, risk, communication, interaction with nature, achievement, optimism. An empirical study of the effectiveness of these strategies has been carried out. Subjects: persons of retirement age, men &mdash; 61&mdash;70, women &mdash; 56&mdash;70 years. The following methods were used: diagnostics of biological age according to Voitenko, questionnaire of personal resources, assessment of individual typological features, correlation analysis. It was found out that the relationship of biological aging with personal resources depends on the socio-demographic characteristics of the individual. Conclusions: to develop an individual-personal anti-aging strategy, it is necessary to take into account the totality of data: gender, age, place of residence, family, children, physique, emotionality, functional asymmetry, interaction style. An effective anti-aging strategy is selected individually for each respondent.</p>
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Ahmed, Amr. "Insulin Sensitizers as Anti-Aging Agents: Unveiling Synergies with Albumin, GLP-1RA, Klotho Protein, and Metformin in the Quest to Combat Aging." Cell & Cellular Life Sciences Journal 9, no. 1 (2024): 1–5. http://dx.doi.org/10.23880/cclsj-16000200.

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This study delves into the synergistic interplay among albumin, insulin, Klotho protein, and relevant medications in the pursuit of a concerted approach to halt aging. Emphasizing albumin's pivotal role in various physiological functions, including transportation and drug distribution, the research underscores its decline's correlation with aging-related cognitive implications. The intricate relationship between insulin and albumin, modulated by Foxo1, underscores its crucial significance. Groundbreaking experiments, utilizing unmodified serum albumin, demonstrate a remarkable increase in lifespan and enhanced physical capabilities, highlighting the potential of an integrative approach. The investigation extends to insulin sensitizers, Klotho protein, metformin, and SGLT2 inhibitors, collectively revealing promising anti-aging effects. The association between Klotho protein and albumin suggests a collaborative mechanism with implications for efficient transport and distribution. This research offers insights into a comprehensive, synergistic strategy harnessing the potential of these elements to counteract aging processes.
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Zimmermann, Andreas, Katharina Kainz, Sebastian J. Hofer, Maria A. Bauer, Sabrina Schroeder, Jörn Dengjel, Federico Pietrocola, et al. "Targeting GATA transcription factors – a novel strategy for anti-aging interventions?" Microbial Cell 6, no. 5 (May 6, 2019): 212–16. http://dx.doi.org/10.15698/mic2019.05.676.

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Bárcena, Clea, Carlos López-Otín, and Guido Kroemer. "Methionine restriction for improving progeria: another autophagy-inducing anti-aging strategy?" Autophagy 15, no. 3 (October 18, 2018): 558–59. http://dx.doi.org/10.1080/15548627.2018.1533059.

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Dissertations / Theses on the topic "Anti-aging strategy"

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Halkoum, Rym. "Rôle du glyoxal dans la sénescence cellulaire : implications dans le vieillissement de la peau." Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS016.pdf.

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La sénescence est une réponse cellulaire associée à des marqueurs spécifiques comme un arrêt irréversible du cycle cellulaire ainsi que la sécrétion d’un ensemble de facteurs comme les cytokines, chimiokines et protéases, constituant le SASP, pour Senescence-Associated Secretory Phenotype. Le SASP peut avoir des rôles autocrine et paracrine qui contribuent au renforcement et à la propagation du phénotype sénescent. La composition du SASP et par conséquent son rôle, dépendent notamment du type cellulaire et de la nature du stress inducteur de sénescence. Du fait de sa fonction de barrière avec l’environnement externe, la peau est particulièrement soumise à différents types de stress induisant la sénescence des cellules et un vieillissement prématuré. Le glyoxal, composé dicarbonylé formé au cours des réactions de glycation, d’auto-oxydation du glucose ou de la peroxydation lipidique, est un précurseur des produits avancés de glycation impliqués dans le vieillissement normal et pathologique. Mes travaux de thèse ont permis de montrer que le glyoxal induit la sénescence de kératinocytes humains normaux ainsi qu’une accumulation d’espèces réactives de l’oxygène et de produits avancés de glycation intracellulaires traduisant un état de stress oxydant. L’initiation de cette sénescence est due à l’activation de la voie d’arrêt du cycle cellulaire AKT/FOXO3a/p27KIP1 suivie d’une phase tardive caractérisée par l’activation de la voie p16INK4A/pRb. La caractérisation du phénotype sécrétoire associé à la phase précoce de cette sénescence, a été réalisée par spectrométrie de masse afin d’identifier des facteurs pouvant être ciblés par des ingrédients sénomorphiques
Senescence is a well-characterized cellular state associated with specific markers such as permanent cell proliferation arrest, and the secretion of messenger molecules by cells expressing the Senescence-Associated Secretory Phenotype (SASP). The SASP can display autocrine and paracrine effects which contribute to the senescent phenotype reinforcement and propagation. The SASP composition depends on many factors such as the cell type or the nature of the stress that induces senescence. Since the skin constitutes a barrier with the external environment, it is particularly subjected to different types of stresses, and consequently prone to premature cellular aging. Glyoxal, a dicarbonyl compound produced during glucose metabolism and lipid peroxidation, is a precursor of Advanced Glycation End-products (AGEs), whose presence marks normal and pathological aging. My thesis work showed that glyoxal treatment provokes oxidative stress by increasing reactive oxygen species and AGEs levels and induce senescence in human keratinocytes. Furthermore, glyoxal-induced senescence bears a unique molecular progression profile: an “early-stage” when AKT-FOXO3a-p27KIP1 pathway mediates cell-cycle arrest, and a “late-stage” senescence maintained by the p16INK4/pRb pathway. Moreover, we characterized the resulting secretory phenotype during early senescence by mass spectrometry in order to find new targets for senomorphic ingredients. Our study provides evidence that glyoxal can affect keratinocyte functions and act as a driver of human skin aging
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Yang, Nai-fang, and 楊迺芳. "The Competitive Strategy for the Anti-aging Maintenance Company in Japan: The Case Study of Saishunkan Co. Ltd." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/60862649688617793827.

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碩士
國立中山大學
國際經營管理碩士班
99
According to the Yearbook of Chemical Industry Statistics of Japan''s Ministry of Economy, Trade and Industry, the total sales of domestic cosmetics market in 2008 was approximately 1,507. 1 billion yen, slightly less than that of 2007. In Japan''s mature cosmetics market, however, the sales of anti-aging skin care products has been growing every year. The reasons are the arrival of an aging society and females'' increasing emphasis on their appearances. There are over 1,200 cosmetics manufacturers in Japan. The distribution of these companies according to their sizes appears to be M-shaped. The top five—SHISEIDO, KAO, Kanebo, Kose, and P&G Japan, have been in heated competition for years, but it does not stop new entrants from joining the battlefield. In the face of fierce competition among the cash-rich giants and the threats of aggressive new entrants, the smaller-scaled companies that lie in between are facing daunting challenges. This study intends to show how such medium-sized companies might come up with a competitive strategy to ensure their survival and profitability. Saishunkan Co. Ltd. is one of those medium-sized cosmetics manufacturers. Saishunkan has modest capital amount of 100 million yen, yet it is able achieve annual sales of over 20 billion yen. The company has unique ways of marketing and sales, clearly targeted customers, products with special ingredients, and a sound market segmentation strategy. This thesis intends to examine, through the case study of Saishunkan, how a smaller-scaled company might compete and survive in a theater filled with cash-rich big companies who enjoy the economy of scale.
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蕭宸婕. "A Strategic Services Approach of Taiwan's Anti-aging Cosmeceutical Industry." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/53413332881037019285.

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碩士
國立交通大學
科技管理研究所
93
This study utilizes the Innovation Intensive Services model to process a strategic approach of Taiwan’s anti-aging cosmeceutical Industry. The IIS model entails a 2-dimensional analysis, containing 4 service packages and 5 innovation fields. Three research methods used for data collection are literature review, professional interview and questionare survey. The IIS model provides a 4╳5 matrix with 20 service cluster positions to analyze the strategic requirements of Taiwan’s anti-aging cosmeceutical service. Potential strategy position and future trends are also investigated in this thesis. Our results indicate that Taiwan’s anti-aging cosmeceutical firms must forward integrate into marketing and servicing to develop capabilities in respectively areas. Results reveal that current position is at restricted service/structural innovation. The future direction is at selective service/market innovation. Results also point out Taiwan’s anti-aging cosmeceutical services must reinforce its service value activities – Modelizing and Holding Innovation Skills, Flexible Servicing Efficiency, Brand and Marketing Ability, Servicing Convey Ability, Channel Service Ability, Financial Management and its externalities – Goodwill, Skill Merchantdizing, Value-chain Integrating Ability, Customer Conducting Interface, Channel Management.
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Book chapters on the topic "Anti-aging strategy"

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López-Lluch, Guillermo. "Coenzyme Q as an Antiaging Strategy." In Emerging Anti-Aging Strategies, 17–39. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7443-4_2.

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Bhoumik, Sukanya, Arun Kumar Yadawa, Parisha Srivastava, and Syed Ibrahim Rizvi. "Intermittent Fasting as an Anti-Aging Strategy." In Emerging Anti-Aging Strategies, 191–206. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7443-4_10.

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Nampoothiri, Madhavan, Kiran Kumar Kolathur, Runali Sankhe, and Sairaj Satarker. "Spermidine, an Autophagy Inducer, as a Therapeutic Antiaging Strategy." In Emerging Anti-Aging Strategies, 135–53. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7443-4_8.

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Singh, Abhishek Kumar, Sandeep Singh, and Syed Ibrahim Rizvi. "Autophagy Induction: A Promising Antiaging Strategy." In Molecular Basis and Emerging Strategies for Anti-aging Interventions, 161–74. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1699-9_11.

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Liang, Yaru, and Zhao Wang. "Which is the Most Reasonable Anti-aging Strategy: Meta-analysis." In Advances in Experimental Medicine and Biology, 267–82. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1117-8_17.

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Saraswat, Komal, Raushan Kumar, and Syed Ibrahim Rizvi. "Inhibition of mTOR Signalling: A Potential Anti-aging Drug Strategy." In Molecular Basis and Emerging Strategies for Anti-aging Interventions, 151–60. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1699-9_10.

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Pandey, Kanti Bhooshan, and Syed Ibrahim Rizvi. "Activation of Plasma Membrane Redox System: A Novel Antiaging Strategy." In Molecular Basis and Emerging Strategies for Anti-aging Interventions, 297–304. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1699-9_19.

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Singh, Bhisham Narayan, Anubha Joshi, Sarada Prasanna Mallick, and Pradeep Srivastava. "Tissue Engineering and Regenerative Medicine: A Translational Research for Antiaging Strategy." In Molecular Basis and Emerging Strategies for Anti-aging Interventions, 47–66. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1699-9_4.

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Yanar, Karolin. "Novel Classification Perspective of Geroprotective and Senolytic Drugs as an Antiaging Strategy." In Molecular Basis and Emerging Strategies for Anti-aging Interventions, 83–96. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1699-9_6.

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Lin, Chih-Li. "Mitophagy and mitohormetics: promising antiaging strategy." In Anti-Aging Drug Discovery on the Basis of Hallmarks of Aging, 279–89. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-323-90235-9.00001-x.

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Conference papers on the topic "Anti-aging strategy"

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Altman, Allison R., Beom Kang Huh, Abhishek Chandra, Wei-Ju Tseng, Ling Qin, and X. Sherry Liu. "3D In Vivo Bone Dynamic Imaging of PTH’s Anabolic Action." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14671.

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Aging shifts bone remodeling toward a negative balance between bone formation and resorption, causing bone loss and increased fracture risk. Anti-resorptive agents are commonly used to inhibit bone resorption and stabilize bone mass. While they are effective to prevent further bone loss, there is also a great need for anabolic agents which can reverse bone deterioration and regain lost skeletal integrity. Intermittent parathyroid hormone (PTH) treatment is the only FDA-approved anabolic treatment for osteoporosis, which greatly stimulates bone formation. Combined therapy of anti-resorptive drugs, such as alendronate (ALN), and PTH have been proposed and are expected to further stimulate bone formation. However, studies show conflicting results regarding the effectiveness of combined treatments: some have reported the addition of ALN to impair PTH function [1, 2], while others suggest an improvement over PTH monotherapy [3, 4]. The first objective of this study is to document the immediate changes of individual trabecular structures due to PTH and combined therapy within 12 days using in vivo micro computed tomography (μCT). As PTH is typically prescribed for 1 to 3 years to osteoporotic patients, a treatment of 12 days for rats (approximately equivalent to one year of human life) may be more clinically relevant than long-term treatment studies on rats. The secondary purpose of this study was to gain insight into the mechanism of combined versus PTH treatments through a bone dynamic imaging strategy to track events over an individual remodeling site. We hypothesized that PTH and combined treatments would immediately enhance bone formation on the trabecular surface.
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Yeger-McKeever, Meira, Alice H. Huang, Ashley F. Stein, and Robert L. Mauck. "Engineered MSC-Laden Cartilage Constructs are Sensitive to Inflammatory Cytokine-Mediated Degradation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176186.

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Degeneration of cartilage resulting from trauma or disease processes is an increasingly prevalent problem in the aging population. Intrinsic repair of cartilage is limited and few methodologies exist, short of prosthetic replacement, for restoring damaged articular surfaces. These realities engender a need for new strategies for extrinsic repair. One strategy, tissue engineering, generates replacement cartilage composed of scaffolds and differentiated chondrocytes [1]. In addition to chondrocytes, recent work has demonstrated that mesenchymal stem cells (MSCs) isolated from bone marrow may be induced to take on a chondrocyte-like phenotype [2]. Tissue engineered constructs of either cell type can yield near-native properties (though those derived from MSCs are typically lower) [3]. While such constructs may be surgically implanted to replace areas denuded of cartilage, one factor to consider is that these defect sites exist in an already inflamed joint [4]. In addition, surgical interventions trigger further inflammatory responses, greatest in the area of intervention [5]. Recent literature has shown that pro-inflammatory cytokines, such as interleukin-1beta (IL-1β), instigate catabolic destruction not only of cartilage explants, but also of chondrocyte-based engineered cartilage constructs [6–9]. Still other studies have shown that un-differentiated MSCs themselves may exert an anti-inflammatory effect on their local environment [10–12]. Thus the current study examined the effect of varying doses of IL-1β exposure on both chondrocyte- and MSC-based engineered cartilage constructs to determine the relative sensitivity of neo-cartilage derived from each cell type to cytokine induced degradation.
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