Relevant bibliographies by topics / Anosognosia for spatial neglect

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Academic literature on the topic 'Anosognosia for spatial neglect'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Anosognosia for spatial neglect"

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Langer, Karen G., and Julien Bogousslavsky. "The Merging Tracks of Anosognosia and Neglect." European Neurology 83, no. 4 (2020): 438–46. http://dx.doi.org/10.1159/000510397.

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Anosognosia and hemineglect are among the most startling neurological phenomena identified during the 20th century. Though both are associated with right hemisphere cerebral dysfunction, notably stroke, each disorder had its own distinct literature. Anosognosia, as coined by Babinski in 1914, describes patients who seem to have no idea of their paralysis, despite general cognitive preservation. Certain patients seem more than unaware, with apparent resistance to awareness. More extreme, and qualitatively distinct, is denial of hemiplegia. Various interpretations of pathogenesis are still deliberated. As accounts of its captivating manifestations grew, anosognosia was established as a prominent symbol of neurological and psychic disturbance accompanying (right-hemisphere) stroke. Although reports of specific neglect-related symptomatology appeared earlier, not until nearly 2 decades after anosognosia’s inaugural definition was neglect formally defined by Brain, paving a path spanning some years, to depict a class of disorder with heterogeneous variants. Disordered awareness of body and extrapersonal space with right parietal lesions, and other symptom variations, were gathered under the canopy of neglect. Viewed as a disorder of corporeal awareness, explanatory interpretations involve mechanisms of extinction and perceptual processing, disturbance of spatial attention, and others. Odd alterations involving apparent concern, attitudes, or belief characterize many right hemisphere conditions. Anosognosia and neglect are re-examined, from the perspective of unawareness, the nature of belief, and its baffling distortions. Conceptual parallels between these 2 distinct disorders emerge, as the major role of the right hemisphere in mental representation of self is highlighted by its most fascinating syndromes of altered awareness.
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Barrett, A. M. "Spatial Neglect and Anosognosia After Right Brain Stroke." CONTINUUM: Lifelong Learning in Neurology 27, no. 6 (December 2021): 1624–45. http://dx.doi.org/10.1212/con.0000000000001076.

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Chen, Peii, and Joan Toglia. "Online and offline awareness deficits: Anosognosia for spatial neglect." Rehabilitation Psychology 64, no. 1 (February 2019): 50–64. http://dx.doi.org/10.1037/rep0000207.

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Mizuno, Katsuhiro, Kengo Tsujimoto, and Tetsuya Tsuji. "Effect of Prism Adaptation Therapy on the Activities of Daily Living and Awareness for Spatial Neglect: A Secondary Analysis of the Randomized, Controlled Trial." Brain Sciences 11, no. 3 (March 9, 2021): 347. http://dx.doi.org/10.3390/brainsci11030347.

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Background: Rehabilitation for unilateral spatial neglect (USN) using prism adaptation (PA) is one of the most widely used methods, and the effectiveness of PA is well-evidenced. Although the effect of PA generalized various neglect symptoms, the effectiveness for some aspects of neglect is not fully proven. The Catherine Bergego Scale (CBS) was developed to identify problems with the activities of daily living (ADL) caused by USN. The CBS is composed of 10 observation assessments and a self-assessment questionnaire. To assess the self-awareness of USN, the anosognosia score is calculated as the difference between the observational scores and the self-assessment scores. To investigate how PA affects ADL and self-awareness in subacute USN patients during rehabilitation, we analyzed each item of the CBS and self-awareness from a randomized, controlled trial (RCT) that we previously conducted (Mizuno et al., 2011). Methods: A double-masked randomized, controlled trial was conducted to evaluate the effects of a 2-week PA therapy on USN in 8 hospitals in Japan. We compared each item of the CBS, anosognosia score, and absolute value of the anosognosia score between the prism group and the control group. Results: Two of ten items (gaze orientation and exploration of personal belongings) were significantly improved in the prism group compared with those in the control group. The absolute value of the anosognosia score was significantly improved by PA. Conclusions: Improvement of oculomotor exploration by PA may generalize the behavioral level in a daily living environment. This study suggested that PA could accelerate the self-awareness of neglect during subacute rehabilitation.
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Hartman-Maeir, Adina, Nachum Soroker, and Noomi Katz. "Anosognosia for Hemiplegia in Stroke Rehabilitation." Neurorehabilitation and Neural Repair 15, no. 3 (September 2001): 213–22. http://dx.doi.org/10.1177/154596830101500309.

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Objective: The purpose of this study was to investigate anosognosia for hemiple gia (AHP) in the rehabilitation phase after onset of stroke. Methods: Forty-six hemi plegic stroke patients, 29 with right hemisphere damage (RHD) and 17 with left hemi sphere damage (LHD) were evaluated ∼1 month after onset of stroke. Anosognosia was evaluated with an implicit measure designed to assess anosognosic behaviors (choosing between unimanual and bimanual tasks), in addition to a traditional ex plicit verbal measure. Results: AHP was found m 28% of the RHD and 24% of the LHD group. The majority of patients with AHP in the RHD group had large lesions involving the frontal, parietal, or temporal lobes and had coexisting sensory deficits and unilateral spatial neglect, whereas the LHD patients with AHP had predominantly small subcortical lesions and no sensory or attentional deficits. The functional out comes of AHP patients in both hemisphere groups revealed their inability to retain safety measures at discharge from rehabilitation (p < 0.036) and their need for assis tance in basic and instrumental activities of daily living at follow-up. Conclusions: AHP presents a significant risk for negative functional outcome in stroke rehabilita tion. The underlying mechanisms of AHP may be different for left and right hemi sphere patients, therefore requiring different intervention approaches. Key Words: Anosognosia—Cerebrovascular accident—Rehabilitation outcome.
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Vossel, Simone, Peter H. Weiss, Philipp Eschenbeck, Jochen Saliger, Hans Karbe, and Gereon R. Fink. "The Neural Basis of Anosognosia for Spatial Neglect After Stroke." Stroke 43, no. 7 (July 2012): 1954–56. http://dx.doi.org/10.1161/strokeaha.112.657288.

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Facchin, Alessio, and Nicoletta Beschin. "Different impact of prism adaptation rehabilitation in spatial neglect and anosognosia for hemiplegia." Annals of Physical and Rehabilitation Medicine 61, no. 2 (March 2018): 113–14. http://dx.doi.org/10.1016/j.rehab.2017.12.007.

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Heilman, K. M., A. M. Barrett, and J. C. Adair. "Possible mechanisms of anosognosia: a defect in self–awareness." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 353, no. 1377 (November 29, 1998): 1903–9. http://dx.doi.org/10.1098/rstb.1998.0342.

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Anosognosia of hemiplegia is of interest for both pragmatic and theoretical reasons. We discuss several neuropsychological theories that have been proposed to explain this deficit. Although for psychological reasons people might deny deficits, the denial hypothesis cannot account for the hemispheric asymmetries associated with this disorder and cannot explain why some patients might deny one deficit and recognize another equally disabling deficit. There is some evidence that faulty feedback from sensory deficits, spatial neglect and asomatognosia might be responsible for anosognosia in some patients. However, these feedback hypotheses cannot account for anosognosia in all patients. Although the hemispheric disconnection hypothesis is appealing, disconnection is probably only a rare cause of this disorder. The feedforward intentional theory of anosognosia suggests that the discovery of weakness is dependent on attempted action and some patients might have anosognosia because they do not attempt to move. We present evidence that supports this theory. The presence of one mechanism of anosognosia, however, does not preclude the possibility that other mechanisms might also be working to produce this disorder. Although a large population study needs to be performed, we suspect that anosognosia might be caused by several of the mechanisms that we have discussed. On the basis of the studies of impaired corporeal self–awareness that we have reviewed, we can infer that normal self–awareness is dependent on several parallel processes. One must have sensory feedback and the ability to attend to both one's body and the space where parts of the body may be positioned or acting. One must develop a representation of the body, and this representation must be continuously modified by expectations (feedforward) and knowledge of results (feedback).
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Othman, Makram, Leila Massoud, Fatma Kamoun, Chahnez Triki, and Khadija Moalla. "Unusual Presentation of Childhood Reversible Angiopathy: Aphasia due to Acute Bilateral Paramedian Thalamic Infarct." Journal of Pediatric Neurology 16, no. 04 (March 9, 2018): 248–52. http://dx.doi.org/10.1055/s-0038-1637722.

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AbstractAn 8-year-old right-handed girl manifested aphasia after bilateral thalamic infarcts. The features of thalamic aphasia are similar to that of previously reported patients with thalamic lesions. Paucity of speech, reduced voice volume, some paraphasia, and severe dysgraphia were present, but comprehension and repetition were preserved. Thalamic aphasia is usually associated with left thalamic lesions. Our patient also had spatial neglect and anosognosia probably due to right thalamic infarction. She had recovered near-normal speech after rehabilitation.
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SASAKI, Tomo, and Masaharu MAEDA. "Association of Lesions and the Pathogenesis of Anosognosia in Hemiplegia in Cases Involving Neither Somatosensory Disturbance nor Unilateral Spatial Neglect." Rigakuryoho Kagaku 36, no. 1 (2021): 101–6. http://dx.doi.org/10.1589/rika.36.101.

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Dissertations / Theses on the topic "Anosognosia for spatial neglect"

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RONCHI, ROBERTA. "Behavioural monitoring disorders in unilateral spatial neglect: productive symptoms and impaired awareness of disease." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2011. http://hdl.handle.net/10281/19801.

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This doctoral thesis investigates the relationships between disorders of behavioural monitoring (including “productive” behaviours and unawareness of neuropsychological deficits) and unilateral spatial neglect in right-brain-damaged patients. One main monitoring disorder is recurrent perseveration, namely a “productive” motor symptom frequently found in target cancellation tasks: we demonstrate, in two specific tasks (Experiments 1 and 2), that the disposition of the stimuli and the type of target modulate its severity. Neglect patients showing perseveration in visuo-motor exploratory tests perseverate also in drawing tasks. No correlation between omission and perseveration errors is found, supporting the functional independence of the two deficits. In the context of a two-component hypothesis, perseveration (the first component) is a specific disorder that manifests in a variety of tasks, particularly those requiring serial graphic production; unilateral spatial neglect (the second component) may trigger and facilitate the production of perseveration errors. Moreover results indicate that patients with perseveration are not disproportionately impaired in tasks assessing executive, visuo-spatial short-term memory, and attentional functions, suggesting the specificity of the monitoring disorder associated with spatial neglect. Lesion analysis indicates damage to the right insula as a relevant neural correlate of perseverative behaviour. Experiment 3 shows that perseverating patients produce a majority of substitution errors during a word reading task, suggesting that also this type of paralexic neglect error can be considered a “productive” manifestation. The clinical, experimental and neural features of another monitoring deficit and “positive” manifestation referring to the personal space, “somatoparaphrenia”, are reviewed: somatoparaphrenia is a symptom usually associated with right-sided hemispheric lesions, most often characterized by a delusion of disownership of contralesional body parts. Possible pathological factors may include a deranged representation of the body concerned with ownership, mainly right-hemisphere-based, and deficits of multisensory integration. Finally, Experiment 4 investigates anosognosia for unilateral spatial neglect by a quantitative assessment. Results indicate that unawareness for spatial attentional and representational deficits is not a pervasive disorder, and that some tasks can evoke different degrees of awareness. In addition, the scores assigned by neglect patients to their performance in spatial tasks are not modulated by the different conditions of the estimation tasks. In conclusion, we demonstrate that: 1) “productive”, as “defective”, manifestations of unilateral spatial neglect are multifarious; 2) these “positive” phenomena are independent of general executive deficits and of the severity of the spatial neglect syndrome; 3) the neural bases of motor productive disorders included the right insula; 4) neglect patients are not globally anosognosic about their spatial defective performances.
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Campbell, Ransom Whitaker. "Dynamic Fluctuations in Emotion and Space Representation: A Functional Cerebral Systems Approach to Right Hemisphere Dysfunction." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/100952.

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This study proposed an experimental test of theoretical models related to emotion and space representation in the brain. Previous research has established that emotion is represented, processed, expressed, and regulated largely by the right hemisphere. Furthermore, there is evidence from experimental paradigms and clinical case reports to suggest that the same hemisphere plays a dominant role in the processing of external space. A conceptual difficulty of clinical and neural network overlap arises when right hemisphere disorders of emotion are compared with those of spatial representation. The current experiment tested some of these hypotheses about emotion regulation and spatial representation in the right hemisphere using nonclinical subjects under a cortical stress paradigm designed to mimic the conditions of cortical duress. An additional goal was an extension of a previous study that examined emotional influence on spatial orientation. Results did not support our initial hypotheses. Subsequent analyses did provide some evidentiary support for some theories related to emotion and brain function. Additionally, patterns of subject performance were observed that support traditionally held theories of differential hemispheric function with regard to emotion and spatial behavior. These findings are discussed within the context of theories of emotion, spatial function, and disorders secondary to right hemisphere damage.
Doctor of Philosophy
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Gerafi, Joel. "A Unified Perspective of Unilateral Spatial Neglect." Thesis, Högskolan i Skövde, Institutionen för kommunikation och information, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-6095.

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The aim of this review is to provide a unified perspective of unilateral spatial neglect (USN). USN is a neurological disorder frequently observed following damage or diseases to the brain. It is particularly associated with strokes to specific anatomical structures within the right hemisphere. Patients with USN fail to respond to or orient towards stimuli located in the hemispace contralateral to the lesion. They also show peculiar behavioral manifestations. There are several distinct subtypes of USN which can affect sensory or motor modalities, spatial representations, the range of space, or pure imagery. This disorder can appear in any sensory modality but the majority of studies have investigated the visual aspect of USN in these subtypes. Theoretical proposals are supported by empirical evidence deriving from neuroimaging which distinguish between these subtypes of USN. Thus, the heterogeneity of the disorder is evident and clinical assessment methods face great difficulties while prevalence rates vary. The neural pathways of spatial attention distinguish between the ventral and dorsal visual streams, both with distinct functional roles and anatomical bases. Prism adaptation (PA) is a common rehabilitation technique among many others and has shown positive effects on USN while having some limitations. A general discussion and concluding remarks are presented in the final section followed by future research suggestions.
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Walker, Robin. "Visual attention with implications for unilateral spatial neglect." Thesis, Durham University, 1992. http://etheses.dur.ac.uk/5611/.

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Recent models of visual attention (eg. Rizzolatti et al., 1987) have suggested that a similar system orients visual attention as is used to produce a saccadic eye movement. This thesis provides further support for the link between the attentional and eye orienting systems and has incorporated ideas from recent models of saccade generation. The time taken by normal subjects to initiate a saccade ('latency'), is examined in Chapters two, three and four. Subjects were given attentional instructions and saccades made to either: unilateral single, or, bilateral double, targets. Latency to attended targets was not greatly enhanced, while latency to non-attended targets was greatly slowed. The results support both the premotor model of visual attention and models of visual attention that emphasise the inhibitory consequence of directed attention. Bilateral double targets produced an additional slowing on saccade latency, which could reflect a further automatic attentional inhibition produced in the contralateral field by the stimulus onset. Fixation point offset (in 'gap' situations) is known to reduce saccade latency, which has been attributed to prior attentional disengagement (Fischer, 1987). In Chapter two, the use of a gap situation produced a generalised speeding which was independent of the effects of directed visual attention. This suggests that active fixation affects a separate component to that involved in orienting visual attention to a spatial location. This idea is incorporated into a model which emphasises the inhibitory consequences of attentive fixation. Chapters six and seven report the findings from an experimental investigation of a patient (B.Q.) with a 'unilateral spatial neglect', a condition often attributed to a deficit of visual attention. The 'gap' paradigm was shown to be effective at reducing the severity of B.Q.'s contralateral neglect. In contrast to normal subjects, bilateral double targets did not have an inhibitory effect on her saccade latency. These findings are explained in terms of a model that neglect results in part from the loss of attentional inhibition for the ipsilesional side of space and in part an inability to switch off contralesional inhibition produced during active fixation. A functional model is proposed in Chapter eight to account for the findings. This supports the close link between the attentional orienting and saccade programming systems. An additional implication of the findings is that models of visual attention and saccade generation need to consider the inhibitory consequences of directing attention to a spatial location.
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FORTIS, PAOLA. "Prism adaptation: a rehabilitation technique for spatial neglect." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/28396.

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This doctoral thesis is divided in six chapters and presents the results of four experiments. It investigates the use of prism adaptation (PA) in the rehabilitation of spatial neglect, how PA may affect spatial cognition, and the specific mechanisms that are influenced by PA. Knowing more about the systems responsive to PA may help our understanding of which symptoms, and which patients, improve optimally after PA training. The Introduction explains concepts and backgrounds useful for the understanding of the experimental projects. First, the definition and characteristics of Unilateral Spatial Neglect are introduced, followed by a review of the distinction in the variety of spatial neglect that separates “perceptual” versus “premotor” neglect and the anatomical and functional dissociation between brain areas associated with these two subtypes of neglect. The Introduction summarizes paradigms that have been used to disentangle the perceptual and premotor components of spatial neglect and the patterns of perceptual and premotor biases reported in neglect patients. A description of different methods that have previously been used in the rehabilitation of spatial neglect is also provided. Among them, the PA technique is described in detail, including the background in which PA has been used, measures to assess the presence of adaptation and aftereffects following the adaptation procedure, processes that have been proposed to be involved in the sensori-motor transformation that occurs during PA, the beneficial effects reported after PA paradigms in neglect patients, and the effects of PA in healthy individuals. Finally, the Introduction gives an overview of the cerebral circuits that appear to be involved in PA based on data derived from brain imaging studies, studies with brain-damaged patients, and studies with primates. The next four chapters report the results of four experiments. These chapters are divided in two main parts. The first section focuses on the feasibility of using ecological visuo-motor activities, based on diverse and engaging visuo-motor tasks, during adaptation to prism. In Experiment 1, 10 neglect patients were submitted to both a standard pointing adaptation training (Frassinetti et al., 2002) and a training involving diverse ecological visuo-motor tasks (Ecological procedure). The effect of the two treatments was compared in a large assessment including a variety of neuropsychological tests as well as functional scales. In Experiment 2, the presence of adaptation and aftereffects was assessed during the ecological procedure, and these measures were compared with those obtained during the traditional pointing task. In Experiment 3, we used a modified version of the paradigm of Schwartz et al. (1997) and Na et al., (1998) to decouple perceptual-attention “where” and motor-intention “aiming” components in visuo-motor tasks (line bisection). The effects of PA on where and aiming components were tested in a large group of neurologically healthy individuals. In Experiment 4, the same effects were tested in a group of five neglect patients. Lastly, the General Discussion summarizes and integrates the results of the four experiments, highlighting the implications for the rehabilitation of spatial neglect. Results from these experiments show that PA training associated with varied visuomotor activities is an effective tool to ameliorate some aspects of spatial neglect as well as functional disabilities, being as effective as the more established pointing task. In the four experiments, measures of adaptation and aftereffects were obtained using three different adaptation procedures: pointing, ecological, and line bisection tasks. It is argued that these measures, especially the aftereffect measures, may be important for establishing the effectiveness of adaptation procedures in neglect rehabilitation. It appears that the three adaptation procedures (pointing, ecological, line bisection) can all induce error correction during the exposure phase. However, the ecological and the pointing procedure seem to create strong and prolonged aftereffects, with the ecological task even better in inducing aftereffects than the pointing task. By contrast, the line bisection task appears to induce weaker aftereffects, suggesting that its use may not be optimal in prism paradigms. Reasons for such differences are explored, focusing on the motivations for the increased aftereffects during the ecological procedure. Indeed, exposure to prism decreased the aiming bias after PA in both studies. In the group of healthy individuals, the initial left aiming bias was reduced after exposure to left-shifting prisms (Experiment 3). In a similar way, in the group of neglect patients the initial right aiming bias improved after exposure to right-shifting prisms (Experiment 4). In addition, in the healthy participants no changes in the aiming bias were found after exposure to right-shifting prisms and control goggles, indicating that the effect of left-shifting prisms was not due to increased familiarity with the task (Experiment 3). These results are interpreted and integrated in light of recent findings. Experiments 1 and 4 also showed the cortical areas associated with neglect in our patients and the responsiveness to the PA paradigm. Results confirmed that patients with right-sided brain lesions in the frontal-parietal cortical and subcortical areas are still able to adapt to the lateral shift induced by prism, and further suggested that adaption to prism and improvement in neglect symptoms can occur even in the presence of an occipital lesion. Finally, the Discussion addresses the question of how to differentiate the effect of the experimental manipulation from spontaneous recovery while testing the efficacy of new rehabilitation methods in brain-damaged individuals. Therefore, evidence for the specificity of our intervention is provided. It is argued that spontaneous recovery cannot fully account for the present findings of improvement in neglect symptoms after PA treatment (Experiment 1 and 4). It is also suggested that performing studies employing neurologically healthy subjects can help in providing evidence for the effect of a treatment on cognitive function. In particular, testing healthy subjects to better understand the functioning of PA in neglect patients is facilitated by the fact that healthy individuals show biases in spatial cognition that mirror the biases in neglect patients.
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Menon, Anita. "Assessment of unilateral spatial neglect post stroke in acute care hospitals : are we neglecting neglect?" Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=80332.

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Timely and accurate assessment of unilateral spatial neglect (USN) post stroke is a critical component of best practice, given that a recent Clinical Practice Guidelines for stroke has recommended the standardized assessment of USN within 48 hours of regaining consciousness following a stroke. This multi-centered, retrospective study using data from medical charts of a representative sample of individuals admitted to 10 Ontario acute care hospitals from July 15th to December 15th 2002, examined the prevalence, timing and frequency of use of standardized assessments to evaluate USN post stroke. Out of the 248 subjects who should have received a USN assessment, 37.5% received an assessment; only 13.31% with a standardized visual perception tool and of these, only 0.81% (n = 2) with a standardized tool specific to USN assessment. All clients receiving a standardized assessment were evaluated for USN in the near extrapersonal space, the hemispace within reaching distance of the patient: no patient received a standardized assessment for USN in the personal space or far extrapersonal space. Three standardized visual perception tools that include a USN component were used: the Clock Drawing Test (n = 22), the Ontario Society of Occupational Therapists (OSOT) Perceptual Evaluation (n = 8) and the Motor-Free Visual Perception Test (n = 1). Only 8 (3.23%) of the 248 clients were screened with a standardized tool within the 2-day critical period as recommended by Stroke Guidelines. Reassessment was rare, even in those with detected USN, such that only 1 subject was ever reassessed with a standardized tool.
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Fisher, Zoe. "The effects of prism adaptation on unilateral spatial neglect." Thesis, Swansea University, 2009. https://cronfa.swan.ac.uk/Record/cronfa43074.

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This thesis concerns the syndrome of visual unilateral spatial neglect (USN). Pisella and Mattingley (2004) argue that two of the core deficits associated with USN, the ipsilesional reaction time (RT) bias (thought to reflect the ipsilesional attention bias) and the ipsilesional detection bias, may have distinct neural substrates and thus may dissociate. In the research reported in this thesis, the 'dissociation hypothesis' was explored in a single patient (JH). JH was impaired at detecting contralesional targets on the visual search task in near space but her far space contralesional target detection was comparable with a healthy control group. However, despite showing 'normal' target detection in far space her far space RTs were significantly slower to detect contralesional targets relative to ipsilesional targets. In fact her RT data was comparable with the far space RT data of 6 patients with USN and was significantly different that that of 10 healthy control participants. This data suggests that the ipsilesional RT and the ipsilesional detection bias can dissociate and may be underpinned by distinct neural processes. The patient was then given PA training. Overall, the analysis showed that the ipsilesional detection bias was ameliorated after PA but the ipsilesional RT gradient remained unchanged. A group study was carried out to explore whether PA ameliorated both the ipsilesional detection bias and the ipsilesional RT bias, (due to the limitations of the case study approach). As research has already shown that PA improves contralesional target detection (Rossetti et al., 1998; Frassinetti et al., 2002) the experiments asked whether increased target detection after PA is accompanied by a normalisation of the ipsilesional RT bias on a visual search task, as would be predicted if PA ameliorated USN by facilitating a redistribution of spatial attention (Rode, 2003; and Pisella, 1999). The findings showed that increased contralesional target detection was not accompanied by a normalisation of the ipsilesional RT gradient. This suggests that a) the ipsilesional detection and the ipsilesional RT bias are not intrinsically related and b) that PA does not facilitate a redistribution of spatial attention. It was argued that PA improves target detection by ameliorating the remapping deficit associated with PA (Pisella and Mattingley, 2004) without ameliorating the ipsilesional attentional bias. It was argued in the rationale section of this thesis that the conventional PA procedure described by Rossetti (1998) is a far space based procedure as patients adapt to the prismatic shift by pointing to objects in far space (beyond arm's reach). However, die patients in Rossetti's (1998) study, and subsequent studies by others, were asked to perform tasks that evaluated the effects of PA only in near space. The findings of Rossetti (1998) and others showed that 'far PA training' ameliorated left USN performance in near space, thereby suggesting that a common underlying mechanism involved in both near and far space processing is ameliorated by PA. A candidate for this mechanism may be the oculomotor system since research has shown that it may be involved in die detection of objects in both far and near space (Previc, 1995). Further, the oculomotor system has been implicated as being involved in the amelioration of USN after PA training (Serino et al., 2007). If it is the case that a mechanism common to processing of both near and far space is ameliorated by PA training, then near space training should also ameliorate USN of far space. On the other hand, there are indications in the literature that the oculomotor system is involved in processing of near and far space to different degrees, being more directly involved in processing of far than near space (Berti and Rizzolatti, 2002) There is also evidence that near and far space are processed by different neural circuits (Rizzolatti et al., 1987, 1985 and 2002). It is conceivable, therefore, that a PA training method based on processing of near space information would have a greater effect on neglect for near space than for far space. A 'near space' training procedure was therefore devised to explore this issue. Specifically, the experiments reported in Chapter 7 asked whether a 'near PA' procedure, which attempted to activate near/reaching circuits (in addition to the oculomotor system), would ameliorate USN in near and far space but to a greater degree in near space. The findings showed that 'near PA' significantly increased contralesional target detection in both near and far space with no enhanced benefit in near space. It was evident when carrying out die group study described above that not all patients benefited from PA training. The group data was re-analysed at a single case level and the findings showed that of the 9 patients who were given PA training only 6 showed a subsequent reduction in symptoms of USN. All of the patients who responded to PA showed error reduction and after effect. Of the patients who did not respond to PA, none showed error reduction and two showed an after effect It was concluded that error reduction and not after effect is the critical predictor of amelioration of USN symptoms after PA. A final longitudinal study explored how long the effects of PA lasted in three patients with USN who responded to PA training. The findings showed that the beneficial effects of PA were maintained for at least two years in one patient but two patients who initially benefited initially from PA lost their training gains over time.
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Malhotra, Paresh Arjun. "Spatial Working Memory and Sustained Attention in the Neglect Syndrome." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487565.

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Spatial neglect is most prominently associated with damage to the right cerebral hemisphere, especially parietal and frontal regions. Although many studies of the neglect syndrome have tended to focus on spatial pathological mechanisms, an increasing body ofdata points to right hemisphere involvement in spatial and nonspatial domains, including spatial working memory (SWM) and sustained attention. The experiments described here critically examine the roles ofSWM, sustained attention and their interaction in the neglect syndrome. By employing a vertical variant of the traditional Corsi blocks it was possible to demonstrate impaired SWM in neglect. A deficit was also found using a second 'purer' task, which required no manual response or memory for sequence order. Impairment on this task correlated with neglect severity, providing evidence for a contributory role for impaired SWM in neglect. Sustained attention was examined by assessing the ability to maintain attention to centrally-displayed visual stimuli. Patients with neglect were impaired, although there was no worsening ofperformance over time, or vigilance decrement. A subsequent study revealed that neglect patients have worsening performance over time only when maintaining attention towards spatial targets... suggesting a previously undescribed interaction between spatial impairment and sustained attention in neglect. Damage to white matter underlying parietal cortex was associated with both impaired SWM and sustained attention, consistent with evidence suggesting involvement for right fronto-parietal networks in both these domains. As impaired SWM and sustained attention appear to contribute to neglect, modulation of one or both of these might lead to improvement. A proof-of-principle trial using guanfacine, anoradrenergic agent previously shown to affect SWM and vigilance, demonstrated that, in selected patients, noradrenergic stimulation leads to improved search. Together, these studies detail the roles of impaired SWM and sustained attention in neglect, in addition to exploring a possible role' for targeted pharmacological treatment.
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Sullivan, Carol-Anne Maria. "The effects of lateralized stimulation on unilateral visuo-spatial neglect." Thesis, University of York, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341860.

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Murphy, Peter. "A study of hemispatial neglect in man and monkey." Thesis, University of Liverpool, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.290408.

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Books on the topic "Anosognosia for spatial neglect"

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Marc, Jeannerod, ed. Neurophysiological and neuropsychological aspects of spatial neglect. Amsterdam: North-Holland, 1987.

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W, Halligan Peter, and Marshall John C, eds. Spatial neglect: Position papers on theory and practice. Hove, East Sussex: Lawrence Erlbaum Associates, 1994.

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W, Halligan Peter, and Marshall John C, eds. Spatial neglect: Position papers on theory and practice. Hove, East Sussex: Lawrence Erlbaum Associates, 1994.

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1961-, Karnath H. O., Milner A. D, and Vallar Giuseppe, eds. The cognitive and neural bases of spatial neglect. Oxford, UK: Oxford University Press, 2002.

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Chen-Wing, Sara L. N. Development of an interface to enhance mobility for persons with hemi-spatial neglect. Ottawa: National Library of Canada, 1996.

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Peter W Halligan. Spatial Neglect. Psychology Press, 2014. http://dx.doi.org/10.4324/9781315804491.

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Vallar, Giuseppe, and Nadia Bolognini. Unilateral Spatial Neglect. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.012.

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Abstract:
Left unilateral spatial neglect is the most frequent and disabling neuropsychological syndrome caused by lesions to the right hemisphere. Over 50% of right-brain-damaged patients show neglect, while right neglect after left-hemispheric damage is less frequent. Neglect patients are unable to orient towards the side contralateral to the lesion, to detect and report sensory events in that portion of space, as well as to explore it by motor action. Neglect is a multicomponent disorder, which may involve the contralesional side of the body or of extra-personal physical or imagined space, different sensory modalities, specific domains (e.g. ‘neglect dyslexia’), and worsen sensorimotor deficits. Neglect is due to higher-order unilateral deficits of spatial attention and representation, so that patients are not aware of contralesional events, which, however, undergo a substantial amount of unconscious processing up to the semantic level. Cross-modal sensory integration is also largely preserved. Neglect is primarily a spatially specific disorder of perceptual consciousness. The responsible lesions involve a network including the fronto-temporo-parietal cortex (particularly the posterior-inferior parietal lobe, at the temporo-parietal junction), their white matter connections, and some subcortical grey nuclei (thalamus, basal ganglia). Damage to primary sensory and motor regions is not associated to neglect. A variety of physiological lateralized and asymmetrical sensory stimulations (vestibular, optokinetic, prism adaptation, motor activation), and transcranial electrical and magnetic stimulations, may temporarily improve or worsen neglect. Different procedures have been successfully developed to rehabilitate neglect, using both ‘top down’ (training the voluntary orientation of attention) and ‘bottom up’ (the above-mentioned stimulations) approaches.
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Spatial Neglect (Neuropsychological Rehabilitation). Psychology Press, 1994.

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Priftis, Konstantinos, Carlo Umiltà, Marco Zorzi, and Mario Bonato, eds. Spatial and Non-Spatial Aspects of Neglect. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-584-8.

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Rizzolatti, G. Neural mechanisms of spatial neglect. 1993.

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Book chapters on the topic "Anosognosia for spatial neglect"

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Bartolomeo, Paolo. "Unilateral Spatial Neglect: Clinical Aspects." In Attention Disorders After Right Brain Damage, 49–83. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5649-9_4.

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Bisiach, E. "The Spatial Features of Unilateral Neglect." In Parietal Lobe Contributions to Orientation in 3D Space, 465–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60661-8_26.

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Danckert, James. "Spatial Neglect: Not Simply Disordered Attention." In The Behavioral Consequences of Stroke, 71–94. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7672-6_5.

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Kerkhoff, Georg, Gilles Rode, and Stephanie Clarke. "Treating Neurovisual Deficits and Spatial Neglect." In Clinical Pathways in Stroke Rehabilitation, 191–217. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-58505-1_11.

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Vallar, Giuseppe, and Roberta Daini. "Visual perceptual processing in unilateral spatial neglect." In Imagery and Spatial Cognition, 337–62. Amsterdam: John Benjamins Publishing Company, 2006. http://dx.doi.org/10.1075/aicr.66.25val.

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Bottini, Gabriella, Martina Gandola, Lorenzo Pia, and Anna Berti. "The impairment of the body image in the unilateral neglect syndrome." In Imagery and Spatial Cognition, 363–79. Amsterdam: John Benjamins Publishing Company, 2006. http://dx.doi.org/10.1075/aicr.66.26bot.

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Jacquin-Ciourtois, Sophie, Jacinta O’Shea, Jacques Luauté, Laure Pisella, Alessandro Farné, Patrice Revol, Gilles Rode, and Yves Rossetti. "Prism Adaptation and the Rehabilitation of Spatial Neglect." In Systems Neuroscience and Rehabilitation, 81–104. Tokyo: Springer Japan, 2011. http://dx.doi.org/10.1007/978-4-431-54008-3_6.

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Silvetti, Massimo, Fabrizio Doricchi, and Eliano Pessa. "Simulating object-centred neglect with head-centred coding of space based on non-linear gaze-dependent units." In Imagery and Spatial Cognition, 381–94. Amsterdam: John Benjamins Publishing Company, 2006. http://dx.doi.org/10.1075/aicr.66.27sil.

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Rossa, Timothé, Pierre Pompidor, Nancy Rodriguez, Arnaud Sallaberry, Pascal Poncelet, Marika Urbanski, Clémence Bourlon, Antoine Seilles, and Guillaume Tallon. "Experimental Web Service and Eye-Tracking Setup for Unilateral Spatial Neglect Assessment." In Digital Human Modeling and Applications in Health, Safety, Ergonomics and Risk Management. Healthcare Applications, 141–55. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22219-2_11.

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Rao, Rajesh P. N., and Dana H. Ballard. "A Computational Model of Spatial Representations that Explains Object-Centered Neglect in Parietal Patients." In Computational Neuroscience, 779–85. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9800-5_121.

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Conference papers on the topic "Anosognosia for spatial neglect"

1

Fairhurst, M. C., R. M. Guest, N. Donnelly, J. Potter, A. Deighton, and M. Patel. "Analysing visuo-spatial neglect through figure-copying tasks." In IEE Third European Workshop on Handwriting Analysis and Recognition. IEE, 1998. http://dx.doi.org/10.1049/ic:19980676.

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Dvorkin, Assaf Y., William Z. Rymer, Krista Settle, and James L. Patton. "Perceptual Assessment of Spatial Neglect Within a Virtual Environment." In 2007 Virtual Rehabilitation. IEEE, 2007. http://dx.doi.org/10.1109/icvr.2007.4362161.

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Fuji, Akira, Makoto Fujimura, and Toshio Higashi. "Virtual Environment of Prism Adaptation for Unilateral Spatial Neglect." In 2018 IEEE 7th Global Conference on Consumer Electronics (GCCE). IEEE, 2018. http://dx.doi.org/10.1109/gcce.2018.8574783.

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Fairhurst, M. C., R. M. Guest, N. Donnelly, J. Potter, A. Deighton, and M. Patel. "Engineering software tools for assessment of visuo-spatial neglect." In IEE Colloquium on Intelligent Decision Support in Clinical Practice. IEE, 1998. http://dx.doi.org/10.1049/ic:19980788.

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Obo, Takenori, Kota Adachi, and Naoyuki Kubota. "Motion analysis for unilateral spatial neglect in computational system rehabilitation." In 2017 6th International Conference on Informatics, Electronics and Vision & 2017 7th International Symposium in Computational Medical and Health Technology (ICIEV-ISCMHT). IEEE, 2017. http://dx.doi.org/10.1109/iciev.2017.8338601.

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Buxbaum, L. J., M. Palermo, D. Mastrogiovanni, M. S. Read, E. Rosenberg-Pitonyak, A. A. Rizzo, and H. B. Coslett. "Assessment of Spatial Neglect with a Virtual Wheelchair Navigation Task." In 2006 International Workshop on Virtual Rehabilitation. IEEE, 2006. http://dx.doi.org/10.1109/iwvr.2006.1707534.

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Ferraro, Federica, Marco Trombini, Romina Truffelli, Marina Simonini, and Silvana Dellepiane. "On the Assessment of Unilateral Spatial Neglect via Digital Tests." In 2021 10th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2021. http://dx.doi.org/10.1109/ner49283.2021.9441471.

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Sugihara, Shunichi, Toshiaki Tanaka, Tomoya Miyasaka, Takashi Izumi, and Koichi Shimizu. "Assessment of visual space recognition in patients with unilateral spatial neglect using head mounted display (HMD) system: Case study with left unilateral spatial neglect." In 2013 IEEE/SICE International Symposium on System Integration (SII). IEEE, 2013. http://dx.doi.org/10.1109/sii.2013.6776681.

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Tsirlin, Inna, Eve Dupierrix, Sylvie Chokron, Theophile Ohlmann, and Sabine Coquillart. "Multimodal virtual reality application for the study of unilateral spatial neglect." In 2010 IEEE Virtual Reality Conference (VR). IEEE, 2010. http://dx.doi.org/10.1109/vr.2010.5444800.

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Hidayat, Sholeh Nurul, and Hendri Kurniawan. "EFFECT OF MIRROR THERAPY ON UNILATERAL SPATIAL NEGLECT IN STROKE PATIENTS." In The 8th International Conference on Public Health 2021. Masters Program in Public Health, Universitas Sebelas Maret, 2021. http://dx.doi.org/10.26911/ab.medicine.icph.08.2021.28.

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