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Relevant bibliographies by topics / Amyotrophic lateral sclerosis – Pathophysiology / Books

Books on the topic 'Amyotrophic lateral sclerosis – Pathophysiology'

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Author: Grafiati

Published: 10 December 2022

Last updated: 29 January 2023

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1

1949-, Chad David A., and Pioro Erik P. 1955-, eds. Amyotrophic lateral sclerosis. Philadelphia: F.A. Davis, 1998.

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2

Brown, Robert H., Michael Swash, and Piera Pasinelli. Amyotrophic Lateral Sclerosis. 2nd ed. London: CRC Press, 2021. http://dx.doi.org/10.1201/9781003076445.

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3

Cosi, V., Ann C. Kato, W. Parlette, P. Pinelli, and M. Poloni, eds. Amyotrophic Lateral Sclerosis. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5302-7.

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4

Clifford, Rose F., ed. Amyotrophic lateral sclerosis. New York, N.Y: Demos, 1990.

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5

G, Farrell Jason, ed. New amyotrophic lateral sclerosis research. New York: Nova Biomedical Books, 2008.

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6

1941-, Smith Richard Alan, ed. Handbook of amyotrophic lateral sclerosis. New York: Dekker, 1992.

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7

Motor neurone diseases: Amyotrophic lateral sclerosis. London: Chapman & Hall, 1995.

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8

Hiroshi, Mitsumoto, ed. Amyotrophic lateral sclerosis: A guide for patients and families. 3rd ed. New York, NY: Demos Health, 2009.

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9

Behind those eyes: Our journey with ALS. Renfrew, Ont: General Store Publishing House, 2013.

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10

Respiratory management of ALS: Amyotrophic lateral sclerosis. Sudbury, Mass: Jones and Bartlett, 2009.

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11

Guion, Lee. Respiratory management of ALS: Amyotrophic lateral sclerosis. Sudbury, Mass: Jones and Bartlett, 2010.

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12

Serratrice, Georges. Pathogenesis and therapy of amyotrophic lateral sclerosis. Philadelphia: Lippincott-Raven, 1995.

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13

Six parts love: One family's battle with Lou Gehrig's disease. New York: Scribner, 1985.

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14

Amyotrophic lateral sclerosis: Hearing before a subcommittee of the Committee on Appropriations, United States Senate, One Hundred Sixth Congress, second session, special hearing. Washington: U.S. G.P.O., 2001.

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15

ALS: Lou Gehrig's disease: Patient advocate. Riverside, CA: Health Scouter - Equity Press, 2009.

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16

Harris, Brent T., Galam A. Khan, and Saed Sadeghi. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0029.

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Although the basic gross and microscopic pathological changes in amyotrophic lateral sclerosis (ALS) have been known for more than 100 years, emerging technology and research into the cellular and molecular changes found in this disease are challenging our understanding about the pathogenesis and pathophysiology. All cell types of the CNS/PNS as well as circulating immune cells have been implicated in the pathology of ALS. Numerous genes, their proteins, and environmental factors have also been associated. However, we still do not understand the specific gene-environmental interactions that bring about and drive this devastating disease in most cases. This short chapter does not address the causal factors and molecular pathogeneses that have been hypothesized and actively researched in the pathology of ALS-as these are discussed in other sections of this text. Here, it shows and discusses the basic pathological changes at the tissue and cellular levels that help to establish the pathological diagnosis of ALS at autopsy.

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17

Jun, Kimura, and Kaji Ryuji, eds. Physiology of ALS and related diseases. Amsterdam: Elsevier, 1997.

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18

1936-, Eisen Andrew, ed. Clinical neurophysiology of motor neuron diseases. Amsterdam: Elsevier, 2004.

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19

Brown, Jr Robert H., Vincent Meininger, and Michael Swash. Amyotrophic Lateral Sclerosis. 2nd ed. Informa Healthcare, 2000.

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20

1952-, Brown Robert H., Meininger Vincent, and Swash Michael, eds. Amyotrophic lateral sclerosis. London: Martin Dunitz, 2000.

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21

MD, Brown Robert H., Swash Michael, and Pasinelli Piera, eds. Amyotrophic lateral sclerosis. 2nd ed. Abingdon [England]: Informa Healthcare, 2006.

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22

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. Amyotrophic lateral sclerosis. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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23

Hiroshi, Mitsumoto, Przedborski Serge, and Gordon Paul H, eds. Amyotrophic lateral sclerosis. Boca Raton: Taylor & Francis, 2006.

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24

Swash, Michael, Robert Brown, and Piera Pasinelli. Amyotrophic Lateral Sclerosis. Taylor & Francis Group, 2005.

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25

Swash, Michael, Robert Brown, and Piera Pasinelli. Amyotrophic Lateral Sclerosis. Taylor & Francis Group, 2006.

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26

Mitsumoto, Hiroshi, Serge Przedborski, and Paul H. Gordon. Amyotrophic Lateral Sclerosis. Taylor & Francis Group, 2005.

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27

Pagnini, Francesco, and Zachary Simmons, eds. Amyotrophic Lateral Sclerosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757726.001.0001.

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Amyotrophic lateral sclerosis: Understanding and optimizing quality of life and psychological well-being presents a comprehensive and up-to-date review of the enhancement of the lives of people with amyotrophic lateral sclerosis (ALS) and their caregivers. ALS is a progressive, fatal neurodegenerative disorder. No current medical therapy can reverse or stop its progression, and the promotion of quality of life and psychological well-being is a central component of ALS care. Health care professionals who work in this field should incorporate attention to psychological, emotional, and relational aspects of the disease into their approach to care. This book provides some of the knowledge and direction necessary for optimizing the quality of care for individuals with ALS and their caregivers. Topics discussed include an ALS-centred view of quality of life, depressive features, anxiety, resilience, cognitive impairment, complementary and alternative medicines, and psychological research. Specific elements of ALS, such as end-of-life concerns and bulbar dysfunction, are described through the lens of their psychological impact. There is extensive discussion of the development of new psychological treatments, as well as the impact and incorporation of new technologies, with the goal of fostering optimal quality of life and psychological well-being as key parts of a holistic approach to care for the patients and for those who are close to such individuals.

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28

Ferraiuolo, Laura, and Stephen J. Kolb. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0026.

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An overriding mystery of ALS pathogenesis orbits around the molecular basis of selective motor neuron vulnerability and clouds our view. There are likely mechanisms involved in the initiation of motor neuron loss and mechanisms involved in the progression of motor neuron loss once initiated. Motor neuron vulnerability is likely related to the unique biological characteristics of these cells. This chapter introduces central molecular pathways that appear to be involved in the pathogenesis of ALS, and highlights why dysregulation of these mechanisms could lead to motor neuron death. Indeed, there are likely mechanisms involved in the initiation of motor neuron loss and mechanisms involved in the progression of motor neuron loss once initiated. Our task is to determine those mechanisms that are relevant to ALS pathogenesis that may be targeted therapeutically to prevent onset and/or halt progression.

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29

Harms, Matthew B., and Timothy M. Miller. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0027.

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Recent advances in sequencing technologies have dramatically expanded the number of genes associated with amyotrophic lateral sclerosis, including rare but highly penetrant causative mutations as well as common risk alleles. This chapter discusses these gene discoveries and how they have implicated a diverse array of biological pathways essential for motor neuron health and have begun to inform our understanding of ALS pathogenesis as a heterogeneous and multistep process. Insights from these discoveries are leading to a new generation of targeted therapies directed at specific genes and are poised to inform how patients with amyotrophic lateral sclerosis are evaluated and treated in the clinic.

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30

Wainger, Brian J. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0028.

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Mouse and cellular models of ALS including stem cells have revealed tremendous insight into the molecular processes that lead to ALS. Models of ALS and other neurodegenerative diseases have led to emergent molecular themes that span several diseases. Future models must account for neuronal subtype specificity of different neurodegenerative diseases, particularly between tightly related diseases such as FTD and ALS. Human iPSC-derived motor neurons offer promise both with regard to the use of human cells and in particular the ability to model sporadic disease, which is critically important given the overwhelming abundance of sporadic disease in ALS and other neurodegenerative diseases.

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31

Mitsumoto, Hiroshi, Serge Przedborski, and Paul H. Gordon, eds. Amyotrophic Lateral Sclerosis. CRC Press, 2005. http://dx.doi.org/10.1201/b14133.

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32

Sattler, Rita, and Jeffrey Rothstein. Amyotrophic Lateral Sclerosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199794591.003.0063.

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This is a digitally enhanced text. Readers can also see the coverage of this topic area in the second edition of Neuroglia. The second edition of Neuroglia was first published digitally in Oxford Scholarship Online and the bibliographic details provided, if cited, will direct people to that version of the text. Readers can also see the coverage of this topic area in the ...

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33

1947-, Brown Robert H., Meininger Vincent, and Swash Michael, eds. Amyotrophic lateral sclerosis. London: Martin Dunitz, 2000.

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34

Araki, Toshiyuki, ed. Amyotrophic Lateral Sclerosis. Exon Publications, 2021. http://dx.doi.org/10.36255/exonpublications.amyotrophiclateralsclerosis.2021.

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35

Maurer, Martin, ed. Amyotrophic Lateral Sclerosis. InTech, 2012. http://dx.doi.org/10.5772/1054.

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36

Gelinas, Deborah, Patricia O'Connor, and Robert G. Miller. Amyotrophic Lateral Sclerosis. Springer Publishing Company, Incorporated, 2005.

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37

Mitsumoto, Hiroshi, Serge Przedborski, and Paul H. Gordon. Amyotrophic Lateral Sclerosis. Taylor & Francis Group, 2019.

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38

Estevez, Alvaro G., Jin Hee Shin, and Fabian H. Rossi. Amyotrophic Lateral Sclerosis. DI Press, 2022.

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39

Gelinas, Deborah, and Miller Robert. Amyotrophic Lateral Sclerosis. Springer Publishing Company, Incorporated, 2010.

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40

Amytotrophic Lateral Sclerosis. Demos Medical Publishing, 2012.

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41

A, Murray Christine, ed. Amyotrophic lateral sclerosis: New research. New York: Nova Biomedical Books, 2006.

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42

Amyotrophic Lateral Sclerosis: Current Research. Murphy & Moore Publishing, 2022.

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43

Murray, Christine A. Amyotrophic Lateral Sclerosis: New Research. Nova Biomedical Books, 2004.

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44

Brown, Robert H., Michael Swash, and Piera Pasinelli. Amyotrophic Lateral Sclerosis Second Edition. Taylor & Francis Group, 2019.

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45

Brown, Robert H., Michael Swash, and Piera Pasinelli. Amyotrophic Lateral Sclerosis, Second Edition. Taylor & Francis Group, 2021.

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46

Brown, Robert H., Michael Swash, and Piera Pasinelli. Amyotrophic Lateral Sclerosis, Second Edition. Taylor & Francis Group, 2021.

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47

O’Reilly, Éilis J. Epidemiology of Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0025.

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The epidemiology of ALS is an emerging field and, like the epidemiology of cardiovascular disease and cancer in the mid last century, requires time for convergence of findings. There appears to be a genetic influence, and one study of twins found that heritability of sporadic ALS is 60%. At present it is thought that 60$ to 70% of genetic mutations responsible for fALS in populations of European ancestry are known. SOD1 mutations were the earliest discoveries in fALS. Subsequently, mutations were identified in TARDBP, which encodes TDP-43 protein found in neuronal inclusions in fALS and frontotemporal dementia (FTD). High rates of ALS have also been reported in military personnel and in certain groups of elite athletes such as soccer players.

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48

Nicholson, Katharine A., and James D. Berry. Amyotrophic Lateral Sclerosis Clinical Trials. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0030.

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The current state of amyotrophic lateral sclerosis (ALS) trial design is best understood within the context of ALS research over the past quarter century. Before the early 1990s, trials in ALS were typically small and clinical trial methodology was less rigorous than it is today. With the discovery of the SOD1 gene mutation in the early 1990s, a new era of excitement and innovation for ALS research began. Since then, the number of ALS trials has steadily increased and trial design and methodology has become increasingly sophisticated.

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49

Foyaca-Sibat, Humberto, and Lurdes de Fátima Ibañez-Valdés, eds. Update on Amyotrophic Lateral Sclerosis. InTech, 2016. http://dx.doi.org/10.5772/61682.

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50

Jr., Robert H. Brown (Editor), Michael Swash (Editor), and Piera Pasinelli (Editor), eds. Amyotrophic Lateral Sclerosis, Second Edition. 2nd ed. Informa Healthcare, 2006.

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