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Books on the topic 'Amyloidosi'

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Published: 9 March 2023

Last updated: 10 March 2023

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1

Marrink, Jan, and Martin H. Van Rijswijk, eds. Amyloidosis. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4309-4.

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2

Glenner, George G., Elliott F. Osserman, Earl P. Benditt, Evan Calkins, Alan S. Cohen, and Dorothea Zucker-Franklin, eds. Amyloidosis. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2199-6.

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3

Gertz, Morie A., and S. Vincent Rajkumar, eds. Amyloidosis. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-631-3.

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4

1927-, Glenner George G., ed. Amyloidosis. New York: Plenum Press, 1986.

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5

Jan, Marrink, and Rijswijk Martin Henri van, eds. Amyloidosis. Dordrecht: M. Nijhoff, 1986.

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6

Thomas, Bardin, Brancaccio Diego, and Gejyo Fumitake, eds. Dialysis amyloidosis. Milan: Wichtig Editore, 1989.

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7

International, Symposium on Amyloidosis (10th 2004 Tours France). Amyloid and amyloidosis. Boca Raton: CRC Press, 2004.

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8

Isobe, Takashi, Shukuro Araki, Fumiya Uchino, Shozo Kito, and Eiro Tsubura, eds. Amyloid and Amyloidosis. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-0298-9.

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9

1935-, Isobe Takashi, ed. Amyloid and amyloidosis. New York: Plenum Press, 1988.

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10

Natvig, Jacob B., Øystein Førre, Gunnar Husby, Anne Husebekk, Bjørn Skogen, Knut Sletten, and Per Westermark, eds. Amyloid and Amyloidosis 1990. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3284-8.

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11

Xavier de Ávila, Diane, and Humberto Villacorta Junior, eds. Amyloidosis and Fabry Disease. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-17759-0.

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12

International Symposium on Amyloidosis (6th 1990 Oslo, Norway). Amyloid and amyloidosis 1990. Dordrecht: Kluwer Academic Publishers, 1991.

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13

United States. National Institutes of Health. National Institute of Diabetes and Digestive and Kidney Diseases., ed. Amyloidosis and kidney disease. [Washington, D.C.?]: National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, 2001.

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14

Amyloidosis: Diagnosis and treatment. New York: Humana Press, 2010.

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15

Hind, Charles R. K. Amyloidosis and amyloid P component. Burnt Mill, Harlow, Essex, England: Longman Scientific & Technical, 1986.

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16

International Symposium on Amyloidosis (11th 2006 Woods Hole, Mass.). XIth International Symposium on Amyloidosis. Edited by Skinner Martha M. D. Boca Raton: Taylor & Francis, 2007.

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17

Amyloidosis and amyloid P component. Harlow: Longman Scientific & Technical, 1986.

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18

Arluison, Véronique, Frank Wien, and Andrés Marcoleta, eds. Bacterial Amyloids. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2529-3.

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19

International Symposium on Amyloidosis ((5th 1987 Hakone-machi, Japan). Amyloid andamyloidosis. New York: Plenum Press, 1988.

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20

G, Husby, ed. Reactive amyloidosis and the acute phase response. London: Baillière Tindall, 1994.

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21

Grateau, Gilles. Les amyloses. Paris: Médecine-Sciences Flammarion, 2000.

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22

1928-, Kyle Robert A., and Gertz Morie A, eds. Amyloid and amyloidosis 1998: The proceedings of the VIIIth International Symposium on Amyloidosis, August 7-11, 1998, Rochester, Minnesota, USA. New York, N.Y: Parthenon Pub. Group, 1999.

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23

Glenner, George G. Amyloidosis. Springer, 2012.

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24

Mittal, Sajjan. Amyloidosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0181.

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Amyloidosis is a multisystem disease caused by the extracellular deposition of insoluble abnormal fibrils that injure tissues and organs. The fibrils are formed by the aggregation of misfolded, normally soluble proteins. Systemic amyloid light-chain (AL) amyloidosis (primary amyloidosis) is the commonest type of amyloidosis in the developed world, accounting for 80% of cases. The remainder are due to AA amyloidosis (secondary or reactive amyloidosis), familial amyloidosis, or other rare types of amyloidosis. The most common clinical features at diagnosis are nephrotic syndrome, heart failure (typically with predominant right heart failure), sensorimotor and/or autonomic peripheral neuropathy, and hepatosplenomegaly.

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25

Feng, Dali, ed. Amyloidosis. InTech, 2013. http://dx.doi.org/10.5772/46140.

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26

Glenner, George G. Amyloidosis. Springer, 2011.

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27

Marrink, J., and M. H. van Rijswijk. Amyloidosis. Springer London, Limited, 2012.

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28

Marrink, J., and M. H. van Rijswijk. Amyloidosis. Springer Netherlands, 2011.

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29

Amyloidosis. Springer, 2011.

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30

Grateau, Gilles, Robert A. Kyle, and Martha Skinner. Amyloid and Amyloidosis. Taylor & Francis Group, 2004.

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31

Isobe, T. Amyloid and Amyloidosis. Springer London, Limited, 2013.

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32

Grateau, Gilles, Robert A. Kyle, and Martha Skinner, eds. Amyloid and Amyloidosis. CRC Press, 2004. http://dx.doi.org/10.1201/9781420037494.

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33

Grateau, Gilles, Robert A. Kyle, and Martha Skinner. Amyloid and Amyloidosis. Taylor & Francis Group, 2004.

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34

Grateau, Gilles, Robert A. Kyle, and Martha Skinner. Amyloid and Amyloidosis. Taylor & Francis Group, 2004.

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35

Grateau, Gilles, Robert A. Kyle, and Martha Skinner. Amyloid and Amyloidosis. Taylor & Francis Group, 2004.

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36

Grateau, Gilles, Robert A. Kyle, and Martha Skinner. Amyloid and Amyloidosis. Taylor & Francis Group, 2004.

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37

Isobe, T. Amyloid and Amyloidosis. Springer, 2013.

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38

Isobe, T. Amyloid and Amyloidosis. Springer, 1989.

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39

Immunology & Alzheimer's Disease (RESEARCH AND PERSPECTIVES IN ALZHEIMER'S DISEASE). Springer, 1988.

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40

Amyloidosis Diet: A Nutritional Guide to Managing Amyloidosis for Beginners. Independently Published, 2022.

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41

Battaglia, S. Amyloids. Not Avail, 1986.

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42

Lachmann, Helen J., and Giampaolo Merlini. The patient with amyloidosis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0152.

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Abstract:

Amyloidosis is a disorder of protein folding in which normally soluble plasma proteins are deposited in the extracellular space in an abnormal insoluble fibrillar form. The process of amyloid formation and deposition causes cytotoxicity and progressive organ dysfunction. Amyloid is remarkably diverse and can be hereditary or acquired, localized or systemic, and lethal or merely an incidental finding. The most important numerically are AL amyloidosis, in which the fibrils are composed of monoclonal immunoglobulin light chains, and AA amyloidosis, in which the acute phase reactant Serum Amyloid A component forms the fibrils.The kidney is involved in 75% of patients with systemic amyloidosis. Heavy proteinuria or nephrotic syndrome is characteristic of most amyloid variants.Without treatment, systemic disease is usually fatal but measures that reduce the supply of amyloid fibril precursor proteins can result in regression of amyloid deposits, prevention of organ failure, and improved quality of life and survival. Early diagnosis, before irreversible organ damage has occurred, is the key to effective treatment. Recent advances in diagnosis and therapy have much improved the outlook of patients with AL amyloidosis, but agents with broader promise are under investigation.

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43

Gertz, Morie A., and S. Vincent Rajkumar. Amyloidosis: Diagnosis and Treatment. Humana, 2012.

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44

Harrison, Jonathan S. Amyloidosis: History and Perspectives. IntechOpen, 2022.

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45

Beating Back AL Amyloidosis. AlternateForum310, 2015.

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46

Amyloidosis: Diagnosis and Treatment. Murphy & Moore Publishing, 2021.

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47

Amyloid and Amyloidosis 1993. Taylor & Francis, 1994.

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48

Zacek, Pavel. Amyloidosis and Cardiac Surgery. INTECH Open Access Publisher, 2012.

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49

Fernandez-Escamilla, Ana Maria, ed. Exploring New Findings on Amyloidosis. InTech, 2016. http://dx.doi.org/10.5772/61496.

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50

Skinner, Martha, John L. Berk, Lawreen H. Connors, and David C. Seldin, eds. XIth International Symposium on Amyloidosis. CRC Press, 2007. http://dx.doi.org/10.1201/9781420043358.

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