Relevant bibliographies by topics / Alzhiemer's disease

Academic literature on the topic 'Alzhiemer's disease'

Author: Grafiati
Published: 6 July 2024

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Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Journal articles on the topic "Alzhiemer's disease":

1

Nevins, A., and S. Cohn. "More Audiovisuals on Alzhiemer's Disease." Gerontologist 25, no. 5 (October 1, 1985): 551–52. http://dx.doi.org/10.1093/geront/25.5.551a.

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Nevins, A., and H. Rzetelny. "More Audiovisuals on Alzhiemer's Disease." Gerontologist 25, no. 5 (October 1, 1985): 552. http://dx.doi.org/10.1093/geront/25.5.552.

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Fontana, Andrea, and Ronald W. Smith. "Alzheimer's Disease Victims: The “Unbecoming” of Self and the Normalization of Competence." Sociological Perspectives 32, no. 1 (March 1989): 35–46. http://dx.doi.org/10.2307/1389006.

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Abstract:
This study examines the importance of routinized practices for the self Alzheimer's disease victims are observed in their daily lives. The deterioration of self is discussed, followed by a discussion of routinized actions and normalization practices by caregivers that allow the patients to be seen as competent selves. Finally, the last remnants of the self in Alzhiemer's patients are discussed. The authors conclude that when the individual self undergoes an “unbecoming” process, due to the mental deterioration caused by the disease, it is largely social practices that allow the self to continue to exist in the eyes of others.
4

Liu-Seifert, Hong, Eric Siemers, Karen Price, Baoguang Han, Katherine Selzler, David Henley, Karen Sundell, et al. "P2-390: COGNITION IMPAIRMENT PRECEDES AND PREDICTS FUNCTIONAL IMPAIRMENT IN MILD ALZHIEMER's DISEASE." Alzheimer's & Dementia 10 (July 2014): P621. http://dx.doi.org/10.1016/j.jalz.2014.05.1070.

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Rozemuller, J. M., P. Eikelenboom, W. Kamphorst, and F. C. Stam. "Lack of evidence for dysfunction of the blood-brain barrier in Alzhiemer's disease: An immunohistochemical study." Neurobiology of Aging 9 (January 1988): 383–91. http://dx.doi.org/10.1016/s0197-4580(88)80085-x.

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Kumari, Rima, Suman Khushwaha, and Rohit Gupta. "P1-086: Alzhiemer's disease: Differentiation from other dementias based on quantitative diffusion tensor imaging on 3 Tesla MRI." Alzheimer's & Dementia 5, no. 4S_Part_7 (July 2009): P198. http://dx.doi.org/10.1016/j.jalz.2009.04.090.

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Apostol, Marcin I., Ashley Wright, Matthew Graf, Jon Scherrer, Ken Kosik, and James Treanor. "P4-156: DISCOVERY OF NOVEL SMALL MOLECULE THERAPEUTICS FOR ALZHIEMER'S DISEASE BY TARGETING STERIC ZIPPERS INVOLVED IN TAU AGGREGATION." Alzheimer's & Dementia 15 (July 2019): P1334. http://dx.doi.org/10.1016/j.jalz.2019.06.3818.

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SR, SHARUNYA, VIJAYALAKSHMI DESAI, MEENAKSHI SINGH, and KUSUMA M. "Survey on Early Detection of Alzhiemer’s Disease Using Capsule Neural Network." International Journal of Artificial Intelligence 7, no. 1 (April 23, 2020): 7–12. http://dx.doi.org/10.36079/lamintang.ijai-0701.65.

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Abstract:
Alzheimer's disease (AD) is an disorder which is irreversible of the brain related to memory loss, mostly found in the old and aged population. Alzheimer's dementia results from the degeneration or loss of brain cells. The brain-imaging technologies most often used to diagnose AD is Magnetic resonance imaging (MRI). MRI or structural magnetic resonance is a very popular and actual technique used to diagnose AD. An MRI uses magnets and powerful radio waves to create a complete view of your brain. To actually detect the presence of Alzheimer’s, the MRI should me studied carefullyImplementation of CBIR Content Based Image Retrival which is a revolutionary computer aided diagnosis technique will create new abilities in MRI Magnetic resonance imaging in related image retrieval and training for recognition of development of AD in early stages
9

Bezprozvanny, I. "Presenilins function as ER calcium leak channels: Implications for Alzhiemer’s disease." Biochemistry (Moscow) Supplement Series A: Membrane and Cell Biology 3, no. 3 (September 2009): 301. http://dx.doi.org/10.1134/s199074780903012x.

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Wojtunik-Kulesza, Karolina A., and Monika Waksmundzka- Hajnos. "Natural terpenes as substances with multidirectional biological activities." Postępy Polskiej Medycyny i Farmacji 6 (May 8, 2019): 43–50. http://dx.doi.org/10.5604/01.3001.0013.2320.

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Abstract:
Terpenes, secondary metabolites, are considered by scientists around the World. Multidirectional biological activities of these substances encourage researchers to their detail analysis towards numerous diseases such as neurodegenerative or metabolic. In the presented paper, authors have focused on Alzheimer’s disease and diabetes as potential direction of terpenes’ activity. According to available literature, terpenes exhibit satisfactory antioxidant activity and ability to inhibition AChE, BuChE (Alzhiemer’s disease) and α-amylase, α-glucosidase (diabetes). Basis of the presented research are both essential oils obtained from plants such as salvia, rosemary, basil and lemon balm being one of the main source of natural terpenes, and single compounds. Special attention is paid to monoterpenes as the main components of numerous essential oils. Among terpenes presented in the paper, activity of ocimene, carvone, pulegone, α-phellandrene exhibiting activity towards both free radical scavenging as well as AChE and BuChE inhibition, and α-pinene, citral, limonene that revealing inhibitory activity towards α-amylase and α-glucosidase should be emphasized.
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Journal articles

Dissertations / Theses on the topic "Alzhiemer's disease":

1

Freer, Rosie. "Molecular origins of tissue vulnerability to aberrant aggregation in protein misfolding diseases." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/275420.

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Neurodegenerative disorders, including Alzheimer’s disease (AD) and Parkinson’s disease (PD), are increasingly common in our ageing society, are remain incurable. A major obstacle encountered by researchers in their attempts to find effective therapies is represented by the current lack of understanding of the molecular origins of these disorders. It is becoming clear that, although the aggregation of specific proteins, including amyloid β (Aβ) and tau in AD and α-synuclein in PD, hallmark these disorders, such behaviour is a consequence of a wider, system-level disruption of protein homeostasis. In order to identify the genetic factors contributing to such a disruption, the transcriptional changes that occur during neurodegenerative disease progression have received considerable scientific attention in recent years. In our approach, we considered another hallmark of these diseases - their characteristic patterns of spreading across the brain - to identify the nature of the transcriptional signature which underlies tissue vulnerability to protein aggregation. By understanding why tissues succumb in their characteristic sequential pattern in neurodegenerative diseases, and why some tissues remain almost completely resistant throughout, we hoped to obtain insight into the molecular origins of these disorders. Our results show that the AD progression can be predicted from a transcriptional signature in healthy brains related to the protein aggregation homeostasis of Aβ, tau, and the wider proteome. We highlight a relationship between a specific subproteome at high risk of aggregation (formed by supersaturated proteins), and the vulnerability to neurodegenerative diseases. We thus identify an AD-specific supersaturated set of proteins - termed the metastable subproteome, whose expression in normal brains recapitulates the staging of AD, with more vulnerable tissues having higher metastable subproteome expression. We find evidence of these vulnerability signatures transcending the tissue level of interrogation, with cellular and subcellular analysis also showing elevated levels of proteins known and predicted to predispose the aberrant aggregation of Aβ and tau. These results characterise the key protein homeostasis pathways in the inception and progression of AD, and establish an approach with the potential to be applied to other protein misfolding diseases, in the brain and beyond.
2

Payne, Magali. "Olfaction, cognition et émotions : liens dans la maladie d'Alzheimer, l'apathie et la COVID-19." Electronic Thesis or Diss., Université Côte d'Azur, 2022. http://www.theses.fr/2022COAZ6013.

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Dans cette thèse nous avons créé un test olfactif et vérifié son utilité pour dépister des troubles en lien avec la pathologie d'Alzheimer ou celle de l'apathie, ou encore dans la COVID-19.Après avoir recueilli l'avis des cliniciens et les recommandations des experts chimistes, nous avons ensuite sélectionné quatorze odeurs, mis au point un test olfactif informatisé qui permet d'obtenir automatiquement le score de seuil de perception et d'identification. Ce travail a permis le dépôt d'un Brevet d'invention pour le "Test Olfactif de Dépistage de la maladie d'Alzheimer et de l'apathie", ou TODA.Nous avons vérifié l'intérêt du TODA en pratique de soins courants dans l'aide au diagnostic de la maladie d'Alzheimer auprès de malades d'Alzheimer en contrôles sains, en France et au Québec. Les scores olfactifs différenciaient très significativement les sujets et étaient corrélés à la mesure cognitive globale. Le TODA permettait aussi de repérer une identification des odeurs différente en fonction de la culture et de la pathologie.Dans une population de sujets présentant un Trouble Cognitif Mineur avec ou sans apathie, les résultats au TODA permettaient de différencier les sujets très significativement, notamment dans la sous-dimension sociale des critères de l'Apathie.Auprès de sujets présentant une post-infection à la COVID-19, le TODA a été utilisé en parallèle du Sniffin'Sticks Test, gold-standard actuel, pour vérifier la validité des résultats du TODA. Le TODA a aussi permis de mettre en évidence des confusions d'odeurs proches, révélant une atteinte de leurs propriétés olfactives. Son utilisation en cas de suspicion d'atteinte sémantique associée a elle aussi été concluante.La comparaison des scores obtenus au TODA par les sujets MA et par les sujets post-COVID-19 a révélé que les résultats étaient similaires. Seule l'étude des odeurs les plus significatives a permis de faire la différence entre les deux pathologies
In this thesis we created an olfactory test to measure its usefullness to detect disorders such as Alzheimer's disease or apathy syndrome or COVID-19.First, we collected the opinions of clinicians and the recommendations of experts chemists. We then selected fourteen odors, and we developed a computerized olfactory test that was able to calculate threshold and identification automatically using theses odors. We submitted a Patent for this test named "Olfactory Test for screening Alzheimer's disease ans apathy" (TODA).We cerified the interest ol TODA in everyday care practice in the diagnosis of Alzheimer's disease. Using subjects with Alzheimer's disease and healthy control subjects, both in France and Quebec, the test scores significantly differenciate AD subjects from control subjects. This test allowed cognitive monitoring of patients and highlighted odor identification regardless of culture and pathology.With a population of subjects with Minor Cognitive Disorder, with and without apathy, the results were able to significantly differenciate subjects, in particular, with a social sub-dimension of Critéria of Apathy.With subjects suffering from post-COVID-19 infection we tested with the TODA and in parallel we used the Sniffin' Stick Test, a gold standard, to verify the validity of the TODA results. The TODA also allowed us to highlight the problems identify similar odors. Finally, we also highlighted the interest of using the TODA in the case of suspected associated semantics impairment.The comparison of the TODA scores obtained by AD subjects and COVID-19 subjects showed that there was no difference between the two groups. Only the study of the most significant odors made it possible to differenciate between the two patologies

Book chapters on the topic "Alzhiemer's disease":

1

Riesner, Detlev. "Protein Aggregation Associated with alzhiemer and Prion Diseases." In Springer Series in Chemical Physics, 225–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-59542-4_11.

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Conference papers on the topic "Alzhiemer's disease":

1

Noel, Rebecca L., Alec J. Batts, and Elisa E. Konofagou. "Focused Ultrasound-Induced Blood-Brain Barrier Opening Delays the Onset of Alzhiemer's Disease-Associated Pathology in Male and Female 3xTg Mice." In 2022 IEEE International Ultrasonics Symposium (IUS). IEEE, 2022. http://dx.doi.org/10.1109/ius54386.2022.9957937.

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2

Shukla, Amar, Rajeev Tiwari, and Shamik Tiwari. "Automated Pipeline Preprocessing Techniques for Alzhiemer Disease Detection." In 2022 10th International Conference on Reliability, Infocom Technologies and Optimization (Trends and Future Directions) (ICRITO). IEEE, 2022. http://dx.doi.org/10.1109/icrito56286.2022.9964957.

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3

Gangyan, Manu, and Pravir Kumar. "VEGF and its role in the treatment of Diabetes and Alzhiemer’s Disease." In 2021 Sixth International Conference on Image Information Processing (ICIIP). IEEE, 2021. http://dx.doi.org/10.1109/iciip53038.2021.9702619.

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4

Lu, Benjamin M., and Abhinav S. Gurram. "revoAD: Revolutionizing Alzhiemer’s Disease Diagnosis through Multimodal Machine Learning for Universal Screening via Speech and Handwriting Patterns." In 2023 8th International Conference on Intelligent Informatics and Biomedical Sciences (ICIIBMS). IEEE, 2023. http://dx.doi.org/10.1109/iciibms60103.2023.10347810.

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