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Books on the topic 'Alzheimers’s disease'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Altman, Harvey J., ed. Alzheimer’s Disease. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-6414-0.

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2

Jha, Arun, and Kaushik Mukhopadhaya. Alzheimer’s Disease. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-56739-2.

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3

Sisodia, Sangram S., and Rudolph E. Tanzi, eds. Alzheimer’s Disease. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-35135-3.

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4

Harris, J. Robin, and Falk Fahrenholz, eds. Alzheimer’s Disease. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/b100942.

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5

Turner, J. D., K. Beyreuther, and F. Theuring, eds. Alzheimer’s Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-03248-0.

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6

Chun, Jerold, ed. Alzheimer’s Disease. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2655-9.

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7

Parks, Peggy J. Alzheimer's disease. San Diego: ReferencePoint Press, 2009.

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8

Sammons, Vivian O. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1987.

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9

Dempsey, Denise P. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1997.

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10

Parliament, Canada Library of. Alzheimer's Disease. Ottawa: Libray of Parliament, 1993.

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11

Molloy, William. Alzheimer's disease. Toronto, Ont: Key Porter Books, 1998.

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12

Brassard, Daniel J. Alzheimer's disease. [Ottawa]: Library of Parliament, Research Branch, 1993.

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13

Roos, R. A. C. Alzheimer's disease. Leiden]: Boerhaave Committee for Post-graduate Education of Medecine, Leiden University, 1998.

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14

Molloy, William. Alzheimer's disease. Buffalo, N.Y: Firefly Books, 2003.

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15

Canada. Library of Parliament. Research Branch., ed. Alzheimer's disease. Ottawa, Ont: Library of Parliament, Research Branch, 1993.

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16

Strock, Margaret. Alzheimer's disease. [Washington, D.C.?]: National Institutes of Health, National Institute of Mental Health, 1994.

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17

Landau, Elaine. Alzheimer's disease. New York: Franklin Watts, 1996.

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18

R, Solomon Paul, ed. Alzheimer's disease. Parsippany, N.J: Crestwood House, 1996.

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19

Gold, Susan Dudley. Alzheimer's disease. Berkeley Heights, N.J: Enslow Publishers, 2001.

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20

R, Solomon Paul, ed. Alzheimer's disease. Berkeley Heights, N.J: Enslow Publishers, 2001.

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21

J, Doka Kenneth, and Hospice Foundation of America, eds. Alzheimer's disease. Washington, DC: Hospice Foundation of America, 2004.

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22

Juergen, Bludau, ed. Alzheimer's disease. Santa Barbara, Calif: Greenwood, 2011.

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23

Twyla, Mueller Racz, ed. Alzheimer's disease. Phoenix, AZ: Oryx Press, 1987.

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24

Adams, Jacqueline. Alzheimer's disease. Detroit: Lucent Books, 2011.

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25

Hooper, Nigel M. Alzheimer's Disease. New Jersey: Humana Press, 1999. http://dx.doi.org/10.1385/1592591957.

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26

Richter, Ralph W., and Brigitte Zoeller Richter. Alzheimer's Disease. New Jersey: Humana Press, 2003. http://dx.doi.org/10.1385/1592596614.

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27

Richter, Ralph Walter. Alzheimer's disease. London: Mosby, 2002.

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28

Landau, Elaine. Alzheimer's disease. New York: Franklin Watts, 1996.

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29

Dempsey, Denise P. Alzheimer's disease. Washington, D.C. (101 Independence Ave., S.E., Washington 20540: Science Reference Section, Science and Technology Division, Library of Congress, 1997.

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30

Landau, Elaine. Alzheimer's disease. New York: F. Watts, 1987.

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31

Alzheimer's disease. Upper Saddle River, NJ: Pearson Prentice Hall, 2004.

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32

Sammons, Vivian O. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1987.

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33

Strock, Margaret. Alzheimer's disease. [Washington, D.C.?]: National Institutes of Health, National Institute of Mental Health, 1994.

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34

Strock, Margaret. Alzheimer's disease. [Washington, D.C.?]: National Institutes of Health, National Institute of Mental Health, 1994.

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35

Sammons, Vivian O. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1987.

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36

Brill, Marlene Targ. Alzheimer's disease. New York: Benchmark Books, 2005.

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37

Pendlebury, William W. Alzheimer's disease. [New Jersey]: Ciba-Geigy Corporation, 1996.

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38

Dempsey, Denise P. Alzheimer's disease. Washington, D.C. (101 Independence Ave., S.E., Washington 20540: Science Reference Section, Science and Technology Division, Library of Congress, 1997.

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39

Govindaraju, Thimmaiah, ed. Alzheimer's Disease. Cambridge: Royal Society of Chemistry, 2022. http://dx.doi.org/10.1039/9781839162732.

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40

Lau, Lit-Fui, and Michael A. Brodney, eds. Alzheimer's Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-74229-6.

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41

Alzheimer's Disease (Diseases and People). Enslow Publishers, 2002.

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42

Lexi-Comp. Diseases Explained: Alzheimer's Disease Wall Chart. Lexi Comp, 2001.

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43

Diseases and Disorders - Alzheimer's Disease (Diseases and Disorders). Lucent Books, 2000.

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44

Hyman, Bradley T., Beatriz G. Perez-Nievas, Isabel Barroeta-Espar, Alberto Serrano-Pozo, Matthew Frosch, and Teresa Gomez-Isla. Alzheimer’s Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0015.

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Alzheimer’s disease is the most common cause of dementia in the developed world, and has a rising incidence due to the strong age-related nature of the risk for the disease. As the population as a whole ages, the number of cases of Alzheimer’s disease will rise in what is essentially an epidemic. The disease is progressive, evolves over years, but is ultimately fatal. Aside from symptomatic therapies that are weakly effective, no treatments are available. The underlying pathobiology, connections to the genetics of the disease, and prospects for research are explored.

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45

Rafii, Michael S. Alzheimer’s Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0016.

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Alzheimer’s disease (AD) is the most common cause of dementia worldwide, and is characterized by a protracted asymptomatic phase estimated to begin approximately 15 to 20 years. Clinically, AD initially manifests itself by progressive memory impairment, specifically, a loss of episodic memory function characterized by impaired free recall that does not improve with cueing. This is followed by a gradual decline in other cognitive domains leading to functional dependency, which essentially defines the dementia phase of the illness and has been the cornerstone of diagnostic criteria. About 50% of all MCI patients progress to Alzheimer’s dementia, and therefore MCI is thought to represent prodromal AD when they harbor the pathological changes associated with AD, including hippocampal atrophy, elevated CSF amyloid and Tau, as well as the presence of amyloid on PET scan.

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46

Jicha, Gregory A., and Frederick A. Schmitt. Alzheimer’s Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0017.

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Advances in the current management and treatment of Alzheimer’s disease have grown directly from our increased understanding of the neurobiology underlying this disease. Currently available pharmacologic and nonpharmacologic treatment strategies remain focused on symptomatic management of disease rather than disease modification. Despite a wealth of evidence supporting the clinical benefits of existing therapies in the management of symptomatic progression, there is limited evidence that these available therapies modify disease progression over the course of dementia progression. More recent research discoveries in the areas of genetics, molecular and cell biology, and environmental risk factors have become the focal point for an explosive growth in experimental disease-modifying strategies designed to prevent, slow, or potentially halt the progression of Alzheimer’s disease.

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47

Taylor, John-Paul, and Alan Thomas. Alzheimer’s disease. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0033.

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Alzheimer’s disease (AD) is a well recognised clinicopathological entity and the most common cause of dementia. This chapter critiques the several sets of newly proposed criteria for making the diagnosis of (AD) at different stages in the disease process, in the context of demand for earlier diagnosis and emerging biomarkers. The differential diagnosis and types of different cognitive symptoms are reviewed and the prevalence and pattern of the many non-cognitive symptoms associated with AD are considered. The management of non-cognitive symptoms is also updated, especially the circumstances in which antipsychotic drugs might be prescribed. And finally a summary of our current understanding of the aetiology (evidence for risk and protective factors for AD ) and the prognosis for AD is provided.

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48

Khachaturian, Zaven S., and Teresa S. Radebaugh, eds. Alzheimer’s Disease. CRC Press, 2019. http://dx.doi.org/10.1201/9780429260353.

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49

Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activity is greatly suppressed in AD. The unique metabolism of ketone bodies allows them to bypass the block at pyruvate dehydrogenase and restore brain metabolism. Recent studies in mouse genetic models of AD and in a human Alzheimer’s patient showed the potential of ketones in maintaining brain energetics and function. Oral ketone bodies might be a promising avenue for treatment of Alzheimer’s disease.

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50

Wisniewski, Thomas, ed. Alzheimer’s Disease. Codon Publications, 2019. http://dx.doi.org/10.15586/alzheimersdisease.2019.

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