Relevant bibliographies by topics / Alpha variation

Academic literature on the topic 'Alpha variation'

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Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Alpha variation"

1

MOON, MARY ANN. "Alpha-Synuclein Gene Variation Associated With Parkinson's." Clinical Psychiatry News 34, no. 11 (November 2006): 44. http://dx.doi.org/10.1016/s0270-6644(06)71903-8.

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2

Dunn, D. S., B. Madhoo, R. Turnbull, and T. Jenkins. "Alpha-1-Antitrypsin Variation in Southern Africa." Human Heredity 36, no. 4 (1986): 238–42. http://dx.doi.org/10.1159/000153633.

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3

Şengül, Hacer, Hüseyin Çakallı, and Mikail Et. "A variation on strongly ideal lacunary ward continuity." Boletim da Sociedade Paranaense de Matemática 38, no. 7 (October 14, 2019): 99–108. http://dx.doi.org/10.5269/bspm.v38i7.46136.

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The main purpose of this paper is to introduce the concept of strongly ideal lacunary quasi-Cauchyness of order (alpha,beta) of sequences of real numbers. Strongly ideal lacunary ward continuity of order (alpha,beta) is also investigated. Interesting results are obtained.
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4

Kunicki, TJ, R. Orchekowski, D. Annis, and Y. Honda. "Variability of integrin alpha 2 beta 1 activity on human platelets." Blood 82, no. 9 (November 1, 1993): 2693–703. http://dx.doi.org/10.1182/blood.v82.9.2693.2693.

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Abstract The activity and surface antigenicity of alpha 2 beta 1 on platelets from 27 normal subjects were found to vary significantly. A fourfold range of surface antigen correlates with a 20-fold variation in the ability of nonactivated, washed platelets to adhere to type I collagen and a fivefold variation in the adhesion of platelets to type III collagen. These differences in surface receptor are reflected in significant variation in the lag time required for type I collagen- induced platelet aggregation in platelet-rich plasma. Among the same individuals, no difference was observed in surface levels or activities of two other platelet integrins, the fibronectin receptor alpha 5 beta 1 and the fibrinogen receptor alpha IIb beta 3. In all cases studied, we observed complimentary differences in the incorporation of 125I into surface alpha 2 beta 1, in quantity of surface alpha 2 beta 1 antigens, and in alpha 2 beta 1 collagen receptor activity. Despite variations in these parameters, there was no difference in the electrophoretic mobility or isoelectric point of either integrin subunit among the individuals studied. The wide range of activity and antigenicity of this platelet collagen receptor may result from polymorphism(s) in the alpha 2 beta 1 genes, or the activity of alpha 2 beta 1 may be variably regulated by another gene product. The heterogeneity of platelet alpha 2 beta 1 that we describe in this report certainly explains previous discrepancies concerning the contributions of this integrin to platelet adhesion to collagens. Most importantly, differences in surface collagen receptor activity may correlate with a long-term risk toward thrombosis, impaired hemostasis, and/or cardiovascular disease.
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Kunicki, TJ, R. Orchekowski, D. Annis, and Y. Honda. "Variability of integrin alpha 2 beta 1 activity on human platelets." Blood 82, no. 9 (November 1, 1993): 2693–703. http://dx.doi.org/10.1182/blood.v82.9.2693.bloodjournal8292693.

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The activity and surface antigenicity of alpha 2 beta 1 on platelets from 27 normal subjects were found to vary significantly. A fourfold range of surface antigen correlates with a 20-fold variation in the ability of nonactivated, washed platelets to adhere to type I collagen and a fivefold variation in the adhesion of platelets to type III collagen. These differences in surface receptor are reflected in significant variation in the lag time required for type I collagen- induced platelet aggregation in platelet-rich plasma. Among the same individuals, no difference was observed in surface levels or activities of two other platelet integrins, the fibronectin receptor alpha 5 beta 1 and the fibrinogen receptor alpha IIb beta 3. In all cases studied, we observed complimentary differences in the incorporation of 125I into surface alpha 2 beta 1, in quantity of surface alpha 2 beta 1 antigens, and in alpha 2 beta 1 collagen receptor activity. Despite variations in these parameters, there was no difference in the electrophoretic mobility or isoelectric point of either integrin subunit among the individuals studied. The wide range of activity and antigenicity of this platelet collagen receptor may result from polymorphism(s) in the alpha 2 beta 1 genes, or the activity of alpha 2 beta 1 may be variably regulated by another gene product. The heterogeneity of platelet alpha 2 beta 1 that we describe in this report certainly explains previous discrepancies concerning the contributions of this integrin to platelet adhesion to collagens. Most importantly, differences in surface collagen receptor activity may correlate with a long-term risk toward thrombosis, impaired hemostasis, and/or cardiovascular disease.
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6

Salter, D. M., J. L. Godolphin, and M. S. Gourlay. "Chondrocyte heterogeneity: immunohistologically defined variation of integrin expression at different sites in human fetal knees." Journal of Histochemistry & Cytochemistry 43, no. 4 (April 1995): 447–57. http://dx.doi.org/10.1177/43.4.7897185.

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During development and at maturity different forms of cartilage vary in morphology and macromolecular content. This reflects heterogeneity of chondrocyte activity, in part involving differential interactions with the adjacent extracellular matrix via specialized cell surface receptors such as integrins. We undertook an immunohistological study on a series of human fetal knee joints to assess variation in the expression of integrins by chondrocytes and potential matrix ligands in articular, epiphyseal, growth plate, and meniscal cartilage. The results show that articular chondrocytes (beta 1+, beta 5 alpha V+, alpha 1+, alpha 2+/-, alpha 5+, weakly alpha 6+, alpha V+) differed from epiphyseal (beta 1+, beta 5 alpha V+, alpha 1+/-, alpha 2+/-, alpha 5+, alpha 6+, alpha V+) growth plate (beta 1+, beta 5 alpha V+, alpha 1-, alpha 2-, alpha 5+, alpha 6+, alpha V+), and meniscal cells (beta 1+, beta 5 alpha V+, alpha 1+, strongly alpha 2+, alpha 5+, alpha 6+, alpha V+ in expression of integrin subunits. There was no expression of beta 3, beta 4, beta 6, or alpha 3 by chondrocytes. These results differ from previous reports on the expression of integrins by adult articular cartilage, where alpha 2 and alpha 6 are not seen. Variation in distribution of matrix ligands was also seen. Fibronectin, laminin and Type VI collagen were expressed in all cartilages but there was restricted expression of tenascin, ED-A and ED-B fibronectin isoforms (articular cartilage and meniscus), and vitronectin (absent from growth plate cartilage). Regulated expression of integrins by chondrocytes, associated with changes in the pericellular matrix composition, is of potential importance in control of cartilage differentiation and function in health and disease.
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7

Ohno, Ichiro. "Temperature Variation of Elastic Properties of .ALPHA.-Quartz up to the .ALPHA.-.BETA. Transition." Journal of Physics of the Earth 43, no. 2 (1995): 157–69. http://dx.doi.org/10.4294/jpe1952.43.157.

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8

MENEZES, J., C. SANTOS, and P. P. AVELINO. "GRAVITATIONAL EFFECTS OF VARYING ALPHA STRINGS." International Journal of Modern Physics A 21, no. 16 (June 30, 2006): 3295–306. http://dx.doi.org/10.1142/s0217751x06031430.

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We study spatial variations of the fine-structure constant in the presence of static straight cosmic strings in the weak-field approximation in Einstein gravity. We work in the context of a generic Bekenstein-type model and consider a gauge kinetic function linear in the scalar field. We determine an analytical form for the scalar field and the string metric at large distances from the core. We show that the gravitational effects of α-varying strings can be seen as a combination of the gravitational effects of global and local strings. We also verify that at large distances to the core the space–time metric is similar to that of a global string. We study the motion of test particles approaching from infinity and show that photons are scattered to infinity while massive particles are trapped in bounded trajectories. We also calculate an overall limit on the magnitude of the variation of α for a GUT string, by considering suitable cosmological constraints coming from the Equivalence Principle.
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9

Claeyssens, A., J. Richard, J. Blaizot, T. Garel, F. Leclercq, V. Patrício, A. Verhamme, et al. "Spectral variations of Lyman $\alpha$ emission within strongly lensed sources observed with MUSE." Monthly Notices of the Royal Astronomical Society 489, no. 4 (September 10, 2019): 5022–29. http://dx.doi.org/10.1093/mnras/stz2492.

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ABSTRACT We present an analysis of ${\rm H\,\rm{\small {I}}}$ Lyman $\alpha$ emission in deep VLT/MUSE observations of two highly magnified and extended galaxies at $z=3.5$ and 4.03, including a newly discovered, almost complete Einstein ring. While these Lyman $\alpha$ haloes are intrinsically similar to the ones typically seen in other MUSE deep fields, the benefits of gravitational lensing allow us to construct exceptionally detailed maps of Lyman $\alpha$ line properties at sub-kpc scales. By combining all multiple images, we are able to observe complex structures in the Lyman $\alpha$ emission and uncover small ($\sim120$ km s−1 in Lyman $\alpha$ peak shift), but significant at $ \gt $4 $\sigma$, systematic variations in the shape of the Lyman $\alpha$ line profile within each halo. Indeed, we observe a global trend for the line peak shift to become redder at large radii, together with a strong correlation between the peak wavelength and line width. This systematic intrahalo variation is markedly similar to the object-to-object variations obtained from the integrated properties of recent large samples. Regions of high surface brightness correspond to relatively small line shifts, which could indicate that Lyman $\alpha$ emission escapes preferentially from regions where the line profile has been less severely affected by scattering of Lyman $\alpha$ photons.
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10

Hauck, Ekkehard W., Arne Hauptmann, Simone M. Haag, Anette Bohnert, Wolfgang Weidner, Gregor Bein, and Holger Hackstein. "Alpha-1-Antitrypsin Levels and Genetic Variation of the Alpha-1-Antitrypsin Gene in Peyronie’s Disease." European Urology 46, no. 5 (November 2004): 623–28. http://dx.doi.org/10.1016/j.eururo.2004.04.028.

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Dissertations / Theses on the topic "Alpha variation"

1

Holmes, Mark D. "Molecular analyses of alpha 1-antitrypsin variation and deficiency /." Title page, table of contents and aims and general introduction only, 1992. http://web4.library.adelaide.edu.au/theses/09MD/09mdh752.pdf.

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Khan, Fayeza Fatima. "Characterization of the alpha defensin copy number variation in humans." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12693/.

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Copy number variation (CNV) has been acknowledged as an important contributor to variation in the human genome, and copy-variable regions harbouring genes are interesting subjects of research for their potential phenotypic effects. However, despite the extensive and continuing discovery of CNVs, multiallelic loci remain technically challenging to measure and study. In this thesis, a PCR-based measurement system for the tandemly repeated CNV that harbours the DEFA1A3 locus has been developed. This locus can either have the DEFA1 or the DEFA3 gene in any given copy of the repeat; the two genes differ by a single nucleotide difference in the coding sequence. This CNV has previously been shown to vary from 4 to 11 copies in a sample of the UK population. The use of this measuring system has allowed a usefully accurate measure and some characterization of this CNV in Europeans, Asians (Chinese and Japanese) and African (Ibadan, Nigeria) samples from the HapMap project, agreeing with the previously found copy number range in Europeans and showing more variability in non-Europeans. Typing of three-generation CEPH families has allowed the inference of haplotype copy numbers from segregation analysis. Combining haplotype data for some CEPH HapMap samples that were part of these families with their SNP genotypes in the same LD block has shown copy number lineages that are surprisingly well-tagged by SNPs. SNP rs4300027 allows the division of copy number haplotypes into low (2 and 3-copy) and high (4 and 5-copy) groups in European HapMap samples, a result that has been corroborated in an independent set of European samples. The Asian and African HapMap samples have failed to show this particular association. However, Japanese samples show copy number lineages tagged by other SNPs, an association not observed in other populations. In the second part of the study, an attempt has been made to explore the phenotypic effects of this variable locus. Copy number-tagging SNPs have been used to investigate published GWAS for indirect signals of association with DEFA1A3 copy number. Preliminary experiments for studying protein expression levels of DEFA1 and DEFA3 have been carried out and the possible role of this CNV as a modifier locus in Cystic Fibrosis disease severity has been investigated through direct typing of CF samples with clinical data, and indirectly through interrogating a CF GWAS.
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3

McKechnie, Victoria Margaret. "Variation in the NS5A gene of Hepatitis C Virus in response to interferon alpha therapy." Thesis, University of Glasgow, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301364.

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4

Wang, Qingfeng. "Rough path properties for local time of symmetric alpha stable processes." Thesis, Loughborough University, 2012. https://dspace.lboro.ac.uk/2134/11052.

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5

Wood, Alice Margaret. "The role of genetic and environmental variation in the respiratory phenotype of alpha 1 antitrypsin deficiency." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/764/.

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Alpha 1 antitrypsin deficiency (AATD) is the only established genetic predisposition to chronic obstructive pulmonary disease (COPD). The development of COPD in AATD is highly variable, probably relating to complex interactions between multiple genetic and environmental factors. This thesis will describe the COPD phenotypes observed in AATD and their inter-relationships, forming the basis for examining phenotypic associations with specific candidate genes and HLA class II type. Finally it examines the potential role of ambient air pollution. Associations were seen for TNFA with chronic bronchitis, SFTPB with FEV1, TGFB with small airways disease and GC with bronchiectasis, consistent with the role of protein products in pathogenesis. Of four MMPs studied, association with gas transfer occurred in two. HLA-DQA1*0301 and HLA-DRB1*04 contributed significantly to gas transfer in regression models, and anti elastin antibodies were higher in HLA-DRB1*04 and HLA-DQA1*0301 homozygotes. Ozone levels contributed to the burden of disease in cross sectional and longitudinal models of pollution exposure, whilst PM10, NO2 and SO2 were associated only in the longitudinal model. In conclusion this thesis demonstrates the importance of genetic variation and environmental factors in determining respiratory phenotype in AATD. It also suggests a key role for adaptive immunity in pathogenesis of emphysema.
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Berengut, Julian Carlo Physics Faculty of Science UNSW. "Isotope shift and relativistic shift in atomic spectra." Awarded by:University of New South Wales. Physics, 2006. http://handle.unsw.edu.au/1959.4/23900.

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At present several groups are analysing quasar absorption spectra to search for variation of the fine structure constant, alpha, across space and time. These studies compare the wavelengths of several transitions observed in the absorption clouds with those seen in the laboratory, and interpret anomalies as variation in alpha. One group has already presented evidence that alpha may have been smaller at an early epoch. Other groups using different telescopes see no variation. These studies use the ???many-multiplet??? method, which relies on the utilisation of many transitions in many ions to enhance the size of the effects and remove sources of systematic error. While this method offers an order-of-magnitude improvement in sensitivity over the previously used alkali-doublet method, the alpha-dependence (relativistic shift) of every transition used in the analysis must be calculated ab initio. In this thesis we present a method for the precise calculation of relativistic shifts, based on an energy calculation involving combination of the configuration interaction method and many-body perturbation theory. The many-multiplet method also introduces a potential systematic error: if the relative isotope abundances of the absorbers differ from terrestrial abundances then there can be spurious shifts in the measured wavelengths, which may be incorrectly interpreted as variation of alpha. A ???conspiracy??? of several isotopic abundances may provide an alternative explanation for the observed spectral anomalies. To account for these systematic errors we need accurate values of the isotope shift. We calculate these shifts using the finite-field method to reduce the problem to that of an energy calculation, which in turn is done using the same method used for the relativistic shift. We present the results of our calculations for a variety of atoms and ions seen in quasar absorption spectra. The results of this research should allow astrophysicists to measure isotope abundances in the absorbers directly. This can provide a test for models of nuclear reactions in stars and supernovae, and of the chemical evolution of the Universe. Our calculations can also be used in conjunction with measurements to extract changes in nuclear charge radii between isotopes.
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Gokcek, Sarac Cigdem. "Study On The Molecular Basis Of Individual Variation In Spatial Memory In Rats." Phd thesis, METU, 2012. http://etd.lib.metu.edu.tr/upload/12614332/index.pdf.

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Despite very extensive studies related to molecular processes underlying memory formation, still little known about the potential differences in the brain biochemistry between &ldquo
good&rdquo
and &ldquo
poor&rdquo
learners belonging to a random population of young animals. In the present study, an attempt was taken to correlate the individual variation in short- and long-term spatial memory in three different lines of young, healthy rats: inbred Wistar (W), outcrossed Wistar/Spraque Dawley (W/S) and pigmented Long-Evans rats, with hippocampal levels of selected enzymes known as &ldquo
memory molecules&rdquo
including neuronal (n), endothelial (e) and inducible (i) NOS, CaMKII&alpha
, PKA and ChAT. Additionally, in order to indirectly estimate the activity of CaMKII&alpha
and PKA, hippocampal levels of their phosphorylated forms (pCaMKII&alpha
and pPKA) were assessed. Rats were classified as &ldquo
good&rdquo
and &ldquo
poor&rdquo
learners on the basis of their performance in a partially baited 12-arm radial maze. The hippocampal protein levels were measured using Western Blot technique. In addition to individual variation in animals&rsquo
learning capacity, strain-depended differences have also been observed. Deficient performance recorded in inbred W rats compared to outcrossed W/S rats, and &ldquo
poor&rdquo
learners from both rat groups had predominantly related to the higher frequency of reference memory errors. The results of biochemical assays showed strain-depended differences in the NOS expression. The overall NOS levels were significantly higher in outcrossed W/S rats compared to inbred W rats. In both rat lines, the rate of learning positively correlated with hippocampal levels of nNOS and negatively correlated with iNOS levels. Hippocampal eNOS levels correlated negatively with animals&rsquo
performance but only in the W rats. These results suggested that all 3 NOS isoforms are implemented in the learning process playing, however, different roles in neural signaling. Experiments carried out on Long-Evans rats did not reveal a significant difference in the basal hippocampal levels of the CaMKII&alpha
, however, the level of the pCaMKII&alpha
, was significantly higher in &ldquo
good&rdquo
learners. Also, hippocampal levels of both PKA and pPKA, as well as that of ChAT were significantly higher in &ldquo
good&rdquo
as compared to &ldquo
poor&rdquo
learners. Taken together, the latter findings indicate that low hippocampal expression of PKA and ChAT as well as low CaMKII&alpha
or PKA activation may cause learning deficits in random population of young rats, and thus, these enzymes can be considered target molecules when looking for cognitive enhancers to treat memory deficits in young subjects.
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8

Böttiger, Anna. "Genetic variation in the folate receptor-alpha and methylenetetrahydrofolate reductase genes as determinants of plasma homocysteine concentrations." Doctoral thesis, Örebro universitet, Hälsoakademin, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-2625.

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Elevated total plasma homocysteine (tHcy) is a risk factor for cardiovascular disease and neurocognitive disease such as dementia. The B vitamins folate and B12 are the main de terminants of tHcy. tHcy concentration can also be affected by mutations in genes coding for receptors, enzymes and transporters important in the metabolism of Hcy. This thesis focuses on mutations in the genes for folate receptor-alpha and methylenetetrahydrofolate reductase (MTHFR) and the effect they have on tHcy concentrations. Six novel mutations in the gene for folate receptor-alpha were described in Paper I. Taken together they exist in a population with a prevalence of approximately 1% and thus are not unusual. There may be an association of –69dupA and –18C>T to tHcy but for the 25-bp deletion, –856C>T, –921T>C and –1043G>A there is probably no association to tHcy. Mutation screening was continued and four additional mutations, 1314G>A, 1816delC, 1841G>A and 1928C>T, were described in Paper II. The prevalences for the heterozygotes were between 0.5% and 13% in an elderly population. There was no significant difference in prevalence between the elderly subjects and patients with dementia. The 1816(–)-allele and the 1841A-allele were in complete linkage and the haplotype 1816(–)-1841A may possibly have a tHcy raising effect. The 1314G>A and 1928C>T mutations had no association to tHcy. The genotype prevalences and haplotype frequencies of the MTHFR 677C>T, 1298A>C and 1793G>A polymorphisms were determined in a population sample of Swedish children and adolescents (Paper III). The MTHFR 677T-allele was associated with increased tHcy concentrations in both children and adolescents. A small elevating effect of the 1298C-allele and a small lowering effect of the 1793A-allele could be shown. In an epidemiological sample of adults from the Canary Islands, Spain, data for serum folate and vitamin B12 were used for a broader study of the nutrigenetic impact on tHcy (Paper IV). The 677T-allele had a significant tHcy increasing effect in men but not in women. The 1298C-allele had a minor elevating effect on tHcy in men with the 677CT genotype. It was not possible to document any effect of the 1793A-allele on tHcy due to its low prevalence. A slightly superior explanatory power for the genetic impact was obtained using the MTHFR haplotypes in the analysis compared to the MTHFR 677C>T genotype-based approach in both the Swedish children and adolescents and in the Spanish adults. Therefore MTHFR haplotypes should be considered when analysing the impact of the MTHFR 677C>T, 1298A>C and 1793G>A polymorphisms on tHcy. Notwithstanding the large geographical distance between our study populations the haplotype composition is quite similar. The MTHFR 677T-allele is slightly more prevalent in Spain compared to Sweden but it has only an effect on tHcy in the Spanish men. Age, gender and factors linked to the ethnicity of the studied subjects, seem to be able to override the nutrigenetic impact of tHcy-raising genotypes or haplotypes in particular settings, such as in the Spanish women in our study. Gene-nutrient interactions on plasma tHcy levels thus may or may not exist in a certain population. The transferability of nutrigenetic findings may therefore be limited, and must be re-evaluated for each particular setting of age-gender-ethnicity.
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9

Žirgulevičiūtė, Jūratė. "P-variacijos indekso vertinimo ekonometrinis tyrimas." Master's thesis, Lithuanian Academic Libraries Network (LABT), 2009. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2008~D_20090908_201800-34444.

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Darbe taikyta Norvaišos ir Salopk (2002) metodologija funkcijos šiurkštumui nagrinėti remiantis modifikuotu funkcijos grafiko dėžučių skaičiaus indeksu. Funkcijos šiurkštumas nusakomas p-variacijos indeksu, kuris prie tam tikrų sąlygų lygus fraktalo dimensijai. Darbe ištirtos tiesinės regresijos, kuri vertina p-variacijos indeksą, liekanos ir pasiūlytas būdas kaip išpildyti balto triukšmo prielaidas. Rezultatai apibendrinti Monte Carlo procedūra. Sukonstruoti p-variacijos indekso pasikliautinieji intevalai -stabiliam ir trupmeniniam Brauno judesio procesams. Ištirtas p-variacijos indekso kintamumas laike „Vallourec” akcijų kainos procesui.
To estimate the roughness of the sample function the methodology introdused in Norvaiša and Salopek (2002) was applied. The roughness is defined as p-variation index of the sample function graph. Methodology is based on linear regression of the oscilation index. This master thesis tests the assumptions of linear regression residuals and constructs estimator which fulfill these assumptions. The model was used for the generated α-stable process and fractional Brownian motion. Conclusions are generalized using Monte-Carlo procedure. The confidence intervals for the p-variation index was constructed making assumption that the process is the realisation of -stable or fractional Brownian motion. The p-variation index was estimated for the „Vallourec” stock price data, sampled at irregular time. In addidion the variability in time of p-variation index was studied for different segments of intervals.
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10

Matharoo, Balwir. "The Glutathione S-Transferases : a study of genetic variation and development of the alpha, mu and pi isoenzymes." Thesis, Keele University, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261477.

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Books on the topic "Alpha variation"

1

Durfy, Sharon Joan. Nucleotide sequence variation , homogenization, and evolution of X chromosome alpha satellite DNA. 1990.

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Warburton, Peter Eyton. Evolution of tandemly repeated DNA: repeat unit variation of human alpha satellite DNA. 1993.

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3

Barber, Sarah. Hepatitis C virus genome variation in the NS5 region and the E2/NS1 hypervariable region: A study of genotypes and genome variability in response to alpha-interferon therapy. 1996.

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4

A, Cooper Paul, Linn Tae W, and Langley Research Center, eds. Sensitivity of space station alpha joint robust controller to structural modal parameter variations. Hampton, Va: National Aeronautics and Space Administration, Langley Research Center, 1991.

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Pfurtscheller, Gert, and Fernando Lopes da Silva. EEG Event-Related Desynchronization and Event-Related Synchronization. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0040.

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Event-related desynchronization (ERD) reflects a decrease of oscillatory activity related to internally or externally paced events. The increase of rhythmic activity is called event-related synchronization (ERS). They represent dynamical states of thalamocortical networks associated with cortical information-processing changes. This chapter discusses differences between ERD/ERS and evoked response potentials and methodologies for quantifying ERD/ERS and selecting frequency bands. It covers the interpretation of ERD/ERS in the alpha and beta bands and theta ERS and alpha ERD in behavioral tasks. ERD/ERS in scalp and subdural recordings, in various frequency bands, is discussed. Also presented is the modulation of alpha and beta rhythms by 0.1-Hz oscillations in the resting state and phase-coupling of the latter with slow changes of prefrontal hemodynamic signals (HbO2), blood pressure oscillations, and heart rate interval variations in the resting state and in relation to behavioral motor tasks. Potential uses of ERD-based strategies in stroke patients are discussed.
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Aspden, Richard, and Jenny Gregory. Morphology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0011.

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The study of joint morphology can help us to understand the risk factors for osteoarthritis (OA), how it progresses, and aids in developing imaging biomarkers for study of the disease. OA results in gross structural changes in affected joints. Growth of osteophytes, deformation of joint components, and loss of joint space where cartilage has broken down are all characteristics of the disorder. Certain bone shapes as well as malalignment predispose people to future OA, or may be a marker for early OA. Geometrical measures, such as the alpha angle or Wiberg’s CE angle, used to be the primary tool for investigating morphology. In recent years, however, statistical shape modelling (SSM) has become increasingly popular. SSM can be used with any imaging modality and has been successfully applied to a number of musculoskeletal conditions. It uses sets of landmark points denoting the anatomy of one or more bones to generate new variables (modes) that describe and quantify the shape variation in a set of images via principal components analysis. With the aid of automated search algorithms for point placement, the use of SSMs is expanding and provides a valuable and versatile tool for exploration of bone and joint morphometry. Whilst the majority of research has focused on hip and knee OA, this chapter provides an overview of joint morphology through the whole skeleton and how it has helped our ability to understand and quantify the risk and progression of osteoarthritis.
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Book chapters on the topic "Alpha variation"

1

Cu, Suong, Sophia Roumeliotis, and Jason Eglinton. "Alpha-Amylase Allelic Variation in Domesticated and Wild Barley." In Advance in Barley Sciences, 57–68. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-4682-4_5.

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Cavender-Bares, Jeannine, Anna K. Schweiger, Jesús N. Pinto-Ledezma, and Jose Eduardo Meireles. "Applying Remote Sensing to Biodiversity Science." In Remote Sensing of Plant Biodiversity, 13–42. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-33157-3_2.

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AbstractBiodiversity is organized hierarchically from individuals to populations to major lineages in the tree of life. This hierarchical structure has consequences for remote sensing of plant phenotypes and leads to the expectation that more distantly related plants will be more spectrally distinct. Applying remote sensing to understand ecological processes from biodiversity patterns builds on prior efforts that integrate functional and phylogenetic information of organisms with their environmental distributions to discern assembly processes and the rules that govern species distributions. Spectral diversity metrics critical to detecting biodiversity patterns expand on the many metrics for quantifying multiple dimensions of biodiversity—taxonomic, phylogenetic, and functional—and can be applied at local (alpha diversity) to regional (gamma diversity) scales to examine variation among communities (beta diversity). Remote-sensing technologies stand to illuminate the nature of biodiversity-ecosystem function relationships and ecosystem service trade-offs over large spatial extents and to estimate their uncertainties. Such advances will improve our capacity to manage natural resources in the Anthropocene.
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Barrow, John D. "Constants and Variations: From Alpha to Omega." In The Cosmology of Extra Dimensions and Varying Fundamental Constants, 207–22. Dordrecht: Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-017-3272-7_29.

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Wilson, Stephen, Rebecca S. Ginger, Tony Dadd, David Gunn, Fei-Ling Lim, Magdalena Sawicka, Melanie Sandel, Paul P. M. Schnetkamp, and Martin R. Green. "NCKX5, a Natural Regulator of Human Skin Colour Variation, Regulates the Expression of Key Pigment Genes MC1R and Alpha-MSH and Alters Cholesterol Homeostasis in Normal Human Melanocytes." In Advances in Experimental Medicine and Biology, 95–107. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_9.

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Invitto, Sara, Antonio Della Torre, and Rosaria Rinaldi. "Neuroprosthetic Haptic Interface and Haptic Stimulation: Neuromorphic Microtransduction and EEG Alpha Variations." In Converging Clinical and Engineering Research on Neurorehabilitation III, 967–71. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-01845-0_194.

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Nusinov, A. A., and V. V. Katyushina. "Lyman-Alpha Line Intensity as a Solar Activity Index in the Far Ultraviolet Range." In The Sun as a Variable Star: Solar and Stellar Irradiance Variations, 201–6. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0950-5_31.

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Gotoda, Takeshi, and Takashi Sakajo. "Enstrophy Variations in the Incompressible 2D Euler Flows and $$\alpha $$ α Point Vortex System." In Mathematical Fluid Dynamics, Present and Future, 401–31. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56457-7_14.

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Seta, N., G. Durand, J. Agneray, J. Féger, and M. Corbic. "VARIATIONS IN THE PROPORTIONS OF MICROHETEROGENEOUS FORMS OF HUMAN ALPHA 1-ACID GLYCOPROTEIN IN ALCOHOLIC CIRRHOSIS." In Proceedings of the Third Symposium, Lyon, France, June 26–28, 1985, edited by Jacques Bienvenu, J. A. Grimaud, and Philippe Laurent, 673–78. Berlin, Boston: De Gruyter, 1986. http://dx.doi.org/10.1515/9783110860757-083.

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Williams, R. S., G. S. Schultz, T. E. Geoghegan, M. C. Steffan, and M. A. Yussman. "Variations in the Level of Transforming Growth Factor-Alpha (TGFα) mRNA During the Human Menstrual Cycle." In Growth Factors and the Ovary, 205–8. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5688-2_20.

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Pany, Gayatri, and Sabyasachi Pani. "Nonlinear Mixed Variational-Like Inequality with Respect to Weakly Relaxed $$\eta -\alpha $$ Monotone Mapping in Banach Spaces." In Mathematical Analysis and its Applications, 185–96. New Delhi: Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-2485-3_14.

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Conference papers on the topic "Alpha variation"

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Banchuin, Rawid, and Rounsan Chaisricharoen. "Alpha power law based model of random variation in nanometer FGMOSFET." In 2017 International Conference on Digital Arts, Media and Technology (ICDAMT). IEEE, 2017. http://dx.doi.org/10.1109/icdamt.2017.7904971.

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AlSalhi, M. S., W. A. Farooq, M. R. Baig, A. H. Al-Fareikh, and S. S. Al-Ghamdi. "CO2 laser induced micro structural variation in alpha-irradiated Polyallydigycol polymer." In 2011 Saudi International Electronics, Communications and Photonics Conference (SIECPC). IEEE, 2011. http://dx.doi.org/10.1109/siecpc.2011.5876927.

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Leroy, Damien, Rémi Gaillard, Erwin Schaefer, Cyrille Beltrando, Shi-Jie Wen, and Richard Wong. "Variation of SRAM Alpha-Induced Soft Error Rate with Technology Node." In 2008 14th IEEE International On-Line Testing Symposium (IOLTS). IEEE, 2008. http://dx.doi.org/10.1109/iolts.2008.38.

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Gonçalves, Gil, Sara Cabral, and António Paulo Lopes. "Annual FEV1% variation before and after augmentation therapy in severe alpha-1-antitrypsin deficiency." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4238.

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Huang, X. "387 Estrogen-related receptor alpha (ESRRA) copy number variation is associated with histological grade in ovarian cancer." In IGCS 2020 Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2020. http://dx.doi.org/10.1136/ijgc-2020-igcs.334.

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Pett, Ryan G., Courtney M. Wheatley, Mary A. Morgan, Eric C. Wong, William T. Foxx-Lupo, Cori L. Daines, Wayne Morgan, and Eric M. Snyder. "Influence Of Genetic Variation Of The Alpha-Subunit Of Enac On Lung Diffusion In Patients With Cystic Fibrosis." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1120.

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Kiss, G. G., D. Galaviz, Gy Gyürky, Z. Elekes, Zs Fülöp, E. Somorjai, K. Sonnabend, et al. "Experimental study of the variation of alpha elastic scattering cross sections along isotopic and isotonic chains at low energies." In ORIGIN OF MATTER AND EVOLUTION OF GALAXIES: The 10th International Symposium on Origin of Matter and Evolution of Galaxies: From the Dawn of Universe to the Formation of Solar System. AIP, 2008. http://dx.doi.org/10.1063/1.2943577.

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Smith, Isaiah E., Amanda Lynn Godbold, and David J. Bottjer. "QUANTIFYING THE INFLUENCE OF INTER-OBSERVER VARIATION AND MEDIUM TYPE (THIN-SECTION VERSUS POLISHED SLAB) ON ALPHA DIVERSITY ESTIMATES." In GSA Annual Meeting in Phoenix, Arizona, USA - 2019. Geological Society of America, 2019. http://dx.doi.org/10.1130/abs/2019am-337566.

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Hamza, Mohamed, Tarek M. Hatem, Dierk Raabe, and Jaafar A. El-Awady. "Hydrogen Diffusion and Segregation in Alpha Iron ∑ 3 (111) Grain Boundaries." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-53118.

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Polycrystalline material generally exhibits degradation in its mechanical properties and shows more tendency for intergranular fracture due to segregation and diffusion of hydrogen on the grain boundaries (GBs). Understanding the parameters affecting the diffusion and binding of hydrogen within GBs will allow enhancing the mechanical properties of the commercial engineering materials and developing interface dominant materials. In practice during forming processes, the coincidence site lattice (CSL) GBs are experiencing deviations from their ideal configurations. Consequently, this will change the atomic structural integrity by superposition of sub-boundary dislocation networks on the ideal CSL interfaces. For this study, the ideal ∑ 3 111 [110] GB structure and its angular deviations in BCC iron within the range of Brandon criterion will be studied comprehensively using molecular statics (MS) simulations. The clean GB energy will be quantified, followed by the GB and free surface segregation energies calculations for hydrogen atoms. Rice-Wang model will be used to assess the embrittlement impact variation over the deviation angles. The results showed that the ideal GB structure is having the greatest resistance to embrittlement prior GB hydrogen saturation, while the 3° deviated GB is showing the highest susceptibility to embrittlement. Upon saturation, the 5° deviated GB appears to have the highest resistance instead due to the lowest stability of hydrogen atoms observed in the free surfaces of its simulation cell. Molecular dynamics (MD) simulations are then applied to calculate hydrogen diffusivity within the ideal and deviated GB structure. It is shown that hydrogen diffusivity decreases significantly in the deviated GB models. In addition, the 5° deviated GB is representing the local minimum for diffusivity results suggesting the existence of the highest atomic disorder and excessive secondary dislocation accommodation within this interface.
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Li, Zhiming, Zhihe Wang, and Zhiyun Xiao. "Alpha Discharge Electron Concentration and Ionization Rate Effect on Spatial Intensity Variation between the All-Metal Slab Waveguide Electrodes of CO2 Laser." In 2011 Symposium on Photonics and Optoelectronics (SOPO 2011). IEEE, 2011. http://dx.doi.org/10.1109/sopo.2011.5780621.

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Reports on the topic "Alpha variation"

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Khaled, Safinaz A., Luc Damé, Mohamed A. Semeida, Magdy Y. Amin, Ahmed Ghitas, Shahinaz Yousef, and Penka Stoeva. Variations of the Hydrogen Lyman Alpha Line throughout Solar Cycle 24 on ESA/PROBA-2 and SORCE/SOLSTICE Data. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, September 2020. http://dx.doi.org/10.7546/crabs.2020.09.10.

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