Relevant bibliographies by topics / Alpha Complex

Academic literature on the topic 'Alpha Complex'

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Published: 6 September 2023

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Journal articles on the topic "Alpha Complex"

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Buenafe, A. C., R. C. Tsu, B. Bebo, A. C. Bakke, A. A. Vandenbark, and H. Offner. "A TCR V alpha CDR3-specific motif associated with Lewis rat autoimmune encephalomyelitis and basic protein-specific T cell clones." Journal of Immunology 158, no. 11 (June 1, 1997): 5472–83. http://dx.doi.org/10.4049/jimmunol.158.11.5472.

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Abstract To investigate TCR V alpha gene expression in the Lewis rat model of experimental autoimmune encephalomyelitis, we obtained V alpha chain sequences from two V beta8.2+-encephalitogenic, BP72-89-specific T cell clones. Two different V alpha genes, a V alpha2 gene and a V alpha23 gene, are utilized, but both were found to contain an asparagine repeat (Asn3+) sequence present in the V alpha CDR3 region. This Asn3+ motif is also present in the previously reported sequence of a BP68-88-specific hybridoma, 510, which utilizes a different V alpha2 gene family member. In further experiments, spinal cord T cells were isolated at the onset of basic protein (BP)-induced disease and sorted for the OX-40 activation marker, which we have previously used to enrich for specifically activated T cells. Analysis of V alpha expression in the OX-40+ population revealed the biased use of three V alpha genes, V alpha1, V alpha2, and V alpha23. The Asn3+ motif was present in the V alpha CDR3 region of V alpha1, V alpha2, and V alpha23 cDNA derived from OX-40+ spinal cord T cells but found to be generally absent in the OX-40- spinal cord population. Since these Asn3+ motif-bearing V alpha chain sequences are nearly identical to those utilized by the BP-specific encephalitogenic clones described, it is likely that these V alpha sequences are derived from disease-associated T cells in the spinal cord. Thus, we demonstrate that the Asn3+ V alpha CDR3 motif is strongly associated with experimental autoimmune encephalomyelitis in the Lewis rat and propose that it plays a role in TCR recognition of a specific BP peptide/MHC complex.
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Harashima, S., A. M. Miller, K. Tanaka, K. Kusumoto, K. Tanaka, Y. Mukai, K. Nasmyth, and Y. Oshima. "Mating-type control in Saccharomyces cerevisiae: isolation and characterization of mutants defective in repression by a1-alpha 2." Molecular and Cellular Biology 9, no. 10 (October 1989): 4523–30. http://dx.doi.org/10.1128/mcb.9.10.4523-4530.1989.

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The alpha 2 protein, the product of the MAT alpha 2 cistron, represses various genes specific to the a mating type (alpha 2 repression), and when combined with the MATa1 gene product, it represses MAT alpha 1 and various haploid-specific genes (a1-alpha 2 repression). One target of a1-alpha 2 repression is RME1, which is a negative regulator of a/alpha-specific genes. We have isolated 13 recessive mutants whose a1-alpha 2 repression is defective but which retain alpha 2 repression in a genetic background of ho MATa HML alpha HMRa sir3 or ho MAT alpha HMRa HMRa sir3. These mutations can be divided into three different classes. One class contains a missense mutation, designated hml alpha 2-102, in the alpha 2 cistron of HML, and another class contains two mat alpha 2-202, in the MAT alpha locus. These three mutants each have an amino acid substitution of tyrosine or acid substitution of tyrosine or phenylalanine for cysteine at the 33rd codon from the translation initiation codon in the alpha 2 cistron of HML alpha or MAT alpha. The remaining 10 mutants make up the third class and form a single complementation group, having mutations designated aar1 (a1-alpha 2 repression), at a gene other than MAT, HML, HMR, RME1, or the four SIR genes. Although a diploid cell homozygous for the aarl and sir3 mutations and for the MATa, HML alpha, and HMRa alleles showed alpha mating type, it could sporulate and gave rise to asci containing four alpha mating-type spores. These facts indicate that the domain for alpha2 repression is separable from that for a1-alpha2 protein interaction or complex formation in the alpha2 protein and that an additional regulation gene, AAR1, is associated with the a1-alpha2 repression of the alpha1 cistron and haploid-specific genes.
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Harashima, S., A. M. Miller, K. Tanaka, K. Kusumoto, K. Tanaka, Y. Mukai, K. Nasmyth, and Y. Oshima. "Mating-type control in Saccharomyces cerevisiae: isolation and characterization of mutants defective in repression by a1-alpha 2." Molecular and Cellular Biology 9, no. 10 (October 1989): 4523–30. http://dx.doi.org/10.1128/mcb.9.10.4523.

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The alpha 2 protein, the product of the MAT alpha 2 cistron, represses various genes specific to the a mating type (alpha 2 repression), and when combined with the MATa1 gene product, it represses MAT alpha 1 and various haploid-specific genes (a1-alpha 2 repression). One target of a1-alpha 2 repression is RME1, which is a negative regulator of a/alpha-specific genes. We have isolated 13 recessive mutants whose a1-alpha 2 repression is defective but which retain alpha 2 repression in a genetic background of ho MATa HML alpha HMRa sir3 or ho MAT alpha HMRa HMRa sir3. These mutations can be divided into three different classes. One class contains a missense mutation, designated hml alpha 2-102, in the alpha 2 cistron of HML, and another class contains two mat alpha 2-202, in the MAT alpha locus. These three mutants each have an amino acid substitution of tyrosine or acid substitution of tyrosine or phenylalanine for cysteine at the 33rd codon from the translation initiation codon in the alpha 2 cistron of HML alpha or MAT alpha. The remaining 10 mutants make up the third class and form a single complementation group, having mutations designated aar1 (a1-alpha 2 repression), at a gene other than MAT, HML, HMR, RME1, or the four SIR genes. Although a diploid cell homozygous for the aarl and sir3 mutations and for the MATa, HML alpha, and HMRa alleles showed alpha mating type, it could sporulate and gave rise to asci containing four alpha mating-type spores. These facts indicate that the domain for alpha2 repression is separable from that for a1-alpha2 protein interaction or complex formation in the alpha2 protein and that an additional regulation gene, AAR1, is associated with the a1-alpha2 repression of the alpha1 cistron and haploid-specific genes.
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4

de Bairros, Thiago, Pedro Pereira, Rausley de Souza, and Michel Yacoub. "Bivariate Complex $\alpha$-$\mu$ Statistics." IEEE Transactions on Vehicular Technology 71, no. 3 (March 2022): 3276–80. http://dx.doi.org/10.1109/tvt.2022.3141232.

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REX SHEU, KWAN-FU, and JOHN P. BLASS. "The alpha-Ketoglutarate Dehydrogenase Complex." Annals of the New York Academy of Sciences 893, no. 1 OXIDATIVE/ENE (November 1999): 61–78. http://dx.doi.org/10.1111/j.1749-6632.1999.tb07818.x.

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Pashaei, Ronak, Amir Pishkoo, Mohammad Sadegh Asgari, and Davood Ebrahimi Bagha. "$\alpha$-Differentiable functions in complex plane." Вестник Самарского государственного технического университета. Серия «Физико-математические науки» 24, no. 2 (2020): 379–89. http://dx.doi.org/10.14498/vsgtu1734.

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В комплексной плоскости вводится взвешенная дробная производная порядка $\alpha$. Относительно многозначной функции $ z ^ {1- \alpha} $ получены дробные уравнения Коши-Римана, которые при $ \alpha = 1 $ совпадают с классическими уравнениями Коши-Римана. Для некоторых функций в комплексной плоскости рассмотрены свойства, относящиеся к комплексной взвешенной дробной производной. Обсуждаются два комплексных дифференциальных уравнения специальной формы. Для некоторых значений $\alpha$ приводятся римановы поверхности их решений и сравниваются их графики.
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Moretti, D. V., A. Prestia, G. Binetti, O. Zanetti, and G. B. Frisoni. "Alpha and Theta EEG Rhythms Activity Reveal Deep Changes in Resting Brain State in Subjects with Prodromal Alzheimer's Disease." Journal of Aging and Gerontology 2, no. 1 (February 5, 2014): 13–23. http://dx.doi.org/10.12974/2309-6128.2014.02.01.3.

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The increase of EEG alpha3/alpha2 frequency power ratio has been associated with AD-converters subjects with mild cognitive impairment (MCI) as well as a reduction in the regional cerebral blood flow (rCBF). In this study, the association between alpha3/alpha2 frequency power ratio with rCBF changes in subjects with MCI was evaluated. The alpha3/alpha2 frequency power ratio was computed in 27 subjects with MCI. Two groups were obtained according to the median values of alpha3/alpha2, at a cut-off of 1.17. In the groups so obtained, the correlation between brain perfusion and EEG markers were detected. In the MCI group with the alpha3/alpha2 frequency power ratio above 1,17 as compared to the group with alpha3/alpha2 frequency power ratio below 1.17 there was: 1) a constant trend to lower rCBF values; 2) smaller hippocampal volumes; 3) higher theta frequency power. The higher EEG alpha3 /alpha2 frequency power ratio individuates two different group of MCI subjects. A complex interplay between alpha and theta rhythms activity MCI patients is suggested in patients with prodromal Alzheimer's disease.
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Forster, B., R. Garunkštis, P. Massopust, and J. Steuding. "Complex B-splines and Hurwitz zeta functions." LMS Journal of Computation and Mathematics 16 (2013): 61–77. http://dx.doi.org/10.1112/s146115701300003x.

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AbstractWe characterize nonempty open subsets of the complex plane where the sum $\zeta (s, \alpha )+ {e}^{\pm i\pi s} \hspace{0.167em} \zeta (s, 1- \alpha )$ of Hurwitz zeta functions has no zeros in $s$ for all $0\leq \alpha \leq 1$. This problem is motivated by the construction of fundamental cardinal splines of complex order $s$.
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KUROKAWA, CHIE, and KIYOSHI KATŌ. "THREE-ALPHA STRUCTURES AND ALPHA-CONDENSATION IN 12C." Modern Physics Letters A 18, no. 02n06 (February 28, 2003): 162–65. http://dx.doi.org/10.1142/s0217732303010168.

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The 3α resonances in 12 C are investigated in a framework of the complex scaling method. Calculated resonance parameters well reproduce the experimental data and we can obtain the second 2+ state just above the third 0+ state.
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Green, Colin P., and Peter Osborne. "ALPHA-ACID-1,2-DIAMINOBENZENE COMPLEX: A CONVENIENT STANDARD FOR ALPHA-ACID ANALYSIS." Journal of the Institute of Brewing 99, no. 4 (July 8, 1993): 347–48. http://dx.doi.org/10.1002/j.2050-0416.1993.tb01174.x.

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Dissertations / Theses on the topic "Alpha Complex"

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Richardson, John Lee. "Structural and kinetic characterization of the leech derived inhibitor haemadin in complex with human [alpha]-thrombin [alpha-thrombin] structural analysis of the tsetse thrombin inhibitor in complex with bovine [alpha]-thrombin [alpha-thrombin] /." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=965976335.

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Ascolani, Gianluca. "EEG, Alpha Waves and Coherence." Thesis, University of North Texas, 2010. https://digital.library.unt.edu/ark:/67531/metadc28389/.

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This thesis addresses some theoretical issues generated by the results of recent analysis of EEG time series proving the brain dynamics are driven by abrupt changes making them depart from the ordinary Poisson condition. These changes are renewal, unpredictable and non-ergodic. We refer to them as crucial events. How is it possible that this form of randomness be compatible with the generation of waves, for instance alpha waves, whose observation seems to suggest the opposite view the brain is characterized by surprisingly extended coherence? To shed light into this apparently irretrievable contradiction we propose a model based on a generalized form of Langevin equation under the influence of a periodic stimulus. We assume that there exist two different forms of time, a subjective form compatible with Poisson statistical physical and an objective form that is accessible to experimental observation. The transition from the former to the latter form is determined by the brain dynamics interpreted as emerging from the cooperative interaction among many units that, in the absence of cooperation would generate Poisson fluctuations. We call natural time the brain internal time and we make the assumption that in the natural time representation the time evolution of the EEG variable y(t) is determined by a Langevin equation perturbed by a periodic process that in this time representation is hardly distinguishable from an erratic process. We show that the representation of this random process in the experimental time scale is characterized by a surprisingly extended coherence. We show that this model generates a sequence of damped oscillations with a time behavior that is remarkably similar to that derived from the analysis of real EEG's. The main result of this research work is that the existence of crucial events is not incompatible with the alpha wave coherence. In addition to this important result, we find another result that may help our group, or any other research group working on the analysis of brain's dynamics, to prove or to disprove the existence of crucial events. We study the diffusion process generated by fluctuations emerging from the same model after filtering out the alpha coherence, and we study the recursion to the origin. We study the survival probability of this process, namely the probability that up to a given time no re-crossing of the origin occurs. We find that this is an inverse power law with a power that depends on whether or not crucial events exist.
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Niranjan, Adityanarayan C. "Normalization of Complex Mode Shapes by Truncation of the Alpha-Polynomial." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1448037029.

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Cingolani, Gino. "Biochemical and crystallographic analysis of the human importin [alpha]/[bêta] complex." Grenoble 1, 1999. http://www.theses.fr/1999GRE10096.

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Le travail presente dans cette these decrit l'analyse structurale du complexe forme par les proteines humaines et importin. Le projet a debute par la caracterisation biochimique des 2 proteines, seules en solution, et du complexe qu'elles forment. Nous avons observe que dans le complexe la proteine importin est resistante a la chymotrypsine. La meme protection a ete observee lorsque la proteine -importin est incubee en presence d'un peptide, synthetise chimiquement, contenant les domaines ibb de la proteine importin. La co-cristallisation de la proteine -importin avec le peptide a donne naissance a 2 formes cristallines dans le groupe d'espace p21, diffractant respectivement a une resolution de 2. 5a et 2. 3a. La structure du complexe a ete resolue dans la premiere forme cristalline par diffusion anomale en utilisant la proteine -importin exprimee en presence de selenio-methionine. La structure a ensuite ete affinee a 2. 5 a de resolution. La deuxieme forme cristalline a ete resolue par remplacement moleculaire et le modele affine jusqu'a 2. 3 a de resolution. Un important changement conformationel a ete observe entre les deux formes cristallines donnant des informations sur la flexibilite interieure de la proteine importin. L'analyse de la structure du complexe ainsi que la comparaison avec les donnees biochimiques obtenues nous ont permis 4 observations majeures : - l'architecture caracteristique des proteines de la superfamille- a ete observe pour la premiere fois. - l'interaction intime entre les proteines et -importin a ete decrite au niveau moleculaire. - le double changement conformationel des proteines et importin observe par les methodes biochimiques a pu etre confirme grace a la structure tridimensionnelle. - une similarite de structure entre le complexe importin : domaines ibb et importin : sequence de localisation nucleaire (nls) a ete observe, confirmant une possible evolution commune de ces proteines.
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Barran, Paul Arthur. "The structure and transcription of a rat RT1 B alpha class II gene." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/26957.

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The major histocompatibility complex of the rat (RT1 complex) encodes two sets of class II molecules referred to as RT1 B and RT1 D. The RT1 Bα gene was isolated from a Sprague-Dawley (RT1b) rat genomic library using a rat RT1 Bα chain cDNA as a hybridization probe. The coding and the majority of the intron DNA sequence was determined. The structure of the RT1 Bα gene is equivalent to that of H-2 and HLA a chain genes. Comparison of the nucleotide and predicted amino acid sequences of the RT1 Bα gene to those of the H-2 and HLA genes revealed a high degree of overall sequence conservation. However, two regions of the first external domain (a1), residues 19-23 and 45-78, exhibit marked sequence variation. Two blocks of conserved nucleotide sequence were identified in the 5' promoter region of the RT1 Bα gene that have been described in all MHC class II genes sequenced to date. These conserved sequences may be involved in the co-ordinate regulation of expression of class II genes. The cloned RT1 Bα gene was efficiently transcribed when transfected into mouse L cells.
Medicine, Faculty of
Medical Genetics, Department of
Graduate
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6

Hurt, Nicholas S. "Electronic detection of DNA polymerase complex formation and dissociation using an alpha-hemolysin nanopore /." Diss., Digital Dissertations Database. Restricted to UC campuses, 2009. http://uclibs.org/PID/11984.

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Sikarwar, Anurag Singh. "Post-translational modifications of thromboxane receptor G-protein alpha q complex in hypoxic PPHN." American Thoracic Society, 2014. http://hdl.handle.net/1993/31664.

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Introduction: Persistent pulmonary hypertension of the newborn (PPHN) is associated with an elevated thromboxane to prostacyclin ratio, pulmonary artery (PA) hyperreactivity and hypersensitivity. Thromboxane receptor (TP), coupling with G-protein Gαq causes pulmonary vasoconstriction; whereas prostacyclin receptor (IP), coupling with Gαs, causes vasodilation and TP phosphorylation via adenylyl cyclase (AC)-cAMP-protein kinase A (PKA), desensitizes TP. Both TP phosphorylation and Gαq palmitoylation play major roles in regulation of signaling through the TP-Gαq complex. We hypothesized that increased Gαq palmitoylation and decreased AC activity could cause hypoxic TP hyperresponsiveness. We studied the impact of hypoxia on selected post-translational modifications of the receptor-G-protein complex, determining TP vasoconstriction: Gαq palmitoylation, TP phosphorylation and upstream AC activity. Methods: Force responses to thromboxane mimetic U46619, palmitoylation inhibition by 2-bromopalmitate (2-BP) and AC activation (forskolin) were studied by myography in hypoxic PPHN and control newborn swine pulmonary artery. Ca2+ mobilization was studied by fluorescent calcium indicators fura-2AM in pulmonary myocytes (PASMC), and fluo-4NW in HEK293 cells. Effects of hypoxia on Gαq palmitoylation were studied by metabolic labeling. Gαq cysteines and TP serines were mutated to determine sites of post-translational modifications. Protein expression and receptor-G-protein coupling were studied by Western blot and co-immunoprecipitation. PKA activity was assayed; and AC activity quantified. Results: Hypoxia increases Gαq palmitoylation, without increasing total palmitate uptake. Palmitoylation inhibition decreases U46619-stimulated force generation as well as Ca2+ mobilization in PPHN PA rings and hypoxic PASMC. Mutation of palmitoylable cysteine and palmitoylation inhibition proportionately decrease U46619-mediated Ca2+ mobilization in HEK293 cells. TP serine phosphorylation is decreased by hypoxia due to decreased PKA activity; this causes TP hypersensitivity and hyper-reactivity. Serine 324 of TPα is the target of PKA-mediated desensitization. AC activator-induced relaxation is reduced in PPHN PA. Basal and receptor-stimulated AC activity are decreased in hypoxic PASMC. Decreased AC activity is not due to decreased AC expression, ATP availability nor increased Gαi activation. Conclusion: Increased Gαq palmitoylation plays a role in TPα hyper-responsiveness in hypoxic PPHN. Hypoxia also reduces responses to agents acting through AC, unleashing TP-mediated vasoconstriction. Reactivation of pulmonary AC might be useful therapeutically to promote vasodilation and TP desensitization.
October 2016
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Huart, Anne-Sophie. "Casein kinase 1 alpha-MDM2 complex : phosphorylation and ubiquitination signals converging on p53 pathway." Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/17282.

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The tumour suppressor p53 is a key regulatory protein that prevents proliferation of damaged cells. Under unperturbed conditions, the ubiquitin ligase murine double minute 2 (MDM2) mediates p53 ubiquitination and further degradation by the proteasome. In consequence p53 is present at low levels, but becomes rapidly stabilised and activated in response to a variety of stimuli, such as DNA damage or virus infection. P53 responds to these diverse stresses to regulate the expression of many target genes that induce cell cycle arrest, DNA repair, or apoptosis. The attenuation of p53 interaction with MDM2 is maintained by enzymes catalysing p53 post-translational modifications such as phosphorylation. Casein kinase 1 α (CK1α) is one such enzyme; it stimulates p53 after DNA virus infection. Surprisingly depletion of CK1α using small interfering RNA or inhibition using a CK1 kinase inhibitor activated the transcription factor p53, indicating that p53 steady-state level is controlled by CK1α. Disrupting MDM2-p53 interaction using small molecule Nutlin-3 displayed similar pharmacological properties to the CK1 inhibitor on p53, indicating that the MDM2-CK1α complex co-regulates p53 stability. Indeed co-immunoprecipitation of endogenous CK1α with MDM2 occurred in undamaged cells. CK1α was shown in vitro to directly bind to and phosphorylate MDM2. Therefore it appears that CK1α must be recruited into specific complexes under different conditions, which can influence its substrate selectivity and explain its dual role on the p53 pathway. Apart from CK1, there are few other kinases whose action can directly contribute to the inhibition of p53. A novel pyrazolo-pyridine analogue showing dual activity against CK1 and Checkpoint kinase 1 led to increased p53 activation. These data highlighted the potential value of dual kinase inhibitors as therapeutics in cancer. The dominant protein-protein interface that stabilises the MDM2-CK1α complex was mapped using a peptide-based approach. One CK1α peptide bound strongly to MDM2, it specifically disrupted the protein-protein interaction, and its transfection was able to reduce cancer cell growth. A peptide phage display approach was finally combined with Next-Generation Sequencing to define the change in MDM2 binding motifs when the CK1α peptide or Nutlin-3 is bound, compared to ligand-free MDM2, and thus will help to understand protein-protein interaction network re-wirings which led to cell growth inhibition.
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Golub, M. V. "Age features of the power spectrum of alpha-band eeg during complex mental activities." Thesis, Sumy State University, 2017. http://essuir.sumdu.edu.ua/handle/123456789/53943.

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Urgency: Electroencephalographic analysis is one of the most informative methods of study of the systemic organization of integrative processes of the human brain in different functional States, mental activity, attention. Objective: to Study the age peculiarities of the organization of the cerebral cortex in the alpha sub-bands with complex mental activities with verbal and figurative components.
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Romano, Dina Lynn. "Characterization of alpha-cyclodextrin inclusion complexes with trans-cinnamic acid in an acid-based beverage system." Thesis, Virginia Tech, 2008. http://hdl.handle.net/10919/42111.

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In response to a need for a natural antimicrobial to replace sodium benzoate, cinnamic acid was chosen. Due to cinnamic acidâ s solubility issues, α-cyclodextrin was used as a host molecule to form an inclusion complex with the cinnamic acid molecule. The cinnamic acid: α-cyclodextrin inclusion complex was then characterized using phase solubility analysis, proton nuclear magnetic resonance (H-NMR), and solid inclusion. Phase solubility analysis verified the maximum amount of cinnamic acid that α-cyclodextrin was able to host. H-NMR was used to determine the complex association constant, determine the chemical shifts of available protons, and yield a stoichiometry for the complex. The solid inclusion complex allowed for a physical formation of the complex, yielding further information in support of the complex stoichiometry. Microbiological tests were also performed to quantify the antimicrobial abilities of the complex, the guest, and the host against the yeast Saccharomyces cerevisiae and mold Paecilomyces variotii. Results indicated that approximately 990.29 ppm in aqueous solution was the maximum amount of cinnamic acid in the complex. The 2:1 stoichiometry yields an association constant of 21.7 M-1. Results also indicated that the cinnamic acid readily conformed to fit within the α-cyclodextrin host molecule, which remained a rigid structure. An 8.9% weight to weight of cinnamic acid was calculated for the solid inclusion again reinforcing a 2:1 stoichiometry. Microbiological studies showed little to no inhibition power by the complex at varying concentrations against S. cerevisiae and P. variotii. Free cinnamic acid showed greater antimicrobial activity compared with free α-cyclodextrin and the complex.
Master of Science in Life Sciences
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Books on the topic "Alpha Complex"

1

Martin, Loren John. Analgesic properties of complex magnetic fields: Pharmacological assessment of the [My] receptor and [Alpha]-2 noradrenergic receptor hypotheses. Sudbury, Ont: Laurentian University, Behavioural Neuroscience Program, 2002.

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Wardle, Antony Morley. An investigation of the effect of alloying additions and thermomechanical processing routes on a complex near-alpha titanium alloy. Birmingham: University of Birmingham, 1985.

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Allen, Julia Margaret. Part 1. Diaziridinium Ions: First Reported Synthesis and Reactivity Studies. Part 2. Tropylium Ion Mediated alpha-Cyanation of Amines. Part 3. Multicatalytic Synthesis of Complex Tetrahydrofurans. [New York, N.Y.?]: [publisher not identified], 2011.

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chen, Shihao. Transcriptional regulation of the human UDP-GlcNac:[alpha]-6-D-mannoside [beta]-1, 2-N-acetylglucosaminyltransferase II gene (MGAT2) which encodes an enzyme that controls complex N-glycan synthesis. Ottawa: National Library of Canada, 1996.

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J, Reed Lester, Roche Thomas E, and Patel Mulchand S, eds. Alpha-keto acid dehydrogenase complexes: Organization, regulation, and biomedical ramifications : a tribute to Lester J. Reed. New York, N.Y: New York Academy of Sciences, 1989.

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Hanrahan, Gareth. Alpha Complex Nights. Mongoose Publishing, 2007.

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S, Patel Mulchand, Roche Thomas E, and Harris Robert A. 1939-, eds. Alpha-keto acid dehydrogenase complexes. Boston: Birkhäuser Verlag, 1996.

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Chen, Guang-Wu. Complex three-dimensional turbulent flow phenomena of high-alpha projectile aerodynamics. 1995.

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Chun, Kathy M. Characterization of mutations in the E1[alpha] subunit of the pyruvate dehydrogenase complex. 1995.

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An Investigation of the Separation of the Componets of the H-alpha Complex of Hydrogen. Hassell Street Press, 2021.

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Book chapters on the topic "Alpha Complex"

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Cheng, Ho-lun, and Tony Tan. "Subdividing Alpha Complex." In FSTTCS 2004: Foundations of Software Technology and Theoretical Computer Science, 186–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-540-30538-5_16.

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Nitschke, Felix, and Peter Schmieder. "Analyses of Covalent Modifications in α-Glucans." In Enzymology of Complex Alpha-Glucans, 34–60. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-3.

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Donohue, Katherine J., Andrea Kuchtova, Craig W. Vander Kooi, and Matthew S. Gentry. "Reversible Phosphorylation in Glycogen and Starch." In Enzymology of Complex Alpha-Glucans, 163–80. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-7.

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Putaux, Jean-Luc. "Morphological and Structural Aspects of α-Glucan Particles from Electron Microscopy Observations." In Enzymology of Complex Alpha-Glucans, 1–17. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-1.

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Ernes, Michael J., Gregory J. MacNeill, and Ian J. Tetlow. "Heteromeric Protein Interactions in Starch Synthesis." In Enzymology of Complex Alpha-Glucans, 259–89. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-11.

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Colleoni, Christophe. "Storage Polysaccharide Metabolism in Micro-Organisms." In Enzymology of Complex Alpha-Glucans, 61–83. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-4.

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Bertoft, Eric. "Starch Granules and their Glucan Components." In Enzymology of Complex Alpha-Glucans, 181–211. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-8.

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Annunziata, Maria Grazia, and John Edward Lunn. "Regulation of Assimilatory Starch Metabolism by Cellular Carbohydrate Status." In Enzymology of Complex Alpha-Glucans, 212–34. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-9.

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Tokarz, Danielle, Richard Cisek, and Virginijus Barzda. "Polarimetric Nonlinear Microscopy of Starch Granules." In Enzymology of Complex Alpha-Glucans, 18–33. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-2.

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Nakamura, Yasunori. "Reserve Starch Metabolism." In Enzymology of Complex Alpha-Glucans, 235–58. First edition. Boca Raton : CRC Press, Taylor & Francis: CRC Press, 2021. http://dx.doi.org/10.1201/b22412-10.

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Conference papers on the topic "Alpha Complex"

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Betan, R. Id, and W. Nazarewicz. "Complex-energy shell model description of alpha decay." In IX LATIN AMERICAN SYMPOSIUM ON NUCLEAR PHYSICS AND APPLICATIONS. AIP, 2012. http://dx.doi.org/10.1063/1.3688779.

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Shinyashiki, Naoki. "Broadband Dielectric Study on Alpha- and Beta-Process for Poly(Ethylene Glycol)-Water Mixtures." In SLOW DYNAMICS IN COMPLEX SYSTEMS: 3rd International Symposium on Slow Dynamics in Complex Systems. AIP, 2004. http://dx.doi.org/10.1063/1.1764248.

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Memon, Hizbullah, Mei Jingbin, and Ghulam Nabi Agha. "Benchmark Drilling Performance in the Undrillable Complex Fold Belt Area." In Gas & Oil Technology Showcase and Conference. SPE, 2023. http://dx.doi.org/10.2118/214335-ms.

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Abstract Alpha Field, a fold belt Oil & Gas field located in the Kirthar mountainous range, has remained as one of most problematic drilling terrains of Pakistan and had been once termed as an undrillable prospect considering the multiple sidetracks at previous wells. Due to formation challenges and multiple sidetracks. One of offset wells had been abandoned because of not reaching to reservoir target and other offset well was eventually reach to well TD after three sidetracks and incurred heavy costs. This paper discusses the case study of phenomenal drilling performance at Well-3 in Alpha field which includes extensive well engineering data reviews to achieve operational excellence. A very detailed offset review has been prepared followed by generation of well plan after careful selection of the drilling plans which included drill bits designs, BHA selections, drilling practices, drilling fluids performance targets, drilling rig performance and extensive level of vigilance to deliver the most complex well with best of the class benchmarking in the field as compared with previous wells and surrounding fields of similar sub-surface challenges. The detailed Well Design and Engineering together with technological selection purely application-based enabling to deliver a successful Alpha field well, both in terms of the lowest cost and in terms of time as the fastest well in Alpha field. The Alpha field due to its complexity, lower success rates and higher costs was one of the most challenging assets in terms of drilling and production delivery. With excellent drilling performance at Well-3 and good production results has opened more opportunities for the Alpha field, and more wells are being planned in the upcoming time.
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Yakovishin, L. A., and E. N. Korzh. "New supramolecular complex of quercetin with triterpene glycoside alpha-hederin." In ACTUAL PROBLEMS OF ORGANIC CHEMISTRY AND BIOTECHNOLOGY (OCBT2020): Proceedings of the International Scientific Conference. AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0068899.

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Ohkura, Michiko, and Wataru Takano. "An Alpha-Wave-Based Motion Control System of a Mechanical Pet for Mental Commitment." In 2007 IEEE/ICME International Conference on Complex Medical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/iccme.2007.4381936.

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Wang, Xiaoping, Huanping Lin, Qiaoxia Wang, Yan Gu, and Yi Xu. "Specific Immunity Elicited by Cross-link Complex Alpha-fetoprotein and Glycoprotein 96." In 2012 International Conference on Biomedical Engineering and Biotechnology (iCBEB). IEEE, 2012. http://dx.doi.org/10.1109/icbeb.2012.361.

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Bakhtiari, Somayeh, Sos Agaian, and Mo Jamshidi. "Complex ensemble empirical mode decomposition and alpha-rooting for image contrast enhancement." In SPIE Defense, Security, and Sensing, edited by Sos S. Agaian, Sabah A. Jassim, and Yingzi Du. SPIE, 2011. http://dx.doi.org/10.1117/12.887424.

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Zhang, Qi, and Tianguang Chu. "$L_{\alpha}$-Regularization-Based Sparse Semi-Supervised Learning for Data with Complex Distributions." In 2019 IEEE 8th Data Driven Control and Learning Systems Conference (DDCLS). IEEE, 2019. http://dx.doi.org/10.1109/ddcls.2019.8908987.

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Wang, Xiaoping, Lijie Zhang, Lijun Sun, Yongxue Zhou, Qiaoxia Wang, Huanping Lin, Xiaoping Ying, and Lansheng Guo. "Antitumor Immunity Induced by Recombinant Vaccine Alpha-Fetoprotein-Heat Shock Protein 70 Complex." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162379.

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Conde-Cespedes, Patricia, Blaise Ngonmang, and Emmanuel Viennet. "Approximation of the Maximal Alpha -- Consensus Local Community Detection Problem in Complex Networks." In 2015 11th International Conference on Signal-Image Technology & Internet-Based Systems (SITIS). IEEE, 2015. http://dx.doi.org/10.1109/sitis.2015.108.

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Reports on the topic "Alpha Complex"

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Rich, Tyvin A., and Sarah Parsons. The Transgenic TGF-Alpha or EGFR1 Overexpression Mouse Model for Symptom Complex Research. Fort Belvoir, VA: Defense Technical Information Center, September 2009. http://dx.doi.org/10.21236/ada533932.

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Lekhanya, Portia Keabetswe, and Kabelo Mokgalaboni. Exploring the effectiveness of vitamin B12 complex and alpha-lipoic acid as a treatment for diabetic neuropathy. Protocol for systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0167.

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Review question / Objective: Does Alpha-Lipoic acid increase the uptake of glucose for better glycaemic control? Does vitamin B12 and Alpha-Lipoic acid improve inflammation? The aim of the study is to explore the effectiveness of Vitamin B12 and Alpha-Lipoic Acid as a possible treatment for diabetic neuropathy with major emphasis on markers of inflammation and glucose metabolism. Condition being studied: Diabetic Neuropathy (DN) is a heterogeneous type of nerve damage associated with diabetes mellitus, the condition most often damages nerves in the legs and feet. It presents both clinically and sub-clinically affecting the peripheral nervous system as a result of an increase in glucose concentration which interferes with nerve signalling. After the discovery of insulin as a treatment for Diabetes Mellitus (DM), the prevalence of DN has since increased significantly due to DM patients having a longer life expectancy. It has been estimated that atleast 50% of DM patients will develop DN in their life, with approximately 20% of these patients experiencing neuropathic pain. Nerves are susceptible to changes in glucose concentrations and insulin makes it impossible for neurons to continue regulating glucose uptake.
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Gunnink, R., W. D. Ruhter, and J. B. Niday. Program listings for GRPANL: A suite of computer programs for analyzing complex Ge and alpha-particle detector spectra: Volume 3. Office of Scientific and Technical Information (OSTI), March 1989. http://dx.doi.org/10.2172/6081998.

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Lande, Lauren, R. J. Newberry, and Evan Twelker. A petrological model for emplacement of the ultramafic Ni-Cu-PGE Alpha complex, Eastern Interior, Alaska (poster): Geological Society of America Cordilleran Section - 111th Annual Meeting, Anchorage, AK, May 11-13, 2015. Alaska Division of Geological & Geophysical Surveys, May 2015. http://dx.doi.org/10.14509/29480.

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Hlushak, Oksana M., Volodymyr V. Proshkin, and Oksana S. Lytvyn. Using the e-learning course “Analytic Geometry” in the process of training students majoring in Computer Science and Information Technology. [б. в.], September 2019. http://dx.doi.org/10.31812/123456789/3268.

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As a result of literature analysis the expediency of free access of bachelors majoring in Computer Sciences and Information Technologies to modern information educational resources, in particular, e-learning courses in the process of studying mathematical disciplines is substantiated. It was established that the e-learning course is a complex of teaching materials and educational services created for the organization of individual and group training using information and communication technologies. Based on the outlined possibilities of applying the e-learning course, as well as its didactic functions, the structure of the certified e-learning course “Analytic Geometry” based on the Moodle platform was developed and described. Features of application of cloud-oriented resources are considered: Desmos, Geogebra, Wolfram|Alpha, Sage in the study of the discipline “Analytic Geometry”. The results of the pedagogical experiment on the basis of Borys Grinchenko Kyiv University and A. S. Makarenko Sumy State Pedagogical University are presented. The experiment was conducted to verify the effectiveness of the implementation of the e-learning course “Analytic Geometry”. Using the Pearson criterion it is proved that there are significant differences in the level of mathematical preparation of experimental and control group of students. The prospect of further scientific research is outlined through the effectiveness of the use of e-learning courses for the improvement of additional professional competences of students majoring in Computer Sciences and Information Technologies (specialization “Programming”, “Internet of Things”).
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Evans, Donald L., Avigdor Eldar, Liliana Jaso-Friedmann, and Herve Bercovier. Streptococcus Iniae Infection in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Towards the Pathogen and Vaccine Formulation. United States Department of Agriculture, February 2005. http://dx.doi.org/10.32747/2005.7586538.bard.

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The objectives of the BARD proposal were to determine the mechanisms of nonspecific cytotoxic cells (NCC) that are necessary to provide heightened innate resistance to infection and to identify the antigenic determinants in Streptococcus iniae that are best suited for vaccine development. Our central hypothesis was that anti-bacterial immunity in trout and tilapia can only be acquired by combining "innate" NCC responses with antibody responses to polysaccharide antigens. These Objectives were accomplished by experiments delineated by the following Specific Aims: Specific aim (SA) #1 (USA) "Clone and Identify the Apoptosis Regulatory Genes in NCC"; Specific aim #2 (USA)"Identify Regulatory Factors that Control NCC Responses to S. iniae"; Specific aim #3 (Israel) "Characterize the Biological Properties of the S. iniae Capsular Polysaccharide"; and Specific aim #4 (Israel) "Development of an Acellular Vaccine". Our model of S. iniae pathogenesis encompassed two approaches, identify apoptosis regulatory genes and proteins in tilapia that affected NCC activities (USA group) and determine the participation of S.iniae capsular polysaccharides as potential immunogens for the development of an acellular vaccine (Israel group). We previously established that it was possible to immunize tilapia and trout against experimental S. difficile/iniaeinfections. However these studies indicated that antibody responses in protected fish were short lived (3-4 months). Thus available vaccines were useful for short-term protection only. To address the issues of regulation of pathogenesis and immunogens of S. iniae, we have emphasized the role of the innate immune response regarding activation of NCC and mechanisms of invasiveness. Considerable progress was made toward accomplishing SA #1. We have cloned the cDNA of the following tilapia genes: cellular apoptosis susceptibility (CAS/AF547173»; tumor necrosis factor alpha (TNF / A Y 428948); and nascent polypeptide-associated complex alpha polypeptide (NACA/ A Y168640). Similar attempts were made to sequence the tilapia FasLgene/cDNA, however these experiments were not successful. Aim #2 was to "Identify Regulatory Factors that Control NCC Responses to S. iniae." To accomplish this, a new membrane receptor has been identified that may control innate responses (including apoptosis) of NCC to S. iniae. The receptor is a membrane protein on teleost NCC. This protein (NCC cationic antimicrobial protein-1/ncamp-1/AAQ99138) has been sequenced and the cDNA cloned (A Y324398). In recombinant form, ncamp-l kills S. iniae in vitro. Specific aim 3 ("Characterize the Biological Properties of the S.iniae Capsular Polysaccharide") utilized an in- vitro model using rainbow trout primary skin epithelial cell mono layers. These experiments demonstrated colonization into epithelial cells followed by a rapid decline of viable intracellular bacteria and translocation out of the cell. This pathogenesis model suggested that the bacterium escapes the endosome and translocates through the rainbow trout skin barrier to further invade and infect the host. Specific aim #4 ("Development of an Acellular Vaccine") was not specifically addressed. These studies demonstrated that several different apoptotic regulatory genes/proteins are expressed by tilapia NCC. These are the first studies demonstrating that such factors exist in tilapia. Because tilapia NCC bind to and are activated by S. iniae bacterial DNA, we predict that the apoptotic regulatory activity of S. iniae previously demonstrated by our group may be associated with innate antibacterial responses in tilapia.
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Citovsky, Vitaly, and Yedidya Gafni. Viral and Host Cell Determinants of Nuclear Import and Export of the Tomato Yellow Leaf Curl Virus in Tomato Plants. United States Department of Agriculture, August 2002. http://dx.doi.org/10.32747/2002.7585200.bard.

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Tomato yellow leaf curl geminivirus (TYLCV) is a major pathogen of cultivated tomato, causing up to 100% crop loss in many parts of the world. In Israel, where TYLCV epidemics have been recorded since the 1960' s, this viral disease is well known and has been of economic significance ever since. In recent years, TYLCV outbreaks also occurred in the "New World" - Cuba, The Dominican Republic, and in the USA, in Florida, Georgia and Louisiana. Thus, TYLCV substantially hinders tomato growth throughout the world. Surprisingly, however, little is known about the molecular mechanisms of TYLCV interaction with the host tomato cells. The present proposal, a continuation of the project supported by BARD from 1994, expanded our understanding of the molecular mechanisms by which TYLCV enters the host cell nucleus for replication and transcription and exits it for the subsequent cell-to-cell spread. Our project sought two objectives: I. To study the roles of the viral capsid protein (CP) and host cell factors in TYLCV nuclear import. II. To study the roles of CP and host cell factors in TYLCV nuclear export. Our research toward these goals have produced the following major achievements: . Developed a one-hybrid assay for protein nuclear export and import (#3 in the List of Publications). . Identified a functional nuclear export signal (NES) in the capsid protein (CP) of TYLCV (#3 in the List of Publications). . Discovered homotypic interactions between intact TYLCV CP molecules and analyzed these interactions using deletion mutagenesis of TYLCV CP (#5 in the List of Publications). . Showed developmental and tissue-specific expression of the host factor required for nuclear import of TYLCV CP, tomato karyopherin alpha 1, in transgenic tomato plants (#14 in the List of Publications). . By analogy to nuclear import of TYLCV ,identified an Arabidopsis VIPI protein that participates in nuclear import of Agrobacterium T -complexes via the karyopherin alpha pathway (#4,6, and 8 in the List of Publications). These research findings provided significant insights into (i) the molecular pathway of TYLCV entry into the host cell nucleus, and (ii) the mechanism by which TYLCV is exported from the nucleus for the cell-to-cell spread of infection. Furthermore, the obtained knowledge will help to develop specific strategies to attenuate TYLCV infection, for example, by blocking viral entry into and/or exit out of the host cell nucleus. Also, as much of our findings is relevant to all geminiviruses, new anti- TYLCV approaches developed based on the results of our research will be useful to combat other members of the Geminivirus family. Finally, in addition to the study of TYLCV nuclear import and export, our research contributed to our understanding of general mechanisms for nucleocytoplasmic shuttling of proteins and nucleic acids in plant cells.
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