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Relevant bibliographies by topics / Alobar holoprosencephaly

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Academic literature on the topic 'Alobar holoprosencephaly'

Author: Grafiati

Published: 30 June 2021

Last updated: 4 February 2022

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Journal articles on the topic "Alobar holoprosencephaly"

1

Selden, Nathan. "Alobar Holoprosencephaly." Pediatric Neurosurgery 33, no. 2 (2000): 112. http://dx.doi.org/10.1159/000028986.

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2

Callahan, Jodi, Casey Harmon, John Aleshire, Bill Hickey, and Brandy Jones. "Alobar Holoprosencephaly With Cebocephaly." Journal of Diagnostic Medical Sonography 33, no. 1 (September 26, 2016): 39–42. http://dx.doi.org/10.1177/8756479316664477.

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Abstract:

Holoprosencephaly (HPE) is a complex brain malformation caused by incomplete fusion of cleavage of the cerebral hemispheres and deep brain structures affecting 6 to 12:10,000 live-born infants. There are three categories of HPE ranging in severity, with alobar holoprosencephaly being the most severe, followed by semilobar holoprosencephaly, and lobar holoprosencephaly being the mildest form. Facial anomalies as well as chromosome anomalies are often associated with HPE. This case study describes a transabdominal sonographic diagnosis of alobar HPE with cebocephaly originally found at 27 weeks 3 days on a patient with no prenatal care.

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3

Bullen, PJ, and SC Robson. "Holoprosencephaly." Fetal and Maternal Medicine Review 12, no. 1 (January 17, 2001): 1–21. http://dx.doi.org/10.1017/s0965539501000110.

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Holoprosencephaly (HPE) is an uncommon and clinically severe abnormality of forebrain development, resulting from failure of septation, or cleavage, of the midline forebrain structures. It encompasses a range of brain pathology classified as lobar, semilobar or alobar HPE reflecting increasing degrees of failed septation. The closely related development of the mid-face area is frequently disturbed, with coexisting facial phenotypes ranging from cyclopia to normal.

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4

Raman, Rajesh, and Geetha Mukunda Jagadesh. "Antenatal Diagnosis of Alobar Holoprosencephaly." Case Reports in Radiology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/724671.

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A twenty-year-old second gravida presented to the department of radiodiagnosis for routine obstetric ultrasound examination. Ultrasonography revealed a live fetus of 17 weeks with absent falx, fused thalami, monoventricle, proboscis, and cyclopia. Fetal MRI was performed and the findings were confirmed. Even though ultrasonography is diagnostic in the detection of fetal anomalies, MRI plays a vital role due to its multiplanar capability and excellent soft tissue resolution. The importance of presenting this classical case of alobar holoprosencephaly is to sensitize the clinicians and radiologists to the imaging manifestations of holoprosencephaly and to stress the importance of early diagnosis. If diagnosed in utero at an early stage of pregnancy, termination can be performed and maternal psychological trauma of bearing a deformed fetus can be avoided.

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5

Khanna, Dolly, and Karandeep S. Bhatti. "Antenatal diagnosis of alobar holoprosencephaly." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 9, no. 5 (April 28, 2020): 2184. http://dx.doi.org/10.18203/2320-1770.ijrcog20201832.

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Holoprosencephaly (HPE), a congenital induction disorder, occurs due to failed segmentation of neural tube and subsequent incomplete separation of the prosencephalon. Because of the defect in the ventral induction, HPE is also associated with multiple facial abnormalities. Mortality correlates with the severity of brain malformation and facial phenotype. Frequent causes of death include respiratory infections, dehydration due to uncontrolled diabetes insipidus, intractable seizures, and brainstem malfunction. This is a case of Alobar holoprosencephaly in the fetus of a 31-year-old G2P1 female, detected on level II obstetric ultrasound at 17 weeks of gestation. The objective of this case report is to discuss the etiology, pathogenesis, prenatal sonographic findings, management and prognosis of HPE which can aid in its early antenatal diagnosis and can help formulate an adequate plan for the parents and fetus.

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6

Mirshekari, Leila, Mehrbanu Amirshahi, Akram Sanagoo, Ashraf Salehi, Azam Kerami, Abdolghani Abdollahimohammad, Fatemeh Mirshekari, Fereshteh Naroei, Leila Mansoorifar, and Marzeeh Mirshekari. "Alobar holoprosencephaly: A case report." Journal of Nursing and Midwifery Sciences 2, no. 4 (2015): 70. http://dx.doi.org/10.18869/acadpub.jnms.2.4.70.

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7

Akpinar, Elif, Mehmet Sabri Gürbüz, Mehmet Özerk Okutan, and Ethem Beşkonakli. "Ventriculoperitoneal Shunting in Alobar Holoprosencephaly." Journal of Craniofacial Surgery 30, no. 6 (September 2019): 1780–81. http://dx.doi.org/10.1097/scs.0000000000005432.

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8

Mizuguchi, M., and Y. Morimatsu. "Histopathological study of alobar holoprosencephaly." Acta Neuropathologica 78, no. 2 (1989): 176–82. http://dx.doi.org/10.1007/bf00688206.

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9

Mizuguchi, M., and Y. Morimatsu. "Histopathological study of alobar holoprosencephaly." Acta Neuropathologica 78, no. 2 (1989): 183–88. http://dx.doi.org/10.1007/bf00688207.

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10

Babaji, Prashant, Shamsher Singh, Seshadri Sekar, Anjani Kumar, Vidit Gupta, and Anurag Singh. "Alobar holoprosencephaly with synopthalmia and proboscis." Journal of Clinical Neonatology 3, no. 3 (2014): 176. http://dx.doi.org/10.4103/2249-4847.140415.

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Dissertations / Theses on the topic "Alobar holoprosencephaly"

1

Elfarawi, Hunaydah. "Alobar Holoprosencephaly: Parental Perspectives on Prenatal Decision-making, Prenatal Provider Prognostication, and Quality of Life." University of Cincinnati / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1617108856885634.

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2

JAMOT, FOURNERON CHRISTINE. "L'holoprosencephalite alobaire et son diagnostic echographique : a propos d'un cas." Saint-Etienne, 1992. http://www.theses.fr/1992STET6221.

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Book chapters on the topic "Alobar holoprosencephaly"

1

"Alobar Holoprosencephaly." In Diagnostic Imaging: Obstetrics, 118–21. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-39256-3.50033-9.

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2

Domm, Christian, Paul-Martin Holterhus, and Heiner Moenig. "19-Year-Old Female with Hypodipsia-Hypernatremia-Syndrome Due to Alobar Holoprosencephaly." In The Endocrine Society's 93rd Annual Meeting & Expo, June 4–7, 2011 - Boston, P3–754—P3–754. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part4.p14.p3-754.

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