Relevant bibliographies by topics / Allergy Immunological aspects

Academic literature on the topic 'Allergy Immunological aspects'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Allergy Immunological aspects"

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Littlewood, J. M. "Allergy. Immunological and Clinical Aspects." Archives of Disease in Childhood 60, no. 3 (March 1, 1985): 292. http://dx.doi.org/10.1136/adc.60.3.292.

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Godfrey, R. C. "Allergy: immunological and clinical aspects." British Journal of Diseases of the Chest 79 (January 1985): 409. http://dx.doi.org/10.1016/0007-0971(85)90081-6.

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David, T. J. "Book Review: Allergy: Immunological and Clinical Aspects." Journal of the Royal Society of Medicine 78, no. 5 (May 1985): 425. http://dx.doi.org/10.1177/014107688507800535.

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Bochner, B. S., B. J. Undem, and L. M. Lichtenstein. "Immunological Aspects of Allergic Asthma." Annual Review of Immunology 12, no. 1 (April 1994): 295–335. http://dx.doi.org/10.1146/annurev.iy.12.040194.001455.

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Ahmed, Syed M., Tar-Ching Aw, and Anil Adisesh. "Toxicological and Immunological Aspects of Occupational Latex Allergy." Toxicological Reviews 23, no. 2 (2004): 123–34. http://dx.doi.org/10.2165/00139709-200423020-00005.

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Roubey, Robert A. S. "Antiphospholipid antibodies: immunological aspects." Clinical Immunology 112, no. 2 (August 2004): 127–28. http://dx.doi.org/10.1016/j.clim.2004.02.010.

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Čolić, Miodrag, Sergej Tomić, and Marina Bekić. "Immunological aspects of nanocellulose." Immunology Letters 222 (June 2020): 80–89. http://dx.doi.org/10.1016/j.imlet.2020.04.004.

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Garrido-Urbani, S., M. Meguenani, F. Montecucco, and B. A. Imhof. "Immunological aspects of atherosclerosis." Seminars in Immunopathology 36, no. 1 (November 9, 2013): 73–91. http://dx.doi.org/10.1007/s00281-013-0402-8.

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Corrigan, Chris J. "Immunological Aspects of Asthma." Clinical Immunotherapeutics 1, no. 1 (January 1994): 31–42. http://dx.doi.org/10.1007/bf03258489.

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Bárány, Peter, and Ingela Fehrman-Ekholm. "Immunological Aspects of Haemodialysis." Clinical Immunotherapeutics 1, no. 6 (June 1994): 469–80. http://dx.doi.org/10.1007/bf03259039.

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Dissertations / Theses on the topic "Allergy Immunological aspects"

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See, Sarah Bihui. "Outer membrane protein immunity to Pasteurella pneumotropica and the interaction of allergy." University of Western Australia. School of Paediatrics and Child Health, 2010. http://theses.library.uwa.edu.au/adt-WU2010.0103.

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[Truncated abstract] Infectious and allergic diseases of the respiratory tract are major contributors to global mortality, morbidity and economic burden. Bacterial infections such as pneumonia and otitis media are important diseases, especially in children, while allergic diseases such as asthma and allergic rhinitis afflict up to 30% of the world's population. A confounding aspect of respiratory disease is the evidence of a complex relationship between respiratory allergy and respiratory infection, with infection suggested to both promote and prevent the pathogenesis of allergic disease. Additionally, allergy is a risk factor for bacterial infection such as otitis media, pneumonia and sinusitis, while respiratory infection can exacerbate allergic symptoms. Given the burden of bacterial respiratory disease and respiratory allergy, the development of preventative treatments for these diseases is needed and will benefit from clearer knowledge of the underlying immune mechanisms. This thesis aimed to to extend current knowledge by using Pasteurella pneumotropica, a similar bacteria to the human pathogen nontypeable Haemophilus influenzae (NTHi), to study respiratory infection and protective anti-outer membrane protein (OMP) immunity as well as the interaction of respiratory infection and allergic inflammation. Homologues of the important NTHi vaccine candidates P4, P6, P26 and D15 were found to be encoded by P. pneumotropica and a high level of amino acid sequence identity was noted between the different P. pneumotropica strains, as well as between other Pasteurellaceae members. ... In contrast, anti-P6his serum antibodies transferred to naïve mice did not confer protection. These results suggested that T-cell–mediated mechanisms were involved in P6his-mediated protection, and showed that the P. pneumotropcia model was useful for elucidating protective mechansims. The interaction of P. pneumotropica infection and papain-induced allergy was studied to investigate immune mechanisms underlying respiratory infection and allergy. Mice with ongoing allergic inflammation were intranasally challenged with bacteria and exhibited reduced pulmonary bacterial numbers, prolonged eosinophilia in the lungs and the induction of Th2 cytokines in the BALF, compared to nonallergic, infected mice. This suggested a protective role for allergic inflammation in this model. The effect of papaininduced inflammation on mice colonised by P. pneumotropica was also examined and allergic inflammation appeared to worsen infection in colonised mice. This suggested that allergic inflammation may also have a role in promoting infection in this model. In conclusion, this thesis explored mechanisms involved in vaccine-mediated immunity and the interaction of respiratory infection and allergy using a P. pneumotropica infection in its natural host. It was shown that intranasally administered recombinant P6 and P4 protected mice from lung infection, which justifies the inclusion of these OMPs as NTHi vaccine candidates. Additionally, it was demonstrated that the interaction of allergy and respiratory infection modulated immune responses. Overall, these results emphasize that a clearer understanding of the complex mechanisms underlying these interactions is required, and may be aided by the development of suitable animal models.
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Noakes, Paul Stanton. "The effects of maternal smoking on infant immune development." University of Western Australia. School of Paediatrics and Child Health, 2006. http://theses.library.uwa.edu.au/adt-WU2006.0080.

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[Truncated abstract] With the dramatic rise in asthma and allergic disease there is an urgent need to define the early life exposures which influence developing immune responses to increase the predisposition to allergic disease. While this is clearly multifactorial, this thesis addresses the effects of maternal smoking as a major adverse, yet avoidable exposure in early life. I hypothesised that the well-documented increased susceptibility to infection in infants of smokers could indicate underlying effects on innate Toll-like receptor (TLR) mediated microbial responses which could in turn contribute to early immune dysregulation and increased risk of allergic disease. In addition to providing the first defence against microbes, TLR-mediated pathways modulate subsequent specific immune response and are of growing interest in the potential inhibition of inappropriate allergic responses. My initial interest in the potential immune effects of smoking in pregnancy was based on preliminary retrospective analyses of a previous cohort (presented in Chapter 3) which suggested possible effects on T cell cytokine responses to mitogens and allergens. Based on this, I recruited a new prospective pregnancy cohort (n=122) of smokers (n=60) and non-smokers (n=62) (as outlined in Chapter 4) to confirm this and test my novel hypothesis that maternal smoking may be affecting important innate (TLR-mediated) immune pathways. … Thus, these findings could indicate that smoking increases the early susceptibility to infection thereby increasing subsequent IgA responses. This is supported by observations that key neonatal TLR responses are attenuated in children who go onto develop wheezy illnesses and lower respiratory tract infections. Together, the study findings suggest that in addition to effects on lung growth, maternal smoking may also influence aspects of neonatal innate immune function that are now believed to play a critical role in microbial-driven postnatal immune development, highlighting that other environmental interactions are also highly relevant to the v
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Xiong, Yelin. "Immune modulation of allergic airways disease." Phd thesis, 1998. http://hdl.handle.net/1885/144680.

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Sawant, Deepali Vijay. "Control of inflammation, helper T cell responses and regulatory T cell function by Bcl6." Thesis, 2014. http://hdl.handle.net/1805/3829.

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Indiana University-Purdue University Indianapolis (IUPUI)
Regulatory T (Treg) cells represent an important layer of immune-regulation indispensible for curtailing exuberant inflammatory responses and maintaining self-tolerance. Treg cells have translational potential for autoimmunity, inflammation, transplantation and cancer. Therefore, delineating the molecular underpinnings underlying the development, suppressor function and stability of Tregs is particularly warranted. The transcriptional repressor Bcl6 is a critical arbiter of helper T cell fate, promoting the follicular helper (Tfh) lineage while repressing Th1, Th2 and Th17 differentiation. Bcl6-deficient mice develop a spontaneous and severe Th2-type inflammatory disease including myocarditis and pulmonary vasculitis, suggesting a potential role for Bcl6 in Treg cell function. Bcl6-deficient Treg cells are competent in controlling Th1 responses, but fail to control Th2 inflammation in an airway allergen model. Importantly, mice with Bcl6 deleted specifically in the Treg lineage develop severe myocarditis, thus highlighting a critical role for Bcl6 in Treg-mediated control of Th2 inflammation. Bcl6-deficient Tregs display an intrinsic increase in Th2 genes and microRNA-21 (miR-21) expression. MiR-21 is a novel Bcl6 gene target in T cells and ectopic expression of miR-21 directs Th2 differentiation in non-polarized T cells. MiR-21 is up-regulated in mouse models of airway inflammation and also in human patients with eosinophilic esophagitis and asthma. Thus, miR-21 is a clinically relevant biomarker for Th2-type pathologies. Our results define a key function for Bcl6 in repressing Gata3 function and miR-21 expression in Tregs, and provide greater understanding of the control of Th2 inflammatory responses by Treg cells.
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"Intracellular signaling mechanisms regulating the mast cell-mediated allergic inflammation." 2007. http://library.cuhk.edu.hk/record=b5893246.

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Ng Sin Man.
Thesis (M.Phil.)--Chinese University of Hong Kong, 2007.
Includes bibliographical references (leaves 120-135).
Abstracts in English and Chinese.
Acknowledgements --- p.i
Abbreviations --- p.iii
Abstract --- p.vi
撮要 --- p.ix
Publications --- p.xi
Table of contents --- p.xiii
Chapter Chapter 1 --- General Introduction
Chapter 1.1 --- Allergic Diseases and Allergic Inflammation --- p.1
Chapter 1.1.1 --- Prevalence of Allergic Diseases --- p.1
Chapter 1.1.2 --- Common Allergic Diseases: Allergic Asthma --- p.1
Chapter 1.1.3 --- Common Allergic Diseases: Atopic Dermatitis --- p.2
Chapter 1.1.4 --- Allergic Inflammation --- p.3
Chapter 1.2 --- The Inflammatory Leukocytes: Mast Cells and Eosinophils --- p.6
Chapter 1.2.1 --- Characteristics of Mast Cells --- p.6
Chapter 1.2.2 --- Mast Cells Distribution --- p.8
Chapter 1.2.3 --- Mast Cells Subtypes --- p.8
Chapter 1.2.4 --- HMC-1 Cells --- p.9
Chapter 1.2.5 --- Characteristics of Eosinophils --- p.12
Chapter 1.3 --- Adhesion Molecules in Allergic Diseases --- p.15
Chapter 1.3.1 --- Adhesion Molecules and Leukocyte Migration --- p.15
Chapter 1.3.2 --- Selectin --- p.17
Chapter 1.3.3 --- Intermolecular Adhesion Molecules --- p.17
Chapter 1.3.4 --- Integrin --- p.18
Chapter 1.4 --- Cytokines and Chemokines in Allergic Diseases --- p.18
Chapter 1.4.1 --- IL-6 --- p.20
Chapter 1.4.2 --- CXCL1 --- p.21
Chapter 1.4.3 --- CXCL8 --- p.21
Chapter 1.4.3 --- CCL2 --- p.22
Chapter 1.5 --- Intercellular Signal Transduction Pathways in Inflammation --- p.24
Chapter 1.5.1 --- RAS-RAF-mitogen-activated Protein Kinases --- p.24
Chapter 1.5.2 --- Janus Kinase/ Signal Transducers and Activators of Transcriptions Pathway --- p.27
Chapter 1.5.3 --- Nuclear Factor-KB Pathway --- p.29
Chapter 1.5.4 --- Phosphoinositide 3-Kinase Pathway --- p.31
Chapter 1.6 --- Aims and Scope of the Study --- p.33
Chapter Chapter 2 --- Materials and Methods
Chapter 2.1 --- Materials --- p.35
Chapter 2.1.1 --- HMC-1 Cell Line --- p.35
Chapter 2.1.2 --- Human Buffer Coat --- p.35
Chapter 2.1.3 --- Human Mast Cell Chymase and TLR ligands --- p.35
Chapter 2.1.4 --- Media and Reagents for Cell Culture --- p.36
Chapter 2.1.5 --- Reagents and Buffers for Purification of Human Eosinophils --- p.37
Chapter 2.1.6 --- Reagents and Buffers for Flow Cytmetry --- p.38
Chapter 2.1.7 --- Reagents and Buffers for Total RNA Extraction --- p.41
Chapter 2.1.8 --- Reagents and Buffers for Reverse Transcription-Polymerase Chain Reaction (RT-PCR) --- p.42
Chapter 2.1.9 --- Reagents and Buffers for Agarose Gel Electrophoresis --- p.45
Chapter 2.1.10 --- Reagents and Buffers for Sodium Dodecyl Sulfate -polyacrylamide Gel Electrophoresis (SDS-PAGE) --- p.46
Chapter 2.1.11 --- Reagents and Buffers for Western Blot Analysis --- p.48
Chapter 2.1.12 --- Chemotactic Migration --- p.51
Chapter 2.1.13 --- Signaling Transduction Inhibitors and Protein Synthesis Inhibitors --- p.51
Chapter 2.2 --- Methods --- p.52
Chapter 2.2.1 --- HMC-1 Cell Cultures --- p.52
Chapter 2.2.2 --- Purification of Buffy Coat Eosinophils by MACS and Eosinophil Culture --- p.52
Chapter 2.2.3 --- Total Cellular RNA Extraction --- p.53
Chapter 2.2.4 --- RT-PCR --- p.54
Chapter 2.2.5 --- Agarose Gel Electrophoresis --- p.55
Chapter 2.2.6 --- Flow Cytometry Analysis --- p.55
Chapter 2.2.7 --- Protein Array Analysis of Cytokine Release --- p.57
Chapter 2.2.8 --- Quantitative Analysis ofCXCLl --- p.58
Chapter 2.2.9 --- Total Protein Extraction --- p.58
Chapter 2.2.10 --- SDS-PAGE --- p.58
Chapter 2.2.11 --- Western Blot Analysis --- p.59
Chapter 2.2.12 --- Chemotactic Migration Analysis --- p.60
Chapter 2.2.13 --- Statistical Analysis --- p.60
Chapter Chapter 3 --- Effects of Mast Cell Derived Chymase on Human Eosinophils and the Signaling Mechanisms: Implication in Allergic Inflammation
Chapter 3.1 --- Introduction --- p.61
Chapter 3.2 --- Results --- p.65
Chapter 3.2.1 --- Effects of Chymase on Eosinophil Survival --- p.65
Chapter 3.2.2 --- Effects of Chymase on the Adhesion Molecule Expression of Eosinophils --- p.68
Chapter 3.2.3 --- Effects of Chymase on the Chemokinetic Properties on Eosinophils --- p.71
Chapter 3.2.4 --- Effects of Chymase on the Release of Chemokines and IL-6 from Eosinophils --- p.73
Chapter 3.2.5 --- Signal Transduction Mechanism Involved in Regulating Chymase-induced Effects on Eosinophils --- p.78
Chapter 3.3 --- Discussion --- p.71
Chapter Chapter 4 --- TLR-mediated Effects and Signal Transduction Mechanism of HMC-1 Cells
Chapter 4.1 --- Introduction --- p.92
Chapter 4.2 --- Results --- p.97
Chapter 4.2.1 --- Expression of Adhesion Molecules on HMC-1 Cells --- p.95
Chapter 4.2.2 --- TLR Expression Profile on HMC-1 Cells --- p.97
Chapter 4.2.3 --- Effects of TLR ligands on HMC-1 Cell Adhesion Molecule Expressions --- p.99
Chapter 4.2.4 --- TLR7-induced Phosphorylation of ERK and Effects of PD98059 on TLR7-induced ERK Phosphorylation --- p.104
Chapter 4.2.5 --- Effect of TLR7 Ligand on HMC-1 Cells Cytokine Release --- p.108
Chapter 4.3 --- Discussion --- p.110
Chapter Chapter 5 --- Conclusions and Future Perspectives
Chapter 5.1 --- Conclusions --- p.115
Chapter 5.2 --- Future Perspectives --- p.117
References --- p.120
Appendix --- p.136
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Glosson, Nicole L. "Development and stability of IL-17-secreting T cells." Thesis, 2014. http://hdl.handle.net/1805/5902.

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Indiana University-Purdue University Indianapolis (IUPUI)
IL-17-producing T cells are critical to the development of pathogen and tumor immunity, but also contribute to the pathology of autoimmune diseases and allergic inflammation. CD8+ (Tc17) and CD4+ (Th17) IL-17-secreting T cells develop in response to a cytokine environment that activates Signal Transducer and Activator of Transcription (STAT) proteins, though the mechanisms underlying Tc17/Th17 development and stability are still unclear. In vivo, Tc17 cells clear vaccinia virus infection and acquire cytotoxic potential, that is independent of IL-17 production and the acquisition of IFN-γ-secreting potential, but partially dependent on Fas ligand, suggesting that Tc17-mediated vaccinia virus clearance is through cell killing independent of an acquired Tc1 phenotype. In contrast, memory Th cells and NKT cells display STAT4-dependent IL-23-induced IL-17 production that correlates with Il23r expression. IL-23 does not activate STAT4 nor do other STAT4-activating cytokines induce Il23r expression in these populations, suggesting a T cell-extrinsic role for STAT4 in mediating IL-23 responsiveness. Although IL-23 is important for the maintenance of IL-17-secreting T cells, it also promotes their instability, often resulting in a pathogenic Th1-like phenotype in vitro and in vivo. In vitro-derived Th17 cells are also flexible when cultured under polarizing conditions that promote Th2 or Th9 differentiation, adopting the respective effector programs, and decreasing IL-17 production. However, in models of allergic airway disease, Th17 cells do not secrete alternative cytokines nor adopt other effector programs, and remain stable IL-17-secretors. In contrast to Th1-biased pro-inflammatory environments that induce Th17 instability in vivo, during allergic inflammatory disease, Th17 cells are comparatively stable, and retain the potential to produce IL-17. Together these data document that the inflammatory environment has distinct effects on the stability of IL-17-secreting T cells in vivo.
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Clark, Kristopher. "Eosinophil activation in a mouse model of allergic airways disease." Thesis, 2003. http://hdl.handle.net/1885/148528.

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Books on the topic "Allergy Immunological aspects"

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Pawankar, Ruby. Allergy frontiers. New York: Springer, 2009.

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International Conference on Immunopharmacology (4th 1988 Osaka, Japan). Abstracts of workshop presentations. Oxford: Pergamon Press, 1988.

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Pawankar, Ruby. Allergy Frontiers: Diagnosis and Health Economics. Tokyo: Springer Japan, 2009.

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1947-, Moqbel Redwan, ed. Allergy and immunity to helminths: Common mechanisms or divergent pathways? London: Taylor & Francis, 1992.

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Allergy and immunology of the eye. 2nd ed. New York: Raven Press, 1993.

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Ch'oe, Min-hŭi. Gutpai at'op'i. Sŏul-si: 21-segi Buksŭ, 2009.

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H, Dunn Denise, ed. Allergies and autism. New York: Nova Science, 2010.

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Dochniak, Michael J. Allergies and autism. Hauppauge, N.Y: Nova Science, 2009.

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Dochniak, Michael J. Allergies and autism. Hauppauge, N.Y: Nova Science, 2009.

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A, Clark D., and Croy B. A, eds. Reproductive immunology 1986: Proceedings of the 3rd International Congress of Reproductive Immunology held in Toronto, Canada, 1-5 July 1986. Amsterdam: Elsevier Science Publishers, 1986.

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Book chapters on the topic "Allergy Immunological aspects"

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Scheynius, Annika. "Immunological Aspects." In Allergic Contact Dermatitis, 4–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80331-4_2.

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Nally, F. F., and J. J. H. Gilkes. "Mucocutaneous Allergic Reactions." In Immunological Aspects of Oral Diseases, 257–74. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4167-0_12.

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Elsner, Jörn, and Alexander Kapp. "Immunological aspects of allergic inflammation: eosinophils." In Immunology and Drug Therapy of Allergic Skin Diseases, 49–71. Basel: Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8464-8_4.

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Dubois, Gerald R., Paul J. Baselmans, and Geert C. Mudde. "Immunological aspects of allergic inflammation: IgE regulation." In Immunology and Drug Therapy of Allergic Skin Diseases, 15–28. Basel: Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8464-8_2.

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Brauer, David Lawrence, and Katharine Margaret Woessner. "Non-pharmacologic Aspects of Management: “Asthma and Allergic and Immunologic Diseases During Pregnancy – A Guide to Management”." In Asthma, Allergic and Immunologic Diseases During Pregnancy, 1–14. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-03395-8_1.

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Martin, R. "Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis and their application for new therapeutic strategies." In Advances in Research on Neurodegeneration, 53–67. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6844-8_6.

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Aalberse, Rob C., and Paolo M. Matricardi. "Immunological Aspects of the Atopic March." In Allergy, Immunity and Tolerance in Early Childhood, 19–31. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-420226-9.00002-4.

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"Food Allergy: Immunological Aspects and Approaches to Safety Assessment." In Immunotoxicology and Immunopharmacology, 631–46. CRC Press, 2006. http://dx.doi.org/10.1201/9781420005448-50.

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Lorenz, Anne-Regine, Stephan Scheurer, and Stefan Vieths. "Food Allergens: Molecular and Immunological Aspects, Allergen Databases and Cross-Reactivity." In Food Allergy: Molecular Basis and Clinical Practice, 18–29. S. Karger AG, 2015. http://dx.doi.org/10.1159/000371647.

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Ma, Songhui. "Immunologic Aspects of Rhinosinusitis." In Sataloff’s Comprehensive Textbook of Otolaryngology: Head and Neck Surgery (Rhinology/Allergy and Immunology) -Volume 2, 391. Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12714_27.

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