Relevant bibliographies by topics / Allergic response

Academic literature on the topic 'Allergic response'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Allergic response"

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Martin, Lynn B., and Courtney A. C. Coon. "Photoperiod-driven variation in an allergic response is independent of allergen exposure." Canadian Journal of Zoology 90, no. 9 (September 2012): 1086–93. http://dx.doi.org/10.1139/z2012-075.

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Allergy prevalence and severity varies seasonally in humans, presumably due to intra-annual changes in allergen exposure. However, it is possible that seasonality of allergic responses is also influenced by seasonal changes in the immune system. Here, we asked whether extended exposure to different day lengths would alter allergic responses to pentadecylcatechol (PDC), an allergenic component of poison ivy ( Toxicodendron radicans (L.) Kuntze), in Siberian hamsters ( Phodopus sungorus (Pallas, 1773)), a species exhibiting extensive seasonal variation in immune functions. We found that contact dermatitis responses were larger in short day-length (SD) housed animals than in long day-length (LD) housed animals even though sensitization and challenge dosages of allergen were identical. Furthermore, SD animals were smaller and had regressed reproductive tissues compared with LD animals, results typically observed in this species in response to photoperiod. These data suggest that endogenous changes in immune functions, perhaps via melatonin, may underlie some seasonal variation in allergic responses.
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Roger, Albert, Maria Basagana, Aina Teniente-Serra, Nathalie Depreux, Yanina Jurgens, Clara Padro, Sira Miquel, Carolina Elduque, and Eva M. Martinez-Caceres. "Immunotheraphy in Allergic Diseases." Current Pharmaceutical Design 24, no. 11 (June 27, 2018): 1174–94. http://dx.doi.org/10.2174/1381612824666180116094048.

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The prevalence of allergic diseases is increasing worldwide. It is estimated that more than 30% of the world population is now affected by one or more allergic conditions and a high proportion of this increase is in young people. The diagnosis of allergy is dependent on a history of symptoms on exposure to an allergen together with the detection of allergen-specific IgE. Accurate diagnosis of allergies opens up therapeutic options. Allergen specific immunotherapy is the only successful disease-modifying therapy for IgE-mediated allergic diseases. New therapeutic strategies have been developed or are currently under clinical trials. Besides new routes of administration, new types of allergens are being developed. The use of adjuvants may amplify the immune response towards tolerance to the antigens. In this review, we analyze different antigen-specific immunotherapies according to administration route, type of antigens and adjuvants, and we address the special case of food allergy.
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Besedovsky, Luciana, Mona Benischke, Jörg Fischer, Amir S. Yazdi, and Jan Born. "Human sleep consolidates allergic responses conditioned to the environmental context of an allergen exposure." Proceedings of the National Academy of Sciences 117, no. 20 (May 4, 2020): 10983–88. http://dx.doi.org/10.1073/pnas.1920564117.

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Allergies are highly prevalent, and allergic responses can be triggered even in the absence of allergens due to Pavlovian conditioning to a specific cue. Here we show in humans suffering from allergic rhinitis that merely reencountering the environmental context in which an allergen was administered a week earlier is sufficient to trigger an allergic response—but only if participants had slept after allergen exposure. This context-conditioning effect was entirely absent when participants stayed awake the night after allergen exposure or were tested in a different context. Unlike in context conditioning, cue conditioning (to an odor stimulus) occurred independently of sleep, a differential pattern that is likewise observed for conditioning in the behavioral domain. Our findings provide evidence that allergic responses can be conditioned to contextual information alone, even after only a single-trial conditioning procedure, and that sleep is necessary to consolidate this rapidly acquired maladaptive response. The results unravel a mechanism that could explain part of the strong psychological impact on allergic responses.
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Jacquet, Alain. "Innate Immune Responses in House Dust Mite Allergy." ISRN Allergy 2013 (February 28, 2013): 1–18. http://dx.doi.org/10.1155/2013/735031.

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Sensitizations to house dust mites (HDM) trigger strong exacerbated allergen-induced inflammation of the skin and airways mucosa from atopic subjects resulting in atopic dermatitis as well as allergic rhinitis and asthma. Initially, the Th2-biased HDM allergic response was considered to be mediated only by allergen B- and T-cell epitopes to promote allergen-specific IgE production as well as IL-4, IL-5, and IL-13 to recruit inflammatory cells. But this general molecular model of HDM allergenicity must be revisited as a growing literature suggests that stimulations of innate immune activation pathways by HDM allergens offer new answers to the following question: what makes an HDM allergen an allergen? Indeed, HDM is a carrier not only for allergenic proteins but also microbial adjuvant compounds, both of which are able to stimulate innate signaling pathways leading to allergy. This paper will describe the multiple ways used by HDM allergens together with microbial compounds to control the initiation of the allergic response through engagement of innate immunity.
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Dr. Mayank Surana, Dr. Vineeta Pande, Dr. Sharad Agarkhedkar, and Dr. Ajit Teegala. "Correlation between Total Serum Immunoglobulin E (IgE) and Absolute Eosinophil Count (AEC) in Allergic Diseases In Children." VIMS Health Science Journal 7, no. 1 (March 6, 2020): 5–9. http://dx.doi.org/10.46858/vimshsj.7102.

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Allergy, is a clinical expression of soluble factors like IgE, histamine or eosinophils found in serum or plasma of such patients. The products that are responsible for allergy are called as Allergens. Allergens normally induce IgE production which leads to type 1 hypersensitivity response on subsequent exposure to the same allergen. The target organs are mostly nose, lung, skin and gastrointestinal tract. Atopy is also considered as a triad of Atopic dermatitis, allergic rhinitis and bronchial asthma. Raised serum IgE and AEC are proven indicators of allergic phenomenon. Various studies show relationship between serum Immunoglobulin E level and total eosinophil count in population suffering from allergic diseases. Serum total Immunoglobulin E, total eosinophil count and specific IgE are all helpful for the diagnosis and treatment of allergic diseases. Objectives: 1.To Evaluate Serum Total IgE level in Children with allergic diseases.2. To Evaluate Absolute Eosinophil Count (AEC) in children with allergic diseases.3. To Correlate Serum Total Immunoglobulin E Level and Absolute Eosinophil Count (AEC) with allergic diseases. Methodology: Cross sectional study with 100 children in the age group 2-12 years with nasopharyngeal allergies (like bronchial asthma and atopic rhinitis) and skin allergies (like atopic dermatitis, urticaria) ,eye allergies were enrolled and serum IgE levels and AEC levels was done. Results: In present study Absolute eosinophil count was raised in 58% of cases Serum IgE was raised in 54% of cases. In present study, of 58% cases with raised Absolute eosinophil count 81% (47 cases) showed raised serum IgE levels. Conclusion: Absolute eosinophil count and serum Total IgE has been considered as a significant marker of allergic state and can be used as a marker of allergic response in atopic individuals. Raised serum IgE and AEC are more in nasobronchial allergy as compare to other systemic allergies. The elevated level of serum Total IgE and Absolute Eosinophil Count both shows Significant Correlation thus can be considered as a dependable laboratory investigation in diagnosing and categorizing allergic diseases.
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Deak, Peter E., Baksun Kim, Amina Abdul Qayum, Jaeho Shin, Girish Vitalpur, Kirsten M. Kloepfer, Matthew J. Turner, et al. "Designer covalent heterobivalent inhibitors prevent IgE-dependent responses to peanut allergen." Proceedings of the National Academy of Sciences 116, no. 18 (April 8, 2019): 8966–74. http://dx.doi.org/10.1073/pnas.1820417116.

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Allergies are a result of allergen proteins cross-linking allergen-specific IgE (sIgE) on the surface of mast cells and basophils. The diversity and complexity of allergen epitopes, and high-affinity of the sIgE–allergen interaction have impaired the development of allergen-specific inhibitors of allergic responses. This study presents a design of food allergen-specific sIgE inhibitors named covalent heterobivalent inhibitors (cHBIs) that selectively form covalent bonds to only sIgEs, thereby permanently inhibiting them. Using screening reagents termed nanoallergens, we identified two immunodominant epitopes in peanuts that were common in a population of 16 allergic patients. Two cHBIs designed to inhibit only these two epitopes completely abrogated the allergic response in 14 of the 16 patients in an in vitro assay and inhibited basophil activation in an allergic patient ex vivo analysis. The efficacy of the cHBI design has valuable clinical implications for many allergen-specific responses and more broadly for any antibody-based disease.
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Simbirtsev, A. S. "Cytokines and their role in immune pathogenesis of allergy." Russian Medical Inquiry 5, no. 1 (2021): 32–37. http://dx.doi.org/10.32364/2587-6821-2021-5-1-32-37.

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Allergy is characterized by inadequate hyperimmune response to one or several antigens with the properties of allergens. A specific pattern of abnormal synthesis of some cytokines (principal molecular mediators of the initiation, development, and regulation of allergic inflammation) has an important pathogenic role in allergy. In recent years, it is generally accepted that allergic disorders are accounted for by impaired immune regulation resulting from increased imbalanced activation of allergen-specific T helper 2 cell clones. Allergic type of immune response is currently referred to as type 2 immune response which normally provides humoral immunity, anthelmintic protection etc. However, pathological response results in allergy. Studies on immune pathogenesis of allergic disorders have improved our understanding of the role of altered synthesis of some cytokines (key mediators of allergic inflammation). Cytokines involved in the initiation and regulation of allergy are produced by epithelial cells, various subsets of leukocytes, innate lymphoid cells, and allergen-activated T helper cell clones. Analysis of the associations of the changes in immune reactivity and clinical manifestations has established pathogenic heterogeneity and identified asthma phenotypes and endotypes. These findings have provided a basis for more reasonable, successful, and personalized approach to biological anti-cytokine treatment for allergic disorders. KEYWORDS: allergy, cytokines, T helper cell clones, phenotype, asthma, anti-cytokine therapy. FOR CITATION: Simbirtsev A.S. Cytokines and their role in immune pathogenesis of allergy. Russian Medical Inquiry. 2021;5(1):32–37. DOI: 10.32364/2587-6821-2021-5-1-32-37.
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Williams, Jesse W., Melissa Y. Tjota, and Anne I. Sperling. "The Contribution of Allergen-Specific IgG to the Development of Th2-Mediated Airway Inflammation." Journal of Allergy 2012 (October 21, 2012): 1–9. http://dx.doi.org/10.1155/2012/236075.

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In both human asthmatics and animal models of allergy, allergen-specific IgG can contribute to Th2-mediated allergic inflammation. Mouse models have elucidated an important role for IgG and Fc-gamma receptor (FcγR) signaling on antigen presenting cells (APC) for the induction of airway inflammation. These studies suggest a positive feedback loop between IgG produced by the adaptive B cell response and FcγR signaling on innate immune cells. Studies of IgG and FcγRs in humans with asthma or allergic lung disease have been more controversial. Some reports have identified associations between allergen-specific IgG and severity of allergic responses, while other studies have found associations of IgG subclass IgG4 with allergic tolerance. In this paper, we review the literature to help define the nature of IgG and FcγR signaling on innate immune cells and how it contributes to the development of allergic immune responses.
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Lei, Dawn K., and Leslie C. Grammer. "An overview of allergens." Allergy and Asthma Proceedings 40, no. 6 (November 1, 2019): 362–65. http://dx.doi.org/10.2500/aap.2019.40.4247.

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Most allergens are proteins or glycoproteins that range in molecular weight from 5000 to 100,000 Da, although polysaccharides and low-molecular-weight substances may also be allergenic. Common allergens include pollens, fungal spores, house-dust mites, and animal epithelial materials but can also include drugs, biologic products, and insect venoms. The allergic response is dependent on the route of exposure. If the exposure is to an inhaled aeroallergen, then the allergic response will be respiratory in nature. Ingested or injected exposure gives rise to gastrointestinal, cutaneous, or anaphylactic reactions. The size of the pollen determines the clinical manifestation of allergy. For example, particles between 20 and 60 μm in diameter can be carried by the wind and cause nasal and ocular symptoms (allergic rhinoconjunctivitis). Particles of <7 μm can deposit in the airways and cause symptoms of asthma. Animals produce allergens in forms unique to each species. Cat allergen, most importantly Fel d 1, is buoyant and “sticky,” which means it easily remains airborne and may last in a home for up to 6 to 9 months after the source is removed. Cat allergen adheres to clothes and can be found in public places, e.g., schools. Dog allergen, particularly Can f 1, is present in dander, saliva, urine, and serum. All dog breeds produce allergenic proteins (even poodles and “hairless” dogs).
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Gushchin, I. S. "Evolutionary background of allergic reactivity: mast cells, FcεRI, IgE - three components of the effector phase of the allergic response." Russian Journal of Allergy 15, no. 4 (December 15, 2018): 5–16. http://dx.doi.org/10.36691/rja131.

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The literature data on the evolution of the main obligatory participants in the effector phase of the IgE-mediated allergic response are presented: mast cells/basophils, immunoglobulin E, and high affinity receptor for the Fcε fragment (FcεRI). Allergic reactivity is considered as the most recent evolutionary immunologically-mediated acquisition of mammals. It is aimed at recognizing small amounts of allergen entering the body in a certain time regime, and organizing an allergen-specific inflammation that carries features of elimination function. The most biologically justified way to prevent allergies is to restore the function of barrier systems and, accordingly, to prevent the need to develop an allergic response.
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Dissertations / Theses on the topic "Allergic response"

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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135731.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress may facilitate and/or consolidate immunological aberrations and thus, may increase the risk for allergic sensitization and exacerbation especially under stressful conditions. In the present review the potential impact of a hyporesponsive as well as a hyperresponsive HPA axis on the onset and chronification of childhood allergy is summarized. Moreover, potential factors that may contribute to the development of an aberrant HPA axis responsiveness in allergy are discussed
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response." Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27671.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress may facilitate and/or consolidate immunological aberrations and thus, may increase the risk for allergic sensitization and exacerbation especially under stressful conditions. In the present review the potential impact of a hyporesponsive as well as a hyperresponsive HPA axis on the onset and chronification of childhood allergy is summarized. Moreover, potential factors that may contribute to the development of an aberrant HPA axis responsiveness in allergy are discussed.
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich.
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Cameron, Elizabeth Anne. "Local isotype switching to IgE within allergic nasal mucosa in response to allergen exposure." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0035/NQ64528.pdf.

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Al, Hamdani A. "The measurement of muscle strength in allergic response." Thesis, University of Salford, 1990. http://usir.salford.ac.uk/14798/.

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The purpose of this thesis was to show a relationship between muscle strength and allergic reactions. Clinics have for many years relied on what have become traditional methods for testing for allergic reactions. These include such tests as the Patch test and the Skin-Prick test. Both these tests cause discomfort and irritation to sensitive patients. The historical development of allergies is outlined, showing that scientists and clinicians in the late 18th and early 19th century were aware of certain reactions which people showed to apparently normal non-reactive foods. The development of methods of diagnosing allergies such as the elimination diet, which dates back to approx 1940, are surveyed. Current practices are also studied with the use of photographs. The reaction of the skin to modern day testing practices is shown. In some cases these reactions are quite violent. The Applied Kinesiology Test is examined as a universally used test which when carried out by an experienced Kinesiologist can give a true but highly subjective response to a patients allergic condition. A system has been developed based on the relationship between muscle strength and allergies, the components of this system were readily available at little cost. Once a basic system was built it was possible to perform trials on patients at an established allergy clinic. With the aid of a data printer it became possible to prove first graphically and second statistically that there was a statistically valid relationship between the measured muscle strength and allergies. The conclusion shows that the final version of the operating system is capable of equaling the performance of an experienced Kinesiologist, showing its results both digitally and graphically rather than subjectively. The various deductions and recommendations which are detailed, point towards a readily marketable device which could represent a potential break through in modern allergy testing. The system will also open the way for clinicians to make an objective assessment of the relationship between muscle strength and allergic sensitivity via further research.
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Spada, Clayton Samuel. "Mediators of acute inflammation and their roles in modulating in vivo leukocyte infiltration and pathobiologic activity in the conjunctiva." Thesis, De Montfort University, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278549.

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Elms, Joanne. "The immune functions of immunoglobulin D (IgD) and the allergic response." Thesis, University of Sheffield, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.370051.

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Zhu, Yan [Verfasser]. "Impact of gram-negative bacteria on the allergic immune response in a mouse model of allergen-induced eczema / Yan Zhu." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2010. http://d-nb.info/1025087461/34.

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Wang, Junji. "Therapeutic vaccines based on myobacterium vaccae for the treatment of an IgE response to ovalbumin in BALB/c mice." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285207.

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McDermott, Jacqueline Ruth. "The influence of dendritic cells on the differentiation of T helper cells." Thesis, University of Surrey, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326509.

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Koprich, James B. "Ontogeny of the fetal immune response to maternal smoking in relation to allergic asthma." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ60854.pdf.

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Books on the topic "Allergic response"

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Hamdani, Alharith Al. The measurement of muscle strength in allergic response. Salford: University of Salford, 1990.

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Chadwick, Derek J., David Evered, and Julie Whelan, eds. Ciba Foundation Symposium 147 - IgE, Mast Cells and the Allergic Response. Chichester, UK: John Wiley & Sons, Ltd., 1989. http://dx.doi.org/10.1002/9780470513866.

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Allergy information for teens: Health tips about allergic reactions to food, pollen, mold, and other substances, including facts about diagnosing, treating, and preventing allergic responses and complications. Detroit, MI: Omnigraphics, 2013.

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Brown, Edgar. Your body's response. Dallas, Tex: Madison Avenue Pub. Co., 1985.

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Mak, Tak W. Handbook of immune response genes. New York: Plenum Press, 1998.

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Benjamini, Eli. Immunology: A short course. 3rd ed. New York: Wiley-Liss, 1996.

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Benjamini, Eli. Immunology: A short course. 2nd ed. New York: Wiley-Liss, 1991.

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Benjamini, Eli. Immunology: A short course. New York: Liss, 1988.

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I was poisoned by my body--I have a gut feeling you could be, too!: The odyssey of a doctor who reversed fibromyalgia, leaky gut syndrome, multiple allergic responses, naturally and her life 10 years after recovery. 2nd ed. Lancaster, OH: Lucky Press, 2007.

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Munster, C. Sorg. Cytokines Regulating the Allergic Response (Cytokines). S Karger Pub, 1989.

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Book chapters on the topic "Allergic response"

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Turner, Keven J. "Epidemiology of the Allergic Response." In Novartis Foundation Symposia, 205–29. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470513866.ch13.

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Liu, G., A. Keane-Myers, D. Miyazaki, A. Tai, and S. J. Ono. "Molecular and Cellular Aspects of Allergic Conjunctivitis." In Immune Response and the Eye, 39–58. Basel: KARGER, 1999. http://dx.doi.org/10.1159/000058748.

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Flint, Kevin Charles. "Mast Cells and the Allergic Response." In The Bloomsbury Series in Clinical Science, 1–12. London: Springer London, 1987. http://dx.doi.org/10.1007/978-1-4471-1458-1_1.

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Gumowski, P. I., S. Dunoyer-Geindre, and J. P. Latgé. "Candida Cell Wall Antigens Inducing Allergic Symptoms." In Fungal Cell Wall and Immune Response, 383–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76074-7_29.

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Nagler-Anderson, Cathryn. "Helminth-Induced Immunoregulation of an Allergic Response to Food." In Parasites and Allergy, 1–13. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000088876.

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Brusasco, Vito, Emanuele Crimi, and Giovanni A. Rossi. "Airway Responsiveness, Late-Phase Response, and Inflammation in Allergic Asthma." In Asthma Treatment, 155–60. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3446-4_13.

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Kalicki, Bolesław, Agnieszka Lipińska-Opałka, Katarzyna Kowalczyk, Katarzyna Mycroft, Joanna Królikowska, Agnieszka Rustecka, and Agata Wawrzyniak. "Excessive Body Weight and Immunological Response in Children with Allergic Diseases." In Advances in Experimental Medicine and Biology, 77–87. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/5584_2019_426.

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Gregory, Greg D., Allison Bickford, Michaela Robbie-Ryan, Mindy Tanzola, and Melissa A. Brown. "MASTering the Immune Response: Mast Cells in Autoimmunity." In Mast Cells and Basophils: Development, Activation and Roles in Allergic/Autoimmune Disease, 215–31. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470033449.ch18.

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Rieber, E. P., U. Pirron, N. Endres, and J. C. Prinz. "Human Fc ε RII/CD23 in the Regulation of the Allergic Immune Response." In New Trends in Allergy III, 82–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46717-2_10.

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Abramson, J., E. A. Barbu, and I. Pecht. "Regulation of Mast Cell Secretory Response to the Type I Fcε Receptor: Inhibitory Elements and Desensitization." In Mast Cells and Basophils: Development, Activation and Roles in Allergic/Autoimmune Disease, 78–94. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470033449.ch6.

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Conference papers on the topic "Allergic response"

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SANJAR, S., D. SMITH, J. MORLEY, L. MAZZONI, and C. TAPPARELLI. "PAF ANTAGONISM AND THE RESPONSE TO ALLERGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644865.

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Intravenous infusion of platelet activating factor (PAF) causes platelets to aggregate and accumulate within the lung. A similar effect is observed when allergen is injected into sensitised animals. Since PAF is released in allergic reactions, it might be considered to be a mediator of this phenomenon. Intrathoracic accumulation of 111-Indium labelled platelets was detected by use of collimated sodium iodide crystal detectors as a part of an automated isotope monitoring system (AIMS 8000, Mumed ltd.). Intravenous infusion of PAF (600 ng/kg/h) caused progressive increase of the intrathoracic platelet content (TPC) (59%). Infusion of small doses of allergen (BGG, 300 ug/kg/h) produced comparable increase of TPC, whether animals were sensitised actively (1 mg/kg BGG+FCA i.p. and boosted two weeks later) (30%) or passively (i.v. injection of 0.25 ml anti-BGG serum) (53%) or received intravenous injections of preformed immune complexes. At a dose of 2 mg/kg/h, ginkgolide B (−5%) or kadsuranone (−1%) fully inhibited increased TPC in response to PAF. However, at higher doses (6 mg/kg/h) ginkgolide B did not diminish TPC in animals that were actively (33%) or passively (60%) sensitised, nor did kadsuranone (6 mg/kg/h) diminish the response in passively sensitised animals (42%) compared to vehicle animals (43%). These observations can be extended to acute bronchospasm and airway hyperreactivity which are secondary to platelet activation in these animals. It can be concluded that PAF formation appears to be a minor determinant of the acute response to allergen in the guinea-pig.
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Harris, R. S., Jose G. Venegas, Vanessa J. Kelly, Tilo Winkler, Mamary Kone, Guido Musch, Marcos F. Vidal Melo, et al. "Ventilation, Perfusion And Airway Response To Segmental Allergen Challenge In Allergic Non-Asthmatic Subjects." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2408.

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Shah, Sajeel, Yanira Riffo Vasquez, Clive Page, and Simon Pitchford. "Platelets roll and adhere to the vascular bed in response to allergen challenge in allergic mice." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.oa4845.

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Chen, Q. L., H. P. Ho, Y. K. Suen, S. K. Kong, Wen J. Li, and C. K. Wong. "Design and fabrication of centrifugal microfluidic disk for allergic response monitoring." In 2009 14th OptoElectronics and Communications Conference (OECC). IEEE, 2009. http://dx.doi.org/10.1109/oecc.2009.5215688.

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Pollak, M., M. Shaw, D. Wilson, and F. A. Ratjen. "Bronchodilator Response in Cystic Fibrosis Patients Diagnosed with Allergic Bronchopulmonary Aspergillosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5351.

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Yuksel, H., E. Tayanc, O. Yilmaz, A. Yasar, and S. Inan. "Epithelial Barrier Dysfunction and Innate Immune System Interaction in Allergic Response." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2198.

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Wheelock, Asa M., Susanna Lundstrom, Jun Yang, Jesper Z. Haeggstrom, Bruce D. Hammock, Johan Grunewald, Magnus Nord, Marianne van Hage, Anders Eklund, and Craig E. Wheelock. "Allergic Asthmatics Exhibit Altered Response In Oxylipin Profile As Compared To Healthy And Asthmatic Controls After Allergen Provocation." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4447.

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Sadigov, Alizaman S. "Latent Tuberculosis Infection Suppresses Airway Allergic Response In Patients With Bronchial Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1434.

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Kelly, JFC, ER Davies, ST Holgate, X. Xu, JA Whitsett, DE Davies, and HM Haitchi. "S89 Soluble adam33 augments the pulmonary immune response promoting allergic airway sensitivity." In British Thoracic Society Winter Meeting 2017, QEII Centre Broad Sanctuary Westminster London SW1P 3EE, 6 to 8 December 2017, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2017. http://dx.doi.org/10.1136/thoraxjnl-2017-210983.95.

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DiGiovanni, FA, R. Ellis, J. Wattie, S. Goncharova, M. Jordana, and MD Inman. "Ozone Exposure Can Facilitate Allergen Sensitization and Enhance the Allergic Response to House Dust Mite in a Mouse Model." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2246.

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Reports on the topic "Allergic response"

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Dua, Shelley, Andrew Clark, Monica Ruiz-Garcia, Simon Bond, Stephen Durham, Ian Kimber, Clare Mills, et al. The effect of sleep deprivation and exercise on reaction threshold in peanut-allergic adults: a randomised controlled study. Food Standards Agency, May 2020. http://dx.doi.org/10.46756/sci.fsa.vjv675.

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This was a randomised cross-over trial that investigated whether common extrinsic factors, such as exercise and sleep deprivation can modulate the threshold of responses to allergenic foods in a representative group of adults from the peanut allergic population.
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Shibata, Yoshimi. Oral Administration of N-acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, May 2008. http://dx.doi.org/10.21236/ada484241.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada442684.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2006. http://dx.doi.org/10.21236/ada454070.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D-Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2007. http://dx.doi.org/10.21236/ada469207.

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Shibata, Yoshimi. Oral Administration of a N-Acetyl-D-Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2004. http://dx.doi.org/10.21236/ada424227.

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McPhedran, R., K. Patel, B. Toombs, P. Menon, M. Patel, J. Disson, K. Porter, A. John, and A. Rayner. Food allergen communication in businesses feasibility trial. Food Standards Agency, March 2021. http://dx.doi.org/10.46756/sci.fsa.tpf160.

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Background: Clear allergen communication in food business operators (FBOs) has been shown to have a positive impact on customers’ perceptions of businesses (Barnett et al., 2013). However, the precise size and nature of this effect is not known: there is a paucity of quantitative evidence in this area, particularly in the form of randomised controlled trials (RCTs). The Food Standards Agency (FSA), in collaboration with Kantar’s Behavioural Practice, conducted a feasibility trial to investigate whether a randomised cluster trial – involving the proactive communication of allergen information at the point of sale in FBOs – is feasible in the United Kingdom (UK). Objectives: The trial sought to establish: ease of recruitments of businesses into trials; customer response rates for in-store outcome surveys; fidelity of intervention delivery by FBO staff; sensitivity of outcome survey measures to change; and appropriateness of the chosen analytical approach. Method: Following a recruitment phase – in which one of fourteen multinational FBOs was successfully recruited – the execution of the feasibility trial involved a quasi-randomised matched-pairs clustered experiment. Each of the FBO’s ten participating branches underwent pair-wise matching, with similarity of branches judged according to four criteria: Food Hygiene Rating Scheme (FHRS) score, average weekly footfall, number of staff and customer satisfaction rating. The allocation ratio for this trial was 1:1: one branch in each pair was assigned to the treatment group by a representative from the FBO, while the other continued to operate in accordance with their standard operating procedure. As a business-based feasibility trial, customers at participating branches throughout the fieldwork period were automatically enrolled in the trial. The trial was single-blind: customers at treatment branches were not aware that they were receiving an intervention. All customers who visited participating branches throughout the fieldwork period were asked to complete a short in-store survey on a tablet affixed in branches. This survey contained four outcome measures which operationalised customers’: perceptions of food safety in the FBO; trust in the FBO; self-reported confidence to ask for allergen information in future visits; and overall satisfaction with their visit. Results: Fieldwork was conducted from the 3 – 20 March 2020, with cessation occurring prematurely due to the closure of outlets following the proliferation of COVID-19. n=177 participants took part in the trial across the ten branches; however, response rates (which ranged between 0.1 - 0.8%) were likely also adversely affected by COVID-19. Intervention fidelity was an issue in this study: while compliance with delivery of the intervention was relatively high in treatment branches (78.9%), erroneous delivery in control branches was also common (46.2%). Survey data were analysed using random-intercept multilevel linear regression models (due to the nesting of customers within branches). Despite the trial’s modest sample size, there was some evidence to suggest that the intervention had a positive effect for those suffering from allergies/intolerances for the ‘trust’ (β = 1.288, p<0.01) and ‘satisfaction’ (β = 0.945, p<0.01) outcome variables. Due to singularity within the fitted linear models, hierarchical Bayes models were used to corroborate the size of these interactions. Conclusions: The results of this trial suggest that a fully powered clustered RCT would likely be feasible in the UK. In this case, the primary challenge in the execution of the trial was the recruitment of FBOs: despite high levels of initial interest from four chains, only one took part. However, it is likely that the proliferation of COVID-19 adversely impacted chain participation – two other FBOs withdrew during branch eligibility assessment and selection, citing COVID-19 as a barrier. COVID-19 also likely lowered the on-site survey response rate: a significant negative Pearson correlation was observed between daily survey completions and COVID-19 cases in the UK, highlighting a likely relationship between the two. Limitations: The trial was quasi-random: selection of branches, pair matching and allocation to treatment/control groups were not systematically conducted. These processes were undertaken by a representative from the FBO’s Safety and Quality Assurance team (with oversight from Kantar representatives on pair matching), as a result of the chain’s internal operational restrictions.
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