Relevant bibliographies by topics / Airway (Medicine)

Academic literature on the topic 'Airway (Medicine)'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Airway (Medicine)"

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Ghosh, Sohini, Allen Cole Burks, and Jason A. Akulian. "Customizable airway stents—personalized medicine reaches the airways." Journal of Thoracic Disease 11, S9 (May 2019): S1129—S1131. http://dx.doi.org/10.21037/jtd.2019.03.100.

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Pimenta de Paula, Fernando, Ariele Patrícia da Silva, and Luciano Alves Matias da Silveira. "Knowledge about airway approach in medicine academics." Journal of Anesthesia & Critical Care: Open Access 13, no. 4 (July 26, 2021): 142–43. http://dx.doi.org/10.15406/jaccoa.2021.13.00485.

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Introduction: The approach to the airways is a common practice in medical life, being a delicate situation susceptible to complications, which requires skill. The objective of this work is to analyze the profile of airway instruction of students in the sixth year of medicine at a Federal University. Methods: Students answered an airway questionnaire. Theoretical knowledge about airway predictors, such as Mallampati and Cormack-Lehane classification, practices and mastery in orotracheal intubation, and use and knowledge of auxiliary devices for airway management were addressed. Answers were coded into spreadsheets for analysis. Results: Fifty students participated in the research (adherence of 62.5%), with a mean age of 25.5 years (22-37 years old) and equal gender divisions. The analysis showed that 100% of academics know the Mallampati Classification, but only 38% know the Cormack-Lehane. As for the practice of orotracheal intubation, 68% performed at least one orotracheal intubation in patients during graduation. About the knowledge of auxiliary and supraglottic devices, 86% say they know the Bougie and 82% at least one supraglottic device. As for the need to establish a surgical airway, 32% reported being able to establish it. Conclusion: Despite the teaching of anesthesiology in the mandatory curriculum at our institution, there is a need for new approaches at different times in the academic course. The practice of activities in the fields of teaching, research and extension aimed at consolidating knowledge of predictors and training in situations of airway management and the use of auxiliary devices should be encouraged.
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Klučka, Jozef, Petr Štourač, Roman Štoudek, Michaela Ťoukálková, Hana Harazim, and Martina Kosinová. "Controversies in Pediatric Perioperative Airways." BioMed Research International 2015 (2015): 1–11. http://dx.doi.org/10.1155/2015/368761.

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Pediatric airway management is a challenge in routine anesthesia practice. Any airway-related complication due to improper procedure can have catastrophic consequences in pediatric patients. The authors reviewed the current relevant literature using the following data bases: Google Scholar, PubMed, Medline (OVID SP), and Dynamed, and the following keywords: Airway/s, Children, Pediatric, Difficult Airways, and Controversies. From a summary of the data, we identified several controversies: difficult airway prediction, difficult airway management, cuffed versus uncuffed endotracheal tubes for securing pediatric airways, rapid sequence induction (RSI), laryngeal mask versus endotracheal tube, and extubation timing. The data show that pediatric anesthesia practice in perioperative airway management is currently lacking the strong evidence-based medicine (EBM) data that is available for adult subpopulations. A number of procedural steps in airway management are derived only from adult populations. However, the objective is the same irrespective of patient age: proper securing of the airway and oxygenation of the patient.
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Russell, Richard J., and Christopher Brightling. "Pathogenesis of asthma: implications for precision medicine." Clinical Science 131, no. 14 (June 30, 2017): 1723–35. http://dx.doi.org/10.1042/cs20160253.

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The pathogenesis of asthma is complex and multi-faceted. Asthma patients have a diverse range of underlying dominant disease processes and pathways despite apparent similarities in clinical expression. Here, we present the current understanding of asthma pathogenesis. We discuss airway inflammation (both T2HIGH and T2LOW), airway hyperresponsiveness (AHR) and airways remodelling as four key factors in asthma pathogenesis, and also outline other contributory factors such as genetics and co-morbidities. Response to current asthma therapies also varies greatly, which is probably related to the inter-patient differences in pathogenesis. Here, we also summarize how our developing understanding of detailed pathological processes potentially translates into the targeted treatment options we require for optimal asthma management in the future.
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Tanabe, Naoya, Kaoruko Shimizu, Kunihiko Terada, Susumu Sato, Masaru Suzuki, Hiroshi Shima, Akira Oguma, et al. "Central airway and peripheral lung structures in airway disease-dominant COPD." ERJ Open Research 7, no. 1 (January 2021): 00672–2020. http://dx.doi.org/10.1183/23120541.00672-2020.

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The concept that the small airway is a primary pathological site for all COPD phenotypes has been challenged by recent findings that the disease starts from the central airways in COPD subgroups and that a smaller central airway tree increases COPD risk. This study aimed to examine whether the computed tomography (CT)-based airway disease-dominant (AD) subtype, defined using the central airway dimension, was less associated with small airway dysfunction (SAD) on CT, compared to the emphysema-dominant (ED) subtype.COPD patients were categorised into mild, AD, ED and mixed groups based on wall area per cent (WA%) of the segmental airways and low attenuation volume per cent in the Kyoto–Himeji (n=189) and Hokkaido COPD cohorts (n=93). The volume per cent of SAD regions (SAD%) was obtained by nonrigidly registering inspiratory and expiratory CT.The AD group had a lower SAD% than the ED group and similar SAD% to the mild group. The AD group had a smaller lumen size of airways proximal to the segmental airways and more frequent asthma history before age 40 years than the ED group. In multivariable analyses, while the AD and ED groups were similarly associated with greater airflow limitation, the ED, but not the AD, group was associated with greater SAD%, whereas the AD, but not the ED, group was associated with a smaller central airway size.The CT-based AD COPD subtype might be associated with a smaller central airway tree and asthma history, but not with peripheral lung pathologies including small airway disease, unlike the ED subtype.
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Sorkness, Ronald L., Kathryn M. Herricks, Renee J. Szakaly, Robert F. Lemanske, and Louis A. Rosenthal. "Altered allergen-induced eosinophil trafficking and physiological dysfunction in airways with preexisting virus-induced injury." American Journal of Physiology-Lung Cellular and Molecular Physiology 292, no. 1 (January 2007): L85—L91. http://dx.doi.org/10.1152/ajplung.00234.2006.

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Although both asthmatics and allergic rhinitics develop an acute inflammatory response to lower airway allergen challenge, only asthmatics experience airway obstruction resulting from chronic environmental allergen exposure. Hypothesizing that asthmatic airways have an altered response to chronic allergic inflammation, we compared the effects of repeated low-level exposures to inhaled Alternaria extract in sensitized rats with preexisting chronic postbronchiolitis airway dysfunction versus sensitized controls with normal airways. Measurements of air space (bronchoalveolar lavage) inflammatory cells, airway goblet cells, airway wall collagen, airway wall eosinophils, airway alveolar attachments, and pulmonary physiology were conducted after six weekly exposures to aerosolized saline or Alternaria extract. Postbronchiolitis rats, but not those starting with normal airways, had persistent increases in airway wall eosinophils, goblet cell hyperplasia in small airways, and loss of lung elastic recoil after repeated exposure to aerosolized Alternaria extract. Despite having elevated airway wall eosinophils, the postbronchiolitis rats had no eosinophils in bronchoalveolar lavage at 5 days after the last allergen exposure, suggesting altered egression of tissue eosinophils into the air space. In conclusion, rats with preexisting airway pathology had altered eosinophil trafficking and allergen-induced changes in airway epithelium and lung mechanics that were absent in sensitized control rats that had normal airways before the allergen exposures.
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Rosenberg, M. B., J. C. Phero, and D. E. Becker. "Essentials of Airway Management, Oxygenation, and Ventilation: Part 2: Advanced Airway Devices: Supraglottic Airways." Anesthesia Progress 61, no. 3 (September 1, 2014): 113–18. http://dx.doi.org/10.2344/0003-3006-61.3.113.

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Abstract Offices and outpatient dental facilities must be properly equipped with devices for airway management, oxygenation, and ventilation. Part 1 in this series on emergency airway management focused on basic and fundamental considerations for supplying supplemental oxygen to the spontaneously breathing patient and utilizing a bag-valve-mask system including nasopharyngeal and oropharyngeal airways to deliver oxygen under positive pressure to the apneic patient. This article will review the evolution and use of advanced airway devices, specifically supraglottic airways, with the emphasis on the laryngeal mask airway, as the next intervention in difficult airway and ventilation management. The final part of the series (part 3) will address airway evaluation, equipment and devices for tracheal intubation, and invasive airway procedures.
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McKune, AJ, and LL Smith. "Airways inflammatory and atopy-related responses in athletes." South African Journal of Sports Medicine 18, no. 2 (February 3, 2009): 46. http://dx.doi.org/10.17159/2413-3108/2006/v18i2a243.

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The prevalence of asthma and airway hyperresponsiveness (AHR) in highly trained endurance athletes is rising. The type of training (i.e. endurance, or speed and power) seems to influence the airway symptoms. High-intensity exercise and training might contribute to the development of asthma or AHR in athletes previously unaffected by these airway disorders. Repeated hyperventilation of unconditioned air, as well as air containing irritants and/or allergens has been suggested to cause thermal, mechanical, or osmotic airway trauma resulting in damage to the airway epithelium. Subsequent airway inflammatory responses may be responsible for the development of atopy-related symptoms in endurance athletes such as those observed in asthma and AHR. Eosinophils and neutrophils are the inflammatory cells that have been frequently observed to be elevated in the airways of endurance athletes. The trafficking of these cells to the airways may possibly be regulated by TH2 cytokines that are expressed in the airways in response to epithelial cell damage. In addition, these airway inflammatory responses may lead to airway remodelling similar to that which occurs in asthma. The effect of the exercise challenge itself may initiate airway atopy-related and inflammatory responses in endurance athletes. While the literature seems to support the role of local airway conditions and/or events in inducing atopy-related symptoms in athletes, it is proposed that alterations in the hormonal and/or cytokine milieu with intense competition and/or training may also play a role. South African Journal of Sports Medicine Vol. 18 (2) 2006: pp. 46-51
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McKune, AJ, and LL Smith. "Airways inflammatory and atopy-related responses in athletes." South African Journal of Sports Medicine 18, no. 2 (February 3, 2006): 46. http://dx.doi.org/10.17159/2078-516x/2006/v18i2a243.

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The prevalence of asthma and airway hyperresponsiveness (AHR) in highly trained endurance athletes is rising. The type of training (i.e. endurance, or speed and power) seems to influence the airway symptoms. High-intensity exercise and training might contribute to the development of asthma or AHR in athletes previously unaffected by these airway disorders. Repeated hyperventilation of unconditioned air, as well as air containing irritants and/or allergens has been suggested to cause thermal, mechanical, or osmotic airway trauma resulting in damage to the airway epithelium. Subsequent airway inflammatory responses may be responsible for the development of atopy-related symptoms in endurance athletes such as those observed in asthma and AHR. Eosinophils and neutrophils are the inflammatory cells that have been frequently observed to be elevated in the airways of endurance athletes. The trafficking of these cells to the airways may possibly be regulated by TH2 cytokines that are expressed in the airways in response to epithelial cell damage. In addition, these airway inflammatory responses may lead to airway remodelling similar to that which occurs in asthma. The effect of the exercise challenge itself may initiate airway atopy-related and inflammatory responses in endurance athletes. While the literature seems to support the role of local airway conditions and/or events in inducing atopy-related symptoms in athletes, it is proposed that alterations in the hormonal and/or cytokine milieu with intense competition and/or training may also play a role. South African Journal of Sports Medicine Vol. 18 (2) 2006: pp. 46-51
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Kim, Ki-Suk, Dong-Hyuk Cho, Hea Jung Yang, Eun-Kyeong Choi, Min Hee Shin, Kang-Hoon Kim, Kwang Seok Ahn, et al. "Effects of the Inhaled Treatment of Liriope Radix on an Asthmatic Mouse Model." American Journal of Chinese Medicine 43, no. 03 (January 2015): 425–41. http://dx.doi.org/10.1142/s0192415x15500275.

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As a treatment for allergic asthma, inhaled treatments such as bronchodilators that contain β2-agonists have an immediate effect, which attenuates airway obstructions and decreases airway hypersensitivity. However, bronchodilators only perform on a one off basis, but not consistently. Asthma is defined as a chronic inflammatory disease of the airways accompanying the overproduction of mucus, airway wall remodeling, bronchial hyperreactivity and airway obstruction. Liriope platyphylla radix extract (LPP), a traditional Korean medicine, has been thoroughly studied and found to be an effective anti-inflammatory medicine. Here, we demonstrate that an inhaled treatment of LPP can attenuate airway hyperresponsiveness (AHR) in an ovalbumin-induced asthmatic mouse model, compared to the saline-treated group (p < 0.01). Moreover, LPP decreases inflammatory cytokine levels, such as eotaxin (p < 0.05), IL-5 (p < 0.05), IL-13 (p < 0.001), RANTES (p < 0.01), and TNF-α (p < 0.05) in the bronchoalveolar lavage (BAL) fluid of asthmatic mice. A histopathological study was carried out to determine the effects of LPP inhalation on mice lung tissue. We performed UPLC/ESI-QTOF-MS, LC/MS, and GC/MS analyses to analyze the chemical constituents of LPP, finding that these are ophiopogonin D, spicatoside A, spicatoside B, benzyl alcohol, and 5-hydroxymethylfurfural. This study demonstrates the effect of an inhaled LPP treatment both on airway AHR and on the inflammatory response in an asthmatic mouse model. Hence, LPP holds significant promise as a nasal inhalant for the treatment of asthmatic airway disease.
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Dissertations / Theses on the topic "Airway (Medicine)"

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Svensson, Holm Ann-Charlotte B. "Platelets and airway remodeling : Mechanisms involved in platelet-induced fibroblast and airway smooth muscle cell proliferation in vitro." Doctoral thesis, Linköpings universitet, Farmakologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-61623.

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Airway remodeling is a contributing cause to the pathological structural changes, such as increased cell proliferation, observed in asthma. Platelets have been found in autopsy lungmaterial obtained from asthmatic patients and are well known to induce proliferation in vitro of a variety of cells. However, the role of platelets in airway remodeling is far from understood. This thesis aims to clarify the involvement of platelets in fibroblast and airway smooth muscle cell (ASMC) proliferation in vitro and to elucidate the importance of HA, FAK, eicosanoid and ROS dependent signaling. The results demonstrate that platelets induce ASMC proliferation through NADPH-oxidase and 5-LOX dependent mechanisms. In addition, platelets also induce a 5-LOX dependent fibroblast proliferation. Furthermore, morphological analysis demonstrates that platelets bind to the extracellular matrix component HA through its receptor CD44 and thereby induce a FAK dependent ASMC proliferation. Taken together, the results obtained in this thesis suggest that platelet/HA interaction mediated through CD44 is of importance for platelets ability to induce cell proliferation. Moreover, the results propose that platelet-induced fibroblast proliferation is 5-LOX dependent and that platelets induce a HA, CD44, FAK, 5-LOX, and ROSdependent ASMC proliferation. This action of platelets represents a potential important and novel mechanism that may have an impact on the remodeling process and in the development of new pharmacological strategies in the treatment of inflammatory respiratory disease such as asthma.
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Noble, Peter Beresford. "Regulation of airway narrowing by dynamic and static mechanical loads." University of Western Australia. School of Biomedical and Chemical Sciences, 2006. http://theses.library.uwa.edu.au/adt-WU2006.0050.

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[Truncated abstract] The extent to which an airway narrows is strongly influenced by mechanical loads on airway smooth muscle (ASM). This thesis considers both dynamic and static mechanical loads. Dynamic load describes the time varying load on airways produced by oscillatory breathing movements. Static load is that present at a fixed lung volume ie. without breathing. In the intact lung static load principally comprises the pressure across the airway wall, that is transmural pressure (Ptm), and elastic after-load arising from distortion of airway and lung tissue by the narrowing airway. The experiments performed in this thesis were designed to answer several outstanding questions relating to how dynamic and static loads regulate airway narrowing. Dynamic load from breathing movements cyclically stretches ASM, which produces a number of physiological and cellular effects. For example in ASM strips a period of cyclical stretch reduces subsequent ASM contraction. However the response of the whole airway to dynamic load may differ from isolated ASM where non-muscle tissue also contributes. The first aim of this thesis was to characterise the response of the whole airway to dynamic load and determine whether the airway wall modifies the effects produced by ASM length cycling. Static after-loads restrict ASM shortening providing a limit to airway narrowing. Two primary sources of airway wall load include cartilage and the mucosal membrane which contribute to airway compliance. The relative importance of cartilage and mucosa to airway wall compliance and airway narrowing is unclear. ... Results demonstrate that airway narrowing is restricted by Ptm but not by parenchymal elastic after-load. The major findings of this thesis are: (1) dynamic loads produced by breathing movements regulate airway responsiveness through cyclical airway expansion and elongation; (2) the reported effects of cyclical stretch on ASM contraction differs in situ 8 possibly due to modification by one or more biomechanical or physiological properties of the airway wall; (3) parenchymal elastic after-loads, previously thought to be important during bronchoconstriction, do not restrict airway narrowing. Given the absence of an effect of parenchymal elastic after-load on airway narrowing, the static mechanical load on ASM therefore comprises Ptm and airway wall stiffness, with important contributions from cartilage and mucosa depending on lung volume.
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Lau, Justine Y. "Novel genes associated with airway smooth muscle proliferation in asthma." Connect to full text, 2008. http://hdl.handle.net/2123/5134.

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Thesis (Ph. D.)--University of Sydney, 2009.
Title from title screen (viewed Aug. 11, 2009) Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Discipline of Pharmacology, Faculty of Medicine. Degree awarded 2009; thesis submitted 2008. Includes bibliographical references. Also available in print form.
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Mohammadi, Hamed. "Cephalometric airway measurements in anterior open bite deformity." Thesis, [Hong Kong : Faculty of Dentistry], The University of Hong Kong, 1997. http://sunzi.lib.hku.hk/HKUTO/record/B3862820X.

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Grainge, Christopher. "Determinants of airway remodelling in asthma." Thesis, University of Southampton, 2010. https://eprints.soton.ac.uk/384164/.

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Fitch, Patrick Stephen. "A study of airway inflammation in childhood asthma." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326411.

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Pandya, Hitesh Champaklal. "Investigations of fetal human airway smooth muscle : potential mechanisms of abnormal airway wall modelling in chronic lung disease of preterm infants." Thesis, King's College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248390.

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Ip, Sau-man Mary. "A pathophysiologic study of airway inflammation in bronchiectasis." [Hong Kong : University of Hong Kong], 1991. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13793895.

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Tang, Shu-sum. "Cephalometric airway measurements in class III skeletal deformity." Click to view the E-thesis via HKUTO, 2000. http://sunzi.lib.hku.hk/HKUTO/record/B38628065.

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葉秀文 and Sau-man Mary Ip. "A pathophysiologic study of airway inflammation in bronchiectasis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1991. http://hub.hku.hk/bib/B31981434.

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Books on the topic "Airway (Medicine)"

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Gareth, Jones J., and Hanning Christopher D, eds. The Upper airway. London: Baillière Tindall, 1995.

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1952-, Howarth Peter, ed. Airway remodeling. New York: Marcel Dekker, 2001.

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Kovacs, George. Emergency airway management. New York, NY: McGraw-Hill, 2008.

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Jonathan, Benger, Nolan Jerry, and Clancy Mike, eds. Emergency airway management. Cambridge: Cambridge University Press, 2009.

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Gorback, Michael S. Emergency airway management. Philadelphia: B.C. Decker, 1990.

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Tony, Eissa N., and Huston David P, eds. Therapeutic targets in airway inflammation. New York: Marcel Dekker, 2003.

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1953-, Rogers D. F., and Lethem M. I. 1957-, eds. Airway mucus: Basic mechanisms and clinical perspectives. Basel: Birkhauser, 1997.

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Workshop on Airway Smooth Muscle (1983 Bethesda, Md.). Report of Workshop on Airway Smooth Muscle, September 25-27, 1983. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1985.

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National Institutes of Health (U.S.), ed. Report of Workshop on Airway Smooth Muscle, September 25-27, 1983. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1985.

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Orlando, Hung, and Murphy Michael F. 1954-, eds. Management of the difficult and failed airway. New York: McGraw-Hill, Medical Pub. Division, 2007.

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Book chapters on the topic "Airway (Medicine)"

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Aggarwal, Aarti, and Chidinma Osineme. "Obstructive Airway Disease." In Family Medicine, 1073–82. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-04414-9_90.

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Forno, Erick, Alejandro Díaz, and Juan Carlos Celedón. "Obstructive Airway Diseases." In Respiratory Medicine, 113–29. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43447-6_10.

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Weinberg, Howard N. "Obstructive Airway Disease." In Family Medicine, 726–33. New York, NY: Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2947-4_83.

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Weinberg, Howard. "Obstructive Airway Disease." In Family Medicine, 652–59. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4757-4005-9_85.

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Weinberg, Howard N. "Obstructive Airway Disease." In Family Medicine, 711–18. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/978-0-387-21744-4_83.

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Riley, Timothy D., and Ashley Morrison. "Obstructive Airway Disease." In Family Medicine, 1–14. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-0779-3_172-1.

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Aggarwal, Aarti, and Chidinma Osineme. "Obstructive Airway Disease." In Family Medicine, 1–10. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-1-4939-0779-3_90-1.

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Riley, Timothy D., and Ashley Morrison. "Obstructive Airway Disease." In Family Medicine, 1169–82. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-54441-6_172.

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Popovich, Marc J. "Airway Management." In Surgical Intensive Care Medicine, 23–35. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4757-6645-5_2.

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Ahmed, Hesham M., Christopher T. Aquina, Vicente H. Gracias, J. Javier Provencio, Mariano Alberto Pennisi, Giuseppe Bello, Massimo Antonelli, et al. "Definitive Airway." In Encyclopedia of Intensive Care Medicine, 688. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_3081.

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Conference papers on the topic "Airway (Medicine)"

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Pirogov, Aleksey, Anna Prikhodko, Evgeniya Afanas'eva, and Yuliy Perelman. "COMPARATIVE ASSESSMENT OF AIRWAY CELLULAR INFLAMMATION IN PATIENTS WITH BRONCHIAL ASTHMA IN RESPONSE TO HYPOSMOLAR AND COLD STIMULES." In XIV International Scientific Conference "System Analysis in Medicine". Far Eastern Scientific Center of Physiology and Pathology of Respiration, 2020. http://dx.doi.org/10.12737/conferencearticle_5fe01d9c45b256.10926397.

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An approach is presented to the study of cellular inflammation using cytological analysis of sputum in patients with bronchial asthma with different types of airway reaction to bronchoprovocation with cold air and distilled water. When the airways are hyperresponsive to hypoosmolar and cold stimuli, it has been established the activation of the neutrophilic component of bronchial granulocytes. Cold airway hyperresponsiveness is associated with an increase in neutrophil content and a concomitant decrease in the number of macrophages in the inflammatory pattern of the bronchi. An increase in sputum cytosis is inherent in a positive airway response to a hypoosmolar test with an unexpressed dynamics of the level of bronchial eosinophils.
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Adams, David C., Jasmin A. Holz, Margit V. Szabari, Lida P. Hariri, R. Scott Harris, Jocelyn L. Cho, Daniel L. Hamilos, Andrew D. Luster, Benjamin D. Medoff, and Melissa J. Suter. "A study of airway smooth muscle in asthmatic and non-asthmatic airways using PS-OCT (Conference Presentation)." In Optical Techniques in Pulmonary Medicine III, edited by Melissa J. Suter, Stephen Lam, and Matthew Brenner. SPIE, 2016. http://dx.doi.org/10.1117/12.2214735.

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Zavrel, Erik A., and Matthew R. Ebben. "A Novel Genioglossal Strengthening Device for Obstructive Sleep Apnea Treatment." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6809.

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Obstructive sleep apnea (OSA) is a condition characterized by temporary diminutions or cessations of breathing caused by repetitive collapse of the upper airway (UA) during sleep [1]. OSA is a common disorder — the American Academy of Sleep Medicine estimates the condition affects 29.4 million Americans — in which the muscles that hold the airway open weaken, resulting in partial or complete UA collapse during inspiration [2]. These pauses in breathing lead to blood oxygen desaturation and induce neurological arousal resulting in sleep disruption and fragmentation. The cycle of airway collapse and arousal can repeat hundreds of times per night.
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Hlastala, M. P. "Airway heat and gas exchange." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761175.

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Hlastala. "Airway Heat And Gas Exchange." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595790.

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Laviola, Marianna, Christian Niklas, Husam Alahmadi, Anup Das, Declan G. Bates, and Jonathan G. Hardman. "High oxygen fraction during airway opening is key to effective airway rescue in obese subjects." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857109.

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Pirogov, Aleksey, Irina Andrievskaya, Anna Prikhodko, Viktor Kolosov, and Yuliy Perelman. "PHOSPHATASE ACTIVITY AND POSSIBILITIES OF LYSOSOMAL SECRETION OF AIRWAY NEUTROPHILS IN PATIENTS WITH BRONCHIAL ASTHMA IN COLD-INDUCED STRESS." In XIV International Scientific Conference "System Analysis in Medicine". Far Eastern Scientific Center of Physiology and Pathology of Respiration, 2020. http://dx.doi.org/10.12737/conferencearticle_5fe01d9c3830b9.60748217.

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An approach to the study of cellular inflammation using methods of cytochemical analysis of sputum in patients with bronchial asthma with different types of airway reactions to bronchoprovocation with cold air is presented. Endotyping of patients contributes to a better understanding of the pathophysiological mechanisms and the choice of treatment tactics for the disease.
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Cheng, Irene, Sharmin Nilufar, Carlos Flores-Mir, and Anup Basu. "Airway Segmentation and Measurement in CT Images." In 2007 29th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2007. http://dx.doi.org/10.1109/iembs.2007.4352410.

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Louis, B., D. Isabey, and J. J. Fredberg. "Airway area by acoustic reflection: A new technique." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761176.

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Szabari, Margit V., Vanessa J. Kelly, Matthew B. Applegate, Chunmin Chee, Khay M. Tan, Lida P. Hariri, R. Scott Harris, Tilo Winkler, and Melissa J. Suter. "Using optical coherence tomography (OCT) imaging in the evaluation of airway dynamics (Conference Presentation)." In Optical Techniques in Pulmonary Medicine III, edited by Melissa J. Suter, Stephen Lam, and Matthew Brenner. SPIE, 2016. http://dx.doi.org/10.1117/12.2216306.

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Reports on the topic "Airway (Medicine)"

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Ma, He, Jifu Zhao, and Zhilei Wang. Efficacy and safety of HuaYu TongFu Method combined with acupuncture in the treatment of Acute Exacerbation of Chronic Obstructive Pulmonary Disease：A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0114.

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Review question / Objective: This study is the protocol for a systematic review to evaluate the efficacy and safety of HuaYu TongFu Method combined with acupuncture in the treatment of Acute Exacerbation of Chronic Obstructive Pulmonary Disease. we conducted a systematic review and meta-analysis of published randomized clinical trials (RCTs) of such combined therapy in the treatment of AECOPD, It provides a reliable scientific basis for clinicians to use this approach to treat AECOPD. Condition being studied: Chronic obstructive pulmonary disease is the third leading cause of death worldwide. AECOPD is the most common cause of hospitalization and death in patients with COPD. As lung function deteriorates and the disease progresses, the risk of alveolar hypoxia and consequent hypoxemia increases. Inflammation plays an important role in the progression of AECOPD. Modern medicine mainly treats AECPD by anti-inflammatory, relief of airway spasm, glucocorticoids, inhalants and other methods. Long-term application can easily lead to bacterial flora imbalance and drug resistance in patients. Comparatively, traditional Chinese medicine and acupuncture therapy are safe and effective.To assess the therapeutic efficacy and safety of HuaYu TongFu Method combined with acupuncture in AECOPD, we created a protocol for a systematic review to inform future clinical applications.
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Schwieger, Alexandra, Kaelee Shrewsbury, and Paul Shaver. Dexmedetomidine vs Fentanyl in Attenuating the Sympathetic Surge During Endotracheal Intubation: A Scoping Review. University of Tennessee Health Science Center, July 2021. http://dx.doi.org/10.21007/con.dnp.2021.0007.

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Purpose/Background Direct laryngoscopy and endotracheal intubation after induction of anesthesia can cause a reflex sympathetic surge of catecholamines caused by airway stimulation. This may cause hypertension, tachycardia, and arrhythmias. This reflex can be detrimental in patients with poor cardiac reserve and can be poorly tolerated and lead to adverse events such as myocardial ischemia. Fentanyl, a potent opioid, with a rapid onset and short duration of action is given during induction to block the sympathetic response. With a rise in the opioid crisis and finding ways to change the practice in medicine to use less opioids, dexmedetomidine, an alpha 2 adrenergic agonist, can decrease the release of norepinephrine, has analgesic properties, and can lower the heart rate. Methods In this scoping review, studies published between 2009 and 2021 that compared fentanyl and dexmedetomidine during general anesthesia induction and endotracheal intubation of surgical patients over the age of 18 were included. Full text, peer-reviewed studies in English were included with no limit on country of study. The outcomes included post-operative reviews of decrease in pain medication usage and hemodynamic stability. Studies that were included focused on hemodynamic variables such as systolic blood pressure, diastolic blood pressure, mean arterial pressure, heart rate, and use of opioids post-surgery. Result Of 2,114 results from our search, 10 articles were selected based on multiple eligibility criteria of age greater than 18, patients undergoing endotracheal intubation after induction of general anesthesia, and required either a dose of dexmedetomidine or fentanyl to be given prior to intubation. Dexmedetomidine was shown to effectively attenuate the sympathetic surge during intubation over fentanyl. Dexmedetomidine showed a greater reduction in heart rate, systolic blood pressure, diastolic blood pressure, mean arterial pressure than fentanyl, causing better hemodynamic stability in patients undergoing elective surgery.Implications for Nursing Practice Findings during this scoping review indicate that dexmedetomidine is a safe and effective alternative to fentanyl during induction of general anesthesia and endotracheal intubation in attenuating the hemodynamic response. It is also a safe choice for opioid-free anesthesia.
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