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Books on the topic 'Agoniste - Antagoniste'

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Published: 4 June 2021
Last updated: 6 February 2022

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1

Freye, Enno. Opioid Agonists, Antagonists and Mixed Narcotic Analgesics. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71854-0.

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2

Albrecht, Fleckenstein, ed. Cardiovascular effects of dihydropyridine-type calcium antagonists and agonists. Berlin: Springer-Verlag, 1985.

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3

Fleckenstein, Albrecht, Cornelius Van Breemen, Rainer Gross, and Friedrich Hoffmeister, eds. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70499-4.

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4

Small peptides: Chemistry, biology, and clinical studies. Amsterdam: Elsevier, 1993.

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5

Akhlaghi, Shirin. The metabolic effects of beta-agonists and antagonists: Focus on beta-1 selectivity. Birmingham: University of Birmingham, 1994.

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6

Freye, Enno. Opioid agonists, antagonists and mixed narcotics analgesics: Theoretical background and considerations for practical use. Berlin: Springer, 1987.

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7

Jain, K. K. Serotonin-modulating drugs: Therapeutic applications and commercial opportunities. [Waltham, MA]: Decision Resources, Inc., 1999.

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8

Neill, Joanna Caroline. The effects of 5-hydroxytryptamine receptor agonists and antagonists on food and fluid intake in the rat. Birmingham: University of Birmingham, 1989.

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9

Organon Round Table Conference (3rd 1992 Paris, France). GnRH, GnRH analogs, gonadotropins, and gonadal peptides: The proceedings of the third Organon Round Table Conference, Paris, 1992. London: Parthenon Pub. Group, 1993.

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10

Angerer, Erwin von. The estrogen receptor as a target for rational drug design. New York: Springer, 1995.

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11

Angerer, Erwin von. The estrogen receptor as a target for rational drug design. New York: Springer, 1995.

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12

Rusk, Ilene Naomi. The effects of selective dopamine D [inferior] 1 and D [inferior] 2 agonists and antagonists on feeding and associated behaviour. Birmingham: University of Birmingham, 1989.

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13

Foreman, John C. Textbook of receptor pharmacology. 3rd ed. Boca Raton: CRC Press, 2011.

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14

Feighner, John Preston. Selective serotonin re-uptake inhibitors: Advances in basic research and clinical practice. 2nd ed. Chichester: Wiley, 1996.

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15

F, Boyer W., ed. Selective serotonin re-uptake inhibitors: The clinical use of citalopram, fluoxetine, fluvoxamine, paroxetine, and sertraline. Chichester: Wiley, 1991.

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16

J, Colatsky Thomas, ed. Potassium channels: Basic function and therapeutic aspects : proceedings of the 29th Annual A.N. Richards Symposium held at Valley Forge, Pennsylvania, May 16-17, 1988. New York: Wiley-Liss, 1990.

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17

Evans, susan Elizabeth Gilbert. Flibanserin, a mixed 5-HT1A agonist/5-HT2A antagonist, decreases palatable food intake in operant and non-operant feeding paradigms in rats. Ottawa: National Library of Canada, 2000.

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18

Z, Langer S., ed. Serotonin receptor subtypes: Pharmacological significance and clinical implications. Basel: Karger, 1992.

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19

Baldi, Elisabetta, and Corrado Bucherelli. Neuroscience. Florence: Firenze University Press, 2017. http://dx.doi.org/10.36253/978-88-6453-638-5.

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This bibliographic material is patrimony of our Laboratory of the Behavior Physiology. This research unit originated in 1972 by will of Aldo Giachetti (until 1990) and with the beginning of the activity of Corrado Bucherelli. In the early 1980s, with Carlo Ambrogi Lorenzini (until 2004), the cataloging became more capillary and systematic, to continue to this day. All the researchers who worked in our laboratory contributed to this collection (Giovanna Tassoni 1986-2000, Benedetto Sacchetti 1996-2002 and Elisabetta Baldi from 1991). The study of learning, memory and behavior requires to follow a broad spectrum of neuroscience topics, ranging from neuronal biochemistry to neuropsychology. The Authors’ idea of publishing this collection comes from believing that a such website, though not exhaustive, might be a useful and targeted tool for the selection of bibliographic material in the field of behavioral neuroscience. The bibliographic references present at the publication (29500), accompanied by a brief comment highlighting the contents, are organized in relation to the topics (represented by the 99 themes) constituting the publication itself. The intersection of several references will point out the topics that represent them simultaneously. Concerning neurotransmitters and neuromodulators, references to agonists, antagonists or molecules interfering with the activity of these synapses have been inserted in the pages of the implicated neurotransmitter (e.g. acetylcholine). The pages including topics that could have been dealt with separately (e.g. active and passive avoidance) are introduced by a short explanatory note. The comment of each publication highlights the animal species used. Each comment is intended to indicate the content rather than the experimental results of paper. This choice comes from wanting to provide the reader with a more objective and less speculative comment.
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20

Essai n° 458: Essai d'activation transcriptionnelle faisant intervenir le récepteur des androgènes humain transfecté de façon stable pour la détection de l'activité androgénique agoniste et antagoniste des produits chimiques. OECD, 2020. http://dx.doi.org/10.1787/9789264264373-fr.

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21

Lambert, David G. Mechanisms and determinants of anaesthetic drug action. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0013.

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This chapter is broken into two main sections: a general description of the principles of ligand receptor interaction and a discussion of the main groups of ‘targets’; and explanation of some common pharmacological interactions in anaesthesia, critical care, and pain management. Agonists bind to and activate receptors while antagonists bind to receptors and block the effects of agonists. Antagonists can be competitive (most common) or non-competitive/irreversible. The main classes of drug target are enzymes, carriers, ion channels, and receptors with examples of anaesthetic relevance interacting with all classes. There are many examples in anaesthesia where multiple interacting drugs are co-administered—polypharmacology. To give an example: neuromuscular blockade. Rocuronium is a non-depolarizing neuromuscular blocker acting as a competitive antagonist at the nicotinic acetylcholine receptor. Rocuronium competes with endogenous acetylcholine to shift the concentration–response curve for contraction to the right. The degree of contractility is less for a given concentration of acetylcholine (agonist) in the presence of rocuronium. Using the same principle, the rightward shift can be compensated by increasing the amount of acetylcholine (as long as the amount of rocuronium presented to the receptor as an antagonist remains unchanged, its action can be overcome by increased agonist). Acetylcholine at the effect site is increased by acetylcholinesterase inhibition with neostigmine. One of the side-effects of neostigmine is that it acts as an indirect parasympathomimetic. In the cardiovascular system this would lead to muscarinic receptor-mediated bradycardia; these effects are routinely reversed by the competitive muscarinic antagonist glycopyrrolate.
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22

Martin, Sarter, Nutt David J. 1951-, and Lister Richard G, eds. Benzodiazepine receptor inverse agonists. New York: Wiley-Liss, 1995.

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23

Opioid Agonists, Antagonists and Mixed Narcotic Analgesics. Springer Verlag, 1987.

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24

Morocco, Martin Terence. Some electroanalytical studies of dopamine agonists and antagonists. 1992.

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25

Gevirtz, Clifford M., Elizabeth Frost, and Alan D. Kaye. Ultrarapid Opiate Detoxification. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190495756.003.0035.

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Extended opioid use results in physical dependence and neural adaptation. Opioid dependence treatment can be carried out with opioid receptor agonists, partial agonists, and antagonists. Conventional treatments have low success rates. Methadone and buprenorphine treatments involve substituting long-duration agonists for the opiate of abuse, essentially substituting one opiate for another. Rapid opiate detoxification is a 3-day process involving large amounts of an opiate antagonist. Both treatments have associated problems. Ultrarapid opiate detoxification (UROD) anesthetizes a patient and precipitates withdrawal while unconscious. It shortens withdrawal, avoiding much of the subjective withdrawal discomfort. Clonidine is critical to reduce catecholamine levels and mitigate central nervous system hyperarousal in acute withdrawal. A UROD advantage is that the withdrawal period is markedly shortened to 8 hours or less versus up to several months for conventional treatments. The patient is anesthetized during the acute withdrawal period and does not experience the unpleasant consequences of acute detoxification.
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26

Berend, Olivier, Wijngaarden I. van, and Soudijn W, eds. Serotonin receptors and their ligands. Amstersam: Elsevier, 1997.

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27

Ren, Ke, and Ronald Dubner. The first crystal structure of an ionotropic glutamate receptor ligand-binding core. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0032.

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The known functional ionotropic glutamate receptors (iGluRs) are composed of three major subtypes: AMPA, NMDA, and kainate. In 1998, in the landmark paper discussed in this chapter, Armstrong et al. provided the first crystal structure of an iGluR-subunit ligand-binding core, the S1S2 region of the rat GluA2 ‘flop’ isoform. They solved its structure with X-ray crystallography from selenomethonine crystals. They also identified residues involved in kainate binding, analysed allosteric sites that regulate affinity and specificity of the agonist, and mapped potential subunit–subunit interaction sites. They also proposed that binding of different agonists may result in variable degrees of domain closure. This work has profound impact on the field and it has been importantly cited. Subsequently, numerous high-resolution crystal structures of ligand-binding domains of iGluRs in complex with ligands, both agonists and antagonists, have been solved.
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28

Povl, Krogsgaard-Larsen, and Hansen J. J. 1944-, eds. Excitatory amino acid receptors: Design of agonists and antagonists. New York: E. Horwood, 1992.

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29

1946-, Höffken K., ed. Peptides in oncology I: LH-RH agonists and antagonists. Berlin: Springer-Verlag, 1992.

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30

Hoffmeister, F., A. Fleckenstein, C. Van Breemen, and R. Groß. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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31

Feng, Alexander J., George C. Chang Chien, and Alan D. Kaye. NMDA Receptor Antagonists, Gabapentinoids, Alpha-2 Agonists, and Dexamethasone. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457006.003.0002.

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Surgical pain is a major obstacle in the recovery of patients. Effective pain management is of upmost importance to optimize a patient’s recovery, decrease medical complications, and increase patient satisfaction. Traditional pain management with opioids and nonsteroidal anti-inflammatory drugs have significant side effect profiles leading to medical complications or insufficient pain management from reluctance of use. Adjuvant analgesic can provide improved pain management with significantly less side effect profile. In addition, the clinician can, with synergistic effects of adjuvant medications, lower the total dosages used, thus lessening the likelihood of the side effects that occur when medications are used alone at a higher dosage. This chapter presents several adjuvant analgesics—NMDA receptor antagonists, gabapentinoids, alpha-2 agonists, and dexamethasone—and evidence for their use. Ultimately, through the use of traditional pain management options along with adjuvant analgesics, the effectiveness of acute pain management can be increased while adverse outcomes are reduced and functional recovery and quality of life improved.
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32

Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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33

1928-, Rech Richard H., Gudelsky Gary A, and American College of Neuropsychopharmacology. Meeting, eds. 5-HT agonists as psychoactive drugs. Ann Arbor, MI: NPP Books, 1988.

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34

H, Bönisch, Trendelenburg U. 1922-, and Weiner Norman 1928-, eds. Catecholamines. Berlin: Springer-Verlag, 1988.

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35

(Editor), A. Fleckenstein, C. Van Breemen (Editor), R. Groß (Editor), and F. Hoffmeister (Editor), eds. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists (Bayer-Symposium). Springer, 1986.

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36

Rodgers, R. J. 5-Ht1a Agonists, 5-Ht3 Antagonists and Benzodiazepines: Their Comparative Behavioural Pharmacology. John Wiley & Sons, 1991.

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37

Fleckenstein, A., C. Van Breemen, and R. Grob. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists (Bayer-Symposium//(Proceedings)). Springer, 1985.

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38

J, Rodger R., and Cooper S. J, eds. 5-HT1A agonists, 5-HT3 antagonists and benzodiazepines: Their comparative behavioural pharmacology. Chichester, West Sussex, Englad: Wiley, 1991.

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39

Opioid Agonists, Antagonists and Mixed Narcotic Analgesics: Theoretical Background and Considerations for Practical Use. Springer, 2012.

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40

Opioid Agonists, Antagonists and Mixed Narcotic Analgesics: Theoretical Background and Considerations for Practical Use. Springer, 1987.

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41

Hoffken, K. Peptides in Oncology I: Lh-Rh Agonists and Antagonists (Recent Results in Cancer Research). Springer-Verlag Telos, 1992.

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42

Hoffken, K. Peptides in Oncology I: LH-Rh Agonists and Antagonists (Recent Results in Cancer Research). Springer, 1992.

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43

KROSGARD. Excitatory Aminoacid Receptors (Ellis Horwood Series in Pharmaceutical Technology Incorporating Pharmacological). Taylor & Francis, 1992.

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44

1929-, Denborough Michael, ed. The Role of calcium in drug action. Oxford: Pergamon Press, 1987.

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45

A, Denborough M., ed. The Roleof calcium in drug action. Oxford: Pergamon, 1987.

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46

KROSGARD. Excitatory Aminoacid Receptors (Ellis Horwood Series in Pharmaceutical Technology Incorporating Pharmacological). Taylor & Francis, 1992.

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47

Sampson, Brett G., and Andrew D. Bersten. Therapeutic approach to bronchospasm and asthma. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0111.

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The optimal management of bronchospasm and acute asthma is reliant upon confirmation of the diagnosis of asthma, detection of life-threatening complications, recognition of β‎2 agonist toxicity, and exclusion of important asthma mimics (such as vocal cord dysfunction and left ventricular failure). β‎2 agonists, anticholinergics, and corticosteroids are the mainstay of treatment. β‎2 agonists should be preferentially administered by metered dose inhaler via a spacer, and corticosteroids by the oral route, reserving nebulized (and intravenous) salbutamol, as well as intravenous hydrocortisone, for situations when these routes are not possible. A single intravenous dose of magnesium may be of benefit in severe asthma, but repeat dosing is likely to cause serious side effects. Parenteral administration of adrenaline may prevent the need for intubation in the patient in extremis. Aminophylline has an unfavourable side effect profile and has not been shown to offer additional benefit in adults. However, it does have a role in paediatric asthma. Unproven medical therapies with potential benefit include ketamine, heliox, inhalational anaesthetics, and leukotriene antagonists. The need for ventilatory support is usually preceded by worsening dynamic hyperinflation, exhaustion, hypoxia, reduced conscious state, or a combination of these. While non-invasive ventilation may have a temporizing role to allow time for response to medical therapy, there is insufficient evidence for its use, and should not delay invasive ventilation. If invasive ventilation is indicated, a strategy of hypoventilation and permissive hypercapnoea, minimizes barotrauma and dynamic hyperinflation. Extracorporeal support may have a role as a rescue therapy.
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48

Mozzarelli, Andrea, and Robert S. Phillips, eds. Enzymes Regulating the Homeostasis of Agonists and Antagonists of the N-Methyl D-Aspartate Receptors. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88963-062-2.

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49

1949-, Bonucelli U., and Rabey J. M. 1945-, eds. Old and new dopamine agonists in Parkinson's disease. Wien: Springer-Verlag, 1995.

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50

Frenier, Susan L. Urethral pressure response to alpha adrenergic agonist and antagonist drugs in the normal male cat. 1990.

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