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Journal articles on the topic 'Aetiology'

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1

King, Hilary. "1 Aetiology." Baillière's Clinical Endocrinology and Metabolism 2, no. 2 (May 1988): 291–305. http://dx.doi.org/10.1016/s0950-351x(88)80033-8.

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2

TARIQ, MOHAMMAD, RABIA BASRI, NAJI ULLAH KHAN, and Said Amin. "AETIOLOGY OF PANCYTOPENIA." Professional Medical Journal 17, no. 02 (June 10, 2010): 252–56. http://dx.doi.org/10.29309/tpmj/2010.17.02.2371.

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Background: Pancytopenia is a reduction in the number of each type of peripheral blood cell. Therefore the role of bone marrow examintion in diagnosis of pancytopenia is important to know etiology of pancytopenia. The objective of the study was to know the aetiology of pancytopenia. Methods: This descriptive (Cross sectional) study was carried out in Khyber teaching hospital. Fifty patients with pancytopenia were included in the study from 1st January 2008 to 30th October 2008. Full blood counts, bone marrow examinations and trephine biopsies were performed according to standard methods. Statistical packages for social science (SPSS.11) was used to analyze data. Results: Out of 50 patients, 36% were of aplastic anaemia, 16% megaloblastic anaemia, 14% myelodysplastic syndrome and 12% acute lymphoblastic leukemia (ALL), Hypersplenism in 10%, 4% non Hodgkin lymphoma (NHL) and 4% multiple myeloma, 2% each of acute myeloblastic leukemia and chronic myelocytic leukemia. All of these disorders were common in male as compared to female. Conclusions: Aplastic anaemia was the commonest cause of pancytopenia followed by megaloblastic anemia and myelodysplastic syndrome in our study.
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3

Herrera-Pérez, Mario, David González-Martín, Mercedes Vallejo-Márquez, Alexandre L. Godoy-Santos, Victor Valderrabano, and Sergio Tejero. "Ankle Osteoarthritis Aetiology." Journal of Clinical Medicine 10, no. 19 (September 29, 2021): 4489. http://dx.doi.org/10.3390/jcm10194489.

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Ankle osteoarthritis affects 1% of the population and, unlike gonarthrosis or coxarthrosis, is secondary to previous trauma in more than 75% of cases. Another peculiarity of this disease is that it affects a younger and active population, with socio-occupational implications. Mechanical factors, such as incongruity, instability, malalignment, and impacts, which increase stress on isolated areas of the ankle cartilage, have been clearly associated with the development of osteoarthritis. However, we cannot ignore the importance of pro-inflammatory mediators present from the moment of fracture as triggers of the cascade that eventually causes chondrocyte cell death, ultimately responsible for ankle osteoarthritis.
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4

Wright, Ian, and Peter Woodruff. "Aetiology of Schizophrenia." CNS Drugs 3, no. 2 (February 1995): 126–44. http://dx.doi.org/10.2165/00023210-199503020-00005.

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5

Jebb, S. A. "Aetiology of obesity." British Medical Bulletin 53, no. 2 (January 1, 1997): 264–85. http://dx.doi.org/10.1093/oxfordjournals.bmb.a011613.

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6

KRAEMER, S. "Aetiology of asthma." Archives of Disease in Childhood 77, no. 2 (August 1, 1997): 183. http://dx.doi.org/10.1136/adc.77.2.183n.

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7

Allan, A. "Aetiology and epidemiology." Current Opinion in Gastroenterology 1, no. 3 (May 1985): 461–67. http://dx.doi.org/10.1097/00001574-198505000-00013.

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8

Deweerdt, Sarah. "Aetiology: Crucial clues." Nature 513, no. 7517 (September 2014): S12—S13. http://dx.doi.org/10.1038/513s12a.

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9

Crow, Timothy J. "Aetiology of schizophrenia." Current Opinion in Psychiatry 7, no. 1 (January 1994): 39–42. http://dx.doi.org/10.1097/00001504-199401000-00011.

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10

Ando, Romeo, Flora Goloncser, and Beata Sperlagh. "Aetiology of depression." International Clinical Psychopharmacology 26 (September 2011): e19-e20. http://dx.doi.org/10.1097/01.yic.0000405654.86415.67.

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11

Brown, Carolyn. "Aetiology: Neighbourhood watch." Nature 540, no. 7631 (November 30, 2016): S4—S6. http://dx.doi.org/10.1038/540s4a.

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12

Mclaren, DonaldS. "AETIOLOGY OF KWASHIORKOR." Lancet 325, no. 8419 (January 1985): 55. http://dx.doi.org/10.1016/s0140-6736(85)91010-4.

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13

Wormsley, K. G. "Aetiology of ulcers." Baillière's Clinical Gastroenterology 2, no. 3 (July 1988): 555–71. http://dx.doi.org/10.1016/s0950-3528(88)80005-8.

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14

Degitz, K., M. Steidl, P. Thomas, G. Plewig, and M. Volkenandt. "Aetiology of tuberculids." Lancet 341, no. 8839 (January 1993): 239–40. http://dx.doi.org/10.1016/0140-6736(93)90101-l.

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15

Fidler, Helen, Graham Rook, NormanMci Johnson, and Clare O'Connor. "Aetiology of tuberculids." Lancet 341, no. 8844 (February 1993): 565–66. http://dx.doi.org/10.1016/0140-6736(93)90335-e.

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16

Duesberg, Peter. "Aetiology of AIDS." Lancet 341, no. 8859 (June 1993): 1544. http://dx.doi.org/10.1016/0140-6736(93)90691-9.

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17

Schechter, MartinT, KevinJ P. Craib, JulioS G. Montaner, ThinhN Lee, MichaelV O'Shaughnessy, KarenA Gelmon, MichaelS Ascher, et al. "Aetiology of AIDS." Lancet 341, no. 8854 (May 1993): 1222–24. http://dx.doi.org/10.1016/0140-6736(93)91060-y.

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18

John Henderson, A. "Aetiology of asthma." Paediatrics and Child Health 23, no. 7 (July 2013): 287–90. http://dx.doi.org/10.1016/j.paed.2012.11.009.

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19

Papp, G. K., Z. Kopa, F. Szabó, and E. Erdei. "Aetiology of haemospermia." Andrologia 35, no. 5 (October 2003): 317–20. http://dx.doi.org/10.1111/j.1439-0272.2003.tb00865.x.

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20

Phillips, C. I. "Aetiology of myopia." British Journal of Ophthalmology 74, no. 1 (January 1, 1990): 47–48. http://dx.doi.org/10.1136/bjo.74.1.47.

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21

Spolveri, Stefano. "Aetiology of intracerebral haemorrage." Reviews in Health Care 2, no. 1S (June 10, 2011): 19. http://dx.doi.org/10.7175/rhc.4121s19-25.

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Spontaneous non traumatic intracerebral haemorrhage (ICH) is usually caused by many different interacting factors, such as the use of alcohol or fibrinolitic drugs, congenital aneurysm, brain tumors, and blood dyscrasia. Age and hypertension-related small vessel diseases, and cerebral amyloid angiopathy are the most common forms of vascular damage which can lead to ICH. Furthermore, a group of inherited cerebral small vessel diseases linked to ICH have been reported recently and the number of these forms is increasing. The presence of leukoaraiosis, lacunar infarcts and microbleeds has been suggested to indicate a higher risk for cerebral hemorrhage. In recent years, MRI and neuroimaging techniques contributed to the understanding and the diagnosis of this disease.
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22

Spolveri, Stefano. "Aetiology of intracerebral haemorrage." Reviews in Health Care 2, no. 1S (June 10, 2011): 19–25. http://dx.doi.org/10.7175/rhc.v2i1s.41.

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Spontaneous non traumatic intracerebral haemorrhage (ICH) is usually caused by many different interacting factors, such as the use of alcohol or fibrinolitic drugs, congenital aneurysm, brain tumors, and blood dyscrasia. Age and hypertension-related small vessel diseases, and cerebral amyloid angiopathy are the most common forms of vascular damage which can lead to ICH. Furthermore, a group of inherited cerebral small vessel diseases linked to ICH have been reported recently and the number of these forms is increasing. The presence of leukoaraiosis, lacunar infarcts and microbleeds has been suggested to indicate a higher risk for cerebral hemorrhage. In recent years, MRI and neuroimaging techniques contributed to the understanding and the diagnosis of this disease.
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23

McMullin, Mary Frances, and Lesley Ann Anderson. "Aetiology of Myeloproliferative Neoplasms." Cancers 12, no. 7 (July 6, 2020): 1810. http://dx.doi.org/10.3390/cancers12071810.

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Myeloproliferative neoplasms (MPNs) have estimated annual incidence rates for polycythemia vera (PV), essential thrombocythemia (ET), and primary myelofibrosis of 0.84, 1.03, and 0.47 per 100,000. Prevalence is much higher, particularly for PV and ET, as mortality rates are relatively low. Patients are often concerned about why they developed an MPN and epidemiological studies enable the identification of potential causative factors. Previous work in small heterogeneous studies has identified a variety of risk factors associated with MPNs including family history of MPN, autoimmune conditions, some occupational exposures, and blood donation. At a population level, germline predisposition factors in various populations have been associated with MPNs. The pilot MOSAICC (Myeloproliferative Neoplasm: An In-depth Case-Control) study is one of the largest epidemiological studies in MPN ever carried out to date. It demonstrated the most effective methods for carrying out a significant epidemiological study in this patient group including the best way of recruiting controls, as well as how to evaluate occupational and lifestyle exposures, evaluate symptoms, and collect biological samples. Significant results linked to MPNs in the pilot study of 106 patients included smoking, obesity, and childhood socioeconomic status. The methodology is now in place for a much larger ongoing MOSAICC study which should provide further insight into the potential causes of MPNs.
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24

Safitri, Jehan. "Aetiology of pedophile Sufferers." European Journal of Social & Behavioural Sciences 9, no. 2 (April 30, 2014): 1417–22. http://dx.doi.org/10.15405/ejsbs.126.

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25

CULLINAN, P., and A. J. NEWMAN TAYLOR. "Aetiology of occupational asthma." Clinical & Experimental Allergy 27 (May 1997): 41–46. http://dx.doi.org/10.1111/j.1365-2222.1997.tb01825.x.

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26

Fielder, Alistair R. "Retinopathy of Prematurity: Aetiology." AVMA Medical & Legal Journal 3, no. 2 (March 1997): 47–51. http://dx.doi.org/10.1177/135626229700300204.

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27

de la Casa Almeida, M., C. Suarez Serrano, J. Rebollo Roldán, and J. J. Jiménez Rejano. "Cellulite’s aetiology: a review." Journal of the European Academy of Dermatology and Venereology 27, no. 3 (July 3, 2012): 273–78. http://dx.doi.org/10.1111/j.1468-3083.2012.04622.x.

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28

Irisarri, C. "Aetiology of Kienböck's Disease." Handchirurgie · Mikrochirurgie · Plastische Chirurgie 42, no. 03 (June 2010): 157–61. http://dx.doi.org/10.1055/s-0030-1253394.

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29

Bannon, M. "Aetiology of multiple sclerosis." QJM 104, no. 5 (April 19, 2011): 381–82. http://dx.doi.org/10.1093/qjmed/hcr064.

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30

Sidebottom, Andrew J., Anne Begley, Paul May, and David Richardson. "Aetiology of nasopharyngeal glioma." International Journal of Oral and Maxillofacial Surgery 29, no. 3 (June 2000): 210–11. http://dx.doi.org/10.1034/j.1399-0020.2000.290311.x.

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31

AMIN, MAGDY, and A. G. D. MARAN. "THE AETIOLOGY OF LARYNGOCOELE." Clinical Otolaryngology 13, no. 4 (August 1988): 267–72. http://dx.doi.org/10.1111/j.1365-2273.1988.tb01130.x.

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32

GRONERT, G. A., J. MOTT, and J. LEE. "AETIOLOGY OF MALIGNANT HYPERTHERMIA." British Journal of Anaesthesia 60, no. 3 (February 1988): 253–67. http://dx.doi.org/10.1093/bja/60.3.253.

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33

VAN HOOREBEECK, BART. "Prospects for Reconstructing Aetiology." Theoretical Criminology 1, no. 4 (November 1997): 501–18. http://dx.doi.org/10.1177/1362480697001004005.

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34

Chionh, E.-H., V.-H. Pham, R. A. Cooke, and I. R. Gough. "AETIOLOGY OF BRANCHIAL CYSTS." ANZ Journal of Surgery 59, no. 12 (December 1989): 949–51. http://dx.doi.org/10.1111/j.1445-2197.1989.tb07637.x.

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35

Murrell, George A. C., and John T. Hueston. "AETIOLOGY OF DUPUYTREN'S CONTRACTURE." ANZ Journal of Surgery 60, no. 4 (April 1990): 247–52. http://dx.doi.org/10.1111/j.1445-2197.1990.tb07362.x.

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36

Heaton, K. W. "Aetiology of acute appendicitis." BMJ 294, no. 6588 (June 27, 1987): 1632–33. http://dx.doi.org/10.1136/bmj.294.6588.1632.

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37

Bannister, B. A., S. Edwards, and G. Ibata. "Aetiology of Kawasaki disease." Archives of Disease in Childhood 64, no. 3 (March 1, 1989): 397–98. http://dx.doi.org/10.1136/adc.64.3.397.

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38

Hopkins, N. "Aetiology of enzootic haematuria." Veterinary Record 118, no. 26 (June 28, 1986): 715–17. http://dx.doi.org/10.1136/vr.118.26.715.

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39

Jaju, P. P. "Aetiology of bifid condyle." Dentomaxillofacial Radiology 39, no. 4 (April 15, 2010): 256. http://dx.doi.org/10.1259/dmfr/32282722.

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40

Adu, D., and J. Stewart Cameron. "Aetiology of Membranous Nephropathy." Nephrology Dialysis Transplantation 4, no. 8 (1989): 757–58. http://dx.doi.org/10.1093/ndt/4.8.757.

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41

CLIFFORD, MICHAEL N., and RONALD WALKER. "The aetiology of scombrotoxicosis." International Journal of Food Science & Technology 27, no. 6 (July 1, 2007): 721–24. http://dx.doi.org/10.1111/j.1365-2621.1992.tb01244.x.

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42

Paulley, J. W. "Aetiology of multiple sclerosis." QJM 87, no. 12 (December 1, 1994): 769. http://dx.doi.org/10.1093/oxfordjournals.qjmed.a068896.

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43

Bhate, Soni, and Steve Wilkinson. "Aetiology of learning disability." Psychiatry 5, no. 9 (September 2006): 298–301. http://dx.doi.org/10.1053/j.mppsy.2006.08.001.

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44

Gaskell, C. J. "Comparative aspects and aetiology." Journal of Small Animal Practice 28, no. 5 (May 1987): 404–6. http://dx.doi.org/10.1111/j.1748-5827.1987.tb01433.x.

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45

Gravitz, Lauren. "Aetiology: The X factor." Nature 484, no. 7393 (April 2012): S2. http://dx.doi.org/10.1038/nature11103.

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46

Curry, Joseph I., Patricia McKinney, James G. Thornton, and Mark D. Stringer. "The aetiology of gastroschisis." BJOG: An International Journal of Obstetrics and Gynaecology 107, no. 11 (November 2000): 1339–46. http://dx.doi.org/10.1111/j.1471-0528.2000.tb11645.x.

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47

Morgan, G. J., F. E. Davies, and M. Linet. "Myeloma aetiology and epidemiology." Biomedicine & Pharmacotherapy 56, no. 5 (July 2002): 223–34. http://dx.doi.org/10.1016/s0753-3322(02)00194-4.

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48

Woolridge, Michael W. "Aetiology of sore nipples." Midwifery 2, no. 4 (December 1986): 172–76. http://dx.doi.org/10.1016/s0266-6138(86)80042-0.

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49

WATSON, H. K., and P. M. GUIDERA. "Aetiology of Kienböck’s Disease." Journal of Hand Surgery 22, no. 1 (February 1997): 5–7. http://dx.doi.org/10.1016/s0266-7681(97)80004-6.

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50

Manchande, Jean-Pierre, Tony Mets, Amy Klion, ValdaM Craddock, D. I. Thurnham, Nubia Munoz, J. Wahrendorf, and M. Crespi. "AETIOLOGY OF OESOPHAGEAL CANCER." Lancet 329, no. 8548 (June 1987): 1499–500. http://dx.doi.org/10.1016/s0140-6736(87)92259-8.

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