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1

Aetiology of urbanisation in India. Allahabad: Govind Ballabh Pant Social Science Institute, 1985.

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2

Lammie, George Alexander. The aetiology of the dental diseases. Bubenhall [Eng.]: G.A. Lammie, 1992.

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3

Ham, R. Limb amputation ; from aetiology to rehabilitation. London: Chapman and Hall, 1991.

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4

Mechelen, Willem Van. Aetiology and prevention of running injuries. Amsterdam: Drukkerij A1/A2, 1992.

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5

Miller, Anthony B., ed. Diet and the Aetiology of Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74376-4.

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6

Malone, P. Colm, and Paul S. Agutter. The Aetiology of Deep Venous Thrombosis. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6650-4.

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7

B, Miller A., ed. Diet and the aetiology of cancer. Berlin: Springer-Verlag, 1989.

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8

Shortt, N. C. Adult thought: Aetiology and stimulation of change. [S.l: The author], 1991.

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9

Obwegeser, Hugo L. Mandibular growth anomalies: Terminology, aetiology, diagnosis, treatment. Berlin: Springer, 2001.

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10

J, Morison M., ed. Pressure sore blueprint: Aetiology, prevention and management. [Hounslow]: Squibb, 1993.

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11

Cleft lip and palate: Epidemiology, aetiology, and treatment. Basel: Karger, 2012.

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12

M, Hutson John, ed. Normal testicular descent and the aetiology of cryptorchidism. Berlin: Springer, 1996.

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13

Hutson, John M., Masaru Terada, Baiyun Zhou, and Martyn P. L. Williams. Normal Testicular Descent and the Aetiology of Cryptorchidism. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-61026-4.

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14

Myers, K. Sara. Ovid's causes: Cosmogony and aetiology in the Metamorphoses. Ann Arbor: University of Michigan Press, 1994.

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15

Chronic pain epidemiology: From aetiology to public health. Oxford: Oxford University Press, 2010.

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16

The foot: Pathology, aetiology, semiology, clinical investigation and therapy. Berlin: Springer-Verlag, 1986.

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17

Sleep, health, and society: From aetiology to public health. Oxford: Oxford University Press, 2010.

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18

D, Parkes J., ed. MPTP and the aetiology of Parkinson's disease: Clinical implications. Wien: Springer-Verlag, 1986.

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19

McMurran, Mary. Young offenders and drinking: Aetiology, assessment and self- management. Birmingham: Universityof Birmingham, 1991.

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20

Das, Eswara. Synopsis of homoeopathic aetiology: A complete work on causation. 2nd ed. New Delhi: B. Jain Publishers Pvt. Ltd., 1988.

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21

Deacon, Elizabeth Mary. The immunopathology and possible viral aetiology of Sjogren's syndrome. Birmingham: University of Birmingham, 1992.

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22

Colloquium Hepatologicum. (6th 1986 Halle an der Saale, Germany). New aspects in aetiology and pathogenesis of liver diseases. Halle (Saale): Martin-Luther-Universität Halle-Wittenberg, 1987.

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23

Newton, Valerie E. Aetiology of bilateral sensori-neural hearing loss in young children. Ashford, Kent: Headley Brothers, 1985.

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24

Sibbons, Paul David. Investigations into the aetiology and pathogenesis of neonatal necrotising enterocolitis. Uxbridge: BrunelUniversity, 1990.

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25

Aetiology of bilateral sensori-neural hearing loss in young children. Ashford, Kent: Invicta Press, 1985.

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26

Birdsey, Tracy Jane. Aetiology of disturbed calcium homeostasis in the offspring of diabetic mothers. Manchester: University of Manchester, 1995.

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27

Brennan, Paul James. An investigation of reproductive factors in the aetiology of rheumatoid arthritis. Manchester: University of Manchester, 1995.

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28

Choi, Alice. The role of occupational factors in brain cancer aetiology: An epidemiological study. Birmingham: University of Birmingham, 1995.

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29

Harper, Catherine Louise. The aetiology of the human corneal endothelium in vivo and in vitro. Manchester: University of Manchester, 1995.

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30

Malone, P. Colm. The aetiology of deep venous thrombosis: A critical, historical and epistemological survey. Dordrecht: Springer, 2008.

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31

The aetiology of idiopathic scoliosis: A review of a century of research. Boca Raton, Fla: CRC Press, 1990.

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32

Leung, Newman Kwok-Cheung. Family interaction and cognitive content in the aetiology and treatment of eating disorders. Birmingham: University of Birmingham, 1999.

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33

Meanwell, Clive Arthur. Age and cervical cancer: An investigation of incidence, aetiology and prognosis in young women. Birmingham: University of Birmingham, 1987.

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34

Fromm, Annette. Vascular aetiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0004.

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Vascular aetiology of young ischaemic stroke covers a broad spectrum of causes. It includes the risk factor-mediated causes considered more common among the elderly on one hand, and a large number of rather rare disorders and conditions typical for younger ages on the other hand. This chapter is focused on atherosclerotic aetiology and comorbidity, small vessel disease and arterial dissection, which account for a majority of young ischaemic strokes worldwide, are treatable, and need to be considered as overall or contributing causes early during investigation. Specific and rare causes of young ischaemic stroke will be presented elsewhere.
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35

Turc, Guillaume, David Calvet, and Jean-Louis Mas. Cardiac aetiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0005.

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Cardiac aetiology accounts for approximately 20% of strokes in young adults. Although atrial fibrillation is a leading cause of stroke in the general population, it is uncommon in young adults. In such patients, more diverse causes of ischaemic stroke are observed, including valvular heart diseases, infective endocarditis, Libman–Sacks endocarditis, dilated cardiomyopathies, congenital heart diseases, myocardial infarction, and intracardiac tumours. Patent foramen ovale is commonly observed in young adults with ischaemic stroke, but this association may be incidental in a sizeable proportion of patients. Young adults who are the most likely to have a stroke-related patent foramen ovale are also those with the lowest recurrence risk.
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36

Parkes, Joanna E., Simon Rothwell, and Janine A. Lamb. Aetiology and pathogenesis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0003.

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The aetiology and pathogenesis of idiopathic inflammatory myopathies (IIM) is poorly understood; IIM are thought to result from exposure to environmental factors in genetically susceptible individuals. Both innate and adaptive immune responses are involved in IIM, and there is increasing evidence that non-inflammatory mechanisms play an important role in disease pathology. Several environmental risk factors, including infectious agents, ultraviolet radiation, cigarette smoking, and exposure to statins, have been implicated. Genetic studies have identified the major histocompatibility complex as the most strongly associated region, while recent large scale genome-wide studies have implicated genes that commonly regulate the adaptive immune response, which overlap with other seropositive autoimmune diseases. Integrating data across these various fields should facilitate refined models of disease pathogenesis.
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37

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff. Aetiology and epidemiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0001.

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Describes how cancer care is structured and explains rationale and components of multidisciplinary teams. This runs through cancer care from diagnosis to terminal care and has resulted in better quality and outcomes for patients
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38

Levy, David. History, epidemiology, and aetiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0001.

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Outline of the key landmarks in the history of Type 1 diabetes. Epidemiology, focusing on its rising incidence and the increasing prevalence of later-onset autoimmune diabetes. Genetics are briefly covered; the evidence for and against the impact of a variety of environmental factors thought to be important in aetiology are emphasized, especially in relation to prospective randomized clinical trials (RCT) in early Type 1 diabetes, aiming to delay the onset of autoimmunity in high-risk individuals or slow the decline in C-peptide levels shortly after clinical diagnosis. The balance-risk hypothesis, which allows for inclusion of protective and promoting factors, is introduced.
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39

Bargiotas, Theodoros. The Aetiology of Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198801900.003.0005.

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This chapter discusses the aetiology of depression. It begins with an overview of reasons why the aetiology of depression has been and still is difficult to define, including its heterogeneity and the wider philosophical and methodological challenges involved. It then considers life events and social determinants of depression as well as psychological factors relevant to depression, including childhood development and early experiences, parenting style, and personality and personality disorders. It also examines cognitive behavioural theories of depression, neuropsychology, and psychodynamic theories, along with the role of unconscious processes and ego psychology in the aetiology of depression. Finally, the chapter describes the biological determinants of depression, paying attention to genetics, neurochemical changes in the brain, and stress and thyroid hormones.
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40

Archer, Nick, and Nicky Manning. Aetiology of fetal heart disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0002.

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This chapter discusses the aetiology of fetal heart disease, including discussion on maternal factors, genetic factors, genetic causes, risk of associated non-cardiac anomalies, prevention of congenital heart disease, and the fetal origins of health and disease.
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41

Figueroa, Eleanor. Ascites: Aetiology, Symptoms and Treatment. Nova Science Publishers, Incorporated, 2016.

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42

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Venous thromboembolism: epidemiology and aetiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0087.

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43

(Editor), John Kingdom, Philip Baker (Editor), and M. Whittle (Preface), eds. Intrauterine Growth Restriction: Aetiology and Management. Springer, 2000.

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44

Archer, Nick, and Nicky Manning. Aetiology of structural congenital heart disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199230709.003.0002.

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Introduction 18Environmental factors 20Genetic factors 26Risk of associated non-cardiac anomalies 32Prevention of congential heart disease 34• The incidence of congenital heart disease (CHD) is approximately 8:1000 lives births but is higher in the prenatal population.• The aetiology of CHD has been considered to be multifactorial—a combination of environmental factors and a genetic predisposition but increasing numbers of specific genetic causes are being identified....
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45

Miller, Anthony B. Diet and the Aetiology of Cancer. Springer, 2012.

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46

Kingdom, John, Philip Baker, and M. Whittle. Intrauterine Growth Restriction: Aetiology and Management. Springer London, Limited, 2012.

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47

Miller, Anthony B. Diet and the Aetiology of Cancer. Springer London, Limited, 2012.

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48

Kingdom, John, Philip Baker, and M. Whittle. Intrauterine Growth Restriction: Aetiology and Management. Springer, 2011.

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49

Ham, R., and L. T. Cotton. Limb Amputation: From Aetiology to Rehabilitation. Springer, 2013.

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50

Agutter, Paul S., and P. Colm Malone. The Aetiology of Deep Venous Thrombosis. Springer, 2008.

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