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Relevant bibliographies by topics / Aetiology

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Academic literature on the topic 'Aetiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 February 2023

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Journal articles on the topic "Aetiology"

1

King, Hilary. "1 Aetiology." Baillière's Clinical Endocrinology and Metabolism 2, no. 2 (May 1988): 291–305. http://dx.doi.org/10.1016/s0950-351x(88)80033-8.

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TARIQ, MOHAMMAD, RABIA BASRI, NAJI ULLAH KHAN, and Said Amin. "AETIOLOGY OF PANCYTOPENIA." Professional Medical Journal 17, no. 02 (June 10, 2010): 252–56. http://dx.doi.org/10.29309/tpmj/2010.17.02.2371.

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Background: Pancytopenia is a reduction in the number of each type of peripheral blood cell. Therefore the role of bone marrow examintion in diagnosis of pancytopenia is important to know etiology of pancytopenia. The objective of the study was to know the aetiology of pancytopenia. Methods: This descriptive (Cross sectional) study was carried out in Khyber teaching hospital. Fifty patients with pancytopenia were included in the study from 1st January 2008 to 30th October 2008. Full blood counts, bone marrow examinations and trephine biopsies were performed according to standard methods. Statistical packages for social science (SPSS.11) was used to analyze data. Results: Out of 50 patients, 36% were of aplastic anaemia, 16% megaloblastic anaemia, 14% myelodysplastic syndrome and 12% acute lymphoblastic leukemia (ALL), Hypersplenism in 10%, 4% non Hodgkin lymphoma (NHL) and 4% multiple myeloma, 2% each of acute myeloblastic leukemia and chronic myelocytic leukemia. All of these disorders were common in male as compared to female. Conclusions: Aplastic anaemia was the commonest cause of pancytopenia followed by megaloblastic anemia and myelodysplastic syndrome in our study.

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Herrera-Pérez, Mario, David González-Martín, Mercedes Vallejo-Márquez, Alexandre L. Godoy-Santos, Victor Valderrabano, and Sergio Tejero. "Ankle Osteoarthritis Aetiology." Journal of Clinical Medicine 10, no. 19 (September 29, 2021): 4489. http://dx.doi.org/10.3390/jcm10194489.

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Ankle osteoarthritis affects 1% of the population and, unlike gonarthrosis or coxarthrosis, is secondary to previous trauma in more than 75% of cases. Another peculiarity of this disease is that it affects a younger and active population, with socio-occupational implications. Mechanical factors, such as incongruity, instability, malalignment, and impacts, which increase stress on isolated areas of the ankle cartilage, have been clearly associated with the development of osteoarthritis. However, we cannot ignore the importance of pro-inflammatory mediators present from the moment of fracture as triggers of the cascade that eventually causes chondrocyte cell death, ultimately responsible for ankle osteoarthritis.

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Wright, Ian, and Peter Woodruff. "Aetiology of Schizophrenia." CNS Drugs 3, no. 2 (February 1995): 126–44. http://dx.doi.org/10.2165/00023210-199503020-00005.

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Jebb, S. A. "Aetiology of obesity." British Medical Bulletin 53, no. 2 (January 1, 1997): 264–85. http://dx.doi.org/10.1093/oxfordjournals.bmb.a011613.

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KRAEMER, S. "Aetiology of asthma." Archives of Disease in Childhood 77, no. 2 (August 1, 1997): 183. http://dx.doi.org/10.1136/adc.77.2.183n.

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Allan, A. "Aetiology and epidemiology." Current Opinion in Gastroenterology 1, no. 3 (May 1985): 461–67. http://dx.doi.org/10.1097/00001574-198505000-00013.

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Deweerdt, Sarah. "Aetiology: Crucial clues." Nature 513, no. 7517 (September 2014): S12—S13. http://dx.doi.org/10.1038/513s12a.

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Crow, Timothy J. "Aetiology of schizophrenia." Current Opinion in Psychiatry 7, no. 1 (January 1994): 39–42. http://dx.doi.org/10.1097/00001504-199401000-00011.

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Ando, Romeo, Flora Goloncser, and Beata Sperlagh. "Aetiology of depression." International Clinical Psychopharmacology 26 (September 2011): e19-e20. http://dx.doi.org/10.1097/01.yic.0000405654.86415.67.

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Dissertations / Theses on the topic "Aetiology"

1

Bradlow, Anthony. "Clinical and laboratory studies into possible relationships between alcohol and musculoskeletal disorders, with emphasis on rheumatoid arthritis, primary osteoarthritis of the hip and Dupuytren's contracture." Thesis, University of Cape Town, 1985. http://hdl.handle.net/11427/25598.

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Fenn, Francis P. "An aetiology of construction disputes." Thesis, University of Manchester, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.488193.

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Rowell, S. L. "The aetiology of running injuries." Thesis, University of Brighton, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234742.

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Kvapilova, Alice. "The aetiology of surface reading pattern /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17813.pdf.

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Thomas, Geraint Emyr Rhys. "The patho-aetiology of hip osteoarthritis." Thesis, University of Oxford, 2014. https://ora.ox.ac.uk/objects/uuid:b4594f27-46ad-47d6-81f1-11ed934e1495.

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Osteoarthritis of the hip frequently occurs in the absence of osteoarthritis in other large joints, suggesting that local factors are important in its pathogenesis. Hip morphology has been recognised as a potential local biomechanical risk factor for the development of hip osteoarthritis. There are no adequate studies examining osteoarthritis development in the hip. Historical cohorts are either limited by a short follow up or by small numbers. This thesis explores the natural history of hip osteoarthritis in a large population cohort with particular attention to hip morphology as a predictor of osteoarthritis development. Software was developed which allows objective measurements of hip morphology in a reproducible manner. Hip morphology was then measured in a 1000 subject cohort. A detailed description of hip morphology is presented in this thesis, with interesting observations of wide variation and a bimodal distribution for alpha angle (a measure of cam-type femoroacetabular impingement). This is suggestive of a discrete pathological entity, which was associated with osteoarthritis in the cross-sectional analysis. No significant changes exist in terms of morphology during the course of the study and no significant relationship exists between age and hip morphology. Longitudinal analysis of hip morphology with radiographic osteoarthritis and total hip replacement revealed a significant association between cam-type femoroacetabular impingement and acetabular dysplasia with both outcome measures. Measurements of hip morphology were independently predictive of outcome when controlling for baseline age, BMI and joint space width, and significantly increased our ability to predict osteoarthritis and total hip replacement. Similar associations were seen when considering hip pain and symptomatic osteoarthritis as the outcome measures of interest. Pincer-type femoroacetabular impingement was not significantly associated with any of the outcome measures of interest and pain remains relatively poorly explained by both hip morphology and/or radiographic change. The understanding of hip morphology and its role in the natural history of osteoarthritis is significantly improved by this research. Further research is now required to determine whether these morphological abnormalities represent modifiable risk factors for osteoarthritis progression.

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Timmerman, Grietje Hermina. "Violent behaviour aetiology and treatment issues /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/77751.

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McOrist, S. "The aetiology of the proliferative enteropathies." Thesis, University of Edinburgh, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383022.

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The entezopathogenicity and antigens of Campylobacter-like organisms and Campylobacter app associated with the proliferative enteropathies were investigated. Two gnotobiotic pigs exposed orally to a filtered suspension of intestinal mucosa designated 284/86 from a naturally infected pig subsequently developed lesions of proliferative enteritis. Culture of the successful mucosal inoculum only revealed a moderate number of C. coli, however an apparently greater number of Campylobacter-like organisms was evident in smears of this inoculum. The pathogenesis of porcine proliferative enteritis was clearer from the results of this study. Ten days after infection, curved bacilli had colonised the ileal and large intestinal crypts. Attachment and entry of Campylobacter-like organisms into crypt enterocytes was also evident, with some proliferation of both bacteria within cells and of the enterocytes themselves. Twenty days after infection there was similar intracellular colonisation of bacteria and proliferative activity, although no luminal bacteria were evident. A moderate sub-acute inflammatory reaction was evident throughout. Conventional hamsters dosed with C. jejuni developed varying degrees of localised acute intestinal inflammation. Hamsters dosed with C. hyointestinalis or C. cola did not develop any lesions. Lesions of proliferative enteritis were detected in hamsters dosed with porcine tissue 284/86. Numerous intra-cytoplasmic Campylobacter-like organisms were detected within enterocytes in affected portions of intestine. Weanling hamsters this proved to be susceptible to the agent of porcine proliferative enteritis by cross-species transmission. Whole cell antigen preparations were made of various Camoylobacter sp. -Indirect immunofluorescence assays incorporating rabbit antisera to each Campylobacter sp gave specific endpoints for each antiserum of 1: 160 to 1: 320. Rabbit antisera prepared to Campylobacter-like organisms partly purified from proliferative enteritis mucosa, by a homogenisation and filtration technique, also gave specific reactions in this assay, up to 1: 610. Intracellular Campylobacter-like organisms were also compared in gel electrophoresis protein profiles and immunoblotting reactions to Campylobacter spp. The intracellular organisms tested had a distinctive protein profile dissimilar to the profiles of the known Campylobacter spp. In immunoblotting reactions, each of the Camoylobacter sp antisera reacted strongly with homologous antigens, but none reacted with Camcylobacter-like organisms prepared from lesions, except for a minor reaction seen with one serum. Similarly antisera to Campylobacter-like organisms showed a strong reaction to 25K to 27K components of homologous antigens, with only minor reactions to various other components of the cultivated Campylobacter app. Therefore it is likely that the intracellular Campylobacter-like organisms have a distinctive antigenic profile and that the 25 and 27K components are major antigenic components. Mouse monoclonal antibodies were produced that were apparently specific to the intracellular Camoylobacter-like organisms. Immunoblotting results showed that these antibodies only bound to a 251 to 27K outer membrane component present in the intracellular organisms. Reactions with this component could not be detected in assays with normal pig intestine, or Camoylobacter sp antigen. Restriction endonuclease digestion of Camoylobacter sp with Bgl II gave suitably resolved DNA fragments of between 2kb and 25kb. Patterns obtained with Bgl II digestion of Camoylobacter sp were dissimilar to those of Camoylobacter-like organisms, and each Camoylobacter sp had a characteristic distinct pattern. Digestion of DNA from porcine tissue samples with Bgl II produced a diffuse smear of fragmented DNA bands between 0.5 and19kb, with no recognizable "ladder" effect. The genome of the Carylobacter-like organisms within enterocytes in proliferative enteritis therefore is different to that of known Camoylobacter spp associated with the disease. This suggests that the differences in antigenic structures between these bacteria axe due to genetic differences. Only a limited number of strains were examined. Looking at the evidence provided by this study, the overall tenor of the results suggests that the intracellular organisms could be a separate, new species of Camp lobacter. If indeed the intracellular organisms are a single, new Cammylobacter PGS/ABST ecies, then a new name may be proposed, such as "C. intracellulare". Verifica nthis side Oni of the validity of such a proposal would require further DNA studies.

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Wilson, Gillian Anne. "The genetic aetiology of lymphoplasmacytic lymphoma." Thesis, University of Sheffield, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434569.

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Balachandran, Aswini Aparna. "Management and aetiology of overactive bladder." Thesis, University of Kent, 2018. https://kar.kent.ac.uk/67149/.

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The aetiology of overactive bladder (OAB) is poorly understood. Current treatment modalities are associated with low success rates and poor long-term compliance. The aim of this thesis was to explore the new and alternative treatment, mirabegron, to assess the efficacy of traditional treatment options such as cystodistension for women with OAB and to ascertain whether underlying baseline parameters (e.g. urodynamic studies, cystoscopic findings, bladder biopsy results) have any impact on treatment outcomes. The aetiology of OAB was also studied to understand the impact of bladder wall thickness and the bladder microbiome in OAB to improve our understanding of the possible mechanisms of OAB. In this thesis, I have reviewed the current literature on OAB, cystodistension and the role of infection in OAB. The work presented in this MD investigates the effectiveness of mirabegron and cystodistension as treatment options for OAB. All studies were conducted in a 'real-life' clinical setting to recreate the challenges in daily clinical practice. Mirabegron was found to be an effective treatment option for patients with OAB. However, the benefits were not striking with 70% of patient reporting improvement of their symptoms at 6 weeks. This was converted into only a minority of patients (36%) reporting their incontinence was "much better" or "very much better". More research should be targeted at identifying patient characteristics that are associated with a better outcome. It was found to be a suitable non-invasive alternative to Botulinum Toxin A (BTXA) in 59% of patients with refractory OAB. However, over two thirds of patients discontinued mirabegron therapy within 1 year. A significant number of patient who persevered with mirabegron were on combination therapy with an antimuscarinic at 1 year. Randomised controlled trial comparing cystodistension with cystoscopy demonstrated no benefit from cystodistension in the treatment of OAB. In this group, the presence of bladder trabeculation on cystoscopy was found to be associated with a direct effect on maximum detrusor muscle contraction with a significant increase in contraction compared to patients with an absence of trabeculation (42.71 cmH2O vs 31.41cmH2O, p = 0.01). The presence of trabeculation also affected the symptoms of OAB with a significant decrease in the Filling Scores of the ICIQ-FLUTS LF questionnaire (8.45 vs 9.58, p=0.04). There was no relationship between urodynamic findings and bladder biopsy on baseline symptomology and outcome of treatment. In this cohort, a feasibility study assessing bladder wall thickness (BWT) recorded a significant increase in BWT in OAB patients with detrusor overactivity (DO) (5.6mm vs 4.2mm, p=0.006). Though ultrasound is unable to replace urodynamic studies, it may be useful tool in understanding the aetiology, disease progression and prediction of treatment outcomes in OAB. The bladder microbiome demonstrated a significant difference between the patients with OAB and healthy controls. Proteus was found to be significantly more prevalent in OAB patients (p=0.01) whilst Lactobacillus was significantly more common in healthy controls (p=0.02). The work in this MD suggests there may be variety of sub-types of OAB with different underlying mechanisms of action that may explain the large variation in outcomes with different treatment modalities. Further research will need to be performed to further explore and confirm these findings.

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Santos, Ricardo Feliciano dos. "Aetiology and epidemiology of grapevine anthracnose." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/11/11135/tde-06042018-155130/.

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Grapevine anthracnose is an important disease, responsible for severe yield losses in humid regions around the world. This study aimed to: i: identify the causal agents of grapevine anthracnose in Brazil; ii: characterize Elsinoë ampelina isolates from Brazil and Australia by means of phylogenetic analyses, morphological features and pathogenicity tests; iii: develop an efficient method for conidial production of E. ampelina; iv: develop and validate a standard area diagram set (SADs) for assessing anthracnose severity on grapevine leaves; and v: study the temporal and spatial progression of anthracnose in a Brazilian vineyard. To identify the causal agents of the disease, leaves, stems and berries with anthracnose symptoms were collected from 38 vineyards in southern and southeastern Brazil and 39 E. ampelina and 13 Colletotrichum spp. isolates were obtained. For E. ampelina isolates, the internal transcribed spacer (ITS), histone H3 (HIS3) and elongation factor 1-α (TEF) sequences were analysed. HIS3 was the most informative region with 55 polymorphic sites. Seven Colletotrichum species were identified: C. siamense, C. gloeosporioides, C. fructicola, C. viniferum, C. nymphaeae, C. truncatum and C. cliviae. In pathogenicity tests, only E. ampelina isolates caused anthracnose symptoms on Vitis vinifera \'Moscato Giallo\' and Vitis labrusca \'Niagara Rosada\'. To characterize E. ampelina from Brazil and Australia, 35 isolates were analysed. ITS and TEF sequences of all isolates were monomorphic. The haplotype network generated from HIS3 dataset showed four distinct haplotypes. High genetic variability was observed in two Brazilian isolates, haplotype EA4, which may have lost the intron region during species evolution. Colonies showed variable coloration, wrinkled texture, absence of spores and slow growth. Brazilian isolates produced conidia larger than conidia from Australian isolates. To induce the conidial production, mycelial fragments were shake-incubated in rainwater and distilled water for 7 days. Isolates produced different concentrations of conidia and the conidial germination was more than 88.5%. In infectivity tests, conidia caused typical anthracnose symptoms on leaves. The SADs developed comprises six true colour diagrams with severity ranging from 1.1 to 27.4%. The use of the SADs improved the accuracy, precision, agreement and inter-rater reliability of the estimates conducted by 12 raters. The temporal and spatial dynamics of anthracnose was carried out in a \'Niagara Rosada\' vineyard in 2014 and 2015. The incidence of vines with diseased leaves, stems and berries and the severity disease on leaves were recorded. Anthracnose symptoms occurred rapidly after bud break and ontogenic resistance was observed for all organs assessed. The monomolecular model showed the best fit to the incidence progress. Temporal analyses suggest that the progress of anthracnose incidence and severity over time is governed mainly by the primary inoculum due to age-related resistance of the vine organs. Spatial analyses showed a predominantly random spatial pattern of diseased vines. In conclusion, this thesis presents a more in-depth understanding of the aetiology and epidemiology of an important grapevine disease.
Antracnose da videira é uma importante doença, responsável por severas perdas de produtividade em regiões húmidas em diversos locais do planeta. Este estudo objetivou: i: identificar o agente causal da antracnose da videira no Brasil; ii: caracterizar isolados de Elsinoë ampelina do Brasil e Austrália através de análises filogenéticas, morfologia e testes de patogenicidade; iii: desenvolver um método eficiente para produção de conídios de E. ampelina; iv: desenvolver e validar uma escala diagramática para avaliar a severidade de antracnose em folhas; e v: estudar o progresso temporal e espacial da antracnose em um vinhedo brasileiro. Para identificar os agentes causais da doença, folhas, ramos e bagas com sintomas de antracnose foram coletados em 38 vinhedos das regiões sul e sudeste do Brasil e 39 isolados de E. ampelina e 13 isolados de Colletotrichum spp. foram obtidos. Para isolados de E. ampelina, sequências de espaçador interno transcrito (ITS), histona H3 (HIS3) e fator de elongação 1-α (TEF) foram analisadas. HIS3 foi a região mais informativa com 55 sítios polimórficos. Foram identificadas sete espécies de Colletotrichum: C. siamense, C. gloeosporioides, C. fructicola, C. viniferum, C. nymphaeae, C. truncatum e C. cliviae. Nos testes de patogenicidade, somente isolados de E. ampelina causaram sintomas de antracnose em Vitis vinifera \'Moscato Giallo\' e Vitis labrusca \'Niagara Rosada\'. Para a caracterização de E. ampelina do Brasil e Austrália, 35 isolados foram analisados. Sequências de ITS e TEF de todos os isolados foram monomórficas. A rede de haplótipos gerada a partir de sequências de HIS3 resultou na formação de quatro haplótipos. Alta diversidade genética foi observada em dois isolados brasileiros, haplótipo EA4, sugerindo a perda do intron durante a evolução da espécie. Colônias apresentaram coloração variável, textura enrugada, ausência de esporos e lento crescimento. Isolados brasileiros apresentaram conídios maiores que conídios de isolados australianos. Para induzir a esporulação, fragmentos de micélio foram agitados e incubados em água da chuva e água destilada durante 7 dias. Isolados produziram diferentes concentrações de conídios e a germinação foi superior a 88,5%. Nos testes de infectividade, os conídios causaram sintomas de antracnose em folhas. A escala diagramática desenvolvida compreende seis diagramas em cores reais com severidade variando de 1,1 a 27,4%. O uso da escala diagramática melhorou a acurácia, precisão, concordância e reprodutibilidade das estimativas conduzidas por 12 avaliadores. A dinâmica temporal e espacial da antracnose foi conduzida em vinhedo de \'Niagara Rosada\' em 2014 e 2015. A incidência de videiras com folhas, ramos e bagas sintomáticos e a severidade em folhas foram registradas. Os sintomas de antracnose ocorreram rapidamente após a brotação sendo observada resistência ontogênica em todos os órgãos avaliados. O modelo monomolecular mostrou o melhor ajuste para o progresso da incidência. As análises temporais sugerem que o progresso da incidência e severidade durante o tempo é influenciado principalmente pelo inóculo inicial devido à resistência ontogênica dos órgãos. As análises espaciais mostraram um padrão espacial predominantemente aleatório de videiras sintomáticas. Em conclusão, esta tese apresenta uma compreensão mais aprofundada da etiologia e epidemiologia de uma importante doença da videira.

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Books on the topic "Aetiology"

1

Aetiology of urbanisation in India. Allahabad: Govind Ballabh Pant Social Science Institute, 1985.

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Lammie, George Alexander. The aetiology of the dental diseases. Bubenhall [Eng.]: G.A. Lammie, 1992.

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Ham, R. Limb amputation ; from aetiology to rehabilitation. London: Chapman and Hall, 1991.

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Mechelen, Willem Van. Aetiology and prevention of running injuries. Amsterdam: Drukkerij A1/A2, 1992.

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Miller, Anthony B., ed. Diet and the Aetiology of Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74376-4.

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Malone, P. Colm, and Paul S. Agutter. The Aetiology of Deep Venous Thrombosis. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6650-4.

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B, Miller A., ed. Diet and the aetiology of cancer. Berlin: Springer-Verlag, 1989.

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Shortt, N. C. Adult thought: Aetiology and stimulation of change. [S.l: The author], 1991.

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Obwegeser, Hugo L. Mandibular growth anomalies: Terminology, aetiology, diagnosis, treatment. Berlin: Springer, 2001.

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J, Morison M., ed. Pressure sore blueprint: Aetiology, prevention and management. [Hounslow]: Squibb, 1993.

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Book chapters on the topic "Aetiology"

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Scadding, J. G., and D. N. Mitchell. "Aetiology." In Sarcoidosis, 546–62. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4899-2971-6_25.

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Zacharin, Robert F. "Aetiology." In Pelvic Floor Anatomy and the Surgery of Pulsion Enterocoele, 65–76. Vienna: Springer Vienna, 1985. http://dx.doi.org/10.1007/978-3-7091-4075-8_3.

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Ward, Christopher. "Aetiology." In A Practical Guide to Heart Failure in Older People, 45–61. Chichester, UK: John Wiley & Sons, Ltd, 2009. http://dx.doi.org/10.1002/9780470742945.ch4.

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Mehlhorn, Heinz. "Aetiology." In Encyclopedia of Parasitology, 67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_3587.

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Mehlhorn, Heinz. "Aetiology." In Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_3587-1.

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Wilkinson, John A. "Aetiology." In Congenital Displacement of the Hip Joint, 1–11. London: Springer London, 1985. http://dx.doi.org/10.1007/978-1-4471-1369-0_1.

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Sherman, C. D., K. C. Calman, S. Eckhardt, I. Elsebai, D. Firat, D. K. Hossfeld, J. P. Paunier, and B. Salvadori. "Aetiology." In Manual of Clinical Oncology, 13–29. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-96995-9_3.

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Hughes, Graham, and Munther A. Khamashta. "Aetiology." In Hughes Syndrome: Highways and Byways, 71–74. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5161-6_15.

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Heimann, Rudi. "Aetiology." In Violence Prevention in Education, School, and Club, 33–44. Wiesbaden: Springer Fachmedien Wiesbaden, 2022. http://dx.doi.org/10.1007/978-3-658-38551-4_3.

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Berg, Charles. "Aetiology." In Clinical Psychology, 23–31. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003251514-2.

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Conference papers on the topic "Aetiology"

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Pratt, D. "The multivariate aetiology of foot pathomechanics." In IEE Colloquium Intelligent Methods in Healthcare and Medical Applications. IEE, 1998. http://dx.doi.org/10.1049/ic:19981032.

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Jira, D., L. Mair, M. Buchberger, and A. Pickhard. "Aetiology of voice prosthesis' leakage after laryngectomy." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686008.

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Mogulkoc, Nesrin, Tarik Simsek, Sanem Nalbantgil, Meral Kayikcioglu, Yasemin Kabasakal, and Hakan Kultursay. "Variation in mortality from pulmonary hypertension by aetiology." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3788.

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Addala, D., RM Mercer, Q. Lu, G. Shepherd, O. Castro, R. Varatharajah, A. Thayanandan, et al. "P102 Discordant exudative pleural effusions: demographics and aetiology." In British Thoracic Society Winter Meeting 2019, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 4 to 6 December 2019, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2019. http://dx.doi.org/10.1136/thorax-2019-btsabstracts2019.245.

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Toma, Varvara, Eugenia Buzoianu, Oana Virban, Ana Maria Sovarel-Moiceanu, Mariana Moiceanu, Liliana Cretu, Daniela Popeia, et al. "P313 Correlation between febrile seizure and aetiology of fever." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.401.

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Duijkers, Ruud, Eduard Burgmeijer, Wim Boersma, and Rene Lutter. "Cytokine-based prediction of aetiology in community acquired pneumonia." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4110.

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Grama, Alina, Lucia Burac, Simona S. Cainap, Cornel Aldea, Dan Delean, Bogdan Bulata, Diana Pacurar, Laura Bodea, Claudia Sirbe, and Tudor L. Pop. "OC50 Acute liver failure in children: aetiology and evolution." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.47.

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Börekçi, Sermin, and Benan Musellim. "Aetiology of bronchiectasis: Evaluation of 319 adult patients with bronchiectasis." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa670.

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Steiger, U., M. Weber, and H. Gerber. "AB0205 Unusual aetiology of erythema nodosum, arthritis, and pleural effusion." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.716.

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Bayston, Roger. "Aetiology, Pathogenic Mechanisms and Treatment of Infections in Spinal Instrumentation." In eccElearning Postgraduate Diploma in Spine Surgery. eccElearning, 2017. http://dx.doi.org/10.28962/01.3.153.

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Reports on the topic "Aetiology"

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LeVoir, Harriet, and Patrick Horner. Aetiology and guidelines for management of urethritis. BJUI Knowledge, August 2022. http://dx.doi.org/10.18591/bjuik.0669.v2.

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O'Kelly, John, and Frank O'Brien. Aetiology and diagnosis of bacterial chronic prostatitis (Type II) and chronic pelvic pain syndrome (CPPS) Type III. BJUI Knowledge, January 2020. http://dx.doi.org/10.18591/bjuik.0059.

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Keating, Louise, Ailish Malone Name, Maire-Brid Casey, Ciaran Bolger, Dara Meldrum, and Catherine Doody. Conservative Primary Care Management for Recent Onset Cervical Radiculopathy – a Systematic Review & Meta-analysis Protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0047.

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Review question / Objective: To investigate the effectiveness of conservative management available in primary care for adults with recent onset (less than 12 weeks) cervical radiculopathy. Conservative management will be compared to any available comparator i.e. no treatment, placebo or any treatment. Eligibility criteria: Inclusion criteria – trials (as defined in item 15) investigating any conservative management (e.g. exercise, advice, manual therapy, traction, acupuncture, pharmacology etc), involving adults with single level CR (as defined in item 10) of any aetiology, with symptom duration of 12 weeks or less, and including 1 or more of the following outcomes i.e. pain, disability, overall improvement or satisfaction with intervention, quality of life or participation restriction. Exclusion criteria – full text not available, not a randomised controlled trial, trials not involving CR (e.g. cervicobrachial pain, neck pain only), trials involving chronic CR, multilevel or bilateral CR (polyradiculopathy) or radiculomyelopathy, major or systemic pathology, post-surgery interventions, trials of surgery or spinal injection only, or involving a paediatric population or not in English language.

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Grueso-Navarro, Elena, Leticia Rodríguez-Alcolado, Ángel Arias, Emilio J. Laserna-Mendieta, and Alfredo J. Lucendo. Influence of HLA-DQA1*05 allele in the response to anti-TNFα drugs in inflammatory bowel diseases. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2023. http://dx.doi.org/10.37766/inplasy2023.2.0076.

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Review question / Objective: Do patients with inflammatory bowel disease and treated with any anti-TNFα drug who had the HLA-DQA1*05 allele (in heterozygosis or homozygosis) have lower response or persistence to those drugs than patients without HLA-DQA1*05 allele? Condition being studied: Inflammatory bowel diseases (IBD), which includes Crohn’s disease (CD) and ulcerative colitis (UC), is a chronic inflammatory condition that may affect any part of the digestive tract (CD) or be limited to the colon (UC). While the specific aetiology of IBD remains unknown, it is believed to involve a complex impairment in the immunity of the gut mucosa due to a combination of several genetic and environmental factors, being the microbiota one of the latest that more attraction has received in recent years. Symptoms of IBD (such as abdominal pain, diarrhoea, fever, tiredness or rectal bleeding) may be either constant or alternate between periods of limited disease activity and flares with remarkable presence of symptoms. As IBD is associated with significant morbidity and disability, pharmacological treatment is required in most cases, especially in CD, aimed at reducing the inflammatory response and attenuating the activity of the immune system. In the moderate and severe forms of the disease, therapy is usually based on immunosuppressant and/or biological drugs. Among the latest, anti-TNFα drugs (infliximab or adalimumab) are normally chosen as the initial biological therapy.

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Aetiology and management of urethritis. BJUI Knowledge, July 2017. http://dx.doi.org/10.18591/bjuik.0669.

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Aetiology and assessment of ureteric obstruction. BJUI Knowledge, July 2018. http://dx.doi.org/10.18591/bjuik.0662.

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Aetiology, presentation and management of prostatic abscess. BJUI Knowledge, December 2018. http://dx.doi.org/10.18591/bjuik.0415.

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Urinary retention in women: aetiology and diagnostic approach. BJUI Knowledge, April 2018. http://dx.doi.org/10.18591/bjuik.0654.

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A cognitive neuroscience review of the aetiology of ADHD. ACAMH, August 2018. http://dx.doi.org/10.13056/acamh.10576.

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A simple neurological explanation has yet to identify an aetiology and pathogenesis of the disorder. However, advancements in imaging techniques should help to give a more detailed understanding of the brain regions that are different to those without ADHD.

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Aetiology of shame and its association with adolescent depression and anxiety - CAMHS around the Campfire. ACAMH, July 2021. http://dx.doi.org/10.13056/acamh.16552.

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For this session we welcomed Professor Thalia Eley, Professor of Developmental Behavioural Genetics, KCL, to discuss her JCPP paper 'Aetiology of shame and its association with adolescent depression and anxiety: results from a prospective twin and sibling study.'

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