Academic literature on the topic 'Aedes aegypti Infection Rate'
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Journal articles on the topic "Aedes aegypti Infection Rate"
Kirstein, Oscar David, Guadalupe Ayora-Talavera, Edgar Koyoc-Cardeña, Daniel Chan Espinoza, Azael Che-Mendoza, Azael Cohuo-Rodriguez, Pilar Granja-Pérez, et al. "Natural arbovirus infection rate and detectability of indoor female Aedes aegypti from Mérida, Yucatán, Mexico." PLOS Neglected Tropical Diseases 15, no. 1 (January 4, 2021): e0008972. http://dx.doi.org/10.1371/journal.pntd.0008972.
Full textIstianah, Siti, Budi Mulyaningsih, Sitti Rahmah Umniyati, and Eggi Arguni. "Aedes aegypti as potential vector of filariasis in Pekalongan, Central Java Province, Indonesia." Jurnal Kedokteran dan Kesehatan Indonesia 12, no. 1 (April 30, 2021): 44–51. http://dx.doi.org/10.20885/jkki.vol12.iss1.art8.
Full textPérez-Pérez, Juliana, Víctor Hugo Peña-García, Arley Calle-Tobón, Marcela Quimbayo-Forero, Raúl Rojo, Enrique Henao, Talya Shragai, and Guillermo Rúa-Uribe. "Entomovirological Surveillance in Schools: Are They a Source for Arboviral Diseases Transmission?" International Journal of Environmental Research and Public Health 18, no. 11 (June 6, 2021): 6137. http://dx.doi.org/10.3390/ijerph18116137.
Full textNoshirma, Monika, Ruben Wadu Willa, Muhammad Kazwaini, and Arief Wibowo. "Deteksi Virus Dengue pada Nyamuk Aedes aegypti (Diptera: Culicidae) yang Tersebar di Kabupaten Sumba Timur dan Sumba Barat Daya." Jurnal Vektor Penyakit 14, no. 1 (June 2, 2020): 57–64. http://dx.doi.org/10.22435/vektorp.v14i1.2421.
Full textGloria-Soria, A., P. M. Armstrong, J. R. Powell, and P. E. Turner. "Infection rate of Aedes aegypti mosquitoes with dengue virus depends on the interaction between temperature and mosquito genotype." Proceedings of the Royal Society B: Biological Sciences 284, no. 1864 (October 4, 2017): 20171506. http://dx.doi.org/10.1098/rspb.2017.1506.
Full textYang, Cihan, Fei Wang, Doudou Huang, Haixia Ma, Lu Zhao, Guilin Zhang, Hailong Li, et al. "Vector competence and immune response of Aedes aegypti for Ebinur Lake virus, a newly classified mosquito-borne orthobunyavirus." PLOS Neglected Tropical Diseases 16, no. 7 (July 18, 2022): e0010642. http://dx.doi.org/10.1371/journal.pntd.0010642.
Full textGRAY, E. M., and T. J. BRADLEY. "Malarial infection in Aedes aegypti : effects on feeding, fecundity and metabolic rate." Parasitology 132, no. 02 (October 3, 2005): 169. http://dx.doi.org/10.1017/s0031182005008966.
Full textSanchez-Vargas, Irma, Laura Harrington, William Black, and Ken Olson. "Analysis of Salivary Glands and Saliva from Aedes albopictus and Aedes aegypti Infected with Chikungunya Viruses." Insects 10, no. 2 (February 1, 2019): 39. http://dx.doi.org/10.3390/insects10020039.
Full textChung, Youne Kow, and Fung Yin Pang. "Dengue virus infection rate in field populations of female Aedes aegypti and Aedes albopictus in Singapore." Tropical Medicine and International Health 7, no. 4 (April 2002): 322–30. http://dx.doi.org/10.1046/j.1365-3156.2002.00873.x.
Full textPeinado, Stephen A., Matthew T. Aliota, Bradley J. Blitvich, and Lyric C. Bartholomay. "Biology and Transmission Dynamics of Aedes flavivirus." Journal of Medical Entomology 59, no. 2 (January 22, 2022): 659–66. http://dx.doi.org/10.1093/jme/tjab197.
Full textDissertations / Theses on the topic "Aedes aegypti Infection Rate"
Schwab, Anne Elisabeth. "The impact of selective oviposition, egg hatchability, food availability and infection with Plagiorchis elegans on the pre-imago population dynamics of Aedes aegypti (Diptera:Culicidae) /." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31534.
Full textWang, Hua. "The role of apoptosis during infection of Aedes aegypti by Sindbis virus." Diss., Kansas State University, 2011. http://hdl.handle.net/2097/11976.
Full textDepartment of Biology
Rollie J. Clem
Each year, over 500 million people are infected with mosquito-borne diseases, including malaria, yellow fever and dengue fever, which cause several million deaths, and long-term disability and suffering. This dissertation focused on the mosquito Aedes aegypti, a vector for dengue virus and yellow fever virus. Since Sindbis virus (SINV) is an arthropod-borne virus (arbovirus) that is vectored by A. aegypti and is well characterized at the molecular level, the SINV - A. aegypti model was used to determine whether apoptosis plays a role in the control of vector competency. In Chapter 2, the effects of inducing or inhibiting apoptosis on SINV replication were tested in mosquito cells. It was observed that recombinant SINVs expressing pro-apoptotic genes caused extensive apoptosis in mosquito cells, with decreased virus production after the cells underwent apoptosis. Infection of mosquito cells with SINV expressing the caspase inhibitor P35 inhibited actinomycin D-induced apoptosis, but had no observable effects on virus replication. This study was the first to test directly whether inducing or inhibiting apoptosis affects arbovirus replication in mosquito cells. Chapter 3 examined the effects of silencing apoptosis regulatory genes on SINV replication and dissemination in A. aegypti. Genes which either positively or negatively regulate apoptosis were silenced by RNA interference in mosquitoes, which were then infected with a recombinant SINV expressing green fluorescent protein (GFP). Reciprocal effects were observed on both the occurrence and intensity of expression of GFP in various tissues. These results suggest that systemic apoptosis positively influences SINV replication in A. aegypti. This was the first direct study to explore the role of apoptosis in determining mosquito vector competence for arboviruses. Finally, in Chapter 4, the mechanisms of apoptosis were explored in A. aegypti. Overexpression of IAP antagonists caused extensive cell death in mosquito cells, while silencing the expression of IAP antagonists attenuated apoptosis. The results showed that the IAP binding motif (IBM) of IAP antagonists was critical for their binding to AeIAP1. The IAP antagonists released initiator and effector caspases from AeIAP1 by competing for the binding sites and caused caspase-dependent apoptosis. These findings imply that the mechanisms of IAP antagonists regulating apoptosis are conserved between mosquitoes and the model insect where apoptosis has been mainly studied, Drosophila melanogaster.
Enguehard, Margot. "Interaction between chikungunya and dengue viruses during co-infection in Aedes mosquito cells and in Aedes aegypti mosquito." Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1161/document.
Full textEmergence and geographical extension of dengue (DENV), Zika (ZIKV) and chikungunya (CHIKV) viruses increase simultaneous outbreak in an increasing number of countries. To date, no vaccine or cure have yet been developed against these diseases those cause a tremendous impact on human health and in the economy worldwide. During recent simultaneous outbreaks, up to 12% of patients have been diagnosed to be co-infected by CHIKV and DENV. In addition, it was shown that the mosquitoes Aedes albopictus could carry and transmit simultaneously CHIKV and DENV. However, the pathology, as well as the epidemiology of a pathogen, relies on the interactions between several infectious agents present within an organism or a community in the environment. It is crucial to consider to which extent a host infected by a first microorganism is modified and whether its reaction to the infection by a second microorganism is consequently altered. However, there is no extensive report of Alphavirus-Flavivirus or Flavivirus- Flavivirus interactions. Our global objective is to characterize these co-infections in both mosquitoes and humans, at the cell and molecular level. To this aim, we started this project by performing sequential co- infection in different cell lines from Aedes albopictus and Aedes aegypti. We found that the permissiveness and production of DENV is enhanced in presence of CHIKV. On the contrary, there is no effect of DENV pre-infection on subsequent CHIKV co-infection. We generalized the synergistic phenomena and we showed that CHIKV pre-infection also increased the infection by DENV-1, DENV-3 and DENV-4, but also by two others re-emerging Flaviviruses, the Yellow Fever Virus (YFV), and the Zika Virus (ZIKV). Remarkably, we succeeded to establish a mosquito model of co-infection of Aedes aegypti mosquito after by different two feedings at 4 days interval. Using this sequential co-infection, we were able to show that a pre-infection of Aedes aegypti by CHIKV increase the level of DENV-2 RNA in salivary glands compare to mono-infected mosquitos. This phenotype is reminiscent of the phenotype we observed in vitro during successive infections. Altogether, our study paves the way to the characterization of molecular interaction between Flaviviruses and Alphaviruses in mosquito in vitro and in vivo. This study can be crucial for a better understanding of disease and epidemiology during simultaneous outbreaks
Chalk, Roderick. "Immunity in Aedes aegypti and the role of antibacterial peptides in Brugia pahangi infection." Thesis, University of Liverpool, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333493.
Full textLowenberger, Carl A. (Carl Arnold). "Plagiorchis elegans from cercariae to infective metacercariae : factors affecting transmission, requirements for development, and behavioural responses of intermediate hosts to infection." Thesis, McGill University, 1993. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=41236.
Full textO'Neill, Katelyn Leigh. "The role of apoptotic factors in Sindbis virus infection and replication in the mosquito vector Aedes aegypti." Diss., Kansas State University, 2013. http://hdl.handle.net/2097/15378.
Full textDepartment of Division of Biology
Rollie J. Clem
Mosquitoes are carriers of a variety of harmful human pathogens, including viruses. In order to be successfully transmitted, a virus must evade mosquito immune responses. In this work, the innate immune role of apoptosis in mosquito-virus interactions was examined utilizing the disease vector Aedes aegypti and Sindbis virus. Ae. aegypti is the main vector for yellow fever and dengue virus, which result in over 100 million infections per year. Sindbis virus (Togaviridae) can be transmitted to vertebrates by Ae. aegypti in the laboratory. Sindbis is also well characterized molecularly, making it a good model system for understanding virus-vector interactions. Sindbis MRE-16 recombinant virus clones were utilized to express either an antiapoptotic or pro-apoptotic gene during virus replication. Mosquitoes were infected with recombinant virus clones during a blood meal or by intrathoracic injection. Midgut tissue and whole body samples were analyzed for virus infection and dissemination. Virus was also quantified in saliva and mosquito survival was assayed. Decreased infection in the midgut and delayed virus replication were observed in mosquitoes that were infected with virus expressing a pro-apoptotic gene. Infection with this virus clone also resulted in less virus in the saliva and reduced survival of infected mosquitoes. In addition, negative selection against pro-apoptotic gene expression during virus replication was observed. Collectively, these data suggest that apoptosis can serve as an antiviral defense in Ae. aegypti and may potentially be exploited to control virus transmission. An additional study included in this dissertation focused on zebrafish development and migration of somitic precursors from the tailbud. The tailbud consists of a population of stem cells at the posterior tip of the embryonic tail. The exit of these stem cells from the tailbud is required for the formation of tail somites. A novel double mutant was identified that lacked the t-box transcription factor spadetail and the BMP inhibitor chordin. Double mutants completely lacked somites and had an enlarged tailbud due to accumulation of stem cells that were unable to exit the tailbud. This study indicates the importance of BMP inhibition and spadetail expression in the proper exit of muscle precursors from the tailbud.
Huang, Yan-Jang. "Identification and characterization of the genetic determinants for yellow fever virus infection and dissemination in Aedes aegypti." Diss., Kansas State University, 2014. http://hdl.handle.net/2097/18149.
Full textDepartment of Diagnostic Medicine/Pathobiology
Stephen Higgs
The genetic composition of arboviruses is a critical determinant of viral infectivity and the capacity for virus dissemination in arthropod vectors. Due to concerns related to a hypothetical potential for loss of attenuation, the supression of vector infection and dissemination is a critical component for the rationale-based design of live-attenuated flavivirus vaccine candidates. The yellow fever virus (YFV) 17D vaccine virus is not only attenuated in vertebrates, but also has low infectivity for Aedes agypti mosquitoes and since it does not disseminate, it is not transmissible. Using a reverse genetics system, the mutations present in the envelope protein YFV 17D virus were characterized in Ae. aegypti to determine the role of mutations in limiting the viral infectivity and dissemination capacity. This knowledge would contribute to the rational design of live attenuated vaccines with the desirable phenotype of being nontransmissible by arthropod vectors. The upper lateral portion of the YFV 17D envelope (E) protein domain III (EDIII) habors the T380R mutation in the FG loop. Experiments demonstrated that the T380R mutation was associated with the viral infectivity phenotype for mosquitoes, but did not influence dissemination into the secondary tissues. The G52R mutation in the molecular hinge region that is located between E protein domains I (EDI) and II, significantly reduced viral infectivity for mosquitoes. In contrast, when cloned into the Asibi wildtype virus genetic backbone, the T173I mutation in the loop structure between the G0 and H0 β- strands did not attenuate viral infection and dissemination. The double mutant virus containing both the G52R and T173I mutations in the E protein, showed a similar attenuated reduced infectivity to the single G52R mutant. The M299I mutation in the linker region between EDI and EDIII resulted in a significantly lower viral infectivity at the initial phase of viral infection at 7 days post-infection in Ae. aegypti. In conclusion, the characterization on four mutations in the YFV 17D vaccine E protein have demonstrated three genetic loci, that can influence the process of YFV infection in Ae. aegypti. These results provide new knowledge and understanding which may have broad applications for the rationale design of safe flavivirus vaccines, via targeting genetic loci and introducing specific mutations that preclude infection of, and transmission by arthropod vectors.
Wallage, Helena Rachelle. "The effects of Plagiorchis elegans (Trematoda: plagiorchiidae) infection on the carbohydrate metabolism of fourth instar Aedes aegypti (DipteraCulicidae) larvae." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0034/MQ64474.pdf.
Full textGrunnill, Martin David. "Inapparent and vertically transmitted infections in two host-virus systems." Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/20866.
Full textWallage, Helena Rachelle. "The effects of Plagiorchis elegans (Trematoda : Plagiorchiidae) infection on the carbohydrate metabolism of fourth instar Aedes aegypti (Diptera : Culicidae) larvae." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30763.
Full textBooks on the topic "Aedes aegypti Infection Rate"
Shieh, Jong-Neng. Influence of host anemia on blood-feeding rate and egg production of Aedes aegypti (L.) (Diptera : Culicidae). 1991.
Find full textMonath, Thomas P., and J. Erin Staples. Yellow fever. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0045.
Full textBook chapters on the topic "Aedes aegypti Infection Rate"
Choudhari, Ranjana Hanumant. "Multidimensional Impact of Climate Change on Human Reproduction and Fertility." In Research Anthology on Environmental and Societal Impacts of Climate Change, 1672–709. IGI Global, 2022. http://dx.doi.org/10.4018/978-1-6684-3686-8.ch083.
Full textChoudhari, Ranjana Hanumant. "Multidimensional Impact of Climate Change on Human Reproduction and Fertility." In Climate Change and Its Impact on Fertility, 278–315. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-4480-8.ch014.
Full textChoudhari, Ranjana Hanumant. "Multidimensional Impact of Climate Change on Human Reproduction and Fertility." In Climate Change and Its Impact on Fertility, 278–315. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-4480-8.ch014.
Full textLyaruu, Lucille. "Chikungunya Virus Transmission." In Chikungunya Virus - A Growing Global Public Health Threat. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100199.
Full textHung, Nguyen Thanh. "Dengue." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha, 1170–74. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0181.
Full textWills, Bridget, and Yee-Sin Leo. "Dengue." In Oxford Textbook of Medicine, edited by Christopher P. Conlon, 845–52. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0090.
Full textA. Fernandez Cerna, Eduardo, Catalina Sherman, and Mercedes Martinez. "Dengue Reduction through Vector Control." In Dengue Fever in a One Health Perspective - Latest Research and Recent Advances [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.109603.
Full textOldstone, Michael B. A. "Yellow Fever." In Viruses, Plagues, and History, 89–122. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190056780.003.0005.
Full text"Virus isolations Mosquito collections obtained during most field trips to the north-west of Western Australia have been processed for virus isolation. Until 1985, virus isolation was undertaken by intracerebral inoculation of suckling mice, but this was then replaced by cell culture using C6/36 mosquito, PSEK, BHK and Vero cells. The use of cell culture has significantly reduced the overall virus isolation rate by largely excluding arboviruses, rhabdoviruses and most bunyaviruses, but is as effective as suckling mice for the isolation of flaviviruses and alphaviruses. MVE virus has been isolated every year that significant numbers of adult mosquitoes have been processed except 1983 (Broom et al. 1989; Broom et al. 1992; Mackenzie et al. 1994c). Isolations of MVE, Kunjin and other flaviviruses are shown in Table 8.2. There was a strong correlation between the number of virus isolates in any given year and the prevailing environmental conditions. Thus those years with a heavy, above average wet season rainfall and subsequent widespread flooding yielded large numbers of virus isolates (1981, 1991, 1993) compared with years with average or below average rainfall and with only localized flooding. Although most MVE virus isolates were obtained from Culex annulirostris mosquitoes, occasional isolates were also obtained from a variety of other species, including Culex quinquefasciatus, Culex palpalis, Aedes normanensis, Aedes pseudonormanensis, Aedes eidvoldensis, Aedes tremulus, Anopheles annulipes, Anopheles bancroftii, Anopheles amictus and Mansonia uniformis (cited in Mackenzie et al. 1994b; Mackenzie and Broom 1995), although the role of these species in natural transmission cycles has still to be determined. Virus carriage rates in Culex annulirostris mosquitoes are shown in Table 8.3 for the Ord River area (Kununurra–Wyndham) and Balgo and Billiluna in south-east Kimberley. Very high mosquito infection rates were observed in those years with above average rainfall. Virus spread and persistence Stanley (1979) suggested that viraemic waterbirds, which are often nomadic, may generate epidemic activity of MVE in south-east Australia and in the Pilbara region. In an attempt to understand the genesis of epidemic activity better, our laboratory initiated a long-term study in the arid south-east Kimberley area at Billiluna and Balgo, two Aboriginal communities on the northern edge of the Great Sandy Desert. Occasional cases of Australian encephalitis had occurred in both communities (1978, 1981). The studies have clearly shown that MVE virus activity only occurs following very heavy, widespread rainfall both locally and in the catchment area of the nearby watercourse, Sturt Creek, which results in extensive flooding across its floodplain (Broom et al. 1992). Localized flooding is insufficient to generate virus activity. Two possible explanations can be proposed to account for the reappearance of MVE virus activity when environmental conditions are suitable: either virus can be reintroduced into the area by viraemic waterbirds arriving from enzootic areas further north; or virus may." In Water Resources, 133–35. CRC Press, 1998. http://dx.doi.org/10.4324/9780203027851-26.
Full textConference papers on the topic "Aedes aegypti Infection Rate"
Yasrul, Rahma Triyana, Sitti Rahmah Umniyati, and Budi Mulyaningsih. "The effect of anticoagulant on the feeding rate, mortality rate, and infection rate of Aedes aegypti (diptera:culicidae) orally infected with dengue virus-3." In INTERNATIONAL CONFERENCE ON BIOINFORMATICS AND NANO-MEDICINE FROM NATURAL RESOURCES FOR BIOMEDICAL RESEARCH: 3rd Annual Scientific Meeting for Biomedical Sciences. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5109977.
Full textAngeliana Kusumaningtiar, Devi, Nur Endah Wahyuningsih, Retno Hestiningsih, and Taufik Rendi Anggara. "Survival Resistance Effects of Cypermethrin on Rate of Aedes Aegypti." In International Conference Recent Innovation. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0009949926502656.
Full textPongsumpun, Puntani. "Dengue Infection Model with Temperature and the biting of Aedes Aegypti and Ades Albopictus in Thailand." In IPMV 2020: 2020 2nd International Conference on Image Processing and Machine Vision. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3421558.3421579.
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