Relevant bibliographies by topics / Adhesion

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Adhesion'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 October 2024

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Journal articles on the topic "Adhesion"

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Yu-Hsing Lin, Pi-Hsin Chen, Ya-Ling Cyue, Keng-Chia Hsu, Shih-Yi Guo, Ya-Peng Wang, Tsung-Han Wu, et al. "Establishment of a postoperative bowel adhesion model in Rats." International Journal of Scholarly Research in Science and Technology 5, no. 1 (August 30, 2024): 001–8. http://dx.doi.org/10.56781/ijsrst.2024.5.1.0028.

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Postoperative adhesive bowel obstruction is a frequent cause of hospital admission in a surgical department. Emergency surgery is needed in a majority of patients with bowel ischemia or peritonitis; most adhesive bowel obstruction can be managed non-operatively. Many studies have investigated benefits of using oral water-soluble contrast to manage adhesive bowel obstruction. Treatment recommendations are still controversial. Intestinal adhesions commonly occur in patients after abdominal surgery, often between intestines, between intestines and the abdominal wall, or within the peritoneum. Adhesions can lead to symptoms such as abdominal pain, constipation, and bloating, and may result in serious complications such as intestinal obstruction, bowel necrosis, acute peritonitis, and sepsis, which can be life-threatening. Therefore, reducing the occurrence of intestinal adhesions is crucial for abdominal surgery. To minimize adhesions, endoscopic techniques can be employed to reduce the abdominal opening area and thus decrease the probability of adhesion formation. However, this approach requires specialized equipment and surgical skills, making it somewhat challenging. Alternatively, anti-adhesion products can be used to lower the risk of adhesion formation. In this study, a murine cecal abrasion adhesion model was established and will be simulated common adhesion scenarios observed in patients after abdominal surgery. The efficacy of anti-adhesion gel materials will be then evaluated to assess their anti-adhesion properties. The record of gross adhesion severity assessment was performed. The adhesion status between the cecum and abdominal wall was observed on postoperative days 3 and 14. The results was shown that three days post-surgery operation, the group exhibited more severe adhesions upon gross examination. By day 14 post-surgery operation (at animal sacrifice), the histopathological analysis revealed that the group exhibited thicker fibrous tissue thickness, increased neovascularization, and higher collagen accumulation in the peritoneum. Therefore, based on the results of this experiment, a postoperative bowel adhesion rat is reliable and can be safely used to test the efficacy of novel anti-adhesive formulations to prevent intra-abdominal adhesions by using this model.
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Willaert, Ronnie G., Yeseren Kayacan, and Bart Devreese. "The Flo Adhesin Family." Pathogens 10, no. 11 (October 28, 2021): 1397. http://dx.doi.org/10.3390/pathogens10111397.

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The first step in the infection of fungal pathogens in humans is the adhesion of the pathogen to host tissue cells or abiotic surfaces such as catheters and implants. One of the main players involved in this are the expressed cell wall adhesins. Here, we review the Flo adhesin family and their involvement in the adhesion of these yeasts during human infections. Firstly, we redefined the Flo adhesin family based on the domain architectures that are present in the Flo adhesins and their functions, and set up a new classification of Flo adhesins. Next, the structure, function, and adhesion mechanisms of the Flo adhesins whose structure has been solved are discussed in detail. Finally, we identified from Pfam database datamining yeasts that could express Flo adhesins and are encountered in human infections and their adhesin architectures. These yeasts are discussed in relation to their adhesion characteristics and involvement in infections.
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Lee, Y. K., and K. Y. Puong. "Competition for adhesion between probiotics and human gastrointestinal pathogens in the presence of carbohydrate." British Journal of Nutrition 88, S1 (September 2002): S101—S108. http://dx.doi.org/10.1079/bjn2002635.

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The adhesion ofLactobacillus rhamnosusGG to human enterocyte-like Caco-2 cells was not inhibited by eight carbohydrates tested, namelyN-acetyl-glucosamine, galactose, glucose, fructose, fucose, mannose, methyl-α-D-mannopyranoside and sucrose. The degree of hydrophobicity predicted the adhesion ofL. rhamnosusGG to Caco-2 cells.L. rhamnosusGG, however, was able to compete withEscherichia coliandSalmonellaspp. of low hydrophobicity and high adhesin–receptor interaction for adhesion to Caco-2 cells. The interference of adhesion of these gastrointestinal (GI) bacteria byL. rhamnosusGG was probably through steric hindrance, and the degree of inhibition was related to the distribution of the adhesin receptors and hydrophobins on the Caco-2 surface. A Carbohydrate Index for Adhesion (CIA) was used to depict the binding property of adhesins on bacteria surfaces. CIA was defined as the sum of the fraction of adhesion in the presence of carbohydrates, with reference to the adhesion measured in the absence of any carbohydrate. The degree of competition for receptor sites betweenLactobacillus caseiShirota and GI bacteria is a function of their CIA distance. There were at least two types of adhesins on the surface ofL. caseiShirota. The study provides a scientific basis for the screening and selection of probiotics that compete with selective groups of pathogens for adhesion to intestinal surfaces. It also provides a model for the characterisation of adhesins and adhesin–receptor interactions.
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Del Canto, Felipe, Douglas J. Botkin, Patricio Valenzuela, Vsevolod Popov, Fernando Ruiz-Perez, James P. Nataro, Myron M. Levine, et al. "Identification of Coli Surface Antigen 23, a Novel Adhesin of Enterotoxigenic Escherichia coli." Infection and Immunity 80, no. 8 (May 29, 2012): 2791–801. http://dx.doi.org/10.1128/iai.00263-12.

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ABSTRACTEnterotoxigenicEscherichia coli(ETEC) is an important cause of diarrhea, mainly in developing countries. Although there are 25 different ETEC adhesins described in strains affecting humans, between 15% and 50% of the clinical isolates from different geographical regions are negative for these adhesins, suggesting that additional unidentified adhesion determinants might be present. Here, we report the discovery of Coli Surface Antigen 23 (CS23), a novel adhesin expressed by an ETEC serogroup O4 strain (ETEC 1766a), which was negative for the previously known ETEC adhesins, albeit it has the ability to adhere to Caco-2 cells. CS23 is encoded by an 8.8-kb locus which contains 9 open reading frames (ORFs), 7 of them sharing significant identity with genes required for assembly of K88-related fimbriae. This gene locus, namedaal(adhesion-associatedlocus), is required for the adhesion ability of ETEC 1766a and was able to confer this adhesive phenotype to a nonadherentE. coliHB101 strain. The CS23 major structural subunit, AalE, shares limited identity with known pilin proteins, and it is more closely related to the CS13 pilin protein CshE, carried by human ETEC strains. Our data indicate that CS23 is a new member of the diverse adhesin repertoire used by ETEC strains.
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Nakamura, N., J. Tanaka, and K. Sobue. "Rous sarcoma virus-transformed cells develop peculiar adhesive structures along the cell periphery." Journal of Cell Science 106, no. 4 (December 1, 1993): 1057–69. http://dx.doi.org/10.1242/jcs.106.4.1057.

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Alteration of the cell/substratum adhesive structures of rat fibroblasts (3Y1 cells) upon transformation by Rous sarcoma virus (RSV) was investigated by immunofluorescence microscopy. In serum-containing culture medium, 3Y1 cells developed focal adhesions as their main adhesive structures, while BY1 cells expressed peculiar close contacts along the cell periphery with the vitronectin receptor integrin, in addition to podosomes. These peripheral close contacts are referred to as the peripheral adhesions. The peripheral adhesions were observed as a darker region than podosomes by interference reflection microscopy. They were more easily destroyed by incubating the cells with RGD-containing peptide than were the focal adhesions. In contrast to focal adhesions and podosomes, actin bundles were not detected within the peripheral adhesions, where pp60v-src and tyrosine-phosphorylated proteins accumulated. Expression of the integrin was determined by the substratum composition when BY1 cells were cultured in serum-free culture medium. Under such conditions, BY1 cells expressed the peripheral adhesions within 3 hours on adhesion molecule-coated glass. On the other hand, in serum-containing medium, they first developed focal adhesions transiently at their early stage of adhesion, and then the peripheral adhesions were predominantly expressed within 12 hours. Podosomes were formed in a time course similar to that of the peripheral adhesions. These findings suggest that the peripheral adhesion is a class of stable adhesive structure distinct from the focal adhesion or podosome of BY1 cells. Similar close contact-type peripheral adhesions with the integrin were also observed in a variety of cultured cells such as normal fibroblasts at their logarithmic growth phase, phorbol ester-treated fibroblasts, and several malignant tumor cells, with poorly organized focal adhesions and stress fibers. These findings further suggest that the peripheral adhesions may be widely involved in the adhesion of cells that inadequately develop stress fibers and focal adhesions.
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Orazov, Mekan R., Viktor E. Radzinsky, Marina B. Khamoshina, Михалева M. Mikhaleva, and Sevinc Ya Ismailzade. "Anti-adhesive barriers in clinical practice: personalized patient management." Gynecology 23, no. 6 (December 15, 2021): 480–84. http://dx.doi.org/10.26442/20795696.2021.6.201292.

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The adhesive process is an urgent problem of operative gynecology. The number of women suffering from complications of the adhesive process is growing every year, not to mention the deaths associated with this problem. Polygenic etiology, low efficiency of treatment of the adhesion process determined the priority of searching for methods to prevent the process of adhesion formation or, at least, to reduce the severity of postoperative adhesion, at least a decrease in the severity of postoperative adhesion. The review presents the current paradigm of the pathogenesis of the formation of postoperative adhesions and the possibility of preventing adhesive disorders in gynecological patients.
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Douglas, L. Julia. "Adhesin - receptor interactions in the attachment ofCandida albicansto host epithelial cells." Canadian Journal of Botany 73, S1 (December 31, 1995): 1147–53. http://dx.doi.org/10.1139/b95-371.

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The ability of Candida albicans to adhere to a variety of host surfaces is thought to be an important factor in the pathogenesis of candidosis. Adhesion of the yeast form of the fungus to epithelial cells can involve several kinds of adhesion – receptor interaction. Yeast adhesins are typically mannoproteins associated with fibrils or fimbriae on the fungal surface. Lectinlike interactions have been identified between the protein portion of two mannoprotein adhesins and glycosides containing L-fucose or N-acetylglucosamine. The fucoside-binding adhesin has been purified and shown to have an affinity for glycosphingolipid receptors carrying the H blood-group antigen. A fimbrial adhesin has also been described that binds to gangliosides containing a βGalNAc(1–4)βGal disaccharide sequence. Other mannoprotein adhesins proposed recently include the factor 6 epitope present on serotype A strains of C. albicans and an integrin analogue. Adhesin expression appears to be regulated by a number of environmental signals, including osmolarity and the availability of iron and sugars. Additional adhesion-dependent signals might trigger further responses such as the initiation of morphogenesis. Key words: Candida albicans, yeast adhesion, epithelial cell adhesion.
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Al-Kaabi, Arshad F. Jassem. "Evaluating The Effect of Humidity on Adhesion Strength of Skin Adhesive." Molecular and Cellular Biomedical Sciences 4, no. 3 (November 2, 2020): 135. http://dx.doi.org/10.21705/mcbs.v4i3.148.

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Background: Skin adhesive has been used for attaching certain medical application to the human skin for functional and/or esthetic purposes. Silicone adhesive is the most common type of skin adhesives that are recently used. This study aims to evaluate the possible effect of humidity on the performance of silicone skin adhesive.Materials and Methods: Twenty-four silicone samples were divided into 2 main groups based on relative humidity (RH) exposure, namely 43% RH and 98% RH. Six samples from each group were tested for adhesion strength after 1 hour of adhesion, and the other 6 samples were tested after 2 hours of adhesion by conducting 180 degree peel test. The data were statistically analyzed for significant difference. Results: The results showed that at 43% RH, the adhesion strength was higher than the 98% RH group. The results also showed that at both humidity settings the adhesion strength after the first hours of adhesion was lower than the adhesion strength after the second hour.Conclusion: The silicone skin adhesive performance can be affected by the increase of relative humidity which needs more time of application to skin to reach the best adhesion function.Keywords: adhesions strength, humidity effect on adhesion, silicone adhesive, skin adhesives
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Taylor, James T., Rebekka Harting, Samer Shalaby, Charles M. Kenerley, Gerhard H. Braus, and Benjamin A. Horwitz. "Adhesion as a Focus in Trichoderma–Root Interactions." Journal of Fungi 8, no. 4 (April 6, 2022): 372. http://dx.doi.org/10.3390/jof8040372.

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Fungal spores, germlings, and mycelia adhere to substrates, including host tissues. The adhesive forces depend on the substrate and on the adhesins, the fungal cell surface proteins. Attachment is often a prerequisite for the invasion of the host, hence its importance. Adhesion visibly precedes colonization of root surfaces and outer cortex layers, but little is known about the molecular details. We propose that by starting from what is already known from other fungi, including yeast and other filamentous pathogens and symbionts, the mechanism and function of Trichoderma adhesion will become accessible. There is a sequence, and perhaps functional, homology to other rhizosphere-competent Sordariomycetes. Specifically, Verticillium dahliae is a soil-borne pathogen that establishes itself in the xylem and causes destructive wilt disease. Metarhizium species are best-known as insect pathogens with biocontrol potential, but they also colonize roots. Verticillium orthologs of the yeast Flo8 transcription factor, Som1, and several other relevant genes are already under study for their roles in adhesion. Metarhizium encodes relevant adhesins. Trichoderma virens encodes homologs of Som1, as well as adhesin candidates. These genes should provide exciting leads toward the first step in the establishment of beneficial interactions with roots in the rhizosphere.
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Kikuchi, Kiyoshi, Kentaro Setoyama, Seiya Takada, Shotaro Otsuka, Kazuki Nakanishi, Kosuke Norimatsu, Akira Tani, et al. "E8002 Inhibits Peripheral Nerve Adhesion by Enhancing Fibrinolysis of l-Ascorbic Acid in a Rat Sciatic Nerve Model." International Journal of Molecular Sciences 21, no. 11 (June 1, 2020): 3972. http://dx.doi.org/10.3390/ijms21113972.

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Perineural adhesions leading to neuropathy are one of the most undesirable consequences of peripheral nerve surgery. However, there are currently no widely used compounds with anti-adhesive effects in the field of peripheral nerve surgery. E8002 is a novel, anti-adhesive, multi-layer membrane that contains L-ascorbic acid (AA). Here, we investigated the effect and mechanism of E8002 in a rat sciatic nerve adhesion model. A total of 21 rats were used. Six weeks after surgery, macroscopic adhesion scores were significantly lower in the E8002 group (adhesion procedure followed by nerve wrapping with E8002) compared to the E8002 AA(−) group (adhesion procedure followed by nerve wrapping with the E8002 membrane excluding AA) and adhesion group (adhesion procedure but no treatment). Correspondingly, a microscopic examination revealed prominent scar tissue in the E8002 AA(−) and adhesion groups. Furthermore, an in vitro study using human blood samples showed that AA enhanced tissue-type, plasminogen activator-mediated fibrinolysis. Altogether, these results suggest that E8002 may exert an anti-adhesive action via AA and the regulation of fibrinolysis.
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Dissertations / Theses on the topic "Adhesion"

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Ponce, Heredia Suomi. "Adhesion of thin structures : frictional peeling and adhesive shells." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066550/document.

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Dans cette thèse, nous nous intéressons à l’adhésion d’élastomères sur des substrats rigides (interactions de van der Waals). Nous revisitons ainsi, en nous appuyant sur une approche expérimentale, deux situations classiques qui permettent la mesure de l’énergie d’adhésion. Dans une première partie dédiée à l’étude du pelage, nous montrons que le frottement peut modifier le processus de détachement d’une bande d’élastomère. Ceci est tout d’abord mis en évidence lors du pelage parallèlement à l’interface, où nous montrons qu’il conduit à la progression d’une zone de frottement jusqu’au détachement de la bande pour une force proportionnelle à l’aire de contact. Nous généralisons par la suite nos résultats au pelage selon un angle quelconque. En particulier, nous montrons comment la dissipation par frottement peut augmenter de façon significative la force de pelage pour des angles faibles. Dans une deuxième partie, nous nous tournons vers la mesure d’adhésion dans une géométrie JKR, c’est à dire lors de l’indentation d’une sphère élastique adhésive avec un substrat rigide. Nous utilisons des coques élastiques dont la réponse mécanique est beaucoup plus souple bien que plus complexe. Nous montrons qu’il est néanmoins possible d’obtenir une mesure de l’adhésion par une méthode très simple et très robuste, puisqu’elle ne suppose pas de connaissance a priori de la mécanique du système. Nous espérons que cette technique permettra dans le futur d’accéder à des énergies d’adhésion très faibles ou mettant en jeu des tissus biologiques particulièrement mous
In this thesis, we are devoted to study the adhesion of elastomers to rigid substrates through van der Waals interactions. We review, from an experimental point of view, two classical methods to measure the adhesion energy. A first part is dedicated to the study of the peeling system, we show that friction can modify the detachment process of an elastomer strip. This is firstly observed in the lap-test configuration, where a sliding front propagates on the interface up until the end of the strip, for a pulling force proportional to the initial contact area. We generalize this results for the finite peeling angle case. In particular, we show how the friction dissipation significantly increases for small peeling angles. In the second part, we study the adhesion measure in the JKR geometry, i.e. for the indentation of an adhesive elastic sphere into a rigid plate. We use elastic thin shells, which elastic response is much softer, as well it is much more complex. However, it is possible to measure the adhesion energy through a very simple and robust method with no need of much details of the mechanical response of the system. We hope this technique will allow to measure the effect of very weak and sensitive adhesive systems such us biological tissues which are particularly soft
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Muniz, Maisonet Maritza. "Topographical Enhancement of Cell Adhesion on Poorly Adhesive Materials." Scholar Commons, 2015. https://scholarcommons.usf.edu/etd/5748.

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The overall thrust of this dissertation is to gain a fundamental understanding of the synergistic effects between surface topography and chemical functionality of poorly adhesive materials on enhancing the adhesion of mouse embryonic fibroblasts. Cellular response to surface topography and chemical functionality have been extensively studied on their own providing valuable information that helps in the design of new and improved biomaterials for tissue engineering applications. However, there is a lack of understanding of the synergistic effect of microscale and nanoscale topography with chemical functionality and the relative impact and contribution of each in modulating cellular behavior. By understanding the relationship between these cues, in particular using materials that are poorly adhesive, this study will provide new clues as to how cells adapt to their environment and also suggest new dimensions of biomaterial design for fine-tuning cellular control. A microstructure that combined non adhesive materials with defined surface topography and surface chemistry is presented, to assess and correlate the enhancement of mouse embryonic fibroblasts cell adhesion and spreading. Poly (N-isopropylacrylamide) or PNIPAAm electrospun fibers were overlaid on PNIPAAm thin films (100 nm) at various time points to investigate the role of topography on such coatings by keeping the chemical functionality the same. After doing this, several topographical patterns were developed, spanning from sparse to dense fiber mats, and cell adhesion strongly depended on the relative available areas for attachment on either the fibers or the supporting surface. To gain a better understanding of this finding, two surface chemistries, non-adhesive (self-assembled monolayer of polyethylene glycol (PEGSAM) alkanethiol on gold) or an adhesive coating (3-aminopropyltriethoxysilane (APTES) on glass) with well characterized adhesive properties were included in this study to assess the effect of topographical cues provided by the PNIPAAm electrospun fibers on cellular responses. With the deposition of the PNIPAAm fibers onto a PEGSAM surface, cell adhesion increased to almost 100%, and unlike the PNIPAAm surface, cell spreading was significantly enhanced. With the deposition of PNIPAAm fibers onto APTES, both cell adhesion and spreading were unaffected up to 60% fiber coverage. For both surfaces, PNIPAAm fiber densities above 60% coverage lead to adhesion and spreading independent of the underlying surface. These findings indicate the presence of a sparse topographical feature can stimulate cell adhesion on a typically non-adhesive material, and that a chemical dissimilarity between the topographic features and the background enhances this effect through greater cell-surface interaction. In addition to the aforementioned studies, cell response was also assessed on PNIPAAm thin films coatings with thicknesses ranging from 100 nm to 7 nm. Cell adhesion and spreading was enhanced as the thickness of the thin film decreased. This change was more noticeable below 30 nm, wherein 7 nm shows the highest cell adhesion and spreading enhancement. The results reported are preliminary results and further experiments will be conducted, to support the data. It is believed that cellular response was enhanced due to a change in surface topography at the nanoscale level.
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Kessler, Amanda. "Intercoat adhesion : How can intercoat adhesion be studied and what influences intercoat adhesion." Thesis, KTH, Skolan för kemi, bioteknologi och hälsa (CBH), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-230693.

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Coatings cover most objects in our daily lives. They can have functional properties such as protecting the coated material or esthetic properties to decorate and provide pleasant appearance. Coatings have historically dried by evaporation of solvents, but now, UV curing coatings are on the uprising. Acrylate functional groups on polymers, oligomers and monomers react through radical chain polymerization initiated by photoinitiators. Photoinitiators are molecules which forms radicals when irradiated with UV light.Coatings are often applied on multiple layers, each layer providing one function for the overall system. To function properly and have desired life time the layers must adhere to each other. The problem of intercoat adhesion has yet to find good techniques for analysis and explanation of what influences it. Many theories have been suggested and most likely many play a part in the overall adhesion.The master thesis project aim to investigate which properties that influences intercoat adhesion and how it can be studied. To relate properties of coatings to the intercoat adhesion one UV curing primer is set to use for all coating systems, and various topcoats have been produced with slight changes in formulation. The study can be divided into two parts; a pre-study and a main study. The pre-study follows up on a previous master thesis conducted at Sherwin-Williams AB. The pre-study has waterbased and waterbased UV-curable topcoats. The main study has UV curable topcoats. Properties which are studied in the main study are chemical backbone of the binder, functionality of monomers, influence of addition of wetting agents, defoamers, fillers and pigment, viscosity, density, pH, curing degree at depth, surface energy, surface tension, surface polarity and monomer to binder ratio.The pull-off method is the best method of analysis of intercoat adhesion today. A dolly is glued to a surface and then lifted, the force of lifting the dolly is measured. The method has one significant drawback; the break must be completely in the interphase of where the adhesion wishes to be analyzed. Throughout this study most interphase failures were between substrate and primer, resulting in no value for intercoat adhesion.No correlation was found between intercoat adhesion and chemical backbone of the binder, functionality of monomers, addition of wetting agents, defoamers, viscosity, pH, surface energy, surface tension or surface polarity. Lower intercoat adhesion was observed for coatings containing talc, calcium carbonate and titanium dioxide. Coating containing titanium dioxide showed insufficient curing above a coating thickness of 40 μm. The insufficient curing could be observed as wrinkles on the surface and liquid coating remaining in the coating interphase. Curing degree in depth of the topcoat is believed to be the main reason to decreased intercoat adhesion for the coating containing titanium dioxide. The insufficient curing could not be confirmed with infrared spectrometry. The reason why talc and calcium carbonate showed decreased intercoat adhesion is not known. Indications suggest that a lower monomer to binder ratio decrease intercoat adhesion, theories to explain this are the high viscosity and the low number of functional groups per volume. A higher number of functional groups per volume could increase the number of crosslinks formed between topcoat and primer.
Färger finns överallt omkring oss. De kan ha funktionella egenskaper så som att skydda det målade materialet eller ge ett önskvärd utseende. Ett bord ska både tåla att få spillt kaffe på sig och se behagligt ut. Ofta appliceras färger i flera lager för att uppnå önskade egenskaper och utseende. Dessa färglager bör sitta bra i varandra för att inte avflagning ska uppstå. Hur bra färgskikten sitter i varandra kallas mellanskiktsvidhäftning. Mellanskiktsvidhäftning har varit en utmaning genom hela färgindustrins historia och ännu har inga entydiga förklaringar på vad som påverkar eller hur man ska mäta det uppkommit.Kommersiella färger har historiskt varit lösningsmedelsbaserade och torkat. Torkning innebär att flyktiga komponenter i färgen avgår genom att evaporera. I och med moderna försök att minska negativa effekter på miljön har vattenbaserade och helt lösningsmedelsfria färger utvecklats. De lösningsmedelfria färgerna torkar inte utan färgfilmen formas genom en kemisk reaktion initierad med hjälp av ultraviolett (UV) ljus. Ett tidigare examensarbete utfört på Sherwin-Williams AB undersökte mellanskiktsvidhäftningen för en UV härdande grund med vattenbaserade topplacker. Detta projekt är menat som en fortsättning på tidigare projekt men fokuserar huvudsakligen på UV härdande topplacker på en låst UV härdande grund. Syftet med studien är att undersöka hur olika egenskaper hos topplacker påverkar mellanskiktsvidhäftningen samt undersöka hur man kan studera mellanskiktsvidhäftning.Den främsta metoden för att undersöka mellanskiktsvidhäftning konstaterades vara dragprovsmätning, då övriga mätmetoder inte ger entydiga resultat. I dragprovningsmetoden limmas en metallknopp på en yta för att sedan dras av vinkelrätt från ytan och kraften som krävs mäts. Nackdelen med metoden är att för att kunna mäta specifikt mellanskiktvidhäftningen måste brottet ske i gränsskiktet mellan färgerna. Brottet uppstod framförallt mellan substrat och grundfärg under studien.Resultaten från studien kunde inte påvisa ett samband mellan den kemiska strukturen av bindemedlet, antalet funktionella grupper på monomer, tillsatser av vätmedel, skumdämpare, viskositet, densitet, pH, ytenergi, ytspänning, ytpolaritet samt krympning av filmen vid härdning. Resultaten indikerar att tillsats av talk, krita eller titandioxid minskar mellanskiktsvidhäftningen. Anledningen till att talk samt krita minskar mellanskiktsvidhäftningen kan ej förklaras. Titandioxiden påverkade uthärdningen av filmen vilket tros vara anledningen till den minskade mellanskiktsvidhäftningen. Det går att se en indikation att minskat förhållande av vikten monomerer mot vikten bindemedel i färgformuleringen minskade mellanskiktsvidhäftnigen. Detta tros bero på en minskad mängd funktionella akrylatgrupper per volym samt ökad viskositet. Förstudien som använde vattenbaserade och vattenbaserade UV härdande topplacker indikerade en försämrad mellanskiktsvidhäftning för rena vattenbaserade system på UV härdande primer.
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Elineni, Kranthi Kumar. "Regulation of Cell Adhesion Strength by Spatial Organization of Focal Adhesions." Scholar Commons, 2011. http://scholarcommons.usf.edu/etd/3088.

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Cell adhesion to extracellular matrix (ECM) is critical to various cellular processes like cell spreading, migration, growth and apoptosis. At the tissue level, cell adhesion is important in the pathological and physiological processes that regulate the tissue morphogenesis. Cell adhesion to the ECM is primarily mediated by the integrin family of receptors. The receptors that are recruited to the surface are reinforced by structural and signaling proteins at the adhesive sites forming focal adhesions that connect the cytoskeleton to further stabilize the adhesions. The functional roles of these focal adhesions extend beyond stabilizing adhesions and transduce mechanical signals at the cell-ECM interface in various signaling events. The objective of this research is to analyze the role of the spatial distribution of the focal adhesions in stabilizing the cell adhesion to the ECM in relation to cell's internal force balance. The central hypothesis was that peripheral focal adhesions stabilize cell adhesion to ECM by providing for maximum mechanical advantage for resisting detachment as explained by the membrane peeling mechanism. Micropatterning techniques combined with robust hydrodynamic shear assay were employed to test our hypothesis. However, technical difficulties in microcontact printing stamps with small and sparse features made it challenging to analyze the role of peripheral focal adhesions in stabilizing cell adhesion. To overcome this limitation, the roof collapse phenomenon in stamps with small and sparse features (low fill factor stamps) that was detrimental to the reproduction of the adhesive geometries required to test the hypothesis was analyzed. This analysis lead to the valuable insight that the non-uniform pressure distribution during initial contact caused by parallelism error during manual microcontact printing prevented accurate replication of features on the substrate. To this end, the template of the stamp was modified so that it included an annular column around the pattern zone that acted as a collapse barrier and prevented roof collapse propagation into the pattern zone. Employing this modified stamp, the required geometries for the cell adhesion analysis were successfully reproduced on the substrates with high throughput. Adhesive areas were engineered with circular and annular patterns to discern the contribution of peripheral focal adhesions towards cell adhesion strength. The patterns were engineered such that two distinct geometries with either constant adhesive area or constant spreading area were obtained. The significance of annular patterns is that for the same total adhesive area as the circular pattern, the annular pattern provided for greater cell spreading thereby increasing the distance of the focal adhesions from the cell's center. The adhesion strength analysis was accomplished by utilizing hydrodynamic shear flow in a spinning disk device that was previously developed. The results indicate that for a constant total adhesive area, the annular patterns provide for greater adhesion strength by enhancing cell spreading area and providing for greater moment arm in resisting detachment due to shear. The next examination was the effect of the cell's internal force balance in stabilizing the cell adhesion. The working hypothesis was that microtubules provide the necessary forces to resist the tensile forces expressed by the cell contractile machinery, thereby stabilizing cell adhesion. Since microtubule disruption is known to enhance cell contractility, its effect on the cell adhesion strength was examined. Moreover, the force balance in cells was altered by engineering adhesive areas so that the cells were either spherical or completely spread and then disrupted microtubules to understand the significance of the force balance in modulating the cell adhesion strength. The results indicated that disruption of microtubules in cells on adhesive islands resulted in a 10 fold decrease in adhesion strength compared to untreated controls whereas no significant change was observed in completely spread cells between treated and untreated controls. This is in surprising contrast to the previous contractility inhibition studies which indicate a less pronounced regulation of adhesion strength for both micropatterned and spread cells. Taken together, these findings suggest that the internal force balance regulated by cell shape strongly modulates the adhesion strength though the microtubule network. In summary, this project elucidates the role of peripheral focal adhesions in regulating the cell adhesion strength. Furthermore, this study also establishes the importance of the internal force balance towards stabilizing the cell adhesion to the ECM through the microtubule network.
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Zhao, Lee Cheng. "Cell adhesion characterization of adhesive forces and effect of topography /." [Gainesville, Fla.] : University of Florida, 2000. http://etd.fcla.edu/etd/uf/2000/ana7043/LCZhao%5FThesis.pdf.

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Thesis (M.S.)--University of Florida, 2000.
Title from first page of PDF file. Document formatted into pages; contains ix, 79 p.; also contains graphics. Vita. Includes bibliographical references (p. 69-77).
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Fredriksson, Fanny. "Outcome and prevention strategies in peritoneal adhesion formation." Doctoral thesis, Uppsala universitet, Barnkirurgi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-282119.

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Peritoneal adhesions occur in up to 93% of adults after peritoneal trauma during surgery. Most adhesions are asymptomatic but can cause female infertility, small bowel obstruction (SBO) and chronic abdominal pain. Adhesion prophylaxis is needed to reduce the significant morbidity and increased health care costs resulting from peritoneal adhesions. This thesis aims to establish a relevant and reproducible experimental adhesion model to simultaneously study the healing processs and adhesion formation and later to examine whether carbazate-activated polyvinyl alcohol (PVAC), an aldehyde-carbonyl scavenger, can reduce adhesion formation or not; and, in a long-term follow-up, to investigate the incidence of and identify risk factors for adhesive SBO requiring surgical treatment after laparotomy during infancy and to survey the prevalence of self-reported chronic abdominal pain and female infertility. Male Sprague-Dawley rats were subjected to laparotomy, cecal abrasion, and construction of a small bowel anastomosis and examined at various time points after surgery. Early elevation of IL-6, IL-1β and TNF-α concentrations in peritoneal fluid but not in plasma correlate to adhesion formation in this rodent adhesion model, indicating that anti-adhesion treatment should be early, local and not systemic. The animals were treated with either peritoneal instillation of PVAC, or the anastomosis was sutured with PVAC-impregnated resorbable polyglactin sutures. At day 7, bursting pressure of the anastomosis was measured and adhesions were blindly evaluated using Kennedy- and Nair scoring systems. PVAC-impregnated sutures reduced adhesion formation without reducing bursting pressure. Infants who underwent laparotomy between 1976 and 2011 were identified (n=1185) and 898 patients were included with a median follow-up time of 14.7 (range 0.0-36.0) years. The median age at first laparotomy was 6 (range 1.0-365.0) days. There were 113 patients (12.6%) with adhesive SBO, with the highest incidence found in patients with Hirschsprung’s disease (19 of 65, 29%), malrotation (13 of 45, 29%), intestinal atresia (11 of 40, 28%) and necrotizing enterocolitis (16 of 64, 25%). Lengthy duration of surgery (hazard ratio (HR) 1.25, 95% CI, 1.07 to 1.45), stoma formation (HR 1.72, 1.15 to 2.56) and postoperative complications (HR 1.81, 1.12 to 2.92) were independent risk factors. Chronic abdominal pain was reported in 180 (24.0%) of 750 patients, and 17 (13.8%) of 123 women reported infertility. The morbidity after laparotomy in neonates and infants is high. Awareness of the risk factors may promote changes in surgical practice.
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Jordan, Allison Clare. "Modelling platelet adhesion." Thesis, University of Leeds, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.410947.

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Coyer, Sean R. "Modulation of cell adhesion strengthening by nanoscale geometries at the adhesive interface." Diss., Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/34763.

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Cell adhesion to extracellular matrices (ECM) is critical to many cellular processes including differentiation, proliferation, migration, and apoptosis. Alterations in adhesive mechanisms are central to the behavior of cells in pathological conditions including cancer, atherosclerosis, and defects in wound healing. Although significant progress has been made in identifying molecules involved in adhesion, the mechanisms that dictate the generation of strong adhesive forces remain poorly understood. Specifically, the role of nanoscale geometry of the adhesive interface in integrin recruitment and adhesion forces remains elusive due to limitations in the techniques available for engineering cell adhesion environments. The objective of this project was to analyze the role of nanoscale geometry in cell adhesion strengthening to ECM. Our central hypothesis was that adhesive interactions are regulated by integrin clusters whose recruitment is determined by the nanoscale geometry of the adhesive interface and whose heterogeneity in size, spacing, and orientation modulates adhesion strength. The objective of this project was accomplished by 1) developing an experimental technique capable of producing nanoscale patterns of proteins on surfaces for cell adhesion arrays, 2) assessing the regulation of integrin recruitment by geometry of the adhesive interface, and 3) determining the functional implications of adhesive interface geometry by systematically analyzing the adhesion strengthening response to nanoscale patterns of proteins. A printing technique was developed that patterns proteins into features as small as 90nm with high contrast and high reproducibility. Cell adhesion arrays were produced by directly immobilizing proteins into patterns on mixed-SAMs surfaces with a protein-resistant background. Colocalization analysis of integrin recruitment to FN patterns demonstrated a concentrating effect of bound integrins at pattern sizes with areas equivalent to small nascent focal adhesions. At adhesion areas below 333 × 333 nm2, the frequency of integrin recruitment events decreased significantly indicating a threshold size for integrin clustering. Functionally, pattern sizes below the threshold were unable to participate in generation of adhesion strength. In contrast, patterns between the threshold and micron sizes showed a relationship between adhesion strength and area of individual adhesion points, independent of the total available adhesion area. These studies introduce a robust platform for producing nanoscale patterns of proteins in biologically relevant geometries. Results obtained using this approach yielded new insights on the role of nanoscale organization of the adhesive interface in modulating adhesion strength and integrin recruitment.
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Marcus, Hilda. "Adherence of Pseudomonas aeruginosa to perfused tracheal epithelium : adhesin [i.e. adhesion] - receptor interactions /." The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487259580261697.

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Sambasivan, Nadarajah. "The adhesion of an optical adhesive to glass substrates for optoelectronics applications." Thesis, University of Surrey, 2001. http://epubs.surrey.ac.uk/843340/.

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Surface characterisation and failure mechanisms of adhesively bonded glass butt joints were studied. Materials of relevance to the adhesive bonding of fibre optic components were employed: a fine annealed Grade A glass (Schott(TM) BK - 7), and a fast curing epoxy based optical adhesive. The joint behaviour and their durability under adverse environmental conditions were investigated, and the subsequent, failed joint fracture surfaces were examined using XPS, ToF-SIMS and SEM. Surface analysis techniques have been employed to characterise components of the adhesive systems and to interrogate the surface of failed joints with a view to establishing the locus of failure. Joints were immersed in pure water for period of 0 - 270 days at 35°C. Substantial reduction in the bond strength within a few days of water immersion was observed. Also for the joints left in for a prolonged exposure periods (> 100 days) the strength values reached a minimum value. The surface analysis of the joints has indicated a cochoidal brittle fracture through the adhesive in dry conditions, and an interfacial failure for wet joint fractures. Calculations of the polymer thickness on the interfacial surfaces indicated a decrease in overlayer thickness. This reduction reached a constant value for joints left in water for more than 100 days. During this investigation surfaces segregation of minor components of the adhesive such as amine and diluent was also identified by ToF-SIMS and XPS. In order to study this phenomenon further, reformulation of the adhesive has been carried out. Six adhesives were formulated, of which three resins were based on the change in diluent content of the system, three were based on the change in amine concentrations. This work has clearly identified amine segregation only at the interfacially failed joints. It is expected that when the joints cures very fast, even if the formulation contains very high proportion of amine, the segregation is minimal. Also, some evidence of diluent migration at the interfaces was observed during the reformulation studies.
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Books on the topic "Adhesion"

1

Horiuchi, Shin, Nao Terasaki, and Takayuki Miyamae, eds. Interfacial Phenomena in Adhesion and Adhesive Bonding. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-4456-9.

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Itzhak, Kahane, Ofek Itzhak, and Bat-Sheva Seminar Toward Anti-Adhesion Therapy of Microbial Diseases (1996 : Zikhron Ya'akov, Israel), eds. Toward anti-adhesion therapy for microbial diseases. New York: Plenum Press, 1996.

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Allen, K. W. Adhesion 14. Dordrecht: Springer Netherlands, 1990.

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Yona, Simon, and Martin Stacey, eds. Adhesion-GPCRs. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7913-1.

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Savage, Dwayne C., and Madilyn Fletcher, eds. Bacterial Adhesion. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4615-6514-7.

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Allen, K. W., ed. Adhesion 9. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4938-6.

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Metcalf, Brian W., Barbara J. Dalton, George Poste, and Judy Schatz, eds. Cellular Adhesion. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2466-3.

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Behrens, Jürgen, and W. James Nelson, eds. Cell Adhesion. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-540-68170-0.

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Allen, K. W., ed. Adhesion 11. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3433-7.

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Allen, K. W., ed. Adhesion 12. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1349-3.

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Book chapters on the topic "Adhesion"

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Horiuchi, Shin, Nao Terasaki, and Takayuki Miyamae. "Introduction—Interfaces in Adhesion and Adhesive Bonding." In Interfacial Phenomena in Adhesion and Adhesive Bonding, 1–15. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4456-9_1.

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AbstractThe social background of research on adhesion and adhesive bonding is reviewed. Next, the interphase in the adhesive bond is defined, and the multi-scale and hierarchical structures contained in the interface are arranged based on their sizes. Finally, the analytical methodologies this book introduces to elucidate interfacial phenomena in adhesion and adhesive bonding are overviewed.
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Hegemann, Johannes H., and Katja Moelleken. "Chlamydial Adhesion and Adhesins." In Intracellular Pathogens I, 97–125. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817329.ch5.

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Horiuchi, Shin. "Interfacial Phenomena in Adhesion and Adhesive Bonding Investigated by Electron Microscopy." In Interfacial Phenomena in Adhesion and Adhesive Bonding, 113–207. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4456-9_3.

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AbstractUsing electron microscopy techniques described in Chap. 2, we investigate interfacial phenomena in adhesion and adhesive bonding. Polymer–polymer interfaces formed via interdiffusion are visualized and characterized by EFTEM. Fractographic studies using high-resolution SEM investigate entanglements at the polymer–polymer interfaces, and the adhesion mechanism is discussed about the interfacial entanglements. The effect of surface treatments of polymers for adhesion improvement is studied in terms of the surface roughness and the chemical functionality of the adherend created by the surface pretreatments. We then describe the role of chemical interactions between polymers and metals on bonding by the analysis of fracture surfaces by the STEM-replica technique. Bonding mechanisms of adhesive bonding and recently developed direct bonding of metal and plastic are also investigated by STEM-EELS/ELNES and STEM-tomography. Finally, we evaluate the toughness and durability of adhesive joints between metal and carbon fiber reinforced plastics (CFRP) and discuss the durability of the adhesive bonding.
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Watts, J. F. "The Interfacial Chemistry of Adhesion: Novel Routes to the Holy Grail?" In Adhesion, 1–16. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch1.

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Wiegemann, M. "Structure Formation in Barnacle Adhesive." In Adhesion, 143–56. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch10.

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Bakowsky, U., C. Ehrhardt, C. Loehbach, P. Li, C. Kneuer, D. Jahn, D. Hoekstra, and C. M. Lehr. "Adhesion Molecule-Modified Cardiovascular Prostheses: Characterization of Cellular Adhesion in a Cell Culture Model and by Cellular Force Spectroscopy." In Adhesion, 157–73. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch11.

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Legeay, G., and F. Poncin-Epaillard. "Surface Engineering by Coating of Hydrophilic Layers: Bioadhesion and Biocontamination." In Adhesion, 175–88. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch12.

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Mülhaupt, R. "New Resins and Nanosystems for High-Performance Adhesives." In Adhesion, 189–203. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch13.

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Hartwig, A., K. Koschek, and A. Lühring. "Influence of Proton Donors on the Cationic Polymerization of Epoxides." In Adhesion, 205–16. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch14.

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Buchman, A., H. Dodiuk-Kenig, T. Brand, Z. Gold, and S. Kenig. "Novel Adhesion Promoters Based on Hyperbranched Polymers." In Adhesion, 217–28. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607307.ch15.

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Conference papers on the topic "Adhesion"

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Gallant, Nathan D., and Kranthi Kumar Elineni. "Regulation of Adhesion Strength by Focal Adhesion Position and Cell Shape." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80832.

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Cell adhesion to extracellular matrices is critical to numerous cellular functions and is primarily mediated by integrin receptors. Binding and aggregation of integrins leads to the formation of focal adhesions (FA) which connect the cytoskeleton to the extracellular matrix in order to reinforce adhesion and transmit signals [1]. Preliminary observations indicated preferential recruitment of FAs to the periphery of the cell spreading area on both uniformly coated and micropatterned fibronectin surfaces (Fig. 1). The current study investigates the biophysical regulation of cell adhesion strength based on the size and position of FA with the central hypothesis that peripheral FAs stabilize adhesion strength. The hypothesis was tested by delineating the cell spreading area from the total cell adhesive area by employing microcontact printing to pattern substrates with a series of circular and annular adhesive islands which control cell shape (Fig. 2). A well characterized hydrodynamic shear assay known as the spinning disk device was used to quantify the adhesion strength of cells adhered to the micropatterns [2].
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Soto, D., A. Parness, N. Esparza, T. W. Kenny, K. Autumn, and M. Cutkosky. "MICROFABRICATED DRY ADHESIVE DISPLAYING FRICTIONAL ADHESION." In 2008 Solid-State, Actuators, and Microsystems Workshop. San Diego: Transducer Research Foundation, 2008. http://dx.doi.org/10.31438/trf.hh2008.36.

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Chirasatitsin, Somyot, Priyalakshmi Viswanathan, Giuseppe Battaglia, and Adam J. Engler. "Directing Stem Cell Fate in 3D Through Cell Inert and Adhesive Diblock Copolymer Domains." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14442.

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Adhesions are important cell structures required to transduce a variety of chemical and mechanics signals from outside-in and vice versa, all of which regulate cell behaviors, including stem cell differentiation (1). Though most biomaterials are coated with an adhesive ligand to promote adhesion, they do not often have a uniform distribution that does not match the heterogeneously adhesive extracellular matrix (ECM) in vivo (2). We have previously shown that diblock copolymer (DBC) mixtures undergo interface-confined de-mixing to form nanodomins of one copolymer in another (3). Here we demonstrate how diblock copolymer mixtures can be made into foams with nanodomains to better recapitulate native ECM adhesion regions and influence cell adhesion.
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Xu, Xi, Taotao Shui, and Yan Jiao. "Adhesive force and adhesion energy of particle-substrate detachment." In Third International Conference on Optoelectronic Science and Materials (ICOSM 2021), edited by Siting Chen and Pei Wang. SPIE, 2021. http://dx.doi.org/10.1117/12.2619740.

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Liu, D. L., J. Martin, and N. A. Burnham. "Optimum Roughness for Minimum Adhesion." In STLE/ASME 2008 International Joint Tribology Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/ijtc2008-71192.

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Surface roughness has a significant affect on adhesion. We used a single-asperity model to describe a smooth tip in contact with a rough surface and predicted that an optimal size of asperity will yield a minimum of adhesion. Experimentally, adhesive forces on silicon wafers with varying roughness were measured using AFM cantilevers with varying tip radii. It was found that minima do exist, and for all tip radii, the adhesion falls significantly for roughness greater than 1–2 nm and drops at higher roughness for larger tips. In addition to RMS roughness, the roughness exponent is another important parameter for the characterization of rough surfaces and its affect on adhesion was also investigated. We developed computer programs to simulate a set of fractal rough surfaces with differing roughness exponents. The adhesive forces between an AFM tip and the fractal surfaces were calculated and the adhesion was seen to decrease as the roughness exponent increases. This work should help minimize MEMS stiction and progress the understanding of nanoscale contact mechanics.
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Slater, John H., Jordan S. Miller, Shann S. Yu, and Jennifer L. West. "Multifaceted Nano- and Micropatterned Surfaces for Cell Adhesion Manipulation." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13177.

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Surfaces displaying nano- and micropatterned cell adhesive ligands have led to numerous discoveries in cell biology. Soft lithography techniques such as microcontact printing are well suited for creating surfaces displaying micropatterns of one ligand type in a single arrangement but are difficult to implement for the creation of multifaceted surfaces that present multiple ligand types with each ligand confined to their own pattern. To better understand the influence of extracellular matrix (ECM) composition on adhesion site formation and gross cell behavior (motility, proliferation, differentiation, etc.) it would be advantageous to posses the ability to create surfaces displaying multiple patterned ligands with length scales ranging from < 0.25 μm2, the typical size of a focal complex to > 1 μm2, the size of focal adhesions. Higher spatial resolution than what is easily achieved with microcontact printing is also desired. Such surfaces would allow for the simultaneous investigations of adhesion site maturation and composition and how changes in these properties can be implemented to engineer cell behavior via cell-surface interactions.
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Li, Yang, Hao Zhang, Geng Xu, Ling Gong, Zhenzhong Yong, Qinwen Li, and Zhendong Dai. "Adhesion performance of gecko-inspired flexible carbon nanotubes dry adhesive." In SPIE Smart Structures and Materials + Nondestructive Evaluation and Health Monitoring, edited by Raúl J. Martín-Palma and Akhlesh Lakhtakia. SPIE, 2013. http://dx.doi.org/10.1117/12.2012009.

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Suthisomboon, Tachadol, Thatthep Rukpanich, Naris Asawalertsak, Pongsiri Borijindakul, Aihong Ji, Zhendong Dai, and Poramate Manoonpong. "VENOM: Versatile, Adhesive, and Soft Material for Various Surface Adhesion." In 2021 IEEE 4th International Conference on Soft Robotics (RoboSoft). IEEE, 2021. http://dx.doi.org/10.1109/robosoft51838.2021.9479242.

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Charles, G., and R. Goodall. "Low adhesion estimation." In IET International Conference on Railway Condition Monitoring. IEE, 2006. http://dx.doi.org/10.1049/ic:20060051.

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Almirall, L., J. Aznar-Salatti, I. Calopa, A. Ordinas, and E. Bastida. "ADHESION OF TUMOR CELLS TO EXTRACELLULAR MATRIX IS MEDIATED BY FIBRONECTIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643207.

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Tumor cell (TC) vessel wall adhesion is thougth to occur at specific sites of exposed extracellular matrix (ECM).To determine the role of fibronectin (FN) in TC/ECM adhesion,we measured; 1)TC adhesion to intact cultured endothelial cell monolayers (EC) or their ECMs,with or without incubation with a polyclonal antibody (Ab) to human FN, or a monoclonal antibody (Mab) to the cell binding site of FN (3E3);2)TC adhesion to EC or their exposed ECMs incubated with specific peptides against bacteria adhesion sites (I 133-79) or the peptide GRGDSP contained in the cell binding sites of several adhesive proteins.TC adhesion was measured as number of 111In-labeled A-549 adenocarcinoma cellsxlO /disc. ECMs were exposed by removing the ECs with N2 flow or EGTA treatment.There was 5.5±1x10 A-549/ EC covered disc (Table). Treatment of ECs with the Abs or peptides had no effect on TC adhesion.TC adhesion to the two ECM preparation were 42 ± 12 and 55±10 respectively.(Table).Blockage of the FN adhesive site by either Ab inhibited TC adhesion (p < 0.01). In contrast,blockage of the bacteria-adhesion site or incubation with GRGDSP had not significant effects in TC adhesion. The table shows the results (mearv±SEM (* p < 0.01).We conclude that TC adhesion to ECM but not to ECs,is dependent upon FN.
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Reports on the topic "Adhesion"

1

Sun, X. S., Donghai Wang, and Zhikai Zhong. Adhesion Performance of Modified Soy Protein Adhesive. Fort Belvoir, VA: Defense Technical Information Center, January 2001. http://dx.doi.org/10.21236/ada414303.

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Bonk, Robert B., John F. Osterndorf, Brenda L. Pettenger, and Annette M. Ambrosio. Methods for Evaluating Adhesive Systems and Adhesion. Fort Belvoir, VA: Defense Technical Information Center, August 1996. http://dx.doi.org/10.21236/ada312617.

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Marín Boyero, Adrián, and Miguel Angel Rodriguez Valverde. Shear adhesion of a sessile drop under a centrifugal field. Fundación Avanza, May 2023. http://dx.doi.org/10.60096/fundacionavanza/1542022.

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Jo, Hyungyung, Hyeyoung Son, Mitchell Rencheck, Jared Gohl, Devin Madigan, Hugh Grennan, Matthew Giroux, Trevor Thiele-Sardina, Chelsea S. Davis, and Kendra A. Erk. Mechanical Properties of Durable Pavement Marking Materials and Adhesion on Asphalt Surfaces. Purdue University, 2022. http://dx.doi.org/10.5703/1288284317357.

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Mechanical properties of commercially available temporary pavement marking (TPM) tapes and thermoplastic materials used as permanent pavement markings (PPM) were investigated using the non-destructive Tape Drape Test and conventional mechanical testing. The impact of temperature and aging on the adhesion of TPM tapes and thermoplastic PPM applied to asphalt core surfaces with various surface roughness and treatments was determined using a modular peel fixture and shear adhesion tests. The adhesion of TPM tapes to model smooth surfaces decreased as surface temperature was increased from 0 to 40°C (32 to 104°F). For some tapes, reduced adhesion and brittle broken fracture were observed at the lowest investigated temperature of -20°C (-4°F). The adhesion of tapes applied to asphalt decreased significantly within 1 week of aging at -25°C (-13°F). Ghost markings were more likely at higher aging temperatures. For PPM thermoplastics, better adhesion to asphalt was observed for higher application temperatures and rougher surfaces. Asphalt emulsion treatments reduced the adhesion of thermoplastics and increased the likelihood of adhesive failure after 5 months of aging at -25°C (-13°F). More ductile PPM thermoplastic materials had better adhesion to both smooth and rough asphalt surfaces compared to thermoplastic materials with a more brittle mechanical response.
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Khanolkar, Amey, Kiyo Fujimoto, and Michael McMurtrey. Laser-based adhesion strength measurements for advanced manufactured sensor adhesion characterization. Office of Scientific and Technical Information (OSTI), July 2021. http://dx.doi.org/10.2172/1813576.

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Patterson, James E. Molecular Basis of Adhesion. Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada567127.

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Gaughen, C. D. Quantifying Sound Coating Adhesion. Fort Belvoir, VA: Defense Technical Information Center, March 2000. http://dx.doi.org/10.21236/ada380878.

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8

Duncan, B., and W. R. Broughton. Characterising strength of adhesion. National Physical Laboratory, June 2023. http://dx.doi.org/10.47120/npl.mgpg72.

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Gorman, D., C. Green, F. Booth-Downs, M. Gee, and T. Fry. Rockwell indentation test for evaluation of adhesion of coatings - aka Daimler-Benz adhesion test. National Physical Laboratory, October 2022. http://dx.doi.org/10.47120/npl.mat107.

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Sabau, Adrian, Harry Meyer III, Jiheon Jun, Jianlin Li, and Donovan Leonard. Laser-Interference Surface Preparation for Enhanced Coating Adhesion and Adhesive Joining of Multi-Materials. Office of Scientific and Technical Information (OSTI), October 2021. http://dx.doi.org/10.2172/1826028.

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