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Relevant bibliographies by topics / Adenoviral; Nervous system; Central

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Academic literature on the topic 'Adenoviral; Nervous system; Central'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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Journal articles on the topic "Adenoviral; Nervous system; Central"

1

Parks, Robin J., and Jonathan L. Bramson. "Adenoviral vectors: prospects for gene delivery to the central nervous system." Gene Therapy 6, no. 8 (August 1999): 1349–50. http://dx.doi.org/10.1038/sj.gt.3301013.

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Davidson, Beverly L., Edward D. Allen, Karen F. Kozarsky, James M. Wilson, and Blake J. Roessler. "A model system for in vivo gene transfer into the central nervous system using an adenoviral vector." Nature Genetics 3, no. 3 (March 1993): 219–23. http://dx.doi.org/10.1038/ng0393-219.

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Schwartz, Kevin L., Susan E. Richardson, Daune MacGregor, Sanjay Mahant, Kamini Raghuram, and Ari Bitnun. "Adenovirus-Associated Central Nervous System Disease in Children." Journal of Pediatrics 205 (February 2019): 130–37. http://dx.doi.org/10.1016/j.jpeds.2018.09.036.

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Zou, Linglong, Heshan Zhou, Lucio Pastore, and Keyi Yang. "Prolonged Transgene Expression Mediated by a Helper-Dependent Adenoviral Vector (hdAd) in the Central Nervous System." Molecular Therapy 2, no. 2 (August 2000): 105–13. http://dx.doi.org/10.1006/mthe.2000.0104.

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Vincent, Arnaud J. P. E., Maria del C. Esandi, Cees J. J. Avezaat, Charles Vecht, Peter Sillevis Smitt, van Bekkum Dirk W., Dinko Valerio, Peter M. Hoogerbrugge, and Abraham Bout. "Preclinical Testing of Recombinant Adenoviral Herpes Simplex Virus-Thymidine Kinase Gene Therapy for Central Nervous System Malignancies." Neurosurgery 41, no. 2 (August 1, 1997): 442–52. http://dx.doi.org/10.1097/00006123-199708000-00023.

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Betz, A. Lorris, Guo-Yuan Yang, and Beverly L. Davidson. "Attenuation of Stroke Size in Rats Using an Adenoviral Vector to Induce Overexpression of Interleukin-1 Receptor Antagonist in Brain." Journal of Cerebral Blood Flow & Metabolism 15, no. 4 (July 1995): 547–51. http://dx.doi.org/10.1038/jcbfm.1995.68.

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Adenoviruses have been proposed as potential vectors for gene therapy in the central nervous system, but there are no reports of their use in the treatment of a brain disease. Because central administration of interleukin-1 receptor antagonist protein (IL-1ra) reduces ischemic brain damage, we determined whether a recombinant adenovirus vector carrying the human IL-1ra cDNA (Ad.RSV IL-1ra) could be used to ameliorate brain injury in permanent focal ischemia. Groups of six rats received intraventricular injections of Ad.RSV IL-1ra or a control adenovirus containing the Escherichia coli β-galactosidase gene (Ad.RSV lacZ). Histochemical staining for β-galactosidase 5 days after virus injection indicated that transgene expression was confined primarily to the cells lining the ventricle. The concentrations of IL-1ra were fivefold to 50-fold higher in the Ad.RSV IL-1ra-injected animals, achieving levels of 9.1 ± 3.3 ng/g in brain and 23.7 ± 22.5 ng/ml in CSF. In these animals, cerebral infarct volume resulting from 24 h of permanent middle cerebral artery occlusion was reduced 64%. These studies demonstrate that adenoviral vectors can be used to deliver genes that attenuate brain injury.

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Shen, J.-S., X.-L. Meng, T. Ohashi, and Y. Eto. "Adenovirus-mediated prenatal gene transfer to murine central nervous system." Gene Therapy 9, no. 12 (June 2002): 819–23. http://dx.doi.org/10.1038/sj.gt.3301700.

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Huang, Yhu-Chering, Sun-Lin Huang, Shih-Perng Chen, Ya-Ling Huang, Chung-Guei Huang, Kuo-Chien Tsao, and Tzou-Yien Lin. "Adenovirus infection associated with central nervous system dysfunction in children." Journal of Clinical Virology 57, no. 4 (August 2013): 300–304. http://dx.doi.org/10.1016/j.jcv.2013.03.017.

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Viola, John J., Zvi Ram, Stuart Walbridge, Eric M. Oshiro, Bruce Trapnell, Jung-Hwa Tao-Cheng, and Edward H. Oldfield. "Adenovirally mediated gene transfer into experimental solid brain tumors and leptomeningeal cancer cells." Journal of Neurosurgery 82, no. 1 (January 1995): 70–76. http://dx.doi.org/10.3171/jns.1995.82.1.0070.

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✓ Among the appealing features of adenoviruses as vectors for transfer of genes into the central nervous system (CNS) are that they are not neurotoxic, they can accommodate the insertion of several large genes, they are not associated with the hazards of insertional mutagenesis, and they can be concentrated to a high-titer preparation. The authors evaluated the feasibility of using adenovirally mediated gene transfer into cultured human glioma cells and in rat models of solid brain tumors and meningeal cancer. Replication-deficient adenoviral vector particles carrying a nuclear-localizing lacZ gene were injected into established 9L cerebral gliomas in Fischer rats. In addition, the adenoviral vector was injected into the subarachnoid space, either simultaneously with intrathecal tumor inoculation or after establishing leptomeningeal cancer. The brains and spinal cords were removed at various intervals for histochemical evaluation for β-galactosidase activity using X-Gal staining. Additional rats received a stereotactic intracerebral injection of the vector into normal brain. No clinical abnormalities were observed in the injected rats. Injection of the adenoviral vector into normal brain resulted in diffuse transduction of astrocytes, microglia, neurons, and endothelial cells at the injection site. Injection of a high-concentration vector preparation into cerebral gliomas resulted in effective tumor transduction. Intrathecal injection of the vector in rats with meningeal cancer resulted in transduction of the infiltrating tumor in the subarachnoid space when injections were given simultaneously with, or 7 days after, tumor inoculation. Transduction rates of both solid and leptomeningeal tumors correlated with the number of injected particles. These results suggest that adenoviral vectors can efficiently transduce solid brain tumors and that the vectors survive in the cerebrospinal fluid for a sufficient period of time to allow leptomeningeal tumor transduction. Adenoviral vector should be evaluated for its potential use in therapeutic gene transfer approaches in malignancies of the CNS.

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Boulis, Nicholas M., Vikas Bhatia, Theodore I. Brindle, Harland T. Holman, Daniel J. Krauss, Mila Blaivas, and Julian T. Hoff. "Adenoviral nerve growth factor and β-galactosidase transfer to spinal cord: a behavioral and histological analysis." Journal of Neurosurgery: Spine 90, no. 1 (January 1999): 99–108. http://dx.doi.org/10.3171/spi.1999.90.1.0099.

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Object. The present study characterizes the time course and loci of gene expression induced by the administration of adenoviral vectors into spinal cord. Although a marked inflammatory response to these vectors occurred, no effect on spinal cord function was seen in the 1st postoperative week. The expression of transgenic genes delivered by viral vectors is being exploited throughout the nervous system. The present study utilized adenoviral vectors containing the Rous sarcoma virus (RSV) promoter and a nuclear localization signal to achieve transgenic expression in mammalian spinal cord. Methods. Initial experiments utilizing the vector Ad.RSVlacZ (1012 particles/ml) injected into the region of the central canal resulted in viral gene expression stretching over approximately 1.2 cm of spinal cord. Gene expression was first detected 3 days following viral administration and lasted until postinjection Day 14 with peak expression at Day 7. A variety of cell types in both white and gray matter expressed lacZ. Transgenic expression of the neurotrophin nerve growth factor (NGF) was achieved using injections of Ad.RSVNGF. On histological examination mononuclear inflammatory infiltrate and gliosis were revealed surrounding the injection sites of spinal cords receiving adenovirus but not vehicle. To assess spinal cord function during viral gene expression, animals previously trained in an operant runway task were tested at 7 days postinjection (the peak of viral gene expression) and demonstrated no changes in spinal cord function. Conclusions. Results of this study using adenoviral neurotrophic gene transfer indicate that it provided an effective tool for the delivery of potentially therapeutic proteins to the injured or diseased spinal cord.

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Dissertations / Theses on the topic "Adenoviral; Nervous system; Central"

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Regardsoe, Emma Louise. "An investigation into the role of Fas ligand as a potential immunomodulatory molecule for CNS gene therapy." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365794.

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Zheng, Luyu. "The role of Coxsackie and Adenovirus receptor in the central nervous system." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=86782.

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The Coxsackie and adenovirus receptor (CAR), a newly described regulator of neurite outgrowth in the developing central nervous system (CNS), is an adhesion molecule of the immunoglobulin superfamily. CAR is highly expressed in the developing brain, but its level is down-regulated in adult nervous system. Recently, CAR has been demonstrated to mediate homophilic cell-cell adhesion in tumor cell-lines and to be a transmembrane component of the tight junction in epithelial cells. We observed that neurons grown in the presence of soluble CAR have longer neurites compared to BSA control. Furthermore, the presence of soluble CAR was able to overcome the inhibitory effect of cytokines (TNFα) and restore the neurite length to control level. In addition, using hippocampal neuron cultures prepared from CAR embryos in which exon 2 of CAR is flanked by LoxP sites ("CAR-flox"), and infected with the adenovirus AdV-CRE-GFP or control vector (AdV-BFP), we demonstrated that both neuron survival and neurite length were affected by knockown of CAR expression. It has been shown that general knockout of CAR results in embryonic lethality; by breeding CAR-floxed mice (a generous gift of Jeffrey Bergelson, U. Pennsylvania) to synapsin-1-Cre transgenic mice we have developed conditional knockout mice in which CAR is deleted specifically in the nervous system. Here, we report some morphological alterations observed in animals lacking CAR in the CNS. The results of this study will further our understanding of the role CAR plays in developmental processes in the brain.
Le récepteur du coxsackievirus et de l'adénovirus (CAR) est une molécule d'adhérence de la famille immunoglobuline. CAR est exprimée dans le cerveau avant la naissance, mais le niveau est réduit dans le cerveau adulte. Récemment, il a été montré que CAR est nécéssaire pour l'adhérence entre les cellules tumorales, et qu'il est aussi un composant de la jonction transmembranaire des cellules épithéliales. Nous avons observé que les neurones mis en culture en présence d'une forme soluble de CAR dévelopent des prologements cellulaires plus longs comparés aux neurones qui sont en présence de BSA. En outre, la présence de la forme soluble de CAR neutralise l'effet inhibiteur du cytokine TNFα. En utilisant des neurones hippocampiques préparé à partir d'embryons CAR dans lequel l'exon 2 de CAR est flanqué de sites loxP (CARFLOX), et infecté par l'adénovirus AdV-CRE-GFP ou vecteur contrôle (AdV-BFP), nous avons démontré que la survie des neurones et la longueur des neurites ont été affectés par la diminution de l'expresssion de CAR. Nouse avons aussi croisé les CARFLOX avec des souris transgéniques exprimant la recombinase Cre sous le contrôle du promoteur de la synapsine I afin de produire des souris dans lesquelles l'expression de CAR serait en baisse spécifiquement dans le système nerveux central. Nous rapportons ici quelques altérations morphologiques observées chez ces animaux. Les résultats de cette étude permettra d'approfondir notre compréhension du rôle que CAR joue dans les processus de développement du cerveau.

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Gonzalez, Sarah Charlotte. "An investigation into retrograde transport of adenovirus vectors in the central nervous system." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398162.

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Hornsey, Mark Alan. "Adenovirus-mediated delivery of transgenes to both the central nervous system and peripheral targets : potential and problems." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393405.

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Nunes, Rafaella Almeida Lima. "Aplicação de técnicas moleculares no diagnóstico laboratorial complementar das infecções virais do sistema nervoso central no Hospital Universitário da USP." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-19032014-160513/.

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Enterovírus (HEV), herpesvírus 1 e 2 (HHV-1 e HHV-2) e adenovírus (HAdV) são importantes agentes de infecções do SNC. Neste trabalho, técnicas moleculares foram aplicadas para a detecção destes vírus em quadros de infecção do SNC. Amostras de líquor foram colhidas de pacientes atendidos no HU-USP entre agosto e novembro/2010 e fevereiro/2012 a janeiro/2013. Através da Nested-PCR HEV foram detectados em 9,8% das amostras, HAdV em 2,5% e HHV-1 e 2 em 1,1%, além de 3 casos de coinfecção, 2 entre HEV e HHV, e 1 entre HEV e HAdV. O material genético viral foi extraído através dos métodos Qiaamp DNA Blood (Qiagen®) e MagMAXTM Viral RNA Isolation (Ambiom), e este último pareceu mais adequado à aplicação na rotina clínica. A análise quimiocitológica do líquor mostrou-se importante no direcionamento da conduta clínica, mas a detecção do vírus é fundamental para a conclusão do diagnóstico. A PCR em tempo real, cuja padronização foi iniciada neste trabalho, consiste em importante ferramenta para a utilização futura no diagnóstico complementar das infecções virais do SNC.
Enteroviruses (HEV), herpesviruses 1 and 2 (HHV-1 and HHV-2) and adenoviruses (HAdV) are important causative agents of infections of the CNS. In this study, molecular techniques were applied to the detection of these viruses. CSF samples were collected from patients treated at the University Hospital of USP, between August and November, 2010, and February 2012 and January 2013. By the Nested-PCR reaction, HEV were detected in 9.8% of the samples, HAdV in 2.5% and HHV-1 and 2 in 1.1%. There were 3 cases of coinfection: 2 with HEV and HHV and other with HEV and HAdV. The viral genetic materials were extracted by QIAamp DNA Blood kit (Qiagen®) and MagMAXTM Viral RNA Isolation (Ambiom), and the second one showed to be more suitable for the application in clinical diagnosis. The CSF chemocytologic analysis proved to be important in directing the clinical conduct, but the detection of viruses is essential for the diagnosis. The real time PCR, which standardization was initiated in this work, will be an important tool for complementary diagnosis of viral infections of the CNS.

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Solomon, Thomas. "Central nervous system infections in Vietnam." Thesis, Open University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340736.

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Zhang, Hui. "Remyelination in the central nervous system." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/8095.

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Multiple Sclerosis (MS) is an inflammatory disease which causes areas of demyelination in the Central Nervous System (CNS) and affects only humans. Current therapies for MS are focused on anti-inflammatory treatment, which reduce the occurrence and clinical relapses of the disease. However, progressive disability of the disease is related to axonal degeneration. After demyelination, remyelination occurs, which helps repair the demyelinated lesions and protects axons from degeneration. However, this endogenous remyelination is inefficient, and currently there are no therapies available to enhance remyelination. The aim of this thesis was to first characterize a fast and reliable model to study CNS remyelination in vitro, and second to investigate the role of semaphorin 3a (Sema3A) and semaphorin 3f (Sema3F) signaling in CNS remyelination. Various in vivo models have been developed to investigate the pathology of multiple sclerosis, and can be used to test remyelination therapies. However, in vivo models are expensive, animal- and time- consuming. Until now, there has been no well-characterized and robust in vitro model for remyelination study. In this thesis, an ex vivo slice culture system with mouse brain and spinal cord was developed, and characterized by immunofluorescent microscopy and transmission electron microscopy, for CNS remyelination study. Automated (re)myelinating quantification by image pro plus software was developed and validated to provide a fast and reliable way for testing factors that change remyelination efficiency. Two such factors are Sema3A and 3F, which were initially identified as axon guidance cues during development. Sema3A (repulsive) and 3F (attractive) were proved to play a role in oligodendrocyte precursor cell (OPC) migration during development, and hypothesized to be important in remyelination. In this thesis, I investigated the effects and mechanisms for this by adding recombinant SEMA3A or SEMA3F or by knockdown their obligatory receptors Neuropilin (Nrp) 1 and 2, using lentivirus induced miRNAi. Slice culture and primary OPC culture were used to determine the effect on OPC survival, migration, proliferation, differentiation and myelination.

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Poland, Stephen D. "Central nervous system infection with human cytomegalovirus." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21311.pdf.

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Hüppi, Petra Susan. "Serum antibodies to central nervous system antigens /." [S.l : s.n.], 1986. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Bernick, Kristin Briana. "Cell biomechanics of the central nervous system." Thesis, Massachusetts Institute of Technology, 2011. http://hdl.handle.net/1721.1/67202.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Biological Engineering, 2011.
Cataloged from PDF version of thesis.
Includes bibliographical references (p. 133-153).
Traumatic brain injury (TBI) is a significant cause of death and morbidity in both the civilian and military populations. The major causes of TBI, such as motor vehicle accidents, falls, sports concussions, and ballistic and explosive blast threats for military personnel, are well established and extensively characterized; however, there remains much to be learned about the specific mechanisms of damage leading to brain injury, especially at the cellular level. In order to understand how cells of the central nervous system (CNS) respond to mechanical insults and stimuli, a combined modeling/experimental approach was adopted. A computational framework was developed to accurately model how cells deform under various macroscopically imposed loading conditions. In addition, in vitro (cell culture) models were established to investigate damage responses to biologically relevant mechanical insults. In order to develop computational models of cell response to mechanical loading, it is essential to have accurate material properties for all cells of interest. In this work, the mechanical responses of neurons and astrocytes were quantified using atomic force microscopy (AFM) at three different loading rates and under relaxation to enable characterization of both the elastic and viscous components of the cell response. AFM data were used to calibrate an eight-parameter rheological model implemented in the framework of a commercial finite element package (Abaqus). Model parameters fit to the measured responses of neurons and astrocytes provide a quantitative measure of homogenized nonlinear viscoelastic properties for each cell type. In order to ensure that the measured responses could be considered representative of cell populations in their physiological environment, cells were also grown and tested on substrates of various stiffness, with the softest substrate mimicking the stiffness of brain tissue. Results of this study showed both the morphology and measured force response of astrocytes to be significantly affected by the stiffness of their substrate, with cells becoming increasingly rounded on soft substrates. Results of simulations suggested that changes in cell morphology were able to account for the observed changes in AFM force response, without significant changes to the cell material properties. In contrast, no significant changes in cell morphology were observed for neurons. These results highlight the importance of growing cells in a biologically relevant environment when studying mechanically mediated responses, such as TBI. To address this requirement, we developed two model systems with CNS cells grown in soft, 3D gels to investigate damage arising from dynamic compressive loading and from a shock pressure wave. These damage protocols, coupled with the single cell computational models, provide a new tool set for characterizing damage mechanisms in CNS cells and for studying TBI in highly controllable in vitro conditions.
by Kristin Briana Bernick.
Ph.D.

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Books on the topic "Adenoviral; Nervous system; Central"

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Central nervous system. Cambridge: Cambridge University Press, 2009.

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Central nervous system angiitis. Boston: Butterworth-Heinemann, 2000.

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Central nervous system infections. Philadelphia, Pennsylvania: Elsevier, 2013.

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Emerich, Dwaine F., Reginald L. Dean, and Paul R. Sanberg, eds. Central Nervous System Diseases. Totowa, NJ: Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-691-1.

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Ahluwalia, Manmeet, Philippe Metellus, and Riccardo Soffietti, eds. Central Nervous System Metastases. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-23417-1.

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Ramakrishna, Rohan, Rajiv S. Magge, Ali A. Baaj, and Jonathan P. S. Knisely, eds. Central Nervous System Metastases. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42958-4.

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Kryzhanovsky, G. N. Central Nervous System Pathology. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-7870-9.

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Allen, Deborah Hutchinson, and Laurie L. Rice. Central nervous system cancers. Pittsburgh, Pa: Oncology Nursing Society, 2010.

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1933-, Voogd J., and Huijzen Chr van, eds. The human central nervous system. 4th ed. New York: Springer, 2008.

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Lacruz, César R., Javier Saénz de Santamaría, and Ricardo H. Bardales. Central Nervous System Intraoperative Cytopathology. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98491-9.

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Book chapters on the topic "Adenoviral; Nervous system; Central"

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Wirth, Thomas, Haritha Samaranayake, and Seppo Ylä-Herttuala. "Glioblastoma Multiforme: Use of Adenoviral Vectors." In Tumors of the Central Nervous System, Volume 2, 335–47. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0618-7_33.

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Kranzler, Justin, Matthew A. Tyler, Ilya V. Ulasov, and Maciej S. Lesniak. "Intracranial Glioma: Delivery of an Oncolytic Adenovirus." In Tumors of the Central Nervous System, Volume 1, 365–70. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0344-5_38.

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Sivasubramaniam, S. D., A. R. Fooks, J. Lee, G. Stacey, and A. D. Jennings. "Neurological Therapy — Adenovirus Mediated Gene Therapy in Cells of the Central Nervous System." In Animal Cell Technology, 51–56. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5404-8_9.

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Tomioka, Ryohei. "Expression of EGFP by Adenovirus-Mediated Gene Transfer in the Central Nervous System." In Methods in Molecular Biology, 97–106. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-559-6_6.

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Timperley, W. R., J. M. MacKenzie, and S. F. D. Robinson. "Central nervous system." In Reporting Histopathology Sections, 366–79. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-7132-6_23.

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Schulz, Volker, Rudolf Hänsel, and Varro E. Tyler. "Central Nervous System." In Rational Phytotherapy, 37–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-97704-6_2.

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Noggle, Chad A., and Jennifer N. Hall. "Central Nervous System." In Encyclopedia of Child Behavior and Development, 319–20. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_488.

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Klingensmith, William C. "Central Nervous System." In The Mathematics and Biology of the Biodistribution of Radiopharmaceuticals - A Clinical Perspective, 161–75. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26704-3_13.

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Yu, Yao, Steve E. Braunstein, Daphne A. Haas-Kogan, and Jean L. Nakamura. "Central Nervous System." In Handbook of Evidence-Based Radiation Oncology, 37–105. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-62642-0_2.

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Farina, Patrizia, Florian Scotté, Chiara Villa, Bertrand Baussart, and Anna Luisa Di Stefano. "Central Nervous System." In Side Effects of Medical Cancer Therapy, 213–47. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70253-7_7.

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Conference papers on the topic "Adenoviral; Nervous system; Central"

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Henoch-Schönlein Purpura, A., Gabriele Simonini, Eleonora Fusco, Ilaria Maccora, Anna Rosati, Rolando Cimaz, and Teresa Giani. "AB1015 CENTRAL NERVOUS SYSTEM VASCULITIS PRECEDING." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4663.

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Guck, Jochen R. "Optomechanical insights into the central nervous system." In Optical Elastography and Tissue Biomechanics VIII, edited by Kirill V. Larin and Giuliano Scarcelli. SPIE, 2021. http://dx.doi.org/10.1117/12.2578567.

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Barač, Anja, Ivona Jerković, and Petra Nimac Kozina. "Primary angiitis of the central nervous system (PACNS)." In NEURI 2015, 5th Student Congress of Neuroscience. Gyrus JournalStudent Society for Neuroscience, School of Medicine, University of Zagreb, 2015. http://dx.doi.org/10.17486/gyr.3.2223.

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Hartwell, Peter. "CeNSE: A central nervous system for the earth." In 2011 IEEE Technology Time Machine (TTM). IEEE, 2011. http://dx.doi.org/10.1109/ttm.2011.6005162.

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Shahidi, Ghavam G. "CeNSE: A central nervous system for the earth." In 2011 IEEE Technology Time Machine (TTM). IEEE, 2011. http://dx.doi.org/10.1109/ttm.2011.6005165.

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Wodarcyk, A. J., and J. G. Wang. "Extensive Central Nervous System Nocardiosis Without Neurologic Manifestations." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4061.

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Phillips, Justin P., Panayiotis A. Kyriacou, Kuriakose J. George, John V. Priestley, and Richard M. Langford. "An Optical Fiber Photoplethysmographic System for Central Nervous System Tissue." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4397523.

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Phillips, Justin P., Panayiotis A. Kyriacou, Kuriakose J. George, John V. Priestley, and Richard M. Langford. "An Optical Fiber Photoplethysmographic System for Central Nervous System Tissue." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.259690.

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Dubrovin, V., M. Zakharova, A. Rashavchenko, and J. Tverdohleb. "Non-pharmacological correction methods of central nervous system disturbances." In 2015 Information Technologies in Innovation Business Conference (ITIB). IEEE, 2015. http://dx.doi.org/10.1109/itib.2015.7355049.

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Laakso, Ilkka, and Takenobu Murakami. "Thresholds of central nervous system stimulation at intermediate frequencies." In 2016 URSI Asia-Pacific Radio Science Conference (URSI AP-RASC). IEEE, 2016. http://dx.doi.org/10.1109/ursiap-rasc.2016.7601395.

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Reports on the topic "Adenoviral; Nervous system; Central"

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Ridgway, Sam H. The Cetacean Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, January 1999. http://dx.doi.org/10.21236/ada381704.

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Albquerque, Edson X. Molecular Targets for Organophosphates in the Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada426356.

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Rowland, Vernon, and Henry Gluck. Attention and Preparatory Processes in the Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, August 1986. http://dx.doi.org/10.21236/ada171316.

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Butler, F. K., and Jr. Central Nervous System Oxygen Toxicity in Closed-Circuit Scuba Divers. Fort Belvoir, VA: Defense Technical Information Center, March 1986. http://dx.doi.org/10.21236/ada170879.

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Clark, J. M., and C. J. Lambertsen. Extension of Central Nervous and Visual System Oxygen Tolerance in Physical Work. Fort Belvoir, VA: Defense Technical Information Center, December 1990. http://dx.doi.org/10.21236/ada239160.

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Mery, Laura, Matthew Wayner, John McQuade, and Erica Anderson. Characterization of the Effects of Fatigue on the Central Nervous System (CNS) and Drug Therapies. Fort Belvoir, VA: Defense Technical Information Center, November 2007. http://dx.doi.org/10.21236/ada489794.

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Catlin, Kristen M. Role of Cytokines and Neurotrophins in the Central Nervous System in Venezuelan Equine Encephalitis Pathogenesis. Fort Belvoir, VA: Defense Technical Information Center, February 2001. http://dx.doi.org/10.21236/ad1012369.

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Li, Yanming, Zhigang Zhao, and Yuanbo Liu. Combined chemotherapy in new diagnosed primary central nervous system lymphoma: a systematic review and network meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2020. http://dx.doi.org/10.37766/inplasy2020.9.0084.

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Carpenter, A. V., W. D. Flanders, E. L. Frome, D. J. Crawford-Brown, and S. A. Fry. Radiation exposure and central nervous system cancers: A case-control study among workers at two nuclear facilities. Office of Scientific and Technical Information (OSTI), March 1987. http://dx.doi.org/10.2172/6646019.

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Qu, Chunrun, Yu Chen, Yuzhen Ouyang, Ruoyu Lu, Yu Zeng, and Zhixiong Liu. Metagenomics Next Generation Sequencing for the Diagnosis of Central Nervous System Infection: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0002.

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