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Relevant bibliographies by topics / Adenosine-signalling

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Journal articles

Academic literature on the topic 'Adenosine-signalling'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "Adenosine-signalling"

1

Fredholm, Bertil B., Giulia Arslan, Bj�rn Kull, Ewa Kontny, and Per Svenningsson. "Adenosine (P1) receptor signalling." Drug Development Research 39, no. 3-4 (1996): 262–68. http://dx.doi.org/10.1002/(sici)1098-2299(199611/12)39:3/4<262::aid-ddr5>3.0.co;2-p.

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2

CARUSO, M., S. HOLGATE, and R. POLOSA. "Adenosine signalling in airways." Current Opinion in Pharmacology 6, no. 3 (2006): 251–56. http://dx.doi.org/10.1016/j.coph.2006.02.002.

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3

Silva, Luis, Mario Subiabre, Joaquín Araos, et al. "Insulin/adenosine axis linked signalling." Molecular Aspects of Medicine 55 (June 2017): 45–61. http://dx.doi.org/10.1016/j.mam.2016.11.002.

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4

Burnstock, Geoffrey. "Purine and purinergic receptors." Brain and Neuroscience Advances 2 (January 2018): 239821281881749. http://dx.doi.org/10.1177/2398212818817494.

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Adenosine 5′-triphosphate acts as an extracellular signalling molecule (purinergic signalling), as well as an intracellular energy source. Adenosine 5′-triphosphate receptors have been cloned and characterised. P1 receptors are selective for adenosine, a breakdown product of adenosine 5′-triphosphate after degradation by ectonucleotidases. Four subtypes are recognised, A1, A2A, A2B and A3 receptors. P2 receptors are activated by purine and by pyrimidine nucleotides. P2X receptors are ligand-gated ion channel receptors (seven subunits (P2X1-7)), which form trimers as both homomultimers and hete

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5

Liang, Bruce T., Tomasz A. Swierkosz, Howard C. Herrmann, Stephen Kimmel, and Kenneth A. Jacobson. "Adenosine and Ischemic Preconditioning." Current Pharmaceutical Design 5, no. 12 (1999): 1029–41. http://dx.doi.org/10.2174/1381612805666230112212126.

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Adenosine is released in large amounts during myocardial ischemia and is capable of exerting potent cardioprotective effects in the heart. Although these observations on adenosine have been known for a long time, how adenosine acts to achieve its anti-ischemic effect remains incompletely understood. However, recent advances on the chemistry and pharmacology of adenosine receptor ligands have provided important and novel information on the function of adenosine receptor subtypes in the cardiovascular system. The development of model systems for the cardiac actions of adenosine has yielded impor

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6

Vlajkovic, Srdjan M., and Peter R. Thorne. "Purinergic Signalling in the Cochlea." International Journal of Molecular Sciences 23, no. 23 (2022): 14874. http://dx.doi.org/10.3390/ijms232314874.

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The mammalian cochlea is the sensory organ of hearing with a delicate, highly organised structure that supports unique operating mechanisms. ATP release from the secretory tissues of the cochlear lateral wall (stria vascularis) triggers numerous physiological responses by activating P2 receptors in sensory, supporting and neural tissues. Two families of P2 receptors, ATP-gated ion channels (P2X receptors) and G protein-coupled P2Y receptors, activate intracellular signalling pathways that regulate cochlear development, homeostasis, sensory transduction, auditory neurotransmission and response

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7

Razak, Azlina A., Lopa Leach, and Vera Ralevic. "Impaired vasocontractile responses to adenosine in chorionic vessels of human term placenta from pregnant women with pre-existing and gestational diabetes." Diabetes and Vascular Disease Research 15, no. 6 (2018): 528–40. http://dx.doi.org/10.1177/1479164118790904.

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Background: There is clinical and experimental evidence for altered adenosine signalling in the fetoplacental circulation in pregnancies complicated by diabetes, leading to adenosine accumulation in the placenta. However, the consequence for fetoplacental vasocontractility is unclear. This study examined contractility to adenosine of chorionic vessels from type 1 diabetes mellitus, gestational diabetes mellitus and normal pregnancies. Methods: Chorionic arteries and veins were isolated from human placenta from normal, gestational diabetes mellitus and type 1 diabetes mellitus pregnancies. Isom

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8

DARCY, P. K., and P. R. FISHER. "Pharmacological evidence for a role for cyclic AMP signalling in Dictyostelium discoideum slug behaviour." Journal of Cell Science 96, no. 4 (1990): 661–67. http://dx.doi.org/10.1242/jcs.96.4.661.

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Phototaxis and thermotaxis by Dictyostelium discoideum slugs on water agar were impaired by the presence in the agar of adenosine, which is a cyclic AMP receptor antagonist in aggregating amoebae. Caffeine, and presumably its analogue theophylline, inhibit cyclic AMP signalling in aggregating amoebae of D. discoideum. Both compounds perturbed slug behaviour in a similar manner to adenosine, as did both ammonium and sulphate ions. (NH4)3SO4 is known to perturb cyclic AMP binding to its receptor, and ammonia is an inhibitor of cyclic AMP signalling in aggregating amoebae. The receptor agonist, c

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9

Przybyła, Tomasz, Monika Sakowicz-Burkiewicz, and Tadeusz Pawełczyk. "Purinergic signalling in B cells." Acta Biochimica Polonica 65, no. 1 (2018): 1–7. http://dx.doi.org/10.18388/abp.2017_1588.

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Adenosine and adenosine triphosphate are involved in purinergic signalling which plays important role in control of immune system. Much data have been obtained regarding impact of purinergic signalling on dendritic cells, macrophages, monocytes and T lymphocytes, however less attention has been paid to purinergic regulation of B cells. This review summarizes present knowledge about ATP- and Ado-dependant signalling in B lymphocytes. Human B cells have been shown to express A2A-R and A3-R and each subtype of P2 receptors. Surface of B cells exhibits two antagonistic ectoenzymatic pathways, on

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10

Gracia, Eduard, Kamil Pérez-Capote, Estefanía Moreno, et al. "A2A adenosine receptor ligand binding and signalling is allosterically modulated by adenosine deaminase." Biochemical Journal 435, no. 3 (2011): 701–9. http://dx.doi.org/10.1042/bj20101749.

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A2ARs (adenosine A2A receptors) are highly enriched in the striatum, which is the main motor control CNS (central nervous system) area. BRET (bioluminescence resonance energy transfer) assays showed that A2AR homomers may act as cell-surface ADA (adenosine deaminase; EC 3.5.4.4)-binding proteins. ADA binding affected the quaternary structure of A2ARs present on the cell surface. ADA binding to adenosine A2ARs increased both agonist and antagonist affinity on ligand binding to striatal membranes where these proteins are co-expressed. ADA also increased receptor-mediated ERK1/2 (extracellular-si

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