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Relevant bibliographies by topics / Addiction relapse / Books

Books on the topic 'Addiction relapse'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Cocaine addiction: Treatment, recovery, and relapse prevention. New York: Norton, 1989.

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2

E, Zweben Joan, ed. Cocaine & methamphetamine addiction: Treatment, recovery, and relapse prevention. New York: Norton, 2009.

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3

T, Gorski Terence, ed. Addiction-free pain management: The relapse prevention counseling workbook. Independence, Mo: Herald House/Independence Press, 1997.

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4

Johan, Egger, and Kalb Mikel, eds. Smoking relapse: Causes, prevention, and recovery. Hauppauge, N.Y: Nova Science Publishers, 2010.

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5

Clancy, Jo. Anger and addiction: Breaking the relapse cycle : a teaching guide for professionals. Madison, Conn: Psychosocial Press, 1996.

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6

Daley, Dennis C. Relapse: A guide to successful recovery : what you must know and do to maintain sobriety during recovery from alcohol or drug dependence. Bradenton, Fla: Human Services Institute, 1987.

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7

Shamil, Wanigaratne, ed. Relapse prevention for addictive behaviours: A manual for therapists. Oxford ; Cambridge, Mass: Blackwell Scientific Publications, 1990.

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8

Bedingungen des Rückfalls bei Rauchern: Die Vorhersagekraft kognitiver Faktoren und der Einfluss von Situationsmerkmalen auf den Rückfall. Frankfurt am Main: P. Lang, 1991.

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9

Koski-Jännes, Anja. Alcohol addiction and self-regulation: A controlled trial of a relapse prevention program for Finnish inpatient alcoholics. Helsinki, Finland: Distributors Akateeminen Kirjakauppam, 1992.

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10

1948-, Gossop Michael, ed. Relapse and addictive behaviour. London: Tavistock/Routledge, 1989.

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11

Chiauzzi, Emil J. Preventing relapse in the addictions. Oxford: Pergamon, 1991.

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12

Newport, John. The wellness-recovery connection: Charting your pathway to optimal health while recovering from alcoholism and drug addiction. Deerfield Beach, Fla: Health Communications, 2004.

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13

B, Baker Timothy, and Cannon Dale S, eds. Assessment and treatment of addictive disorders. New York: Praeger, 1988.

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14

Kenneth, Blum, ed. Overload: Attention deficit disorder and the addictive brain. Kansas City, Mo: Andrews and McMeel, 1996.

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15

Preventing relapse in the addictions: A biopsychosocial approach. New York: Pergamon Press, 1991.

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16

Wragg, Jeffrey. An evaluation of a model of drug education. Canberra: Australian Government Publishing Service, 1992.

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17

Daley, Dennis C. Kicking addictive habits once and forall: A relapse-prevention guide. Lexington, Mass: Lexington Books, 1991.

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18

Daley, Dennis C. Kicking addictive habits once and for all: A relapse-prevention guide. Lexington, Mass: Lexington Books, 1991.

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19

Daley, Dennis C. Kicking addictive habits once and for all: A relapse prevention guide. San Francisco: Jossey-Bass Publishers, 1998.

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20

Neharika, Chawla, and Marlatt G. Alan, eds. Mindfulness-based relapse prevention for addictive behaviors: A clinician's guide. New York: Guilford Press, 2011.

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21

Alan, Marlatt G., and Donovan Dennis M, eds. Relapse prevention: Maintenance strategies in the treatment of addictive behaviors. 2nd ed. New York: Guilford Press, 2005.

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22

White knuckles and wishful thinking: Breaking the chain of compulsive reaction and relapse in alcoholism and other addictions. Seattle: Hogrefe & Huber, 1992.

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23

Proven Holistic Treatment for Addiction & Chronic Relapse. Tate Publishing, 2006.

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24

Cocaine Addiction: Treatment, Recovery, and Relapse Prevention. W. W. Norton & Company, 1991.

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25

Daley, Dennis C., and Antoine Douaihy. Relapse Prevention Counseling: Clinical Strategies to Guide Addiction Recovery and Reduce Relapse. PESI, 2015.

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26

Daley, Dennis C., and Antoine Douaihy. Relapse Prevention Counseling: Clinical Strategies to Guide Addiction Recovery and Reduce Relapse. PESI, 2015.

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27

Douaihy, Antoine. Relapse Prevention Counseling: Clinical Strategies to Guide Addiction Recovery and Reduce Relapse. PESI Publishing & Media, 2015.

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28

Daley, Dennis C. Relapse: Conceptual Research and Clinical Perspectives. Haworth Press, 1989.

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29

Daley, Dennis C. Relapse: Conceptual Research and Clinical Perspectives. Haworth Press, 1989.

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30

Washton, Arnold, and Joan Ellen Zweben. Cocaine and Methamphetamine Addiction: Treatment, Recovery, and Relapse Prevention. W. W. Norton, 2008.

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31

Gold, Michelle. Fighting Addiction: A Powerful Book to Prevent You from Relapse. Independently Published, 2019.

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32

Kelly, Mary T. Relapse Prevention: A Workbook for Shopping Addiction Long Term Sobriety. Independently Published, 2018.

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33

Mulian, Bertha. Stop Shopping Addiction for Good: A Relapse Prevention and Recovery Workbook. Independently Published, 2019.

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34

Christopher, Evans J., Brigitte L. Kieffer, David Jentsch, and Rafael J. Maldonado. Animal Models of Addiction. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0043.

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Abstract:

Drug addiction, now officially diagnosed as substance use disorder (SUD), is a chronic brain syndrome characterized by the compulsive use of drugs, loss of control over drug taking in spite of its adverse consequences, and relapse even after long periods of drug abstinence. Animal models have played a critical role in our understanding of the molecules, circuits, and behaviors associated with substance use disorders. This chapter reviews animal models that have been widely used to assess all stages of the addiction cycle: from drug initiation, through drug seeking, to withdrawal and relapse. We discuss the power of genetics, especially in generating rodent models for the discovery of essential proteins and pathways regulating behaviors exhibited during the different stages of the addiction cycle. Preclinical research in animal models will undoubtedly continue to reveal therapeutic strategies for substance use disorders.

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35

Nutt, David J., and Liam J. Nestor. Nicotine addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0011.

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Cigarette smoking presents with considerable health risks and induces high costs on healthcare resources. People continue to smoke cigarettes in the face of adversity because they contain nicotine, which is highly addictive. Nicotine is a stimulant that exerts its effects within the brain by acting at nicotinic acetylcholine receptors (nAChRs). nAChRs are located in areas of the brain involved in reward processing, motivation, and cognitive control, which results in disruptions to behaviour when nicotine addiction has developed. Disturbances to the brain and behaviour are particularly evident during early nicotine abstinence when people are in withdrawal. Importantly, treatments (e.g. varenicline, bupropion) that attenuate disturbances to reward and cognition in the brain during withdrawal in early nicotine abstinence are conferred with the efficacy to promote smoking cessation and protect against relapse.

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36

Nutt, David J., and Liam J. Nestor. Appetite hormones and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0012.

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Many of the same behavioural and brain disturbances observed in addiction are also seen in obesity and binge-eating disorder. This suggests that there are shared neural substrates between substance addiction and compulsive food consumption. Food intake and appetite are regulated by numerous appetite hormones that exert their effects through brain systems involved in reward sensitivity, stress, impulsivity, and compulsivity. There is now emerging evidence that appetite hormones (e.g. ghrelin, glucagon-like peptide-1, orexin) can modulate addictive behaviours (e.g. craving) and the intake of alcohol and drugs. Therefore, there is an emerging shift into a new field of testing drugs that affect appetite hormones and their receptors in the brain, and their use in regulating the brain mechanisms that lead to relapse in addiction disorders.

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37

Nutt, David J., and Liam J. Nestor. The glutamate system and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0009.

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Glutamate is the primary excitatory neurotransmitter in the brain. Glutamate is involved in synaptic plasticity, particularly within dopamine systems of the brain that are involved in reward. Glutamate-dependent plasticity is involved in the development of substance addiction through its actions at NMDA receptors during long-term potentiation (LTP) related learning and memory processes. This plasticity within brain circuitry involved in learning and memory is sustained during substance abstinence and may provide a neural substrate for a vulnerability to addiction relapse. Medications that possess the efficacy to reduce glutamate tone in certain brain circuits may reduce craving, and ultimately, relapse in substance dependence. Further research is required, however, to show that the modulation of glutamate transmission in the brain confers clinical benefits in substance addiction.

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38

Summer, Edward. Relapse No More: A Therapeutic Community Addiction Recovery Prompt Journal Writing Notebook. Independently Published, 2019.

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39

Nutt, David J., and Liam J. Nestor. The opioid system and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0010.

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The opioid system of the brain is the major target for opiate drugs such as morphine and heroin, and has been implicated in processes such as pain, stress and reward. Many of these effects take place at the mu opioid receptor (mOR), which is distributed throughout the brain. Significantly, genetic polymorphisms at the mOR may confer a greater dopamine response to the reinforcing effects of alcohol, and it has been suggested that addiction per se may be associated with alterations to the opioid system. There is evidence for the potential efficacy of mOR antagonists (e.g. naltrexone) in reducing drug and alcohol relapse, increasing treatment retention and attenuating the subjective effects of substances of abuse. Medications with partial agonist activity at the kappa opioid receptor (e.g. nalmefene) may also confer an additional clinical advantage by reducing binging following relapse.

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40

Wallace, Wendy, Shamil Wanigaratne, Jane Pullin, and Francis Keaney. Relapse Prevention for Addictive Behaviours: A Manual for Therapists. Blackwell Science, 1990.

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41

Nutt, David J., and Liam J. Nestor. Pharmacodynamics of addictive substances. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0006.

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Pharmacodynamics refers to the effects addictive substances have on the body. Acutely, substances of addiction target various neurotransmitter systems in the brain. The pharmacological actions of these substances at receptors results in their physiological and psychological effects. The chronic abuse of these substances, however, leads to pharmacodynamic tolerance at receptors within neurotransmitter systems and leads to addiction. This tolerance means that a greater amount of the substance is required to achieve the desired effect and it has significant implications for relapse during early abstinence in addiction. Treatments target these neurotransmitter disturbances in addiction to prevent relapse.

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42

Understanding Drug and Alcohol Addiction: An LDS Perspective. CFI (Cedar Fort, Inc.), 2004.

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43

Block, Carolyn Bryant, Stanley H. Block, Guy du Plessis, and Robert Weathers. Mind-Body Workbook for Addiction: Effective Tools for Substance-Abuse Recovery and Relapse Prevention. New Harbinger Publications, 2016.

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44

Recovery training and self-help: Relapse prevention and aftercare for drug addicts. Rockville, MD (5600 Fishers Lane, Rockville 20857): U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Institute on Drug Abuse, Office of Science Policy, Education, and Legislation, Community and Professional Education Branch, 1993.

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45

Reissner, Kathryn J., and Peter W. Kalivas. Cellular and Molecular Mechanisms of Addiction. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0046.

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Exposure to drugs of abuse can be a reinforcing experience that, in vulnerable individuals, can lead to continued use and the development of an addiction disorder. Evidence indicates that the escalation in use and compulsive motivation to obtain the drug is linked to long-lasting cellular changes within the brain reward neurocircuitry. In this chapter we describe the stages of transition in use from social use to habitual relapse, and within that context we describe the implicated neurocircuitry, and the enduring cellular and molecular changes that occur within that circuitry, that may mediate the preoccupation with drug seeking in addiction-vulnerable individuals.

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46

Fran, Mel. Stop Your Addiction Prayer Journal: Daily Sobriety and Relapse Prevention Lined Writing Personal Intercession Notebook. Independently Published, 2019.

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47

Sam, Sathiya. Last Relapse: Realize Your Potential, Reclaim Intimacy, and Resolve the Root Issues of Porn Addiction. SMG Publishing, 2022.

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48

Sam, Sathiya. Last Relapse: Realize Your Potential, Reclaim Intimacy, and Resolve the Root Issues of Porn Addiction. SMG Publishing, 2022.

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49

Steele, Vaughn R., Vani Pariyadath, Rita Z. Goldstein, and Elliot A. Stein. Reward Circuitry and Drug Addiction. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0044.

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Addiction is a complex neuropsychiatric syndrome related to dysregulation of brain systems including the mesocorticolimbic dopamine reward circuit. Dysregulation of reward circuitry is related to each of the three cyclical stages in the disease model of addiction: maintenance, abstinence, and relapse. Parsing reward circuitry is confounded due to the anatomical complexity of cortico-basal ganglia-thalamocortical loops, forward and backward projections within the circuit, and interactions between neurotransmitter systems. We begin by introducing the neurobiology of the reward system, specifically highlighting nodes of the circuit beyond the basal ganglia, followed by a review of the current literature on reward circuitry dysregulation in addiction. Finally, we discuss biomarkers of addiction identified with neuroimaging that could help guide neuroprediction models and development of targets for effective new interventions, such as noninvasive brain stimulation. The neurocircuitry of reward, especially non-prototypical nodes, may hold essential keys to understanding and treating addiction.

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50

Maultsby, Maxie C. Stay Sober and Straight : How to Prevent Addiction Relapse with the Rational Self-Help Treatment Method. Rational Self-Help Books, 1990.

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