Academic literature on the topic 'Acidosis'
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Journal articles on the topic "Acidosis"
Areas, Jorge, Sevag Balian, Dianna Slemmer, Mario Belledonne, and Harry G. Preuss. "Renal ammoniagenesis following glutamine loading in intact dogs during acute metabolic acid–base perturbations." Clinical Science 72, no. 1 (January 1, 1987): 61–69. http://dx.doi.org/10.1042/cs0720061.
Full textWilliams, Bryan, Ellis Layward, and John Walls. "Skeletal muscle degradation and nitrogen wasting in rats with chronic metabolic acidosis." Clinical Science 80, no. 5 (May 1, 1991): 457–62. http://dx.doi.org/10.1042/cs0800457.
Full textChalla, A., R. J. Krieg, M. A. Thabet, J. D. Veldhuis, and J. C. Chan. "Metabolic acidosis inhibits growth hormone secretion in rats: mechanism of growth retardation." American Journal of Physiology-Endocrinology and Metabolism 265, no. 4 (October 1, 1993): E547—E553. http://dx.doi.org/10.1152/ajpendo.1993.265.4.e547.
Full textMoody, Mikayla, Tannin A. Schmidt, Ruchir Trivedi, and Alix Deymier. "Administration of alendronate exacerbates ammonium chloride-induced acidosis in mice." PLOS ONE 18, no. 9 (September 15, 2023): e0291649. http://dx.doi.org/10.1371/journal.pone.0291649.
Full textCaso, Giuseppe, Barbara A. Garlick, George A. Casella, Dawn Sasvary, and Peter J. Garlick. "Acute metabolic acidosis inhibits muscle protein synthesis in rats." American Journal of Physiology-Endocrinology and Metabolism 287, no. 1 (July 2004): E90—E96. http://dx.doi.org/10.1152/ajpendo.00387.2003.
Full textTETA, Daniel, Alan BEVINGTON, Jeremy BROWN, David THROSSELL, Kevin P. G. HARRIS, and John WALLS. "Effects of acidosis on leptin secretion from 3T3-L1 adipocytes and on serum leptin in the uraemic rat." Clinical Science 97, no. 3 (August 6, 1999): 363–68. http://dx.doi.org/10.1042/cs0970363.
Full textTüre, Hatice, Özgül Keskin, Ülkem Çakır, Canan Aykut Bingöl, and Uğur Türe. "The frequency and severity of metabolic acidosis related to topiramate." Journal of International Medical Research 44, no. 6 (October 27, 2016): 1376–80. http://dx.doi.org/10.1177/0300060516669897.
Full textPrice, S. R., X. Wang, and J. L. Bailey. "Tissue-specific responses of branched-chain alpha-ketoacid dehydrogenase activity in metabolic acidosis." Journal of the American Society of Nephrology 9, no. 10 (October 1998): 1892–98. http://dx.doi.org/10.1681/asn.v9101892.
Full textKarinch, Anne M., Cheng-Mao Lin, Christopher L. Wolfgang, Ming Pan, and Wiley W. Souba. "Regulation of expression of the SN1 transporter during renal adaptation to chronic metabolic acidosis in rats." American Journal of Physiology-Renal Physiology 283, no. 5 (November 1, 2002): F1011—F1019. http://dx.doi.org/10.1152/ajprenal.00106.2002.
Full textLucioni, Alvaro, Christopher Womack, Mark W. Musch, Flavio L. Rocha, Cres Bookstein, and Eugene B. Chang. "Metabolic acidosis in rats increases intestinal NHE2 and NHE3 expression and function." American Journal of Physiology-Gastrointestinal and Liver Physiology 283, no. 1 (July 1, 2002): G51—G56. http://dx.doi.org/10.1152/ajpgi.00529.2001.
Full textDissertations / Theses on the topic "Acidosis"
Force, Lisa Marie. "Traumatic brain injury and acidosis /." view abstract or download text of file, 2006. http://hdl.handle.net/1794/3913.
Full textMaruta, Celso Akio. "Comparação da susceptibilidade de bovinos das raças Jersey e Gir à acidose láctica ruminal, induzida experimentalmente com sacarose." Universidade de São Paulo, 2000. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-24072007-082404/.
Full textFour Jersey (J) and four Gir (G) rumen-cannulated steers were used. The steers were fed, for two months before the beginning of the rumen lactic acidosis (RLA) induction, a standard diet of hay and concentrates. The RLA was induced experimentally through the administration of sucrose into the rumen, according to the corrected metabolic weight, after ORTOLANI (1995). Blood, rumen fluid, urine, and fecal samples were collected and clinical examination carried out in the following times after the induction: zero, 14, 16, 18, 20, 22 and 24 hours. The pH, the total lactic acid and its L and D isomers were determined in all samples. The hematocrit, acid-base variables and the creatinine concentration were determined in the blood samples; creatinine was also determined in the urine samples. All the rumen content was evacuated in order to evaluate its volume at the 24th h. A intense rumen acidosis was reached; no differences in the rumen fluid pH and in the concentration of the total lactic acid and its isomers were found in both studied breeds. A moderate level of systemic metabolic acidosis was reached in both breeds, but lower overall mean of bicarbonate and TCO2 (p < 0.0001) as well as blood pH (p < 0.025) were found in the J steers. These steers absorbed higher amounts of total lactic and its D isomer than the G animals; the higher the blood D-lactate concentration, the lower the blood pH (r = - O.78) in the former breed. Better blood pH homeostasis were kept, at the end of induction, by the G steers, probably by their higher efficiency to metabolize L-lactate. However, the G steers exhibited a higher level of dehydration as seen by the greater hematocrit and plasma volume deficit (p < 0.00001). They also presented a lower glomerular filtration as evidenced by the higher creatinine serum levels (p < 0.00001), its lower urinary clearance (p < 0.003) and the lower estimated urinary volume (p < 0.05). There were no differences in the fecal pH values presented by both breeds. There was a negative correlation between the fecal total lactate concentration and the fecal pH (r = - 0.65).
Pinto, Ana Carolina Janssen. "Adaptação a dietas de alta energia para bovinos Nelore submetidos previamente à restrição nutricional ou consumo de concentrados e efeitos nas características ruminais." Universidade Estadual Paulista (UNESP), 2017. http://hdl.handle.net/11449/152743.
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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
O objetivo deste estudo foi avaliar o efeito da restrição alimentar prévia ou exposição anterior a ingredientes concentrados sobre o tempo para adaptação à dietas de alto concentrado, sob os parâmetros da fermentação ruminal e o perfil de microrganismos ruminais de bovinos Nelore canulados confinados. Foram utilizados 6 bovinos da raça Nelore, castrados, com peso vivo inicial aproximado de 236 ± 23 kg, 15 meses de idade e canulados no rúmen, os quais foram divididos em dois quadrados latinos 3 x 3. Os tratamentos diferiram somente sobre o tipo de alimentação estabelecida previamente ao período de adaptação: Controle (forragem ad libitum + suplemento mineral); Restrição (forragem restrita a 1,4% do peso vivo + suplemento mineral) e Concentrado (forragem ad libitum + 0,5% do peso vivo de ingredientes concentrados e suplemento mineral). A duração deste estudo foi de 119 dias, sendo compostos por 3 períodos experimentais (33 dias cada período) e dois intervalos de washout (10 dias) entre os períodos 1 e 2, e 2 e 3. Os períodos foram divididos em: 14 dias de pré- adaptação, 6 dias de adaptação 1 (72% de concentrado), 6 dias de adaptação 2 (79% de concentrado) e 7 dias de dieta de terminação (86% de concentrado). O pH ruminal foi monitorado por meio da utilização de data loggers a cada 10 minutos. Foram avaliados a concentração de ácidos graxos de cadeia curta e amônia ruminal; a quantificação relativa das bactérias celulolíticas e utilizadoras de lactato por meio da técnica de PCR e a contabilidade total e diferencial de protozoários (0,4,8 e 12 horas após o trato). Os animais restritos, na fase de adaptação, tiveram maior concentração de AGCC (P< 0,01), maior duração de pH abaixo de 6,2 (P<0,01) e menor pH máximo nos dias 15 e 16 (P≤ 0,10) em relação ao tratamento controle. Na fase de terminação, esses animais tiveram menor CMS (P=0,02), maior pH médio e menor área de pH abaixo de 6,2 (P≤0,10) em relação ao tratamento controle. Os animais expostos previamente ao concentrado, na fase de adaptação, tiveram menor concentração de butirato e NH3 ruminal (P≤0,10) em relação ao tratamento controle. Apresentaram também menor quantidade relativa de Fibrobacter succinogenes (P=0,10) e maior CMS (P<0,01) em relação ao controle. Já na fase de terminação o CMS não teve diferença significativa em relação ao tratamento controle. Os animais que passaram por restrição alimentar ou por exposição de concentrado na fase de pré-adaptação apresentam características ruminais semelhantes durante a fase a terminação aos animais em forragem ad libitum.
The objective of this study was to evaluate the effect of diet restriction or prior exposure to concentrate ingredientes prior to the adaption period to high concentrate diet on parameters ruminal fermentation patterns and ruminal microorganisms profile of cannulated Nellore cattle. Six Nelore steers was used, with initial body weight about 350 kg, 20 months and cannulated in the rumen, which will be divided into two Latin squares 3 x 3. The treatments differed only with respect to the type of diets prior to the adaptation period: Control (forage ad libitum + mineral supplement); Restriction (forage restricted to 1.4% of body weight + mineral supplement) and concentrate (forage ad libitum + 0.5% of the body weight of concentrated ingredients + mineral supplement). The study was last 119 days, in which animals was submitted to three experimental periods (33 days each one) and two washout intervals (10 days) between the periods 1 and 2, 2 and 3; each period was divided as follows: 14 days of pre-adaptation, 6 days of adaptation 1 (72% concentrate), 6 days of adaptation 2 (79% concentrate) and 7 days of finishing diet (88% concentrate). The rumen pH and temperature was be monitored through the use of data loggers it was evaluated the rumen production of short chain fatty acids and ammonia concentration; the relative quantification of cellulolytic, amylolytic and lactate bacteria utilizing PCR and total and differential quantification of protozoa. Thus, the hypothesis is to test if Nelore cattle exposed to feed restriction or comsuption of concentrate ingredientes prior to the adaptation period present same rumen fermentation patters and microrganism profile when compared to animals of control treatment in the finish diet. Restricted animals, in the adaptation phase, had a higher concentration of SCFA (P<0.01), a higher duration of pH below 6.2 (P <0.01) and lower maximum pH on days 15 and 16 (P≤ 0.10) in relation to the control treatment. In the finish diet, these animals had lower DMI (P = 0.02), higher mean pH and lower pH area below 6.2 (P≤0.10) in relation to the control treatment. The animals previously exposed to the concentrate in the adaptation diet had a lower concentration of butyrate and ruminal NH3 (P≤0.10) in relation to the control treatment. They also presented lower amount of Fibrobacter succinogenes (P = 0.10) and higher DMI (P <0.01) in relation to the control. In the termination diet the DMI did not affect in relation to the control treatment. Cattle previously submitted to either nutricional restriction or intake of concentrate have similar ruminal characteristics during the finishing diet of the animals in control treatment.
2016/04262-8
Vivan, Maria Carolina Ribeiro [UNESP]. "Correlação dos níveis de lactato sanguíneo com o estado neurológico e cardiorrespiratório de filhotes de cães nascidos de parto normal ou cesariana sob anestesia geral inalatória." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/92186.
Full textCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
A avaliação da perfusão tecidual com macroparâmetros não permite a detecção precoce de alteração na microvasculatura. A anestesia da gestante requer avaliação da perfusão e a eficácia do lactato na identificação de complicações em crianças após o parto já foi descrita. O presente estudo objetivou validar o lactato sanguíneo e correlacioná-lo a métodos, na avaliação de neonatos de parto normal ou cesariana eletiva sob anestesia geral inalatória. Foram utilizadas oito cadelas para realização de parto normal ou cesariana eletiva, com o protocolo composto de morfina, propofol e sevofluorano. Foram avaliados 24 neonatos de parto normal (GN) e 30 de cesariana (GC), com exames de sangue umbilical no nascimento para dosagem de lactato, hemogasometria, hematócrito, glicose e eletrólitos. No nascimento e aos 10 minutos de vida foram realizadas avaliações Apgar e neurológica. O lactato foi avaliado aos 10 minutos, 4 e 30 dias de vida. Os filhotes apresentaram acidose respiratória, acidemia e hipoxemia ao nascimento, mais elevada no GC. Os animais do GC apresentaram notas de Apgar e resposta neurológica menores ao nascimento, com melhora aos 10 minutos. O lactato sanguíneo foi maior nos animais do GN no nascimento, e foi maior nos animais que morreram. A correlação entre o lactato e as variáveis ocorreu em GN. O lactato sanguíneo associado aos demais parâmetros foi útil na avaliação dos neonatos do GN, porém nos animais do GC não houve correlação com a condição clínica dos animais no momento do nascimento. O procedimento anestésico influencia nos valores de lactato, e a determinação do melhor intervalo para sua avaliação nesses pacientes é necessária.
The assessment of tissue perfusion using macro parameters does not allow early detection of changes in the microvasculature. Anesthesia for pregnant patient requires evaluation of perfusion, and the lactate effectiveness in identifying complications in children after birth has been described. This study aimed to validate the blood lactate and its correlation with other methods, in the evaluation of neonates born from vaginal delivery or elective cesarean section under inhalator anesthesia. Eight dogs were used to perform normal delivery or cesarean section, with the protocol consisting of morphine, propofol and sevoflurane. At birth were evaluated 24 neonates born from vaginal delivery (NG) and 30 born from cesarean section (CG) using umbilical blood to test lactate, blood gas, hematocrit, glucose and electrolytes. Apgar score and neurologic tests were performed at birth and at 10 minutes of life. Lactate levels were evaluated at 10 minutes, 4 and 30 days of life. The puppies showed respiratory acidosis, hypoxemia and acidemia at birth, higher in the CG. The animals of CG presented lower Apgar scores and neurological response at birth, which improved in 10 minutes. Blood lactate was higher in NG animals at birth, and was higher in those who died. The correlation between lactate and the variables occurred on NG. Association of blood lactate with other parameters was useful in the evaluation of neonates on NG, but in the animals of CG there was no correlation with the clinical condition of animals at birth. Anesthesia influences the lactate values, and it is necessary to determine the best interval for evaluation in these patients.
Pereira, Ismael de Castro. "Estudo meta-analítico da flutuação de ingestão de massa seca no desempenho, comportamento ingestivo e saúde ruminal de bovinos confinados com dietas de alto concentrado." Botucatu, 2016. http://hdl.handle.net/11449/141503.
Full textResumo: O presente estudo teve o objetivo de avaliar o impacto da flutuação natural da ingestão de massa seca no desempenho, características de carcaça, comportamento ingestivo, perfil metabólico sanguíneo e aspectos morfofisiológicos ruminais de bovinos alimentados com dietas de alta inclusão de concentrado, por meio de estudo meta-analítico. Os experimentos contidos na meta-análise utilizaram animais com grupamento genético similar ao utilizado em confinamentos comerciais brasileiros. A meta-análise teve como base de dados os resultados de dez experimentos conduzidos no Confinamento Experimental da Faculdade de Medicina Veterinária e Zootecnia da UNESP, em Botucatu, entre os anos de 2006 e 2015. Os dados foram coletados a partir 838 bovinos, divididos em 238 baias, consideradas as unidades experimentais para o estudo. Os valores de flutuação de ingestão de massa seca na baia foram calculados pela diferença da ingestão de massa seca entre dois dias consecutivos e dividido pelo dia anterior para encontrar o valor em porcentagem. Com base na mediana geral de 5,62%, os animais foram divididos em dois grupos: alta e baixa flutuação de massa seca. As análises estatísticas foram feitas com auxílio do programa computacional R (versão 3.2.3). Utilizou-se um modelo de regressão linear misto para analisar os dados, no qual os experimentos e baias foram considerados como variáveis aleatórias e grupos de flutuação como variável fixa. Os grupos de baixa e alta flutuação apresentaram flutuação mé... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: This study aimed to evaluate the inpact of natural dry matter intake fluctuation on performance, carcass characteristics, feeding behavior, blood metabolic profile and morphophysiological ruminal aspects of cattle fed with diets of high inclusion of concentrate. The experiments used for this meta-analysis used cattle with genetic group similar to used in Brazilian commercial feedlots. The meta-analysis was conducted using a database of results of ten experiments conducted in Experimental Feedlot of Faculty of Veterinary Medicine and Animal Science, UNESP, Botucatu, between the years 2006 and 2015. Data were collected from 838 bulls, divided into 238 pens, considered the experimental units for the study. The values of dry matter intake fluctuation were calculated by the difference in dry matter intake between consecutive days and divided by the previous day to find the value in percentage. Based on the overall median of 5.62% cattle were classified into two groups: high- or low-fluctuation. All data analysis was performed in R using a mixed model approach where pens and year were random variables and fluctuation group fixed. The low and high fluctuation groups presented a dry matter intake fluctuation average of 4.79% and 6.74%, respectively. Low fluctuation group showed higher dry matter intake as percent of body weight (P <0.01), higher average daily gain (P = 0.05), higher dry matter intake in kg (P = 0.06), greater total weight gain (P = 0.08), as well as greater depositio... (Complete abstract click electronic access below)
Mestre
Bethell, H. W. L. "Potassium, acidosis and ventricular repolarisation during ischaemia." Thesis, University of Cambridge, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596607.
Full textReaich, David. "Protein and carbohydrate metabolism in metabolic acidosis." Thesis, University of Aberdeen, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308003.
Full textHerbst, Andreas. "Acidemia at birth risk factors, diagnosis and prognosis, with special reference to maternal fever in labour /." Lund : Dept. of Obstetrics and Gynaecology, University of Lund, 1997. http://books.google.com/books?id=dF9rAAAAMAAJ.
Full textLi, Ping-An. "Mechanisms of acidosis-mediated ischemic brain damage histopathology and pathophysiology /." Lund : Lund University, 1996. http://catalog.hathitrust.org/api/volumes/oclc/38158955.html.
Full textKey, M. L. "Acidosis, a fundamental regulator of bone cell function." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1361011/.
Full textBooks on the topic "Acidosis"
E. Wesson, Donald, ed. Metabolic Acidosis. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8.
Full textMuñoz, Ricardo, ed. Renal Tubular Acidosis in Children. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-91940-5.
Full textLangbroek, Aart Jan Mattheüs. Metabolic acidosis and ventilatory response. Groningen: Drucker:] van Denderen, 1988.
Find full textArieff, Allen I., ed. Hypoxia, Metabolic Acidosis, and the Circulation. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4614-7542-2.
Full text1938-, Arieff Allen I., and American Physiological Society (1887- ), eds. Hypoxia, metabolic acidosis, and the circulation. New York: Published for the American Physiological Society by Oxford University Press, 1992.
Find full textParker, James N., and Philip M. Parker. The Official patient's sourcebook on renal tubular acidosis. San Diego, Calif: Icon Health Publications, 2002.
Find full textRai, Sharadindu. Protective effects of acidosis against stretch-induced lung injury. Ottawa: National Library of Canada, 2002.
Find full textChŏn, Yang-suk. Chongyang ŭi sansŏnghwa e ŭihan HIF-1[alpha] kwabarhyŏn kijŏn kyumyŏng kwa saeroun hangam chʻiryo tʻaget ŭi palgul =: Mechanism of HIF-1[alpha] overexpression in acidified tumor and novel target for anticancer therapy. [Seoul]: Pogŏn Pokchibu, 2007.
Find full textJohn, Gennari F., ed. Acid-base disorders and their treatment. Boca Raton: Taylor & Francis, 2005.
Find full textSimonsohn, Barbara. Healing power of papaya: A holistic handbook on how to avoid acidosis, allergies, and other health disorders. Twin Lakes, WI: Lotus Light Publications, 2000.
Find full textBook chapters on the topic "Acidosis"
Mehlhorn, Heinz. "Acidosis." In Encyclopedia of Parasitology, 57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4426.
Full textMehlhorn, Heinz. "Acidosis." In Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4426-1.
Full textBöning, Dieter, Michael I. Lindinger, Damian M. Bailey, Istvan Berczi, Kameljit Kalsi, José González-Alonso, David J. Dyck, et al. "Acidosis." In Encyclopedia of Exercise Medicine in Health and Disease, 5–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_3.
Full textGoraya, Nimrit, and Donald E. Wesson. "Overview of Acid–Base Physiology." In Metabolic Acidosis, 1–6. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_1.
Full textAmodu, Afolarin, and Matthew K. Abramowitz. "Effects of Metabolic Acidosis on Skeletal Muscle." In Metabolic Acidosis, 101–10. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_10.
Full textE. Wesson, Donald. "Metabolic Acidosis Effects on Bone and Its Metabolism." In Metabolic Acidosis, 111–20. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_11.
Full textE. Wesson, Donald. "Endocrine Consequences of Metabolic Acidosis." In Metabolic Acidosis, 121–30. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_12.
Full textKovesdy, Csaba P. "Metabolic Acidosis and Progression of Chronic Kidney Disease." In Metabolic Acidosis, 131–43. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_13.
Full textE. Wesson, Donald. "Management of Chronic Metabolic Acidosis." In Metabolic Acidosis, 145–53. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_14.
Full textLaski, Melvin E. "Metabolic Acidosis: Physiology, Presentation, and Diagnosis." In Metabolic Acidosis, 7–16. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3463-8_2.
Full textConference papers on the topic "Acidosis"
Onanyan, M., I. Garrison, R. White, M. Crook, T. Stevens, and J. Y. Lee. "Extrinsic Acidosis Suppresses Lung Endothelium Metabolism While Intrinsic Acidosis Does Not." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1988.
Full textNechipurenko, Yury Dmitrievich, Denis Aleksandrovich Semyonov, Igor Andreevich Lavrinenko, Denis Anatolievich Lagutkin, Evgenii Aleksandrovich Generalov, Anna Yurevna Zaitceva, Olga Vyacheslavovna Matveeva, and Yegor Evgenievich Yegorov. "PATHOGENESIS OF COVID-19: THE ROLE OF ACIDOSIS." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.43.
Full textRaoof, S. "Metformin Associated Lactic Acidosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4805.
Full textTsay, Junchieh J., Derrick Raptis, and David R. Schwartz. "Lactic Acidosis In Metastatic Melanoma." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4609.
Full textLeiser, A., R. Virdee, and P. Kinniry. "Severe Epinephrine-Induced Lactic Acidosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3414.
Full textGanta, Ashwin, Ratna Priya Gangi, Wilson Gonsalves, Sonica Saini, and Tammy Wichman. "Inhalational Beta Agonist Induced Lactic Acidosis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1343.
Full textJeyaraman, Alagusutha, Neil Wright, and Marta Cohen. "227 Mind the gap!! uncommon cause of high anion gap metabolic acidosis due to pyroglutamic acidosis (PGA)." In RCPCH Conference Singapore. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjpo-2021-rcpch.124.
Full textGheorghe, MC, R. Dadu, F. Berianu, R. Vazquez, Y. Feng, C. Blot, A. Balasingham, D. Kaufman, and CA Manthous. "Hyperchloremic Metabolic Acidosis Following Resuscitation of Shock." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1595.
Full textMoe, Orson W., Daniel G. Fuster, Xiao-Song Xie, James C. Williams, Andrew P. Evan, James E. Lingeman, and James A. McAteer. "Distal Renal Tubular Acidosis and Calcium Nephrolithiasis." In RENAL STONE DISEASE 2: 2nd International Urolithiasis Research Symposium. AIP, 2008. http://dx.doi.org/10.1063/1.2998065.
Full textEl-Kenawi, Asmaa E., Arig A. Ibrahim-Hashim, Kimberly A. Luddy, Shari A. Pilon-Thomas, Robert A. Gatenby, and Robert J. Gillies. "Abstract 3213: Extracellular acidosis alters polarization of macrophages." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3213.
Full textReports on the topic "Acidosis"
Rodriquez, Fernando, Mark A. Rasmussen, and Milton J. Allison. Discovery of a Probiotic to Reduce the Risk of Lactic Acidosis in Cattle. Ames (Iowa): Iowa State University, January 2007. http://dx.doi.org/10.31274/ans_air-180814-563.
Full textChiquette, Johanne, Milton J. Allison, and Mark A. Rasmussen. Use of Prevotella bryantii 25A as a Probiotic to Reduce the Risk of Ruminal Acidosis in Dairy Cows. Ames (Iowa): Iowa State University, January 2008. http://dx.doi.org/10.31274/ans_air-180814-161.
Full textVisser, Wesley, Elma van de Braak, Anneke de Mik - van Egmond, Anna van der Burgh, Nicole de Roos, Inez Jans, Iris van der Hoef, Joanne Olieman, Ewout Hoorn, and David Severs. Correcting metabolic acidosis and the nutritional status of patients with non-dialysis dependent chronic kidney disease: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2023. http://dx.doi.org/10.37766/inplasy2023.5.0085.
Full textGuerreiro, Hugo, Rute Borrego, and Lino Mendes. β-alanine supplementation for athletic performance in female athletes: a protocol for a systematic review of randomized control trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0041.
Full textBannink, A., J. Dijkstra, and S. J. Koopmans. Relaties tussen acidose in de pens, in de darm en in het metabolisme van melkvee tijdens de vroege lactatie : deskstudie. Wageningen: Wageningen Livestock Research, 2018. http://dx.doi.org/10.18174/465646.
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