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Relevant bibliographies by topics / ACE

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Academic literature on the topic 'ACE'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

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Journal articles on the topic "ACE"

1

Sapak, Z., A. N. Mohd Faisol Mahadeven, Nurul Farhana M.H., Norsahira S., and Mohd Zafri A.W. "A review of common diseases of pineapple: the causal pathogens, disease symptoms, and available control measures." Food Research 5, S4 (November 26, 2021): 1–14. http://dx.doi.org/10.26656/fr.2017.5(s4).004.

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Pineapple is a perennial fruit-bearing tropical plant that belongs to the Bromeliaceae family, which has more than 2500 species. Pineapple is known excellent source of minerals and vitamins. It produces substantial calcium, potassium, glucose, the proteindigesting enzyme bromelain, fibre, vitamin A, B and C. In Malaysia, twelve registered varieties of pineapple have been introduced and commercially planted such as Moris (AC1), Sarawak (AC2), Gandul (AC3), Maspine (AC4), Josapine (AC5) Yankee (AC6) Moris Gajah (AC7), N36 (AC8), MD2 (AC9), View of Sunset (AC10), Madu Kaca (AC11), and Keningau Diamond (AC12). The disease is one of the important factors that can contribute to the huge losses of pineapple yield worldwide. This review paper aimed to discuss the main diseases of pineapple and their control management, particularly in Malaysia. The common diseases of pineapple that cause significant yield losses in the farms such as mealybug wilt-associated virus, bacterial heart rot, fruit collapse, butt rot, fruitlet core rot, black rot, yeasty and fusariosis are highlighted and discussed in detail on the causal pathogens, disease symptoms and signs, disease infection and development. The available control measures for managing pineapple diseases were also included in this paper.

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Alam, Md Amirul, Abdul Shukor Juraimi, M. Y. Rafii, Azizah Abdul Hamid, and Farzad Aslani. "Screening of Purslane (Portulaca oleraceaL.) Accessions for High Salt Tolerance." Scientific World Journal 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/627916.

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Purslane (Portulaca oleraceaL.) is an herbaceous leafy vegetable crop, comparatively more salt-tolerant than any other vegetables with high antioxidants, minerals, and vitamins. Salt-tolerant crop variety development is of importance due to inadequate cultivable land and escalating salinity together with population pressure. In this view a total of 25 purslane accessions were initially selected from 45 collected purslane accessions based on better growth performance and subjected to 5 different salinity levels, that is, 0.0, 10.0, 20.0, 30.0, and 40.0 dS m−1NaCl. Plant height, number of leaves, number of flowers, and dry matter contents in salt treated purslane accessions were significantly reduced (P≤0.05) and the enormity of reduction increased with increasing salinity stress. Based on dry matter yield reduction, among all 25 purslane accessions 2 accessions were graded as tolerant (Ac7 and Ac9), 6 accessions were moderately tolerant (Ac3, Ac5, Ac6, Ac10, Ac11, and Ac12), 5 accessions were moderately susceptible (Ac1, Ac2, Ac4, Ac8, and Ac13), and the remaining 12 accessions were susceptible to salinity stress and discarded from further study. The selected 13 purslane accessions could assist in the identification of superior genes for salt tolerance in purslane for improving its productivity and sustainable agricultural production.

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Jourdan, Karen B., Nicola A. Mason, Lu Long, Peter G. Philips, Martin R. Wilkins, and Nicholas W. Morrell. "Characterization of adenylyl cyclase isoforms in rat peripheral pulmonary arteries." American Journal of Physiology-Lung Cellular and Molecular Physiology 280, no. 6 (June 1, 2001): L1359—L1369. http://dx.doi.org/10.1152/ajplung.2001.280.6.l1359.

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Activation of adenylyl cyclase (AC), of which there are 10 diversely regulated isoforms, is important in regulating pulmonary vascular tone and remodeling. Immunohistochemistry in rat lungs demonstrated that AC2, AC3, and AC5/6 predominated in vascular and bronchial smooth muscle. Isoforms 1, 4, 7, and 8 localized to the bronchial epithelium. Exposure of animals to hypoxia did not change the pattern of isoform expression. RT-PCR confirmed mRNA expression of AC2, AC3, AC5, and AC6 and demonstrated AC7 and AC8 transcripts in smooth muscle. Western blotting confirmed the presence of AC2, AC3, and AC5/6 proteins. Functional studies provided evidence of cAMP regulation by Ca2+ and protein kinase C-activated but not Gi-inhibited pathways, supporting a role for AC2 and a Ca2+-stimulated isoform, AC8. However, NKH-477, an AC5-selective activator, was more potent than forskolin in elevating cAMP and inhibiting serum-stimulated [3H]thymidine incorporation, supporting the presence of AC5. These studies demonstrate differential expression of AC isoforms in rat lungs and provide evidence that AC2, AC5, and AC8 are functionally important in cAMP regulation and growth pathways in pulmonary artery myocytes.

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Long, Qi, Ming-Hui Sun, Xiao-Xue Fan, Zong-Bing Cai, Kai-Yao Zhang, Si-Yi Wang, Jia-Xin Zhang, et al. "First Identification and Investigation of piRNAs in the Larval Gut of the Asian Honeybee, Apis cerana." Insects 14, no. 1 (December 23, 2022): 16. http://dx.doi.org/10.3390/insects14010016.

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Piwi-interacting RNAs (piRNAs), a class of small non-coding RNAs (ncRNAs), play pivotal roles in maintaining the genomic stability and modulating biological processes such as growth and development via the regulation of gene expression. However, the piRNAs in the Asian honeybee (Apis cerana) are still largely unknown at present. In this current work, on the basis of previously gained high-quality small RNA-seq datasets, piRNAs in the larval gut of Apis cerana cerana, the nominated species of A. cerana, were identified for the first time, followed by an in-depth investigation of the regulatory roles of differentially expressed piRNAs (DEpiRNAs) in the developmental process of the A. c. cerana. Here, a total of 621 piRNAs were identified in A. c. cerana larval guts, among which 499 piRNAs were shared by 4-(Ac4 group), 5-(Ac5 group), and 6-day-old (Ac6 group) larval guts, while the numbers of unique ones equaled 79, 37, and 11, respectively. The piRNAs in each group ranged from 24 nucleotides (nt) to 33 nt in length, and the first base of the piRNAs had a cytosine (C) bias. Additionally, five up-regulated and five down-regulated piRNAs were identified in the Ac4 vs. Ac5 comparison group, nine of which could target 9011 mRNAs; these targets were involved in 41 GO terms and 137 pathways. Comparatively, 22 up-regulated piRNAs were detected in the Ac5 vs. Ac6 comparison group, 21 of which could target 28,969 mRNAs; these targets were engaged in 46 functional terms and 164 pathways. The results suggested an overall alteration of the expression pattern of piRNAs during the developmental process of A. c. cerana larvae. The regulatory network analysis showed that piR-ace-748815 and piR-ace-512574 in the Ac4 vs. Ac5 comparison group as well as piR-ace-716466 and piR-ace-828146 in the Ac5 vs. Ac6 comparison group were linked to the highest number of targets. Further investigation indicated that targets of DEpiRNAs in the abovementioned two comparison groups could be annotated to several growth and development-associated pathways, such as the Jak/STAT, TGF-β, and Wnt signaling pathways, indicating the involvement of DEpiRNAs in modulating larval gut development via these crucial pathways. Moreover, the expression trends of six randomly selected DEpiRNAs were verified using a combination of stem-loop RT-PCR and RT-qPCR. These results not only provide a novel insight into the development of the A. c. cerana larval gut, but also lay a foundation for uncovering the epigenetic mechanism underlying larval gut development.

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Strait, Kevin A., Peter K. Stricklett, Mark Chapman, and Donald E. Kohan. "Characterization of vasopressin-responsive collecting duct adenylyl cyclases in the mouse." American Journal of Physiology-Renal Physiology 298, no. 4 (April 2010): F859—F867. http://dx.doi.org/10.1152/ajprenal.00109.2009.

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Little is known about collecting duct adenylyl cyclase (AC) isoforms or regulation in the mouse. We performed RT-PCR for AC isoforms 1–9 in microdissected cortical (CCD) and outer medullary (OMCD) and acutely isolated inner medullary (IMCD) collecting duct. All collecting duct regions contained AC3, AC4, and AC6 mRNA, while CCD and OMCD, but not IMCD, also contained AC5 mRNA. Acutely isolated IMCD expressed AC3, AC4, and AC6 proteins by Western blot analysis. The mIMCD3 cell line expressed AC2, AC3, AC4, AC5, and AC6 mRNA; M-1 CCD cells expressed AC2, 3, 4, and 6, while mpkCCD cell lines contained AC3, AC4, and AC6 mRNA. AVP stimulated cAMP accumulation in acutely isolated mouse IMCD; this was reduced by chelation of extracellular calcium (EGTA) and almost completely abolished by blockade of calmodulin (W-7). Blockade of calmodulin kinase with KN-93 or endoplasmic reticulum calcium ATPase (thapsigargin) also reduced the AVP response. A similar inhibitory effect of W-7, KN-93, and thapsigargin was seen on forskolin-stimulated cAMP content in acutely isolated mouse IMCD. These three agents had the same pattern of blockade of AVP- or forskolin-stimulated AC activity in acutely isolated rat IMCD. AVP responsiveness in primary cultures of mouse IMCD was also reduced by W-7, KN-93, and thapsigargin. Small interfering RNA (siRNA) designed to knock down AC3 or AC6 in primary cultured mouse IMCD significantly reduced AVP-stimulated cAMP accumulation. Together, these data are consistent with a role of AC3 and AC6 in the activation of mouse collecting duct by AVP.

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Yang, Jin, Xuhui Feng, Qiong Zhou, Wei Cheng, Ching Shang, Pei Han, Chiou-Hong Lin, Huei-Sheng Vincent Chen, Thomas Quertermous, and Ching-Pin Chang. "Pathological Ace2-to-Ace enzyme switch in the stressed heart is transcriptionally controlled by the endothelial Brg1–FoxM1 complex." Proceedings of the National Academy of Sciences 113, no. 38 (September 6, 2016): E5628—E5635. http://dx.doi.org/10.1073/pnas.1525078113.

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Genes encoding angiotensin-converting enzymes (Ace and Ace2) are essential for heart function regulation. Cardiac stress enhances Ace, but suppresses Ace2, expression in the heart, leading to a net production of angiotensin II that promotes cardiac hypertrophy and fibrosis. The regulatory mechanism that underlies the Ace2-to-Ace pathological switch, however, is unknown. Here we report that the Brahma-related gene-1 (Brg1) chromatin remodeler and forkhead box M1 (FoxM1) transcription factor cooperate within cardiac (coronary) endothelial cells of pathologically stressed hearts to trigger the Ace2-to-Ace enzyme switch, angiotensin I-to-II conversion, and cardiac hypertrophy. In mice, cardiac stress activates the expression of Brg1 and FoxM1 in endothelial cells. Once activated, Brg1 and FoxM1 form a protein complex on Ace and Ace2 promoters to concurrently activate Ace and repress Ace2, tipping the balance to Ace2 expression with enhanced angiotensin II production, leading to cardiac hypertrophy and fibrosis. Disruption of endothelial Brg1 or FoxM1 or chemical inhibition of FoxM1 abolishes the stress-induced Ace2-to-Ace switch and protects the heart from pathological hypertrophy. In human hypertrophic hearts, BRG1 and FOXM1 expression is also activated in endothelial cells; their expression levels correlate strongly with the ACE/ACE2 ratio, suggesting a conserved mechanism. Our studies demonstrate a molecular interaction of Brg1 and FoxM1 and an endothelial mechanism of modulating Ace/Ace2 ratio for heart failure therapy.

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Lv, Yunyun, Yanping Li, Yunhai Yi, Lijun Zhang, Qiong Shi, and Jian Yang. "A Genomic Survey of Angiotensin-Converting Enzymes Provides Novel Insights into Their Molecular Evolution in Vertebrates." Molecules 23, no. 11 (November 9, 2018): 2923. http://dx.doi.org/10.3390/molecules23112923.

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Angiotensin-converting enzymes, ACE and ACE2, are two main elements in the renin–angiotensin system, with a crucial role in the regulation of blood pressure in vertebrates. Previous studies paid much attention to their physiological functions in model organisms, whereas the studies on other animals and related evolution have been sparse. Our present study performed a comprehensive genomic investigation on ace and ace2 genes in vertebrates. We successfully extracted the nucleotide sequences of ace and ace2 genes from high-quality genome assemblies of 36 representative vertebrates. After construction of their evolutionary tree, we observed that most of the phylogenetic positions are consistent with the species tree; however, certain differences appear in coelacanths and frogs, which may suggest a very slow evolutionary rate in the initial evolution of ace and ace2 in vertebrates. We further compared evolutionary rates within the entire sequences of ace and ace2, and determined that ace2 evolved slightly faster than ace. Meanwhile, we counted that the exon numbers of ace and ace2 in vertebrates are usually 25 and 18 respectively, while certain species may occur exon fusion or disruption to decrease or increase their exon numbers. Interestingly, we found three homologous regions between ace and ace2, suggesting existence of gene duplication during their evolutionary process. In summary, this report provides novel insights into vertebrate ace and ace2 genes through a series of genomic and molecular comparisons.

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Zhang, Ruifeng, Yingli Wu, Meng Zhao, Chuanxu Liu, Lin Zhou, Shaoming Shen, Shihua Liao, Kun Yang, Qingyun Li, and Huanying Wan. "Role of HIF-1α in the regulation ACE and ACE2 expression in hypoxic human pulmonary artery smooth muscle cells." American Journal of Physiology-Lung Cellular and Molecular Physiology 297, no. 4 (October 2009): L631—L640. http://dx.doi.org/10.1152/ajplung.90415.2008.

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Angiotensin-converting enzyme (ACE) enhances the proliferation and migration of pulmonary artery smooth muscle cells (PASMCs), which contribute to the pathogenesis of hypoxic pulmonary hypertension (HPH). Previous reports have demonstrated that hypoxia upregulates ACE expression, but the underlying mechanism is unknown. Here, we found that ACE is persistently upregulated in PASMCs on the transcriptional level during hypoxia. Hypoxia-inducible factor 1α (HIF-1α), a key transcription factor activated during hypoxia, was able to upregulate ACE protein expression under normoxia, whereas knockdown of HIF-1α expression in PASMCs inhibited hypoxia-induced ACE upregulation. Furthermore, HIF-1α can bind and transactivate the ACE promoter directly. Therefore, we report that ACE is a novel target of HIF-1α. Recently, a homolog of ACE, ACE2, was reported to counterbalance the function of ACE. In contrast to ACE, we found that ACE2 mRNA and protein levels increased during the early stages of hypoxia and decreased to near-baseline levels at the later stages after HIF-1α accumulation. Thus HIF-1α inhibited ACE2 expression, and the accumulated ANG II catalyzed by ACE is a key mediator in the downregulation of ACE2 by HIF-1α. Moreover, a reduction of ACE2 expression in PASMCs by RNA interference was accompanied by significantly enhanced proliferation and migration during hypoxia. We conclude that ACE is directly regulated by HIF-1α, whereas ACE2 is regulated in a bidirectional way during hypoxia and may play a protective role during the development of HPH. In sum, these findings contribute to the understanding of the pathogenesis of HPH.

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Afifah, Nisa Nur, Yani Mulyani, and Ari Yuniarto. "Review: Pengaruh Tanaman Obat Yang Beraktivitas Hipertensi Terhadap Ekspresi Gen Reseptor ACE-1 dan ACE 2." Jurnal Mandala Pharmacon Indonesia 7, no. 1 (June 30, 2021): 9–31. http://dx.doi.org/10.35311/jmpi.v7i1.64.

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Hipertensi adalah salah satu penyakit dengan angka kesakitan dan kematian yang terus meningkat, termasuk di Indonesia. Dalam mengatasi hipertensi obat-obatan seperti ACE inhibitor berperan dalam menurunkan tekanan darah diastol dan sistol, namun tanaman obat seperti ekstrak buah hawthorn, buah zaitun (Olea europaea L.), Hibiscus Sabdariffa, Allium Sativum dan Allium Cepa juga memiliki efek sebagai antihipertensi dengan harga yang relatif murah, mudah didapat, efek samping yang lebih rendah dibandingkan dengan obat sintesis atau kimia lainnya. Review jurnal ini ditujukan untuk mengetahui berbagai tanaman obat yang memiliki aktivitas hipertensi dan berpengaruh terhadap ekspresi gen reseptor hipertensi ACE1 dan ACE2. Penelusuran referensi dilakukan melalui database PubMed, Science Direct, dan Google Scholar, dengan kata kunci “Medicinal Plant”, “Gene expression”, “Angiotensin Converting Enzyme 1”, “Angiotensin Converting Enzyme 2”, dan“Antihypertension”. Tanaman obat digunakan sebagai terapi alternatif penurun tekanan darah tinggi dan merupakan salah satu cara pengobatan non farmakologis hipertensi. Hal ini menunjukkan bahwa tanaman obat memiliki pengaruh dalam menurunkan tekanan darah tinggi karena kandungan senyawa yang ada dalam masing-masing tanaman sehingga mampu menghambat reseptor hipertensi ACE1 dan ACE2 dengan berbagai metode ekspresi gen. Banyak tanaman obat yang telah diteliti memiliki aktivitas sebagai antihipertensi. Dari 14 tanaman obat dengan aktivitas sebagai anti hipertensi, sebanyak 90% tanaman berpengaruh terhadap ekspresi gen Angiotensin Converting Enzyme 1 (ACE 1), dan sebanyak 10% tanaman memiliki pengaruh terhadap Angiotensin Converting Enzyme 2 (ACE 2). Tanaman obat yang telah ditemukan dan memiliki aktivitas terhadap ekspresi gen Angiotensin Converting Enzyme 1 (ACE 1) paling banyak merupakan tanaman obat dengan family Poaceae, Oleaceae, dan Zingiberaceae.

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Alam, Md Amirul, Abdul Shukor Juraimi, M. Y. Rafii, and Azizah Abdul Hamid. "Effect of Salinity on Biomass Yield and Physiological and Stem-Root Anatomical Characteristics of Purslane (Portulaca oleraceaL.) Accessions." BioMed Research International 2015 (2015): 1–15. http://dx.doi.org/10.1155/2015/105695.

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13 selected purslane accessions were subjected to five salinity levels 0, 8, 16, 24, and 32 dS m−1. Salinity effect was evaluated on the basis of biomass yield reduction, physiological attributes, and stem-root anatomical changes. Aggravated salinity stress caused significant (P<0.05) reduction in all measured parameters and the highest salinity showed more detrimental effect compared to control as well as lower salinity levels. The fresh and dry matter production was found to increase in Ac1, Ac9, and Ac13 from lower to higher salinity levels but others were badly affected. Considering salinity effect on purslane physiology, increase in chlorophyll content was seen in Ac2, Ac4, Ac6, and Ac8 at 16 dS m−1salinity, whereas Ac4, Ac9, and Ac12 showed increased photosynthesis at the same salinity levels compared to control. Anatomically, stem cortical tissues of Ac5, Ac9, and Ac12 were unaffected at control and 8 dS m−1salinity but root cortical tissues did not show any significant damage except a bit enlargement in Ac12 and Ac13. A dendrogram was constructed by UPGMA based on biomass yield and physiological traits where all 13 accessions were grouped into 5 clusters proving greater diversity among them. The 3-dimensional principal component analysis (PCA) has also confirmed the output of grouping from cluster analysis. Overall, salinity stressed among all 13 purslane accessions considering biomass production, physiological growth, and anatomical development Ac9 was the best salt-tolerant purslane accession and Ac13 was the most affected accession.

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Dissertations / Theses on the topic "ACE"

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Ma, Chao. "ACE Student Tracker." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1306789622.

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Keenan, Frank. "Agile context enquiry (ACE)." Thesis, University of Ulster, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529536.

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Hutton, Thomas C. 1965. "ACE vs. Six Sigma." Thesis, Massachusetts Institute of Technology, 2004. http://hdl.handle.net/1721.1/17898.

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Thesis (M.B.A.)--Massachusetts Institute of Technology, Sloan School of Management, 2004.
Includes bibliographical references (leaf 68).
In the early 1980's and 1990's, companies began to build upon the principles of Total Quality Management and developed there own unique quality systems. The most popular and well known of these systems is Six Sigma that was developed by Motorola and successfully adopted by others such as Allied Signal (now Honeywell) and most notably, General Electric. Six Sigma can be characterized as a highly formalized, process oriented improvement tool that is data focused. The Six Sigma process is normally performed by a diverse team, who attack a quality/process problem by analyzing process variation or in statistical terms, sigma. The foundations of Six Sigma are commitment from upper management, detailed training and a regimented diagnostic approach. Another quality operating system is the less known, but very successful, Achieving Competitive Excellence (ACE) operating system. This system was developed and is practiced by United Technologies Corporation (UTC). The ACE system is broader based than the Six-Sigma approach, however, ACE is not as data oriented as the Six Sigma approach. ACE revolves around the three principle categories of process improvement and waste elimination tools, decision-making tools, and problem solving tools. These tools impact issues as diverse, but not limited to, factory floor cleanliness, market feedback analysis, machine tool preventative maintenance and set up reduction. ACE is a combination of lean manufacturing and quality improvement philosophies. This paper provides an analysis of both the Six Sigma and ACE Quality Operating Systems. In the paper the systems are compared and contrasted. Further, strengths and weaknesses of each system are discussed. In particular, the analysis focuses on how ACE can leverage elements
(cont.) and aspects of Six Sigma. The analysis concludes that there are elements of Six Sigma that would benefit ACE. The paper identifies that the strength of Six Sigma's statistical approach and its positive impact on process certification could be beneficially applied to the ACE system. Further, there are recommendations for UTC to place more of an emphasis on ACE training and to accelerate its current efforts to better link quality and lean improvement to product engineering and design.
by Thomas C. Hutton.
M.B.A.

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Ohgren, Michael. "Ace in the hole." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1400069423.

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Dolson, R., D. Morelen, Julia Dodd, and Andrea D. Clements. "Pocket Ace: Neglect of Child Sexual Abuse Survivors in the ACE Study Questionnaire." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etsu-works/7222.

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Dolson, Robyn A., Diana M. Morelen, Julia Dodd, and Andrea Clements. "Pocket Ace: Neglect of Child Sexual Abuse Survivors in the ACE Study Questionnaire." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/124.

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Twenty years ago, a seminal study on adverse childhood experiences (ACEs) and subsequent increased health risks catapulted ACEs into the zeitgeist of research and application. Though a validated construct, the questionnaire, particularly the child sexual abuse (CSA) item is not without limitation and yet is used by the Centers for Disease Control and state agencies to quantify need and allocate resources to services accordingly. Currently, CSA is counted only when the perpetrator is 5-years or older than the victim. This requirement makes neglect of sibling and peer assault very likely. Accordingly, this study aimed to assess whether individuals with CSA experiences within an age gap smaller than 5 years are missed by the 5-year modifier embedded in CSA assessment wording and whether this missed group would otherwise qualify for services if detected. The study also aimed to assess whether this missed group has equivalently poor health outcomes to CSA groups currently captured by the 5-year modifier and whether outcomes for all CSA groups were higher than those who did not have a CSA history. An international sample of 974 women aged 18 to 50 completed an online survey hosted by Reddit regarding their substance use, multiple domains of current health, and CSA history using the original ACEs questionnaire and an experimental version of the CSA item without the 5-year modifier. All statistical analyses were completed in R. Results indicated there was a group of survivors with CSA experiences missed by the 5-year modifier and this had implications for reducing their total ACE scores. This group was nearly equal in size to CSA groups captured by the 5-year modifier and demonstrated equivalently poor health and substance use outcomes. On nearly all variables, CSA groups demonstrated poorer health outcomes than those who had never experienced CSA. These findings suggest the language of how CSA is assessed must be thoughtfully revised to include all CSA experiences as all are equally at risk for adverse outcomes and thus all warrant consideration for services currently afforded those with CSA histories and high ACE scores.

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Kanakamedala, Keerthy. "ROLE OF ANGIOTENSIN CONVERTING ENZYMES ACE AND ACE2 IN DIABETES INDUCED CARDIOVASCULAR DYSFUNCTION." Wright State University / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=wright1196272791.

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Dolson, Robyn. "Pocket ACE: Neglect of Child Sexual Abuse Survivors in the ACEs Study Questionnaire." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etd/3573.

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In 1998, a seminal study on adverse childhood experiences (ACEs) and subsequent health risks catapulted ACEs and the study questionnaire into the zeitgeist. However, its childhood sexual abuse (CSA) item is problematic as it requires the perpetrator have been 5-years or older than the victim. To assess whether some survivors’ CSA is not identified by the current item, whether their exclusion prevents access to services requiring a four-threshold ACE score, and how their health outcomes compared to other CSA groups and controls, an international sample of 974 women completed an online survey assessing their current health and CSA history using the original item and an experimental item without the 5-year modifier. Results indicated many CSA survivors are not identified by a 5-year modifier, exclusion has service implications for some, and on most variables, they had increased adverse health outcomes compared to controls. Means of assessing CSA must be thoughtfully revised.

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Cecil, Anne N. "ACE tourism : trekking in Nepal." Honors in the Major Thesis, University of Central Florida, 2002. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/263.

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This item is only available in print in the UCF Libraries. If this is your Honors Thesis, you can help us make it available online for use by researchers around the world by following the instructions on the distribution consent form at http://library.ucf.edu/Systems/DigitalInitiatives/DigitalCollections/InternetDistributionConsentAgreementForm.pdf You may also contact the project coordinator, Kerri Bottorff, at kerri.bottorff@ucf.edu for more information.
Bachelors
Business Administration
Hospitality Management

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Обухова, Ольга Анатоліївна, Ольга Анатольевна Обухова, Olha Anatoliivna Obukhova, and K. M. Sheikh. "Ace gene and physical activity." Thesis, Publisher SumDU, 2010. http://essuir.sumdu.edu.ua/handle/123456789/6754.

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Books on the topic "ACE"

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Ace of aces. Florida: Leo Publishing LLC, 2013.

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Ace. London: Gollancz, 1991.

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Ace. Washington, DC: Word Works, 2009.

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Mike, Terry, ed. Ace. London: Puffin, 1999.

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1949-, Graham-Yooll Elizabeth, ed. Ace. Harmondsworth: Puffin Books, 1991.

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Carr, Richard. Ace. Washington, DC: Word Works, 2009.

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Dick Bong: America's ace of aces. 2nd ed. Superior, Wis: Richard I. Bong WWII Heritage Center, 2003.

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Bong, Carl. Ace of aces: The Dick Bong story. Mesa, Ariz: Champlin Fighter Museum Press, 1985.

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D’Orléans-Juste, Pedro, and Gérard E. Plante, eds. ACE Inhibitors. Basel: Birkhäuser Basel, 2001. http://dx.doi.org/10.1007/978-3-0348-7579-0.

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1957-, D'Orléans-Juste P., and Plante G. E, eds. ACE inhibitors. Basel: Birkhäuser Verlag, 2001.

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Book chapters on the topic "ACE"

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Hodges, John R., and Andrew J. Larner. "Addenbrooke’s Cognitive Examinations: ACE, ACE-R, ACE-III, ACEapp, and M-ACE." In Cognitive Screening Instruments, 109–37. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-44775-9_6.

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van den Meiracker, A. H. "ACE-remmers." In Het Cardiovasculair Formularium, 208–18. Houten: Bohn Stafleu van Loghum, 2010. http://dx.doi.org/10.1007/978-90-313-7366-6_17.

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Allgeier, J., and G. F. Hauf. "ACE-Hemmer." In Herzkrankheiten, 1365–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-97605-6_54.

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Chiu, M. C., U. I. Von-Mehlem, C. E. Willey, T. M. Betenbaugh, J. J. Maynard, J. A. Krein, R. F. Conde, et al. "Ace Spacecraft." In The Advanced Composition Explorer Mission, 257–84. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4762-0_13.

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Peter, Helga, and Thomas Penzel. "ACE-Hemmer." In Springer Reference Medizin, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-642-54672-3_290-1.

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Hitzenberger, G. "ACE-Hemmer." In Herzerkrankungen und Interventions-möglichkeiten, 331–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80404-5_18.

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Sturrock, Edward D., and K. Ravi Acharya. "ACE Inhibitors." In Encyclopedia of Molecular Pharmacology, 1–11. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_1-1.

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Sturrock, Edward D., and K. Ravi Acharya. "ACE Inhibitors." In Encyclopedia of Molecular Pharmacology, 8–17. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57401-7_1.

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van den Meiracker, A. H. "ACE-remmers." In Het cardiovasculair formularium, 263–74. Houten: Bohn Stafleu van Loghum, 2013. http://dx.doi.org/10.1007/978-90-313-9985-7_17.

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Cooper, Mark E. "ACE and diabetes." In ACE Inhibitors, 177–84. Basel: Birkhäuser Basel, 2001. http://dx.doi.org/10.1007/978-3-0348-7579-0_14.

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Conference papers on the topic "ACE"

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Wang, Di, Chuangang Ren, Sriram Govindan, Anand Sivasubramaniam, Bhuvan Urgaonkar, Aman Kansal, and Kushagra Vaid. "ACE." In the ACM SIGMETRICS/international conference. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2465529.2465536.

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Gordon, Kiel, Matthew Davis, Zachary Birnbaum, and Andrey Dolgikh. "ACE." In CCS '18: 2018 ACM SIGSAC Conference on Computer and Communications Security. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3264888.3264891.

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Raghavachari, Mukund, and Anne Rogers. "Ace." In the sixth ACM SIGPLAN symposium. New York, New York, USA: ACM Press, 1997. http://dx.doi.org/10.1145/263764.263777.

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Yan, Haijin, David K. Lowenthal, and Kang Li. "ACE." In the international workshop. New York, New York, USA: ACM Press, 2005. http://dx.doi.org/10.1145/1065983.1065992.

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Whyte, Todd C., and Darrell W. Starks. "ACE." In the 28th conference. New York, New York, USA: ACM Press, 1996. http://dx.doi.org/10.1145/256562.256941.

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Nath, Suman. "ACE." In the 10th international conference. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2307636.2307640.

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Byrne, Anthony, Shripad Nadgowda, and Ayse K. Coskun. "ACE." In Middleware '20: 21st International Middleware Conference. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3429880.3430098.

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Backes, Michael, Aniket Kate, and Esfandiar Mohammadi. "Ace." In the 2012 ACM workshop. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2381966.2381974.

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Strezoski, Gjorgji, Arumoy Shome, Riccardo Bianchi, Shruti Rao, and Marcel Worring. "ACE." In MM '19: The 27th ACM International Conference on Multimedia. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3343031.3350588.

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Bosco, Piergiorgio, Giovanni Martini, and Corrado Moiso. "TINA ACE." In the 19th international conference. New York, New York, USA: ACM Press, 1997. http://dx.doi.org/10.1145/253228.253503.

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Reports on the topic "ACE"

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Brauer, Douglas C., Daniel Henry, and George A. Matzkanin. ACE/AACE Inspection and Analysis Handbook. Part 1. Management. Fort Belvoir, VA: Defense Technical Information Center, June 1985. http://dx.doi.org/10.21236/ada210047.

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Brauer, Douglas C., Daniel Henry, and George A. Matzkanin. ACE/AACE Inspection and Analysis Handbook. Part 2. Engineering. Fort Belvoir, VA: Defense Technical Information Center, June 1985. http://dx.doi.org/10.21236/ada210048.

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Brauer, Douglas C., Daniel Henry, and George A. Matzkanin. ACE/AACE Inspection and Analysis Handbook. Part 3. Profiling. Fort Belvoir, VA: Defense Technical Information Center, June 1985. http://dx.doi.org/10.21236/ada210049.

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Feng-Berman, S. K. ACE program/UNIX user manual. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/6298543.

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Feng-Berman, S. K. ACE program/UNIX user manual. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/10165284.

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Jabbour, Kamal. Advanced Course in Engineering (ACE). Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada434301.

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McCoyd, Gerald, and Adrienne Raglin. ACE: Lessons Learned. Volume 1. Fort Belvoir, VA: Defense Technical Information Center, September 1992. http://dx.doi.org/10.21236/ada267979.

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Conlin, Jeremy Lloyd. Listing of Available ACE Data Tables. Office of Scientific and Technical Information (OSTI), January 2017. http://dx.doi.org/10.2172/1342828.

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O'Connor, Dennis J. Application Center of Excellence (ACE) Program. Fort Belvoir, VA: Defense Technical Information Center, October 1991. http://dx.doi.org/10.21236/ada248694.

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Conlin, Jeremy Lloyd, and Paul Romano. A Compact ENDF (ACE) Format Specification. Office of Scientific and Technical Information (OSTI), September 2019. http://dx.doi.org/10.2172/1561065.

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