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Academic literature on the topic 'Abhijit Sen'
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Journal articles on the topic "Abhijit Sen"
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Ashis Sengupta. "Abhijit Sen, <i>Rabindranath Tagore’s Theatre: From Page to Stage</i>." Asiatic: IIUM Journal of English Language and Literature 16, no. 1 (June 24, 2022): 190–94. http://dx.doi.org/10.31436/asiatic.v16i1.2503.
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Chakravartty, Gargi. "Shahin Akhtar and Mousumi Bhowmik (eds.), Janana Mahfil (Selected Writings of Bengali Muslim Women Writers 1904-1938). Calcutta: Stree. 1998. 316 pages. Rs. 220. Abhijit Sen and Abhijit Bhattacharya (eds.), Swarnakumari Devi's Collection of Essays (Bengali). Calcutta: Bikalpa Publication. 1998. 229 pages. Rs. 75." Indian Journal of Gender Studies 8, no. 2 (September 2001): 330–32. http://dx.doi.org/10.1177/097152150100800212.
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Chakravarty, Gargi. "Book Reviews : Sambuddha Chakravarty, Andare Antare: Unish Shatake Bangali Bhadramahila. Calcutta: Stree Prakashan, 1995. 274 pages. Rs. 150. Abhijit Sen and Jasodhara Bagchi (eds.), Shatavarshe Ashalata Sen. School of Women's Studies, Jadavpur University: Stree Prakashan, 1995. 95 pages. Rs. 65." Indian Journal of Gender Studies 4, no. 2 (September 1997): 281–84. http://dx.doi.org/10.1177/097152159700400210.
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Abraham, Joseph. "Abject Poverty and Multiple Deprivations in Rural India Based on SECC 2011 in Comparison with NSSO and NFHS: Summary Findings Analyzed." IRA-International Journal of Management & Social Sciences (ISSN 2455-2267) 6, no. 1 (February 10, 2017): 67. http://dx.doi.org/10.21013/jmss.v6.n1.p10.
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<em>This paper analyzes latest findings from the recently completed Socio Economic and Caste Census 2011(SECC2011), by focusing on rural abject poverty and multi-dimensionality of it by the pre-set seven deprivation parameters across rural India .As per schema of SECC2011 for analyzing the various facets of multi-dimensional poverty, firstly one set of households will be excluded on the basis of 13 automatic exclusion parameters, and subsequently another set of households will be automatically included on the basis of five parameters and finally the remaining set would be subjected to verifications by seven deprivations. Thereby, the SECC 2011 had set in motion an effort to capture some specifics of multidimensional poverty as desired by the Ministry of Rural Development (MoRD) in the Government of India. It is surmised here that the union of automatically included and deprived households will provide a base line of the number of poor through a multi-dimensional mode. The intersection of automatically included households with the seven deprivations variables will also identify the socio economic characteristics of the abjectly poor. Besides presenting the above analysis of SECC data, an attempt is made to compare these findings with those based on the unidimensional National Sample Survey (NSSO) poverty ratios ( by S.Tendulker 2009, C Rangarajan 2012) and multi-dimensional (R. Radhakrishna et al 2010) NFHS data based studies. A separate set of multi-dimensional poverty numbers were arrived at in the past for three Five Year Plans (1992-97, 1997- 02, 2002-07) through the Below Poverty Line (BPL) Censuses that were under taken by the Ministry of Rural Development (MoRD) to identify the poor households through the State/UT Governments. These later estimates of poor households were never permitted to exceed the official poverty ratio worked out by the Planning Commission for respective State/UT governments. The concepts used to arrive at these poor households will be briefly reviewed here as a prelude to explaining the modes operandi of identifying multi dimensional poverty via SECC 2011. A committee was set in up in February 2013 under the Chairmanship of Abhijit Sen , then Planning Commission Member, to examine the SECC indicators for data analysis, to recommend appropriate methodologies for determining classes of beneficiaries for different rural development programmes. Some of the recommendations of the committee would also be put to scrutiny. </em>
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Dr. Dilip Bhatt. "Application of Rasa Theory in Ravindranath Tagore’s The Waterfall." International Peer Reviewed E Journal of English Language & Literature Studies - ISSN: 2583-5963 1, no. 2 (December 10, 2019): 1–7. http://dx.doi.org/10.58213/ell.v1i2.9.
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Veer Rasa or Heroic has its roots in the higher kind of people, splendour, enormity, goodness,power, liveliness, and the ability to take action. Attention to detail and a strong desire forsuccess can be seen in this character's demeanour. Strength and endurance are two traits thatoften indicate a person's potential for heroism. In other words, when faced with danger orperil, one is firm and does not budge. A strong sense of persistence supports this firmness.The dynamic energy or the spirit of veer is what keeps them both going. The traits offirmness and patience are sustained by a positive outlook and energy. The focus of this paperis on the play's intensity and heroism. When an engineer named Bibhuti offers to build a damon the river, the King of Uttarkut accepts. Thus, the villagers' livelihoods are impacted: theirfishing, farming, and community activities are all disrupted by the disaster. Baba Dhananjaybegins a non-violent revolt at this critical juncture. The public's movement grows strongerand more rebellious over time. Abhijit, the king's son, also succeeds in bursting the dam bysacrificing himself. The Muktadhara River has been restored to its natural state. This paperalso discusses issues related to the human-machine conflict.
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Putri, Indah Merdeka, and Ali Muhyidin. "Cambodian and Thai Political Actors Interest in Preah Vihear Temple Border Conflict in 2008-2011." Jurnal Politik 4, no. 2 (March 31, 2019): 215. http://dx.doi.org/10.7454/jp.v3i2.73.
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This paper examines how domestic interest of political actors in particular country may spark conflict and create reconciliation with other country. The case of the Preah Vihear border temple dispute between Cambodia and Thailand in 2008-2011 shows that distinctive political interest within each country has made the the relation of both countries in up and down situation, in term of escalating conflict or initiating reconciliation, in that period of time. By discussing the interest of Cambodia’s political actor represented by Prime Minister Hun Sen with the Cambodian's People Party (CPP) and Thailand’s elites represented by Prime Minister Abhisit Vejjajiva with the Democracy Party (PD) and, later, by Yingluck Shinawatra with the Pheu Thai Party, the article argues that Preah Vihear conflict has been used by these political actors for their domestic political gains. Based on this study, it can be concluded that a border conflict can be basically influenced by political actor interest to maintain or gain domestic power. By underlining the domestic political actor interest, this study gives a different dimension compare to other studies at the same topic that tend to explain the conflict by using descriptive or chronological approach.
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Dozmorov, Mikhail, Maggie Marshall, Narmeen Rashid, Jacqueline Grible, Aaron D. Valentine, Amy Olex, Kavita Murthy, et al. "Abstract P1-13-23: Carboplatin resistance-associated changes in the 3D chromatin landscape of a triple-negative breast cancer Patient-Derived Xenograft." Cancer Research 83, no. 5_Supplement (March 1, 2023): P1–13–23—P1–13–23. http://dx.doi.org/10.1158/1538-7445.sabcs22-p1-13-23.
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Abstract Changes in the three-dimensional (3D) structure of the genome are an emerging hallmark of cancer. Cancer-associated copy number variants and single nucleotide polymorphisms promote rewiring of chromatin loops, disruption of topologically associating domains (TADs), active/inactive chromatin state switching, leading to oncogene expression and silencing of tumor suppressors. However, little is known about 3D changes during cancer progression to a chemotherapy-resistant state. We integrated chromatin conformation capture (Hi-C), RNA-seq, and whole-genome sequencing obtained from triple-negative breast cancer patient-derived xenograft primary tumors (UCD52) and carboplatin-resistant samples and found increased short-range (< 2Mb) interactions, chromatin looping, formation of topologically associating domains (TAD), chromatin state switching into a more active state, and amplification of ATP-binding cassette (ABC) transporters. Transcriptome changes suggested the role of long-noncoding RNAs in carboplatin resistance. Rewiring of the 3D genome was associated with TP53, TP63, BATF, FOS-JUN family of transcription factors and led to activation of aggressiveness-, metastasis- and other cancer-related pathways. Integrative analysis highlighted increased ribosome biogenesis and oxidative phosphorylation, suggesting the role of mitochondrial energy metabolism. Our results suggest that 3D genome remodeling may be a key mechanism underlying carboplatin resistance. Citation Format: Mikhail Dozmorov, Maggie Marshall, Narmeen Rashid, Jacqueline Grible, Aaron D. Valentine, Amy Olex, Kavita Murthy, Abhijit Chakraborty, Joaquin Reyna, Daniela Salgado Figueroa, Da-Inn Lee, Brittany Baur, Sushmita Roy, Ferhat Ay, Chuck Harrell. Carboplatin resistance-associated changes in the 3D chromatin landscape of a triple-negative breast cancer Patient-Derived Xenograft [abstract]. In: Proceedings of the 2022 San Antonio Breast Cancer Symposium; 2022 Dec 6-10; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2023;83(5 Suppl):Abstract nr P1-13-23.
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Moyer, Cassandra L., Abhijit Mazumdar, Jamal Hill, Martin E. Sanders, and Powel Brown. "Abstract P4-01-14: The RXR agonist, IRX4204, increases the anti-tumor activity of HER2-targeted therapies in HER2-amplified breast cancer." Cancer Research 82, no. 4_Supplement (February 15, 2022): P4–01–14—P4–01–14. http://dx.doi.org/10.1158/1538-7445.sabcs21-p4-01-14.
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Abstract Background: HER2 targeted monoclonal antibodies and tyrosine kinase inhibitors (TKI) are effective treatments for HER2-amplified primary tumors. However, the challenge persists that primary and acquired resistance to anti-HER2 therapy is common, and that anti-HER2 targeted therapies fail to achieve a cure in the metastatic setting. There is a critical need for safe and effective therapies that can overcome anti-HER2 drug resistance and eliminate breast cancer metastases. Here, we present data demonstrating the activity of IRX4204, a highly specific agonist of the nuclear retinoid X receptor (RXR), to inhibit the growth of HER2-amplified breast cancer cell lines alone and in combination with current anti-HER2 therapies. Methods: In vitro cell growth assays were conducted to evaluate the efficacy of IRX4204 alone and in combination with trastuzumab, tucatinib, neratinib or T-DM1, using a panel of breast cancer cell lines expressing various levels of HER2. A calculated coefficient of drug interaction (CDI) and isobologram analysis was used to assess the additive, synergistic or antagonistic effects of IRX4204 with each anti-HER2 targeted therapy. Results: IRX4204 treatment alone inhibited the growth of HER2-overexpressing cell lines, SkBr3, AU565, HCC1419 and ZR75-1 in vitro, but not the growth of HER2-normal cell lines MCF7, MDA-MB-231, or HCC1143. When combined with trastuzumab, tucatinib, neratinib or T-DM1, additive or synergistic effects were observed in all of the drug-sensitive cell lines. Moreover, IRX4204 in combination with neratinib or tucatinib inhibited the growth of a TKI-resistant breast cancer cell line (HCC1954). Conclusion: These data demonstrate a novel use of an RXR agonist, IRX4204, to inhibit the growth of HER2-amplified breast cancer cell lines and to enhance the activity of anti-HER2 therapy in vitro. These results also demonstrate that IRX4204 can overcome anti-HER2 resistance to inhibit the growth of HER2-positive cells. These data demonstrate that IRX4204 can enhance the anti-cancer activity of anti-HER2 therapies even in TKI-resistant cells. In vivo experiments and mechanistic studies are ongoing. Citation Format: Cassandra L Moyer, Abhijit Mazumdar, Jamal Hill, Martin E Sanders, Powel Brown. The RXR agonist, IRX4204, increases the anti-tumor activity of HER2-targeted therapies in HER2-amplified breast cancer [abstract]. In: Proceedings of the 2021 San Antonio Breast Cancer Symposium; 2021 Dec 7-10; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2022;82(4 Suppl):Abstract nr P4-01-14.
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Szabo, Elizabeth, Emily Blackburn, Alexander Radecki, Abhijit Rath, and Christopher Heinen. "Abstract 5615: Functional analysis of variants of uncertain significance in the MSH6 mismatch repair gene." Cancer Research 84, no. 6_Supplement (March 22, 2024): 5615. http://dx.doi.org/10.1158/1538-7445.am2024-5615.
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Abstract Lynch syndrome (LS) is a hereditary condition that increases patients’ lifetime risk of cancer, primarily colorectal cancer. LS is caused by germline mutations in mismatch repair (MMR) genes, MLH1, MSH2, MSH6, and PMS2. Identification and classification of these mutations is important in LS diagnosis for guiding treatment plans and preventative care for patients’ and their families. However, the consequences of some variants in these genes are not immediately obvious, granting them classification as variants of uncertain significance (VUS). Laboratory functional analysis to determine whether these variants disrupt MMR function in human cells can provide important evidence for understanding the pathogenicity of VUS and reclassifying them. Half of known variants in the MMR gene MSH6 are VUS and, thus, the goal of my project is to provide evidence for a subset of these variants to help provide evidence for reclassification for these variants. We are calibrating and validating functional assays for MSH6 variants, using known MSH6 pathogenic and benign controls. We will then test MSH6 VUS identified in patients as reported in the InSiGHT database, ClinVar database, and current literature. We are using CRISPR gene editing to recreate these variants in the endogenous MSH6 loci in human embryonic stem cells (hESC). Using these edited hESC lines we are examining the impact of the variants on RNA and protein stability, repair of DNA microsatellites, which is a hallmark of normal MMR function, and induction of the MMR-dependent DNA damage response. With this compilation of data, we will convert the results into a quantitative Odds of Pathogenicity value. This Odds of Pathogenicity score can be used by expert variant interpretation committees to help readily reclassify these variants and provide a clearer diagnosis for these suspected Lynch syndrome patients. Citation Format: Elizabeth Szabo, Emily Blackburn, Alexander Radecki, Abhijit Rath, Christopher Heinen. Functional analysis of variants of uncertain significance in the MSH6 mismatch repair gene [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2024; Part 1 (Regular Abstracts); 2024 Apr 5-10; San Diego, CA. Philadelphia (PA): AACR; Cancer Res 2024;84(6_Suppl):Abstract nr 5615.
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Xiao, Lanbo, Abhijit Parolia, Yuanyuan Qiao, Pushpinder Bawa Pushpinder, Sanjana Eyunni, Rahul Mannan, Sandra E. Carson, et al. "Abstract 5469: Targeting SWI/SNF ATPases in enhancer-addicted prostate cancer." Cancer Research 82, no. 12_Supplement (June 15, 2022): 5469. http://dx.doi.org/10.1158/1538-7445.am2022-5469.
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Abstract The switch/sucrose non-fermentable (SWI/SNF) complex plays a crucial role in chromatin remodeling and is recurrently altered in over 20% of human cancers. Here, we developed a proteolysis targeting chimera (PROTAC) degrader of ATPase subunits of the SWI/SNF complex, SMARCA2 and SMARCA4. Intriguingly, we found androgen receptor (AR)/forkhead box A1 (FOXA1)-positive prostate cancer to be exquisitely sensitive to dual SMARCA2 and SMARCA4 degradation relative to benign prostate as well as other cancer cell lines, including those with inactivating SMARCA4 mutations. Mechanistically, SWI/SNF inhibition rapidly compacts the cis-regulatory elements that are bound and activated by transcription factors that drive cancer proliferation, namely AR, FOXA1, ERG, and MYC. This ensues in chromatin untethering of these oncogenic drivers, chemical decommissioning of their core enhancer circuitry, and attenuation of downstream gene programs. Furthermore, we found SWI/SNF inhibition to disrupt super-enhancer and promoter DNA looping interactions that wire supra-physiologic expression of the AR, FOXA1, and MYC oncogenes, thereby tempering their expression in cancer cells. Monotherapy with the SMARCA2/4 degrader induced potent inhibition of tumor growth in cell line-derived xenograft models of prostate cancer and remarkably synergized with AR antagonists, inducing disease remission in models of castration-resistant prostate cancer. We also found the combinatorial treatment to significantly inhibit the growth of enzalutamide resistant disease using in vitro as well as patient-derived xenograft models. Notably, no major toxicities were seen in mice upon prolonged treatment with the SMARCA2/4 degrader, including no indications of thrombocytopenia, gastrointestinal goblet cell depletion, or germ cell degeneration. Taken together, these results suggest that impeding enhancer accessibility through SWI/SNF ATPase inactivation represents a novel therapeutic approach in enhancer addicted human cancers. Citation Format: Lanbo Xiao, Abhijit Parolia, Yuanyuan Qiao, Pushpinder Bawa Pushpinder, Sanjana Eyunni, Rahul Mannan, Sandra E. Carson, Yu Chang, Xiaoju Wang, Yuping Zhang, Josh Vo, Steven Kregel, Stephanie A. Simko, Andrew D. Delekta, Mustapha Jaber, Heng Zheng, Ingrid Apel, Lisa McMurry, Fengyun Su, Rui Wang, Sylvia Wang, Sanjita Sasmal, Leena K. Satyam, Subhendu Mukherjee, Chandrasekhar AbbinenI, Kiran Aithal, Mital S. Bhakta, Jay Ghurye, Xuhong Cao, Nora M. Navone, Alexey Nesvizhskii, Rohit Mehra, Ulka Vaishampayan, Marco Blanchette, Yuzhuo Wang, Susanta Samajdar, Murali Ramachandra, Arul M. Chinnaiyan. Targeting SWI/SNF ATPases in enhancer-addicted prostate cancer [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 5469.
Books on the topic "Abhijit Sen"
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Singh, Abhijit. India’s Naval Interests in the Pacific. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199479337.003.0011.
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Indian naval analyst, Abhijit Singh examines the reasons behind India’s naval engagement in Southeast Asia and Indian perspectives of China’s activities in the South China Sea. Singh argues that in recent years, there has been a discernable shift in India’s maritime posture in the Pacific. While the Indian Navy still identifies the Western Pacific as a secondary area of interest, its operational deployments to Southeast Asia have been gradually rising, signalling an enhanced appreciation of Indian strategic stakes in the region. In many ways, India’s principal drivers for security operations in the Pacific have their origins in the Indian Ocean where New Delhi has for long harboured geopolitical ambitions.