Relevant bibliographies by topics / Abdominal aneurysm

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Academic literature on the topic 'Abdominal aneurysm'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 January 2023

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Journal articles on the topic "Abdominal aneurysm"

1

Genyk, Stepan. "Aneurysmal Disease of the Main Arteries." Archive of Clinical Medicine 22, no. 2 (December 8, 2016): 201627. http://dx.doi.org/10.21802/acm.2016.2.7.

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The high incidence of aneurysms of different arterial systems in case of abdominal aortic aneurysm causes the need for the examination of all patients with aortic aneurysm in order to detect aneurysmal process in all main arteries. The use of the mentioned predictors of aneurism wall failure in daily clinical practice provides an opportunity to improve the results of surgical treatment. Active surgical approach in relation to aortic aneurysm and main arteries is indicated in the presence of aneurismal disease. The choice of treatment depends on the severity of the patient’s condition, clinical manifestations, localization of the aneurysm and its morphology, with the preference for endovascular approach.
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Sebayang, Abed Nego Okthara, and Niko Azhari Hidayat. "Endovascular Aortic Repair (EVAR) Method in The Management of Abdominal Aortic Aneurysm." SCRIPTA SCORE Scientific Medical Journal 2, no. 1 (August 28, 2020): 53–7. http://dx.doi.org/10.32734/scripta.v2i1.3530.

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Aortic disease is a collection of diseases of the aorta, which includes aortic aneurysms; acute aortic infections consisting of aortic dissection, intramular hematoma, penetration of atherosclerotic ulcers (PAU) and traumatic injury to the aorta; pseudoaneurysm; aortic rupture; Marfan syndrome; and congenital abnormalities such as coarctation of the aorta. One of the aortic diseases that cause the death rate to increase according to the 2010 Global Burden Disease is aortic aneurysm. Abdominal aortic aneurysm (AAA) is a focal dilatation of the aortic segment. The diagnosis of AAA is done by history taking, physical examination and supporting examination. Management at AAA aims to prevent aortic wall rupture. An alternative procedure without open surgery is endovascular aortic repair (EVAR) using prostheses. It is expected that through the EVAR method, mortality and morbidity due to AAA can be reduced. Keywords: abdominal aortic aneursym, EVAR, prostheses Penyakit aorta merupakan kumpulan penyakit pada aorta yang meliputi aneurisma aorta; sindrom aorta akut berupa diseksi aorta, hematoma intramular, penetrating atherosclerosis ulcer (PAU) dan cedera akibat trauma pada aorta; pseudoaneurysm; ruptur aorta; sindrom Marfan; serta penyakit kongenital seperti koarktasio aorta. Salah satu penyakit aorta yang menyebabkan angka kematian meningkat menurut Global Burden Disease 2010 adalah aneurisma aorta. Aneurisma aorta abdominalis (AAA) merupakan dilatasi fokal pada segmen aorta. Penegakan diagnosis AAA dilakukan dengan anamnesis, pemeriksaan fisik dan pemeriksaan penunjang. Penatalaksanaan pada AAA bertujuan untuk mencegah pecahnya dinding aorta. Prosedur alternatif tanpa pembedahan terbuka yang dijadikan pilihan adalah endovascular aortic repair (EVAR) menggunakan protesa. Diharapkan melalui metode EVAR angka mortalitas dan morbiditas akibat AAA dapat diturunkan. Kata kunci: aneurisma aorta abdominalis, EVAR, protesa
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Pereira, Thiago Scremin Boscolo, Vanessa Belentani Marques, Elizandra Moura dos Santos, Ana Letícia Daher Aprígio da Silva, Eduardo Martini Romano, and Carla Patricia Carlos. "Association Between Abdominal Aortic and Common Iliac Artery Aneurysms: Case Report." Journal of Biology and Life Science 10, no. 2 (June 30, 2019): 71. http://dx.doi.org/10.5296/jbls.v10i2.14714.

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The infrarenal abdominal aortic aneurysm is the most common among arterial aneurysms; it happens when there is an abnormal and irreversible enlargement of the blood vessel. This disease usually compromises other arterial segments and is linked to high mortality rates, mainly due to its rupture. Given its importance, we present a case study of an abdominal aortic aneurysm associated with a common iliac artery aneurysm. During a dissection practice in the Morphofunctional Laboratory at FACERES Medical School, we observed the presence of a mild stenosis in the abdominal aorta below the renal arteries, as well as the formation of an infrarenal abdominal aortic aneurysm. In addition, we noticed that the infrarenal abdominal aortic aneurysm was associated with a bilateral common iliac artery aneurysm. Morphological analyses carried out in the blood vessels showed a large quantity of atheromatous plaques, which are the probable cause of the pathology. The information herein may broaden the knowledge on the infrarenal abdominal aortic aneurysmal disease, thus reducing its complications and mortality rates.
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Weiss, Norbert, Roman N. Rodionov, and Adrian Mahlmann. "Medical management of abdominal aortic aneurysms." Vasa 43, no. 6 (November 1, 2014): 415–21. http://dx.doi.org/10.1024/0301-1526/a000388.

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Abdominal aortic aneurysms (AAA) are the most common arterial aneurysms. Endovascular or open surgical aneurysm repair is indicated in patients with large AAA ≥ 5.5 cm in diameter as this prevents aneurysm rupture. The presence even of small AAAs not in need of immediate repair is associated with a very high cardiovascular risk including myocardial infarction, stroke or cardiovascular death. This risk by far exceeds the risk of aneurysm rupture. These patients therefore should be considered as high-risk patients and receive optimal medical treatment and life-style modificiation of their cardiovascular risk factors to improve their prognosis. In addition, these patients should be followed-up for aneurysm growth and receive medical treatment to decrease aneurym progression and rupture rate. Treatment with statins has been shown to reduce cardiovascular mortality in these patients, and also slows the rate of AAA growth. Use of beta-blockers, ACE inhibitors and AT1-receptor antagonists does not affect AAA growth but may be indicated for comorbidities. Antibiotic therapy with roxithromycin has a small effect on AAA growth, but this effect must be critically weighed against the potential risk of wide-spread use of antibiotics.
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Batinic, Nikola, Tijana Kokovic, Dragan Nikolic, Vladimir Manojlovic, Viktor Till, and Slavko Budinski. "The impact of abdominal aortic aneurysm diameter on the outcome of endovascular aortic repair." Medical review 74, no. 11-12 (2021): 347–53. http://dx.doi.org/10.2298/mpns2112347b.

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Introduction. Abdominal aortic aneurysm diameter is one of the most important parameters in the diagnostic and therapeutic algorithm for aneurysm follow-up. Currently, two therapeutic modalities are used: open surgery and endovascular aortic repair. The aim of this study is to analyze the impact of the maximum transverse diameter of the abdominal aortic aneurysm on the incidence of general and specific complications. Material and Methods. The retrospective study included 75 patients with infrarenal abdominal aortic aneurysm who underwent endovascular aortic repair in the period from July 2008 to January 2020. The patients were divided into two groups: group A with an abdominal aortic aneurysm size ? 5.9 cm, and group B with an abdominal aortic aneurysm size ? 6.0 cm. Results. A total of 41.3% of patients presented with a maximum transverse aneurysm diameter of ? 5.9 cm, and 58.7% of patients had ? 6.0 cm. Of comorbid diseases, chronic obstructive pulmonary disease was more prevalent in patients with a large abdominal aortic aneurysm (group A 25.8%; group B 59.1%). None of the other comorbidities showed a statistically significant difference between the two groups of patients. Early complications were present in a total of 14.7% of patients, of which 12.9% of patients with a small and 15.9% with a large abdominal aortic aneurysm. Late complications occurred in a total of 18.7% of patients, in 9.7% of patients with a small and 25% of patients with a large abdominal aortic aneurysm. Conclusion. Patients with abdominal aortic aneurysms with a maximum transverse diameter of 6 cm and larger, present with a higher rate of late postoperative complications, increase in aneurysmal sac on control multislice computed tomography angiography, and have a worse prognosis compared to patients with smaller abdominal aortic aneurysms.
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Babic, Srdjan, Petar Popov, Miroslav Milicic, Nenad Ilijevski, Dragoslav Nenezic, Slobodan Tanaskovic, Predrag Gajin, et al. "Surgery of infrarenal inflammatory aneurysm of abdominal aorta infected with methicillin resistant Staphylococcus aureus in a patient undergoing haemodialysis." Srpski arhiv za celokupno lekarstvo 136, no. 9-10 (2008): 529–32. http://dx.doi.org/10.2298/sarh0810529b.

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INTRODUCTION Inflammatory abdominal aortic aneurysm accounts for 5% to 10% of all cases of abdominal aortic aneurysm and differs from typical atherosclerotic abdominal aortic aneurysm in many important ways. Although both inflammatory and atherosclerotic abdominal aortic aneurysms most commonly affect the infrarenal portion of the abdominal aorta, patients with the inflammatory variant are younger and usually symptomatic, chiefly from back or abdominal pain. Unlike patients with atherosclerotic abdominal aortic aneurysm, most with the inflammatory variant have an elevated erythrocyte sedimentation rate or abnormalities of other serum inflammatory markers. Computed tomography and magnetic resonance imaging are both sensitive for demonstrating the cuff of soft tissue inflammation surrounding the aneurysm that is characteristic of inflammatory abdominal aortic aneurysm. Inflammatory abdominal aortic aneurysm can be primarily infected by degenaration of an infected artery (in less than 1% of cases), or can become secondary infected in the already existing aneurysm. Secondary infection of the pre-existing aneurysm has big influence on treatment choice, but is also rare. Clinically non-symptomatic infection, also known as bacterial collonisation, can be very frequent, regarding a greatly increased number of positive intraoperative findings (10-15%). Prolonged intravascular catheterization, vascular grafting, repeated punctures with large bore needles, and decreased immune defense mechanism make uraemic patients undergoing hemodialysis more likely to develop Staphylococcus aureus bacteraemia and its complications. CASE OUTLINE The case shows a gigantic inflammatory aneurysm of the abdominal aorta, localized infrarenally, which was solved successfully by resection of the aneurysm of the abdominal aorta, and interposition of Dacron tubular graft 22 mm. Bacterial examination of the aneurysmal sac was positive: methicillin-resistent Staphylococcus aureus was detected. CONCLUSION There were no postoperative complications, and the final outcome was fully satisfactory. Control CT scans after 3, 6 and 12 months were regular, with signs of regression fibrosis of the retroperitoneum.
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Kostic, Dusan, Lazar Davidovic, Drago Milutinovic, Radomir Sindjelic, Marko Dragas, and Momcilo Colic. "Ex vivo repair of renal artery aneurysm associated with surgical treatment of abdominal aortic aneurysm." Srpski arhiv za celokupno lekarstvo 132, no. 7-8 (2004): 250–53. http://dx.doi.org/10.2298/sarh0408250k.

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INTRODUCTION Renal artery aneurysms is relatively uncommon with reported incidence ranges from 0.3% to 1%. However, considering all visceral artery aneurysms the percentage of renal artery aneurysms is relatively high between 15-25%. The distal forms of renal artery aneurysms sometimes require "ex vivo" reconstruction and kidney autotransplantation. CASE REPORT A 75-year-old male presented with the right abdominal and back pain. He suffered from a long history of arterial hypertension and chronic renal failure over the last few months (urea blood = 19.8 mmol/l; creatinine = 198 mmol/l). Duplex ultrasonography showed abdominal aortic aneurysm. Subsequent translumbarangiography revealed juxtarenal abdominal aortic aneurysm associated with distal right renal artery aneurysm. The operation was performed under combined thoracic epidural analgesia and general anesthesia using transperitoneal approach. After the laparotomy, the ascending colon was mobilized and reflected medially followed by Kocher maneuver. The result was visualization of the anterior aspect of the right kidney, the collecting system, ureter as well as the right renal vein and artery with large saccular aneurysm located distally. After mobilization of the renal vessels and careful dissection of the ureter, the kidney was explanted. The operation was continued by two surgical teams. The first team performed abdominal aortic aneurysm resection and reconstruction with bifurcated Dacron graft. The second team performed ex vivo reparation of renal artery aneurysm. All time during the explantation, the kidney was perfused by Collins' solution. The saccular right renal artery aneurysm 4 cm in diameter was located at the kidney hilus at the first bifurcation. Three branches originated from the aneurysm. The aneurysm was resected completely. The longest and widest of three branches arising from the aneurysmal sac was end-to-end anastomized with 6 mm PTFE graft. After this intervention, one of shorter arteries was implanted into the long artery, and another one into PTFE graft. After 30 minutes of explanation, autotransplantation of the kidney into the right iliac fossa was performed. The right renal vein was implanted into the inferior vein cava, and PTFE graft into the right limb of Dacron graft. Immediately following the completion of both anastomoses, large volume of urine was evident. Finally, ureteneocystostomy was performed with previous insertion of double "J" catheter. In the immediate postoperative period, renal function was restored to normal, while postoperative angiography revealed all patent grafts. DISCUSSION The most common causes of renal artery aneurysms are arteriosclerosis, as in our case, and fibro-muscular dysplasia. Very often, renal artery aneurysms are asymptomatic and discovered only during angiography in patients with aneurysmal and occlusive aortic disease. Other cases include: arterial hypertension, groin pain and acute or chronic renal failure. Due to relatively small number of evaluated cases, the risk of aneurysmal rupture is not known. According to some authors, the overall rupture rate of renal artery aneurysm is 5%, however, the rupture risk becomes higher in young pregnant woman. Several standard surgical procedures are available for the repair of renal artery aneurysms. These include saphenous vein angioplasty, bypass grafting, as well as ex vivo reconstruction with reimplantation or autotransplantation. Furthermore, interventional embolization therapy, as well as endovascular treatment with ePTFE covered stent, or autologous vein-coverage stent graft, have been also reported to be successful. CONCLUSION The major indications for surgical treatment of renal artery aneurysms are to eliminate the source of thromboembolism which leads to fixed renal hypertension and kidney failure, as well as prevention of aneurysmal rupture.
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Baykova, A. V., A. Ya Bedrov, A. A. Moiseev, and V. V. Baykov. "Gender based differences in histopathology of abdominal aorta in patients with abdominal aortic aneurysm and aortoiliac occlusive disease." Scientific Notes of the Pavlov University 29, no. 3 (July 4, 2022): 106–17. http://dx.doi.org/10.24884/1607-4181-2022-29-3-106-117.

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Relevance. Evidence exists that infrarenal aortic aneurisms and aortoiliac occlusive disease in women are different than in men in terms of anatomical features and more severe clinical course. Gender differences in histopathology of abdominal aorta are not fully studied.The objective was to study gender based differences in histopathology of abdominal aorta in patients with aortic aneurisms and aortoiliac occlusive disease.Methods and materials. The study included 96 biopsy specimens from 71 patients with aorto-iliac lesions (23 women and 48 men). A number of morphological characteristics and expression of matrix metalloproteinase 9 (MMP-9) were analyzed. The data obtained were processed statistically.Results. Adventitial infiltrate and medial sclerosis are more intense in the aneurysm than in occlusive-stenotic lesions, differences in the grade of fibrosis were proven only in men. Regardless of the patients’ gender, adventitial infiltration is denser in the aneurysmal body than in the neck; other changes in the anatomic parts of the aneurysm are similar. MMP-9 expression in the adventitia in the aneurysmal neck is higher in women, than in men. MMP-9 expression in the media and adventitia is higher in men with aneurysm, then with occlusive or stenotic lesion. Almost all indices of the aortic wall remodeling and MMP-9 expression correlate with each other in men with the aneurysm, in contrast to women.Conclusion. Gender based features in histopathology of abdominal aorta and the degree of their correlation may determine differences in the anatomy and course of abdominal aortic aneurysm and aortoiliac occlusive disease in women and men.
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Rajab, T., Miriam Beyene, Farhang Yazdchi, and Matthew Menard. "Aortic Aneurysm Eroding into the Spine." AORTA 06, no. 02 (April 2018): 068–69. http://dx.doi.org/10.1055/s-0038-1669416.

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AbstractAortic aneurysms are usually asymptomatic until catastrophic rupture occurs. Ruptured abdominal aortic aneurysms classically present with acute back pain, shock, and a pulsatile abdominal mass. The natural history of some aortic aneurysms also includes a stage of contained rupture. This occurs when extravasation of blood from the ruptured aneurysm is contained by surrounding tissues. Here, the authors report the case of a chronic contained abdominal aortic aneurysm rupture that resulted in erosion of the spine.
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da Silva, Erasmo S., Vitor C. Gornati, Ivan B. Casella, Ricardo Aun, Andre EV Estenssoro, Pedro Puech-Leão, and Nelson De Luccia. "The similarities and differences among patients with abdominal aortic aneurysms referred to a tertiary hospital and found at necropsy." Vascular 23, no. 4 (September 23, 2014): 411–18. http://dx.doi.org/10.1177/1708538114552095.

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Objective To analyze the characteristics of patients with abdominal aortic aneurysms referred to a tertiary center and to compare with individuals with abdominal aortic aneurysm found at necropsy. Methods We have retrospectively analyzed the medical records of 556 patients with abdominal aortic aneurysm and 102 cases abdominal aortic aneurysm found at necropsy. Results At univariated analysis, hypertension, tobacco use and maximum diameter were significant risk factors for symptomatic aneurysm, while diabetes tended to be a protective factor for rupture. By logistic regression analysis, the largest transverse diameter was the only one significantly associated with abdominal aortic aneurysm rupture ( p < .0001, odds ratio 1.7, 95% confidence interval 1.481–1.951). Intact abdominal aortic aneurysm found at necropsy showed similarities with outpatients in relation to abdominal aortic aneurysm diameter and risk factors. Conclusion Intact abdominal aortic aneurysm at necropsy and at outpatients setting showed similarities that confirmed that abdominal aortic aneurysm repair is less offered to women, and they died more frequently with intact abdominal aortic aneurysm from other causes.
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Dissertations / Theses on the topic "Abdominal aneurysm"

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Rossaak, Jeremy Ian, and n/a. "The genetics of abdominal aortic aneurysms." University of Otago. Dunedin School of Medicine, 2004. http://adt.otago.ac.nz./public/adt-NZDU20070502.143818.

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Abdominal Aortic Aneurysms (AAA) are amongst the top ten most common cause of death in those over 55 years of age. The disease is usually asymptomatic, often being diagnosed incidentally. Once diagnosed, elective repair of an AAA results in excellent long-term survival with a 3-5% operative mortality. However, up to one half of patients present with ruptured aneurysms, a complication that carries an 80% mortality in the community, and of those reaching hospital, a 50% mortality. Clearly early diagnosis and treatment results in improved survival. Screening for AAA, with ultrasound, would detect aneurysms early, prior to rupture. However, debate continues over the cost effectiveness of population based screening programmes. The identification of a sub-population at a higher risk of developing AAA would increase the yield of a screening prograrmne. A number of populations have been examined, none of which have received international acceptance. About 20% of patients with an AAA have a family history of an aneurysm. The disease is also considered to be a disease of Caucasians, both facts suggesting a strong genetic component to the disease. Perhaps a genetically identified sub-population at a high risk of developing an AAA would prove to be an ideal population for screening. This thesis examines the incidence of aneurysms and the family histories of patients with AAA in the Otago region of New Zealand. Almost twenty percent of the population has a family history of AAA. DNA was collected from each of these patients for genetic analysis. The population was divided into familial AAA and non-familial AAA for the purpose of genetic analysis and compared to a control population. AAA is believed to be a disease of Caucasians; a non-Caucasian population with a low incidence of AAA may prove to be a good control population for genetic studies. A literature review demonstrated a higher incidence of AAA in Caucasians than other ethnic groups and within Caucasians a higher incidence in patients of Northern European origin. The incidence was low in Asian communities, even in studies involving of migrant Asian populations. The New Zealand Maori are believed to have originated from South East Asia, therefore could be expected to have a low incidence of AAA and would make an ideal control population for genetic studies. A pilot study was undertaken to examine the incidence of AAA in the New Zealand Maori. The age standardised incidence of AAA proved to be at least equal in Maori to non-Maori, with a more aggressive form of the disease in Maori, manifesting with a younger age at presentation and a higher incidence of ruptured aneurysms at diagnosis. It is well known that at the time of surgery, an AAA is at the end stage in its life. At this time, inflammation and matrix metalloproteinases (MMP) enzymes are prevalent within the aneurysm wall and have destroyed the wall of the aorta. One of the most important genetic pathways regulating these enzymes is the plasminogen activator inhibiter 1-Tissue plasminogen activator-plasmin pathway. Genetic analysis of this pathway demonstrated an association of the 4G5G polymorphism in the promoter of the PAl-1 gene with familial AAA. In this insertion:deletion polymorphism, the 5G variant binds an activator and repressor, resulting in reduced PAI-1 expression and ultimately increased MMP activation. This allele was associated with familial aneurysms, 47% versus 62% non-familial AAA and 61% controls (p=0.024). A polymorphism within the tissue plasminogen activator gene was also examined and no association was found with AAA. Another way the MMPs expression could be increased is from mutations or polymorphisms in their own genetic structure. Stromelysin 3 is itself a MMP capable of destroying the aortic wall and it has a role in activating other MMPs. A 5A6A insertion:deletion polymorphism exists in the promoter of this gene. The 5A allele variant results in increased stromelysin expression and is associated with AAA 46% versus 33% in controls p=0. 0006. The actions of the MMPs are themselves inhibited by the tissue inhibitors of matrix metalloproteinases. The TIMP genes have been sequenced; two polymorphisms have been identified in the non-coding promoter area of the TIMP 1 gene. Further studies are necessary to examine the effect of these polymorphisms. Inflammation has been implicated in aneurysm progression. One of the roles of the inflammatory cells found in an aneurysm is to deliver the MMP�s to the AAA. The HLA system is integral in controlling this inflammation and was therefore examined. From this series of studies it is concluded that there is a genetic component to AAA. This thesis presents the first genetic polymorphism associated with familial AAA and explores the role of a genetic pathway in the formation of AAA.
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Svensjö, Sverker. "Screening for Abdominal Aortic Aneurysm." Doctoral thesis, Uppsala universitet, Kärlkirurgi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-198677.

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Randomised controlled trials have demonstrated that mortality from Abdominal Aortic Aneurysm (AAA) can be cost-effectively reduced by ultrasound-screening of men. Evidence for screening women is insufficient. Reports of falling AAA incidence are emerging. In an effort to study screening for AAA in a contemporary setting, two cross-sectional multi-centre population-based studies of one-time screening of 65-year-old men, and 70-year-old women in Middle Sweden were undertaken. Cost-efficiency of one-time screening of 65-year-old men was evaluated in a decision-analysis model. Five-year outcomes in men invited to screening at age 65 and age 70, were studied in a longitudinal cohort study. A lower than expected (1.7%) prevalence of AAA in 65-year-old men was found, as well as a very low (0.4%) prevalence in 70-year-old women. Smoking was the dominating risk factor associated with AAA, but the association was stronger in women. The main cause of reduced contemporary prevalence was falling smoking rates in the population since 30 years. One-time screening of 65-year-old men was found to be cost-effective and deliver significant clinical impact. The cost per quality adjusted life-year gained, at 13-years follow-up, was €14706, which was below the recommended UK NICE threshold of €25000. 15 lives were saved by inviting 10000 to screening. Prevalence of AAA and the rate of incidental detection of AAAs in the population were important factors affecting cost-efficiency. New AAAs developed after 5 years in men screened normal at age 65, predominantly in men with sub-aneurysmal aortas (25-29mm) at 65, and smokers. The 5-year rate of AAA repair was high among men with screening detected AAAs, as was non-AAA related mortality. Ruptures were only documented among non-attenders. Conclusions: A lower than expected prevalence of AAA among 65-year-old men, an unchanged repair rate, and improved longevity of the elderly population was found. Although one-time screening for AAA was still cost-effective within a contemporary context, several issues need to be addressed; the threshold diameter for follow-up, the current rate of opportunistic detection of AAA in the population, re-screening of the entire population at a higher age, and targeted screening of smokers. Screening 70-year-old women who do not smoke is likely to be futile, thus ruling out population screening of women for AAA.
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Chinien, Ganessen. "Molecular genetics of abdominal aortic aneurysm." Thesis, King's College London (University of London), 2012. https://kclpure.kcl.ac.uk/portal/en/theses/molecular-genetics-of-abdominal-aortic-aneurysm(e269485a-e71a-41a7-9a8e-ae40eb968dd4).html.

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Abdominal aortic aneurysm (AAA) is a common disorder and a major cause of death. Pathological processes involved in AAA formation include inflammation, proteolysis, angiogenesis and apoptosis. It has also a strong familial predisposition with linkage studies identifying chromosomes 19q13 and 4q31 as susceptible loci. AAA is likely to be a polygenic disorder. Aims The aims of this study were to carry out a whole transcriptome analysis in order to identify novel genes and pathways that are differentially expressed between aneurysmal (AAA), atheromatous (AOD) and normal (NA) aortic tissue and to confirm a set of these differentially expressed genes using quantitative real time polymerase chain reaction (qRT-PCR). Methods RNA samples were prepared from full thickness aortic walls obtained during open repair of AAA, aortic bypass for AOD and transplant patients for NA. The quality of the RNA was assessed using the Bioanalyzer 2100 (Agilent) and Nanodrop. RNA was then reverse transcribed to cDNA which was then hybridised to the Human Genome (HG) -U133 plus 2.0 microarray (Affymetrix) that interrogates the whole human genome. The robustness of the genearray was assessed using data output quality control as defined by Affymetrix. Statistical analysis was then carried out using the GeneSpring software. Genes were considered to be significantly differentiated if they had at least a two-fold change and a P-value < 0.05 following Benjamini-Hochberg multiple correction testing. Genes were then classified according to their molecular functions. A set of consistently differentially expressed genes were confirmed using qRT-PCR with Taqman probes on a larger sample size compared with the microarray experiment. All pathway and network analysis on the differentially expressed genes were conducted using MetaCore software Version 6.3 (GeneGo, Inc). Results A total of 3320 genes and 233 genes were differentially expressed when comparing AAA with NA and AAA with AOD respectively.
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Malina, Martin. "Endovascular repair of abdominal aortic aneurysms aspects on a novel technique /." Lund : Dept. of Vascular and Renal Diseases, Lund University, Malmö University Hospital, 1998. http://books.google.com/books?id=hWBsAAAAMAAJ.

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Lowe, Christopher. "Three-dimensional ultrasound in the management of abdominal aortic aneurysm." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/threedimensional-ultrasound-in-themanagement-of-abdominal-aorticaneurysm(b8950db7-847b-4d11-a6a5-2a06b3bb66d0).html.

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Objectives: Clinical implementation of 3D ultrasound (3D-US) in vascular surgery is in its infancy. The aim of this thesis was to develop novel clinical applications for 3D-US in the diagnosis and management of abdominal aortic aneurysm (AAA). Methods: Four principle clinical applications were investigated. 1) Intraoperative imaging – The ability of 3D-US to detect and classify endoleaks was compared with digital subtraction angiography in patients undergoing EVAR. 2) Detection and classification of endoleaks following endovascular aneurysm repair (EVAR) – The abilityof 3D-US to accurately detect and classify endoleaks following EVAR was compared to CTA and the final multi-disciplinary team decision. 3) AAA volume measurement – measurements using magnetic and optically-tracked 3D-US were compared to CTA. 4) Biomechanical analysis – the challenges of using 3D-US to generate surface models for biomechanical simulation was explored by development of an interactive segmentation technique and comparison of paired CT and 3D-US datasets. Optimal results were used in finite element analysis (FEA) and computational fluid dynamic(CFD) simulations. Results: 3D-US out-performed uniplanar angiography for the detection of endoleaks during EVAR. This approach allowed contrast-free EVAR to be performed in patients with poor renal function. 3D contrast-enhanced ultrasound was superior to CTA for endoleak detection and classification when compared with the final decision of the multi-disciplinary team. Optimal results for AAA volume measurements were gained using an optically tracked 3D-US system in EVAR surveillance. However, there remained a significant mean difference of 13.6ml between CT and 3D-US. Complete technical success of generating geometries for use in biomechanical analysis using 3D-US was only 5%. When the optimal results were used, a comparable CFD analysis under the conditions of steady, laminar and Newtonian flow was achieved. Using basic modelling assumptions in FEA, peak von Mises and principle wall stress was found to be at the same anatomical location on both the CT and 3D-US models but the 3D-US model overestimated the wall stress values by 41% and 51% respectively. Conclusions: 3D-US could be clinically implemented for intra-operative imaging and EVAR surveillance in specific cases. 3D-US volume measurement is feasible but future work should aim to improve accuracy and inter-observer reliability. Although the results of biomechanical analysis using the optimal results was encouraging and provided a proof-of-principal, there are a number of technical developments required to make this approach feasible in a larger number of patients.
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Djavani, Gidlund Khatereh. "Intra-abdominal Hypertension and Colonic Hypoperfusion after Abdominal Aortic Aneurysm Repair." Doctoral thesis, Uppsala universitet, Institutionen för kirurgiska vetenskaper, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-149241.

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Colonic ischaemia (CI), Intra-abdominal hypertension (IAH) and abdominal compartment syndrome (ACS) are devastating complications after abdominal aortic aneurysm (AAA) surgery. The aims of this thesis were to study the incidence and clinical consequences of IAH/ACS and the association between CI and intra-abdominal pressure (IAP) among patients undergoing OR for ruptured AAA (rAAA), to compare extraluminal pHi monitoring, with standard intra-luminal monitoring among patients operated on for AAA, and to study the frequency and clinical consequences of IAH/ACS after endovascular repair (EVAR) for rAAA. The incidence of ACS was 26% in a retrospective study of 27 patients undergoing OR for rAAA. Consensus definitions on IAH/ACS were appropriate for patients after OR for rAAA: 78% (7/9) of patients with IAH grade III or IV developed organ failure and all patients who developed CI had some degree of IAH. Active fluid resuscitation treating hypovolaemia to avoid CI may partly cause IAH. The association between CI and IAP was investigated in a prospective study on 29 patients operated on for rAAA, 86% (25/29) were treated for hypovolaemia and ten (34%) had both IAH and CI. Since monitoring colonic perfusion is very important and there is no ideal method, a new technique, extraluminal colonic tonometry to detect colonic perfusion was compared with standard intraluminal tonometry. Although, this new method was not able to determine the severity of ischaemia it may serve as a screening test. EVAR of rAAA is feasible and patients may benefit from this less invasive procedure. Of 29 patients treated with this technique, 10% developed ACS, and all patients except one with preoperative shock developed some degree of IAH. In conclusion, IAP/ACS is common after both OR and EVAR for rAAA, and is associated with adverse outcome. Monitoring IAP and colonic perfusion with timely intervention may improve outcome.
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Boyle, Jonathan Robert. "New perspectives in abdominal aortic aneurysm management." Thesis, University of Leicester, 2000. http://hdl.handle.net/2381/29606.

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A better understanding of the pathophysiology of abdominal aortic aneurysms has recently been established paving the way for potential targeted pharmacotherapy aimed at inhibiting the growth of small aneurysms. In particular the matrix metallopropteinase enzymes have been implicated in the destruction of the aortic wall. To this end the first part of this thesis investigates the potential therapeutic role of doxycycline, a non-specific metalloproteinase inhibitor, in an established model of aneurysmal disease. Subsequently the role of Amlodipine a calcium antagonist and metalloproteinase potentiator is investigated in the same model. Endovascular AAA repair is a new minimally invasive technique that allows treatment of aortic aneurysms without major abdominal surgery. The feasibility of this technique has been established, however a number of important questions remain unanswered. The second half of this thesis investigates the invasiveness of endovascular repair in comparison to conventional surgery. In particular the impact both procedures have on respiratory, cardiac, renal and metabolic responses is studied in comparative cohorts undergoing both conventional and endovascular AAA repair. Finally the implications of offering a tertiary referral service for AAA treatment is investigated. The results presented in this thesis demonstrate that doxycycline inhibits MMP activity and thus elastin destruction in a porcine model of aneurysmal disease. In the same model however, Amlodipine potentiates MMP activity and accelerates elastin degradation. There may be a therapeutic role for doxycycline in reducing the growth rate of small aneurysms. The clinical investigations of this thesis show that endovascular AAA repair attenuates the respiratory, cardiovascular, renal and metabolic responses associated with conventional aneurysm surgery. There were however still considerable insults from endoluminal surgery. Endovascular AAA repair may reduce morbidity and mortality rates after elective AAA surgery. The last experimental chapter illustrates that offering endovascular AAA repair as a tertiary centre has considerable clinical and financial implications. Finally a number of problems remain with endovascular AAA surgery which require evaluation by randomised controlled trial before its widespread use.
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Choke, Tieng Chek. "Molecular mechanisms of abdominal aortic aneurysm rupture." Thesis, St George's, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.511897.

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Watton, Paul N. "Mathematical modelling of the abdominal aortic aneurysm." Thesis, University of Leeds, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411948.

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Holmström, Ami. "Abdominal Aortic Aneurysm Screening : an Ethical Discussion." Thesis, Örebro universitet, Institutionen för medicinska vetenskaper, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-72994.

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Introduction: Abdominal aortic aneurysms (AAA) have a prevalence of approximately 2%, and are more common in men. AAAs are generally asymptomatic, but if ruptured and untreated, the mortality rate is close to 100%. Screening programs for AAAs are implemented in Sweden, the UK, and the US. This study describes the different views of AAA screening with a special emphasis on underlying ethical issues. Aim: To analyze the scientific background of AAA screening in order to be able to discuss its ethical basis. Methods: This was a qualitative literature study with an analysis of arguments using a hermeneutic method. Articles were obtained through a literature search and consisted of official articles, scientific articles, and debate articles. Results: A recent dissertation has questioned the value of AAA screening because of decreased AAA mortality and risk for overdiagnosis. However, most studies and official recommendations are in favor of AAA screening because disease specific mortality decreases and the screening program is considered cost-effective. Conclusion: This study shows that intellectual passion has created an unusually polarized discussion. It seems that benefit outweighs harm. Since AAA screening is the first screening program which could lead to the death of a previously asymptomatic individual, well founded informed consent is extremely important. Finally, both decisions to act and not to act have moral consequences.
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Books on the topic "Abdominal aneurysm"

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Starnes, Benjamin W., Manish Mehta, and Frank J. Veith, eds. Ruptured Abdominal Aortic Aneurysm. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-23844-9.

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G, Hakaim Albert, ed. Current endovascular treatment of abdominal aortic aneurysms. Oxford: Blackwell Pub., 2005.

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David, Tilson M., Kuivaniemi Helena, and Upchurch Gilbert R, eds. The abdominal aortic aneurysm: Genetics, pathophysiology, and molecular biology. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

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Webster, Ellis Lorenzo. Analysis of tissue inhibitor of metalloproteases (TIMP) as the unifying entity in the etiology of abdominal aortic aneurysms. [S.l: s.n.], 1991.

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J, Doyle Barry, ed. 3D imaging of abdominal aortic aneurysms: Techniques and applications. Hauppauge, N.Y: Nova Science, 2010.

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Anderson, Michael A. B. Organ injury following ruptured abdominal aortic aneurysm is mediated by oxidants. Ottawa: National Library of Canada, 2000.

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Klein, Lazar Victor. Discordant effects of interleukin-10 upon organ injury in a model of ruptured abdominal aortic aneurysm. Ottawa: National Library of Canada, 2002.

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Hakaim, Albert G., ed. Current Endovascular Treatment of Abdominal Aortic Aneurysms. Oxford, UK: Blackwell Publishing, 2006. http://dx.doi.org/10.1002/9780470753156.

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Dueck, Andrew D. Care of ruptured abdominal aortic aneurysms in Ontario. Ottawa: National Library of Canada, 2003.

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Savage, Edward Bruce. Hydrocortisone induces aortic rupture in inbred blotchy mice: Implications for abdominal aortic aneurysmal disease in humans. [New Haven: s.n.], 1985.

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Book chapters on the topic "Abdominal aneurysm"

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Friedewald, Vincent E. "Abdominal Aortic Aneurysm." In Clinical Guide to Cardiovascular Disease, 1–13. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7293-2_1.

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Dharmarajan, T. S., T. S. Dharmarajan, T. S. Dharmarajan, and T. S. Dharmarajan. "Abdominal Aortic Aneurysm." In Geriatric Gastroenterology, 631–36. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-1623-5_68.

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March, Robert J. "Abdominal Aortic Aneurysm." In Common Surgical Diseases, 107–10. New York, NY: Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2945-0_25.

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d’Audiffret, A., and J. P. Becquemin. "Abdominal Aortic Aneurysm." In Vascular Surgery, 11–16. London: Springer London, 2003. http://dx.doi.org/10.1007/978-1-4471-3870-9_2.

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Dharmarajan, T. S., and Nilesh N. Balar. "Abdominal Aortic Aneurysm." In Geriatric Gastroenterology, 1–16. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-90761-1_87-1.

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Danzer, Daniel, and Jean-Pierre Becquemin. "Abdominal Aortic Aneurysm." In Vascular Surgery, 15–24. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-356-5_2.

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Becquemin, Jean-Pierre, and Alexandre d’Audiffret. "Abdominal Aortic Aneurysm." In Vascular Surgery, 13–21. London: Springer London, 2006. http://dx.doi.org/10.1007/1-84628-211-x_2.

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Schein, Moshe. "Ruptured Abdominal Aneurysm." In Schein’s Common Sense Emergency Abdominal Surgery, 267–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-88133-6_30.

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Greenleaf, Erin K., and Faisal Aziz. "Abdominal Aortic Aneurysm." In Clinical Algorithms in General Surgery, 551–53. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98497-1_135.

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Champion, Howard R., Nova L. Panebianco, Jan J. De Waele, Lewis J. Kaplan, Manu L. N. G. Malbrain, Annie L. Slaughter, Walter L. Biffl, et al. "Abdominal Aortic Aneurysm." In Encyclopedia of Intensive Care Medicine, 5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1019.

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Conference papers on the topic "Abdominal aneurysm"

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Zeinali-Davarani, S., A. Sheidaei, and S. Baek. "Towards Patient-Specific Modeling of an Enlarging Abdominal Aortic Aneurysm." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205488.

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There has been a clear need for better understanding of the progression of abdominal aortic aneurysm (AAA) and obtaining reliable prediction of the AAA rupture. Finite element analysis (FEA) using non-axisymmetric models of AAAs provides better estimation of stress distribution in the aneurysmal wall with complex shapes [1]. However, FEA alone does not provide a mathematical description for the evolution of an AAA through growth and remodeling (G&R). A computational framework for modeling stress-mediated growth and structural remodeling of the arterial wall under physiological and pathological conditions has been suggested using a constrained mixture assumption [2]. Stress-mediated enlargement of intracranial aneurysms has been investigated using idealized axisymmetric geometries [3,4]. The kinetics of stress-mediated turnover of collagen fiber families and degradation of elastin were found to have particular importance in the G&R of aneurysmal wall.
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Gee, Michael W., and Wolfgang A. Wall. "Model Complexity and Prestressing in Abdominal Aortic Aneurysm Simulation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204593.

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Rupture of abdominal aortic aneurysm (AAA) is the 13th leading cause of death in western society and is fatal in 70–90%. In consequence, precise prediction of AAA rupture risk is essential. With the current, well established CT-morphological parameters such as maximum aortic diameter, aneurysm shape and AAA expansion, only at best the relative, but not the individual rupture risk can be determined. Hence, AAA rupture may occur unexpectedly in small aneurysms below the critical diameter limits whereas many large aneurysms may remain stable throughout patient’s lifetime, without prophylactic surgery.
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Martufi, Giampaolo, Jose F. Rodriguez, and Ender A. Finol. "Anisotropic Wall Mechanics of Abdominal Aortic Aneurysms." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192265.

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The prevalence of AAA is growing along with population age and according to different studies AAA rupture is the 13th most common cause of death in the U.S., causing an estimated 15,000 deaths per year. In biomechanical terms, AAA rupture is a phenomenon that occurs when the developing mechanical stresses within the aneurysm inner wall, as a result of the exerted intraluminal pressure, exceed the failure strength of the aortic tissue. To obtain a reliable estimation of wall stress, it is necessary to perform an accurate three-dimensional reconstruction of the AAA geometry and model an appropriate constitutive law for the aneurysmal tissue material characterization. In this regard, a recent study on the biaxial mechanical behavior of human AAA tissue specimens [1] demonstrates that aneurysmal arterial tissue behaves mechanically anisotropic. The objectives of the present work are to determine the effect of material anisotropy of the aneurysmal abdominal aorta on wall stress distribution and to establish a comparison of wall mechanics between ruptured and unruptured aneurysms.
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Embong, A. H., A. M. Al-Jumaily, G. Mahadevan, A. Lowe, and S. Sugita. "Development of an Abdominal Aortic Aneurysm Ruptures Mechanism Using a Geometric Analytical Technique." In ASME 2014 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/imece2014-39823.

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Current ultrasound approaches practice probe for diagnosing instantaneous abdominal aortic aneurysms (AAA) based on arterial tissue deformation. However, tracking the progression of potential aneurysms, and predicting the risk of rupture is based on the diameter of the aneurysm and is still an insufficient method: Larger diameter aneurysms do not always lead to ruptures, and smaller diameter aneurysms unexpectedly rupture. In order to improve diagnostic accuracy of ultrasound imaging techniques, this paper presents geometric analyses of patient-specific instant deformations as a means to develop an aneurysm rupture mechanism. Segmented AAA images were used to analyze dependent elements that contribute to a three-dimensional (3-D) aneurysm reconstructive model using proposed Patient-Specific Aneurysm Rupture Predictor (P-SARP) method. The outcomes indicate that the proposed technique has the ability to associate the distortion of wall deformation with geometric analyses. This method can positively be integrated with established ultrasound techniques for improvements in the accuracy of future diagnoses of potential AAA ruptures.
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Washington, Christopher B., Judy Shum, Satish C. Muluk, and Ender A. Finol. "Abdominal Aortic Aneurysm Growth: The Association of Aortic Wall Mechanics and Geometry." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53977.

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In an effort to prevent rupture, patients with known AAA undergo periodic abdominal ultrasound or CT scan surveillance. When the aneurysm grows to a diameter of 5.0–5.5 cm or is shown to expand at a rate greater than 1 cm/yr, elective operative repair is undertaken. While this strategy certainly prevents a number of potentially catastrophic ruptures, AAA rupture can occur at sizes less than 5 cm. From a biomechanical standpoint, aneurysm rupture occurs when wall stress exceeds wall strength. By using non-invasive techniques, such as finite element analysis (FEA), wall stress can be estimated for patient specific AAA models, which can perhaps more carefully predict the rupture potential of a given aneurysm, regardless of size. FEA is a computational method that can be used to evaluate complicated structures such as aneurysms. To this end, it was reported earlier that AAA peak wall stress provides a better assessment of rupture risk than the commonly used maximum diameter criterion [1]. What has yet to be examined, however, is the relationship between wall stress and AAA geometry during aneurysm growth. Such finding has the potential for providing individualized predictions of AAA rupture potential during patient surveillance. The purpose of this study is to estimate peak wall stress for an AAA under surveillance and evaluate its potential correlation with geometric features characteristic of the aneurysm’s morphology.
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Ayyalasomayajula, Avinash, Bruce R. Simon, and Jonathan P. Vande Geest. "Porohyperelastic Simulation of Abdominal Aortic Aneurysms." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193147.

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Abdominal aortic aneurysm (AAA) is a progressive dilation of the infrarenal aorta and results in a significant alteration in local hemodynamic environment [1]. While an aneurysmal diameter of 5.5cm is typically classified as being of high risk, recent studies have demonstrated that maximum wall stress could be a better indicator of an AAA rupture than maximum diameter [2]. The wall stress is greatly influenced by the blood pressure, aneurysm diameter, shape, wall thickness and the presence of thrombus. The work done by Finol et al. suggested that hemodynamic pressure variations have an insignificant effect on AAA wall stress and that primarily the shape of the aneurysm determines the stress distribution. They noted that for peak wall stress studies the static pressure conditions would suffice as the in vivo conditions. Wang et al have developed an isotropic hyperelastic constitutive model for the intraluminal thrombus (ILT). Such models have been used to study the stress distributions in patient specific AAAs [3, 4].
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Trachet, Bram, Marjolijn Renard, Joris Bols, Steven Staelens, Bart Loeys, and Patrick Segers. "Hemodynamics in Ascending and Abdominal Aorta Aneurysm Formation in the ApoE−/− Angiotensin II Mouse Model." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80243.

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Aortic aneurysm is a pathological dilatation of the aorta that can be life-threatening when it ruptures. Aneurysms occur throughout the entire aorta but there is a predisposition for the ascending and the abdominal aorta, an observation that cannot be fully explained by the current knowledge of the disease pathophysiology. ApoE −/− mice infused with angiotensin II have recently been reported to develop not only abdominal [1], but also ascending aortic aneurysms [2]. These animals thus provide the perfect model to compare aneurysm progression in both aortic locations and to investigate whether disturbed hemodynamics play a role in the initial phase of aneurysm growth. In this study, both imaging and computational biomechanics techniques were used to elucidate the flow field at the location of the aneurysm prior to onset of the disease.
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Thubrikar, Mano J., Michel Labrosse, Jihad Al-Soudi, Brett Fowler, and Francis Robicsek. "Material Properties of Abdominal Aortic Aneurysm Wall From Uniaxial Tests." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2541.

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Abstract Abdominal aortic aneurysms (AAA) rupture when the aortic wall cannot withstand the stresses and strains induced by the pulsatile blood pressure. In recent years, different mechanical models of aneurysms have been presented (Vorp et al., 1998, Di Martino et al., 1998, Thubrikar et al., 1999). Although powerful modeling tools such as finite elements are available, there is still a need for experimental data concerning the mechanical properties of the aneurysm wall.
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Ene, Florentina, Carine Gachon, Patrick Delassus, and Liam Morris. "Investigating the Effect of Intraluminal Thrombus in Abdominal Aortic Aneurysm by Computational and Experimental Methods." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206636.

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Abdominal aortic aneurysm (AAA) represents an abnormal dilatation and weakening of the abdominal aorta with high risk of rupture. Most aneurysms of the infrarenal aorta possess an asymmetrical fusiform morphology.
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Habib, S., and J. Dehmeshki. "Automatic Segmentation of Abdominal Aortic Aneurysm." In 2018 IEEE 13th International Scientific and Technical Conference on Computer Sciences and Information Technologies (CSIT). IEEE, 2018. http://dx.doi.org/10.1109/stc-csit.2018.8526709.

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Reports on the topic "Abdominal aneurysm"

1

Karmy-Jones, R. Abdominal Aortic Aneurysm and Pheochromocytoma. Science Repository, June 2019. http://dx.doi.org/10.31487/j.ijscr.2019.01.02.

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Jianqing, Deng, Jie Liu, Dan Rong, Yangyang Ge, Hongpeng Zhang, and Xiaoping Liu. Locoregional Anesthesia Versus General Anesthesia in Endovascular Repair of Ruptured Abdominal Aortic Aneurysm: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review Protocols, March 2020. http://dx.doi.org/10.37766/inplasy2020.3.0010.

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Fewer wound hernias occur if mesh is used to reinforce abdominal aortic aneurysm surgery. National Institute for Health Research, September 2018. http://dx.doi.org/10.3310/signal-000644.

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Abdominal aortic aneurysm screening for women is unlikely to be a fair use of NHS resources. National Institute for Health Research, November 2018. http://dx.doi.org/10.3310/signal-000676.

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Endovascular aortic repair (EVAR) surgery more beneficial for ruptured abdominal aortic aneurysms than open repair. National Institute for Health Research, August 2018. http://dx.doi.org/10.3310/signal-000638.

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No clear difference between open and keyhole surgery for the repair of ruptured abdominal aortic aneurysms. National Institute for Health Research, May 2016. http://dx.doi.org/10.3310/signal-000234.

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