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Academic literature on the topic 'Abdomina Aortic Aneurysm'

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Published: 14 March 2023

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Journal articles on the topic "Abdomina Aortic Aneurysm"

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Genyk, Stepan. "Aneurysmal Disease of the Main Arteries." Archive of Clinical Medicine 22, no. 2 (December 8, 2016): 201627. http://dx.doi.org/10.21802/acm.2016.2.7.

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The high incidence of aneurysms of different arterial systems in case of abdominal aortic aneurysm causes the need for the examination of all patients with aortic aneurysm in order to detect aneurysmal process in all main arteries. The use of the mentioned predictors of aneurism wall failure in daily clinical practice provides an opportunity to improve the results of surgical treatment. Active surgical approach in relation to aortic aneurysm and main arteries is indicated in the presence of aneurismal disease. The choice of treatment depends on the severity of the patient’s condition, clinical manifestations, localization of the aneurysm and its morphology, with the preference for endovascular approach.
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Sebayang, Abed Nego Okthara, and Niko Azhari Hidayat. "Endovascular Aortic Repair (EVAR) Method in The Management of Abdominal Aortic Aneurysm." SCRIPTA SCORE Scientific Medical Journal 2, no. 1 (August 28, 2020): 53–7. http://dx.doi.org/10.32734/scripta.v2i1.3530.

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Aortic disease is a collection of diseases of the aorta, which includes aortic aneurysms; acute aortic infections consisting of aortic dissection, intramular hematoma, penetration of atherosclerotic ulcers (PAU) and traumatic injury to the aorta; pseudoaneurysm; aortic rupture; Marfan syndrome; and congenital abnormalities such as coarctation of the aorta. One of the aortic diseases that cause the death rate to increase according to the 2010 Global Burden Disease is aortic aneurysm. Abdominal aortic aneurysm (AAA) is a focal dilatation of the aortic segment. The diagnosis of AAA is done by history taking, physical examination and supporting examination. Management at AAA aims to prevent aortic wall rupture. An alternative procedure without open surgery is endovascular aortic repair (EVAR) using prostheses. It is expected that through the EVAR method, mortality and morbidity due to AAA can be reduced. Keywords: abdominal aortic aneursym, EVAR, prostheses Penyakit aorta merupakan kumpulan penyakit pada aorta yang meliputi aneurisma aorta; sindrom aorta akut berupa diseksi aorta, hematoma intramular, penetrating atherosclerosis ulcer (PAU) dan cedera akibat trauma pada aorta; pseudoaneurysm; ruptur aorta; sindrom Marfan; serta penyakit kongenital seperti koarktasio aorta. Salah satu penyakit aorta yang menyebabkan angka kematian meningkat menurut Global Burden Disease 2010 adalah aneurisma aorta. Aneurisma aorta abdominalis (AAA) merupakan dilatasi fokal pada segmen aorta. Penegakan diagnosis AAA dilakukan dengan anamnesis, pemeriksaan fisik dan pemeriksaan penunjang. Penatalaksanaan pada AAA bertujuan untuk mencegah pecahnya dinding aorta. Prosedur alternatif tanpa pembedahan terbuka yang dijadikan pilihan adalah endovascular aortic repair (EVAR) menggunakan protesa. Diharapkan melalui metode EVAR angka mortalitas dan morbiditas akibat AAA dapat diturunkan. Kata kunci: aneurisma aorta abdominalis, EVAR, protesa
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Batinic, Nikola, Tijana Kokovic, Dragan Nikolic, Vladimir Manojlovic, Viktor Till, and Slavko Budinski. "The impact of abdominal aortic aneurysm diameter on the outcome of endovascular aortic repair." Medical review 74, no. 11-12 (2021): 347–53. http://dx.doi.org/10.2298/mpns2112347b.

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Introduction. Abdominal aortic aneurysm diameter is one of the most important parameters in the diagnostic and therapeutic algorithm for aneurysm follow-up. Currently, two therapeutic modalities are used: open surgery and endovascular aortic repair. The aim of this study is to analyze the impact of the maximum transverse diameter of the abdominal aortic aneurysm on the incidence of general and specific complications. Material and Methods. The retrospective study included 75 patients with infrarenal abdominal aortic aneurysm who underwent endovascular aortic repair in the period from July 2008 to January 2020. The patients were divided into two groups: group A with an abdominal aortic aneurysm size ? 5.9 cm, and group B with an abdominal aortic aneurysm size ? 6.0 cm. Results. A total of 41.3% of patients presented with a maximum transverse aneurysm diameter of ? 5.9 cm, and 58.7% of patients had ? 6.0 cm. Of comorbid diseases, chronic obstructive pulmonary disease was more prevalent in patients with a large abdominal aortic aneurysm (group A 25.8%; group B 59.1%). None of the other comorbidities showed a statistically significant difference between the two groups of patients. Early complications were present in a total of 14.7% of patients, of which 12.9% of patients with a small and 15.9% with a large abdominal aortic aneurysm. Late complications occurred in a total of 18.7% of patients, in 9.7% of patients with a small and 25% of patients with a large abdominal aortic aneurysm. Conclusion. Patients with abdominal aortic aneurysms with a maximum transverse diameter of 6 cm and larger, present with a higher rate of late postoperative complications, increase in aneurysmal sac on control multislice computed tomography angiography, and have a worse prognosis compared to patients with smaller abdominal aortic aneurysms.
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Pereira, Thiago Scremin Boscolo, Vanessa Belentani Marques, Elizandra Moura dos Santos, Ana Letícia Daher Aprígio da Silva, Eduardo Martini Romano, and Carla Patricia Carlos. "Association Between Abdominal Aortic and Common Iliac Artery Aneurysms: Case Report." Journal of Biology and Life Science 10, no. 2 (June 30, 2019): 71. http://dx.doi.org/10.5296/jbls.v10i2.14714.

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The infrarenal abdominal aortic aneurysm is the most common among arterial aneurysms; it happens when there is an abnormal and irreversible enlargement of the blood vessel. This disease usually compromises other arterial segments and is linked to high mortality rates, mainly due to its rupture. Given its importance, we present a case study of an abdominal aortic aneurysm associated with a common iliac artery aneurysm. During a dissection practice in the Morphofunctional Laboratory at FACERES Medical School, we observed the presence of a mild stenosis in the abdominal aorta below the renal arteries, as well as the formation of an infrarenal abdominal aortic aneurysm. In addition, we noticed that the infrarenal abdominal aortic aneurysm was associated with a bilateral common iliac artery aneurysm. Morphological analyses carried out in the blood vessels showed a large quantity of atheromatous plaques, which are the probable cause of the pathology. The information herein may broaden the knowledge on the infrarenal abdominal aortic aneurysmal disease, thus reducing its complications and mortality rates.
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Joshi, Nikhil V., Maysoon Elkhawad, Rachael O. Forsythe, Olivia M. B. McBride, Nikil K. Rajani, Jason M. Tarkin, Mohammed M. Chowdhury, et al. "Greater aortic inflammation and calcification in abdominal aortic aneurysmal disease than atherosclerosis: a prospective matched cohort study." Open Heart 7, no. 1 (March 2020): e001141. http://dx.doi.org/10.1136/openhrt-2019-001141.

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ObjectiveUsing combined positron emission tomography and CT (PET-CT), we measured aortic inflammation and calcification in patients with abdominal aortic aneurysms (AAA), and compared them with matched controls with atherosclerosis.MethodsWe prospectively recruited 63 patients (mean age 76.1±6.8 years) with asymptomatic aneurysm disease (mean size 4.33±0.73 cm) and 19 age-and-sex-matched patients with confirmed atherosclerosis but no aneurysm. Inflammation and calcification were assessed using combined 18F-FDG PET-CT and quantified using tissue-to-background ratios (TBRs) and Agatston scores.ResultsIn patients with AAA, 18F-FDG uptake was higher within the aneurysm than in other regions of the aorta (mean TBRmax2.23±0.46 vs 2.12±0.46, p=0.02). Compared with atherosclerotic control subjects, both aneurysmal and non-aneurysmal aortae showed higher 18F-FDG accumulation (total aorta mean TBRmax2.16±0.51 vs 1.70±0.22, p=0.001; AAA mean TBRmax2.23±0.45 vs 1.68±0.21, p<0.0001). Aneurysms containing intraluminal thrombus demonstrated lower 18F-FDG uptake within their walls than those without (mean TBRmax2.14±0.43 vs 2.43±0.45, p=0.018), with thrombus itself showing low tracer uptake (mean TBRmax thrombus 1.30±0.48 vs aneurysm wall 2.23±0.46, p<0.0001). Calcification in the aneurysmal segment was higher than both non-aneurysmal segments in patients with aneurysm (Agatston 4918 (2901–8008) vs 1017 (139–2226), p<0.0001) and equivalent regions in control patients (442 (304-920) vs 166 (80-374) Agatston units per cm, p=0.0042).ConclusionsThe entire aorta is more inflamed in patients with aneurysm than in those with atherosclerosis, perhaps suggesting a generalised inflammatory aortopathy in patients with aneurysm. Calcification was prominent within the aneurysmal sac, with the remainder of the aorta being relatively spared. The presence of intraluminal thrombus, itself metabolically relatively inert, was associated with lower levels of inflammation in the adjacent aneurysmal wall.
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Samura, Makoto, Nobuya Zempo, Yoshitaka Ikeda, Masaaki Hidaka, Yoshikazu Kaneda, Kazuhiro Suzuki, Hidetoshi Tsuboi, and Kimikazu Hamano. "Single-stage thoracic and abdominal endovascular aneurysm repair for multilevel aortic disease." Vascular 22, no. 1 (May 13, 2013): 55–60. http://dx.doi.org/10.1177/1708538112473965.

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This investigation evaluated the results of single-stage thoracic endovascular aneurysm repair (TEVAR) and endovascular aneurysm repair (EVAR) for multilevel aortic disease in a series of nine patients. The lesions repaired included thoracic and abdominal aortic aneurysms ( n = 7) and subacute type B dissections with abdominal aortic aneurysms ( n = 2). All procedures were successfully performed, and none of the patients experienced postoperative stroke or spinal cord ischemia. The median follow-up period for these patients was 18.9 months (range 1.7–31.4 months) and none of the patients exhibited any signs of type I endoleaks or aneurysmal diameter enlargements more than 5 mm. In conclusion, single-stage TEVAR and EVAR procedures for multilevel aortic disease were found to be safe and feasible modalities for high-risk patients.
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Faries, Peter L., Luis A. Sanchez, Michael L. Marin, Richard E. Parsons, Ross T. Lyon, Steve Oliveri, and Frank J. Veith. "An Experimental Model for the Acute and Chronic Evaluation of Intra-Aneurysmal Pressure." Journal of Endovascular Therapy 4, no. 3 (August 1997): 290–97. http://dx.doi.org/10.1177/152660289700400310.

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Purpose: To develop an animal model for the acute and chronic monitoring of pressure within abdominal aortic aneurysms (AAAs) to be treated with endovascular grafts. Methods: A strain-gauge pressure transducer was placed within an AAA created from a prosthetic vascular graft. Prosthetic aneurysms were implanted into 17 canine infrarenal aortas. The intra-aneurysmal pressure was monitored and correlated with noninvasive forelimb sphygmomanometry for 2 weeks. After this time, an intravascular manometer catheter was passed into the aneurysm. Simultaneous pressure measurements were obtained using the implanted strain-gauge pressure transducer, the manometer catheter, and the forelimb sphygmomanometer. Angiography was performed to assess intraluminal morphology, aneurysm anastomoses, and adjoining aortic vessels. In addition, two control animals underwent intra-aneurysmal pressure monitoring after standard surgical aneurysm repair. Results: There was excellent correlation (r = 0.97) between the pressure measurements obtained with the implanted strain-gauge pressure transducer and the intravascular manometer. Close correlation was also observed between the implanted strain-gauge transducer and the forelimb sphygmomanometer (r = 0.88) during postprocedural monitoring. Intra-aneurysmal pressure was lowered dramatically by surgical exclusion (aneurysm: 15/5 ± 7/4 mmHg; systemic: 124/66 ± 34/17 mmHg; p < 0.001). The prosthetic aneurysms were successfully imaged with angiography. Conclusions: This animal model provides an accurate and reproducible means for measuring intra-aneurysmal pressure on an acute and chronic basis. It may be possible to use this model in the assessment of endovascular devices to determine their efficacy in reducing intra-aneurysmal pressure. Evaluation of complications associated with their use, such as patent aneurysm side branches, perigraft channels, and perianastomotic reflux, may also be possible.
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Weiss, Norbert, Roman N. Rodionov, and Adrian Mahlmann. "Medical management of abdominal aortic aneurysms." Vasa 43, no. 6 (November 1, 2014): 415–21. http://dx.doi.org/10.1024/0301-1526/a000388.

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Abdominal aortic aneurysms (AAA) are the most common arterial aneurysms. Endovascular or open surgical aneurysm repair is indicated in patients with large AAA ≥ 5.5 cm in diameter as this prevents aneurysm rupture. The presence even of small AAAs not in need of immediate repair is associated with a very high cardiovascular risk including myocardial infarction, stroke or cardiovascular death. This risk by far exceeds the risk of aneurysm rupture. These patients therefore should be considered as high-risk patients and receive optimal medical treatment and life-style modificiation of their cardiovascular risk factors to improve their prognosis. In addition, these patients should be followed-up for aneurysm growth and receive medical treatment to decrease aneurym progression and rupture rate. Treatment with statins has been shown to reduce cardiovascular mortality in these patients, and also slows the rate of AAA growth. Use of beta-blockers, ACE inhibitors and AT1-receptor antagonists does not affect AAA growth but may be indicated for comorbidities. Antibiotic therapy with roxithromycin has a small effect on AAA growth, but this effect must be critically weighed against the potential risk of wide-spread use of antibiotics.
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Kontopodis, Nikolaos, Eleni Metaxa, Yannis Papaharilaou, Emmanouil Tavlas, Dimitrios Tsetis, and Christos Ioannou. "Advancements in identifying biomechanical determinants for abdominal aortic aneurysm rupture." Vascular 23, no. 1 (April 22, 2014): 65–77. http://dx.doi.org/10.1177/1708538114532084.

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Abdominal aortic aneurysms are a common health problem and currently the need for surgical intervention is determined based on maximum diameter and growth rate criteria. Since these universal variables often fail to predict accurately every abdominal aortic aneurysms evolution, there is a considerable effort in the literature for other markers to be identified towards individualized rupture risk estimations and growth rate predictions. To this effort, biomechanical tools have been extensively used since abdominal aortic aneurysm rupture is in fact a material failure of the diseased arterial wall to compensate the stress acting on it. The peak wall stress, the role of the unique geometry of every individual abdominal aortic aneurysm as well as the mechanical properties and the local strength of the degenerated aneurysmal wall, all confer to rupture risk. In this review article, the assessment of these variables through mechanical testing, advanced imaging and computational modeling is reviewed and the clinical perspective is discussed.
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Dolmaci, Onur B., Sulayman El Mathari, Antoine H. G. Driessen, Robert J. M. Klautz, Robert E. Poelmann, Jan H. N. Lindeman, and Nimrat Grewal. "Are Thoracic Aortic Aneurysm Patients at Increased Risk for Cardiovascular Diseases?" Journal of Clinical Medicine 12, no. 1 (December 29, 2022): 272. http://dx.doi.org/10.3390/jcm12010272.

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Objectives: Abdominal aortic aneurysms are associated with a sharply increased cardiovascular risk. Cardiovascular risk management is therefore recommended in prevailing guidelines for abdominal aneurysm patients. It has been hypothesized that associated risk relates to loss of aortic compliance. If this hypothesis is correct, observations for abdominal aneurysms would also apply to thoracic aortic aneurysms. The objective of this study is to test whether thoracic aneurysms are also associated with an increased cardiovascular risk burden. Methods: Patients who underwent aortic valve or root surgery were included in the study (n = 239). Cardiovascular risk factors were studied and atherosclerosis was scored based on the preoperative coronary angiographies. Multivariate analyses were performed, controlling for cardiovascular risk factors and aortic valve morphology. Comparisons were made with the age- and gender-matched general population and non-aneurysm patients as control groups. A thoracic aortic aneurysm was defined as an aortic aneurysm of ≥45 mm. Results: Thoracic aortic aneurysm was not associated with an increased coronary atherosclerotic burden (p = 0.548). Comparison with the general population revealed a significantly higher prevalence of hypertension (61.4% vs. 32.2%, p < 0.001) and a lower prevalence of diabetes (1.4% vs. 13.1%, p = 0.001) in the thoracic aneurysm group. Conclusions: The extreme cardiovascular risk associated with abdominal aortic aneurysms is location-specific and not explained by loss of aortic compliance. Thoracic aortic aneurysm, in contrast to abdominal, is not part of the atherosclerotic disease spectrum and, therefore, cardiovascular risk management does not need to be implemented in treatment guidelines of isolated thoracic aneurysms. Hypertension should be treated.
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Dissertations / Theses on the topic "Abdomina Aortic Aneurysm"

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Rossaak, Jeremy Ian, and n/a. "The genetics of abdominal aortic aneurysms." University of Otago. Dunedin School of Medicine, 2004. http://adt.otago.ac.nz./public/adt-NZDU20070502.143818.

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Abdominal Aortic Aneurysms (AAA) are amongst the top ten most common cause of death in those over 55 years of age. The disease is usually asymptomatic, often being diagnosed incidentally. Once diagnosed, elective repair of an AAA results in excellent long-term survival with a 3-5% operative mortality. However, up to one half of patients present with ruptured aneurysms, a complication that carries an 80% mortality in the community, and of those reaching hospital, a 50% mortality. Clearly early diagnosis and treatment results in improved survival. Screening for AAA, with ultrasound, would detect aneurysms early, prior to rupture. However, debate continues over the cost effectiveness of population based screening programmes. The identification of a sub-population at a higher risk of developing AAA would increase the yield of a screening prograrmne. A number of populations have been examined, none of which have received international acceptance. About 20% of patients with an AAA have a family history of an aneurysm. The disease is also considered to be a disease of Caucasians, both facts suggesting a strong genetic component to the disease. Perhaps a genetically identified sub-population at a high risk of developing an AAA would prove to be an ideal population for screening. This thesis examines the incidence of aneurysms and the family histories of patients with AAA in the Otago region of New Zealand. Almost twenty percent of the population has a family history of AAA. DNA was collected from each of these patients for genetic analysis. The population was divided into familial AAA and non-familial AAA for the purpose of genetic analysis and compared to a control population. AAA is believed to be a disease of Caucasians; a non-Caucasian population with a low incidence of AAA may prove to be a good control population for genetic studies. A literature review demonstrated a higher incidence of AAA in Caucasians than other ethnic groups and within Caucasians a higher incidence in patients of Northern European origin. The incidence was low in Asian communities, even in studies involving of migrant Asian populations. The New Zealand Maori are believed to have originated from South East Asia, therefore could be expected to have a low incidence of AAA and would make an ideal control population for genetic studies. A pilot study was undertaken to examine the incidence of AAA in the New Zealand Maori. The age standardised incidence of AAA proved to be at least equal in Maori to non-Maori, with a more aggressive form of the disease in Maori, manifesting with a younger age at presentation and a higher incidence of ruptured aneurysms at diagnosis. It is well known that at the time of surgery, an AAA is at the end stage in its life. At this time, inflammation and matrix metalloproteinases (MMP) enzymes are prevalent within the aneurysm wall and have destroyed the wall of the aorta. One of the most important genetic pathways regulating these enzymes is the plasminogen activator inhibiter 1-Tissue plasminogen activator-plasmin pathway. Genetic analysis of this pathway demonstrated an association of the 4G5G polymorphism in the promoter of the PAl-1 gene with familial AAA. In this insertion:deletion polymorphism, the 5G variant binds an activator and repressor, resulting in reduced PAI-1 expression and ultimately increased MMP activation. This allele was associated with familial aneurysms, 47% versus 62% non-familial AAA and 61% controls (p=0.024). A polymorphism within the tissue plasminogen activator gene was also examined and no association was found with AAA. Another way the MMPs expression could be increased is from mutations or polymorphisms in their own genetic structure. Stromelysin 3 is itself a MMP capable of destroying the aortic wall and it has a role in activating other MMPs. A 5A6A insertion:deletion polymorphism exists in the promoter of this gene. The 5A allele variant results in increased stromelysin expression and is associated with AAA 46% versus 33% in controls p=0. 0006. The actions of the MMPs are themselves inhibited by the tissue inhibitors of matrix metalloproteinases. The TIMP genes have been sequenced; two polymorphisms have been identified in the non-coding promoter area of the TIMP 1 gene. Further studies are necessary to examine the effect of these polymorphisms. Inflammation has been implicated in aneurysm progression. One of the roles of the inflammatory cells found in an aneurysm is to deliver the MMP�s to the AAA. The HLA system is integral in controlling this inflammation and was therefore examined. From this series of studies it is concluded that there is a genetic component to AAA. This thesis presents the first genetic polymorphism associated with familial AAA and explores the role of a genetic pathway in the formation of AAA.
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Mello, Flávia Moerbeck Casadei de [UNESP]. "Aneurisma da aorta abdominal infra-renal: avaliação ultra-sonográfica em homens acima de 50 anos." Universidade Estadual Paulista (UNESP), 2003. http://hdl.handle.net/11449/87358.

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Com o objetivo de avaliar a ocorrência de aneurisma da aorta abdominal infra-renal (AAAIR), estudou-se uma amostra da população masculina do Município de Marília, com idade igual ou acima de 50 anos, no período de 2000 a 2002. Foram avaliados 240 homens por meio da ultrasonografia abdominal (USAb), com média de idade de 65,1 anos (±9,8 anos). A aorta abdominal foi medida no sentido ânteroposterior (AP) e látero-lateral (LL) aproximadamente a 2cm abaixo da artéria mesentérica superior (AMS) e 2cm acima de sua bifurcação. O critério utilizado para considerar aneurisma foi o maior diâmetro encontrado igual ou maior que 3,1cm. Também por questionário, foram avaliados os fatores de risco (tabagismo, sedentarismo, alimentação) e as doenças associadas (HAS, DPOC, IM, DM, AOP ou hiperlipidemia). Nos 240 homens, foram encontrados 11 aneurismas, sendo, portanto, a freqüência de 4,6%. Desses 11 aneurismas, 8 mediam entre 3,1 e 4cm (72,7%) e 3, entre 4,1 e 5cm (27,3%). O maior diâmetro da aorta aneurismática foi de 5 cm (sentido AP a 2cm abaixo da AMS). Foi encontrada uma associação significativa entre aneurisma e AOP e DM, não ocorrendo o mesmo com os demais fatores de risco ou outras doenças associadas. A freqüência de aneurisma encontrada em nossa amostra não foi diferente da referida nos estudos populacionais publicados na literatura, o que mostra a importância da doença em nosso meio, e os indivíduos com AOP e DM têm risco maior de desenvolver a doença.
In order to evaluate the occurrence of Infra-Renal Abdominal Aortic Aneurysm (AAAIR), a sample of the male population in the city of Marília aged 50 years or older was studied from 2000 to 2002. A group of 240 men with mean age of 65,1 years (±9,8 years) was evaluated through abdominal ultra-sonography examination. The abdominal aorta was measured in the anteroposterior (AP) and in the latero-lateral directions (LL) approximately 2cm below the superior mesenteric artery and 2cm above its bifurcation. The largest diameter equal or larger than 3.1cm found was the criterion used for aneurysm. Risk factors such as smoking, eating, and exercise habits and associated diseases (systemic arterial hypertension, chronic obstructive pulmonary disease, myocardial infarction, diabetes mellitus, occlusive peripheral arterial disease, or hyperlipidemia) were also evaluated through questionnaires. Eleven aneurysms were found in the 240 men, which meant a frequency of 4,6%. Out of these 11 aneurysms, 8 measured from 3.1 to 4cm (72,7%) and 3 measured from 4.1 to 5cm (27,3%). The largest diameter of the aneurysmatic aorta was 5cm (AP direction approximately 2cm of the superior mesenteric artery). A significant association between aneurysm and peripheral vascular disease and diabetes mellitus was found. The same did not occur with the other risk factors or other associated diseases. The frequency of aneurysm found in our sample was not different from the frequency mentioned in population studies published in the literature, which shows the importance of the disease in our environment and that patients with peripheral vascular disease and diabetes mellitus have a higher risk to develop the disease.
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Mello, Flávia Moerbeck Casadei de. "Aneurisma da aorta abdominal infra-renal : avaliação ultra-sonográfica em homens acima de 50 anos /." Botucatu : [s.n.], 2003. http://hdl.handle.net/11449/87358.

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Orientador: Hamilton Almeida Rollo
Resumo: Com o objetivo de avaliar a ocorrência de aneurisma da aorta abdominal infra-renal (AAAIR), estudou-se uma amostra da população masculina do Município de Marília, com idade igual ou acima de 50 anos, no período de 2000 a 2002. Foram avaliados 240 homens por meio da ultrasonografia abdominal (USAb), com média de idade de 65,1 anos (±9,8 anos). A aorta abdominal foi medida no sentido ânteroposterior (AP) e látero-lateral (LL) aproximadamente a 2cm abaixo da artéria mesentérica superior (AMS) e 2cm acima de sua bifurcação. O critério utilizado para considerar aneurisma foi o maior diâmetro encontrado igual ou maior que 3,1cm. Também por questionário, foram avaliados os fatores de risco (tabagismo, sedentarismo, alimentação) e as doenças associadas (HAS, DPOC, IM, DM, AOP ou hiperlipidemia). Nos 240 homens, foram encontrados 11 aneurismas, sendo, portanto, a freqüência de 4,6%. Desses 11 aneurismas, 8 mediam entre 3,1 e 4cm (72,7%) e 3, entre 4,1 e 5cm (27,3%). O maior diâmetro da aorta aneurismática foi de 5 cm (sentido AP a 2cm abaixo da AMS). Foi encontrada uma associação significativa entre aneurisma e AOP e DM, não ocorrendo o mesmo com os demais fatores de risco ou outras doenças associadas. A freqüência de aneurisma encontrada em nossa amostra não foi diferente da referida nos estudos populacionais publicados na literatura, o que mostra a importância da doença em nosso meio, e os indivíduos com AOP e DM têm risco maior de desenvolver a doença.
Abstract: In order to evaluate the occurrence of Infra-Renal Abdominal Aortic Aneurysm (AAAIR), a sample of the male population in the city of Marília aged 50 years or older was studied from 2000 to 2002. A group of 240 men with mean age of 65,1 years (±9,8 years) was evaluated through abdominal ultra-sonography examination. The abdominal aorta was measured in the anteroposterior (AP) and in the latero-lateral directions (LL) approximately 2cm below the superior mesenteric artery and 2cm above its bifurcation. The largest diameter equal or larger than 3.1cm found was the criterion used for aneurysm. Risk factors such as smoking, eating, and exercise habits and associated diseases (systemic arterial hypertension, chronic obstructive pulmonary disease, myocardial infarction, diabetes mellitus, occlusive peripheral arterial disease, or hyperlipidemia) were also evaluated through questionnaires. Eleven aneurysms were found in the 240 men, which meant a frequency of 4,6%. Out of these 11 aneurysms, 8 measured from 3.1 to 4cm (72,7%) and 3 measured from 4.1 to 5cm (27,3%). The largest diameter of the aneurysmatic aorta was 5cm (AP direction approximately 2cm of the superior mesenteric artery). A significant association between aneurysm and peripheral vascular disease and diabetes mellitus was found. The same did not occur with the other risk factors or other associated diseases. The frequency of aneurysm found in our sample was not different from the frequency mentioned in population studies published in the literature, which shows the importance of the disease in our environment and that patients with peripheral vascular disease and diabetes mellitus have a higher risk to develop the disease.
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Malina, Martin. "Endovascular repair of abdominal aortic aneurysms aspects on a novel technique /." Lund : Dept. of Vascular and Renal Diseases, Lund University, Malmö University Hospital, 1998. http://books.google.com/books?id=hWBsAAAAMAAJ.

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Tenório, Emanuel Júnio Ramos. "Expressão dos níveis plasmáticos dos miRNA-191 e miRNA-455-3P em pacientes com aneurisma de aorta abdominal e suas relações com a evolução clínica após tratamento endovascular." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17137/tde-10042018-142246/.

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Introdução: O aneurisma de aorta abdominal (AAA) é uma importante causa de morbimortalidade na população idosa. O tratamento endovascular está associado a menor morbimortalidade que o tratamento convencional, no entanto, necessita de um seguimento rigoroso com exames de imagem contrastados para confirmação da exclusão do saco aneurismático. Considerando que a formação de um aneurisma é um processo multifatorial complexo, envolvendo a remodelação destrutiva do tecido conjuntivo em todo o segmento afetado da parede da aorta e que este processo envolve uma inflamação crônica local, uma diminuição no número de células do músculo liso da túnica média, e fragmentação da matriz extracelular da aorta e ainda que um perfil de expressão aberrante de miRNAs tem sido associada a doenças humanas, incluindo disfunção cardiovascular propôs-se então a realização deste estudo envolvendo todo este processo. O objetivo principal foi quantificar e avaliar a resposta da expressão dos miRNAs à correção endovascular de aneurisma de aorta abdominal com base em dosagens séricas no seguimento de seis meses. População e Método: Foram recrutados 30 pacientes consecutivos com AAA sem outras doenças inflamatórias associadas, do Ambulatório de Cirurgia Vascular e Endovascular do HCFMRPUSP com indicação de tratamento endovascular. Foram escolhidos para estudo e dosagens séricas os miRNA-191 e miRNA-455-3p. A expressão diferencial dos miRNAs foi realizada pelo método de PCR em tempo real, após extração do RNA das amostras de sangue total em dois momentos, pré- operatório e após 6 meses de pós-operatório. Além disso, ferramentas de bioinformática foram utilizadas para determinar vias fisiopatológicas relacionadas ao AAA. Foram Colhidos dados de perfil demográfico, de seguimento clinico e exames de imagem com angiotomografia no pré-operatórios e após 6 meses. Resultados: Foi observado uma hiperexpressão dos miR-191 e miR-455-3p no sangue total dos pacientes com AAA. O tratamento endovascular dos pacientes com AAA resultou em diminuição significativa das expressões dos miRNAs estudados, indicando que a exclusão do saco aneurismático altera as expressões dos mesmos. Adicionalmente, as expressões dos miR-191 e miR-455-3p não apresentaram correlação com o diâmetro do aneurisma e a análise da influência dos diversos tipos de dispositivos utilizados para o tratamento endovascular dos AAA, não mostrou diferenças significativas nas expressões dos miR-191 e miR-455-3p. Conclusões: A hiperexpressão dos miR-191 e miR-455-3p com sua significativa redução apos o tratamento endovascular, pode sugerir a utilização dessas moléculas como potenciais biomarcadores no seguimento desses pacientes. Novos estudos com maior número de casos devem ser realizados com o objetivo de validar os dados obtidos incluindo pacientes com eventuais vazamentos.
Background: Abdominal aortic aneurysm (AAA) is an important cause of morbidity and mortality in the elderly population. Endovascular treatment is associated with lower morbidity and mortality than conventional treatment, however, it requires a rigorous follow-up with contrast imaging tests to confirm the aneurysmal sac exclusion. Considering that the formation of an aneurysm is a complex multifactorial process, involving the destructive remodeling of the connective tissue throughout the affected segment of the aortic wall and that this process involves a chronic local inflammation, a decrease in the number of smooth muscle cells of the media tunic, and fragmentation of the extracellular matrix of the aorta and although an aberrant expression profile of miRNAs has been associated with human diseases, including cardiovascular dysfunction, it was proposed to carry out this study involving this whole process. The main objective was to quantify and evaluate miRNA expression response to endovascular correction of abdominal aortic aneurysm based on serum dosages at the six-month follow-up. Population and Method: We recruited 30 consecutive patients with AAA without other associated inflammatory diseases from the Ambulatory of Vascular and Endovascular Surgery of the HCFMRPUSP with indication of endovascular treatment. The miRNA-191 and miRNA-455-3p were selected for study and serum dosages. The differential expression of the miRNAs was performed by the real-time PCR method, after extraction of RNA from the whole blood samples at two moments, preoperatively and after 6 months of follow-up. In addition, bioinformatics tools were used to determine pathophysiological pathways related to AAA. Demographic profile, clinical follow-up and imaging examinations with angiotomography performed in the preoperative period and after 6 months were collected. Results: Hyperexpression of miR-191 and miR-455-3p in whole blood of AAA patients was observed. The endovascular treatment of patients with AAA resulted in a significant decrease in the expression of the miRNAS studied, indicating that the exclusion of the aneurysmal sac altered their expression. In addition, the expression of miR-191 and miR-455-3p showed no correlation with the diameter of the aneurysm and analysis of the influence of the various types of devices used for the endovascular treatment of AAA did not show significant differences in the expression of miR-191 And miR-455-3p. Conclusions: The hyperexpression of miR- 191 and miR-455-3p with its significant reduction after endovascular treatment may suggest the use of these molecules as potential biomarkers in the follow-up of these patients. New studies with a greater number of cases should be performed with the objective of validating the data obtained including patients with possible endoleaks.
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Svensjö, Sverker. "Screening for Abdominal Aortic Aneurysm." Doctoral thesis, Uppsala universitet, Kärlkirurgi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-198677.

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Randomised controlled trials have demonstrated that mortality from Abdominal Aortic Aneurysm (AAA) can be cost-effectively reduced by ultrasound-screening of men. Evidence for screening women is insufficient. Reports of falling AAA incidence are emerging. In an effort to study screening for AAA in a contemporary setting, two cross-sectional multi-centre population-based studies of one-time screening of 65-year-old men, and 70-year-old women in Middle Sweden were undertaken. Cost-efficiency of one-time screening of 65-year-old men was evaluated in a decision-analysis model. Five-year outcomes in men invited to screening at age 65 and age 70, were studied in a longitudinal cohort study. A lower than expected (1.7%) prevalence of AAA in 65-year-old men was found, as well as a very low (0.4%) prevalence in 70-year-old women. Smoking was the dominating risk factor associated with AAA, but the association was stronger in women. The main cause of reduced contemporary prevalence was falling smoking rates in the population since 30 years. One-time screening of 65-year-old men was found to be cost-effective and deliver significant clinical impact. The cost per quality adjusted life-year gained, at 13-years follow-up, was €14706, which was below the recommended UK NICE threshold of €25000. 15 lives were saved by inviting 10000 to screening. Prevalence of AAA and the rate of incidental detection of AAAs in the population were important factors affecting cost-efficiency. New AAAs developed after 5 years in men screened normal at age 65, predominantly in men with sub-aneurysmal aortas (25-29mm) at 65, and smokers. The 5-year rate of AAA repair was high among men with screening detected AAAs, as was non-AAA related mortality. Ruptures were only documented among non-attenders. Conclusions: A lower than expected prevalence of AAA among 65-year-old men, an unchanged repair rate, and improved longevity of the elderly population was found. Although one-time screening for AAA was still cost-effective within a contemporary context, several issues need to be addressed; the threshold diameter for follow-up, the current rate of opportunistic detection of AAA in the population, re-screening of the entire population at a higher age, and targeted screening of smokers. Screening 70-year-old women who do not smoke is likely to be futile, thus ruling out population screening of women for AAA.
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Lowe, Christopher. "Three-dimensional ultrasound in the management of abdominal aortic aneurysm." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/threedimensional-ultrasound-in-themanagement-of-abdominal-aorticaneurysm(b8950db7-847b-4d11-a6a5-2a06b3bb66d0).html.

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Objectives: Clinical implementation of 3D ultrasound (3D-US) in vascular surgery is in its infancy. The aim of this thesis was to develop novel clinical applications for 3D-US in the diagnosis and management of abdominal aortic aneurysm (AAA). Methods: Four principle clinical applications were investigated. 1) Intraoperative imaging – The ability of 3D-US to detect and classify endoleaks was compared with digital subtraction angiography in patients undergoing EVAR. 2) Detection and classification of endoleaks following endovascular aneurysm repair (EVAR) – The abilityof 3D-US to accurately detect and classify endoleaks following EVAR was compared to CTA and the final multi-disciplinary team decision. 3) AAA volume measurement – measurements using magnetic and optically-tracked 3D-US were compared to CTA. 4) Biomechanical analysis – the challenges of using 3D-US to generate surface models for biomechanical simulation was explored by development of an interactive segmentation technique and comparison of paired CT and 3D-US datasets. Optimal results were used in finite element analysis (FEA) and computational fluid dynamic(CFD) simulations. Results: 3D-US out-performed uniplanar angiography for the detection of endoleaks during EVAR. This approach allowed contrast-free EVAR to be performed in patients with poor renal function. 3D contrast-enhanced ultrasound was superior to CTA for endoleak detection and classification when compared with the final decision of the multi-disciplinary team. Optimal results for AAA volume measurements were gained using an optically tracked 3D-US system in EVAR surveillance. However, there remained a significant mean difference of 13.6ml between CT and 3D-US. Complete technical success of generating geometries for use in biomechanical analysis using 3D-US was only 5%. When the optimal results were used, a comparable CFD analysis under the conditions of steady, laminar and Newtonian flow was achieved. Using basic modelling assumptions in FEA, peak von Mises and principle wall stress was found to be at the same anatomical location on both the CT and 3D-US models but the 3D-US model overestimated the wall stress values by 41% and 51% respectively. Conclusions: 3D-US could be clinically implemented for intra-operative imaging and EVAR surveillance in specific cases. 3D-US volume measurement is feasible but future work should aim to improve accuracy and inter-observer reliability. Although the results of biomechanical analysis using the optimal results was encouraging and provided a proof-of-principal, there are a number of technical developments required to make this approach feasible in a larger number of patients.
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Chinien, Ganessen. "Molecular genetics of abdominal aortic aneurysm." Thesis, King's College London (University of London), 2012. https://kclpure.kcl.ac.uk/portal/en/theses/molecular-genetics-of-abdominal-aortic-aneurysm(e269485a-e71a-41a7-9a8e-ae40eb968dd4).html.

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Abdominal aortic aneurysm (AAA) is a common disorder and a major cause of death. Pathological processes involved in AAA formation include inflammation, proteolysis, angiogenesis and apoptosis. It has also a strong familial predisposition with linkage studies identifying chromosomes 19q13 and 4q31 as susceptible loci. AAA is likely to be a polygenic disorder. Aims The aims of this study were to carry out a whole transcriptome analysis in order to identify novel genes and pathways that are differentially expressed between aneurysmal (AAA), atheromatous (AOD) and normal (NA) aortic tissue and to confirm a set of these differentially expressed genes using quantitative real time polymerase chain reaction (qRT-PCR). Methods RNA samples were prepared from full thickness aortic walls obtained during open repair of AAA, aortic bypass for AOD and transplant patients for NA. The quality of the RNA was assessed using the Bioanalyzer 2100 (Agilent) and Nanodrop. RNA was then reverse transcribed to cDNA which was then hybridised to the Human Genome (HG) -U133 plus 2.0 microarray (Affymetrix) that interrogates the whole human genome. The robustness of the genearray was assessed using data output quality control as defined by Affymetrix. Statistical analysis was then carried out using the GeneSpring software. Genes were considered to be significantly differentiated if they had at least a two-fold change and a P-value < 0.05 following Benjamini-Hochberg multiple correction testing. Genes were then classified according to their molecular functions. A set of consistently differentially expressed genes were confirmed using qRT-PCR with Taqman probes on a larger sample size compared with the microarray experiment. All pathway and network analysis on the differentially expressed genes were conducted using MetaCore software Version 6.3 (GeneGo, Inc). Results A total of 3320 genes and 233 genes were differentially expressed when comparing AAA with NA and AAA with AOD respectively.
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Djavani, Gidlund Khatereh. "Intra-abdominal Hypertension and Colonic Hypoperfusion after Abdominal Aortic Aneurysm Repair." Doctoral thesis, Uppsala universitet, Institutionen för kirurgiska vetenskaper, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-149241.

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Colonic ischaemia (CI), Intra-abdominal hypertension (IAH) and abdominal compartment syndrome (ACS) are devastating complications after abdominal aortic aneurysm (AAA) surgery. The aims of this thesis were to study the incidence and clinical consequences of IAH/ACS and the association between CI and intra-abdominal pressure (IAP) among patients undergoing OR for ruptured AAA (rAAA), to compare extraluminal pHi monitoring, with standard intra-luminal monitoring among patients operated on for AAA, and to study the frequency and clinical consequences of IAH/ACS after endovascular repair (EVAR) for rAAA. The incidence of ACS was 26% in a retrospective study of 27 patients undergoing OR for rAAA. Consensus definitions on IAH/ACS were appropriate for patients after OR for rAAA: 78% (7/9) of patients with IAH grade III or IV developed organ failure and all patients who developed CI had some degree of IAH. Active fluid resuscitation treating hypovolaemia to avoid CI may partly cause IAH. The association between CI and IAP was investigated in a prospective study on 29 patients operated on for rAAA, 86% (25/29) were treated for hypovolaemia and ten (34%) had both IAH and CI. Since monitoring colonic perfusion is very important and there is no ideal method, a new technique, extraluminal colonic tonometry to detect colonic perfusion was compared with standard intraluminal tonometry. Although, this new method was not able to determine the severity of ischaemia it may serve as a screening test. EVAR of rAAA is feasible and patients may benefit from this less invasive procedure. Of 29 patients treated with this technique, 10% developed ACS, and all patients except one with preoperative shock developed some degree of IAH. In conclusion, IAP/ACS is common after both OR and EVAR for rAAA, and is associated with adverse outcome. Monitoring IAP and colonic perfusion with timely intervention may improve outcome.
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Alves, Lais Missae Murakami Domingues Estraiotto. "Estudo da expressão sérica do microRNA-1281, proteína C reativa e avaliação da função renal em indivíduos com aneurisma de aorta abdominal antes e após tratamento endovascular." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17137/tde-28052018-160605/.

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Introdução: O aneurisma de aorta abdominal (AAA) é uma doença prevalente e silenciosa também relacionada com a atividade inflamatória. Atualmente, a abordagem endovascular tem sido utilizada como principal técnica devido à inúmeras vantagens. Porém tem uma maior taxa de reintervenções e necessita de seguimento periódico com angiotomografias, o que aumenta custos e tem implicações como alteração da função renal além do acúmulo progressivo de radiação. Tais condições justificam a busca por possíveis biomarcadores que possam contribuir para um melhor seguimento. Objetivos: Neste estudo, buscou-se correlacionar o microRNA-1281, proteína C reativa (PCR) e a avaliação da função renal de indivíduos com AAA com a evolução dos mesmos após o tratamento endovascular. Pacientes e métodos: Foram selecionados 30 pacientes consecutivos do Ambulatório de Cirurgia Vascular e Endovascular do HCFMRP-USP, no período de janeiro de 2104 a novembro de 2015, com aneurisma de aorta abdominal e com indicação para tratamento endovascular. As dosagens séricas e avaliações angiotomográficas foram feitas no pré-operatório e 6 meses após a intervenção. Resultados: Houve uma hiperexpressão do microRNA-1281 nos pacientes com aneurisma e uma significativa redução dos seus níveis séricos após a correção endovascular. A expressão do miRNA-1281 apresentou correlação positiva com o clearence de creatinina. Houve também correlação positiva da PCR com a presença do aneurisma, e com seu diâmetro e não houve alteração significativa da função renal mensurada através das dosagens séricas de uréia, creatinina e cálculo indireto de clearence. Conclusão: O estudo mostrou que o miRNA 1281 tem boa correlação com a evolução favorável pós-tratamento endovascular do AAA, não se observando o mesmo com a proteína C reativa. Novos estudos são necessários para validar e complementar tais achados.
Introduction: Abdominal aortic aneurysm (AAA) is a prevalent and silent disease. Currently, the endovascular approach has been widely used and is the main technique due to the innumerable advantages. However, it has a higher rate of reintervention and requires periodic follow-up with tomography over the years, which increases its costs and has implications such as altered renal function besides the accumulation of radiation. Such conditions justify the search for possible biomarkers that may perhaps replace CT. Objectives: In this study, we sought to correlate the microRNA-1281, Creactive protein (CRP) and the renal function evaluation of individuals with AAA with their evolution after endovascular treatment. Patients and methods: We selected 30 consecutive patients from the Ambulatory of Vascular and Endovascular Surgery of the HCFMRP-USP, in the period from January of 2104 until November of 2015, with abdominal aortic aneurysm and with indication for endovascular treatment. Serum dosages were made preoperatively and 6 months after the intervention Results: There was a hyperexpression of the micro-RNA -1281 in patients with aneurysm and a significant reduction of their serum levels after endovascular correction. Expression of miRNA-1281 showed a positive correlation with creatinine clearence. There was also a positive correlation of CRP with the presence of the aneurysm, and with its diameter, and there was no significant alteration of renal function measured through serum urea, creatinine and indirect clearance calculations. Conclusion: The study showed that 1281 miRNAs may prove to be a potential biomarker for eventual follow-up of patients undergoing AAA endovascular repair. New studies are needed to validate and complement these findings.
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Books on the topic "Abdomina Aortic Aneurysm"

1

E, Pierce George, ed. Abdominal aortic aneurysms. Philadelphia: Saunders, 1989.

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Starnes, Benjamin W., Manish Mehta, and Frank J. Veith, eds. Ruptured Abdominal Aortic Aneurysm. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-23844-9.

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1956-, Calligaro Keith D., Dougherty Matthew J, and Hollier Larry H, eds. Diagnosis and treatment of aortic and peripheral arterial aneurysms. Philadelphia: W.B. Saunders, 1999.

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G, Hakaim Albert, ed. Current endovascular treatment of abdominal aortic aneurysms. Oxford: Blackwell Pub., 2005.

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Hakaim, Albert G., ed. Current Endovascular Treatment of Abdominal Aortic Aneurysms. Oxford, UK: Blackwell Publishing, 2006. http://dx.doi.org/10.1002/9780470753156.

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J, Doyle Barry, ed. 3D imaging of abdominal aortic aneurysms: Techniques and applications. Hauppauge, N.Y: Nova Science, 2010.

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David, Tilson M., Kuivaniemi Helena, and Upchurch Gilbert R, eds. The abdominal aortic aneurysm: Genetics, pathophysiology, and molecular biology. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

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W, Calvert N., and Trent Institute for Health Services Research. Working Group on Acute Purchasing., eds. The use of endovascular stents for abdominal aortic aneurysm. [Sheffield]: Trent Institute for Health Services Research, 1999.

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David, Tilson M., and Boyd Charles D, eds. The abdominal aortic aneurysm: Genetics, pathophysiology, and molecular biology. New York, N.Y: New York Academy of Sciences, 1996.

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Dueck, Andrew D. Care of ruptured abdominal aortic aneurysms in Ontario. Ottawa: National Library of Canada, 2003.

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Book chapters on the topic "Abdomina Aortic Aneurysm"

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Lillvis, John H., Guy M. Lenk, and Helena Kuivaniemi. "Genetics of Abdominal Aortic Aneurysms." In Aortic Aneurysms, 1–26. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-204-9_1.

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Englesbe, Michael J. "Abdominal Aortic Aneurysms in Transplant Patients." In Aortic Aneurysms, 277–88. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-204-9_19.

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Patel, Sheela T., and C. Parodi Juan. "Endovascular Repair of Abdominal Aortic Aneurysms." In Aortic Aneurysms, 121–32. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-204-9_7.

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Friedewald, Vincent E. "Abdominal Aortic Aneurysm." In Clinical Guide to Cardiovascular Disease, 1–13. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7293-2_1.

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Dharmarajan, T. S., T. S. Dharmarajan, T. S. Dharmarajan, and T. S. Dharmarajan. "Abdominal Aortic Aneurysm." In Geriatric Gastroenterology, 631–36. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-1623-5_68.

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March, Robert J. "Abdominal Aortic Aneurysm." In Common Surgical Diseases, 107–10. New York, NY: Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2945-0_25.

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d’Audiffret, A., and J. P. Becquemin. "Abdominal Aortic Aneurysm." In Vascular Surgery, 11–16. London: Springer London, 2003. http://dx.doi.org/10.1007/978-1-4471-3870-9_2.

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Dharmarajan, T. S., and Nilesh N. Balar. "Abdominal Aortic Aneurysm." In Geriatric Gastroenterology, 1–16. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-90761-1_87-1.

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Danzer, Daniel, and Jean-Pierre Becquemin. "Abdominal Aortic Aneurysm." In Vascular Surgery, 15–24. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-356-5_2.

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Becquemin, Jean-Pierre, and Alexandre d’Audiffret. "Abdominal Aortic Aneurysm." In Vascular Surgery, 13–21. London: Springer London, 2006. http://dx.doi.org/10.1007/1-84628-211-x_2.

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Conference papers on the topic "Abdomina Aortic Aneurysm"

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Trachet, Bram, Marjolijn Renard, Joris Bols, Steven Staelens, Bart Loeys, and Patrick Segers. "Hemodynamics in Ascending and Abdominal Aorta Aneurysm Formation in the ApoE−/− Angiotensin II Mouse Model." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80243.

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Aortic aneurysm is a pathological dilatation of the aorta that can be life-threatening when it ruptures. Aneurysms occur throughout the entire aorta but there is a predisposition for the ascending and the abdominal aorta, an observation that cannot be fully explained by the current knowledge of the disease pathophysiology. ApoE −/− mice infused with angiotensin II have recently been reported to develop not only abdominal [1], but also ascending aortic aneurysms [2]. These animals thus provide the perfect model to compare aneurysm progression in both aortic locations and to investigate whether disturbed hemodynamics play a role in the initial phase of aneurysm growth. In this study, both imaging and computational biomechanics techniques were used to elucidate the flow field at the location of the aneurysm prior to onset of the disease.
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Ene, Florentina, Carine Gachon, Patrick Delassus, and Liam Morris. "Investigating the Effect of Intraluminal Thrombus in Abdominal Aortic Aneurysm by Computational and Experimental Methods." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206636.

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Abdominal aortic aneurysm (AAA) represents an abnormal dilatation and weakening of the abdominal aorta with high risk of rupture. Most aneurysms of the infrarenal aorta possess an asymmetrical fusiform morphology.
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Martufi, Giampaolo, Jose F. Rodriguez, and Ender A. Finol. "Anisotropic Wall Mechanics of Abdominal Aortic Aneurysms." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192265.

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The prevalence of AAA is growing along with population age and according to different studies AAA rupture is the 13th most common cause of death in the U.S., causing an estimated 15,000 deaths per year. In biomechanical terms, AAA rupture is a phenomenon that occurs when the developing mechanical stresses within the aneurysm inner wall, as a result of the exerted intraluminal pressure, exceed the failure strength of the aortic tissue. To obtain a reliable estimation of wall stress, it is necessary to perform an accurate three-dimensional reconstruction of the AAA geometry and model an appropriate constitutive law for the aneurysmal tissue material characterization. In this regard, a recent study on the biaxial mechanical behavior of human AAA tissue specimens [1] demonstrates that aneurysmal arterial tissue behaves mechanically anisotropic. The objectives of the present work are to determine the effect of material anisotropy of the aneurysmal abdominal aorta on wall stress distribution and to establish a comparison of wall mechanics between ruptured and unruptured aneurysms.
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Washington, Christopher B., Judy Shum, Satish C. Muluk, and Ender A. Finol. "Abdominal Aortic Aneurysm Growth: The Association of Aortic Wall Mechanics and Geometry." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53977.

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In an effort to prevent rupture, patients with known AAA undergo periodic abdominal ultrasound or CT scan surveillance. When the aneurysm grows to a diameter of 5.0–5.5 cm or is shown to expand at a rate greater than 1 cm/yr, elective operative repair is undertaken. While this strategy certainly prevents a number of potentially catastrophic ruptures, AAA rupture can occur at sizes less than 5 cm. From a biomechanical standpoint, aneurysm rupture occurs when wall stress exceeds wall strength. By using non-invasive techniques, such as finite element analysis (FEA), wall stress can be estimated for patient specific AAA models, which can perhaps more carefully predict the rupture potential of a given aneurysm, regardless of size. FEA is a computational method that can be used to evaluate complicated structures such as aneurysms. To this end, it was reported earlier that AAA peak wall stress provides a better assessment of rupture risk than the commonly used maximum diameter criterion [1]. What has yet to be examined, however, is the relationship between wall stress and AAA geometry during aneurysm growth. Such finding has the potential for providing individualized predictions of AAA rupture potential during patient surveillance. The purpose of this study is to estimate peak wall stress for an AAA under surveillance and evaluate its potential correlation with geometric features characteristic of the aneurysm’s morphology.
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Looyenga, Eric M., and Stephen P. Gent. "Examination of Fluid-Structure Interaction in Stent Grafts and its Hemodynamic Implications." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6872.

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Every year in the United States, 4,500 deaths occur from abdominal aortic aneurysm (AAA) rupture. Aneurysms develop when the arterial wall weakens. Many risk factors can contribute to aneurysm formation, including age, sex, ethnicity, smoking and hypertension [1]. AAAs are the most common form of aneurysm because the aorta experiences the highest wall shear stress (WSS) of any vessels in the human body. These aneurysms are 5–6% prevalent in men and 1–2% in women, both over 65 years of age [2]. In the aorta, high WSS causes plaque formation, but in peripheral arteries where the flow rate is lower, atherosclerosis can also trigger aneurysm formation.
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Zeinali-Davarani, S., A. Sheidaei, and S. Baek. "Towards Patient-Specific Modeling of an Enlarging Abdominal Aortic Aneurysm." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205488.

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There has been a clear need for better understanding of the progression of abdominal aortic aneurysm (AAA) and obtaining reliable prediction of the AAA rupture. Finite element analysis (FEA) using non-axisymmetric models of AAAs provides better estimation of stress distribution in the aneurysmal wall with complex shapes [1]. However, FEA alone does not provide a mathematical description for the evolution of an AAA through growth and remodeling (G&R). A computational framework for modeling stress-mediated growth and structural remodeling of the arterial wall under physiological and pathological conditions has been suggested using a constrained mixture assumption [2]. Stress-mediated enlargement of intracranial aneurysms has been investigated using idealized axisymmetric geometries [3,4]. The kinetics of stress-mediated turnover of collagen fiber families and degradation of elastin were found to have particular importance in the G&R of aneurysmal wall.
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Vorp, David A., and David H. J. Wang. "Use of Finite Elasticity in Abdominal Aortic Aneurysm Research." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-1928.

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Abstract Abdominal aortic aneurysm (AAA) is a condition whereby the terminal aorta permanently dilates to dangerous proportions, risking rupture. Rupture of AAA is a significant cause of death in this country that is generally avoided by surgical repair. In order to improve our understanding and diagnosis of this disease, we call upon first principles of finite elasticity. We use such principles to estimate the AAA wall strength and acting wall stress, which in turn allow evaluation of an individual aneurysm’s risk of rupture. In this paper, we summarize the work that has been performed to date in applying finite elasticity to abdominal aortic aneurysm research.
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Finol, Ender A., and Cristina H. Amon. "Secondary Flow and Wall Shear Stress in Three-Dimensional Steady Flow AAA Hemodynamics." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23013.

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Abstract Abdominal Aortic Aneurysms (AAAs) are balloon-shaped expansions commonly found in the infrarenal segment of the abdominal aorta, between the renal arteries and the iliac bifurcation. The mean age of patients with AAA is 67 years and males are affected more often than women in a ratio of 4:1. Abdominal aortic aneurysm rupture is the 13th leading cause of death in the United States, affecting 1 in 250 individuals greater than 50 years of age. AAAs usually remain asymptomatic while slowly enlarging over a period of years or even decades. Factors that are known to affect the risk of aneurysm rupture are: maximum transverse dimension of the aneurysm, its expansion rate, its relative size compared to the patient’s body size, smoking, and family history of the patient. The five-year survival rate is only 19% and the overall mortality rate following rupture may exceed 90% [1]. Therefore, aneurysm screening and determination of the factors that may have an important role in aneurysm growth and rupture have become important elements in the investigation of this clinical problem.
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Gee, Michael W., and Wolfgang A. Wall. "Model Complexity and Prestressing in Abdominal Aortic Aneurysm Simulation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204593.

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Rupture of abdominal aortic aneurysm (AAA) is the 13th leading cause of death in western society and is fatal in 70–90%. In consequence, precise prediction of AAA rupture risk is essential. With the current, well established CT-morphological parameters such as maximum aortic diameter, aneurysm shape and AAA expansion, only at best the relative, but not the individual rupture risk can be determined. Hence, AAA rupture may occur unexpectedly in small aneurysms below the critical diameter limits whereas many large aneurysms may remain stable throughout patient’s lifetime, without prophylactic surgery.
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Swillens, Abigail, Lieve Lanoye, Julie De Backer, Nikos Stergiopulos, Frank Vermassen, Pascal Verdonck, and Patrick Segers. "The Impact of an Abdominal Aortic Aneurysm on Aortic Wave Reflection." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175514.

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The economical growth and increased welfare in the Western world have a reverse side, with an increased death toll due to cardiovascular diseases. Among these, aortic aneurysms (a local dilation) are particularly lethal as they may grow unnoticed until rupture occurs. In this study, we assessed the impact of the presence of an abdominal aortic aneurysm on arterial hemodynamics and wave reflection in particular. Experimental and numerical methods were applied. Linear wave separation was used to quantify the reflections; wave intensity analysis was applied to assess the nature of the reflected waves. In both the experimental and numerical models, negative reflections were found in the upper aorta corresponding to a backward expansion wave caused by the sudden expansion of the aorta. A numerical parameter study demonstrated that larger diameters and more compliant aneurysms generate stronger negative reflections.
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Reports on the topic "Abdomina Aortic Aneurysm"

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Karmy-Jones, R. Abdominal Aortic Aneurysm and Pheochromocytoma. Science Repository, June 2019. http://dx.doi.org/10.31487/j.ijscr.2019.01.02.

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Jianqing, Deng, Jie Liu, Dan Rong, Yangyang Ge, Hongpeng Zhang, and Xiaoping Liu. Locoregional Anesthesia Versus General Anesthesia in Endovascular Repair of Ruptured Abdominal Aortic Aneurysm: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review Protocols, March 2020. http://dx.doi.org/10.37766/inplasy2020.3.0010.

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Endovascular aortic repair (EVAR) surgery more beneficial for ruptured abdominal aortic aneurysms than open repair. National Institute for Health Research, August 2018. http://dx.doi.org/10.3310/signal-000638.

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Fewer wound hernias occur if mesh is used to reinforce abdominal aortic aneurysm surgery. National Institute for Health Research, September 2018. http://dx.doi.org/10.3310/signal-000644.

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Abdominal aortic aneurysm screening for women is unlikely to be a fair use of NHS resources. National Institute for Health Research, November 2018. http://dx.doi.org/10.3310/signal-000676.

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No clear difference between open and keyhole surgery for the repair of ruptured abdominal aortic aneurysms. National Institute for Health Research, May 2016. http://dx.doi.org/10.3310/signal-000234.

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