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Relevant bibliographies by topics / A9 neurons

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Journal articles

Academic literature on the topic 'A9 neurons'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "A9 neurons"

1

Haynes, John M., Shanti M. Sibuea, Alita A. Aguiar, Fangwei Li, Joan K. Ho та Colin W. Pouton. "Inhibition of β-catenin dependent WNT signalling upregulates the transcriptional repressor NR0B1 and downregulates markers of an A9 phenotype in human embryonic stem cell-derived dopaminergic neurons: Implications for Parkinson’s disease". PLOS ONE 16, № 12 (2021): e0261730. http://dx.doi.org/10.1371/journal.pone.0261730.

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In this study we investigate how β-catenin-dependent WNT signalling impacts midbrain dopaminergic neuron (mDA) specification. mDA cultures at day 65 of differentiation responded to 25 days of the tankyrase inhibitor XAV969 (XAV, 100nM) with reduced expression of markers of an A9 mDA phenotype (KCNJ6, ALDH1A1 and TH) but increased expression of the transcriptional repressors NR0B1 and NR0B2. Overexpression of NR0B1 and or NR0B2 promoted a loss of A9 dopaminergic neuron phenotype markers (KCNJ6, ALDH1A1 and TH). Overexpression of NR0B1, but not NR0B2 promoted a reduction in expression of the β-c

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2

Fiorenzano, Alessandro, Edoardo Sozzi, Malin Parmar, and Petter Storm. "Dopamine Neuron Diversity: Recent Advances and Current Challenges in Human Stem Cell Models and Single Cell Sequencing." Cells 10, no. 6 (2021): 1366. http://dx.doi.org/10.3390/cells10061366.

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Human midbrain dopamine (DA) neurons are a heterogeneous group of cells that share a common neurotransmitter phenotype and are in close anatomical proximity but display different functions, sensitivity to degeneration, and axonal innervation targets. The A9 DA neuron subtype controls motor function and is primarily degenerated in Parkinson’s disease (PD), whereas A10 neurons are largely unaffected by the condition, and their dysfunction is associated with neuropsychiatric disorders. Currently, DA neurons can only be reliably classified on the basis of topographical features, including anatomic

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3

GRENHOFF, J., L. UGEDO, and T. H. SVENSSON. "Firing patterns of midbrain dopamine neurons: differences between A9 and A10 cells." Acta Physiologica Scandinavica 134, no. 1 (1988): 127–32. http://dx.doi.org/10.1111/j.1748-1716.1988.tb08468.x.

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4

Pujo, J., G. De Palma, J. Lu, S. M. Collins, and P. Bercik. "A9 DORSAL ROOT GANGLIA NEURONAL RESPONSES AND SUBSTANCE P PRODUCTION ARE HIGHER IN MALE MICE." Journal of the Canadian Association of Gastroenterology 4, Supplement_1 (2021): 10–11. http://dx.doi.org/10.1093/jcag/gwab002.008.

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Abstract Background Abdominal pain is a common complaint in patients with chronic gastrointestinal disorders. Accumulating evidence suggests that gut microbiota is an important determinant of gut function, including visceral sensitivity. Germ-free (GF) mice have been shown to display visceral hypersensitivity, which normalizes after colonization. Sex also appears to play a key role in visceral sensitivity, as women report more abdominal pain than men. Thus, both gut bacteria and sex are important in the regulation of gut nociception, but the underlying mechanisms remain poorly understood. Aims

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5

Yang, S., L. C. Edman, J. A. Sanchez-Alcaniz, et al. "Cxcl12/Cxcr4 signaling controls the migration and process orientation of A9-A10 dopaminergic neurons." Journal of Cell Science 126, no. 22 (2013): e1-e1. http://dx.doi.org/10.1242/jcs.145136.

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6

Stockton, Marsha E., and Kurt Rasmussen. "Electrophysiological Effects of Olanzapine, a Novel Atypical Antipsychotic, on A9 and A10 Dopamine Neurons." Neuropsychopharmacology 14, no. 2 (1996): 97–104. http://dx.doi.org/10.1016/0893-133x(94)00130-r.

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7

Yang, S., L. C. Edman, J. A. Sanchez-Alcaniz, et al. "Cxcl12/Cxcr4 signaling controls the migration and process orientation of A9-A10 dopaminergic neurons." Development 140, no. 22 (2013): 4554–64. http://dx.doi.org/10.1242/dev.098145.

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8

German, Dwight C., and Kebreten F. Manaye. "Midbrain dopaminergic neurons (nuclei A8, A9, and A10): Three-dimensional reconstruction in the rat." Journal of Comparative Neurology 331, no. 3 (1993): 297–309. http://dx.doi.org/10.1002/cne.903310302.

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9

Bye, Christopher R., Lachlan H. Thompson, and Clare L. Parish. "Birth dating of midbrain dopamine neurons identifies A9 enriched tissue for transplantation into Parkinsonian mice." Experimental Neurology 236, no. 1 (2012): 58–68. http://dx.doi.org/10.1016/j.expneurol.2012.04.002.

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10

Goldstein, Jeffrey M., Linda C. Litwin, E. B. Sutton, and Jeffrey B. Malick. "D-2 dopamine antagonist-like effects of SCH 23390 on A9 and A10 dopamine neurons." Life Sciences 40, no. 11 (1987): 1039–44. http://dx.doi.org/10.1016/0024-3205(87)90565-0.

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