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1

Lee, H., M. Ko, S. Oh, and K. Shin. "(665)." Journal of Pain 7, no. 4 (April 2006): S22. http://dx.doi.org/10.1016/j.jpain.2006.01.067.

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2

Wong, F. "(665)." Journal of Pain 8, no. 4 (April 2007): S17. http://dx.doi.org/10.1016/j.jpain.2007.02.076.

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Mashburn, Danielle, Katherine Ward, Cameron Gunville, Emily Dobyns, Beth Wathen, and Jen Roth. "665." Critical Care Medicine 41 (December 2013): A163. http://dx.doi.org/10.1097/01.ccm.0000439903.14323.f0.

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O’Brien, Kevin, Ed Wong, Perry Lubens, and Christopher Babbitt. "665." Critical Care Medicine 42 (December 2014): A1520. http://dx.doi.org/10.1097/01.ccm.0000458162.91746.74.

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5

Cartron, Alexander, William Pastor, and Venkat Shankar. "665." Critical Care Medicine 43 (December 2015): 168. http://dx.doi.org/10.1097/01.ccm.0000474493.98991.a2.

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6

Brothers, R. Matthew. "665." Medicine & Science in Sports & Exercise 50, no. 5S (May 2018): 142. http://dx.doi.org/10.1249/01.mss.0000535553.48834.f9.

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7

Gombas, Michelle, Benny Joyner, Elizabeth Fitzgerald, and Anthony Charles. "665." Critical Care Medicine 47 (January 2019): 313. http://dx.doi.org/10.1097/01.ccm.0000551417.92596.30.

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8

Lajoie, Lidie, and Robert Schulze. "665." Critical Care Medicine 40 (December 2012): 1–328. http://dx.doi.org/10.1097/01.ccm.0000424881.78509.11.

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9

Carroll, Megan, Brajesh Lal, Shahab Toursavadkohi, and Kathleen To. "665." Critical Care Medicine 48 (January 2020): 313. http://dx.doi.org/10.1097/01.ccm.0000626400.69995.e0.

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10

Adams, Gregory R. "665." Medicine & Science in Sports & Exercise 39, Supplement (May 2007): S35—S36. http://dx.doi.org/10.1249/01.mss.0000273024.12956.49.

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11

McViety, J., M. Horan, A. Timmis, and K. Parkins. "ABSTRACT 665." Pediatric Critical Care Medicine 15 (May 2014): 150. http://dx.doi.org/10.1097/01.pcc.0000449391.56698.6a.

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12

Evans, M. I., and D. W. Britt. "P-665." Fertility and Sterility 86, no. 3 (September 2006): S379—S380. http://dx.doi.org/10.1016/j.fertnstert.2006.07.1044.

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13

Xanthos, Martin. "665. SINOBAMBUSA TOOTSIK." Curtis's Botanical Magazine 27, no. 1 (March 2010): 2–9. http://dx.doi.org/10.1111/j.1467-8748.2010.01679.x.

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14

Lin, Yongqiang, Mengjia Li, Gaofeng Rao, Wenfu Zhang, and Xuyan Chen. "Inhibition of miR-665 alleviates neuropathic pain by targeting SOCS1." Tropical Journal of Pharmaceutical Research 19, no. 8 (November 18, 2020): 1591–97. http://dx.doi.org/10.4314/tjpr.v19i8.4.

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Purpose: To investigate the effect of miR-665 in neuropathic pain and the possible molecular mechanism involved.Methods: A neuropathic pain model was established using chronic constriction injury (CCI) methods in Sprague Dawley (SD) rats. Mechanical and thermal hyperalgesia were measured using paw withdrawal threshold (PWT) and paw withdrawal latency (PWL), respectively. The inflammation response was determined by assessing the production of inflammation factors. The target relationship of miR-665 and suppressor of cytokine signaling 1 (SOCS1) was verified by luciferase assay.Results: In the CCI rat model, PWT and PWL decreased following treatment with miR-665 (p < 0.01). MiR-665 was elevated in the spinal cord and microglia of CCI rats at different time points (p < 0.01). Down-regulation of miR-665 increased PWT and PWL and inhibited the production of interleukin (IL)-1β, IL-6, and tumor necrosis factor (TNF)-α in CCI rats (p < 0.01). Luciferase assay results indicate that SOCS1 was the target of miR-665 (p < 0.01). SOCS1 decreased in CCI rats (p < 0.01) after treatment with miR-665. MiR-665 negatively regulated the expression of SOCS1 (p < 0.01). Down-regulation of SOCS1 reversed the alleviating effect of decreased miR-665 on pain sensitivity and inflammationresponse (p < 0.01).Conclusion: Down-regulation of miR-665 alleviates neuropathic pain by targeting SOCS1, and hence making miR-665 a promising therapeutic target for neuropathic pain. Keywords: MiR-665, SOCS1, Neuropathic pain, CCI, Spinal cord
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15

Qi, Yongjie, Yue Han, Chen Chen, Yingchang Xue, Nianhui Jiao, and Xuejun Li. "Inhibition of microRNA-665 Alleviates Septic Acute Kidney Injury by Targeting Bcl-2." Journal of Healthcare Engineering 2022 (March 3, 2022): 1–9. http://dx.doi.org/10.1155/2022/2961187.

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Sepsis can easily cause acute kidney injury (AKI) and seriously endanger human health. This article aims to investigate and study the role of microRNA-665 (miR-665) in septic AKI and the underlying molecular mechanism. Lipopolysaccharide (LPS) was used to construct cell and animal models of septic AKI. The expression of miR-665 in cells and kidney tissues was detected by quantitative reverse-transcription polymerase chain reaction (RT-PCR). The contents of inflammatory factors (TNF-α, IL-1β, and IL-6) in the cell supernatant were detected using commercial kits. Renal tissue damage was observed by hematoxylin-eosin (HE) staining. Kidney function was assessed by serum Cr, serum BUN, and urine NAG levels. The apoptosis of HK-2 cells was analyzed by flow cytometry and TUNEL staining. Luciferase activity assay was performed for the verification of the target of miR-665. The expression of miR-665 was increased in the cell model and animal model of septic AKI constructed by LPS. By transfecting miR-665 inhibitor in HK-2 cells and injecting miR-665 antagomir (antagomiR-665) through the tail vein of rats, the expression of miR-665 in HK-2 cells and rat kidneys was remarkably reduced. Silencing miR-665 dramatically inhibited the expression of inflammatory factors (TNF-α, IL-1β, and IL-6) in LPS-induced HK-2 cells and reduced LPS-induced apoptosis in HK-2 cells. At the same time, the levels of serum Cr, serum BUN, and urine NAG decreased markedly, and the damage of the kidney was also alleviated. Finally, luciferase reporter experiments demonstrated that miR-665 directly targets Bcl-2. We revealed that miR-665 expression was increased in septic AKI, and silencing miR-665 could inhibit LPS-induced inflammation and apoptosis of the kidney by targeting Bcl-2, thereby improving renal function.
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16

Liu, Xiting, Ze Chen, Yuqiang Wu, Feng Gu, Dong Yan, Lei Yang, Qin Ma, and Caihong Fu. "Circ_0078767 Inhibits the Progression of Non-Small-Cell Lung Cancer by Regulating the GPX3 Expression by Adsorbing miR-665." International Journal of Genomics 2022 (March 17, 2022): 1–15. http://dx.doi.org/10.1155/2022/6361256.

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Non-small-cell lung cancer (NSCLC) is one of the most serious cancers. The circular RNA_0078767 (circ_0078767) expression was decreased in NSCLC tissues. However, the molecular mechanism of circ_0078767 remains unknown. The expression of circ_0078767, microRNA-665 (miR-665), and glutathione peroxidase 3 (GPX3) was detected by quantitative real-time fluorescence polymerase chain reaction (qRT-PCR). Cell proliferation, migration, and invasion were detected by colony formation assay and transwell assay, respectively. The lactate production and glucose consumption were tested by glycolysis. Western blot examined the protein levels of hexokinase-2 (HK2), matrix metalloproteinase-9 (MMP9), and GPX3 cells. Circinteractome predicted the relationship between miR-665 and circ_0078767 or GPX3 and was verified by dual luciferase reporter assays. The xenotransplantation model was established to study the role of circ_0078767 in vivo. The expression of circ_0078767 and GPX3 was decreased in NSCLC tissues, while the expression of miR-665 was increased. Circ_0078767 can sponge miR-665, and GPX3 is the target of miR-665. In vitro complement experiments showed that knockdown of circ_0078767 significantly promoted malignant behavior of NSCLC, while cotransfection of miR-665 inhibitor partially reduced this change. In addition, the GPX3 overexpression decreased the promoting effects of miR-665 upregulation on proliferation, migration, and invasion of NSCLC cells. Mechanically, circ_0078767 regulates the GPX3 expression in NSCLC cells by spongy miR-665. In addition, in vivo studies have shown that downregulation of circ_0078767 promotes tumor growth. Circ_0078767 silencing promotes proliferation, migration, invasion, and glycolysis of NSCLC cells by regulating the miR-665/GPX3 axis, suggesting that circ_0078767/miR-665/GPX3 axis may be a potential regulatory mechanism for the treatment of NSCLC.
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17

Heck, Robert W., John A. Lombardo, and Richard A. Fischer. "665 SHOULDER INJURY???FOOTBALL." Medicine & Science in Sports & Exercise 25, Supplement (May 1993): S120. http://dx.doi.org/10.1249/00005768-199305001-00667.

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18

 . "665 CTG/ZAIO-Circulaires." Zorg en Financiering 5, no. 5 (May 2006): 69–71. http://dx.doi.org/10.1007/bf03092489.

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19

Ning, Qiaoyi, Yiting He, Wukai Ma, Fang Tang, Ying Huang, and Xueming Yao. "Inhibition of miR-665 alleviates lipopolysaccharide-induced inflammation via up-regulation of SOCS7 in chondrogenic ATDC5 cells." Tropical Journal of Pharmaceutical Research 19, no. 10 (November 25, 2020): 2067–72. http://dx.doi.org/10.4314/tjpr.v19i10.7.

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Purpose: To examine the effect and mechanism of action of miR-665 in osteoarthritis.Methods: An in vitro inflammatory injury model of osteoarthritis was established using chondrogenic ATDC5 cells with lipopolysaccharide (LPS) treatment. The expression levels of inflammatory cytokines were determined by enzyme-linked immunosorbent assays (ELISAs) and by quantitative real-time polymerase chain reaction (qRT-PCR). A binding target for miR-665 was predicted using TargetScan and then evaluated using a dual-luciferase reporter assay.Results: Treatment with LPS significantly up-regulated the inflammatory cytokine expressions of interleukin-1β (IL-1β), IL-6, and tumor necrosis factor-alpha (TNF-α), in ATDC5 cells (p < 0.01), and the expression of miRNA-665 was significantly increased in LPS-treated ATDC5 cells (p < 0.01).Knockdown of miR-665 down-regulated the expression levels of these inflammatory cytokines. Suppressor of cytokine signaling 7 (SOCS7) was identified as a target of miR-665. Data from qRT-PCR and western-blot analyses indicated that SOCS7 expression was promoted by miR-665 inhibition and inhibited by miR-665 over-expression. LPS treatment significantly decreased the expression of SOCS7 protein in ATDC5 cells (p < 0.01), and over-expression of SOCS7 attenuated the LPS-stimulated inflammatory injury. In addition, over-expression of miR-655 enhanced the inflammatory injury and reversed the protective effect of SOCS7 against LPS-stimulated inflammation.Conclusion: Inhibition of miR-665 alleviated LPS-stimulated inflammatory injury in ATDC5 cells via the up-regulation of SOCS7, suggesting a potential therapeutic target for osteoarthritis. Keywords: MiR-665, Lipopolysaccharide, Inflammation, SOCS7, Chondrogenic, ATDC5
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20

Mohamed, Amal Ahmed, Abdellah Abosrie Ali Omar, Rehab R. EL-Awady, Sally Mohamed Aboelsayed Hassan, Waleed Mohamed Soliman Eitah, Rehab Ahmed, Amir khater, Omnia Mohamed Saad Tantawi, and Ahmed Abdelhafeez Mohamed. "MiR-155 and MiR-665 role as potential non-invasive biomarkers for hepatocellular carcinoma in Egyptian patients with chronic hepatitis C virus infection." Journal of Translational Internal Medicine 8, no. 1 (May 9, 2020): 32–40. http://dx.doi.org/10.2478/jtim-2020-0006.

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AbstractBackground and ObjectivesHepatocellular carcinoma (HCC) is the fourth leading cause of cancer associated death globally. Serum micro RNAs are full of potential as noninvasive biomarkers. Here, we aim to assess the performance of serum MicroRNA-155 and MicroRNA-665 as diagnostic biomarker for HCC comparing to AFP.MethodsSerum samples were collected from 200 subjects (40 healthy control, 80 chronic hepatitis C patients with cirrhosis and without HCC (LC) and 80 HCC patients currently infected by hepatitis C infection and didn’t start the treatment). The HCC patients didn’t include alcoholic liver disease, nonalcoholic fatty liver disease nor autoimmune liver disease. MicroRNA-155 and MicroRNA-665 expression were measured by real-time quantitative PCR (RT-qPCR), while AFP level was assessed by ELISA method.ResultsBoth miR-155 and miR-665 were significantly elevated in HCC group as compared to both control and LC groups. The comparison between LC and HCC patients revealed that the serum level of miR-155 was a significant increase in HCC patients compared to LC patients; however, the serum level of miR-665 didn’t show any significant difference between the same two groups. MiR-665 expression level showed a direct correlation with tumor size in HCC patients.ConclusionsUsing measurement against AFP level in serum, miR-665 is considered a promising serum biomarker for the diagnosis of HCC patients among the LC patients without HCC. MiR-155 didn’t provide a better performance than serum AFP as a diagnostic biomarker among the same group. MiR-665 may serve as a good indicator for HCC prognosis.
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Arpaci, Ibrahim, Thabet Abdeljawad, Mustafa Baloğlu, Şahin Kesici, and Ibrahim Mahariq. "Mediating Effect of Internet Addiction on the Relationship Between Individualism and Cyberbullying: Cross-Sectional Questionnaire Study." Journal of Medical Internet Research 22, no. 5 (May 28, 2020): e16210. http://dx.doi.org/10.2196/16210.

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Background Among a variety of dynamics that may have effects on internet-related behaviors, cultural orientation is particularly important. Previous studies suggest that individualism is a strong determinant of certain behaviors. In addition, findings suggest that vertical individualism may lead to the development of more tolerance for addiction and aggression on the internet. Objective This study aimed to investigate whether vertical individualism has significant positive effects on cyberbullying and internet addiction and whether horizontal individualism has significant negative effects on cyberbullying and internet addiction. A theoretical model was specified to test the relationships among vertical versus horizontal individualism, cyberbullying, and internet addiction. Methods A total of 665 college students were selected using a convenience sampling method and willingly participated in the study. Participants’ ages ranged from 17 to 19 years (mean 17.94 years, SD 1.12 years). Of the group, 462 were women (462/665, 69.5%), and 203 were men (203/665, 30.5%). Study majors represented were mathematics (113/665, 17%), sciences (102/665, 15.3%), instructional technology (99/665, 14.9%), psychology (98/665, 14.7%), and others (253/665, 38.1%). Self-report instruments were used to measure vertical/horizontal individualism, cyberbullying, and internet addiction. Results Results show a significant positive effect of vertical individualism (effect size 0.10) and significant negative effect of horizontal individualism (effect size –0.12) on cyberbullying. In addition, the direct effect of vertical individualism on internet addiction was significant (effect size 0.28), but the direct effect of horizontal individualism was not (effect size –0.05). Internet addiction had a significant direct effect on cyberbullying (effect size 0.39) as well as an intervening effect on the relationship between vertical individualism and cyberbullying. Results also indicate significant gender differences in cultural patterns and internet addiction. Conclusions The findings suggest that horizontal and vertical individualism have significant effects on internet addiction. The findings also suggest that vertical individualists are more vulnerable to internet addiction. Further, the findings indicate a significant relationship between internet addiction and cyberbullying.
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Rahmawati, Selvi, Riswi Haryatfrehni, Afra Meilianda, Matin Nuhamunada, Listia Pradani, and Bagas Lantip Prakasa. "PIGMENTS CHARACTERIZATION OF MACROALGAE IN DRINI BEACH, GUNUNGKIDUL, YOGYAKARTA FOR SYSTEMATICS STUDY." KnE Life Sciences 2, no. 1 (September 20, 2015): 294. http://dx.doi.org/10.18502/kls.v2i1.161.

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<p>Drini Beach is one of beaches in Gunungkidul, D.I. Yogyakarta with dead-coral reefs as the landform. Dead-coral reef is a good habitat for macroalgae. Based on the pigment content, the macroalgae are divided into Chlorophyta, Phaeophyta, and Rhodophyta with their specific pigments chlorophyta, carotenoid and phycobiliprotein, respectively. These pigments are potentially used as a marker for identification and classification. The research aimed to identify and quantify pigments content of macroalgae in Drini Beach, Gunungkidul, DIY. Samples collection was done in June, 2013. Pigment extractions were done for polar and nonpolar pigments and then the extracts were analyzed using spectrophotometry UV-VIS Shimadzu Genesys <br />10. The result showed that nonpolar extraction of Chaetomorpha had a maximum absorption peak between 320-380, 400-500, and 670 nm ; Borgesenia has maximum peak of 340, 440, and 670 nm; Cladophora in wavelenght of 340, 415, 435, 470, and 665 nm ; and Dictyosphaeria in wavelenght between 330-370, 415, 435, 470, and 665 nm of macroalgae samples; Padina in wavelenght of 340, 380, 440, 665 ; Gelidiella between 305-345 nm ; Gracilaria in wavelenght 330 nm ; Laurencia in wavelength between 310-340, 500, 550, 625, and 680 nm ; and Amphiroa in wavelenght 330 dan 400 nm. Besides, polar extraction using distilled water of Rhodhophyta showed maximum absorption peak of genus Gelidiella between 330-340, 410, and 665 nm; Gracilaria between 330-345, 410, 565, 665 nm ; Laurencia between 330-345, 435, and 665 nm ; and Amphiroa in wavelenght 335, 415, and 665 nm. Based on the results, it could be concluded that Chlorophyta has chlorophyll a, b and carotenoid; Phaeophyta has chlorophyll a, b, and, c; and Rhodophyta has chlorophyll a, carotenoid, and phycoeritrine.</p><p><br /><strong>Keywords</strong> :, macroalgae, Gunung Kidul, pigment, spectrophotometry</p>
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Wang, Zhengyang, Minmin Lin, Lulu He, Hongyan Qi, Jing Shen, and Kejing Ying. "Exosomal lncRNA SCIRT/miR-665 Transferring Promotes Lung Cancer Cell Metastasis through the Inhibition of HEYL." Journal of Oncology 2021 (July 24, 2021): 1–13. http://dx.doi.org/10.1155/2021/9813773.

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Lung cancer remains the leading cause of cancer-related death worldwide. Recently, extracellular vesicles such as exosomes have attracted considerable interest both as a source for theranostic biomarkers and an essential participant in lung cancer progression. However, how specific exosomal cargos, such as noncoding RNAs, are selectively packaged into exosomes and promote lung cancer progression remains unclear. In this study, we identified miR-665 as the most elevated exosomal miRNA from both non-small-cell lung cancer (NSCLC) and small-cell lung cancer (SCLC) patients. We further demonstrated that lncRNA SCIRT was also increased in cancer cell exosomes and may facilitate the exosomal loading of miR-665 with the help of hnRNPA1. As a consequence, exosomal miR-665 promoted lung cancer cell invasion and migration by targeting Notch downstream transcription factor HEYL. In addition, we found that miR-665 and SCIRT were significantly upregulated in tumor tissue and plasma of patients with lung cancer, and both of them showed increased expression in metastatic disease samples. Our findings suggest that the exosomal transferring of miR-665 and SCIRT is a functional and mechanism-driven pathway that contributes to cancer progression and, thus, may provide novel diagnostic and therapeutic targets for lung cancer.
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24

Li, Jia, Gang Zhang, and Guoqiang Wu. "Effect of paeonol on proliferation, apoptosis, migration, invasion and glutamine of gastric cancer cells via circSFMBT2/miR-665 axis." Cellular and Molecular Biology 66, no. 8 (December 31, 2020): 33–40. http://dx.doi.org/10.14715/cmb/2020.66.8.6.

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This experiment was performed to investigate the effect of paeonol on the proliferation, apoptosis, migration, invasion and glutamine of gastric cancer HGC-27 cells and its possible mechanism. For this purpose, the MTT method was used to detect cell viability; Flow cytometry experiment was used to detect cell apoptosis; Transwell chamber experiment was used to detect cell migration and invasion; Western blotting was used to detect the expression levels of MMP2 and MMP9 protein; The decomposition of glutamine was evaluated by detecting the expression levels of glutamine, glutamic acid and α-ketoglutarate (α-KG). This study used RT-PCR to detect the expression of circSFMBT2 and miR-665. The targeting relationship between circSFMBT2 and miR-665 was verified by the dual-luciferase report experiment and RIP experiment. Results showed that different concentrations of Paeonol could significantly inhibit the proliferation, migration, invasion and glutamine decomposition of HGC-27 cells, and induce cell apoptosis in a dose-dependent manner. In gastric cancer tissues and cells, the expression of circSFMBT2 was up-regulated, and the expression of miR-665 was down-regulated. Over-expression of circSFMBT2 could partially restore the effects of paeonol on the proliferation, apoptosis, migration, invasion and glutamine of HGC-27 cells. CircSFMBT2 could target and negatively regulate the expression of miR-665. Overexpression of miR-665 could partially restore the effects of Pae and circSFMBT2 on the proliferation, apoptosis, migration, invasion and glutamine of HGC-27 cells. It was concluded that paeonol can inhibit the proliferation, migration, invasion and glutamine decomposition of gastric cancer HGC-27 cells via circSFMBT2/miR-665 axis, and also induce cell apoptosis.
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MARTINS, F., G. HORTA, M. CARNEIRO, T. FARIA, D. PEREIRA, C. GOMES, and M. CARVALHO. "665: Accidental Overdose of Epidural Sufentanil." Regional Anesthesia and Pain Medicine 33, no. 5 (September 2008): e61-e61. http://dx.doi.org/10.1016/j.rapm.2008.07.124.

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WEBER, E. "665 Cefazolin specific side chain hypersensitivity." Journal of Allergy and Clinical Immunology 97, no. 1 (January 1996): 349. http://dx.doi.org/10.1016/s0091-6749(96)80883-9.

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Martins, F., G. Horta, M. Carneiro, T. Faria, D. Pereira, C. Gomes, and M. Carvalho. "665. Accidental Overdose of Epidural Sufentanil." Regional Anesthesia & Pain Medicine 33, Suppl 1 (September 2008): e61.2-e61. http://dx.doi.org/10.1136/rapm-00115550-200809001-00113.

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YOKOYAMA, Takuya, and Katsuhiko YAMADA. "665 Relative Position Control of Spacecraft." Proceedings of Conference of Tokai Branch 2008.57 (2008): 451–52. http://dx.doi.org/10.1299/jsmetokai.2008.57.451.

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Azimi, E., V. B. Reddy, S. B. Elmariah, T. Luo, and E. A. Lerner. "665 Pruritogen activation of human MRGPRX2." Journal of Investigative Dermatology 139, no. 5 (May 2019): S114. http://dx.doi.org/10.1016/j.jid.2019.03.741.

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Azimi, E., V. Reddy, and E. A. Lerner. "665 Mas-related GPCRs and itch." Journal of Investigative Dermatology 137, no. 5 (May 2017): S114. http://dx.doi.org/10.1016/j.jid.2017.02.687.

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Zholobak, Galina, Oksana Sybirtseva, Mariana Vakolyuk, and Yuliia Zakharchyk. "Remote monitoring of the state of winter wheat during the spring-summer vegetation of 2016 year, by using vegetation indices of Sentinel-2A satellite (case study by foreststeppe area of Ukraine)." Ukrainian journal of remote sensing, no. 15 (February 11, 2018): 23–30. http://dx.doi.org/10.36023/ujrs.2017.15.115.

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The spectral vegetation indices NDVI (842, 665), NDVI (740, 665) and GreenNDVI received from the survey data of new generation satellite Sentinel2A, were analyzed in publication for studying the vegetation of two cultivars of winter wheat, grown up of crops production for the harvest in 2016 year of Grain Alliance Ukraine (Berezan, Kyiv oblast, Ukraine).
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32

Parks, Taryn. "http://www.jaoa.org/content/114/8/665.full." Journal of the American Osteopathic Association 114, no. 8 (August 2014): 666–67. http://dx.doi.org/10.7556/jaoa.2014.134.

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Dasadia, S., M. Sun, C. Sarazin, A. Morandi, M. Markevitch, D. Wik, L. Feretti, G. Giovannini, F. Govoni, and V. Vacca. "A STRONG MERGER SHOCK IN ABELL 665." Astrophysical Journal 820, no. 1 (March 17, 2016): L20. http://dx.doi.org/10.3847/2041-8205/820/1/l20.

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34

Olsson, H. O. M., and S. Jahnson. "665 SUB STAGING OF T1 BLADDER TUMOURS." European Urology Supplements 8, no. 4 (March 2009): 287. http://dx.doi.org/10.1016/s1569-9056(09)60660-6.

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35

Hughes, John P., and Yasuo Tanaka. "The X-ray spectrum of Abell 665." Astrophysical Journal 398 (October 1992): 62. http://dx.doi.org/10.1086/171835.

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Carcso, M. D., Y. Dror, S. Butchart, W. Vanek, V. Blanchette, and M. H. Freedman. "#665 Familial thrombocytopenia with hypoplastic bone marrow." Journal of Pediatric Hematology/Oncology 20, no. 4 (July 1998): 397. http://dx.doi.org/10.1097/00043426-199807000-00102.

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 . "665 Resultaat Cao Gehandicaptenzorg Weer Van Tafel." Zorg en Financiering 8, no. 5 (May 2009): 66. http://dx.doi.org/10.1007/bf03098334.

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38

Ball, George E. "Das Amara-Subgenus Xenocelia subg. n. (Coleoptera: Carabidae)." Coleopterists Bulletin 57, no. 4 (December 2003): 494. http://dx.doi.org/10.1649/665.

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39

Rohde, Randi, Barbara Rudolph, Kristina Ruthe, Francisco G. Lorea-Hernández, Pedro Luis Rodrigues de Moraes, Jie Li, and Jens G. Rohwer. "Neither Phoebe nor Cinnamomum – the tetrasporangiate species of Aiouea (Lauraceae)." Taxon 66, no. 5 (October 24, 2017): 1085–111. http://dx.doi.org/10.12705/665.

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Han, Z. S., E. T. Zhang, and A. D. Craig. "Nociceptive and thermoreceptive lamina I neurons are anatomically distinct." Nature Neuroscience 1, no. 3 (July 1998): 218–25. http://dx.doi.org/10.1038/665.

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Lungo, Mario. "Planificación urbana y economía en las ciudades centroamericanas." Realidad: Revista de Ciencias Sociales y Humanidades, no. 60 (December 29, 1997): 651–65. http://dx.doi.org/10.5377/realidad.v0i60.4989.

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Alfaro, Salvador Orlando. "Gramsci y la sociología del conocimiento: un análisis de la concepción del mundo de las clases subalternas." Realidad: Revista de Ciencias Sociales y Humanidades, no. 83 (October 29, 2001): 651–65. http://dx.doi.org/10.5377/realidad.v0i83.4670.

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Molina, Mercedes. "Sistema educativo y educación en derechos humanos: consideraciones desde la sociología de la educación en torno del caso argentino." Realidad: Revista de Ciencias Sociales y Humanidades, no. 95 (October 28, 2003): 647–65. http://dx.doi.org/10.5377/realidad.v0i95.4223.

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González, Julián. "Juan Antonio Estrada, Imágenes de Dios." Realidad: Revista de Ciencias Sociales y Humanidades, no. 110 (December 13, 2017): 665–67. http://dx.doi.org/10.5377/realidad.v0i110.3437.

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Miller, Katherine. "Oscurantismo iluminado: lectura para una polémica rediviva." Realidad: Revista de Ciencias Sociales y Humanidades, no. 114 (October 20, 2017): 659–65. http://dx.doi.org/10.5377/realidad.v0i114.3988.

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Ginocchio, David González. "Juan Fernando Sellés: Los hábitos intelectuales según Tomás de Aquino, Pamplona: Eunsa 2008, 665 pp." Tópicos, Revista de Filosofía 35, no. 1 (November 28, 2013): 157. http://dx.doi.org/10.21555/top.v35i1.143.

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Editor, El. "Discurso de la toma de posesión del presidente de la República, Elías Antonio Saca." ECA: Estudios Centroamericanos 59, no. 668 (June 30, 2004): 659–65. http://dx.doi.org/10.51378/eca.v59i668.5373.

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Bozinovska, L., S. Mancevska, J. Gligorovska, E. Sivevska, and S. Bozinovski. "665 EXG parameters in children with emotional immaturity." International Journal of Psychophysiology 30, no. 1-2 (September 1998): 252. http://dx.doi.org/10.1016/s0167-8760(98)90664-x.

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Oegerle, William R., Michael J. Fitchett, John M. Hill, and Paul Hintzen. "Dynamics of the microwave-decrement cluster Abell 665." Astrophysical Journal 376 (July 1991): 46. http://dx.doi.org/10.1086/170254.

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Knowles, G., S. J. Sweeney, T. E. Sale, and A. R. Adams. "Self-heating effects in red (665 nm) VCSELs." IEE Proceedings - Optoelectronics 148, no. 5 (December 1, 2001): 256–60. http://dx.doi.org/10.1049/ip-opt:20010843.

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