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Thematische Bibliographien / Wound healing Physiology

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Zeitschriftenartikel
Dissertationen
Bücher
Buchteile
Konferenzberichte

Auswahl der wissenschaftlichen Literatur zum Thema „Wound healing Physiology“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 31. Januar 2023

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Zeitschriftenartikel zum Thema "Wound healing Physiology"

1

Strodtbeck, Frances. "Physiology of wound healing." Newborn and Infant Nursing Reviews 1, no. 1 (March 2001): 43–52. http://dx.doi.org/10.1053/nbin.2001.23176.

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Rhee, John S., David Hom, and Timothy Lian. "Wound Healing and Flap Physiology." Otolaryngology–Head and Neck Surgery 143, no. 5 (November 2010): 718. http://dx.doi.org/10.1016/s0194-5998(10)02297-7.

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Silver, I. A. "The physiology of wound healing." Journal of Wound Care 3, no. 2 (March 2, 1994): 106–9. http://dx.doi.org/10.12968/jowc.1994.3.2.106.

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Flanagan, M. "The physiology of wound healing." Journal of Wound Care 9, no. 6 (June 2000): 299–300. http://dx.doi.org/10.12968/jowc.2000.9.6.25994.

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Rhee, John S., David Hom, and Timothy Lian. "Wound Healing and Flap Physiology." Otolaryngology - Head and Neck Surgery 143, no. 5 (November 2010): 718. http://dx.doi.org/10.1016/j.otohns.2010.09.045.

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6

Young, Alistair, and Clare-Ellen McNaught. "The physiology of wound healing." Surgery (Oxford) 29, no. 10 (October 2011): 475–79. http://dx.doi.org/10.1016/j.mpsur.2011.06.011.

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Harper, Daniel, Alistair Young, and Clare-Ellen McNaught. "The physiology of wound healing." Surgery (Oxford) 32, no. 9 (September 2014): 445–50. http://dx.doi.org/10.1016/j.mpsur.2014.06.010.

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Singh, Shailendra, Alistair Young, and Clare-Ellen McNaught. "The physiology of wound healing." Surgery (Oxford) 35, no. 9 (September 2017): 473–77. http://dx.doi.org/10.1016/j.mpsur.2017.06.004.

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9

Hunt, Thomas K. "The physiology of wound healing." Annals of Emergency Medicine 17, no. 12 (December 1988): 1265–73. http://dx.doi.org/10.1016/s0196-0644(88)80351-2.

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10

Norris, Susan O’Brien, Barbara Provo, and Nancy A. Stotts. "Physiology of Wound Healing and Risk Factors that Impede the Healing Process." AACN Advanced Critical Care 1, no. 3 (November 1, 1990): 545–52. http://dx.doi.org/10.4037/15597768-1990-3010.

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In the critically ill patient, wound repair can be impeded by processes inherent to the illness, its treatment, and the critical care environment. This vulnerability to wound complications increases patient morbidity and mortality as well as length of stay, resource consumption, and hospital cost. The physiology of wound healing and factors that impede wound repair are discussed. Those factors commonly seen in critical illness include advanced age, diabetes mellitus, compromised immunocompetence, inadequate perfusion, and oxygenation, infection, malnutrition, obesity, and preoperative illness.

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Dissertationen zum Thema "Wound healing Physiology"

1

Rippon, Mark Geoffrey. "The physiology of wound healing." Thesis, Manchester Metropolitan University, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240980.

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Kesl, Shannon Lynn. "Metabolic Therapy for Age-Dependent Impaired Wound Healing." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6104.

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Chronic wounds represent an under-acknowledged socioeconomic epidemic, affecting 1.8 million new patients per year and costing the US health care system upwards of $25 billion annually. This substantial cost is rapidly growing due to a disproportionate occurrence in the ever-aging population. Key features associated with age-related impairment of wound healing include limited energy and nutrient exchange, unremitting inflammations, increased reactive oxygen species (ROS), and diminished blood flow. Most chronic wound therapies target specific molecular mechanisms; however, there are often mult

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Andreatta-Van, Leyen Sheila. "Experimental approaches for enhancing wound healing and inhibiting tumor growth." Case Western Reserve University School of Graduate Studies / OhioLINK, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=case1061557930.

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Mari, Walid Omran Dr. "Extracellular Microvesicles as a Novel Biomarker for Wound Healing." Wright State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright1495270509788421.

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Menke, Nathan. "A COMPUTATIONAL BIOLOGY APPROACH TO THE ANALYSIS OF COMPLEX PHYSIOLOGY: COAGULATION, FIBRINOLYSIS, AND WOUND HEALING." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/2093.

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The birth of complexity research derives from the logical progression of advancement in the scientific field afforded by reductionist theory. We present in silico models of two complex physiological processes, wound healing and coagulation/fibrinolysis based on two common tools in the study of complex physiology: ordinary differential equations (ODE) and Agent Based Modeling (ABM). The strengths of these two approaches are well-suited in the analysis of clinical paradigms such as wound healing and coagulation. The complex interactions that characterize acute wound healing have stymied the d

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Roach, Necrisha. "The Development of a Novel Multi-dimensional Product for Wound Healing Applications." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/2131.

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A characteristic feature of chronic wounds is a prolonged inflammatory response as well as susceptibility to infection. Studies have shown that during the inflammatory response, there is a significant increase in the levels of neutrophil-derived enzymes. The purpose of this work was to determine whether the anionic macromolecule polystyrene sulfonate (PSS) and five of its salt forms, namely PSS-calcium, PSS-chlorhexidine, PSS-doxycycline, PSS-glutathione and PSS-silver are able to inhibit the activity of three of the enzymes whose levels are elevated in chronic wounds: elastase, cathepsin G an

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Marshall, Nicholas John. "The influence of insulin-like growth factor 1 and its analogues on fibroblasts and dermal wound healing." Title page, table of contents and synopsis only, 1998. http://web4.library.adelaide.edu.au/theses/09MD/09mdm3685.pdf.

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Includes bibliography (leaves 191-219). Examines the levels of insulin-like growth factor and the presence of IGF binding proteins in human wound fluid. Tests the potency of IGF-1 and 2 analogues in in vitro models of fibroblast activity and their effect on healing in normal and diabetic rodent wounds. Shows that IGF-1, IGF-2 and their binding proteins are present in fluid from a partial thickness cutaneous wound; that the binding proteins negatively modulate the activity of insulin-like growth factors in vitro, but that the IGFs do not necessarily show enhanced activity in vivo at the wound s

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Sinno, Hani. "Role of collagen, complement C3, and C5 on cutaneous wound healing: topical formulation, preparation, and «in-vivo» evaluation in experimental rats." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66713.

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The growing rates of problematic wounds in the population and the subsequent increase in morbidity and mortality warrant further understanding of wound healing and the development of therapeutic agents targeted to alleviate these devastating concerns. The complement system is composed of bactericidal and hemolytic proteins that increase capillary leakage and inflammatory cell migration. It allows for an anaphylactic reaction and the recruitment of inflammatory cells. Fibroblast recruitment and subsequent collagen deposition in wounds is responsible for wound healing and is re

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Robertson, James Gray. "Insulin-like growth factors and insulin-like growth factor binding proteins in wounds /." Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phr6509.pdf.

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Horobin, Adele Jayne. "Maggots and wound healing : the effects of Lucilia sericata larval secretions upon interactions between human dermal fibroblasts and extracellular matrix proteins." Thesis, University of Nottingham, 2005. http://eprints.nottingham.ac.uk/11516/.

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The introduction of necrophagous fly larvae (maggots) into chronic wounds for the purpose of inducing healing is an ancient practice that has recently undergone a renaissance in Western medicine. Through clinical observations, maggots are broadly recognised to debride the wound of necrotic tissue, cleanse the wound of infection and promote granulation tissue formation. Despite such recognition, little research at the biological level has been undertaken to identify the mechanisms by which maggots accomplish such feats. The dermal fibroblast is a major cellular component of granulation tissue a

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Bücher zum Thema "Wound healing Physiology"

1

Cutting, Keith F. Wound physiology & moist wound healing. Holsworthy: Medical Communications UK, 2003.

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2

Peter, Altmeyer, ed. Wound healing and skin physiology. New York: Springer-Verlag, 1994.

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3

Altmeyer, Peter, Klaus Hoffmann, Stephan el Gammal, and Jerry Hutchinson, eds. Wound Healing and Skin Physiology. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-77882-7.

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4

Comprehensive wound management. 2nd ed. Thorofare, NJ: SLACK Inc., 2010.

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5

The care of wounds: A guide for nurses. Oxford: Blackwell Scientific Publications, 1994.

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6

Dealey, Carol. The Care of Wounds. New York: John Wiley & Sons, Ltd., 2008.

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7

Percival, Steven L. Microbiology of wounds. Boca Raton, FL: CRC Press, 2010.

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8

A, Bryant Ruth, and International Association for Enterostomal Therapy., eds. Acute and chronic wounds: Nursing management. St. Louis: Mosby Year Book, 1992.

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Heino, Jyrki, and Veli-Matti Ka ha ri. Cell invasion. Georgetown, Tex: Landes Bioscience, 2002.

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Bianca, C. Towards a mathematical theory of complex biological systems. Singapore: World Scientific, 2011.

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Buchteile zum Thema "Wound healing Physiology"

1

Hatz, R. A., R. Niedner, W. Vanscheidt, and W. Westerhof. "Physiology of Wound Healing." In Wound Healing and Wound Management, 1–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79195-6_1.

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Theoret, Christine. "Physiology of Wound Healing." In Equine Wound Management, 1–13. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781118999219.ch1.

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3

Horch, Raymund E., Oliver Bleiziffer, and Ulrich Kneser. "Physiology and Wound Healing." In Plastic and Reconstructive Surgery, 3–10. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-513-0_1.

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4

Gupta, Ankit. "Classification of Wounds and the Physiology of Wound Healing." In Wound Healing Research, 3–53. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-2677-7_1.

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Mayya, Chaithra, Sumit Kharbhanda, Ashadul Haque, and Dhiraj Bhatia. "Mechanisms of Collective Cell Migration in Wound Healing: Physiology and Disease." In Wound Healing Research, 55–74. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-2677-7_2.

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6

Cohen, I. K., and J. H. Haynes. "Fetal Wound Repair." In Wound Healing and Skin Physiology, 27–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-77882-7_3.

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Toia, Francesca, Fernando Rosatti, and Adriana Cordova. "Wound Healing: Physiology and Pathology." In Textbook of Plastic and Reconstructive Surgery, 15–25. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-82335-1_2.

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Wadström, T., and Å. Ljungh. "Pathogenesis of Wound Infections." In Wound Healing and Skin Physiology, 393–411. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-77882-7_38.

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9

Rompel, R., and J. Petres. "Wound Healing in Oncologic Dermatosurgery." In Wound Healing and Skin Physiology, 551–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-77882-7_53.

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10

Abatangelo, G., P. Brun, and R. Cortivo. "Collagen Metabolism and Wound Contraction." In Wound Healing and Skin Physiology, 71–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-77882-7_7.

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Konferenzberichte zum Thema "Wound healing Physiology"

1

Pryse, Kenneth M., Teresa M. Abney, Guy M. Genin, and Elliot L. Elson. "Probing Cytoskeletal Mechanics Using Biochemical Inhibitors." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19451.

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Quantifying the mechanics of the cytoskeletons of living cells is important for understanding several physiologic and pathologic cellular functions, such as wound healing and cellular migration in cancer. Our laboratory develops three-dimensional tissue constructs for assaying cytoskeletal mechanics in controlled conditions. These tissue constructs consist of defined components such as chick embryo fibroblasts and reconstituted rat tail collagen; fibroblasts remodel the collagen extracellular matrix (ECM) and develop a structural environment representative of that which would exist in a natura

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Sharma, Puja, Kevin Sheets, and Amrinder S. Nain. "The Influence of Polymeric Fiber Stiffness and Alignment on Cytoplasmic Bleb Dynamics and Migration of Glioblastoma Multiforme Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80923.

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Cell migration is a tightly regulated phenomenon necessary for regular physiologic processes such as wound healing, immune response, embryonic development, growth, and regeneration [1–3]. Consequences of abnormal migratory behaviors include autoimmune diseases and metastasis during cancer progression [4, 5]. Described as one of the hallmarks of cancer, metastasis is a complex multistep process, and is responsible for 90% of cancer deaths in humans. A better understanding of the process of metastasis is of paramount importance in developing efficient cancer treatment therapies and drugs [6].

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Grabowski, F. E. "RHEOLOGY AND PRIMARY HEMOSTASIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643986.

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Overview The adhesion-aggregation of platelets to a site of vessel wall injury is a quintessential blood flow phenomenon. Firstly, platelets are driven to the vicinity of the vessel wall by a form of convective diffusion in which red cells both mechanically augment the effective platelet diffusivity (Turitto et al., Ind. Eng. Chem. Fund. 11:216-223, 1972; Grabowski et al., Ind. Eng. Chem. Fund. 11:224-232, 1972) and enhance the near-wall piatelet concentration (Ti11es and Eckstein, Microvasc Res., In press, 1987). Secondly, red cells subjected to physiologic shear forces are capable of secreti

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