Zeitschriftenartikel: „Ulcerative syndrome“

1

Nand, N., M. K. Ward und A. R. Morley. „Nephrotic Syndrome in Ulcerative Colitis“. Nephrology Dialysis Transplantation 6, Nr. 3 (01.01.1991): 227. http://dx.doi.org/10.1093/ndt/6.3.227.

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2

Anti, M., G. Marra, G. L. Rapaccini und G. Fedeli. „Ulcerative esophagitis in Behcet's syndrome“. Gastrointestinal Endoscopy 31, Nr. 4 (August 1985): 289. http://dx.doi.org/10.1016/s0016-5107(85)72190-6.

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3

Kudo, Takahiro, Yoh Aoyagi, Tohru Fujii, Yoshikazu Ohtsuka, Satoru Nagata und Toshiaki Shimizu. „Ulcerative colitis and aortitis syndrome“. Pediatrics International 52, Nr. 1 (Februar 2010): e43-e45. http://dx.doi.org/10.1111/j.1442-200x.2009.02985.x.

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4

Hyodo, Hiromi, Yuichiro Tomita, Kohta Hirai, Hitoshi Hirakawa, Shigeru Ueno und Hiroyuki Ishiguro. „Turner Syndrome with Ulcerative Colitis“. Clinical Pediatric Endocrinology 18, Nr. 4 (2009): 101–5. http://dx.doi.org/10.1297/cpe.18.101.

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5

Resende, Ludimila O., Alexandre J. Dwan und Marcello MS Nico. „Trigeminal trophic syndrome: an important simulator of discoid cutaneous lupus erythematosus – a case series“. Lupus 29, Nr. 10 (26.06.2020): 1282–86. http://dx.doi.org/10.1177/0961203320935169.

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Trigeminal trophic syndrome occurs secondary to trigeminal nerve injury, leading to anaesthesia and paraesthesia, with consequent vigorous facial skin manipulation and lesion production, simulating other facial diseases such as ulcerative discoid lupus erythematosus, tumours and other artificially produced lesions. Ulceration and destruction of the ala nasi is a typical feature besides scratching end excoriations in the cutaneous segment affected. In this series, we present the features of five patients with trigeminal trophic syndrome, highlighting possible confusion with cutaneous lupus. Differential diagnoses, including discoid lupus erythematosus, are discussed, as well as possible treatment modalities.

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6

Turnbull, A. J., A. Law und T. J. Peters. „Ulcerative Colitis Associated with Kallmann's Syndrome“. Journal of the Royal Society of Medicine 81, Nr. 6 (Juni 1988): 354–55. http://dx.doi.org/10.1177/014107688808100619.

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7

Mantaka, Aikaterini N., Dimitrios N. Samonakis, Michail Liontiris und Ioannis E. Koutroubakis. „Ulcerative colitis and Budd–Chiari syndrome“. European Journal of Gastroenterology & Hepatology 26, Nr. 11 (November 2014): 1306. http://dx.doi.org/10.1097/meg.0000000000000183.

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8

Liu, Zhengru, Ke Zhou, Shan Tian und Weiguo Dong. „Ulcerative colitis with Guillain–Barré syndrome“. Medicine 97, Nr. 25 (Juni 2018): e11013. http://dx.doi.org/10.1097/md.0000000000011013.

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9

Takaya, Junji, Masayuki Teraguchi, Yumiko Ikemoto, Ken Yoshimura, Fumiko Yamato, Hirohiko Higashino, Yohnosuke Kobayashi und Kazunari Kaneko. „Turner Syndrome Associated with Ulcerative Colitis“. Clinical Pediatric Endocrinology 15, Nr. 3 (2006): 97–100. http://dx.doi.org/10.1297/cpe.15.97.

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10

Malheiros, Anna Paula Rocha, Magaly Gemio Teixeira, Maria Denise Fonseca Takahashi, Maristela Gomes de Almeida, Desidério Roberto Kiss und Ivan Cecconello. „Sweet syndrome associated with ulcerative colitis“. Inflammatory Bowel Diseases 13, Nr. 12 (Dezember 2007): 1583–84. http://dx.doi.org/10.1002/ibd.20227.

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11

Eshraghi, Hamoon, Aria Mahtabfar und Mohammad H. Dastjerdi. „A Case of Peripheral Ulcerative Keratitis Associated with Autoimmune Hepatitis“. Case Reports in Medicine 2017 (2017): 1–3. http://dx.doi.org/10.1155/2017/3939413.

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Purpose. To describe a case of peripheral ulcerative keratitis in the setting of autoimmune hepatitis and possible overlap syndrome with primary sclerosing cholangitis. Case Report. A 48-year-old African American female with autoimmune hepatitis with possible overlap syndrome with primary sclerosing cholangitis presented with tearing, irritation, and injection of the left eye that was determined to be peripheral ulcerative keratitis. The patient was treated with topical and systemic steroids, immunosuppressant drugs (azathioprine and mycophenolate mofetil), a biologic (rituximab), and surgery (conjunctival resection), and the peripheral ulcerative keratitis epithelialized but ultimately led to corneal perforation. Conclusion. In this unique case, a patient with peripheral ulcerative keratitis who underwent treatment ultimately had a corneal perforation. This case may suggest a possible relationship between autoimmune hepatitis and peripheral ulcerative keratitis.

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12

Suvorov, A. P., V. F. Orkin, A. I. Zavyalov, A. L. Bakulev, E. V. Rumyantseva und G. Kh Hlybochko. „About Behcet's syndrome“. Kazan medical journal 79, Nr. 3 (15.05.1998): 214–15. http://dx.doi.org/10.17816/kazmj64066.

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More than half a century ago, the Turkish dermatologist Huluzi Behcet first described a kind of symptom complex, consisting of a combination of ulcerative lesions of the oral cavity, genitals and the choroid. Further study of the disease showed that its clinical manifestations are much more diverse.

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13

Ali, Massud, und Donald R. Duerksen. „Ulcerative Colitis and Sweet’s Syndrome: A Case Report and Review of the Literature“. Canadian Journal of Gastroenterology 22, Nr. 3 (2008): 296–98. http://dx.doi.org/10.1155/2008/960585.

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A 47-year-old man with a history of ulcerative colitis on prednisone and azathioprine was admitted to the hospital with a four-day history of fever, skin rash, arthralgias and leukocytosis. A skin biopsy demonstrated neutrophilic infiltration of the dermis that was consistent with Sweet’s syndrome. He improved after several days with an increase in his prednisone and azathioprine. Sweet’s syndrome is a rare cutaneous manifestation of inflammatory bowel disease, with approximately 40 cases reported in the literature. In a previously reported case of a patient with ulcerative colitis-associated Sweet’s syndrome who was on azathioprine at the time of the skin eruption, the azathioprine was stopped, raising the possibility of drug-induced Sweet’s syndrome. In the present case, the azathioprine was actually increased with complete resolution of the skin manifestations. This would support the theory that immunosuppressive therapy is the mainstay of therapy for this condition. In conclusion, Sweet’s syndrome is a neutrophilic dermatosis that is rarely associated with ulcerative colitis. It may occur while on immunosuppressive therapy and responds to an intensification of immunosuppression.

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14

Chinabut, Supranee. „Epizootic Ulcerative Syndrome: Information up to 1997.“ Fish Pathology 33, Nr. 4 (1998): 321–26. http://dx.doi.org/10.3147/jsfp.33.321.

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15

TSUBOI, Hiromi, und Kensei KATSUOKA. „Ulcerative lichen planus associated with Sjögren's syndrome“. Journal of Dermatology 34, Nr. 2 (16.01.2007): 131–34. http://dx.doi.org/10.1111/j.1346-8138.2006.00232.x.

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16

Federman, Daniel G., Jeffrey D. Kravetz, Christopher B. Ruser, Peter H. Judson und Robert S. Kirsner. „Vogt-Koyanagi-Harada Syndrome and Ulcerative Colitis“. Southern Medical Journal 97, Nr. 2 (Februar 2004): 169–71. http://dx.doi.org/10.1097/01.smj.0000100263.99708.10.

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17

Chesner, I. M., S. Muller und J. Newman. „Ulcerative colitis complicated by Budd-Chiari syndrome.“ Gut 27, Nr. 9 (01.09.1986): 1096–100. http://dx.doi.org/10.1136/gut.27.9.1096.

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18

Stewart, C. M., und I. Bhattacharyya. „Chronic ulcerative stomatitis in primary Sjögren’s syndrome“. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology 102, Nr. 3 (September 2006): 335–36. http://dx.doi.org/10.1016/j.tripleo.2006.06.036.

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19

Peltonen, S., und M. Kankuri-Tammilehto. „Brooke-Spiegler Syndrome Associated with Ulcerative Rectosigmoiditis“. Acta Dermato Venereologica 93, Nr. 1 (2013): 112–13. http://dx.doi.org/10.2340/00015555-1391.

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20

Yılmaz, Bülent, Seyfettin Köklü und Yusuf Bayraktar. „Ulcerative colitis presenting with Budd–Chiari syndrome“. Journal of Crohn's and Colitis 7, Nr. 2 (März 2013): e74-e75. http://dx.doi.org/10.1016/j.crohns.2012.07.005.

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21

Kerimova, S. Sh. „ULCERATIVE COLITIS AND INTRAHEPATIC CHOLESTASIS“. Hepatology and Gastroenterology 7, Nr. 2 (Dezember 2023): 146–50. http://dx.doi.org/10.25298/2616-5546-2023-7-2-146-150.

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Background. Ulcerative colitis (UC) is an inflammatory bowel disease with unpredicted clinical course and outcomes. Histology is an important part of UC diagnosis and plays an increasing role in management of patients, since more and more histological signs with prognostic value are being identified and confirmed. Objective. To describe a clinical case of death of a UC patient with intrahepatic cholestasis. Material and methods. The article presents autopsy and pathology findings in UC. Results. A patient admitted to the hospital for unspecified acute hepatitis, nonspecific ulcerative colitis and gastrointestinal bleeding died because of ulcerative colitis complicated by sclerosing cholangitis. The course of ulcerative colitis was complicated by bleeding from ulcerated areas; by intrahepatic and extrahepatic cholestasis as well as necrosis of hepatocytes associated with sclerosing cholangitis; by the development of hepatorenal syndrome and sepsis, that resulted in fatal outcome. Conclusions. Multiple severe disturbances occurring in UC pathogenesis result in mutual aggravation syndrome involving interaction of pathophysiological processes with subsequent patient's condition deterioration as well as increased risk of complications and death.

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22

Casellas, F., M. Papo, F. Guarner, M. Antolín, J. R. Armengol und J. R. Malagelada. „Intraluminal Colonic Release of Immunoreactive Tumour Necrosis Factor in Chronic Ulcerative Colitis“. Clinical Science 87, Nr. 4 (01.10.1994): 453–58. http://dx.doi.org/10.1042/cs0870453.

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1. Tumour necrosis factor is a proinflammatory macrophage-derived polypeptide cytokine. Its participation in disease processes has been usually inferred from data obtained from experiments in vitro or from measurements of its plasma circulating levels. To investigate its role in chronic ulcerative colitis, we have quantified in vivo the steady-state release of tumour necrosis factor into the colonic lumen. 2. We studied 19 patients with untreated active ulcerative colitis and seven patients with irritable bowel syndrome as controls. A group of seven patients with active ulcerative colitis were studied before and after 4 weeks on treatment with oral 5-aminosalicylic acid. By means of an intracolonic double-lumen perfusion tube, an isotonic solution was continuously infused 50 cm from the anal verge at a rate of 5 ml/min, and was recovered 30 cm distally by siphonage. Effluents were assayed for tumour necrosis factor by a specific e.l.i.s.a. and for prostaglandin E2 and leukotriene B4 by specific r.i.a.s. 3. The intracolonic release of tumour necrosis factor was undetectable in patients with irritable bowel syndrome, whereas measurable release occurred in 15 out of 19 patients with active ulcerative colitis (P < 0.01). Prostaglandin E2 and leukotriene B4 release were also increased in active ulcerative colitis by comparison with irritable bowel syndrome (P < 0.01). Five out of seven patients with colitis improved with 5-aminosalicylic acid treatment, and tumour necrosis factor release became undetectable or decreased markedly (P < 0.05 compared with before treatment). However, tumour necrosis factor release remained high in the non-responder patients. 4. These findings indicate that intracolonic immunoreactive tumour necrosis factor release is enhanced in active chronic ulcerative colitis, becoming undetectable when mucosal lesions are healed. These results suggest that the luminal release of tumour necrosis factor may serve as an objective index of inflammatory activity in patients with chronic ulcerative colitis.

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23

Xu, Hong-Hui, Ting Xiao, Xing-Hua Gao und Hong-Duo Chen. „Ulcerative Sweet syndrome accompanied by interstitial lung disease and myelodysplastic syndrome“. European Journal of Dermatology 19, Nr. 4 (Juli 2009): 411–12. http://dx.doi.org/10.1684/ejd.2009.0709.

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24

Bruce-Hickman, Damian, Shanaz Matthew Sajeed, Yin Huei Pang, Choon Sheong Seow, Weihao Chen und Monika Gulati Kansal. „Bowel ulceration following tocilizumab administration in a COVID-19 patient“. BMJ Open Gastroenterology 7, Nr. 1 (August 2020): e000484. http://dx.doi.org/10.1136/bmjgast-2020-000484.

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Tocilizumab, a monoclonal antibody against interleukin-6, has been used to treat cytokine release syndrome (CRS) in a subset of patients with severe COVID-19 disease. Acute ulcerative bowel disease has been only rarely documented in patients treated for rheumatological conditions. The gastrointestinal side effects seen when used in the context of COVID-19 are unknown. We present a case of COVID-19 CRS in which acute terminal ileum and perforated caecal ulceration evolved after tocilizumab exposure. We raise awareness of a possible causal relationship between even a single dose of tocilizumab and gut ulceration in patients with COVID-19. Any such drug enteropathy relationship requires watchful monitoring during upcoming trials of tocilizumab in patients with COVID-19.

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25

Karásková, Eva, Mária Véghová-Velgáňová, Miloš Geryk, Vratislav Smolka, Kamila Michálková und Dagmar Pospíšilová. „Posterior Reversible Encephalopathy Syndrome in a child with ulcerative colitis“. Gastroenterologie a hepatologie 75, Nr. 1 (24.02.2021): 46–50. http://dx.doi.org/10.48095/ccgh202146.

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Posterior Reversible Encephalopathy Syndrome (PRES) is a clinical-neuroradiological entity with acute onset, defined as a vasogenic brain edema associated with neurotoxic impairment. We report the case of an eight-year-old boy with severe ulcerative colitis and celiac disease, who was treated with infliximab and azathioprine. On the 10th day after infliximab administration, a 45-minute unconsciousness developed with generalized convulsions and pupil miosis, accompanied by arterial hypertension. The boy was treated with anticonvulsants and antihypertensives. The state of consciousness improved ad integrum. Magnetic resonance imaging (MRI) of the brain revealed symmetric areas of high-intensity signal involving occipital and fronto-parietal lobes. Lumbar puncture was negative. Repeat MRI, seven days from the onset, showed resolution of the previous changes. The condition was assessed as PRES that developed as a complication of treatment (infliximab, azathioprine) in a patient with active ulcerative colitis. The case indicates a possible risk of developing PRES in patients with uncompensated inflammatory bowel disease, receiving immunosuppressive and/or biological treatment and/or developing arterial hypertension

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26

Revajova, V., M. Levkut, M. Kozak, J. Bilek, D. Magic und R. Herich. „Canine histiocytic syndrome manifested as ulcerative gastroenterocolitis, skin lesions and lymphadenopathy – a case report“. Veterinární Medicína 49, No. 8 (29.03.2012): 312–16. http://dx.doi.org/10.17221/5710-vetmed.

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Histiocytic syndrome was diagnosed in a 7-year-old boxer bitch using histological and immunohistochemical methods. Necropsy confirmed the presence of enlarged superficial lymph nodes, two large ulcerated oval cutaneous masses one on the left lateral thoracic wall and one near to the vulva. In the gastrointestinal tract there were multiple ulcers apparently overlying nodules located in the submucosa of stomach, ulcers in the ileocaecal valve, and enlargement of lymphoid nodules in the small intestine manifested ulcerative gastroenteritis. Histologically, the thoracic wall mass showed infiltration of the tissue by macrophages with cytoplasmic vacuoles. The vacuoles contained PAS-positive polysaccharides. The macrophages were positive for alpha-1-antitrypsin and lysozyme by immunostaining. Lysozyme is a marker for phagocytic macrophages/histiocytes and may be used to confirm cells of this lineage in cases when there is any doubt.

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27

GÜNDOĞDU, Barış, Demet ÇİÇEK und Süleyman Serdar KOCA. „Ulcerative Type Pyoderma Gangrenosum in Primary Sjogren's Syndrome“. Turkiye Klinikleri Journal of Case Reports 26, Nr. 4 (2018): 223–26. http://dx.doi.org/10.5336/caserep.2018-60781.

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28

Fornaciari, Giovanni, Sergio Maccari, Pier Paolo Borgatti, Roberto Rustichelli, Nicola Amelio, Ivana Lattuada und Angelo Cesare Plancher. „Nephrotic Syndrome from 5-ASA for Ulcerative Colitis?“ Journal of Clinical Gastroenterology 24, Nr. 1 (Januar 1997): 37–39. http://dx.doi.org/10.1097/00004836-199701000-00008.

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29

Toshniwal, Jay, Romesh Chawlani, Mukesh Bang und Sachin Kale. „Infectious ulcerative colitis resulting in incomplete Reiter’s syndrome“. Endoscopy 47, S 01 (12.06.2015): E229—E230. http://dx.doi.org/10.1055/s-0034-1391654.

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30

Unić, Josipa, Matea Kovačić, Gordana Jakovljević, Ana Tripalo Batoš, Tonći Grmoja und Iva Hojsak. „Lemierre Syndrome in Adolescent with Active Ulcerative Colitis“. Pediatric Gastroenterology, Hepatology & Nutrition 21, Nr. 3 (2018): 214. http://dx.doi.org/10.5223/pghn.2018.21.3.214.

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31

John, K. Riji, und M. Rosalind George. „Viruses Associated with Epizootic Ulcerative Syndrome: An Update“. Indian Journal of Virology 23, Nr. 2 (September 2012): 106–13. http://dx.doi.org/10.1007/s13337-012-0108-x.

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32

Uza, Norimitsu, Hiroshi Nakase, Kazuhiko Nishimura, Shunichi Yoshida, Kenji Kawabata und Tsutomu Chiba. „Solitary rectal ulcer syndrome associated with ulcerative colitis“. Gastrointestinal Endoscopy 63, Nr. 2 (Februar 2006): 355–56. http://dx.doi.org/10.1016/j.gie.2005.09.005.

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33

Ho, Bo-Lin, Fang-Jung Yu, Chiou-Lian Lai und Hsin-Hsin Lin. „Avellis syndrome as presenting manifestation of ulcerative colitis“. Journal of the Neurological Sciences 325, Nr. 1-2 (Februar 2013): 160–61. http://dx.doi.org/10.1016/j.jns.2012.11.015.

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34

HAGA, Hiroshi, Shuichi SHIGETOMI, Ken KATOH, Hiroshi KOHNO, Hideo TOSAKI, Kazuya MORI, Shinji KIN, Atsuo MATSUNAGA, Kiyonobu TANAKA und Soitu FUKUCHI. „A case of Turner's syndrome with ulcerative colitis.“ Nihon Naika Gakkai Zasshi 78, Nr. 10 (1989): 1486–87. http://dx.doi.org/10.2169/naika.78.1486.

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35

Bento-Miranda, Mario, David N. Perdigoto, Sofia Mendes und Francisco Portela. „Sweet’s Syndrome as Manifestation of Active Ulcerative Colitis“. GE - Portuguese Journal of Gastroenterology 27, Nr. 2 (06.08.2019): 138–40. http://dx.doi.org/10.1159/000501399.

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36

Smith, Zachary, Ghouri Yezaz, Jamal A. Ibdah und Veysel Tahan. „1527 Ulcerative Colitis With Concomitant Serrated Polyposis Syndrome“. American Journal of Gastroenterology 114, Nr. 1 (Oktober 2019): S845. http://dx.doi.org/10.14309/01.ajg.0000595636.51034.79.

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37

Natale, M., P. Spennato, A. Bocchetti, M. Fratta, L. Savarese und M. Rotondo. „Ulcerative and mutilating variant of carpal tunnel syndrome“. Acta Neurochirurgica 147, Nr. 8 (11.02.2005): 905–8. http://dx.doi.org/10.1007/s00701-004-0478-y.

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38

Bashir, Anam, Jose M. Cabrera, Mariko Suchi und Barry J. Pelz. „Hypereosinophilic Syndrome with Pulmonary Involvement in Ulcerative Colitis“. JPGN Reports 4, Nr. 3 (09.06.2023): e320. http://dx.doi.org/10.1097/pg9.0000000000000320.

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39

H Mahchouche, F Saoudi und R Lattafi. „Oral ulceration as a revealing sign of granulomatosis with polyangiitis“. International Journal of Scholarly Research in Science and Technology 2, Nr. 2 (30.04.2023): 001–7. http://dx.doi.org/10.56781/ijsrst.2023.2.2.0021.

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Granulomatosis with polyangiitis (GPA), formerly called Wegener's granulomatosis, is a rare systemic disease of unknown etiology that can affect all areas of the body, including the oral cavity. Typical oral manifestations present as nonspecific erosive/ulcerative lesions of the oral mucosa or appear with hyperplastic gingivitis, termed “strawberry gingivitis”. We report here a case of cheek ulceration as the first sign of recurrence of GPA in the absence of oral manifestations in the primary disease. A 37-year-old woman was referred to our oral pathology and surgery department for oral ulcerations. The patient had already been diagnosed with GOODPASTURE syndrome (pneumorenal syndrome) two years previously and treated with cyclophosphamide and Azathioprine. Its relapse and the appearance of oral manifestations made it possible to readjust the diagnosis and the treatment.

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40

Xinmei, Zhang, und Chen Yan. „Nephrotic syndrome and moya-moya syndrome in a patient with ulcerative colitis“. Inflammatory Bowel Diseases 18, Nr. 4 (April 2012): E798—E800. http://dx.doi.org/10.1002/ibd.21850.

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41

Benton, EC, D. Rutherford und JA Hunter. „Sweet´s syndrome and pyoderma gangrenosum associated with ulcerative colitis“. Acta Dermato-Venereologica 65, Nr. 1 (01.01.1985): 77–80. http://dx.doi.org/10.2340/00015555657780.

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A young woman with ulcerative colitis developed pyoderma gangrenosum during the active phase of the disease and Sweet´s syndrome (acute febrile neutrophilic dermatosis) three months after panproctocolectomy.

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42

Van Howe, Robert S. „Sexually Transmitted Infections and Male Circumcision: A Systematic Review and Meta-Analysis“. ISRN Urology 2013 (16.04.2013): 1–42. http://dx.doi.org/10.1155/2013/109846.

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The claim that circumcision reduces the risk of sexually transmitted infections has been repeated so frequently that many believe it is true. A systematic review and meta-analyses were performed on studies of genital discharge syndrome versus genital ulcerative disease, genital discharge syndrome, nonspecific urethritis, gonorrhea, chlamydia, genital ulcerative disease, chancroid, syphilis, herpes simplex virus, human papillomavirus, and contracting a sexually transmitted infection of any type. Chlamydia, gonorrhea, genital herpes, and human papillomavirus are not significantly impacted by circumcision. Syphilis showed mixed results with studies of prevalence suggesting intact men were at great risk and studies of incidence suggesting the opposite. Intact men appear to be of greater risk for genital ulcerative disease while at lower risk for genital discharge syndrome, nonspecific urethritis, genital warts, and the overall risk of any sexually transmitted infection. In studies of general populations, there is no clear or consistent positive impact of circumcision on the risk of individual sexually transmitted infections. Consequently, the prevention of sexually transmitted infections cannot rationally be interpreted as a benefit of circumcision, and any policy of circumcision for the general population to prevent sexually transmitted infections is not supported by the evidence in the medical literature.

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43

Hendrickx, T., M. Peetermans, A. D’Hoore, K. Claes, A. Van Hootegem und J. Sabino. „STEC colitis mimicking acute severe colitis with life-threatening consequences: a case report“. Acta Gastro Enterologica Belgica 87, Nr. 1 (März 2024): 37–39. http://dx.doi.org/10.51821/87.1.11652.

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Acute colitis is a common feature of infection with Shiga-toxin producing Escherichia coli (STEC) and can mimic acute severe ulcerative colitis. Early recognition is important as there is a risk of developing Shiga toxin-induced haemolytic uremic syndrome (STEC-HUS), defined by the triad of microangiopathic haemolytic anemia, thrombocytopenia and organ damage. In severe cases STEC-HUS can cause severe neurological complications and can be fatal. Case: We present a patient with a medical history of refractory ulcerative colitis, where making the diagnosis of STEC-HUS was challenging since the initial clinical presentation was difficult to differentiate from a flare of ulcerative colitis. Conclusion: This case illustrates that STEC induced colitis can mimic acute severe ulcerative colitis. This finding is of utmost clinical importance because of the potential life-threatening complications of STEC-HUS. Therefore it should be excluded promptly in patients with acute severe ulcerative colitis by using multiplex-PCR assay on a faecal sample.

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Lekatompessy, Deasly, Nor Sa’adah und Nirmalasari Idha Wijaya. „Identifikasi Penyakit Pada Terumbu Karang di Pulau Gili Labak, Sumenep, Madura“. Journal of Marine Research 12, Nr. 4 (25.10.2023): 604–12. http://dx.doi.org/10.14710/jmr.v12i4.41474.

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Infeksi penyakit pada karang telah diidentifikasi sebagai salah satu faktor utama yang memperburuk kondisi terumbu karang global. Munculnya penyakit karang dicirikan dengan adanya perubahan warna, kerusakan dari skeleton biota karang, sampai dengan kehilangan jaringannya. Contoh penyakit karang yang tersebar di perairan indo-pasifik adalah black band disease (BBD), White Band Disease (WBD), White Syndromes (WS), Pink Blotch (PB), Ulcerative White Spot (UWS), Red Band Disease (RDB), Yellow Band Disease (YBD). Pulau Gili Labak atau yang lebih dikenal dengan Wisata Gili Labak merupakan salah satu wisata bahari yang terletak di sebuah pulau kecil Madura. Pulau Gili Labak saat ini sudah mulai dikembangkan di Kabupaten Sumenep karena adanya potensi pertumbuhan terumbu karang yang cukup baik. Penelitian ini bertujuan mengidentifikasi penyakit pada terumbu karang, prevalensi penyakit karang, dan life form terumbu karang. Pengambilan data dilakukan dengan metode belt transek di tiga stasiun pengamatan di Pulau Gili Labak. Identifikasi penyakit dilakukan secara deskriptif berdasarkan buku coral dieses handbook dan referensi jurnal. life form yang ditemukan di pulau Gili Labak ada 9 tipe life form. Jenis penyakit terumbu karang yang ditemukan di Pulau Gili Labak ada 4 jenis penyakit yaitu White Band Disease, Ulcerative White Spot, White Syndrome, dan Pink Line Syndrome. Prevalensi penyakit karang di Pulau Gili Labak dengan nilai rata-rata prevalensi sebesar (1,7%). Coral disease infections have been identified as one of the main factors deteriorating the condition of coral reefs globally. The emergence of coral disease is characterized by changes in color, damage to the skeleton of coral biota, and even loss of their tissues. There are examples of coral diseases that are spread in Indo-Pacific waters are black band disease (BBD), White Band Disease (WBD), White Syndromes (WS), Pink Blotch (PB), Ulcerative White Spot (UWS), Red Band Disease (RDB) , Yellow Band Disease (YBD). Gili Labak Island or better known as Gili Labak Tourism is one of marine tourism site located on the small island of Madura. Gili Labak Island is currently starting to be developed in Sumenep Regency because of the potential for quite good coral reef growth. This research aims to identify diseases on coral reefs, the prevalence of coral diseases, and the life form of coral reefs. Data collection was carried out using the belt transect method at three observation stations on Gili Labak Island. Disease identification was carried out descriptively based on the coral disease handbook and journal references. There are 9 types of life forms found on Gili Labak Island. There are 4 types of coral reef diseases found on Gili Labak Island, called White Band Disease, Ulcerative White Spot, White Syndrome, and Pink Line Syndrome. The prevalence of coral disease on Gili Labak Island with an average prevalence (1.7%).

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Iskhakova, D. G., D. O. Subkhangulova, D. D. Safina, L. M. Ibragimova und E. V. Khazova. „Hypercoagulation in patient with ulcerative colitis associated with antiphospholipid syndrome and latent inherited thrombophilia after COVID-19: a clinical case“. Siberian Journal of Clinical and Experimental Medicine 38, Nr. 2 (10.07.2023): 293–304. http://dx.doi.org/10.29001/2073-8552-2023-38-2-293-304.

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Patients with ulcerative colitis exacerbation associated with COVID-19 infection represent a particular group who require additional attention and follow-up due to an increased risk of thrombosis. The article presents a clinical case of a patient born in 1971 with ulcerative colitis in association with antiphospholipid syndrome. Additionally the patient had a latent inherited thrombophilia, which activation was probably caused by exacerbation of ulcerative colitis. Nevertheless, there may have been additional triggering activity of a COVID-19 infection that the patient suffered before the exacerbation of ulcerative colitis. The patient usually had a fever in the morning during an exacerbation of ulcerative colitis. During current hospitalization, fever was also presented. To clarify the cause of the fever patient’s examination was performed. Infectious and inflammatory processes, neoplasms, connective tissue diseases were excluded. However, genetic testing revealed the presence of an increased risk of thrombosis in the patient (ITGA2 C/T, ITGB3 T/C were presented). At the same time, thrombotic masses in the portal vein, subtotal thrombosis of the right hepatic branch of the portal vein, thrombosis of the upper mesenteric vein up to 30%, lower mesenteric vein up to 70% were detected during ultrasound and computer tomography (CT) examination of the abdominal cavity. It should be noted that the management of patients with exacerbation of ulcerative colitis, especially with association with COVID-19 infection, requires an individual approach considering the risk assessment of thrombotic complications. Comprehensive examination is highly recommended, including genetic testing of patients in the clinical management of such patients.

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Sagara, Shiho. „Acute respiratory distress syndrome associated with severe ulcerative colitis“. World Journal of Gastroenterology 16, Nr. 19 (2010): 2440. http://dx.doi.org/10.3748/wjg.v16.i19.2440.

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Bawazeer, Ahmed M., und W. Bruce Jackson. „Marginal Infiltrative Ulcerative Keratitis Secondary to Churg–Strauss Syndrome“. Cornea 19, Nr. 3 (Mai 2000): 402–4. http://dx.doi.org/10.1097/00003226-200005000-00030.

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Sayeed, Sana, Areeba Imtiaz und Faiza Naz. „SAPHO Syndrome in an Adult Male with Ulcerative Colitis“. Journal of the College of Physicians and Surgeons Pakistan 29, Nr. 07 (01.07.2019): 671–73. http://dx.doi.org/10.29271/jcpsp.2019.07.671.

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Praderio, Luisa, Lorenzo Dagna, Paolo Longhi, Gianpaolo Rubin und Maria Grazia Sabbadini. „Budd-Chiari Syndrome in a Patient With Ulcerative Colitis“. Journal of Clinical Gastroenterology 30, Nr. 2 (März 2000): 203–4. http://dx.doi.org/10.1097/00004836-200003000-00016.

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Porcelli, Piero, und Gioacchino Leandro. „Bowel Obsession Syndrome in a Patient With Ulcerative Colitis“. Psychosomatics 48, Nr. 5 (September 2007): 448–50. http://dx.doi.org/10.1176/appi.psy.48.5.448.

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