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Thematische Bibliographien / Ulcerative syndrome

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Zeitschriftenartikel
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Bücher
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Auswahl der wissenschaftlichen Literatur zum Thema „Ulcerative syndrome“

Autor: Grafiati

Veröffentlicht am 18. Mai 2024

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Zeitschriftenartikel zum Thema "Ulcerative syndrome"

1

Nand, N., M. K. Ward und A. R. Morley. „Nephrotic Syndrome in Ulcerative Colitis“. Nephrology Dialysis Transplantation 6, Nr. 3 (01.01.1991): 227. http://dx.doi.org/10.1093/ndt/6.3.227.

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Anti, M., G. Marra, G. L. Rapaccini und G. Fedeli. „Ulcerative esophagitis in Behcet's syndrome“. Gastrointestinal Endoscopy 31, Nr. 4 (August 1985): 289. http://dx.doi.org/10.1016/s0016-5107(85)72190-6.

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Kudo, Takahiro, Yoh Aoyagi, Tohru Fujii, Yoshikazu Ohtsuka, Satoru Nagata und Toshiaki Shimizu. „Ulcerative colitis and aortitis syndrome“. Pediatrics International 52, Nr. 1 (Februar 2010): e43-e45. http://dx.doi.org/10.1111/j.1442-200x.2009.02985.x.

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Hyodo, Hiromi, Yuichiro Tomita, Kohta Hirai, Hitoshi Hirakawa, Shigeru Ueno und Hiroyuki Ishiguro. „Turner Syndrome with Ulcerative Colitis“. Clinical Pediatric Endocrinology 18, Nr. 4 (2009): 101–5. http://dx.doi.org/10.1297/cpe.18.101.

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Resende, Ludimila O., Alexandre J. Dwan und Marcello MS Nico. „Trigeminal trophic syndrome: an important simulator of discoid cutaneous lupus erythematosus – a case series“. Lupus 29, Nr. 10 (26.06.2020): 1282–86. http://dx.doi.org/10.1177/0961203320935169.

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Trigeminal trophic syndrome occurs secondary to trigeminal nerve injury, leading to anaesthesia and paraesthesia, with consequent vigorous facial skin manipulation and lesion production, simulating other facial diseases such as ulcerative discoid lupus erythematosus, tumours and other artificially produced lesions. Ulceration and destruction of the ala nasi is a typical feature besides scratching end excoriations in the cutaneous segment affected. In this series, we present the features of five patients with trigeminal trophic syndrome, highlighting possible confusion with cutaneous lupus. Differential diagnoses, including discoid lupus erythematosus, are discussed, as well as possible treatment modalities.

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Turnbull, A. J., A. Law und T. J. Peters. „Ulcerative Colitis Associated with Kallmann's Syndrome“. Journal of the Royal Society of Medicine 81, Nr. 6 (Juni 1988): 354–55. http://dx.doi.org/10.1177/014107688808100619.

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Mantaka, Aikaterini N., Dimitrios N. Samonakis, Michail Liontiris und Ioannis E. Koutroubakis. „Ulcerative colitis and Budd–Chiari syndrome“. European Journal of Gastroenterology & Hepatology 26, Nr. 11 (November 2014): 1306. http://dx.doi.org/10.1097/meg.0000000000000183.

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Liu, Zhengru, Ke Zhou, Shan Tian und Weiguo Dong. „Ulcerative colitis with Guillain–Barré syndrome“. Medicine 97, Nr. 25 (Juni 2018): e11013. http://dx.doi.org/10.1097/md.0000000000011013.

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Takaya, Junji, Masayuki Teraguchi, Yumiko Ikemoto, Ken Yoshimura, Fumiko Yamato, Hirohiko Higashino, Yohnosuke Kobayashi und Kazunari Kaneko. „Turner Syndrome Associated with Ulcerative Colitis“. Clinical Pediatric Endocrinology 15, Nr. 3 (2006): 97–100. http://dx.doi.org/10.1297/cpe.15.97.

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Malheiros, Anna Paula Rocha, Magaly Gemio Teixeira, Maria Denise Fonseca Takahashi, Maristela Gomes de Almeida, Desidério Roberto Kiss und Ivan Cecconello. „Sweet syndrome associated with ulcerative colitis“. Inflammatory Bowel Diseases 13, Nr. 12 (Dezember 2007): 1583–84. http://dx.doi.org/10.1002/ibd.20227.

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Dissertationen zum Thema "Ulcerative syndrome"

1

John, K. Riji. „Characterization of reovirus-like agents associated with snakehead fish and cell culture“. Thesis, University of Stirling, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244645.

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Kanchanakhan, Somkiat. „Field and laboratory studies of rhabdoviruses associated with epizootic ulcerative syndrome (EUS) of fishes“. Thesis, University of Stirling, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319799.

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Lilley, James H. „Studies on the comparative biology of Aphanomyces invadans“. Thesis, University of Stirling, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244692.

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Vento, Pälvi. „Nitric oxide and neuropeptides in the gut : changes in ulcerative colitis, pouchitis and short bowel syndrome“. Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/biola/vk/vento/.

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Villani, Alexandra-Chloé. „Genetic investigation of inflammatory bowel disease and post-infectious irritable bowel syndrome : the contribution of innate immunity candidate risk variants“. Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115888.

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The gastro-intestinal (GI) tract represents the largest surface of the body and is continuously exposed to the microbial environment. In such anatomy, the survival of the host requires that the intestinal microbial flora be contained without excessive immune-reactivity to commensal bacteria while retaining the ability to respond to episodic pathogens. The discriminative recognition between beneficial commensal bacteria and potentially harmful pathogens demands an accurate interpretation by the GI mucosal immune system. Any defects in the processes of innate immune recognition and killing may lead to the development and perpetuation of chronic intestinal inflammation, namely inflammatory bowel disease (i.e. Crohn's disease (CD) and ulcerative colitis (UC)) and post-infectious irritable bowel syndrome (PI-IBS). The aim of ours studies was to evaluate the contribution of candidate genes, involved in the homeostasis and regulation of the intestinal innate immune response, to the susceptibility to CD, DC, and PI-IBS. In the first phase, we describe functional and genetic association results supporting NLRP3, encoding NALP3/cryopyrin, as a novel CD susceptibility gene. We subsequently report that the MEFV gene, encoding pyrin, known to interact with and be involved in the same pathway as NALP3/cryopyrin, does not contribute to CD and DC susceptibility. No CD or DC additional associations were observed upon NLRP3-MEFV gene-gene interaction analyses. In the third phase, we report the first association study evaluating genetic determinants for PI-IBS, using the well-characterized Walkerton population cohort. We uncovered variants in the TLR9, CDH1, and IL6 regions associated with PI-IBS susceptibility. These results are in keeping with the pathophysiologic changes observed in patients with PI-IBS, which include increased intestinal permeability and intestinal immune activation.
Overall, these results contribute to a better understanding of the genetic susceptibility to CD, DC and PI-IBS and shed light on new pathogenic signaling pathways in the development of these diseases.

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Lio-Po, Gilda. „Studies on several virus isolates, bacteria and a fungus associated with Epizootic Ulcerative Syndrome (EUS) of several fishes in the Philippines“. Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0018/NQ37727.pdf.

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Reinders, Claudia I. „Nitric oxide : a surrogate marker of bowel inflammation /“. Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-491-0/.

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Гуріна, С. В., А. М. Азарєнков und В. В. Віннікова. „Мікробіоценоз трофічних виразок та імунореактивність у людей з синдромом діабетичної стопи“. Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27034.

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Вікторова, Ю. А., und А. О. Ярмолюк. „Суглобовий синдром при хронічних запальних захворюванннях кишечника“. Thesis, Сумський державний університет, 2016. http://essuir.sumdu.edu.ua/handle/123456789/47232.

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Актуальність Неспецифічний виразковий коліт та хвороба Крона – це хронічні запально-некротичні захворювання товстого кишечнику. Вони можуть супроводжуватися суглобовим синдромом з ураженням хребта і периферичних суглобів. Суглобові прояви зустрічаються у 20-40% хворих. Їх відносять до серонегативних спондилоартритів.. Мета Вивчення особливостей патогенезу та клініки суглобового синдрому у хворих на хронічні запальні захворювання кишечника (ХЗЗК).

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Miles, David J. C. „Studies on host responses to Aphanomyces invadans“. Thesis, University of Stirling, 2002. http://hdl.handle.net/1893/63.

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Aphanomyces invadans is the pathogen that causes epizootic ulcerative syndrome (EUS), an economically devastating fish disease in southern Asia. The present thesis considered possible improvements to current methods of monitoring EUS, and examined the mechanisms of the host immune response to A. invadans in order to establish whether they could be enhanced to reduce the impact of EUS on aquaculture. Monoclonal antibody (MAb) technology was considered as a possible improvement to the histopathological methods currently used in diagnosis of EUS. Five MAbs were raised to day-old A. invadans germlings. Four gave weak reactions to A. invadans and cross-reacted with other Aphanomyces spp, though they may be useful for future studies on A. invadans. The other, designated MAb 3gJC9, only cross-reacted with the crayfish plague pathogen, A. astaci, and was used for the development of an immunohistochemistry protocol that may be of use in diagnosis. Immunohistochemistry with MAb 3gJC9, which recognised an extracellular product (ECP) of A. invadans, was specific to A. invadans in fish tissue, although it also recognised A. astaci in plague-infected crayfish. It also recognised the mycelium in fish infected with ulcerative mycosis, indicating that ulcerative mycosis is synonymous with EUS. Preliminary observations indicated that both ECPs and what appeared to be a hitherto unreported early stage of the mycelium are important in the pathology of EUS. Studies in vitro on the macrophages of EUS-susceptible giant gourami Osphronemus gouramy and silver barb Barbodes gonionotus, and EUS-resistant Nile tilapia Oreochromis niloticus, found that their macrophages were able to inhibit the growth of A. invadans. The macrophages of striped snakehead Channa striata did not inhibit A. invadans, which may account for their high EUS-susceptibility, especially as A. invadans strongly inhibited the respiratory burst of snakehead macrophages. Studies on humoral immune responses revealed that complement inhibited A. invadans in the case of snakeheads, gourami and barbs but not tilapia or swamp eels Monopterus albus. The humoral responses of the latter were very different to the four other species, and not elucidated. Low levels of anti A. invadans antibodies were found in tilapia and gourami from an EUS-endemic region, and high levels in snakehead. Snakehead antibodies appeared to be able to inhibit A. invadans even when complement was removed, but lower levels were produced at the low temperatures typically associated with EUS. A range of potential immunostimulants were screened for the ability to enhance resistance to EUS. The two successful products were administered as feed supplements to snakeheads and barbs that were subsequently injected intramuscularly with A. invadans. One, the algal extract Ergosan, showed some beneficial effects on snakeheads although the challenge was inconclusive. The other, the vitamin supplement Salar-bec, accelerated the cellular immune response and reduced mortality in snakeheads and barbs, and enhanced antibody production in snakeheads. The antibody response of snakeheads was further studied by comparing the anti- A. invadans antibody level, inhibitory activity of sera in vitro and protective capacity of sera from EUS-naïve snakeheads to that of snakeheads recently exposed to EUS and those subject to long term EUS-exposure. Sera of populations recently exposed to EUS showed an increased level of antibodies, but little improvement in inhibitory or protective activity. Sera from snakeheads that had endured long term exposure showed a wide range of antibody levels, but marked increases in inhibitory and protective activity. Antibodies cross-reacted with non-pathogenic Aphanomyces spp. in all cases.

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Bücher zum Thema "Ulcerative syndrome"

1

University, Barkatullah, Hrsg. Investigations on the epizoolitic ulcerative syndrome of fishes in the water bodies in and around Bhopal with special references to Halali reservoir: Project completion report. [Bhopal: Barktullah University], 1997.

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Sindermann, Carl J. Epizootic ulcerative syndromes in coastal/estuarine fish. Woods Hole, Mass: U.S. Department of Commerce, National Oceanic and Atmospheric Administration, National Marine Fisheries Service, Northeast Fisheries Center, 1988.

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Epizootic Ulcerative Fish Disease Syndrome. Elsevier, 2016. http://dx.doi.org/10.1016/c2014-0-03152-8.

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Kar, Devashish. Epizootic Ulcerative Fish Disease Syndrome. Elsevier Science & Technology Books, 2015.

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Kar, Devashish. Epizootic Ulcerative Fish Disease Syndrome. Elsevier Science & Technology Books, 2015.

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Publications, Black. IBD Journal: Irritable Bowel Syndrome - Food Diary and Symptoms Tracker for Ulcerative Colitis, Crohn's Disease and Other Digestive Disorders. Independently Published, 2021.

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Monastyrsky, Konstantin. Fiber Menace: The Truth About the Leading Role of Fiber in Diet Failure, Constipation, Hemorrhoids, Irritable Bowel Syndrome, Ulcerative Colitis, Crohn's Disease, and Colon Cancer. Ageless Press, 2005.

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Publication, Asifesisler. Colitis and Crohn's Disease Journal and Planner: Log Your Pain Levels, Meals, Symptoms and More. Use for Irritable Bowel Syndrome , Ulcerative Colitis, Celiac Log Book Diary. Independently Published, 2022.

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Sparks, Orora P. My IBD Food Journal: Food Diary and Tracker for Ulcerative Colitis, Crohns, IBS and Other Digestive Disorders Symptom Management Log Food Sensitivity Journal Book Ibs Awareness Eating for Ibs Food Journal for Intolerance down Syndrome Diary. Independently Published, 2021.

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Jacquet, Gabrielle, und Lawrence Page. Odontogenic Infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0013.

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Odontogenic infections often arise from dental caries (usually the mandibular teeth) or from dental extraction. Acute necrotizing ulcerative gingivitis (ANUG) is more common in immunocompromised patients. These infections may spread into the parapharyngeal and retropharyngeal spaces, involving the airway and mediastinum. Airway management is critical as odontogenic infections can compromise airways via mass effect. Complications include the following: abscess, facial or orbital cellulitis/abscess, intracranial invasion, Ludwig’s angina, Lemierre syndrome, carotid artery erosion, descending necrotizing mediastinitis, airway compromise, hematogenous dissemination to distant organs, intraoral or dentocutaneous fistula formation, and cardiovascular disease. Antibiotics are not a substitute for definitive airway management. In addition, many cases of odontogenic infection will require surgical drainage, either at the bedside in the emergency department or in the operating room. Prior to this, consider using a nerve block to obtain anesthesia to the affected area of the face. Patients with necrotizing infections need emergent surgery with wide local debridement.

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Buchteile zum Thema "Ulcerative syndrome"

1

Kamilya, Dibyendu, und Kollanoor Riji John. „Epizootic ulcerative syndrome (Aphanomyces invadans).“ In Climate change and infectious fish diseases, 291–302. Wallingford: CABI, 2020. http://dx.doi.org/10.1079/9781789243277.0291.

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Ginsburg, Philip M., und Theodore M. Bayless. „Management of Irritable Bowel Syndrome in the Patient with Ulcerative Colitis“. In Medical Therapy of Ulcerative Colitis, 431–37. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1677-1_40.

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Chakravorty, Hitesh, und Prodipto Das. „Image Processing Technique and SVM for Epizootic Ulcerative Syndrome Fish Image Classification“. In Communications in Computer and Information Science, 273–81. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-47221-3_24.

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Kolluri, Raghu. „Antiphospholipid Antibody Syndrome-induced Ulceration“. In Atlas of Clinical Vascular Medicine, 110–11. Oxford, UK: Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118618189.ch55.

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Chong, Roger Sie-Maen. „Epizootic ulcerative syndrome“. In Aquaculture Pathophysiology, 621–27. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-812211-2.00050-0.

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Kar, Devashish. „Pathology of Epizootic Ulcerative Syndrome“. In Epizootic Ulcerative Fish Disease Syndrome, 83–93. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802504-8.00005-5.

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Kar, Devashish. „Monitoring of Epizootic Ulcerative Syndrome“. In Epizootic Ulcerative Fish Disease Syndrome, 247–56. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802504-8.00011-0.

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Kar, Devashish. „Introduction“. In Epizootic Ulcerative Fish Disease Syndrome, 1–19. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802504-8.00001-8.

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Kar, Devashish. „Origin of the Epizootic Ulcerative Syndrome Problem“. In Epizootic Ulcerative Fish Disease Syndrome, 21–39. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802504-8.00002-x.

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Kar, Devashish. „Efforts in Unraveling the Enigmatic Epizootic Ulcerative Syndrome and Review of Current Status on its Research and Development“. In Epizootic Ulcerative Fish Disease Syndrome, 41–61. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802504-8.00003-1.

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Konferenzberichte zum Thema "Ulcerative syndrome"

1

Wichert, PV, und P. Barth. „A Brochocolitic Syndrome – The Lung in Ulcerative Colitis.“ In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2900.

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Smith, SPF, A. Abdulla, Z. Zaidi, P. Deb, N. Croft, S. Naik und A. Kadir. „OC38 Posterior reversible encephalopathy syndrome following immunosuppressive treatment for ulcerative colitis“. In Abstracts of the BSPGHAN Annual Meeting 2023. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/flgastro-2023-bspghan.37.

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Rachman, F., MNS Akbar und E. Putera. „FISH DISEASE DETECTION OF EPIZOOTIC ULCERATIVE SYNDROME USING DEEP LEARNING IMAGE PROCESSING TECHNIQUE“. In International Conference on Fisheries and Aquaculture. The International Institute of Knowledge Management, 2023. http://dx.doi.org/10.17501/23861282.2023.8102.

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Vijayalakshmi, M., A. Sasithradevi und P. Prakash. „Transfer Learning Approach for Epizootic Ulcerative Syndrome and Ichthyophthirius Disease Classification in Fish Species“. In 2023 International Conference on Bio Signals, Images, and Instrumentation (ICBSII). IEEE, 2023. http://dx.doi.org/10.1109/icbsii58188.2023.10181046.

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Rocha de Moura Rodrigues de Aguiar, Débora, Daniela Blanco Mecking, Paulo Phillipe do Valle Ricardo Moreira, Luiz Felipe Dipe Prates Miranda und Luciana de Abreu e Lima Pamplona. „PERIPHERAL ULCERATIVE KERATITIS REFRACTORY IN AN OVERLAP OF SJÖGREN'S SYNDROME AND SARCOIDOSIS: A CASE REPORT“. In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17034.

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Sujatha, Kamepalli, und Pakanati Mounika. „Evaluation of ML Models for Detection and Prediction of Fish Diseases: A Case Study on Epizootic Ulcerative Syndrome“. In 2023 Second International Conference on Electrical, Electronics, Information and Communication Technologies (ICEEICT). IEEE, 2023. http://dx.doi.org/10.1109/iceeict56924.2023.10156914.

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Parker, C. J., D. E. Huber, A. R. Hedges und V. V. Kakkar. „ARE THE SYMPTOMS OF THE POST PHLEBITIC LIMB ALWAYS POST THROMBOTIC“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644200.

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The term ‘post phlebitic syndrome’ implies a previous history of deep venous thrombosis. To test the validity of this assumption, 106 patients who had routine post-operative bilateral ascending venography following total hip replacement were reviewed five years later.Patients were assessed clinically for symptoms and signs of the ‘post phlebitic syndrome’ (pain, swelling, induration, pigmentation, ulceration and varicose veins). Haemodynamic changes were assessed by foot volumetry and an objective score was derived by computer analysisThirty patients (28%) had post-operative DVT. 50% of all patients had at least one symptom or sign of the post phlebitic limb. Pain was present in 9 limbs; swelling in 13; induration in 18; pigmentation in 55, and varicose veins in 53 limbs.At five years there was no significant difference in the incidence of symptoms, signs or haemadynamic changes of the post phlebitic syndrome between limbs with or without a previous DVT. We conclude that deep vein thrombosis is not the only factor involved in the aetiology of the post phlebitic limb.

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Berichte der Organisationen zum Thema "Ulcerative syndrome"

1

Zhang, Liangkun, Jian Chen, Wenchao Gu, Hailiang Huang und Tao Han. Comparing the Efficacy and Safety of Three Xinkai Kujiang Decoctions in Treatment of Ulcerative Colitis with Cold-heat Syndrome: Network Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, Juli 2020. http://dx.doi.org/10.37766/inplasy2020.7.0055.

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