Zeitschriftenartikel zum Thema „Tubular-interstitial fibrosis“
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Wyczanska, Maja, Jana Rohling, Ursula Keller, Marcus R. Benz, Carsten Kirschning und Bärbel Lange-Sperandio. „TLR2 mediates renal apoptosis in neonatal mice subjected experimentally to obstructive nephropathy“. PLOS ONE 18, Nr. 11 (28.11.2023): e0294142. http://dx.doi.org/10.1371/journal.pone.0294142.
Der volle Inhalt der QuelleChristensen, Erik I., und Pierre J. Verroust. „Interstitial fibrosis: tubular hypothesis versus glomerular hypothesis“. Kidney International 74, Nr. 10 (November 2008): 1233–36. http://dx.doi.org/10.1038/ki.2008.421.
Der volle Inhalt der QuelleRascio, Federica, Paola Pontrelli, Giuseppe Stefano Netti, Elisabetta Manno, Barbara Infante, Simona Simone, Giuseppe Castellano et al. „IgE-Mediated Immune Response and Antibody-Mediated Rejection“. Clinical Journal of the American Society of Nephrology 15, Nr. 10 (09.09.2020): 1474–83. http://dx.doi.org/10.2215/cjn.02870320.
Der volle Inhalt der QuelleEskild-Jensen, Anni, Lene Fogt Paulsen, Lise Wogensen, Ping Olesen, Lea Pedersen, Jørgen Frøkiær und Jens Randel Nyengaard. „AT1 receptor blockade prevents interstitial and glomerular apoptosis but not fibrosis in pigs with neonatal induced partial unilateral ureteral obstruction“. American Journal of Physiology-Renal Physiology 292, Nr. 6 (Juni 2007): F1771—F1781. http://dx.doi.org/10.1152/ajprenal.00479.2006.
Der volle Inhalt der QuelleWang, Shi-Nong, und Raimund Hirschberg. „Growth factor ultrafiltration in experimental diabetic nephropathy contributes to interstitial fibrosis“. American Journal of Physiology-Renal Physiology 278, Nr. 4 (01.04.2000): F554—F560. http://dx.doi.org/10.1152/ajprenal.2000.278.4.f554.
Der volle Inhalt der QuelleThomas, S. E., S. Anderson, K. L. Gordon, T. T. Oyama, S. J. Shankland und R. J. Johnson. „Tubulointerstitial disease in aging: evidence for underlying peritubular capillary damage, a potential role for renal ischemia.“ Journal of the American Society of Nephrology 9, Nr. 2 (Februar 1998): 231–42. http://dx.doi.org/10.1681/asn.v92231.
Der volle Inhalt der QuelleLeong, Khai Gene, Elyce Ozols, John Kanellis, David J. Nikolic-Paterson und Frank Y. Ma. „Cyclophilin A Promotes Inflammation in Acute Kidney Injury but Not in Renal Fibrosis“. International Journal of Molecular Sciences 21, Nr. 10 (22.05.2020): 3667. http://dx.doi.org/10.3390/ijms21103667.
Der volle Inhalt der QuelleWang, Shudan, Ming Wu, Luis Chiriboga, Chaim Putterman, Anna Broder und H. Michael Belmont. „4336 Renal Tubular Complement C9 Deposition is Associated with Renal Tubular Damage and Fibrosis in Lupus Nephritis“. Journal of Clinical and Translational Science 4, s1 (Juni 2020): 144. http://dx.doi.org/10.1017/cts.2020.424.
Der volle Inhalt der QuellePichler, R. H., N. Franceschini, B. A. Young, C. Hugo, T. F. Andoh, E. A. Burdmann, S. J. Shankland, C. E. Alpers, W. M. Bennett und W. G. Couser. „Pathogenesis of cyclosporine nephropathy: roles of angiotensin II and osteopontin.“ Journal of the American Society of Nephrology 6, Nr. 4 (Oktober 1995): 1186–96. http://dx.doi.org/10.1681/asn.v641186.
Der volle Inhalt der QuelleWang, Hao, Yujiao Deng, Limeng He, Yan Deng und Wei Zhang. „Renal Interstitial Fibrosis Detected on 18F-AlF-NOTA-FAPI-04 PET/CT in a Patient With Multiple Myeloma“. Clinical Nuclear Medicine 48, Nr. 10 (02.09.2023): 896–98. http://dx.doi.org/10.1097/rlu.0000000000004804.
Der volle Inhalt der QuelleShappell, S. B., T. Gurpinar, J. Lechago, W. N. Suki und L. D. Truong. „Chronic obstructive uropathy in severe combined immunodeficient (SCID) mice: lymphocyte infiltration is not required for progressive tubulointerstitial injury.“ Journal of the American Society of Nephrology 9, Nr. 6 (Juni 1998): 1008–17. http://dx.doi.org/10.1681/asn.v961008.
Der volle Inhalt der QuelleWen, Jin, Zhengwei Ma, Man J. Livingston, Wei Zhang, Yanggang Yuan, Chunyuan Guo, Yutao Liu, Ping Fu und Zheng Dong. „Decreased secretion and profibrotic activity of tubular exosomes in diabetic kidney disease“. American Journal of Physiology-Renal Physiology 319, Nr. 4 (01.10.2020): F664—F673. http://dx.doi.org/10.1152/ajprenal.00292.2020.
Der volle Inhalt der QuelleEddy, A. A. „Experimental insights into the tubulointerstitial disease accompanying primary glomerular lesions.“ Journal of the American Society of Nephrology 5, Nr. 6 (Dezember 1994): 1273–87. http://dx.doi.org/10.1681/asn.v561273.
Der volle Inhalt der QuelleForbes, Michael S., Barbara A. Thornhill, Jordan J. Minor, Katherine A. Gordon, Carolina I. Galarreta und Robert L. Chevalier. „Fight-or-flight: murine unilateral ureteral obstruction causes extensive proximal tubular degeneration, collecting duct dilatation, and minimal fibrosis“. American Journal of Physiology-Renal Physiology 303, Nr. 1 (01.07.2012): F120—F129. http://dx.doi.org/10.1152/ajprenal.00110.2012.
Der volle Inhalt der QuelleWang, S., M. Wu, L. Chiriboga, C. Putterman, B. Goilav, A. R. Broder und H. M. Belmont. „OP0043 RENAL TUBULAR COMPLEMENT C9 DEPOSITION IS ASSOCIATED WITH RENAL TUBULAR DAMAGE AND FIBROSIS IN LUPUS NEPHRITIS“. Annals of the Rheumatic Diseases 79, Suppl 1 (Juni 2020): 28.2–29. http://dx.doi.org/10.1136/annrheumdis-2020-eular.2394.
Der volle Inhalt der QuelleHaas, Mark. „Chronic allograft nephropathy or interstitial fibrosis and tubular atrophy“. Current Opinion in Nephrology and Hypertension 23, Nr. 3 (Mai 2014): 245–50. http://dx.doi.org/10.1097/01.mnh.0000444811.26884.2d.
Der volle Inhalt der QuelleMuramatsu, Masaki, Yoji Hyodo, Abigail Lee, Atsushi Aikawa, Carmelo Puliatti, Magdi Yaqoob und Michael Sheaff. „Transplant nephrectomy; pathological features of 124 consecutive cases in a single center study over 10 years“. Journal of Nephropathology 8, Nr. 3 (21.06.2019): 23. http://dx.doi.org/10.15171/jnp.2019.23.
Der volle Inhalt der QuelleMao, Haiping, Zhilian Li, Yi Zhou, Zhijian Li, Shougang Zhuang, Xin An, Baiyu Zhang et al. „HSP72 attenuates renal tubular cell apoptosis and interstitial fibrosis in obstructive nephropathy“. American Journal of Physiology-Renal Physiology 295, Nr. 1 (Juli 2008): F202—F214. http://dx.doi.org/10.1152/ajprenal.00468.2007.
Der volle Inhalt der QuelleWei, Qingqing, Jennifer Su, Guie Dong, Ming Zhang, Yuqing Huo und Zheng Dong. „Glycolysis inhibitors suppress renal interstitial fibrosis via divergent effects on fibroblasts and tubular cells“. American Journal of Physiology-Renal Physiology 316, Nr. 6 (01.06.2019): F1162—F1172. http://dx.doi.org/10.1152/ajprenal.00422.2018.
Der volle Inhalt der QuelleGupta, Kanishk. „Karyomegalic Interstitial Nephritis-A Rare Cause Of Chronic Tubulointerstitial Nephritis“. Nephrology & Renal Therapy 6, Nr. 3 (31.12.2020): 1–3. http://dx.doi.org/10.24966/nrt-7313/100042.
Der volle Inhalt der QuelleWarner, Gina M., Jingfei Cheng, Bruce E. Knudsen, Catherine E. Gray, Ansgar Deibel, Justin E. Juskewitch, Lilach O. Lerman, Stephen C. Textor, Karl A. Nath und Joseph P. Grande. „Genetic deficiency of Smad3 protects the kidneys from atrophy and interstitial fibrosis in 2K1C hypertension“. American Journal of Physiology-Renal Physiology 302, Nr. 11 (01.06.2012): F1455—F1464. http://dx.doi.org/10.1152/ajprenal.00645.2011.
Der volle Inhalt der QuelleWu, Jinhao, Chao Huang, Gang Kan, Hanyu Xiao, Xiaoping Zhang und Jun Yang. „Silymarin Regulates Tgf-β1/Smad3 Signaling Pathway and Improves Renal Tubular Interstitial Fibrosis Caused by Obstructive Nephropathy“. Current Topics in Nutraceutical Research 19, Nr. 4 (17.03.2021): 508–13. http://dx.doi.org/10.37290/ctnr2641-452x.19:508-513.
Der volle Inhalt der QuelleKimura, Kuniko, Masayuki Iwano, Debra F. Higgins, Yukinari Yamaguchi, Kimihiko Nakatani, Koji Harada, Atsushi Kubo et al. „Stable expression of HIF-1α in tubular epithelial cells promotes interstitial fibrosis“. American Journal of Physiology-Renal Physiology 295, Nr. 4 (Oktober 2008): F1023—F1029. http://dx.doi.org/10.1152/ajprenal.90209.2008.
Der volle Inhalt der QuelleVIELHAUER, VOLKER, HANS-JOACHIM ANDERS, MATTHIAS MACK, JOSEF CIHAK, FRANK STRUTZ, MANFRED STANGASSINGER, BRUNO LUCKOW, HERMANN-JOSEF GRÖNE und DETLEF SCHLÖNDORFF. „Obstructive Nephropathy in the Mouse: Progressive Fibrosis Correlates with Tubulointerstitial Chemokine Expression and Accumulation of CC Chemokine Receptor 2- and 5-Positive Leukocytes“. Journal of the American Society of Nephrology 12, Nr. 6 (Juni 2001): 1173–87. http://dx.doi.org/10.1681/asn.v1261173.
Der volle Inhalt der QuelleKuruş, Meltem, Murat Ugras und Mukaddes Esrefoglu. „Effect of resveratrol on tubular damage and interstitial fibrosis in kidneys of rats exposed to cigarette smoke“. Toxicology and Industrial Health 25, Nr. 8 (September 2009): 539–44. http://dx.doi.org/10.1177/0748233709346755.
Der volle Inhalt der QuelleBurdmann, E. A., T. F. Andoh, C. C. Nast, A. Evan, B. A. Connors, T. M. Coffman, J. Lindsley und W. M. Bennett. „Prevention of experimental cyclosporin-induced interstitial fibrosis by losartan and enalapril“. American Journal of Physiology-Renal Physiology 269, Nr. 4 (01.10.1995): F491—F499. http://dx.doi.org/10.1152/ajprenal.1995.269.4.f491.
Der volle Inhalt der QuellePang, Maoyin, Jagan Kothapally, Haiping Mao, Evelyn Tolbert, Murugavel Ponnusamy, Y. Eugene Chin und Shougang Zhuang. „Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy“. American Journal of Physiology-Renal Physiology 297, Nr. 4 (Oktober 2009): F996—F1005. http://dx.doi.org/10.1152/ajprenal.00282.2009.
Der volle Inhalt der QuelleQuimby, Jessica M., Shannon M. McLeland, Rachel E. Cianciolo, Katharine F. Lunn, Jody P. Lulich, Andrea Erikson und Lara B. Barron. „Frequency of histologic lesions in the kidneys of cats without kidney disease“. Journal of Feline Medicine and Surgery 24, Nr. 12 (Dezember 2022): e472-e480. http://dx.doi.org/10.1177/1098612x221123768.
Der volle Inhalt der QuelleDebelle, Frédéric D., Joëlle L. Nortier, Eric G. De Prez, Christian H. Garbar, Anne R. Vienne, Isabelle J. Salmon, Monique M. Deschodt-Lanckman und Jean-Louis Vanherweghem. „Aristolochic Acids Induce Chronic Renal Failure with Interstitial Fibrosis in Salt-Depleted Rats“. Journal of the American Society of Nephrology 13, Nr. 2 (Februar 2002): 431–36. http://dx.doi.org/10.1681/asn.v132431.
Der volle Inhalt der QuelleRekhtina, I. G., E. V. Kazarina, E. S. Stolyarevich, A. M. Kovrigina, V. N. Dvirnyk, S. M. Kulikov und L. P. Mendeleeva. „Morphological and immunohistochemical predictors of renal response to therapy patients with myeloma cast nephropathy and dialysis-dependent acute kidney injury“. Terapevticheskii arkhiv 92, Nr. 7 (01.09.2020): 63–69. http://dx.doi.org/10.26442/00403660.2020.07.000776.
Der volle Inhalt der QuelleTampe, Désirée, Laura Schridde, Peter Korsten, Philipp Ströbel, Michael Zeisberg, Samy Hakroush und Björn Tampe. „Different Patterns of Kidney Fibrosis Are Indicative of Injury to Distinct Renal Compartments“. Cells 10, Nr. 8 (06.08.2021): 2014. http://dx.doi.org/10.3390/cells10082014.
Der volle Inhalt der QuelleSun, Ke, Zhenliang Fan und Junfeng Fan. „A study on the mechanism of cordycepin in regulating autophagy and alleviating renal tubular interstitial fibrosis“. Tropical Journal of Pharmaceutical Research 23, Nr. 3 (14.04.2024): 529–35. http://dx.doi.org/10.4314/tjpr.v23i3.6.
Der volle Inhalt der QuelleRanganathan, Punithavathi, Calpurnia Jayakumar und Ganesan Ramesh. „Proximal tubule-specific overexpression of netrin-1 suppresses acute kidney injury-induced interstitial fibrosis and glomerulosclerosis through suppression of IL-6/STAT3 signaling“. American Journal of Physiology-Renal Physiology 304, Nr. 8 (15.04.2013): F1054—F1065. http://dx.doi.org/10.1152/ajprenal.00650.2012.
Der volle Inhalt der QuelleHuang, Ming, Shuai Zhu, Huihui Huang, Jinzhao He, Kenji Tsuji, William W. Jin, Dongping Xie et al. „Integrin-Linked Kinase Deficiency in Collecting Duct Principal Cell Promotes Necroptosis of Principal Cell and Contributes to Kidney Inflammation and Fibrosis“. Journal of the American Society of Nephrology 30, Nr. 11 (25.10.2019): 2073–90. http://dx.doi.org/10.1681/asn.2018111162.
Der volle Inhalt der QuelleWang, Xiaohua, Yang Zhou, Ruoyun Tan, Mingxia Xiong, Weichun He, Li Fang, Ping Wen, Lei Jiang und Junwei Yang. „Mice lacking the matrix metalloproteinase-9 gene reduce renal interstitial fibrosis in obstructive nephropathy“. American Journal of Physiology-Renal Physiology 299, Nr. 5 (November 2010): F973—F982. http://dx.doi.org/10.1152/ajprenal.00216.2010.
Der volle Inhalt der QuelleGui, Yuan, und Chunsun Dai. „mTOR Signaling in Kidney Diseases“. Kidney360 1, Nr. 11 (03.09.2020): 1319–27. http://dx.doi.org/10.34067/kid.0003782020.
Der volle Inhalt der QuelleYamashita, Noriyuki, Tetsuro Kusaba, Tomohiro Nakata, Aya Tomita, Tomoharu Ida, Noriko Watanabe-Uehara, Kisho Ikeda et al. „Intratubular epithelial-mesenchymal transition and tubular atrophy after kidney injury in mice“. American Journal of Physiology-Renal Physiology 319, Nr. 4 (01.10.2020): F579—F591. http://dx.doi.org/10.1152/ajprenal.00108.2020.
Der volle Inhalt der QuelleKida, Yujiro, Kinji Asahina, Hirobumi Teraoka, Inna Gitelman und Tetsuji Sato. „Twist Relates to Tubular Epithelial-Mesenchymal Transition and Interstitial Fibrogenesis in the Obstructed Kidney“. Journal of Histochemistry & Cytochemistry 55, Nr. 7 (19.03.2007): 661–73. http://dx.doi.org/10.1369/jhc.6a7157.2007.
Der volle Inhalt der QuelleGinley, Brandon, Kuang-Yu Jen, Seung Seok Han, Luís Rodrigues, Sanjay Jain, Agnes B. Fogo, Jonathan Zuckerman et al. „Automated Computational Detection of Interstitial Fibrosis, Tubular Atrophy, and Glomerulosclerosis“. Journal of the American Society of Nephrology 32, Nr. 4 (23.02.2021): 837–50. http://dx.doi.org/10.1681/asn.2020050652.
Der volle Inhalt der QuelleHart, Allyson, Scott Jackson, Bertram L. Kasiske, Michael S. Mauer, Behzad Najafian, Arthur J. Matas, Richard Spong und Hassan N. Ibrahim. „Uric Acid and Allograft Loss From Interstitial Fibrosis/Tubular Atrophy“. Transplantation 97, Nr. 10 (Mai 2014): 1066–71. http://dx.doi.org/10.1097/01.tp.0000440952.29757.66.
Der volle Inhalt der QuelleWilson, Parker C., Michael Kashgarian und Gilbert Moeckel. „Interstitial inflammation and interstitial fibrosis and tubular atrophy predict renal survival in lupus nephritis“. Clinical Kidney Journal 11, Nr. 2 (31.08.2017): 207–18. http://dx.doi.org/10.1093/ckj/sfx093.
Der volle Inhalt der QuelleMorales, Enrique, Hernando Trujillo, Teresa Bada, Marina Alonso, Eduardo Gutiérrez, Esther Rodríguez, Elena Gutiérrez, María Galindo und Manuel Praga. „What is the value of repeat kidney biopsies in patients with lupus nephritis?“ Lupus 30, Nr. 1 (20.10.2020): 25–34. http://dx.doi.org/10.1177/0961203320965703.
Der volle Inhalt der QuelleCahyawati, Putu Nita, Ngatidjan ., Dwi Cahyani Ratna Sari, Muhammad Mansyur Romi, Nur Arfian, Muhammad Mansyur Romi, Muhammad Mansyur Romi, Nur Arfian und Nur Arfian. „SIMVASTATIN ATTENUATES RENAL FAILURE IN MICE WITH A 5/6 SUBTOTAL NEPHRECTOMY“. International Journal of Pharmacy and Pharmaceutical Sciences 9, Nr. 5 (01.05.2017): 12. http://dx.doi.org/10.22159/ijpps.2017v9i5.12261.
Der volle Inhalt der QuelleCui, Wenpeng, Hasiyeti Maimaitiyiming, Xinyu Qi, Heather Norman, Qi Zhou, Xiaojun Wang, Jian Fu und Shuxia Wang. „Increasing cGMP-dependent protein kinase activity attenuates unilateral ureteral obstruction-induced renal fibrosis“. American Journal of Physiology-Renal Physiology 306, Nr. 9 (01.05.2014): F996—F1007. http://dx.doi.org/10.1152/ajprenal.00657.2013.
Der volle Inhalt der QuelleYao, Lan, M. Frances Wright, Brandon C. Farmer, Laura S. Peterson, Amir M. Khan, Jianyong Zhong, Leslie Gewin, Chuan-Ming Hao, Hai-Chun Yang und Agnes B. Fogo. „Fibroblast-specific plasminogen activator inhibitor-1 depletion ameliorates renal interstitial fibrosis after unilateral ureteral obstruction“. Nephrology Dialysis Transplantation 34, Nr. 12 (10.04.2019): 2042–50. http://dx.doi.org/10.1093/ndt/gfz050.
Der volle Inhalt der QuelleFine, L. G., und J. T. Norman. „Renal growth responses to acute and chronic injury: routes to therapeutic intervention.“ Journal of the American Society of Nephrology 2, Nr. 10 (April 1992): S206. http://dx.doi.org/10.1681/asn.v210s206.
Der volle Inhalt der QuelleYang, Junwei, und Youhua Liu. „Delayed administration of hepatocyte growth factor reduces renal fibrosis in obstructive nephropathy“. American Journal of Physiology-Renal Physiology 284, Nr. 2 (01.02.2003): F349—F357. http://dx.doi.org/10.1152/ajprenal.00154.2002.
Der volle Inhalt der QuelleTorsello, Barbara, Sofia De Marco, Silvia Bombelli, Ingrid Cifola, Ivana Morabito, Lara Invernizzi, Chiara Meregalli et al. „High glucose induces an activated state of partial epithelial-mesenchymal transition in human primary tubular cell cultures“. PLOS ONE 18, Nr. 2 (24.02.2023): e0279655. http://dx.doi.org/10.1371/journal.pone.0279655.
Der volle Inhalt der QuelleEddy, A. A. „Molecular insights into renal interstitial fibrosis.“ Journal of the American Society of Nephrology 7, Nr. 12 (Dezember 1996): 2495–508. http://dx.doi.org/10.1681/asn.v7122495.
Der volle Inhalt der QuelleMa, Frank Y., Jian Liu, A. Richard Kitching, Carl L. Manthey und David J. Nikolic-Paterson. „Targeting renal macrophage accumulation via c-fms kinase reduces tubular apoptosis but fails to modify progressive fibrosis in the obstructed rat kidney“. American Journal of Physiology-Renal Physiology 296, Nr. 1 (Januar 2009): F177—F185. http://dx.doi.org/10.1152/ajprenal.90498.2008.
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