Auswahl der wissenschaftlichen Literatur zum Thema „Troubles cognitifs mineurs“
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Zeitschriftenartikel zum Thema "Troubles cognitifs mineurs"
Mallet, J., N. Ramoz, P. Gorwood und C. Dubertret. „Cannabis et schizophrénie : approche clinique, cognitive et neurologique dans la définition d’un nouveau phénotype“. European Psychiatry 28, S2 (November 2013): 7–8. http://dx.doi.org/10.1016/j.eurpsy.2013.09.018.
Der volle Inhalt der QuelleEl Kadiri, Wafa, Benoit Delpont, Sophie Mohr, Corinne Leloup, Laurent Brondel, Yannick Bejot und Agnès Jacquin-Piques. „Étude de la gustation chez les patients ayant des troubles cognitifs mineurs ou majeurs de type Alzheimer, par l’analyse des potentiels évoqués gustatifs“. Revue Neurologique 178 (April 2022): S3. http://dx.doi.org/10.1016/j.neurol.2022.02.124.
Der volle Inhalt der QuelleKrebs, M. O., O. Gay, G. Martinez und I. Amado. „Signes neurologiques mineurs et contrôle moteur : ce qu’ils nous apprennent sur la schizophrénie, ses mécanismes et ses frontières“. European Psychiatry 29, S3 (November 2014): 580–81. http://dx.doi.org/10.1016/j.eurpsy.2014.09.282.
Der volle Inhalt der QuellePechenot-Paillot, Audrey, Thierry Moulin und Elisabeth De Bustos Medeiros. „Troubles cognitifs post AVC chez des patients victimes d’un accident vasculaire mineur ou d’un AIT suivi en hôpital de jour de neuro-vasculaire à Besançon : facteurs de risques et conséquences“. Revue Neurologique 175 (April 2019): S69. http://dx.doi.org/10.1016/j.neurol.2019.01.197.
Der volle Inhalt der QuelleMenez, Cindy, Christelle Filleau, Lyne Daumas, Alexandra Konig, Justine Lemaire und Guillaume Sacco. „Apport de l’activité physique sur la motivation des patients atteints de troubles cognitifs mineurs à majeurs“. Soins Gérontologie, Juli 2023. http://dx.doi.org/10.1016/j.sger.2023.06.005.
Der volle Inhalt der QuelleDissertationen zum Thema "Troubles cognitifs mineurs"
Payne, Magali. „Olfaction, cognition et émotions : liens dans la maladie d'Alzheimer, l'apathie et la COVID-19“. Electronic Thesis or Diss., Université Côte d'Azur, 2022. http://www.theses.fr/2022COAZ6013.
Der volle Inhalt der QuelleIn this thesis we created an olfactory test to measure its usefullness to detect disorders such as Alzheimer's disease or apathy syndrome or COVID-19.First, we collected the opinions of clinicians and the recommendations of experts chemists. We then selected fourteen odors, and we developed a computerized olfactory test that was able to calculate threshold and identification automatically using theses odors. We submitted a Patent for this test named "Olfactory Test for screening Alzheimer's disease ans apathy" (TODA).We cerified the interest ol TODA in everyday care practice in the diagnosis of Alzheimer's disease. Using subjects with Alzheimer's disease and healthy control subjects, both in France and Quebec, the test scores significantly differenciate AD subjects from control subjects. This test allowed cognitive monitoring of patients and highlighted odor identification regardless of culture and pathology.With a population of subjects with Minor Cognitive Disorder, with and without apathy, the results were able to significantly differenciate subjects, in particular, with a social sub-dimension of Critéria of Apathy.With subjects suffering from post-COVID-19 infection we tested with the TODA and in parallel we used the Sniffin' Stick Test, a gold standard, to verify the validity of the TODA results. The TODA also allowed us to highlight the problems identify similar odors. Finally, we also highlighted the interest of using the TODA in the case of suspected associated semantics impairment.The comparison of the TODA scores obtained by AD subjects and COVID-19 subjects showed that there was no difference between the two groups. Only the study of the most significant odors made it possible to differenciate between the two patologies
Gay, Olivier. „Marqueurs neurodéveloppementaux en psychiatrie : intérêt dans les troubles schizophréniques“. Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB016/document.
Der volle Inhalt der QuelleThe term neurodevelopment in its broadest sense refers to all of the processes encompassing development of the nervous system from the earliest stages of formation in utero to later stages of maturation during adolescence to produce the fully functional adult nervous system. Work over the last thirty years has led to a neurodevelopmental model of human psychiatric disorders, including schizophrenia, based on genetic, epidemiological and imaging evidence. This model asserts that disease is fundamentally linked to or develops from abnormality(s) in the formation processes (early neurodevelopment) and maturation (late neurodevelopment) of the nervous system due to a combination of genetic and environmental factors. In this context this thesis aims to clarify the effects of neurodevelopmental abnormalities on psychiatric disorders, including schizophrenia, through the study of different markers. The first study aims to investigate correlations between markers of early brain development: a clinical marker (neurological soft signs) and an imaging marker (sulcation of the cerebral cortex) in a population of subjects with schizophrenia. A correlation between these two markers is presented: the sulcation index was found to be lower in subjects that had significant neurological soft signs. We concluded that the combined study of different markers may help to isolate subgroups of patients with greater early neurodevelopmental damage. The second study aims to characterize effects of different markers of early neurodevelopmental abnormalities on cognitive functioning in patients with schizophrenia. Effects on executive control (as measured by the Trail Making Test) were correlated with clinical markers (neurological soft signs, handedness) and imaging (sulcation of the anterior cingulate cortex and enlargment of the ventricles). We found interactions between different markers with a mainly non-linear summation effect. Our interpretation is that different markers reflect separate insults, though all early, on brain development with a common final effect on executive function. The third study aims to clarify the specificity of sulcation as a marker of early neurodevelopmental abnormalities by studying a population of adult subjects with autism spectrum disorder (ASD), a patholody beginning in early childhood and linked with evidence of early neurodevelopmental damage. Sulcation abnormalities of the anterior cingulate cortex, similar to those observed in patients with schizophrenia are detected in patients with ASD. These results suggest early neurodevelopmental abnormalities are shared by different psychiatric disorders and that changes in cortical sulcation are not specific to a given disorder but the early damage. In conclusion, we suggest that the study of neurodevelopmental abnormalities should be integrated into a dimensional approach in psychiatry
Dervaux, Alain. „Influence de la consommation de substances sur l'émergence et l'évolution des troubles psychotiques“. Phd thesis, Université Pierre et Marie Curie - Paris VI, 2010. http://tel.archives-ouvertes.fr/tel-00814790.
Der volle Inhalt der QuelleAndriuta, Daniela. „Les fonctions exécutives, le ralentissement de l'action et leurs corrélats anatomiques dans les phases précoces de la Maladie d'Alzheimer“. Electronic Thesis or Diss., Amiens, 2019. http://www.theses.fr/2019AMIE0044.
Der volle Inhalt der QuelleAlzheimer’s disease (AD) progresses from preclinical isolated subjective cognitive complaints (SCC) to mild neurocognitive disorders (NCD) and then major NCD and is “typically” characterized by prominent memory impairment. In early stages, the intensity of ED and action slowing and their imaging correlates are undetermined. The main objective was to establish in early stages of AD, the intensity of executive dysfunction (ED) and action slowing as well as their imaging correlates. In the Study 1, we showed that executive functions were impaired to a similar extent in AD (with positive CSF biomarkers) and VCI. In the Study 2 we performed a meta-analysis (327 mild NCD and 468 healthy controls) and showed that mean simple reaction time (SRT) was longer in mild NCD group. In the Study 3 we investigated the imaging correlates of ED in MEMENTO cohort (2323 mild NCD or SCC). ED were related to cortical thickness in the left pars orbitalis, right precuneus, right supramarginal (SMG) and the right isthmus cingulate regions. In the Study 4 we determined the imaging correlates (using voxelwise based morphometry) of action slowing in 30 patients with NCD (25 AD, 3 Lewy body dementia, 2 behavioral frontotemporal degeneration). SRT slowing was associated with right dorso-lateral prefrontal and left SMG cortices atrophy. To conclude in early stages of AD, action slowing and ED were a part of the cognitive profile. The atrophy of inferior parietal (including the SMG) and to a lesser extent, of prefrontal cortices accounted for ED and action slowing. In the future studies these results will have to be confirmed in prodromal and preclinical AD
Martinez, Gilles. „Continuum autisme-schizophrénie : apport de l’étude de la cognition sociale et de marqueurs phénotypiques développementaux“. Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB065/document.
Der volle Inhalt der QuelleAutism and schizophrenia are both neurodevelopmental psychiatric disorders. Research on early-onset schizophrenia, commonly associated to autism spectrum disorders (ASD), suggested a possible developmental continuum between both of these disorders. Clinical and epidemiological evidence, and research from molecular genetics or brain imaging, come to support this hypothesis. In this context, social cognition is a matter of special interest. Impairments are reported both in the two disorders, but with inconsistent results, revealing common features as well as differences. Otherwise, links between social cognition impairments and neurodevelopmental burden have been until now poorly explored. Through the contribution of our three studies, we confirmed the importance of social cognition impairment in autism and schizophrenia. The MASC test (Movie for the Assessment of Social Cognition), an original tool which was by our findings validated in a French version, revealed higher overall impairment of mentalizing capabilities in ASD than in schizophrenia. Animated Shapes (non verbal test of attribution of intentions) revealed qualitative differences: whereas hypomentalizing is common both to ASD and schizophrenia, overmentalizing seemed to be more important in schizophrenia. Furthermore, along a continuum between autism and schizophrenia, social cognition impairment was linked to thought and language disorganization, and to neurological soft signs (a marker for neurodevelopmental load). In addition, in subjects with schizophrenia, overmentalizing was correlated to the precocity of onset of the disease. Altogether, our results highlight the need to screen developmental feature in adulthood. In that way, we presented preliminary results in order to validate a developmental disorders screening self-rated questionnaire. As a conclusion, our results bring evidence in favour of a hypothesis of a continuum between autism and schizophrenia, showing a social cognition impairment in both disorders, correlated to the neurodevelopmental load existing in both of them in a transnosographic way. We contributed to emphasize the sub-group of subjects with schizophrenia with early-onset of disease, characterized by a tendency to overmentalizing and presenting a marked disorganization. Our work provides avenue to further studies, integrating neuroimaging and genetic data, that will help to advance in a deeper comprehension of the pathophysiology of autism and schizophrenia. Furthermore, we used and validated in this work promising tools to improve finely psychopathological evaluation and differential diagnosis in adults suffering from autism and from schizophrenia
Mallet, Jasmina. „Marqueurs neurodéveloppementaux, cognition et facteurs environnementaux précoces et tardifs dans le phénotype psychotique des pathologies mentales Heavy cannabis use prior psychosis in schizophrenia : clinical, cognitive and neurological evidences for a new endophenotype? Etude et apport de la latéralité comme marqueur neurodéveloppemental dans les troubles schizophréniques et bipolaires Cigarette smoking and schizophrenia : a specific clinical and therapeutic profile? Results from the Face-Schizophrenia cohort Tobacco smoking is associated with antipsychotic medication, physical aggressiveness and alcohol use disorder in schizophrenia : results from the Face-SZ national cohort Tabagisme et schizophrénie, impact sur la cognition Tobacco smoking and psychotic-like experiences in a general population sample Poster congrès français de psychiatrie 2018 : Expériences psychotiques chez 50 patients adolescents hospitalisés pour la 1ère fois : approche trans-diagnostique et prospective avec la PQ16“. Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2403&f=17360.
Der volle Inhalt der QuelleMental diseases represent a very heterogeneous categorical group, even within a given nosographic entity. Multifactorial approaches allow accounting for the clinical heterogeneity of mental disorders, the continuum between certain clinical dimensions, and even between the normal and the pathological. Among such dimensions, the psychotic phenotype constitutes an essential dimension of schizophrenic disorder. The dimensional approach allows for the search of psychotic experiences in most mental disorders as well as in the general population. We make the general hypothesis that certain psychiatric disorders with psychotic symptoms could be the result of the interaction between early- (obstetric traumas for example) and late- environmental factors (toxics, traumatisms) and the neurodevelopment of the individual. The initial step in this thesis work was to better define the concepts of vulnerability in psychiatry, and, based on the example of schizophrenia, to conduct a review of the literature on risk factors according to their early or late interaction with neurodevelopment. Subsequently, the first axis of research of the present thesis was to evaluate early neurodevelopmental markers (neurological soft signs, laterality, cognition). Our first work concerned the clinical, neurological and cognitive characterization of 64 patients suffering from schizophrenia, according to their cannabis use (or not) prior to psychosis. It provided evidence for a lower burden of neurodevelopment in cannabis users, and the potential impact of this substance on vulnerable individuals. Our second work concerns the clinical and cognitive impact of lateralization in patients with schizophrenia (n = 667) and bipolar disorder (n = 2445). We bring arguments for a neurodevelopmental weight (measured with this lateralization index) that is more important in schizophrenia. Our second axis of research focused on tobacco smoking as a late environmental factor in schizophrenia and psychotic phenotype. We showed in two studies on the FACE-SZ cohort (n = 361, n = 474) that SZ patients consumed almost twice as much as the general population and that they could represent a SZ subgroup with specific socio-demographic and clinical characteristics. In a third study, we compare the cognitive functions of these patients (n = 785) and show that the self-medication hypothesis alone cannot account for the high prevalence of their smoking. In a fourth work, we studied the impact of smoking on the psychotic phenotype with a dimensional approach, and showed an association between smoking and certain psychotic-type experiences in a representative sample of the US general population (NESARC, n = 34653). Finally, in a last line of research, we evaluated the psychotic phenotype in a population of adolescents and young adults hospitalized for a first psychiatric episode (n = 50). In a preliminary study, we show a high prevalence of psychotic-like experiences in these young adults, regardless of the diagnosis made six months afterwards, highlighting the trans-nosographic character of the psychotic phenotype during the emergence of different mental disorders. Overall, the present thesis underscores the clinical heterogeneity of mental illnesses and the importance of dimensional and trajectory approaches in identifying risk (or protective) factors, towards a better etiopathogenic understanding, better prevention opportunities, and a personalized patient care