Auswahl der wissenschaftlichen Literatur zum Thema „Transport neuronal“
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Zeitschriftenartikel zum Thema "Transport neuronal"
Sammler, Esther, Stefan Titz und Sheriar Hormuzdi. „Neuronal chloride transport tuning“. Lancet 385 (Februar 2015): S85. http://dx.doi.org/10.1016/s0140-6736(15)60400-7.
Der volle Inhalt der QuelleJODAL, M. „Neuronal influence on intestinal transport“. Journal of Internal Medicine 228, S732 (November 1990): 125–32. http://dx.doi.org/10.1111/j.1365-2796.1990.tb01484.x.
Der volle Inhalt der QuelleBenaïssa, Ibtissem. „Analogie du transport neuronal au transport électronique en nanotechnologie“. Journal of Renewable Energies 12, Nr. 1 (26.10.2023): 9–28. http://dx.doi.org/10.54966/jreen.v12i1.115.
Der volle Inhalt der QuelleMENZIKOV, SERGEY A. „NEURONAL MULTIFUNCTIONAL ATPase“. Biophysical Reviews and Letters 08, Nr. 03n04 (Dezember 2013): 213–27. http://dx.doi.org/10.1142/s1793048013300065.
Der volle Inhalt der QuelleKaye, D. M., S. D. Wiviott, L. Kobzik, R. A. Kelly und T. W. Smith. „S-nitrosothiols inhibit neuronal norepinephrine transport“. American Journal of Physiology-Heart and Circulatory Physiology 272, Nr. 2 (01.02.1997): H875—H883. http://dx.doi.org/10.1152/ajpheart.1997.272.2.h875.
Der volle Inhalt der QuellePerry, Rotem Ben-Tov, und Mike Fainzilber. „Nuclear transport factors in neuronal function“. Seminars in Cell & Developmental Biology 20, Nr. 5 (Juli 2009): 600–606. http://dx.doi.org/10.1016/j.semcdb.2009.04.014.
Der volle Inhalt der QuelleStiess, Michael, und Frank Bradke. „Neuronal transport: myosins pull the ER“. Nature Cell Biology 13, Nr. 1 (12.12.2010): 10–11. http://dx.doi.org/10.1038/ncb2147.
Der volle Inhalt der QuelleStaff, N. P., E. E. Benarroch und C. J. Klein. „Neuronal intracellular transport and neurodegenerative disease“. Neurology 76, Nr. 11 (14.03.2011): 1015–20. http://dx.doi.org/10.1212/wnl.0b013e31821103f7.
Der volle Inhalt der QuelleBrenner, S. R., N. P. Staff, E. E. Benarroch und C. J. Klein. „Neuronal intracellular transport and neurodegenerative disease“. Neurology 77, Nr. 21 (21.11.2011): 1932. http://dx.doi.org/10.1212/wnl.0b013e318239bf96.
Der volle Inhalt der QuelleBakshi, Rachit, Shuchi Mittal, Zhixiang Liao und Clemens R. Scherzer. „A Feed-Forward Circuit of EndogenousPGC-1αandEstrogen Related Receptor αRegulates the Neuronal Electron Transport Chain“. Parkinson's Disease 2016 (2016): 1–9. http://dx.doi.org/10.1155/2016/2405176.
Der volle Inhalt der QuelleDissertationen zum Thema "Transport neuronal"
MacAskill, A. F. N. „Control of neuronal mitochondrial transport“. Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19495/.
Der volle Inhalt der QuelleMahato, Deependra. „Mutation of Polaris, an Intraflagellar Transport Protein, Shortens Neuronal Cilia“. Thesis, University of North Texas, 2005. https://digital.library.unt.edu/ark:/67531/metadc4856/.
Der volle Inhalt der QuelleChen, Liang. „Single molecule and single particle studies of neuronal axonal transport /“. May be available electronically:, 2009. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.
Der volle Inhalt der QuelleJohnson, Christopher M. „Investigating the Slow Axonal Transport of Neurofilaments: A Precursor for Optimal Neuronal Signaling“. Ohio University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1452018547.
Der volle Inhalt der QuellePekergin, Mehmet Ferhan. „Optimisation combinatoire par le calcul neuronal et parallelisme optimal“. Paris 5, 1992. http://www.theses.fr/1992PA05S017.
Der volle Inhalt der QuelleCoats, Charles Jason. „Development of primary neuronal culture of embryonic rabbit dorsal root ganglia for microfluidic chamber analysis of axon mediated neuronal spread of Bovine Herpesvirus type 1“. Thesis, Manhattan, Kan. : Kansas State University, 2010. http://hdl.handle.net/2097/4115.
Der volle Inhalt der QuelleLi, Yunyun [Verfasser], und Peter [Akademischer Betreuer] Hänggi. „Noise assisted transport in artificial channels and neuronal membranes / Yunyun Li. Betreuer: Peter Hänggi“. Augsburg : Universität Augsburg, 2011. http://d-nb.info/1077700296/34.
Der volle Inhalt der QuelleCopp, Steven Wesley. „Enzymatic regulation of skeletal muscle oxygen transport: novel roles for neuronal nitric oxide synthase“. Diss., Kansas State University, 2013. http://hdl.handle.net/2097/15512.
Der volle Inhalt der QuelleDepartment of Anatomy and Physiology
Timothy I. Musch
Nitric oxide (NO) is synthesized via distinct NO synthase (NOS) enzymes and constitutes an essential cardiovascular signaling molecule. Whereas important vasomotor contributions of endothelial NOS (eNOS) have been well-described, the specific vasomotor contributions of nNOS-derived NO in healthy subjects during exercise are unknown. The purpose of this dissertation is to test the global hypothesis that nNOS-derived NO is a critical regulator of exercising skeletal muscle vascular control. Specifically, we utilized the selective nNOS inhibitor S-methyl-L-thiocitrulline (SMTC) to investigate the effects of nNOS-derived NO on skeletal muscle vascular function within established rodent models of exercise performance. The first investigation (Chapter 2) identifies that nNOS inhibition with SMTC increases mean arterial pressure (MAP) and reduces rat hindlimb skeletal muscle blood flow at rest whereas there are no effects during low-speed (20 m/min) treadmill running. In Chapter 3 it is reported that nNOS inhibition with SMTC reduces blood flow during high-speed treadmill running (>50 m/min) with the greatest relative effects found in highly glycolytic fast-twitch muscles and muscle parts. Chapter 4 demonstrates that nNOS-derived NO modulates contracting skeletal muscle blood flow (increases), O2 consumption (VO2, increases), and force production (decreases) in the rat spinotrapezius muscle and thus impacts the microvascular O2 delivery-VO2 ratio (which sets the microvascular partial pressure of O2, PO2mv, and represents the pressure head that drives capillary-myocyte O2 diffusion). In Chapter 5 we report that systemic administration of the selective nNOS inhibitor SMTC does not impact lumbar sympathetic nerve discharge. This reveals that the SMTC-induced peripheral vascular effects described herein reflect peripheral nNOS-derived NO signaling as opposed to centrally-derived regulation. In conclusion, nNOS-derived NO exerts exercise-intensity and muscle fiber-type selective peripheral vascular effects during whole-body locomotor exercise. In addition, nNOS-derived NO modulates skeletal muscle contractile and metabolic function and, therefore, impacts the skeletal muscle PO2mv. These data identify novel integrated roles for nNOS-derived NO within healthy skeletal muscle and have important implications for populations associated with reduced NO bioavailability and/or impaired nNOS structure and/or function specifically (e.g., muscular dystrophy, chronic heart failure, advanced age, etc.).
Naudon, Laurent. „Recherche d'une participation du transporteur neuronal de la dopamine et du transporteur vésiculaire à l'adaptation neuronale“. Rouen, 1994. http://www.theses.fr/1994ROUES066.
Der volle Inhalt der QuelleDaoust, Alexia. „IRM du manganèse (MEMRI) : couplage à l'imagerie chimique par microsonde synchrotron pour optimiser l'imagerie fonctionnelle du transport neuronal“. Phd thesis, Université de Grenoble, 2012. http://tel.archives-ouvertes.fr/tel-00770158.
Der volle Inhalt der QuelleBücher zum Thema "Transport neuronal"
Gribkoff, Valentin K. Structure, function, and modulation of neuronal voltage-gated ion channels. Hoboken, N.J: Wiley, 2009.
Den vollen Inhalt der Quelle finden1955-, Gonzalez-Lima Francisco, Hrsg. Cytochrome oxidase in neuronal metabolism and Alzheimer's disease. New York: Plenum Press, 1998.
Den vollen Inhalt der Quelle findenJ, Bean Andrew, Hrsg. Protein trafficking in neurons. Amsterdam: Elsevier/Academic Press, 2007.
Den vollen Inhalt der Quelle findenA, Lappi Douglas, Hrsg. Suicide transport and immunolesioning. Austin: R.G. Landes, 1994.
Den vollen Inhalt der Quelle findenVeli, Himanen, Nijkamp Peter, Reggiani Aura und Raitio Juha, Hrsg. Neural networks in transport applications. Aldershot, Hants, England: Ashgate, 1998.
Den vollen Inhalt der Quelle findenMurdoch, Ritchie J., Keynes R. D und Bolis Liana, Hrsg. Ion channels in neural membranes: Proceedings of the 11th International Conference on Biological Membranes held at Crans-sur-Sierre, Switzerland, June 10-14, 1985. New York: A.R. Liss, 1986.
Den vollen Inhalt der Quelle findenJ, Alvarez-Leefmans F., Russell John M. 1942- und International Brain Research Organization. Congress, Hrsg. Chloride channels and carriers in nerve, muscle, and glial cells. New York: Plenum Press, 1990.
Den vollen Inhalt der Quelle finden1953-, Iverson Linda E., Conn P. Michael und Rudy Bernardo, Hrsg. Ion channels. San Diego: Academic Press, 1992.
Den vollen Inhalt der Quelle findenCytoskeleton of the nervous system. New York: Springer, 2011.
Den vollen Inhalt der Quelle findenSuter, Daniel M., und Kyle E. Miller, Hrsg. Neuronal Mechanics and Transport. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-823-8.
Der volle Inhalt der QuelleBuchteile zum Thema "Transport neuronal"
Knight, Adam L., Yanmin Chen, Tao Sun und Zu-Hang Sheng. „Neuronal Mitochondrial Transport“. In The Functions, Disease-Related Dysfunctions, and Therapeutic Targeting of Neuronal Mitochondria, 166–85. Hoboken, NJ: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781119017127.ch7.
Der volle Inhalt der QuelleVoelzmann, André, und Natalia Sanchez-Soriano. „Drosophila Primary Neuronal Cultures as a Useful Cellular Model to Study and Image Axonal Transport“. In Methods in Molecular Biology, 429–49. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1990-2_23.
Der volle Inhalt der QuelleAtkin, Talia A., Andrew F. MacAskill und Josef T. Kittler. „Neuronal Mitochondrial Transport and Dysfunction“. In Mitochondrial Dysfunction in Neurodegenerative Disorders, 157–73. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-701-3_10.
Der volle Inhalt der QuelleStephenson, F. Anne, und Kieran Brickley. „Mechanisms of Neuronal Mitochondrial Transport“. In Folding for the Synapse, 105–19. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7061-9_6.
Der volle Inhalt der QuelleDeitmer, Joachim W. „pH regulation and acid/base-mediated transport in glial cells“. In Glial ⇔ Neuronal Signaling, 263–77. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-1-4020-7937-5_10.
Der volle Inhalt der QuelleLedeen, R. W., D. A. Aquino, M. Sbaschnig-Agler, C. M. Gammon und K. K. Vaswani. „Fundamentals of Neuronal Transport of Gangliosides. Functional Implications“. In Gangliosides and Modulation of Neuronal Functions, 259–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71932-5_21.
Der volle Inhalt der QuelleCaputto, R., B. L. Caputto, M. S. Domowicz und S. C. Kivatinitz. „Gangliosides: Biosynthesis, Transport and Location in the Plasma Membrane“. In Gangliosides and Modulation of Neuronal Functions, 251–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71932-5_20.
Der volle Inhalt der QuellePichiule, P., J. C. Chavez, R. J. Przybylski und J. C. LaManna. „Increase of Neuronal Nitric Oxide Synthase during Chronic Hypoxia“. In Oxygen Transport to Tissue XX, 319–23. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4863-8_37.
Der volle Inhalt der QuelleMiller, Richard J. „Modulation and Functions of Neuronal Ca2+ Permeable Channels“. In Calcium Transport and Intracellular Calcium Homeostasis, 3–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-83977-1_1.
Der volle Inhalt der QuelleAquino, D. A., M. A. Bisby und R. W. Ledeen. „Axonal Transport of Gangliosides and Neutral Glycolipids in the Peripheral Nervous System. Identification of Ganglioside Types in Motoneurons of the PNS“. In Gangliosides and Neuronal Plasticity, 161–69. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4757-5309-7_14.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Transport neuronal"
Zhu, Liang, und Robert Flower. „Role of Vasomotion in Control of Retina Edema in Diabetic Retinopathy: Quantification of Fluid Transport Through Retinal Capillaries“. In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-189507.
Der volle Inhalt der QuelleSmith, Jennifer A., Gregoriy Smiyun, Leslie Wilson, Stuart Feinstein und Mary Ann Jordan. „Abstract 5294: Inhibition of mitochondrial transport in neuronal cells by microtubule-targeting drugs eribulin, ixabepilone, paclitaxel, and vincristine“. In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5294.
Der volle Inhalt der QuelleLee, Sung Jin, Jingjing Sun, Michael King, Huikai Xie und Malisa Sarntinoranont. „Viscoelastic Property Changes of Acute Rat Brain Tissue Slices as a Function of Cell Viability“. In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53909.
Der volle Inhalt der QuelleStork, Larissa Rosa, Lucca Stephani Ribeiro, Izabella Savergnini Deprá, Luísa D’Ávila Camargo und Maria Angélica Santos Novaes. „Tau protein and its role in Alzheimer’s disease physiopathology: a literature review“. In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.132.
Der volle Inhalt der QuelleBrooks, Joseph Bruno Bidin. „De novo variant in the MAPK8IP3 gene in the differential diagnosis of global development delay. Case report.“ In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.181.
Der volle Inhalt der QuelleGarrido Rodriguez, Maria Concepcion. „Análisis de la calidad del servicio en el transporte público mediante redes neuronales artificiales“. In CIT2016. Congreso de Ingeniería del Transporte. Valencia: Universitat Politècnica València, 2016. http://dx.doi.org/10.4995/cit2016.2016.4120.
Der volle Inhalt der QuelleMudrakola, Harsha V., Chengbiao Wu, Kai Zhang und Bianxiao Cui. „Single Molecule Imaging of Axonal Transport in Live Neurons“. In Laser Science. Washington, D.C.: OSA, 2009. http://dx.doi.org/10.1364/ls.2009.lsthb3.
Der volle Inhalt der QuelleYang, Chun-Lin, Nandan Shettigar und C. Steve Suh. „A Proposition for Describing Real-World Network Dynamics“. In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-73360.
Der volle Inhalt der QuelleSchmitt-Böhrer, A., JF Kolter, A. Kreis, C. Hamann, C. Bodden, N. Sachser, E. Asa und K.-P. Lesch. „Impact of different life histories on neuronal morphology in serotonin transporter deficient mice“. In Abstracts of the 2nd Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402985.
Der volle Inhalt der QuelleBecker, Nicole Bernd, Allan Marinho Alcará, Isadora Ghilardi, Vitoria Pimentel, Giulia Pinzetta, Laura Provenz, Gabriel Leal et al. „Mesenchymal stem cells modulate the gene expression of cationchloride co-transporter KCC2 in epileptogenesis“. In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.693.
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