Auswahl der wissenschaftlichen Literatur zum Thema „Synapse activity“
Geben Sie eine Quelle nach APA, MLA, Chicago, Harvard und anderen Zitierweisen an
Inhaltsverzeichnis
Machen Sie sich mit den Listen der aktuellen Artikel, Bücher, Dissertationen, Berichten und anderer wissenschaftlichen Quellen zum Thema "Synapse activity" bekannt.
Neben jedem Werk im Literaturverzeichnis ist die Option "Zur Bibliographie hinzufügen" verfügbar. Nutzen Sie sie, wird Ihre bibliographische Angabe des gewählten Werkes nach der nötigen Zitierweise (APA, MLA, Harvard, Chicago, Vancouver usw.) automatisch gestaltet.
Sie können auch den vollen Text der wissenschaftlichen Publikation im PDF-Format herunterladen und eine Online-Annotation der Arbeit lesen, wenn die relevanten Parameter in den Metadaten verfügbar sind.
Zeitschriftenartikel zum Thema "Synapse activity"
Hu, Xiaoge, Jian-hong Luo und Junyu Xu. „The Interplay between Synaptic Activity and Neuroligin Function in the CNS“. BioMed Research International 2015 (2015): 1–13. http://dx.doi.org/10.1155/2015/498957.
Der volle Inhalt der QuellePettem, Katherine L., Daisaku Yokomaku, Hideto Takahashi, Yuan Ge und Ann Marie Craig. „Interaction between autism-linked MDGAs and neuroligins suppresses inhibitory synapse development“. Journal of Cell Biology 200, Nr. 3 (28.01.2013): 321–36. http://dx.doi.org/10.1083/jcb.201206028.
Der volle Inhalt der QuelleKo, Jaewon, Gilberto J. Soler-Llavina, Marc V. Fuccillo, Robert C. Malenka und Thomas C. Südhof. „Neuroligins/LRRTMs prevent activity- and Ca2+/calmodulin-dependent synapse elimination in cultured neurons“. Journal of Cell Biology 194, Nr. 2 (25.07.2011): 323–34. http://dx.doi.org/10.1083/jcb.201101072.
Der volle Inhalt der QuelleGaidarova, Svetlana, JianWu Li, Laura G. Corral, Emilia Glezer, Peter H. Schafer, Weilin Xie, Antonia Lopez-Girona, Bruce D. Cheson und Brydon Bennett. „Lenalidomide Alone and in Combination with Rituximab Enhances NK Cell Immune Synapse Formation in Chronic Lymphocytic Leukemia (CLL) Cells in Vitro through Activation of Rho and Rac1 GTPases.“ Blood 114, Nr. 22 (20.11.2009): 3441. http://dx.doi.org/10.1182/blood.v114.22.3441.3441.
Der volle Inhalt der QuelleMoss, Brenda L., Abby D. Fuller, Christie L. Sahley und Brian D. Burrell. „Serotonin Modulates Axo-Axonal Coupling Between Neurons Critical for Learning in the Leech“. Journal of Neurophysiology 94, Nr. 4 (Oktober 2005): 2575–89. http://dx.doi.org/10.1152/jn.00322.2005.
Der volle Inhalt der QuelleWei, Wei, und Xiao-Jing Wang. „Downstream Effect of Ramping Neuronal Activity through Synapses with Short-Term Plasticity“. Neural Computation 28, Nr. 4 (April 2016): 652–66. http://dx.doi.org/10.1162/neco_a_00818.
Der volle Inhalt der QuelleLiu, Kang K. L., Michael F. Hagan und John E. Lisman. „Gradation (approx. 10 size states) of synaptic strength by quantal addition of structural modules“. Philosophical Transactions of the Royal Society B: Biological Sciences 372, Nr. 1715 (05.03.2017): 20160328. http://dx.doi.org/10.1098/rstb.2016.0328.
Der volle Inhalt der QuelleZhao, Qing-Tai, Fengben Xi, Yi Han, Andreas Grenmyr, Jin Hee Bae und Detlev Gruetzmacher. „Ferroelectric Devices for Neuromorphic Computing“. ECS Meeting Abstracts MA2022-02, Nr. 32 (09.10.2022): 1183. http://dx.doi.org/10.1149/ma2022-02321183mtgabs.
Der volle Inhalt der QuelleWilson, Emily S., und Karen Newell-Litwa. „Stem cell models of human synapse development and degeneration“. Molecular Biology of the Cell 29, Nr. 24 (26.11.2018): 2913–21. http://dx.doi.org/10.1091/mbc.e18-04-0222.
Der volle Inhalt der QuelleBloom, Ona, Emma Evergren, Nikolay Tomilin, Ole Kjaerulff, Peter Löw, Lennart Brodin, Vincent A. Pieribone, Paul Greengard und Oleg Shupliakov. „Colocalization of synapsin and actin during synaptic vesicle recycling“. Journal of Cell Biology 161, Nr. 4 (19.05.2003): 737–47. http://dx.doi.org/10.1083/jcb.200212140.
Der volle Inhalt der QuelleDissertationen zum Thema "Synapse activity"
Ghezali, Grégory. „Control of synaptic transmission by astroglial connexin 30 : molecular basis, activity-dependence and physiological implication“. Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066423/document.
Der volle Inhalt der QuellePerisynaptic astrocytes are active partners of neurons in cerebral information processing. A key property of astrocytes is to express high levels of the gap junction forming proteins, the connexins (Cxs). Strikingly, astroglial Cx30 was suggested early on to be involved in cognitive processes; however, its specific role in neurophysiology has yet been unexplored. We recently reveal that Cx30, through an unconventional non-channel function, controls hippocampal glutamatergic synaptic strength and plasticity by directly setting synaptic glutamate levels through astroglial glutamate clearance. Yet the cellular and molecular mechanisms involved in such control, its dynamic regulation by activity and its impact in vivo in a physiological context were unknown. To answer these questions, I demonstrated during my PhD that: 1) Cx30 drives the morphological maturation of hippocampal astrocytes via the modulation of a laminin signaling pathway regulating cell polarization; 2) Cx30 expression, perisynaptic localization and functions are modulated by neuronal activity; 3) Cx30-mediated control of astrocyte synapse coverage in the supraoptic nucleus of the hypothalamus sets basal plasmatic level of the neurohormone oxytocin and hence promotes appropriate oxytocin-based social abilities. Taken together, these data shed new light on astroglial Cxs activity-dependent regulations and roles in the postnatal development of neuroglial networks, as well as in astrocyte-synapse structural interactions mediating behavioral processes
Mardinly, Alan Robert. „Regulation of Synapse Development by Activity Dependent Transcription in Inhibitory Neurons“. Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:10739.
Der volle Inhalt der QuelleBrown, Rosalind. „Role of activity in neuromuscular synaptic degeneration : insights from Wlds mice“. Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/6523.
Der volle Inhalt der QuelleXiao, Wei. „Class 5 semaphorins mediate synapse elimination and activity-dependent synaptic plasticity in hippocampal neurons“. Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/60340.
Der volle Inhalt der QuelleMedicine, Faculty of
Graduate
Jay, Taylor Reagan. „The TREM2 Receptor Directs Microglial Activity in Neurodegeneration and Neurodevelopment“. Case Western Reserve University School of Graduate Studies / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1560181547156823.
Der volle Inhalt der QuelleAtaman, Bulent. „The Molecular Mechanisms of Activity-Dependent Wingless (Wg)/Wnt Signaling at a Drosophila Glutamatergic Synapse: a Dissertation“. eScholarship@UMMS, 2008. https://escholarship.umassmed.edu/gsbs_diss/353.
Der volle Inhalt der QuelleSjölin, Hanna. „Regulation of NK cell activity : studies of DAP12-associated receptors in immune synapse formation and in responses to cytomegalovirus infection /“. Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-985-8/.
Der volle Inhalt der QuelleLouçã, Mathilde. „Functional impacts of Huntingtin lowering on the synaptic maturation and activity of neuronal networks derived from human induced pluripotent stem cells“. Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL054.
Der volle Inhalt der QuelleHuntington's disease (HD) is a neurodegenerative disorder caused by a mutation in the Huntingtin gene (HTT). Reducing the expression of mutant HTT is an obvious therapeutic approach explored in patients. However, targeting mutant HTT often leads to a simultaneous reduction in non-mutant HTT. The consequences of losing this protein on neuronal health remain poorly understood.My doctoral work addresses this question using in vitro models of human neuronal networks differentiated from induced pluripotent stem cells. My research demonstrates that HTT loss induces developmental and homeostatic abnormalities in these networks. My results suggest that therapies targeting both mutant and non-mutant HTT indiscriminately could compromise the health of targeted neuronal circuits
McMahon, Catherine. „The mechanisms underlying normal spike activity of the primary afferent synapse in the cochlea and its dysfunction : an investigation of the possible mechanisms of peripheral tinnitus and auditory neuropathy“. University of Western Australia. School of Biomedical and Chemical Sciences, 2004. http://theses.library.uwa.edu.au/adt-WU2003.0034.
Der volle Inhalt der QuelleKatona, Linda. „The role of cell-type selective synaptic connections in rhythmic neuronal network activity in the hippocampus“. Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:cebe42e9-4040-486b-8ff4-fa1bf642bea0.
Der volle Inhalt der QuelleBücher zum Thema "Synapse activity"
Mamalyga, L. M., Hrsg. Simulation of Neural Networks Based on Self-Assembly of Reaction-Diffusion Electrical Synapses and their Nonlinear Electrophysiological Activity, 164 p. [in Russian]. Moscow: Moscow Pedagogical State University, Department of Biology & Chemistry, 2012.
Den vollen Inhalt der Quelle findenBaldi, Elisabetta, und Corrado Bucherelli. Neuroscience. Florence: Firenze University Press, 2017. http://dx.doi.org/10.36253/978-88-6453-638-5.
Der volle Inhalt der QuelleGrant, Seth G. N. Synaptic Mechanisms of Psychotic Disorders. Herausgegeben von Dennis S. Charney, Eric J. Nestler, Pamela Sklar und Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0017.
Der volle Inhalt der QuelleFlavell, Steven Willem. Regulation of synapse development by the activity-regulated transcription factor MEF2. 2009.
Den vollen Inhalt der Quelle findenSturgill, James Fitzhugh. Activity-dependent regulation of synapse structure and function: Roles of PSD-95 and the metabolic sensor, AMPK. 2010.
Den vollen Inhalt der Quelle findenBay, Mihee J., und Bruce K. Shapiro. Attention Deficit-Hyperactivity Disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0060.
Der volle Inhalt der QuelleBeninger, Richard J. Neuroanatomy and dopamine systems. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0011.
Der volle Inhalt der QuelleBeninger, Richard J. Mechanisms of dopamine-mediated incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0012.
Der volle Inhalt der QuelleBuchteile zum Thema "Synapse activity"
Palm, Daniel, und Frank Entschladen. „Neoneurogenesis and the Neuro-Neoplastic Synapse“. In Neuronal Activity in Tumor Tissue, 91–98. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000100049.
Der volle Inhalt der QuelleZänker, Kurt S. „The Neuro-Neoplastic Synapse: Does it Exist?“ In Neuronal Activity in Tumor Tissue, 154–61. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000100075.
Der volle Inhalt der QuelleUlbricht, Carolin, Ruth Leben, Yu Cao, Raluca A. Niesner und Anja E. Hauser. „Combined FRET-FLIM and NAD(P)H FLIM to Analyze B Cell Receptor Signaling Induced Metabolic Activity of Germinal Center B Cells In Vivo“. In The Immune Synapse, 91–111. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3135-5_6.
Der volle Inhalt der QuelleBarber, Michael J., und Jeff W. Lichtman. „Resolving the Paradoxical Effect of Activity on Synapse Elimination“. In Computational Neuroscience, 131–35. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4831-7_22.
Der volle Inhalt der QuelleGorman, Julia, Konstantin Holzhausen, Joyce Reimer und Jørgen Riseth. „Realizing Synaptic Signal Transmission During Astrocyte-Neuron Interactions within the EMI Framework“. In Computational Physiology, 65–78. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-25374-4_5.
Der volle Inhalt der QuelleBera, Sujoy, Gonca Bayraktar, Katarzyna M. Grochowska, Michelle Melgarejo da Rosa und Michael R. Kreutz. „Activity Dependent Protein Transport from the Synapse to the Nucleus“. In Dendrites, 111–24. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56050-0_5.
Der volle Inhalt der QuelleBorodinsky, Laura N., und Nicholas C. Spitzer. „Mechanisms of Synapse Formation: Activity-Dependent Selection of Neurotransmitters and Receptors“. In Co-Existence and Co-Release of Classical Neurotransmitters, 1–12. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09622-3_3.
Der volle Inhalt der QuelleGraham, Bruce. „Multiple Forms of Activity-Dependent Plasticity Enhance Information Transfer at a Dynamic Synapse“. In Artificial Neural Networks — ICANN 2002, 45–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-46084-5_8.
Der volle Inhalt der QuelleFarsi, Zohreh, und Andrew Woehler. „Imaging Activity-Dependent Signaling Dynamics at the Neuronal Synapse Using FRET-Based Biosensors“. In Methods in Molecular Biology, 261–75. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6688-2_18.
Der volle Inhalt der QuelleVarfolomeev, Sergey, Viktor Bykov und Svetlana Tsybenova. „Kinetic modelling of processes in the cholinergic synapse. Mechanisms of functioning and control methods“. In ORGANOPHOSPHORUS NEUROTOXINS, 127–39. ru: Publishing Center RIOR, 2020. http://dx.doi.org/10.29039/22_127-139.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Synapse activity"
Tsai, Chiou-Tsun, Norihiro Watanabe und Maksim Mamonkin. „313 Enhancing anti-cancer activity of therapeutic T-cells with a synapse-stabilizing receptor“. In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0313.
Der volle Inhalt der QuelleDanaei, Farzaneh, Fariba Bahrami und Mahyar Janahmadi. „Alzheimer's disease can cause epileptic seizure activity in a CA3-CA1 tripartite synapse: A computational study“. In 2014 22nd Iranian Conference on Electrical Engineering (ICEE). IEEE, 2014. http://dx.doi.org/10.1109/iraniancee.2014.6999870.
Der volle Inhalt der QuelleMejias, J. F., J. J. Torres, Joaquín Marro, Pedro L. Garrido und Pablo I. Hurtado. „Memory and pattern storage in neural networks with activity dependent synapses“. In MODELING AND SIMULATION OF NEW MATERIALS: Proceedings of Modeling and Simulation of New Materials: Tenth Granada Lectures. AIP, 2009. http://dx.doi.org/10.1063/1.3082323.
Der volle Inhalt der QuelleSergeeva, Svetlana. „ELECTRICAL SYNAPSES ON NERVE BRANCHES FORM THE REVERBERATION ACTIVITY OF A NEURON“. In XVIII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m2922.sudak.ns2022-18/305.
Der volle Inhalt der QuelleIbanez, Jorge, Haley Houke, Michaela Meehl, Jennifer Ocasio, Nikhil Hebbar, Paulina Velasquez, Suzanne Baker und Giedre Krenciute. „231 Dysfunctional immune synapses restrain anti-DIPG activity of CAR T cells“. In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0231.
Der volle Inhalt der QuelleWerner, T., D. Garbin, E. Vianello, O. Bichler, D. Cattaert, B. Yvert, B. De Salvo und L. Perniola. „Real-time decoding of brain activity by embedded Spiking Neural Networks using OxRAM synapses“. In 2016 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2016. http://dx.doi.org/10.1109/iscas.2016.7539048.
Der volle Inhalt der QuelleYuniati, Anis, und Retno Dwi Astuti. „Neural Network Synchronization of the Morris-Lecar Neuron Model Coupled with Short-Term Plasticity (STP)“. In The 6th International Conference on Science and Engineering. Switzerland: Trans Tech Publications Ltd, 2024. http://dx.doi.org/10.4028/p-ymnn4n.
Der volle Inhalt der QuelleBadica, C., M. Teodorescu, C. Spahiu, A. Badica und C. Fox. „Integrating role activity diagrams and hybrid IDEF for business process modeling using MDA“. In Seventh International Symposium on Symbolic and Numeric Algorithms for Scientific Computing (SYNASC'05). IEEE, 2005. http://dx.doi.org/10.1109/synasc.2005.40.
Der volle Inhalt der QuelleGabrielli, Ângelo, Camila Sousa Bragunce Alves, Bruna Oliveira Bicalho und Débora Pimenta Alves. „Benefits and Challenges of Cannabis Use in the Treatment of Refractory Epilepsy“. In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.239.
Der volle Inhalt der QuelleTsukimata, Márcio Yutaka, Bianca Lumi Inomata da Silva und Jennison Alves Guimarães. „Açaí: potential anticonvulsant agent“. In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.064.
Der volle Inhalt der Quelle