Auswahl der wissenschaftlichen Literatur zum Thema „Susceptibilité au stress“
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Zeitschriftenartikel zum Thema "Susceptibilité au stress"
Tiret, Laurence. „Gene-environment interaction: a central concept in multifactorial diseases“. Proceedings of the Nutrition Society 61, Nr. 4 (November 2002): 457–63. http://dx.doi.org/10.1079/pns2002178.
Der volle Inhalt der QuelleBenajiba, N., J. De Leiris, B. Lyan, A. Derouiche, N. Mokhtar und H. Aguenaou. „Effet de l’huile d’argan sur la contractilité de l’aorte : susceptibilité au stress oxydatif“. Oléagineux, Corps gras, Lipides 13, Nr. 1 (Januar 2006): 76–80. http://dx.doi.org/10.1051/ocl.2006.0076.
Der volle Inhalt der QuelleChakroun Feki, Nozha, Patrice Therond, Pierre Jouannet und Jacques Auger. „Composition lipidique des spermatozoides humains et susceptibilité au stress oxydant avant et après migration dans le mucus cervical“. Andrologie 13, Nr. 4 (Dezember 2003): 381–92. http://dx.doi.org/10.1007/bf03035205.
Der volle Inhalt der QuelleAuxéméry, Y. „L’état de stress post-traumatique comme conséquence de l’interaction entre une susceptibilité génétique individuelle, un évènement traumatogène et un contexte social“. L'Encéphale 38, Nr. 5 (Oktober 2012): 373–80. http://dx.doi.org/10.1016/j.encep.2011.12.003.
Der volle Inhalt der QuellePospichal, A., D. Pokorova, T. Vesely und V. Piackova. „Susceptibility of the topmouth gudgeon (Pseudorasbora parva) to CyHV-3 under no-stress and stress conditions“. Veterinární Medicína 63, No. 5 (29.05.2018): 229–39. http://dx.doi.org/10.17221/88/2017-vetmed.
Der volle Inhalt der QuelleParé, William P. „Prior stress and susceptibility to stress ulcer“. Physiology & Behavior 36, Nr. 6 (Januar 1986): 1155–59. http://dx.doi.org/10.1016/0031-9384(86)90493-2.
Der volle Inhalt der QuelleSalim, Samina, Hesong Liu und Fatin Atrooz. „Early life stress, stress-resilience/susceptibility and oxidative stress“. Free Radical Biology and Medicine 120 (Mai 2018): S165. http://dx.doi.org/10.1016/j.freeradbiomed.2018.04.542.
Der volle Inhalt der QuelleMajlingová, A., M. Závacká und D. Kliment. „An assessment of hucava mountain stream catchment susceptibility to flooding“. Journal of Forest Science 58, No. 12 (11.12.2012): 553–59. http://dx.doi.org/10.17221/75/2011-jfs.
Der volle Inhalt der QuelleEbner, Karl, und Nicolas Singewald. „Individual differences in stress susceptibility and stress inhibitory mechanisms“. Current Opinion in Behavioral Sciences 14 (April 2017): 54–64. http://dx.doi.org/10.1016/j.cobeha.2016.11.016.
Der volle Inhalt der QuellePohl, Tobias, und Hanna Hörnberg. „SOCIAL STRESS DURING ADOLESCENCE: NEUROIMMUNOLOGICAL SIGNATURES OF STRESS SUSCEPTIBILITY“. IBRO Neuroscience Reports 15 (Oktober 2023): S47. http://dx.doi.org/10.1016/j.ibneur.2023.08.2148.
Der volle Inhalt der QuelleDissertationen zum Thema "Susceptibilité au stress"
Merlot, Elodie. „Modulation de la production de cytokines par l'environnement social et susceptibilité aux infections“. Paris, Institut national d'agronomie de Paris Grignon, 2003. https://pastel.archives-ouvertes.fr/pastel-00000790.
Der volle Inhalt der QuelleIn social species, social environment contributes to the development and expression of diseases. Indeed, the social position in a group modulates susceptibility to infections. However, the endocrine and immune mechanisms involved in these differences are not known. Furthermore, instability in the social organisation generates a severe stress of which immune consequences are still controversial. The purposes of this thesis are (1) to describe the influence of social status on the endocrine and immune systems, (2) to specify the effects of social stress on cytokine production and susceptibility to infections and (3) to look for possible sources of variability in the immune consequences of social stress. Excepted a single study on piglets, experiments were performed in mice. Results show an influence of social status. In the absence of stress, dominants show higher basal corticosterone levels and specific response to tuberculin than subordinates. After social defeat, dominants are more affected. Social defeat increases inflammatory reactivity but does not clearly modify the balance between Th1 and Th2 cytokines production and does not affect the development of specific immunity against a mycobacterial infection. Immune effects of social defeat can be observed only when the stress involves significant fights and injuries. Our work also shows that the response to stress depends on individual social experience, in particular on social status, and on the recent immune experience of the individual
Blein, Sophie. „Étude de la variabilité du génome mitochondrial comme facteur de susceptibilité au cancer du sein“. Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10240/document.
Der volle Inhalt der QuelleA large part of the genetic component of breast cancer risk (BCR) is still unexplained. Therefore I studied if variants of the mitochondrial genome (mtDNA) might explain a part of this risk. In fact, mitochondria is the main source of reactive oxygen species (ROS), which contribute to genomic instability and tumor development. As a first axis of research, I studied potential interactions between some nuclear and mitochondrial variants, in conjugation to alcohol consumption. Despite the large dimensions of our dataset, the lack of statistical significant interaction in our data might reveal that former published results that show such interactions were not robust. I also studied if mitochondrial haplogroups could be considered as modificators of known association between BCR and pathogenic mutations in the BRCA1/2 genes. I identified haplogroup T1a1 such as modificator for individuals carrying a mutated BRCA2. Finally, I characterized by NGS mitochondrial genome of women diagnosed for a familial breast cancer, but tested negative for known pathogenic BRCA1/2 mutations. Several variants were identified as potentially damaging. Two genes, MT-ATP6 and MT-CYB are specifically enriched both in terms of distinct variants and in the number of individuals carrying these variants. They are both essential structural components of the mitochondrial respiratory chain, the main ROS production source in the cell. All these analyses contribute to enrich the knowledge about associations between BCR and variability of mtDNA, by integrating questions linked to interactions between genomic variants, environmental exposure, and effect modifications related to mitochondrial haplogroups
Rakotobé, Malalaniaina. „Bases neuro-développementales de la susceptibilité à l’anxiété : apports de l’étude du système habénulo-interpédonculaire“. Electronic Thesis or Diss., Université Côte d'Azur, 2021. http://www.theses.fr/2021COAZ6002.
Der volle Inhalt der QuelleReward circuits of the brain are neuronal networks that modulate positively or negatively the motivation to act according to the situation. They are essential for the survival of the subject and of species by selecting the appropriate behaviors, such as fleeing the danger or searching for food. Dysfunctions of these circuits can lead to mood disorders and addictions. Although these psychiatric disorders can emerge in adulthood, according to the neurodevelopmental hypothesis they can find their origins at a much earlier stage. Indeed, there are periods of development, called critical periods of development, during which genetic and environmental factors influence the maturation of neuronal circuits. A stressful environment and/or defaults in the expression of developmental genes during these critical periods can lead to the malformation of neuronal circuits, including the reward circuits, creating a predisposition to psychiatric disorders. The habenulo-interpeduncular system (HIPS), composed of the medial habenula (MHb) and its main target, the interpeduncular nucleus (IPN), emerges as a key component of the reward circuits. It is notably involved in disorders such as addiction, anxiety and depression. Our team has recently identified the Otx2 transcription factor as an essential genetic factor for the formation of the MHb and IPN. Otx2 expression is maintained in the HIPS throughout life, strongly in the last neurons generated in the MHb (Otx2High neurons) and in neurons of the lateral and central subnucleus of the caudal IPN, suggesting that it might also play a role in the maturation and functioning of the HIPS. We already know that in the ventral tegmental area (VTA), another actor of the reward circuits, a chronic stress, endured during a specific period of childhood decreases the expression level of Otx2 and predisposes to depression and anxiety. It is thus possible that Otx2 might play a similar function in the HIPS response to stress during periods of development. This thesis work aimed to better understand how genetic and environmental factors affecting the development of the HIPS could lead to the development of psychiatric disorders.The first part of this thesis shows that Otx2High neurons of the MHb project on the Otx2+ neurons of the IPN, revealing an Otx2-dependant sub-circuit in the HIPS. The MHb was then chemogenetically activated with the use of DREADD to verify that Otx2High neurons of the MHb are indeed able to modulate the activity of the Otx2+ neurons of the IPN and confirmed the idea that these neurons constitute a sub-circuit of the HIPS, labelled with the expression of Otx2, that we will call the HIPOPS (Habenulo-InterPeduncular-Otx2-Positive-System).In the second part of this project, a critical period of development during which the HIPS is more sensitive to stress has been searched. By using Fos and Egr1, two neuronal markers of activity, we identified a preadolescent period during which the HIPS is more responsive to chronic stress. Interestingly, that chronic stress involves more particularly the activity of the HIPOPS, emphasizing the implication of that sub-circuit in the stress response.In the third part, a “2-hit” stress protocol of chronic stress has been developed and revealed that a chronic stress endured during preadolescence predisposes to anxiety but not to depression in the long-term. In the last part, the effects of Otx2 conditional deletion in the HIPS has been assessed. This study shows that Otx2 deletion in the MHb protects from the deleterious effects of preadolescent chronic stress on anxiety symptoms. This work, therefore, demonstrates that the interaction between environment (stress) and genes (Otx2) during preadolescence determines the susceptibility to develop chronic anxiety in the long term and that one of the cellular support of that susceptibility is the HIPOPS, a circuit lodged inside the HIPS, recognizable by Otx2 expression
Moulin, Sophie. „Rôle de l'hypoxia-inducible factor-1 dans la susceptibilité myocardique à l'ischémie-reperfusion induite par l'hypoxie intermittente“. Thesis, Université Grenoble Alpes (ComUE), 2018. http://www.theses.fr/2018GREAV034/document.
Der volle Inhalt der QuelleObstructive sleep apnea syndrome (OSAS) is a major public health problem that is considered an independent risk factor for the occurrence of myocardial infarction (MI). The cardiovascular alterations associated with OSA are mainly due to the chronic intermittent hypoxia (IH). In particular, activation by IH, the hypoxia-inducible factor-1 (HIF-1) transcription factor likely contributes to enhance myocardial vulnerability to ischemia-reperfusion injury. The aim of this thesis was to study the role of HIF-1 in the mechanisms involved in the increase in MI induced by chronic IH. This work has highlighted two new deleterious consequences of IH exposure, namely endoplasmic reticulum (ER) stress and mitochondrial alterations. Through genetic and/or pharmacological inhibition of HIF-1, we have shown that HIF-1 appears to be a primordial actor in all the deleterious mechanisms of IH, including those discovered during this thesis. HIF-1 also appears to play a major role in the IH-induced increase in MI size. In parallel, its myocardial activation is correlated with the apnea-hypopnea index in apnoeic, compared to non-apnoeic, patients with coronary heart disease. Therefore, HIF-1 activation could serve as a diagnostic marker of OSA in patients with cardiovascular risk. HIF 1 could also be a target for new therapeutic approaches, in complement or replacement of standard treatments
Le, guernic Antoine. „Effets sublétaux d'une contamination métallique liée à des rejets miniers uranifères sur l'épinoche à trois épines (Gasterosteus aculeatus L.). Implication dans la susceptibilité envers un stress biologique“. Thesis, Reims, 2015. http://www.theses.fr/2015REIMS027.
Der volle Inhalt der QuelleUranium extraction has resulted in a remobilisation of this actinide into mine surrounding ecosystems. Uses of metal salts during mining site rehabilitation, and the natural presence of metals have increased the metal contamination in hydrosystems submitted to mine tailings.In situ experiments were conducted in two former French uranium mining sites. Three-spined stickleback caging was used to determine the sublethal effects of this metal mixture on this freshwater fish, as well as its effects on fish susceptibility to a sudden biological stress.This pollution, characterised by higher metal concentrations (especially for uranium), has led to an oxidative stress in sticklebacks visible through several biomarkers, and other effects dependent on the study site. The polymetallic contamination has modified the stickleback responses to the biological stress, by preventing their phagocytic and antioxidant responses. This work has reinforced the interest of the caging technique during environmental studies and that of immunomarkers in a multi-biomarker approach
Pujol, Marine. „Variabilité comportementale en réponse au stress social : influence du variant allélique VGLUT3-p.T8I sur l'acétylcholine et la dopamine dans le noyau accumbens“. Electronic Thesis or Diss., Sorbonne université, 2024. http://www.theses.fr/2024SORUS098.
Der volle Inhalt der QuelleStress is a risk factor for the development of psychiatric conditions such as generalized anxiety and major depressive disorder which are often associated with sleep disturbances. The behavioral adaptation to stress is influenced by a combination of environmental and genetic factors, leading to significant variability among individuals. The nucleus accumbens (NAc) plays a critical role in stress susceptibility and sleep regulation. In the NAc, the behavioral adaptations associated with resilience or susceptibility to stress are modulated by dopamine (DA) and acetylcholine (ACh) signals. The latter is predominantly released by cholinergic interneurons (CINs). Notably, CINs have the ability to signal with both ACh and glutamate through the expression of vesicular acetylcholine transporter (VAChT) and vesicular glutamate transporter 3 (VGLUT3). Our lab recently showed that the VGLUT3-p.T8I allelic variant results in a reduced ACh release from CINs without affecting glutamate release.The objective of my PhD was to elucidate the role of the p.T8I variant in shaping behavioral and sleep responses to stress as well as the stress-associated neuronal ACh and DA dynamics in the NAc. To address these questions, I took advantage of a mouse line carrying the p.T8I variant (VGLUT3T8I/T8I) to study social, anxiety- and depressive-like behaviors as well as sleep-wake patterns in male and female mice both before and after chronic social defeat stress (CSDS) exposure. Then, I measured in vivo ACh and DA dynamics in the NAc using fiber photometry coupled with fluorescent biosensors to assess their release in response to stress. The findings indicate that the p.T8I variant is associated with enhanced social cue discrimination characterized by high target-dependent social avoidance, and elevated anxiety-like levels, accompanied by distinct acute stress-induced sleep changes. Moreover, these behaviors are associated with reduced stress-induced ACh release in the NAc in male mice. Together, these findings reveal the intricate relationship between the p.T8I variant, diminished cholinergic transmission within the NAc and susceptibility to stress
Etain, Bruno. „Contribution à l'étude des facteurs génétiques et environnementaux de susceptibilité aux troubles bipolaires : études du trouble bipolaire à début précoce et des traumatismes affectifs de l’enfance“. Thesis, Paris Est, 2009. http://www.theses.fr/2009PEST0048.
Der volle Inhalt der QuelleBipolar disorders (BD) are psychiatric diseases with a complex determinism in which genetic and environmental susceptibility factors are involved. Attempts to identify genetic factors have produced conflicting results and environmental factors remain unknown. Early-onset bipolar disorder (EO-BD) is a clinical entity that is characterized by a strong familial aggregation ; a specific focus on this subtype might facilitate the identification of susceptibility genes. A genome-wide scan in EO-BD has suggested eight regions of linkage (chromosomal regions 2p21, 2q14, 3p14, 5q33, 7q36, 10q23, 16q23 and 20p12). We have demonstrated an association between EO-BD and the SNAP25 gene (located at 20p12 and involved in exocytosis). We have demonstrated an association between BD and the ASMT gene (involved in the synthesis of melatonin). Finally, a genome-wide association study has suggested the involvement of two phosphatidyl-inositol pathway related genes in the susceptibility to EO-BD (PLEKHA5 et PLCXD3). Concerning environmental susceptibility factors, childhood affective traumatic events (mainly emotional abuse) are associated with BD, might influence two core dimensions of BD (affective lability and affect intensity) and might interact with the serotonin transporter genelinked polymorphic region to modulate the age of onset of the disorder. These studies illustrate the relevance of focusing on the early onset subgroup of the disease to identify susceptibility genes, the need to further explore early stressors as environmental factors associated with BD and to investigate the complex relationships between these two kinds of susceptibility factors
Jones, C. A. „A study of stress susceptibility in pigs“. Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372043.
Der volle Inhalt der QuelleThomas, Gladys Ruth. „Susceptibility of Brugia malayi to oxidative and nitrosative stress“. Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267808.
Der volle Inhalt der Quelle鍾慧虹 und Wai-hung Angela Chung. „Cognitive vulnerability, stress, and postpartum depressive symptomatology: a prospective study in HongKong“. Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B37101055.
Der volle Inhalt der QuelleBücher zum Thema "Susceptibilité au stress"
Gillis, A. R. Cultural susceptibility to crowding: An empirical analysis. Toronto: Centre for Urban and Community Studies, University of Toronto, 1986.
Den vollen Inhalt der Quelle findenLyle, Fred F. Stress-corrosion cracking susceptibility of weldments in duplex stainless steels. St. Louis, Missouri: Materials Technology Institute of the Chemical Process Industries, 1989.
Den vollen Inhalt der Quelle findenMadra, Moneek. Interactions between the BDNF (Val66Met) variant and stress as a model of susceptibility to Anorexia Nervosa. [New York, N.Y.?]: [publisher not identified], 2015.
Den vollen Inhalt der Quelle findenInternational Berzelius Symposium. (6th 1985 Malmö, Sweden). Social support--health and disease. Herausgegeben von Isacsson Sven-Olof, Janzon Lars und Sweden. Delegationen för social forskning. Stockholm, Sweden: Almqvist & Wiksell International, 1986.
Den vollen Inhalt der Quelle findenJ, Sholar Clyde. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. Louisville, Ky. (2301 Bradley Ave., Louisville 40217): U.S. Dept. of the Interior, U.S. Geological Survey, 1991.
Den vollen Inhalt der Quelle findenSholar, Clyde J. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. Louisville, Ky. (2301 Bradley Ave., Louisville 40217): U.S. Dept. of the Interior, U.S. Geological Survey, 1991.
Den vollen Inhalt der Quelle findenB, Ludvigsen J., European Association for Animal Production. Commission on Animal Management and Health., European Association for Animal Production. Commission on Pig Production. und European Association for Animal Production. Meeting, Hrsg. Stress susceptibility and meat quality in pigs: Proceeding of Commis[s]ion on Animal Management and Health and Commission on Pig Production joint session, Halkidiki, Greece, 30. Sept.-5. Oct. 1985. [Rome?]: European Association for Animal Production, 1985.
Den vollen Inhalt der Quelle findenL, Power Michael, und Schulkin Jay, Hrsg. Birth, distress, and disease: Placental-brain interactions. Cambridge: Cambridge University Press, 2005.
Den vollen Inhalt der Quelle findenTsai, Jack, Natalie Jones, Robert H. Pietrzak, Ilan Harpaz-Rotem und Steven M. Southwick. Susceptibility, Resilience, and Trajectories. Herausgegeben von Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad und Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0019.
Der volle Inhalt der QuelleWho Gets Ptsd?: Issues of Posttraumatic Stress Vulnerability. Charles C. Thomas Publisher, 2006.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Susceptibilité au stress"
Miroshnychenko, Mykola, Oleksandr Kruglov, Pavlo Nazarok und Stanislav Kovalenko. „Identification of the Structure of Soil Cover by Magnetic Susceptibility“. In Soils Under Stress, 57–68. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68394-8_6.
Der volle Inhalt der QuelleMohanty, Prasanna, Vladimir D. Kreslavski, Vyacheslav V. Klimov, Dmitry A. Los, Mamoru Mimuro, Robert Carpentier und Suleyman I. Allakhverdiev. „Heat Stress: Susceptibility, Recovery and Regulation“. In Photosynthesis, 251–74. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1579-0_12.
Der volle Inhalt der QuelleLaudenslager, Mark L. „Psychosocial Stress and Susceptibility to Infectious Disease“. In Viruses, Immunity, and Mental Disorders, 391–402. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1799-9_38.
Der volle Inhalt der QuelleNguyen, Chi Cong, Phuoc Vo, Viet Long Doan, Quang Binh Nguyen, Tien Cuong Nguyen und Quoc Dinh Nguyen. „Assessment of the Effects of Rainfall Frequency on Landslide Susceptibility Mapping Using AHP Method: A Case Study for a Mountainous Region in Central Vietnam“. In Progress in Landslide Research and Technology, Volume 1 Issue 2, 2022, 87–98. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-18471-0_7.
Der volle Inhalt der QuelleHaycock, J. W., G. Falkous und D. Mantle. „Differential susceptibility of skeletal muscle proteins to free radical-induced oxidative damage in vitro“. In Oxidative Stress in Skeletal Muscle, 157–76. Basel: Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8958-2_10.
Der volle Inhalt der QuelleJiles, D. C., und P. Garikepati. „Detection of Stress in Steels from Differential Magnetic Susceptibility“. In Nondestructive Characterization of Materials, 524–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-84003-6_62.
Der volle Inhalt der QuelleKathirvel, Paramasivam, und Alan L. Archibald. „The Halothane Gene, Leanness and Stress Susceptibility in Pigs“. In Animal Models — Disorders of Eating Behaviour and Body Composition, 173–90. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9662-6_10.
Der volle Inhalt der QuelleErzagian, Egy, Wahyu Wilopo und Teuku Faisal Fathani. „Landslide Susceptibility Zonation Using GIS-Based Frequency Ratio Approach in the Kulon Progo Mountains Area, Indonesia“. In Progress in Landslide Research and Technology, 115–26. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-44296-4_3.
Der volle Inhalt der QuelleMagiri, Royford, Kaampwe Muzandu, George Gitau, Kennedy Choongo und Paul Iji. „Impact of Climate Change on Animal Health, Emerging and Re-emerging Diseases in Africa“. In African Handbook of Climate Change Adaptation, 1835–51. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-45106-6_19.
Der volle Inhalt der QuelleStephenson, Kale J., Yugo Ashida, Jeremy T. Busby und Gary S. Was. „Stress Corrosion Crack Initiation Susceptibility of Irradiated Austenitic Stainless Steels“. In Proceedings of the 15th International Conference on Environmental Degradation of Materials in Nuclear Power Systems — Water Reactors, 1187–201. Cham: Springer International Publishing, 2011. http://dx.doi.org/10.1007/978-3-319-48760-1_72.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Susceptibilité au stress"
Jindal, Rajni, Chahat Kumar, Gaurav Jawla und Harshit Goyal. „Predicting Susceptibility to Covid Stress Using Data Mining“. In 2022 International Conference on Emerging Smart Computing and Informatics (ESCI). IEEE, 2022. http://dx.doi.org/10.1109/esci53509.2022.9758247.
Der volle Inhalt der QuelleBayles, Robert A., R. K. Singh Raman, Steven P. Knight und Jy-An Wang. „Evaluating Stress-Corrosion Cracking Susceptibility Using a Torsion Test“. In ASME 2005 Pressure Vessels and Piping Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/pvp2005-71782.
Der volle Inhalt der QuelleFernandes, Carlos, und Osimar Simiao. „Methodology development for pipeline stress corrosion cracking susceptibility evaluation“. In Rio Pipeline Conference and Exhibition. IBP, 2023. http://dx.doi.org/10.48072/2447-2069.rpc.2023.187.
Der volle Inhalt der Quelle„Distinguishing Effect of Buffing Operation on Surface Residual Stress Distribution and Susceptibility of 304L SS and 321 SS Welds to Chloride Induced SCC“. In Residual Stresses 2018. Materials Research Forum LLC, 2018. http://dx.doi.org/10.21741/9781945291890-22.
Der volle Inhalt der QuelleRund, C. O. „Development of a Miniature Portable X-Ray Instrument for the Non-Destructive Measurement of Stresses“. In ASME/JSME 2004 Pressure Vessels and Piping Conference. ASMEDC, 2004. http://dx.doi.org/10.1115/pvp2004-2825.
Der volle Inhalt der QuelleKuzyk, M. G., K. D. Singer, H. E. Zahn und L. A. King. „Controlling the Second Order Nonlinear Optical Tensor Properties of Poled Films With Stress“. In Nonlinear Optical Properties of Materials. Washington, D.C.: Optica Publishing Group, 1988. http://dx.doi.org/10.1364/nlopm.1988.wa3.
Der volle Inhalt der QuelleChen, Bo, Michael W. Spindler, David J. Smith und Peter E. J. Flewitt. „Effect of Thermo-Mechanical History on Reheat Cracking in 316H Austenitic Stainless Steel Weldments“. In ASME 2010 Pressure Vessels and Piping Division/K-PVP Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/pvp2010-25088.
Der volle Inhalt der Quelle„Early life stress leads to enhanced susceptibility to social stress in adult male C57BL/6 mice“. In SYSTEMS BIOLOGY AND BIOINFORMATICS. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/sbb-2019-16.
Der volle Inhalt der QuelleZiomek-Moroz, M., und J. Flis. „Susceptibility of Carbon Steel to Stress Corrosion Cracking in Sodium Hydroxide“. In ASME 2005 Pressure Vessels and Piping Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/pvp2005-71783.
Der volle Inhalt der QuelleMizutani, T., A. Sato, A. Watanabe, Y. Hamakawa, K. Uemasu, N. Tanabe, S. Sato und T. Hirai. „Susceptibility to Oxidative Stress Characterizes Phenotypes in Murine Model of BPD“. In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5488.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Susceptibilité au stress"
Christman. L51577 Prediction of SCC Susceptibility Based on Mechanical Properties of Line Pipe Steels. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), August 1988. http://dx.doi.org/10.55274/r0010278.
Der volle Inhalt der QuelleColleen Shelton-Davis. Fuel Canister Stress Corrosion Cracking Susceptibility Experimental Results. Office of Scientific and Technical Information (OSTI), März 2003. http://dx.doi.org/10.2172/911541.
Der volle Inhalt der QuelleLeis. L51838 Cyclic Stress Strain Behavior and SCC Susceptibility of Line Pipe Steels. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), März 2001. http://dx.doi.org/10.55274/r0010355.
Der volle Inhalt der QuelleKoch, Beavers und Berry. L51491 Effect of Temperature on Stress-Corrosion Cracking of Pre-Cracked Line-Pipe Steel. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), Februar 1985. http://dx.doi.org/10.55274/r0010078.
Der volle Inhalt der QuelleRahman, M. M., und K. N. Strafford. USA09-01 Appraisal and Assessment of Factors Involved in the Maintenance of the Integrity of Pipelines. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), September 1992. http://dx.doi.org/10.55274/r0011770.
Der volle Inhalt der QuelleSubramanian, K. STRESS CORROSION CRACKING SUSCEPTIBILITY OF HIGH LEVEL WASTE TANKS DURING SLUDGE MASS REDUCTION. Office of Scientific and Technical Information (OSTI), Oktober 2007. http://dx.doi.org/10.2172/920661.
Der volle Inhalt der QuellePsaila-Dombrowski, M. J., W. A. Van Der Sluys und B. P. Miglin. GRI-97-0001 Investigation of Pipeline Stress Corrosion Cracking Under Controlled Chemistry Conditions. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), August 1997. http://dx.doi.org/10.55274/r0012043.
Der volle Inhalt der QuelleDenys und Lefevre. L52229 SCC Avoidance in Ethanol Pipelines. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), März 2005. http://dx.doi.org/10.55274/r0011363.
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