Thematische Bibliographien / Structural cell model

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Structural cell model“

Autor: Grafiati
Veröffentlicht am 28. Juni 2021
Zuletzt aktualisiert am 18. Februar 2022

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Zeitschriftenartikel zum Thema "Structural cell model"

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Watanabe, Hiroshi C., Kai Welke, Franziska Schneider, Satoshi Tsunoda, Feng Zhang, Karl Deisseroth, Peter Hegemann und Marcus Elstner. „Structural Model of Channelrhodopsin“. Journal of Biological Chemistry 287, Nr. 10 (11.01.2012): 7456–66. http://dx.doi.org/10.1074/jbc.m111.320309.

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Drits, V. A., B. A. Sakharov, A. L. Salyn und A. Manceau. „Structural Model for Ferrihydrite“. Clay Minerals 28, Nr. 2 (Juni 1993): 185–207. http://dx.doi.org/10.1180/claymin.1993.028.2.02.

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AbstractThe structure of 6-line and 2-line ferrihydrite (Fh) has been reconsidered. X-ray diffraction (XRD) curves were first simulated for the different structural models so far proposed, and it is shown that neither of these corresponds to the actual structure of ferrihydrite. On the basis of agreement between experimental and simulated XRD curves it is shown that Fh is a mixture of three components: (i) Defect-free Fh consisting of anionic ABACA . . . close packing in which Fe atoms occupy only octahedral sites with 50% probability; the hexagonal unit-cell parameters are a = 2-96 Å and c = 9-40 Å, and the space group is P1c. (ii) Defective Fh in which Ac1Bc2A and Ab1Cb2A structural fragments occur with equal probability and alternate completely at random; Fe atoms within each of these fragments have identical ordered distribution with in the hexagonal super-cell with a = 5.26 Å. (iii) Ultradispersed hematite with mean dimension of coherent scattering domains (CSD) of 10-20 Å. The main structural difference between 6-line and 2-line Fh is the size of their CSD which is extremely small for the latter structure. Nearest Fe-Fe distances calculated for this new structural model are very close to those determined by EXAFS spectroscopy on the same samples.
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Wu, D. L. „Three-cell model and 5D braided structural composites“. Composites Science and Technology 56, Nr. 3 (Januar 1996): 225–33. http://dx.doi.org/10.1016/0266-3538(95)00136-0.

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Rozhdestvenskaya, I. V., T. Kogure, E. Abe und V. A. Drits. „A structural model for charoite“. Mineralogical Magazine 73, Nr. 5 (Oktober 2009): 883–90. http://dx.doi.org/10.1180/minmag.2009.073.2.883.

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AbstractThe crystal structure of charoite was investigated mainly by using selected-area electron diffraction (SAED), X-ray diffraction (XRD) and high-resolution electron microscopy (HREM). SAED and XRD patterns indicate that the structure has a monoclinic cell: a = 32.296, b = 19.651, c = 7.16 Å, β = 96.3° and V = 4517 Å3. The space group inferred from systematic absences and HREM images is P21/m. A model of the charoite structure is proposed that is based on the features of related Ca-alkaline silicate structures and HREM images. The structure of charoite consists of three different silicon-oxygen radicals (polymerized SiO4 tetrahedra) which are located between Ca polyhedra. Two of these radicals form continuous tubular structures comprising pectolite-like tetrahedral chains. Calcium polyhedra are joined to form blocks, each of which consists of four columns sharing edges and apices. Potassium and H2O molecules are probably located inside the tubular silicate radicals. From these results, a general formula is derived: K6-7(Ca,Na)18[(Si6O17)(Si12O30)(Si18O45)](OH,F)2.nH2O with two formula units in the unit cell (Z = 2).
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Ptitsyn, O. B., A. V. Finkelstein und A. G. Murzin. „Structural model for interferons“. FEBS Letters 186, Nr. 2 (08.07.1985): 143–48. http://dx.doi.org/10.1016/0014-5793(85)80697-9.

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Kaduk, James A., und Thomas N. Blanton. „An improved structural model for cellulose II“. Powder Diffraction 28, Nr. 3 (23.04.2013): 194–99. http://dx.doi.org/10.1017/s0885715613000092.

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A sample of cellulose II, prepared by deacetylation of cellulose acetate, has permitted more precise determination of the unit-cell parameters by the Rietveld method. Cellulose II is monoclinic, with space group P21c-axis unique (or P1121) (No. 4) and refined unit-cell parameters a = 8.076(13), b = 9.144(10), c = 10.386(20) Å, γ = 117.00(8)°, and V = 683.5(18) Å3. A density functional geometry optimization using these fixed unit-cell parameters has resulted in an improved structural model for cellulose II. A powder pattern calculated from this new model has been submitted to the ICDD for inclusion in future releases of the Powder Diffraction File.
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Ishida, Hideki, und Yoshinobu Shigenaka. „Cell model contraction in the ciliatespirostomum“. Cell Motility and the Cytoskeleton 9, Nr. 3 (1988): 278–82. http://dx.doi.org/10.1002/cm.970090310.

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Jones, P. M., und A. M. George. „A New Structural Model for P-Glycoprotein“. Journal of Membrane Biology 166, Nr. 2 (15.11.1998): 133–47. http://dx.doi.org/10.1007/s002329900455.

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Alaimo, Andrea, Federico Marino und Stefano Valvano. „BCC lattice cell structural characterization“. Reports in Mechanical Engineering 2, Nr. 1 (26.04.2021): 77–85. http://dx.doi.org/10.31181/rme200102077v.

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In this work, a numerical characterization of BCC lattice cells is performed through the use of an homogenization approach. The main goal is to establish a relationship among those properties and the relative density of the cubic unit cell. The BCC cell struts diameter are the inputs parameters of the homogenization analysis campaing in order to vary the relative density of the unit cell. A linear periodic condition has been applied to the model in order to simulate a clear probing situation. Traction load tests are used in order to evaluate the Young modulus and the Poisson coefficient, differently a pure shear load case is employed for the evaluation of the shear modulus. Hence the final results will be presented in a graphic visualization.
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Rosich, Albert, Fatiha Nejjari und Ramon Sarrate. „Fuel Cell System Diagnosis based on a Causal Structural Model“. IFAC Proceedings Volumes 42, Nr. 8 (2009): 534–39. http://dx.doi.org/10.3182/20090630-4-es-2003.00089.

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Dissertationen zum Thema "Structural cell model"

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Dennison, Kelly J. „Development of a structural model of human T-cell leukemia virus type-I protease“. Thesis, Georgia Institute of Technology, 2002. http://hdl.handle.net/1853/30060.

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Granja, Vasquez Jochen. „Analysis of Secreted Phosphoprotein-24 and its Effects During Osteoblast Differentiation in a Mesenchymal Stem Cell Model“. VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1884.

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Musculoskeletal diseases, in particular osteoporosis, are increasingly becoming more prevalent in the U.S. due to the ageing population (Figure1). It is estimated that one-sixth of 300 million people in U.S. suffer from bone disorders or loss. About 10 million of those people above age 50 suffer from osteoporosis. Patients that suffer from osteoporosis have high morbidity and mortality rates. For instance, patients have decreased bone mineral density (BMD), a measurement of bone density that reflects the strength of bone as represented by calcium content. A decrease in BMD typically leads to an increased risk of bone fractures. In particular, hip fractures have an associated 20% mortality rate 1 year after injury among senior citizens 1. Patients that suffer from musculoskeletal diseases and from bone injuries, not associated with disease, account for 130 million hospital visit per year. Not to mention, 245 billion dollars of healthcare expenditure 2. Over that last 30 years, there has been much improvement in the field of bone research and its application to medicine. It has changed the quality of life and prolonged the life expectancy of patients suffering from bone disease. However, many details remain unknown about the underlying mechanism that control bone metabolism, formation, and healing. Furthermore, current effective therapies to combat bone disorders have limitations including unwanted side effects and prohibitive costs. For example, treatment with glucocorticoids which is a known inducer of osteoblastogenesis in vitro has been shown to produce an osteoporotic phenotype in vivo. Recognizing the importance of bone health and its affordability to the public makes the advancement of therapeutic targets work worth doing. Work in this field will eventually lead to the prevention, treatment, and cure for bone disease. A potential therapeutic candidate that maybe involved directly or indirectly with bone formation is secreted phosphoprotein-24 (Spp24). The following research aims to establish an importance and role for Spp24 in bone differentiation. A novel antibody that detects Spp24 which we have developed and characterized, has allowed us to feasibly study the protein. Our results demonstrate localization of Spp24 in different tissue, the processing of the protein during osteoblastogenesis, and have allowed us to conceptualize possible functions based on our data.
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Maluš, Miroslav. „Komplexní model turbulence pro různé velikosti cel“. Master's thesis, Vysoké učení technické v Brně. Fakulta elektrotechniky a komunikačních technologií, 2021. http://www.nusl.cz/ntk/nusl-442416.

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This diploma thesis is about creating a program to model turbulent cells of different sizes on the chosen transmission path. The initial part of the work is devoted to the formation of atmospheric turbulence and the mathematical description of the extent of turbulence and its effect on optical waves. The methods of the turbulence generation and their physical description of formation are described below. The practical part is devoted to the created program in the MATLAB.
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Santana, Bonilla Alejandro, Rafael Gutierrez, Sandonas Leonardo Medrano, Daijiro Nozaki, Alessandro Paolo Bramanti und Gianaurelio Cuniberti. „Structural distortions in molecular-based quantum cellular automata: a minimal model based study“. Royal Society of Chemistry, 2014. https://tud.qucosa.de/id/qucosa%3A36371.

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Molecular-based quantum cellular automata (m-QCA), as an extension of quantum-dot QCAs, offer a novel alternative in which binary information can be encoded in the molecular charge configuration of a cell and propagated via nearest-neighbor Coulombic cell–cell interactions. Appropriate functionality of m-QCAs involves a complex relationship between quantum mechanical effects, such as electron transfer processes within the molecular building blocks, and electrostatic interactions between cells. The influence of structural distortions of single m-QCA are addressed in this paper within a minimal model using an diabatic-to-adiabatic transformation. We show that even small changes of the classical square geometry between driver and target cells, such as those induced by distance variations or shape distortions, can make cells respond to interactions in a far less symmetric fashion, modifying and potentially impairing the expected computational behavior of the m-QCA.
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Minassian, Anuka [Verfasser], und Peter [Gutachter] Kloppenburg. „Stem Cell Therapy For Stroke. Modulation of structural and functional adjustments after stem cell implantation in a mouse model of cortical stroke / Anuka Minassian ; Gutachter: Peter Kloppenburg“. Köln : Universitäts- und Stadtbibliothek Köln, 2018. http://d-nb.info/1200096991/34.

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Börsum, Jakob. „Estimating Causal Effects Of Relapse Treatment On The Risk For Acute Myocardial Infarction Among Patients With Diffuse Large B-Cell Lymphoma“. Thesis, Uppsala universitet, Statistiska institutionen, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-447241.

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This empirical register study intends to estimate average causal effects of relapse treatment on the risk for acute myocardial infarction (AMI) among patients with Diffuse B-Cell Lymphoma (DLBCL) within the potential outcome framework. The report includes a brief introduction to causal inference and survival anal- ysis and mentions specific causal parameters of interest that will be estimated. A cohort of 2887 Swedish DLBCL patients between 2007 and 2014 were included in the study where 560 patients suffered a relapse. The relapse treatment is hypothesised to be cardiotoxic and induces an increased risk of heart diseases. The identifiability assumptions need to hold to estimate average causal effects and are assessed in this report. The patient cohort is weighted using inverse probability of treatment and censoring weights and potential marginal survival curves are estimated from marginal structural Cox models. The resulting point estimate indicates a protective causal effect of relapse treatment on AMI but estimated bootstrap confidence intervals suggest no significant effect on the 5% significance level.
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Mason, Nena Lundgreen. „The Anatomy of Porcine and Human Larynges: Structural Analysis and High Resolution Magnetic Resonance Imaging of the Recurrent Laryngeal Nerve“. BYU ScholarsArchive, 2015. https://scholarsarchive.byu.edu/etd/5783.

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The recurrent laryngeal nerve (RLN) innervates all the intrinsic muscles of the larynx that are responsible for human vocalization and language. The RLN runs along the tracheoesophageal groove bilaterally and is often accidentally damaged or transected during head and neck surgical procedures. RLN palsy and vocal cord paralysis are the most common and serious post op complications of thyroid surgeries. Patients who suffer from RLN injury can develop unilateral or bilateral vocal fold paralysis (BVFP). Theoretically, selective reinnervation of the posterior cricoarytenoid muscle would be the best treatment for BVFP. The phrenic nerve has been shown in several studies to be the best candidate to anastomose to the distal end of a severed RLN to restore glottal abduction. Successful PCA reinnervation has been sporadically achieved in both human patients and in animal models. Another notable ramification of recurrent laryngeal nerve injury is vocal instability caused by the alteration of mechanical properties within the larynx. In phonosurgery, alterations to the position and framework of the laryngeal apparatus are made to improve voice quality. Accurate and realistic synthetic models are greatly needed to predict the outcome of various adjustments to vocal cord tension and position that could be made surgically. Despite the sporadically successful attempts at PCA reinnervation, thus far, there are still several deficits in our anatomical familiarity and technological capability, which hinder the regularity of successful PCA reinnervation surgeries and our capacity to generate synthetic models of the human larynx that are both realistic and functional. We will address three of these deficits in this project using the porcine larynx as a model. Firstly, we will identify the anatomical variations of the porcine recurrent laryngeal nerve branches. A microscribe digitizer will be used to create three-dimensional mapping of the recurrent laryngeal nerve branches that are relevant to the posterior cricoarytenoid muscle and the abduction of the vocal folds. Secondly, we will develop a magnetic resonance imaging technique to correlate recurrent laryngeal nerve branching patterns with high-resolution MR images that can be used to determine the branching patterns present in a given specimen without surgery. Lastly, we will determine the distribution and composition of different tissue types found within human vocal folds. High resolution MRI, and Mallory's trichrome and H&E histological staining will be used to distinguish and identify the tissue composition of the vocal folds and surrounding laryngeal structures. Detailed information regarding vocal fold tissue composition and histological geometry will enable laryngeal modelers to select more sophisticated and life-like materials with which to construct synthetic vocal fold models.
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Silparasetty, Shobha Lavanya. „Cloning of "Animal Cryptochrome" cDNA from the Model Organism CHLAMYDOMONAS REINHARDTII for Functional Analysis of Its Protein Product“. TopSCHOLAR®, 2009. http://digitalcommons.wku.edu/theses/117.

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reinhardtii, a unicellular green alga, is a model organism to study the circadian clock. Cryptochromes are the blue light photoreceptors that entrain the clock in some organisms. The CPH1 protein of C. reinhardtii resembles the cryptochromes of the plant model Arabidopsis, but whether CPH1 entrains the circadian clock in C. reinhardtii is not yet known. Recent reports have suggested the existence of one more cryptochrome in C. reinhardtii, which resembles the cryptochromes of animals. However, the amino acid sequence of this protein shows even higher sequence similarity with the 6-4 DNA photolyase of Arabidopsis. DNA photolyases are involved in the repair of UV light-induced DNA damage using the energy of blue light. In order to determine, if the “animal cryptochrome” gene of C. reinhardtii actually encodes a 6-4 DNA photolyase rather than a photoreceptor, an experimental design was developed to test whether the protein product is able to rescue an E. coli mutant defective in its DNA photolyase gene. The design is as follows: In a first step, the coding region of the “animal cryptochrome” cDNA is cloned. In a second step, the cDNA is inserted in-frame into an E. coli expression vector. In a third step, the construct is transformed into an E. coli photolyase mutant, its expression induced, and the strain tested for better survival after UV light exposure. To accomplish the first step, the cloning of “animal cryptochrome” cDNA, total RNA was successfully extracted from C. reinhardtii 4 hrs into the light phase of a 12 h light/12 h dark cycle and reverse transcribed into cDNA using oligo(dT) primers. After initially unsuccessful attempts at amplifying animal cryptochrome from cDNA or genomic template with a variety of primers and conditions, a short fragment with the expected size of 186 bp was amplifiable with both templates. However, even this fragment was not reliably obtained in every PCR assay. Because of this difficulty, real-time PCR was finally performed in the presence of DMSO (Dimethylsulfoxide) and Betaine. These two adjuvants were reported to improve amplifications particularly for GC-rich templates. C. reinhardtii DNA is especially GC-rich with an average of 64% Gs and Cs. The improved conditions allowed the reliable amplification of the 186 bp fragment from genomic template. It also enabled the amplification of a larger fragment of 528 bp from the same template. The results suggest that a combination of 5% DMSO and 1M Betaine is optimal for the amplification of C. reinhardtii DNA and thus can serve as the basis for successful amplification of the entire 1788 bp coding region of the animal cryptochrome cDNA.
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Orlová, Lucie. „Výpočtové modelování mechanických zkoušek živočišné buňky“. Master's thesis, Vysoké učení technické v Brně. Fakulta strojního inženýrství, 2021. http://www.nusl.cz/ntk/nusl-443747.

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Předkládaná diplomová práce se zabývá stavbou živých živočišných buněk a jejich odezvou na mechanické zatěžování. Zobecněným zaměřením práce je popis mechanického chování buňky nejenom ve fyziologickém, ale i v patologickém stavu. Výchozím předpokladem pro úspěšné řešení zadané úlohy je vysoce interdisciplinární přístup kombinující výpočtové přístupy mechaniky těles (v~tomto případě metodu konečných prvků) s lékařským výzkumem. Nejdůležitějším bodem při tvorbě výpočtového modelu, pomocí něhož je možné aproximovat chování živé buňky při zatížení, je zejména identifikace mechanicky významných komponent a~jejich materiálových parametrů. V tomto případě jsou jako mechanicky význačné identifikovány spojité součásti jádro, membrána a cytoplazma, které jsou nově propojeny s prvky diskrétními (mitochondriální sítí) v hybridním modelu, jehož platnost je ověřena pomocí experimentálních dat. Tento model slouží jako podklad k vyhodnocení míry vlivu mitochondrií na celkovou tuhost buňky.
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Yamamoto, Akihisa. „Mesoscopic structural dynamics and mechanics of cell membrane models“. 京都大学 (Kyoto University), 2015. http://hdl.handle.net/2433/198928.

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Bücher zum Thema "Structural cell model"

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Wilson, John W. Multiple lesion track structure model. Hampton, Va: Langley Research Center, 1992.

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Ponnamperuma, Cyril, und Julian Chela-Flores, Hrsg. Chemical Evolution: Structure and Model of the First Cell. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0105-9.

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1923-, Ponnamperuma Cyril, Chela-Flores Julian, Unesco und Trieste Conference on Chemical Evolution (3rd : 1994), Hrsg. Chemical evolution--the structure and model of the first cell. Dordrecht: Kluwer Academic Publishers, 1995.

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Michael, Kraus. Structured biological modelling: A new approach to biophysical cell biology. Boca Raton: CRC Press, 1995.

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Mesgari, M. Saadi. Topological cell-tuple structures for three-dimensional spatial data. [Netherlands: University of Twente], 2000.

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Chen, Luonan. Modeling biomolecular networks in cells: Structures and dynamics. London: Springer, 2010.

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Jafferali, R. A stochastic model to simulate the structure and performance of microfiltration and the growth of animal cell cultures. Manchester: UMIST, 1995.

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Roux, Benoît. Molecular machines. Hackensack, NJ: World Scientific, 2011.

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Ignatʹev, V. A. Thin-walled cellular structures: Methods for their analysis. Rotterdam: Balkema, 1999.

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Real-time biomolecular simulations: The behavior of biological macromolecules from a cellular systems perspective. New York: McGraw Hill, 2007.

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Buchteile zum Thema "Structural cell model"

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Folan, J. C. „Paraganglionic Cell Response to Chronic Imipramine: A Structural Model“. In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia, 266–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_46.

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Calvete, Juan José. „Elements for a Structural/Functional Model of Human Platelet Plasma Membrane Fibrinogen Receptor, the Glycoprotein IIb/IIIa Complex (Integrin αIIb/β3)“. In Cell Adhesion Molecules, 63–91. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2830-2_6.

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Miki, Masao. „Structural Changes Between Regulatory Proteins and Actin: A Regulation Model by Tropomyosin-Troponin Based on FRET Measurements“. In Results and Problems in Cell Differentiation, 191–203. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-540-46558-4_14.

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Bentz, Joe, und Harma Ellens. „A Structural Model for the Mass Action Kinetic Analysis of P-gp Mediated Transport Through Confluent Cell Monolayers“. In Methods in Molecular Biology, 289–316. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-758-7_14.

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Gao, Fei, Benjamin Blunier und Abdellatif Miraoui. „Model Structural and Functional Approaches“. In Proton Exchange Membrane Fuel Cells Modeling, 49–52. Hoboken, NJ USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118562079.ch6.

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McCarthy, James B., Daniel J. Mickelson, Cynthia G. Fields und Gregg B. Fields. „The use of collagen-model peptides to correlate collagen primary and secondary structural effects with the mechanisms of tumor cell adhesion, motility and invasion“. In Peptides 1992, 109–10. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1470-7_38.

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Dyson, Janet, und Glenn F. Webb. „A Cell Population Model Structured by Cell Age Incorporating Cell–Cell Adhesion“. In Mathematical Oncology 2013, 109–49. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0458-7_4.

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Bansaye, Vincent, und Sylvie Méléard. „Splitting Feller Diffusion for Cell Division with Parasite Infection“. In Stochastic Models for Structured Populations, 79–87. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21711-6_8.

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Terashima, N., K. Fukushima, L.-F. He und K. Takabe. „Comprehensive Model of the Lignified Plant Cell Wall“. In Forage Cell Wall Structure and Digestibility, 247–70. Madison, WI, USA: American Society of Agronomy, Crop Science Society of America, Soil Science Society of America, 2015. http://dx.doi.org/10.2134/1993.foragecellwall.c10.

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Hansen-Goos, Hendrik, und Seth Lichter. „Geometric Models of Protein Secondary-Structure Formation“. In Nano and Cell Mechanics, 411–35. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118482568.ch16.

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Konferenzberichte zum Thema "Structural cell model"

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Agmon, Eran, James A. Glazier und Randall D. Beer. „Structural coupling of a Potts model cell“. In Proceedings of the 14th European Conference on Artificial Life ECAL 2017. Cambridge, MA: MIT Press, 2017. http://dx.doi.org/10.7551/ecal_a_008.

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Venkatesan, Vidyashankar, und Nilay Mukherjee. „Finite Element Model of a Cell Incorporating Cell Membrane, Cytoskeletal Structure and Intracellular Fluid Pressure“. In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32667.

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Compressive loading is intrinsic to certain tissues in our body like articular cartilage and bone (1). In situ experiments in cartilage suggest that chondrocytes can undergo significant deformation due to compressive loading on the tissue (2). In situ and isolated cell experiments have concluded that cells are quite resilient to compressive loading, aspiration etc. and exhibit a moduli in the range of 0.6 to 2 kPa (3). However, few studies have attempted to understand the compressive behavior of cells in terms of its structural components. The structural components of a cell consist of a membrane and a dense network of at least three elements (actin, microtubules and intermediate filaments). Using finite element analysis techniques we wanted to explore the role of these structural components in determining the ability of the cell to withstand compression.
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3

Coghlan, Karen M., Patrick McGarry, Mohammad R. K. Mofrad und Peter E. McHugh. „Development of a Discrete Finite Element Cell Model“. In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176734.

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Computational models have proven useful in the study of cell mechanics and mechanotransduction. While most finite element (FE) models of cells are commonly described in terms of the laws of continuum mechanics, a model that can accurately represent the microstructure of the filamentous network of the cytoskeleton would be required to relate mechanics to biology at the microscale level. An alternative approach to a continuum is presented here, whereby the discrete nature of the cytoskeleton of the cell is emphasized and the known structural properties of the cytoskeleton of the cell are utilized.
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Inoue, Gen, Yosuke Matsukuma und Masaki Minemoto. „Evaluation of Two-Phase Condition and Mass Transfer in GDL With Pore Network Model“. In ASME 2009 7th International Conference on Fuel Cell Science, Engineering and Technology. ASMEDC, 2009. http://dx.doi.org/10.1115/fuelcell2009-85152.

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In order to improve the output performance of PEFC, it is important to investigate the two-phase condition in gas diffusion layer (GDL). In this study, the simulated GDL structure was developed by numerical analysis including the random orientation of carbon fibers and binders. And detailed structural estimation was carried out. As structural properties, pore size distribution, electrical resistivity and tortuosity were calculated, and these values almost agreed with actual measurement values. Furthermore, our past two-phase network model was improved, and the model based on an actual structure was developed by a direct 3D networking porous structure. And the influence of GDL structure on the two-phase condition with accumulated water was evaluated, and effective diffusion coefficient of oxygen in GDL with liquid water was calculated.
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Iqbal, Gulfam, Huang Guo und Bruce S. Kang. „Reliability Model of SOFC Anode Material Under Thermo-Mechanical and Fuel Gas Contaminants Effects“. In ASME 2009 7th International Conference on Fuel Cell Science, Engineering and Technology. ASMEDC, 2009. http://dx.doi.org/10.1115/fuelcell2009-85026.

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Solid Oxide Fuel Cells (SOFCs) work under severe environment which deteriorate anode material properties and reduce its serviceable life. Besides electrochemical performance, structural integrity of SOFC anode is essential for successful long-term operation. SOFC anode material is subjected to stresses at high temperature, thermal and redox cycles and fuel gas contaminants effects on its structure during long-term operation. These mechanisms can degrade anode microstructure and decrease electrochemical performance and structural properties. In this research an anode material degradation model is developed and implemented in finite element analysis. The model incorporates thermo-mechanical and fuel gas contaminants degradation effects and predicts long-term structural integrity of SOFC anode. An analytical solution is also developed for button cell deformation under uniform pressure to establish correlation between the degradation model and experimental measurements. Preliminary results of the model application on the planar co-flow SOFC are also presented.
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Christie, Jaryd R., Mohamed Abdelrazek, Pencilla Lang und Sarah A. Mattonen. „A multi-modality radiomics-based model for predicting recurrence in non-small cell lung cancer“. In Biomedical Applications in Molecular, Structural, and Functional Imaging, herausgegeben von Barjor S. Gimi und Andrzej Krol. SPIE, 2021. http://dx.doi.org/10.1117/12.2586233.

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Lua, Jim, Jag Sankar und Devdas Pai. „A Four-Cell Decomposition Model for Unbalanced Woven Fabric Composites Subjected to Thermal-Mechanical Loading“. In 47th AIAA/ASME/ASCE/AHS/ASC Structures, Structural Dynamics, and Materials Conference
14th AIAA/ASME/AHS Adaptive Structures Conference
7th. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2006. http://dx.doi.org/10.2514/6.2006-1690.

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Bum, Hong, Kwang Soo, Kyeong Mo und Sang Kil. „Effect of coral cell on beach deformation by 3 D hydraulic model experiment“. In Seventh International Conference on Advances in Civil Structural and Environmental Engineering ACSEE 2018. Institute of Research Engineers and Doctors, 2018. http://dx.doi.org/10.15224/978-1-63248-158-0-13.

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González, Paula, Michaela Reichenzeller, Roland Eils und Evgeny Gladilin. „Contactless Investigation of Nuclear Mechanics of Normal and Lamin Mutant Cells Using a 3D Image- and Model-Based Framework“. In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204663.

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Apart from storing most of the DNA in eukaryotic cells, the cell nucleus provides mechanical protection through its nuclear envelope to ensure the integrity of the genome. The nuclear lamina is known to play an important role in this respect by supplying a structural framework for the nucleus [1]. The severe diseases arising from mutations in the LMNA gene confirm the importance of the lamin proteins for normal cell functionality [2]. Most experimental techniques for investigation of the cell mechanics are based on the application of external forces onto the cell boundary [3]. Thus, the quantitative determination of the mechanical properties of intracellular structures in situ, still represents a challenging task. In our previous works, we proposed a 3D image- and model-based framework for analysis of intracellular mechanics [4]. In this work, we extend this approach to a fully contactless investigation of nuclear mechanics of normal and LMNA–/– mutant cells. Differently from previous approaches, cellular deformation was induced by chemical agents, i.e., without any mechanical contact with the cell boundary. In particular, we focus on (i) comparative analysis of 3D structural response of nuclear matter with respect to external forces in normal and pathological cell, as well as (ii) determination of the scarcely-investigated nuclear compressibility (i.e. the Poisson’s ratio).
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Iqbal, Gulfam, Suryanarayana R. Pakalapati, Francisco Elizalde-Blancas, Huang Guo, Ismail Celik und Bruce Kang. „Anode Structure Degradation Model for Planar-SOFC Configuration Under Fuel Gas Contaminants“. In ASME 2010 8th International Conference on Fuel Cell Science, Engineering and Technology. ASMEDC, 2010. http://dx.doi.org/10.1115/fuelcell2010-33183.

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Solid Oxide Fuel Cells (SOFCs) is one of the enabling technologies that are being extensively researched for clean power generation from coal-derived syngas. Anode structural degradation is one of the problems that limit the SOFCs operation lifetime and it is further aggravated by some common contaminants found in coal syngas such as phosphine. An accurate model for predicting the degradation patterns inside an SOFC anode operating under different conditions will be an effective tool for advancement of this technology. In this study, a structural durability model developed earlier for button SOFC anodes is extended to simulate the planar-SOFC anodes. The model accounts for thermo-mechanical and fuel gas contaminants effects on the anode material properties to predict evolution, in space and time, of degradation patterns inside the anode and consequently its lifetime. The temperature field and contaminant concentration distribution inside the SOFC anode are the required inputs for the degradation model which are obtained from DREAM-SOFC: a multi-physics code for SOFC modeling. Due to larger active areas compared to button cell, planar-SOFCs bear greater spatial and temporal temperature gradients which lead to higher thermo-mechanical degradation. Moreover, fuel contaminants are distributed on the anode surface which leads to non-uniform microstructure degradation along the fuel flow. For the case of co-flow configuration, anode thermo-mechanical degradation is severe at the anode-electrolyte interface at the fuel outlet. Whereas the fuel gas contaminants effects on the anode microstructure begin at the fuel inlet and propagate through the anode thickness and along the fuel flow. This research will be useful to establish control parameters to achieve desired service life of SOFC stacks working under coal syngas.
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Berichte der Organisationen zum Thema "Structural cell model"

1

Stupakov, Gennady. Random Walk Model for Cell-To-Cell Misalignments in Accelerator Structures. Office of Scientific and Technical Information (OSTI), September 2000. http://dx.doi.org/10.2172/765008.

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Sparks, Paul, Jesse Sherburn, William Heard und Brett Williams. Penetration modeling of ultra‐high performance concrete using multiscale meshfree methods. Engineer Research and Development Center (U.S.), September 2021. http://dx.doi.org/10.21079/11681/41963.

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Terminal ballistics of concrete is of extreme importance to the military and civil communities. Over the past few decades, ultra‐high performance concrete (UHPC) has been developed for various applications in the design of protective structures because UHPC has an enhanced ballistic resistance over conventional strength concrete. Developing predictive numerical models of UHPC subjected to penetration is critical in understanding the material's enhanced performance. This study employs the advanced fundamental concrete (AFC) model, and it runs inside the reproducing kernel particle method (RKPM)‐based code known as the nonlinear meshfree analysis program (NMAP). NMAP is advantageous for modeling impact and penetration problems that exhibit extreme deformation and material fragmentation. A comprehensive experimental study was conducted to characterize the UHPC. The investigation consisted of fracture toughness testing, the utilization of nondestructive microcomputed tomography analysis, and projectile penetration shots on the UHPC targets. To improve the accuracy of the model, a new scaled damage evolution law (SDEL) is employed within the microcrack informed damage model. During the homogenized macroscopic calculation, the corresponding microscopic cell needs to be dimensionally equivalent to the mesh dimension when the partial differential equation becomes ill posed and strain softening ensues. Results of numerical investigations will be compared with results of penetration experiments.
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Azadi, Paratoo. 8th Annual Glycoscience Symposium: Integrating Models of Plant Cell Wall Structure, Biosynthesis and Assembly. Office of Scientific and Technical Information (OSTI), September 2015. http://dx.doi.org/10.2172/1221374.

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