Auswahl der wissenschaftlichen Literatur zum Thema „Stress oxydante“
Geben Sie eine Quelle nach APA, MLA, Chicago, Harvard und anderen Zitierweisen an
Inhaltsverzeichnis
Machen Sie sich mit den Listen der aktuellen Artikel, Bücher, Dissertationen, Berichten und anderer wissenschaftlichen Quellen zum Thema "Stress oxydante" bekannt.
Neben jedem Werk im Literaturverzeichnis ist die Option "Zur Bibliographie hinzufügen" verfügbar. Nutzen Sie sie, wird Ihre bibliographische Angabe des gewählten Werkes nach der nötigen Zitierweise (APA, MLA, Harvard, Chicago, Vancouver usw.) automatisch gestaltet.
Sie können auch den vollen Text der wissenschaftlichen Publikation im PDF-Format herunterladen und eine Online-Annotation der Arbeit lesen, wenn die relevanten Parameter in den Metadaten verfügbar sind.
Zeitschriftenartikel zum Thema "Stress oxydante"
Mezdour, Hichem, Mourad Hanfer, Ahmed Menad und Souad Ameddah. „Oxidative stress and its relationship with the emergence of various stomach damages“. Batna Journal of Medical Sciences (BJMS) 4, Nr. 2 (31.12.2017): 145–48. http://dx.doi.org/10.48087/bjmsra.2017.4204.
Der volle Inhalt der QuelleThorin-Trescases, Nathalie, Guillaume Voghel, Nada Farhat, Annick Drouin, Marie-Ève Gendron und Éric Thorin. „Âge et stress oxydant“. médecine/sciences 26, Nr. 10 (Oktober 2010): 875–80. http://dx.doi.org/10.1051/medsci/20102610875.
Der volle Inhalt der QuelleGeny, B., A. L. Charles, A. Lejay und A. Meyer. „Pollution et stress oxydant“. Revue Française d'Allergologie 59, Nr. 3 (April 2019): 174–76. http://dx.doi.org/10.1016/j.reval.2019.02.195.
Der volle Inhalt der QuelleRoussel, Anne-Marie, und Monique Ferry. „Stress oxydant et vieillissement“. Nutrition Clinique et Métabolisme 16, Nr. 4 (Dezember 2002): 285–91. http://dx.doi.org/10.1016/s0985-0562(02)00173-5.
Der volle Inhalt der QuelleBonnefont-Rousselot, D. „Obésité et stress oxydant“. Obésité 9, Nr. 1 (08.10.2013): 8–13. http://dx.doi.org/10.1007/s11690-013-0408-3.
Der volle Inhalt der QuelleBarouki, Robert. „Stress oxydant et vieillissement“. médecine/sciences 22, Nr. 3 (März 2006): 266–72. http://dx.doi.org/10.1051/medsci/2006223266.
Der volle Inhalt der QuelleLeverve, Xavier. „Stress oxydant et antioxydants ?“ Cahiers de Nutrition et de Diététique 44, Nr. 5 (Oktober 2009): 219–24. http://dx.doi.org/10.1016/j.cnd.2009.09.001.
Der volle Inhalt der QuelleROCHETTE, L. „Stress oxydant et sepsis“. Réanimation 17, Nr. 6 (September 2008): 1–4. http://dx.doi.org/10.1016/s1624-0693(08)73932-x.
Der volle Inhalt der QuelleDelattre, J., M. Gardès und D. Jore. „Stress oxydant et diabète sucré“. Journal de la Société de Biologie 195, Nr. 4 (2001): 375–76. http://dx.doi.org/10.1051/jbio/2001195040375.
Der volle Inhalt der QuelleFavier, A. „Stress oxydant et pathologies humaines“. Annales Pharmaceutiques Françaises 64, Nr. 6 (November 2006): 390–96. http://dx.doi.org/10.1016/s0003-4509(06)75334-2.
Der volle Inhalt der QuelleDissertationen zum Thema "Stress oxydante"
Corroyer, Sophie. „Agression oxydante et processus de lesion-reparation : role du systeme des cyclines et du systeme des insulin-like growth factors“. Paris 5, 1998. http://www.theses.fr/1998PA05N130.
Der volle Inhalt der QuelleVentura, Emilie. „Stress oxydant et vieillissement : modulation de la NADPH oxydase“. Montpellier 2, 2008. http://www.theses.fr/2008MON20128.
Der volle Inhalt der QuelleOxidative stress is the result of an imbalance between production of oxidants and antioxidant defense mechanisms. The NADPH oxidase is a key enzyme for excessive production of oxidants, strengthening the interest of its modulation. NADPH oxidase is involved in the initiation and progression of age-related diseases such as cardiovascular diseases and dementia. Through an epidemiological study consisted of 517 subjects (79. 5 ± 7. 1 years), we determined NADPH oxidase activation and its main determinants with age. Among the factors tested, homocysteine and inflammation were significantly associated with NADPH oxidase activity in a multivariate analysis. These data are confirmed in vitro because homocysteine thiolactone and lipopolysaccharide induce a dose-dependent activation and expression of NADPH oxidase in cell line THP-1. In an in vitro on THP-1 and animal studies, we studied the negative modulation of the NADPH oxidase by natural antioxidants (polyphenols) and enzymatic antioxidants (melon extract rich in SOD and catalase). These nutritional antioxidants negatively modulate the activity but also the expression of NADPH oxidase. In conclusion, NADPH oxidase activity is associated with age and is positively regulated by inflammation and homocysteine. The superoxide anion production is associated with cardiovascular and neurodegenerative diseases. NADPH oxidase may be modulated in vitro by natural antioxidants. Links with dementia, cardiovascular disease or mortality must be further study. The antioxidant supplements efficiency must be clarified in animal models and then on a clinical study
Rossary, Adrien. „Implication de la NADPH oxydase dans les phénomènes de stress oxydant“. Lyon 1, 2007. http://www.theses.fr/2007LYO10271.
Der volle Inhalt der QuelleFalvelly, Diane de. „Réponse immunitaire, sida et cancer: rôle du stress oxydant et des vitamines anti-oxydantes“. Paris 5, 1997. http://www.theses.fr/1997PA05P131.
Der volle Inhalt der QuellePage, Audrey. „Etude de la modulation de la réponse cellulaire au stress oxydatif par les protéines VP24 des virus Marburg et Ebola“. Phd thesis, Ecole normale supérieure de lyon - ENS LYON, 2012. http://tel.archives-ouvertes.fr/tel-00671994.
Der volle Inhalt der QuelleAllanore, Yannick. „Implication du stress oxydant et propriètés anti-oxydantes des inhibiteurs calciques dihydropyridiniques au cours de la sclérodermie systémique“. Paris 5, 2004. http://www.theses.fr/2004PA05N08S.
Der volle Inhalt der QuelleFree radicals are suspected to play a key role in the successive vascular, dysmmune and fibrotic lesions which occur in sistemic sclerosis. Our results emphasize monocyte free radical superoxide anion production wich may account for the various oxidative stress circulating markers observed in the disease. We also demonstrate that calcium channel blockers of dihydropyridine type can inhibit monocyte activation and superoxyde anion production. These drugs seem to act in modulation of superoxyde anion production through inhibition of PMA-induced phosphorylations and protein kinase c activity inhibition. These results, together with previous data on their impact on microvasculature, highlith the major role of dihydropyridine type calcium channel blockers in the treatment of systemic sclerosis
Rangel, Manuel. „Analyse du rôle de la NADPH oxydase et du stress oxydant dans les cellules dendritiques“. Phd thesis, Université de Grenoble, 2012. http://tel.archives-ouvertes.fr/tel-00767151.
Der volle Inhalt der QuelleGoux, Aurélie. „NADPH Oxydase et Stress Oxydant au cours de l'Insuffisance Rénale Chronique : modulation par les HDL“. Thesis, Montpellier 2, 2010. http://www.theses.fr/2010MON20166/document.
Der volle Inhalt der QuelleCardiovascular (CV) diseases are the first cause of mortality during chronic kidney disease (CKD) and cannot only be explained by traditional risk factors (age, gender, dyslipidemia, hypertension). Oxidative stress, which has been associated with CKD, appears as a non-traditional risk factor closely interconnected with inflammation and malnutrition.This study aimed at investigating oxidative stress in CV complications in uremic rats. Then, HDL proteomic profile and in vitro functionality of HDL were compared between hemodialyzed (HD) patients and control subjects.First, an animal model of CKD associated with malnutrition, the adenine-fed rats, was set up in order to study CV oxidative stress. NADPH oxidase activity was increased three-fold, but the maximal activity of mitochondrial respiratory chain complexes and SOD were not different between groups. Superoxide anion output was associated with accumulation of osteopontin and of pro-collagen type I. In a second part, HDL proteomic study from HD and control subjects was performed to characterize qualitative modifications associated with the decrease in HDL observed in CKD. HDL anti-oxidative activities from these subjects were studied in vitro in a model of copper-induced LDL oxidation and in a cellular model of NADPH oxidase activation. Compared to control, HDL from HD patients failed to protect LDL oxidation. By contrast, HDL modulation of NADPH activity is maintained in HD patients but could be impaired by elevated inflammation.These results suggest that oxidative stress is a key event in cardiac complications during CKD. Among protective endogenous mechanisms, HDL anti-oxidative properties could be impaired in HD patients
Duroc, Yann. „Etude des relations structure-fonction de la protéine ORF 138 responsable de la stérilité mâle cytosplasmique Ogura“. Paris 11, 2004. http://www.theses.fr/2004PA112187.
Der volle Inhalt der QuelleThe ORF138 protein (encoded by the mitochondrial genome) is a mitochondrial membrane protein responsible of Ogura cytoplasmic male sterility (CMS) in Brassicaceae. In sterile plants microspore abortion is apparently due to premature degeneration of the anther tapetum, by a still unknown mechanism. This is the only induced phenotype whereas ORF138 is present in all tissues. The objectives of this work were to develop molecular and biochemical tools for the study of this protein, and to obtain relevent information for setting up hypotheses about the mechanism of male sterility. We showed that the association of the hydrophobic and hydrophilic domains of the protein is essential for its effect. A domain of 15 amino acids, sharing predicted secondary structure with two other proteins of unrelated CMS's, was highlighted and showed to play a role in this effect. We showed that ORF138 was integrated in the mitochondrial inner membrane of rapeseed and formed homooligomers. A proteic complex of size ranging between 750 and 900 kDa implying ORF138 was also identified. We also proved that ORF138 can be copurified with mitochondrial nucleoproteic components of male sterile plants. An increased accumulation of mitochondrial alternative oxydase probably accompanied by an increased activity, was observed in the sterile plants compared to fertiles. The connection between oxidative stress and alternative oxydase activity is well-known, and a possible bond between an oxidizing stress and the premature death of the tapetum constitutes one of the most promising tracks for the elucidation of the sterility mechanism
Joffe, Ricardo. „The role of mesophyll conductance in the regulation of photosynthesis under ozone-induced oxidative stress in poplar : ecophysiological, anatomical and biochemical aspects“. Electronic Thesis or Diss., Université de Lorraine, 2022. http://www.theses.fr/2022LORR0289.
Der volle Inhalt der QuelleOzone (O₃) is one of the most harmful and widespread air pollutants considerably affecting photosynthesis and thus crop yields and plant health worldwide. The CO₂ diffusion conductance within leaf mesophyll (gm) is widely considered to be a major limiting factor of photosynthesis under stress and non-stress conditions. This work aimed to investigate the effects of O₃-induced oxidative stress on gm and to decipher underlying mechanisms driving its regulation at a physiological, anatomical and biochemical level. A selection of several genotypes of the tree model Populus × canadensis Moench was submitted to chronic O₃ exposure at 120 ppb for 21d under controlled conditions in phytotronic chambers in order to analyse gas exchange, antioxidants pool and leaf structural and ultrastructural traits. Chronic O₃ caused a substantial decrease of gm, which was thus a major limiting factor of the net assimilation rate (Anet), ahead of stomatal conductance to CO₂ (gsc) and of the maximum carboxylation capacity of the Rubisco enzyme (Vcmax). Total pools of the main antioxidations ascorbate and glutathione were not related to gm but the signalling molecule malondialdehyde (MDA) might have mitigated O₃-caused decrease of gm. On a structural level, this observed decrease was mainly driven by O₃-induced modifications of subcellular traits, notably cell wall and cytoplasm thickening, inter-chloroplast spacing as well as the ratio of exposed chloroplasts to mesophyll surface area (Sc/Sm). However, the observed relationship between gm and anatomy but also the gm response to O₃ were genotype-dependent. The dynamic response of gm to O₃ was also investigated testing a single genotype during alternating periods of short-term acute O₃ exposure at 200 ppb and recovery. The assumed contribution of biochemical determinants of gm was analysed by quantifying the expression levels of several potential CO₂-diffusion facilitating carbonic anhydrases (CA). Under acute O₃ stress, gm was highly responsive but exhibited also some degree of post-stress resilience. The response of the expression level of α-CA1 to the applied treatment may suggest the involvement of this CA isoform in the short-term regulation of gm. This work underlines the importance of gm as a major limitation of Anet but also the necessity of its consideration in a comprehensive assessment of photosynthesis response to environmental constrains. These results further suggest that gm might be a very promising target for plant engineering in order to enhance photosynthetic yield and stress resistance of field and biomass crops
Bücher zum Thema "Stress oxydante"
Delattre, Jacques. Radicaux libres et stress oxydant: Aspects biologiques et pathologiques. Londres: Editions TEC & DOC et Editions médicales nationales, 2005.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Stress oxydante"
Orban, J. C. „Oxygène, stress oxydant“. In Désordres métaboliques et réanimation, 427–37. Paris: Springer Paris, 2011. http://dx.doi.org/10.1007/978-2-287-99027-4_22.
Der volle Inhalt der QuelleGilbert, T., J. Drai und M. Bonnefoy. „Stress oxydant et maladie d’Alzheimer“. In Traité sur la maladie d’Alzheimer, 175–94. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0443-9_10.
Der volle Inhalt der QuelleZeghal, K. M., und Z. Sahnoun. „La réaction inflammatoire et le stress oxydant“. In Abrégé de physiologie à l’usage des acupuncteurs et des réflexothérapeutes, 47–53. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0361-6_4.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Stress oxydante"
Boogaerts, M. A., P. Zachée, M. P. Emonds, W. Goossens, R. L. Verwilghen und R. L. Lins. „ERYTHROCYTES(RBC) AS SUICIDAL ENDOGENOUS SCAVENGERS IN IMMUNE TRIGGERED GRANULOCYTE(PMN)MEDIATED VASCULAR DAMAGE“. In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643162.
Der volle Inhalt der Quelle