Thematische Bibliographien / Sensitivity of the cancer cells

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Sensitivity of the cancer cells“

Autor: Grafiati
Veröffentlicht am 28. Juni 2021
Zuletzt aktualisiert am 29. Juli 2025

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Zeitschriftenartikel zum Thema "Sensitivity of the cancer cells"

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Kozak, T. P. "MODIFICATION OF BREAST CANCER CELLS' SENSITIVITY TO METFORMIN DUE TO CO-CULTIVATION WITH B. ANIMALIS." Biotechnologia Acta 18, no. 2 (2025): 54–56. https://doi.org/10.15407/biotech18.02.054.

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Glucose metabolism (GM) disturbances are well-known risk factors for the development of breast cancer (BC). The GM regulator metformin is used as an adjunctive therapy for BC. Another potent modulator of GM in BC cells is the microbiota, particularly bifidobacteria. The combined action of these factors may lead to unpredictable effects on the sensitivity of malignant cells to antitumor agents. Aim. To investigate the effect of Bifidobacterium animalis on the sensitivity of BC cells to the cytotoxic/cytostatic effects of metformin. Materials and Methods. The impact of B. animalis on GM in BC ce
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De Maria, R. "5 Drug sensitivity of cancer stem cells." European Journal of Cancer Supplements 8, no. 7 (2010): 10. http://dx.doi.org/10.1016/s1359-6349(10)71708-0.

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Chang, Yu-Yao, Tsung-Ying Yang, and Gwo-Tarng Sheu. "Association of Wild-Type TP53 with Downregulation of Lovastatin Sensitivity in Human Non-Small Cell Lung Cancer Cells." Current Issues in Molecular Biology 46, no. 9 (2024): 10130–39. http://dx.doi.org/10.3390/cimb46090604.

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Statins inhibit 3-hydroxy-3-methylglutaryl-CoA reductase (HMGCR), the rate-limiting enzyme of the mevalonate pathway, and reduce cholesterol synthesis. They also have been demonstrated to improve prognosis in patients with various cancers, suggesting a potential anti-cancer effect of statins. However, there is no consensus on the molecular targets of statins for their anti-cancer effects. Docetaxel (DOC) is a microtubule-stabilizing agent currently used as a chemotherapeutic drug in several cancers, including lung cancer. Interestingly, the anti-cancer effects of either drug that are related t
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Pummarin, Siwanut, Natcha Madared, Sotida Kayem, et al. "Different Doxorubicin Sensitivity Across Various Human Cancer Cell Lines." Trends in Sciences 21, no. 12 (2024): 8566. http://dx.doi.org/10.48048/tis.2024.8566.

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Doxorubicin (Dox) is a highly potent chemotherapeutic agent, approved by FDA since 1974, for treating a broad spectrum of cancers. However, development of severe side effects and drug resistance are main issues that limit the clinical use of Dox. Herein, we aimed to investigate the sensitivity of Dox in a variety of human cancer cell lines. Eleven cell lines were tested in this study including 2 hepatocellular carcinoma (HCC) cell lines (HepG2 and Huh7), 4 bladder cancer (BlCa) cell lines (UMUC-3, VMCUB-1, TCCSUP and BFTC-905), a lung cancer cell line (A549), a cervical carcinoma cell line (He
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Watanabe, Takayuki, Takaaki Oba, Keiji Tanimoto, Tomohiro Shibata, Shinobu Kamijo, and Ken-ichi Ito. "Tamoxifen resistance alters sensitivity to 5-fluorouracil in a subset of estrogen receptor-positive breast cancer." PLOS ONE 16, no. 6 (2021): e0252822. http://dx.doi.org/10.1371/journal.pone.0252822.

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Sequential treatment with endocrine or chemotherapy is generally used in the treatment of estrogen receptor (ER)-positive recurrent breast cancer. To date, few studies have investigated the effect of long-term endocrine therapy on the response to subsequent chemotherapy in ER-positive breast cancer. We examined whether a preceding endocrine therapy affects the sensitivity to subsequent chemotherapy in ER-positive breast cancer cells. Three ER-positive breast cancer cell lines (T47D, MCF7, BT474) and tamoxifen-resistant sublines (T47D/T, MCF7/T, BT474/T) were analyzed for sensitivity to 5-fluor
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Noh, Woo-Chul, Wallace H. Mondesire, Junying Peng, et al. "Determinants of Rapamycin Sensitivity in Breast Cancer Cells." Clinical Cancer Research 10, no. 3 (2004): 1013–23. http://dx.doi.org/10.1158/1078-0432.ccr-03-0043.

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Zhang, Yi, Wendy Schulte, Desmond Pink, et al. "Sensitivity of Cancer Cells to Truncated Diphtheria Toxin." PLoS ONE 5, no. 5 (2010): e10498. http://dx.doi.org/10.1371/journal.pone.0010498.

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Candido, Saverio, Stephen L. Abrams, Linda Steelman, et al. "Metformin influences drug sensitivity in pancreatic cancer cells." Advances in Biological Regulation 68 (May 2018): 13–30. http://dx.doi.org/10.1016/j.jbior.2018.02.002.

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Karagkounis, Georgios, Jennifer DeVecchio, Sylvain Ferrandon, and Matthew F. Kalady. "Simvastatin enhances radiation sensitivity of colorectal cancer cells." Surgical Endoscopy 32, no. 3 (2017): 1533–39. http://dx.doi.org/10.1007/s00464-017-5841-1.

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Shariat Razavi, Seyedeh Mahya, Reyhaneh Mahmoudzadeh Vaziri, Gholamreza Karimi, et al. "Crocin Increases Gastric Cancer Cells’ Sensitivity to Doxorubicin." Asian Pacific Journal of Cancer Prevention 21, no. 7 (2020): 1959–67. http://dx.doi.org/10.31557/apjcp.2020.21.7.1959.

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Dissertationen zum Thema "Sensitivity of the cancer cells"

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Tong, Zhimin. "The mechanisms of cellular sensitivity to photodynamic therapy /." *McMaster only, 2001.

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Landry, Benjamin D. "Tumor-stroma interactions differentially alter drug sensitivity based on the origin of stromal cells." eScholarship@UMMS, 2018. https://escholarship.umassmed.edu/gsbs_diss/1011.

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Tumor heterogeneity observed between patients has made it challenging to develop universal or broadly effective cancer therapies. Therefore, an ever-growing movement within cancer research aims to tailor cancer therapies to individual patients or specific tumor subtypes. Tumor stratification is generally dictated by the genomic mutation status of the tumor cells themselves. Importantly, non-genetic influences – such as interactions between tumor cells and other components of the tumor microenvironment – have largely been ignored. Therefore, in an effort to increase treatment predictability and
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Ferguson, Paul R. "The role of thymidylate synthase in modulating sensitivity to chemotherapeutic agents." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326399.

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Fung, Ka-lai. "Significance of MAD2 in mitotic checkpoint control and cisplatin sensitivity of testicular germ cell tumour cells." View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B38588912.

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Kanwal, Shahzina. "Effect of O-GlcNAcylation on tamoxifen sensitivity in breast cancer derived MCF-7 cells." Phd thesis, Université René Descartes - Paris V, 2013. http://tel.archives-ouvertes.fr/tel-00912341.

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One of the hallmarks of cancer cells is to exhibit increased uptake and consumption of glucose.3-5% of the glucose entering into the cell leads to a minor pathway of the glucose metabolismknown as the hexosamine biosynthetic pathway (HBP). UDP-N-acetylglucosamine is the endproduct of HBP and is used as substrate by OGT (O-GlcNAc transferase) to modify diverserange of nuclear and cytoplasmic proteins with a recently characterized post-translationalmodification called O-GlcNAcylation. It corresponds to the addition of sugar moiety O-linked β-N-acetylglucosamine (O-GlcNAc) on serine or threonine
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Sureechatchaiyan, Paichat [Verfasser]. "Role of Purinergic Ligands to Enhance Cisplatin Sensitivity in Ovarian Cancer Cells / Paichat Sureechatchaiyan." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2017. http://d-nb.info/1148066756/34.

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Fung, Ka-lai, and 馮家禮. "Significance of MAD2 in mitotic checkpoint control and cisplatin sensitivity of testicular germ cell tumour cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39357727.

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Zhang, Pingde, та 张萍德. "TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47752944.

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Ovarian cancer is the most lethal gynecological malignancy. Most of ovarian cancer patients relapse and subsequently die due to the development of resistance to chemotherapy. P73 belongs to the tumor suppressor p53 family. Like p53, the transcriptionally active TAp73 can bind specifically to p53 responsive elements and transactivates some of the p53 target genes, and finally leads to cell cycle arrest and apoptosis. TAp73 can be induced by DNA damage to enhance cellular sensitivity to anticancer agents in human cancer cells. However, the functions of TAp73 in ovarian cancer cells and t
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Newbury, Golnar. "A Numerical Study of a Delay Differential Equation Model for Breast Cancer." Thesis, Virginia Tech, 2007. http://hdl.handle.net/10919/34420.

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In this thesis we construct a new model of the immune response to the growth of breast cancer cells and investigate the impact of certain drug therapies on the cancer. We use delay differential equations to model the interaction of breast cancer cells with the immune system. The new model is constructed by combining two previous models. The first model accounts for different cell cycles and includes terms to evaluate drug treatments, but ignores quiescent tumor cells. The second model includes quiescent cells, but ignores the immune response and drug treatments. The new model is obtained by co
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Jokinen, E. (Elina). "Targeted therapy sensitivity and resistance in solid malignancies." Doctoral thesis, Oulun yliopisto, 2014. http://urn.fi/urn:isbn:9789526205755.

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Abstract Cancer is a major global killer and a challenge for the healthcare worldwide. Earlier cancer has been treated with surgery, radiation, chemotherapy and hormonal therapy. Unfortunately the efficiency of these therapies has shown to be limited and this has raised an enthusiasm for development of new, targeted cancer therapies that are based on activated oncogenes. The challenge of the targeted therapies is therapy resistance, de novo, adaptive and acquired. This work investigated targeted therapy sensitivity and resistance in lung cancer, breast cancer, colorectal cancer, and melanoma c
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Bücher zum Thema "Sensitivity of the cancer cells"

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Steinberg, Ken. Effects of structural domains of Raf-1 on the sensitivity of MCF-7 breast cancer cells to Taxol (Paclitaxel). Laurentian University, School of Graduate Studies, 2005.

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Farrar, William L., ed. Cancer Stem Cells. Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511605536.

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Yu, John S., ed. Cancer Stem Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-280-9.

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Rajasekhar, Vinagolu K., ed. Cancer Stem Cells. John Wiley & Sons, 2014. http://dx.doi.org/10.1002/9781118356203.

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Wiestler, O. D., B. Haendler, and D. Mumberg, eds. Cancer Stem Cells. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-70853-7.

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Papaccio, Gianpaolo, and Vincenzo Desiderio, eds. Cancer Stem Cells. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7401-6.

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Bapat, Sharmila, ed. Cancer Stem Cells. John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470391594.

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Pat, Moyer Mary, and Poste George, eds. Colon cancer cells. Academic Press, 1990.

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Papaccio, Federica, and Gianpaolo Papaccio, eds. Cancer Stem Cells. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3730-2.

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Sharmila, Bapat, ed. Cancer stem cells. John Wiley & Sons, 2008.

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Buchteile zum Thema "Sensitivity of the cancer cells"

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Saeed, Mohamed, Henry Johannes Greten, and Thomas Efferth. "Collateral Sensitivity in Drug-Resistant Tumor Cells." In Resistance to Targeted Anti-Cancer Therapeutics. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7070-0_10.

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Lebert-Ghali, Charles-Etienne, Joanne Margaret Ramsey, Alexander Thompson, and Janetta Bijl. "Sensitivity of Hematopoietic and Leukemic Stem Cells to Hoxa Gene Levels." In Stem Cells and Cancer Stem Cells, Volume 4. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2828-8_2.

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Moissoglu, Konstadinos, Stephen J. Lockett, and Stavroula Mili. "Visualizing and Quantifying mRNA Localization at the Invasive Front of 3D Cancer Spheroids." In Cell Migration in Three Dimensions. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2887-4_16.

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AbstractLocalization of mRNAs at the front of migrating cells is a widely used mechanism that functionally supports efficient cell movement. It is observed in single cells on two-dimensional surfaces, as well as in multicellular three-dimensional (3D) structures and in tissue in vivo. 3D multicellular cultures can reveal how the topology of the extracellular matrix and cell-cell contacts influence subcellular mRNA distributions. Here we describe a method for mRNA imaging in an inducible system of collective cancer cell invasion. MDA-MB-231 cancer cell spheroids are embedded in Matrigel, induce
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Isik, Omer. "Hyperthermia: Causes, Symptoms, Prevention and Treatment." In Immunotherapy in Human Cancers. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359388.16.

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Hyperthermia is a type of cancer treatment in which body tissue is exposed to high temperatures to damage and kill cancer cells. The objective of this treatment is to raise the temperature in tumor up to such a therapeutic level that cell death occurs. Hyperthermia, the use of elevated temperatures to treat cancer, has emerged as a promising adjuvant therapy. By raising the temperature of tumor tissues to 40-45°C, hyperthermia enhances the effectiveness of radiation and chemotherapy. This therapeutic approach can damage and kill cancer cells with minimal harm to normal tissues, primarily by ca
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Yoshida, Tatsushi, and Toshiyuki Sakai. "Agents that Regulate DR5 and Sensitivity to TRAIL." In Sensitization of Cancer Cells for Chemo/Immuno/Radio-therapy. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-474-2_4.

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Stahl, Sebastian, Fabian Mueller, Ira Pastan, and Ulrich Brinkmann. "Factors that Determine Sensitivity and Resistances of Tumor Cells Towards Antibody-Targeted Protein Toxins." In Resistance to Targeted Anti-Cancer Therapeutics. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17275-0_3.

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Brown, J. Martin, and Bradly G. Wouters. "Does Apoptosis Contribute to Tumor Cell Sensitivity to Anticancer Agents?" In Apoptosis and Cancer Chemotherapy. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-720-8_1.

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Quinones, Addison, and Anne Le. "The Multifaceted Glioblastoma: From Genomic Alterations to Metabolic Adaptations." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_4.

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AbstractGlioblastoma multiforme (GBM) develops on glial cells and is the most common as well as the deadliest form of brain cancer. As in other cancers, distinct combinations of genetic alterations in GBM subtypes induce a diversity of metabolic phenotypes, which explains the variability of GBM sensitivity to current therapies targeting its reprogrammed metabolism. Therefore, it is becoming imperative for cancer researchers to account for the temporal and spatial heterogeneity within this cancer type before making generalized conclusions about a particular treatment’s efficacy. Standard therap
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Crescimanno, M., N. Borsellino, V. Leonardi, L. Rausa, and N. D’Alessandro. "Effects of Tumor Necrosis Factor-Alpha on Growth and Doxorubicin Sensitivity of Multidrug Resistant Tumor Cell Lines." In Cancer Therapy. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84613-7_16.

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Derweesh, Ithaar H., Luis Molto, Charles Tannenbaum, et al. "Alterations in T-Cell Signaling Pathways and Increased Sensitivity to Apoptosis." In Cancer Immunotherapy at the Crossroads. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-743-7_7.

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Konferenzberichte zum Thema "Sensitivity of the cancer cells"

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Li, Han, Zheng Dang, Xuemei Ma, and Pengxiang Zhao. "Hydrogen Nanobubbles Enhance Sensitivity of Drug-Resistant Cancer Cells." In 2024 IEEE 1st International Workshop on Future Intelligent Technologies for Young Researchers (FITYR). IEEE, 2024. http://dx.doi.org/10.1109/fityr63263.2024.00016.

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Torres-Torres, Carlos, Blanca Estela García-Pérez, and Hilario Martines-Arano. "Ablation and Dynamic Distributions Exhibited by Lung Epithelial Cancer Cells Monitored by Chaotic Attractors." In Latin America Optics and Photonics Conference. Optica Publishing Group, 2024. https://doi.org/10.1364/laop.2024.w1b.5.

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Changes in distribution of A549 cancer cells were identified by a straightforward single-beam measurement of a modulated optical transmittance. The assistance of a chaotic attractor improves the sensitivity dependent on resonant conditions of the sample.
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Dimitriu, Gabriel, Vasile lucian Boiculese, Mihaela Moscalu, and Cristina gena Dascalu. "GLOBAL SENSITIVITY ANALYSIS APPLIED TO A CANCER IMMUNOTHERAPY MODEL." In eLSE 2019. Carol I National Defence University Publishing House, 2019. http://dx.doi.org/10.12753/2066-026x-19-177.

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Sensitivity analysis methods have a long history and have been widely applied in different fields, such as environmental modeling, economical modeling for decision making, parameter estimation and control, chemical kinetics, and biological modeling analysis, with metabolic networks, signaling pathways and genetic circuits. Cancer immunotherapy is based on the idea of immune surveillance. Immunotherapy, also named biologic therapy, represents a type of cancer treatment that boosts the body's natural defenses to fight cancer. It uses substances made by the body or in a laboratory to improve or r
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Tang, Xin, Tony Cappa, Theresa B. Kuhlenschmidt, Mark S. Kuhlenschmidt, and Taher A. Saif. "Studying the Mechanical Sensitivity of Human Colon Cancer Cells Through a Novel Bio-MEMS Force Sensor." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13237.

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Cancer deaths are primarily caused by metastases, not by the parent tumor. During the metastasis, malignant cancer cells detach from the parent tumor, and spread through the patient’s circulatory system to invade new tissues and organs [1]. To study the role played by the mechanical microenvironment on the cancer cell growth and malignancy promotion, we cultured human colon carcinoma (HCT-8) cells in vitro on substrates with varied mechanical stiffness, from the physiologically relevant 1 kPa, 20 kPa to very stiff 3.5 GPa. A novel and versatile micro-electromechanical systems (Bio-MEMS) force
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Song, Ji-Hong, Woo-Young Kim, and Yong-Yeon Cho. "Abstract 356: Mutaome-based magnolin sensitivity in ovarian cancer cells." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-356.

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Fan, Z. Hugh, Weian Sheng, Thomas George, and Chen Liu. "Abstract IA21: Enumeration of circulating tumor cells for studying cancer drug sensitivity." In Abstracts: AACR Precision Medicine Series: Drug Sensitivity and Resistance: Improving Cancer Therapy; June 18-21, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.pms14-ia21.

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Kulsum, Safeena, Sindhu VG, Ramanan Somasundara Pandian, et al. "Abstract A08: Cancer stem cell-like cells in drug resistance of head and neck squamous cell carcinoma." In Abstracts: AACR Precision Medicine Series: Drug Sensitivity and Resistance: Improving Cancer Therapy; June 18-21, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.pms14-a08.

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Liu, Jingchun, Darryl T. Martin, Marcia A. Wheeler, and Robert M. Weiss. "Abstract B21: Chemoresistant bladder cancer cells induces epithelial mesenchymal transition." In Abstracts: AACR Precision Medicine Series: Drug Sensitivity and Resistance: Improving Cancer Therapy; June 18-21, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.pms14-b21.

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Pelosof, Lorraine, Sashidhar Yerram, Nilofer Azad, and James Herman. "Abstract 652:GPX3promoter hypermethylation predicts platinum sensitivity in colorectal cancer cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-652.

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Chang, Shih-Shin, Hirohito Yamaguchi, and Mien-Chie Hung. "Abstract B03: Cisplatin-resistant cells possess collateral sensitivity to folate antimetabolites." In Abstracts: AACR Precision Medicine Series: Drug Sensitivity and Resistance: Improving Cancer Therapy; June 18-21, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.pms14-b03.

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Berichte der Organisationen zum Thema "Sensitivity of the cancer cells"

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McConkey, David J. Cell Cycle Dependence of TRAIL Sensitivity in Prostate Cancer Cells. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada466697.

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McConkey, David J. Cell Cycle Dependence of TRIAL Sensitivity in Prostate Cancer Cells. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada481365.

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Londono-Joshi, Angelina I. Sensitivity of Breast Cancer Stem Cells to TRA-8 Anti-DR5 Monoclonal Antibody. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada577680.

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Joshi, Angelina L. Sensitivity of Breast Cancer Stem Cells to TRA-8 Anti-DR5 Monoclonal Antibody. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada560119.

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Kahwati, Leila, Matthew Avenarius, Leslie Brouwer, et al. Blood-Based Tests for Multiple Cancer Screening: A Systematic Review. AHRQ, 2025. https://doi.org/10.23970/ahrqepcsrmultiple.

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Objectives. Screening for multiple cancers in a single blood test is potentially transformative. The objective of this review was to assess the benefits, harms, and accuracy of screening with blood-based multicancer screening tests (MCST) in asymptomatic adults. Data sources. Medline, Cochrane Library, trial registries, relevant government and commercial websites through December 2024; surveillance was conducted through March 31, 2025. Study Selection. Eligible designs included controlled studies for benefit outcomes (e.g., cancer mortality, cancer detection, quality of life), controlled and u
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Langland, Gregory T. Establishment of an 'In Vitro Cell-Based System' to Assay Radiation Sensitivity in Breast Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada472420.

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7

Sriuranpong, Virote. The role of SHP-1 promoter 2 hypermethylation detection of lymph node micrometastasis in resectable nonmetastasis NSCLC as a prognostic marker of disease. Chulalongkorn University, 2010. https://doi.org/10.58837/chula.res.2010.16.

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Introduction; Despite adequate surgical management of stage I non-small cell lung cancer, many patients still have relapsed of disease which leads to mortality. Micrometastasis of tumor is the postulate mechanism which might not be detected by standard H&E method. The author conducted the study of epithelial methylation marker, SHP-1 Promoter 2 (SHP1P2) methylation as a potential molecular marker to detect high risk relapsed of disease in stage I resectable non-small cell lung cancer (NSCLC). Method; To explore the potential role of SHP1P2 methylation to detect micrometastasis, Lymph node
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Markovic, Dubravka, and Edward P. Cohen. Treatment of Breast Cancer with Immunogenic Cells Transfected with DNA from Breast Cancer Cells. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396744.

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9

J, Bull Richard, and Larry E. Anderson. Sensitivity to Radiation-Induced Cancer in Hemochromatosis. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/833475.

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10

Doxsey, Stephen. Midbody Accumulation in Breast Cancer Cells. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada516482.

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