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Thematische Bibliographien / Rheumatoid arthritis / Dissertationen

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Dissertationen zum Thema „Rheumatoid arthritis“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 25. Juli 2025

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1

Ulfgren, Ann-Kristin. "Cytokines in rheumatoid arthritis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-3823-7/.

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2

DeLaura, Angela. "Rheumatoid arthritis : an overview /." Online version of thesis, 1989. http://hdl.handle.net/1850/11502.

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3

Thomson, W. "Immunogenetics of rheumatoid arthritis." Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383908.

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4

Kalla, Asgar Ali. "Osteoporosis in rheumatoid arthritis." Master's thesis, University of Cape Town, 1989. http://hdl.handle.net/11427/26297.

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The literature is replete with reports of osteoporosis in rheumatoid arthritis, but the mechanism of bone loss remains obscure. This is probably due to the overlap with bone loss of aging and the menopause, whose exact mechanisms are also poorly understood. Against this background, a study was designed to evaluate generalised bone loss in young, premenopausal (if female), patients with rheumatoid arthritis. The protocol was designed to record demographic data, as well as information pertaining to the disease. Cortical bone mass was measured at the metacarpals and left femur, using an automated

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5

Toms, Tracey. "Dyslipidaemia in rheumatoid arthritis." Thesis, University of Manchester, 2012. https://www.research.manchester.ac.uk/portal/en/theses/dyslipidaemia-in-rheumatoid-arthritis(e7808bd7-52e6-40a0-84cb-e4aadbf7505c).html.

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Introduction: Rheumatoid arthritis (RA) is known to be associated with an increased risk of cardiovascular disease (CVD), resulting in excess mortality. Dyslipidaemia has been identified as a major CVD risk factor in the general population. Current evidence would suggest that lipid metabolism is altered in RA due to inflammation, and that use of anti-inflammatory therapy may reverse some of these changes. However, the impact of such lipid changes on CVD in RA remains unknown. Data regarding the effects of RA/drug therapy on lipid structure and function are sparse. Genetic factors are important

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6

Vingsbo, Lundberg Carina. "Chronic autoimmune arthritis in rats pathogenesis and genetic factors /." Lund : Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/68945081.html.

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7

Riding, S. Barbara. "The arthritic pain experience of children with juvenile rheumatoid arthritis." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/27731.

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This study was designed to investigate the experience of having arthritic pain from the children's perspective. Previous research on how Canadian children perceive and manage arthritic pain and how it affects their daily lives is nonexistent. Therefore the purpose of this qualitative descriptive study was to explore and describe the arthritic pain experience of school age children with juvenile rheumatoid arthritis (JRA) and to understand the impact/influence of various factors on the construction of that experience. Ten children, aged 10 to 13 years, with either early (at 2 to 4 years) or la

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8

Gutowska-Owsiak, Danuta. "NKT Cells in Rheumatoid Arthritis." Thesis, University of Liverpool, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526938.

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9

Eurenius, Eva. "Physical activity in rheumatoid arthritis /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-697-2/.

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10

Reynolds, Sophie L. "Vascular dysfunction in rheumatoid arthritis." Thesis, Cardiff University, 2010. http://orca.cf.ac.uk/54162/.

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These findings suggest that systemic and vascular wall levels of matrix metalloproteinase-9, related to inflammation at the joint site, may play a prominent role in the development of vascular dysfunction in this experimental model. This thesis goes someway to elucidating the potential mechanisms of vascular dysfunction in rheumatoid arthritis.

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11

Wright, Helen Louise. "Neutrophil Function in Rheumatoid Arthritis." Thesis, University of Liverpool, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.510936.

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12

Emery, P. "Immune responses in rheumatoid arthritis." Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598845.

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13

Duke, O. L. "Immunological observations in rheumatoid arthritis." Thesis, University of Cambridge, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598674.

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14

Rantapää, Dahlqvist Solbritt. "Genetic markers in rheumatoid arthritis." Doctoral thesis, Umeå universitet, Reumatologi, 1985. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-101305.

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Genetic as well as environmental factors are believed to be of importance in the etiology of rheumatoid arthritis (RA). There are a number of previous studies of genetic markers in RA, but so far no genetic linkage and only a few associations have been found. Of the associations only one (with the HLA antigen DR4) appears to be well documented. In most previous association studies the patients have not been divided according to sex and family history of RA. In this investigation the HLA antigens A, B and DR and five serum protein systems (Bf, C3, Pi, Hp and Tf) were studied in patients with er

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15

Lacroix, Brigitte. "Pharmacometric Modeling in Rheumatoid Arthritis." Doctoral thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-247917.

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Biologic therapies have revolutionized the treatment of rheumatoid arthritis, a common chronic inflammatory disease, mainly characterized by the chronic inflammation of the joints. The activity and progression of the disease are highly variable, both between subjects and between the successive assessments for the same subject. Standardized assessments of clinical variables have been developed to reflect the disease activity and evaluate new therapies. Pharmacokinetics-pharmacodynamic (PKPD) models and methods for analyzing the generated time-course data are needed to improve the interpretation

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16

Pritchard, M. L. "Psychological aspects of rheumatoid arthritis." Thesis, University of Exeter, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381050.

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17

Martin, Rosemary H. "Dietary factors in rheumatoid arthritis." Thesis, University of Ulster, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268590.

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18

Houssien, Dhiya Taj Alhaj. "Outcome studies in rheumatoid arthritis." Thesis, King's College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298543.

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19

Bedwell, A. E. "Immunological abnormalities of rheumatoid arthritis." Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372005.

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20

Adlan, Ahmed. "Autonomic function in rheumatoid arthritis." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6705/.

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Rheumatoid arthritis (RA) is a chronic inflammatory condition with poorly understood pathophysiology and increased cardiovascular risk. The mechanisms for increased cardiovascular risk are not fully known, however one novel mechanism explored in this thesis is autonomic nervous system (ANS) dysfunction. The thesis comprises of: a systematic literature review; two case-control studies (n=30 RA patients, n=34 controls; a longitudinal case-study (n=1 RA patient)); a cohort study (n=112 RA patients); and a randomised placebo controlled crossover study (n=10 healthy controls). The work presented in

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21

Hildalgo, Ester. "T cells in Rheumatoid Arthritis." Thesis, University of Birmingham, 2011. http://etheses.bham.ac.uk//id/eprint/1715/.

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Identification of the role of T cells and their interaction with other cell types remains a major challenge to our understanding of the pathogenesis of rheumatoid arthritis. In this study we have investigated the regulation of the response of T cells infiltrating the rheumatoid joint to IL-6. Furthermore we have investigated the level of T cell activation in the early stages of rheumatoid arthritis. Interleukin-6 is an important regulator of T cell differentiation and survival. It exerts its biological function by either directly binding to the complete IL-6 receptor consisting of CD126 CD130

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22

Hutchinson, David. "Cigarette smoking and rheumatoid arthritis." Thesis, University of Leicester, 2003. http://hdl.handle.net/2381/29431.

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The principle aim of this study was to test the hypothesis that heavy smoking is an aetiological factor in RA and generates a distinct subgroup of the disease definable in terms of clinical phenotype, particularly severity. A second aim was to investigate possible molecule mechanisms linking smoking with RA and what I believe to be candidate mechanisms involving the detoxifying glutathione S transferase Mu 1 (GST M1) gene and oxidative damage to alpha 1 proteinase (alpha1 PI). These studies involved a review of the literatures regarding the link between RA and both smoking and alpha1 PI defici

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23

Fang, Jierui. "Responsive wearables for rheumatoid arthritis." Thesis, Massachusetts Institute of Technology, 2020. https://hdl.handle.net/1721.1/127855.

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Thesis: S.B. in Art and Design, Massachusetts Institute of Technology, Department of Architecture, May, 2020<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references (pages 34-36).<br>The purpose of this thesis is to investigate and create more responsive and adaptive assistive technology for patients with rheumatoid arthritis (RA), using computational design methods to embed individualized data within the design and materiality. Rheumatoid arthritis is a chronic, autoimmune disease that attacks the joints and causes progressive deformity and bone erosion directed mostly

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24

Scott, Ian Clifford. "Risk prediction in rheumatoid arthritis." Thesis, King's College London (University of London), 2014. https://kclpure.kcl.ac.uk/portal/en/theses/risk-prediction-in-rheumatoid-arthritis(e69fd700-7819-41d6-96ae-dc26e1896e1a).html.

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As rheumatoid arthritis (RA) is a heterogeneous disease whose course and treatment response varies between patients, a stratified approach to its management is required. This thesis aimed to facilitate the risk prediction that underpins stratified medicine in RA. Its primary aim was to improve the knowledge of which clinical and genetic factors predict RA’s onset, disease course and treatment responses. Its secondary aim was to develop a prediction modelling framework that harnessed these factors to inform clinical care. There were five key findings. Firstly, it demonstrated a significant inve

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25

MacKay, Kirsten Robyn. "Genetic susceptibility to rheumatoid arthritis." Thesis, University of Bath, 2003. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288240.

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The heritability of Rheumatoid Arthritis is approximately 60%. Although association with the HLA region is well recognised, these genes account for only 40% of the total genetic contribution. Linkage mapping and association studies need to proceed in parallel to identify which non-MHC genes are involved in the remaining 60% of the genetic contribution. The work described in this thesis represents a systematic approach to identifying these non-HLA effects, using genome-wide linkage mapping and candidate gene methods. To identify regions exhibiting genetic linkage to rheumatoid arthritis a syste

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26

Spooner, Luke. "Preventing rheumatoid arthritis : understanding factors that influence decisions to take preventative treatments for rheumatoid arthritis." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/62681.

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27

Iaquinta, Monica L. "The phenomenological lived experience of rheumatoid arthritis." Morgantown, W. Va. : [West Virginia University Libraries], 2001. http://etd.wvu.edu/templates/showETD.cfm?recnum=1842.

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28

Chan, Vivien. "Use of leflunomide in rheumatoid arthritis /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18573.pdf.

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29

Bishop, Carole Marie. "Coping with pain in rheumatoid arthritis." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/29207.

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This research investigated the role of coping strategies in reducing the pain experience of rheumatoid arthritis (RA) patients over a seven-day period. Sixty-three patients completed a twice-daily structured dairy consisting of an eight scale revision of the Ways of Coping (WOC), the depression subscale of the Affects Balance Scale (ABS), and a pain visual analogue scale (VAS). Multivariate analyses for repeated measures identified two coping strategies, Self-Care and Positive Reappraisal, as significantly effective in pain reduction. Self-Care includes behavioral attempts to manage the sympto

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30

Harney, SineÌad M. J. "Major histocompatability genetics of rheumatoid arthritis." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419294.

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31

Saravanan, Vadivelu. "Small Airway Obstruction in Rheumatoid Arthritis." Thesis, University of Newcastle Upon Tyne, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519460.

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32

Mewar, Devesh. "Studies on autoantigens in rheumatoid arthritis." Thesis, University of Sheffield, 2003. http://etheses.whiterose.ac.uk/3455/.

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This thesis describes the use of phage display for the isolation of autoantigens in rheumatoid arthritis. The potential of the technology is demonstrated by the isolation of an autoantigen, eukaryotic translation elongation factor 1a1 (eEF 1 (x 1)) from a fibroblast cDNA library using rounds of selective enrichment with IgG from RA patients. Subsequently in order to isolate joint-specific antigens a phage-displayed cDNA library from rheumatoid pannus was generated and screened with analogous procedures. From the clones isolated, putative candidate autoantigens were identified. The presence of

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33

Abdel-Nour, A. N. "Cell mediated immunity in rheumatoid arthritis." Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375007.

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34

Côrte, Ana Filipa Terleira Camacho da. "Cervical spine instability in rheumatoid arthritis." Master's thesis, Faculdade de Medicina da Universidade do Porto, 2010. http://hdl.handle.net/10216/61051.

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35

Adams, Joanna Elizabeth. "Hand function in early rheumatoid arthritis." Thesis, University of Southampton, 2006. https://eprints.soton.ac.uk/368402/.

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This thesis is about what people can do with their hands in the early stages of rheumatoid arthritis (RA). The wrist and hand are affected early in RA. Wrist and hand joint swelling, pain and deformity are all likely to contribute to the functional performance of the hand. Static splinting alongside joint protection education and active exercise is one of the most common conservative intervention strategies used by therapists. Splinting attempts to reduce local inflammation and pain, correctly position joints, minimise the occurrence or progression of deformity and maintain hand function. This

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36

Olinyk, O. Yu. "Metabolic syndrome in rheumatoid arthritis patients." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18595.

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37

Germond, Sean Alan. "Rheumatoid arthritis : a cognitive-behavioural intervention." Master's thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/13551.

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Bibliography: leaves 190-206.<br>This study investigated both the mediating role of psychological adjustment in determining pain experience, disease · status, and immune function in Rheumatoid Arthritis (RA), and the value of cognitive-behavioural intervention in improving the overall health status of such patients. Two related hypotheses were tested in a matched-random assigned two-groups design, with pre-, mid-, and post-intervention assessment. Fourteen (N = 14) female RA outpatients, selected along established inclusion criteria, were allocated to either treatment (n=8) or control (n=6) gr

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38

Prahalad, Sampath. "Juvenile Rheumatoid Arthritis and Familial Autoimmunity." University of Cincinnati / OhioLINK, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=ucin991251421.

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39

Meltzer, Janet R. "Psychological adjustment in juvenile rheumatoid arthritis /." The Ohio State University, 1987. http://rave.ohiolink.edu/etdc/view?acc_num=osu148758564557603.

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40

Hammaker, Deepa Rajan. "Monoclonal antibody therapy of rheumatoid arthritis." Diss., The University of Arizona, 1999. http://hdl.handle.net/10150/289074.

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Objectives. To (a) determine the immunological effects of a PRIMATIZED® anti-CD4 antibody alone or in combination with methotrexate in RA patients, (b) determine the immunological effects of a chimeric anti-CD25 antibody in RA patients who are partially refractive to methotrexate and (c) compare interleukin-15 levels in the serum of RA patients and healthy controls and determine if there is a correlation between this cytokine and serum TNF-α, CD 122 expression, and disease activity. Patients and methods. (a) Eight RA patients were selected, four received anti-CD4+ placebo and the other four re

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41

Côrte, Ana Filipa Terleira Camacho da. "Cervical spine instability in rheumatoid arthritis." Dissertação, Faculdade de Medicina da Universidade do Porto, 2010. http://hdl.handle.net/10216/61051.

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42

Oeser, Christian. "Polymorphismen in Kandidatengenen der Apoptose als genetische Risikofaktoren für Rheumatoide Arthritis." Doctoral thesis, Universitätsbibliothek Leipzig, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-89381.

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Die Rheumatoide Arthritis (RA) ist eine chronisch-entzündliche Systemerkrankung des Bindegewebes mit autoimmunem Charakter. In dieser Studie wurden 7 Kandidatengene, welche in zentrale Abläufe der Apoptose involviert sind (CFLAR, XIAP, NFKB1, RELA, BCL2L1, FAS, FASLG), selektiert. Innerhalb dieser Gene wurden 23 Einzel-Basen-Polymorphismen (single nucleotide polymorphisms bzw. SNPs) sowie ein Insertions-Deletions-Polymorphismus in 300 französich-kaukasischen Individuen (100 RA-Trio-Familien) mittels Einzelbasenverlängerung (Single Base Extension bzw. SBE) in einer massenspektrometrischen Analy

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43

Edwards, Bryan Michael. "Collagen epitopes in arthritis." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265003.

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44

Gibbon, Wayne William. "Imaging in inflammatory arthritis : a multidisciplinary team approach /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18870.pdf.

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45

Muhammad, Khalid. "Longterm impact of anti-CD20 mediated transient B cell depletion on memory B cells in patients with rheumatoid arthritis." Doctoral thesis, kostenfrei, 2009. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-36319.

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B-Lymphozyten leisten unterschiedliche Beiträge zur Pathophysiologie der Rheumatoiden Arthritis. Sie produzieren Autoantikörper, präsentieren Autoantigene und schütten verschiedene Zytokine, die am proinflammatorischen Prozess beteiligt sind, aus. Aufbauend auf diesen Ergebnissen wurden in den letzten Jahren Therapien entwickelt, die gezielt B-Lymphozyten ansteuern um direkt oder indirekt in den autoimmunen Krankheitsverlauf einzugreifen. Die zeitlich begrenzte B-Zell-Depletion mit Rituximab (anti CD20-Antikörper) hat dabei in den letzten Jahren einen hohen Stellenwert erlangt und wird im klin

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46

Brentano, Ackermann Fabia. "Toll-like receptor signalling in rheumatoid arthritis /." [S.l.] : [s.n.], 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000281181.

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47

Kamarova, Halina. "CD4+161+ T Cells in Rheumatoid Arthritis." Thesis, University of Liverpool, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.490658.

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Rheumatoid arthritis (RA) patients develop a chronic inflammation in synovial joints and often in other tissues. Since efforts to isolate microbial agents from the involved sites have failed to confirm an infectious origin, it is generally agreed that RA is an autoimmune disease. However, the mechanisms initiating and maintaining the abnormal immune response remain poorly understood. The number ofNKT cells is reduced in several human autoimmune disorders and in animal models of autoimmunity. The purpose of this study was to investigate whether the frequency of CD161+ NKT cells in RA correlates

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48

Mcgovern, Amanda Jane. "Functional characterisation of rheumatoid arthritis risk loci." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/functional-characterisation-of-rheumatoid-arthritis-risk-loci(9c2cfbf0-3a1e-424f-942e-a58b108b7b94).html.

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Rheumatoid arthritis (RA) is a complex autoimmune disease affecting approximately 1% of the population. Multiple factors contribute to the development of RA, with genetic factors accounting for around 60% of the disease risk. Over the last few years, genome-wide association studies (GWAS) have successfully been used to identify regions of the genome predisposing to complex disease. There are now 101 confirmed RA risk loci, but for the vast majority of these loci the causal gene and causal variant remain unidentified and therefore, their function in disease is unexplored. The majority of geneti

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49

Roy, Suzette M. "Living with chronic pain of rheumatoid arthritis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq21268.pdf.

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50

Miranda, Letícia Algarves. "Rheumatoid arthritis as a modifier of periodontitis /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-047-3/.

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