Thematische Bibliographien / Resistance

Auswahl der wissenschaftlichen Literatur zum Thema „Resistance“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 22. Juni 2024

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Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Zeitschriftenartikel zum Thema "Resistance":

1

Pink, D. A. C., und P. Hand. „Plant resistance and strategies for breeding resistant varieties“. Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (01.01.2002): S9—S14. http://dx.doi.org/10.17221/10310-pps.

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An explanation of the ‘boom-bust’ cycle of resistance breeding was provided by the gene-for-gene relationship between a pathogen and its host. Despite this understanding, most R genes continued to be deployed singly and resistance has been ephemeral. The reasons for breeding ‘single R gene’ varieties are discussed. Alternative strategies for the deployment of R genes and the use of quantitative race non-specific resistance have been advocated in order to obtain durable resistance. The feasibility of both of these approaches is discussed taking into account the impact of technologies such as plant transformation and marker-assisted selection. A change in focus from durability of the plant phenotype to that of the crop phenotype is advocated.
2

Çelik, Cem. „Increasing antimicrobial resistance in nosocomial pathogens; multidrug-resistant extensively drug-resistant and pandrug-resistant Acinetobacter baumannii“. Journal of Microbiology and Infectious Diseases 4, Nr. 1 (01.03.2014): 7–12. http://dx.doi.org/10.5799/ahinjs.02.2014.01.0116.

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3

Gokalp, Deniz, Alpaslan Tuzcu, Ster Irmak, Mithat Bahceci und Ozlem Demirpence. „FREQUENCY OF ASPIRIN RESISTANCE IN PATIENTS WITH TYPE 1 AND 2 DIABETES MELLITUS AND ITS ASSOCIATION WITH METABOLIC PARAMETERS“. International Journal of Surgery and Medicine 2, Nr. 3 (2016): 122. http://dx.doi.org/10.5455/ijsm.aspirin-resistance-diabetes.

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4

Kumar, Saneesh, Priya Mitra und Rupak Roy. „Antibiotic Resistance Profile of Amoxicillin-Resistant Bacteria Isolated from Chicken Liver“. International Journal of Research Publication and Reviews 5, Nr. 1 (24.01.2024): 5607–32. http://dx.doi.org/10.55248/gengpi.5.0124.0366.

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5

Андреевская, С. Н., Т. Г. Смирнова, Е. Е. Ларионова, И. Ю. Андриевская, Л. Н. Черноусова und А. Эргешов. „Изониазид-резистентные Mycobacterium tuberculosis: частота выявления, спектры резистентности и генетические детерминанты устойчивости“. Phylogenetic Analysis, Nr. 1;2020 (2020): 22–28. http://dx.doi.org/10.24075/vrgmu.2020.001.

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Отсутствие ускоренной диагностики туберкулеза с устойчивостью возбудителя к изониазиду с сохраненной чувствительностью к рифампицину (ИР-ТБ) может быть причиной низкой эффективности терапии и приводить к амплификации лекарственной резистентности, в том числе к формированию множественной лекарственной устойчивости. Целью работы было определить частоту встречаемости ИР-ТБ в современной популяции, охарактеризовать фенотипическую чувствительность и генетические детерминанты устойчивости к изониазиду представителей этой группы M. tuberculosis на репрезентативном материале. Анализировали результаты определения лекарственной чувствительности, полученные при исследовании молекулярно- генетическими и/или культуральными методами изолятов M. tuberculosis / ДНК M. tuberculosis, выделенных от больных туберкулезом легких из клинических отделений Центрального научно-исследовательского институа туберкулеза за период 2011–2018 гг. Частота ИР-ТБ составила 12% от всех выявленных случаев туберкулеза. M. tuberculosis с ИР были как монорезистентными к изониазиду (45%), так и полирезистентными (устойчивыми к 2–6 противотуберкулезным препаратам), а устойчивость к изониазиду была обусловлена мутациями в гене katG, приводящими к высокому уровню резистентности. На основании анализа литературных данных и собственных наблюдений подчеркивается важность разработки и внедрения новых простых молекулярных тестов для определения устойчивости одновременно к рифампицину и изониазиду.
6

Singh, Amresh Kumar. „Resistance patterns and trends of extensively drug-resistant tuberculosis: 5-year experience“. Journal of Microbiology and Infectious Diseases 03, Nr. 04 (01.12.2013): 169–75. http://dx.doi.org/10.5799/ahinjs.02.2013.04.0103.

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7

Kumar, Sinha Ritesh, und Chandra Satish. „Insulin resistance“. Asian Pacific Journal of Health Sciences, Supplimentary 2014 (2014): 71–78. http://dx.doi.org/10.21276/apjhs.2014.1.1s.15.

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K Mammen, Ninu. „Antimicrobial Resistance“. International Journal of Science and Research (IJSR) 12, Nr. 5 (05.05.2023): 1207. http://dx.doi.org/10.21275/sr23512132813.

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9

Wang, S. C., und P. S. Wei. „Modeling Dynamic Electrical Resistance During Resistance Spot Welding“. Journal of Heat Transfer 123, Nr. 3 (28.11.2000): 576–85. http://dx.doi.org/10.1115/1.1370502.

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Dynamic electrical resistance during resistance spot welding has been quantitatively modeled and analyzed in this work. A determination of dynamic resistance is necessary for predicting the transport processes and monitoring the weld quality during resistance spot welding. In this study, dynamic resistance is obtained by taking the sum of temperature-dependent bulk resistance of the workpieces and contact resistances at the faying surface and electrode-workpiece interface within an effective area corresponding to the electrode tip where welding current primarily flows. A contact resistance is composed of constriction and film resistances, which are functions of hardness, temperature, electrode force, and surface conditions. The temperature is determined from the previous study in predicting unsteady, axisymmetric mass, momentum, heat, species transport, and magnetic field intensity with a mushy-zone phase change in workpieces, and temperature and magnetic fields in the electrodes of different geometries. The predicted nugget thickness and dynamic resistance versus time show quite good agreement with available experimental data. Excluding expulsion, the dynamic resistance curve can be divided into four stages. A rapid decrease of dynamic resistance in stage 1 is attributed to decreases in contact resistances at the faying surface and electrode-workpiece interface. In stage 2, the increase in dynamic resistance results from the primary increase of bulk resistance in the workpieces and an increase of the sum of contact resistances at the faying surface and electrode-workpiece interface. Dynamic resistance in stage 3 decreases, because increasing rate of bulk resistance in the workpieces and contact resistances decrease. In stage 4 the decrease of dynamic resistance is mainly due to the formation of the molten nugget at the faying surface. The molten nugget is found to occur in stage 4 rather than stage 2 or 3 as qualitatively proposed in the literature. The effects of different parameters on the dynamic resistance curve are also presented.
10

Shahina, Sk jasmine, Sheeba Ali Siddiqui und Dr Padma Krishnan. „Nasal Carriage of Methicillin Resistant Staphylococci with Inducible Clindamycin Resistance and Pvl Gene“. International Journal of Scientific Research 3, Nr. 6 (01.06.2012): 392–94. http://dx.doi.org/10.15373/22778179/june2014/132.

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Zeitschriftenartikel Dissertationen Bücher

Dissertationen zum Thema "Resistance":

1

Börjesson, Stefan. „Antibiotic Resistance in Wastewater : Methicillin-resistant Staphylococcus aureus (MRSA)and antibiotic resistance genes“. Doctoral thesis, Linköpings universitet, Medicinsk mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-17709.

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A large part of the antibiotics consumed ends up in wastewater, and in the wastewater the antibiotics may exert selective pressure for or maintain resistance among microorganisms. Antibiotic resistant bacteria and genes encoding antibiotic resistance are commonly detected in wastewater, often at higher rates and concentrations compared to surface water. Wastewater can also provide favourable conditions for the growth of a diverse bacterial community, which constitutes a basis for the selection and spread of antibiotic resistance. Therefore, wastewater treatment plants have been suggested to play a role in the dissemination and development of antibiotic resistant bacteria. Methicillin-resistant Staphylococcus aureus (MRSA) is a large problem worldwide as a nosocomial pathogen, but knowledge is limited about occurrence in non-clinical environments, such as wastewater, and what role wastewater plays in dissemination and development of MRSA.   In this thesis we investigated the occurrence of MRSA in a full-scale wastewater treatment plant (WWTP). We also investigated the concentration of genes encoding resistance to aminoglycosides (aac(6’)-Ie+aph(2’’)), β-lactam antibiotics (mecA) and tetracyclines (tetA and tetB) in three wastewater-associated environments: (1) soil from an overland flow area treating landfill leachates, (2) biofilm from a municipal wastewater treatment plant, and (3) sludge from a hospital wastewater pipeline. In addition, concentrations of mecA, tetA and tetB were investigated over the treatment process in the WWTP. These investigations were performed to determine how the prevalence and concentration of MRSA and the antibiotic resistence genes are affected in wastewater and wastewater treatment processes over time. The occurrence of MRSA was investigated by cultivation and a commercially available real-time PCR assay. In order to determine concentrations of the genes aac(6’)-Ie+aph(2’’), mecA, tetA and tetB in wastewater we developed a LUXTM real-time PCR assay for each gene.   Using cultivation and real-time PCR we could for the first time describe the occurrence of MRSA in wastewater and show that it had a stable occurrence over time in a WWTP. MRSA could mainly be detected in the early treatment steps in the WWTP, and the wastewater treatment process reduced the number and diversity of cultivated MRSA. However, our results also indicate that the treatment process selects for strains with more extensive resistance and possibly higher virulence. The isolated wastewater MRSA strains were shown to have a close genetic relationship to clinical isolates, and no specific wastewater lineages could be detected, indicating that they are a reflection of carriage in the community. Taken together, these data indicate that wastewater may be a potential reservoir for MRSA and that MRSA are more prevalent in wastewater than was previously thought.   The real-time PCR assays, for aac(6’)-Ie+aph(2’’), mecA, tetA, and tetB that we developed, were shown to be sensitive, fast, and reproducible methods for detection and quantification of these genes in wastewater environments. The highest concentrations of all genes were observed in the hospital pipeline, and the lowest in the overland flow system, with tetA and aac(6´)-Ie+aph(2´´) detected in all three environments. In the full-scale WWTP, we continuously detected mecA, tetA and tetB over the treatment process and over time. In addition, it was shown that the treatment process reduces concentrations of all three genes. The data presented in this thesis also indicate that the reduction for all three genes may be connected to the removal of biomass, and in the reduction of tetA and tetB, sedimentation and precipitation appear to play an important role.
2

OUNNOUGHENE, MARC Kahn Jean-Pierre. „LA DEPRESSION RESISTANTE QUELS TRAITEMENTS POUR QUELLE RESISTANCE ? /“. [S.l.] : [s.n.], 2000. http://www.scd.uhp-nancy.fr/docnum/SCDMED_T_2000_OUNNOUGHENE_MARC.pdf.

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3

Pfeltz, Richard F. Wilkinson Brian J. „The effects of vancomycin resistance selection and magnesium on resistance expression in methicillin-resistant Staphylococcus aureus“. Normal, Ill. Illinois State University, 1999. http://wwwlib.umi.com/cr/ilstu/fullcit?p9927774.

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Thesis (Ph. D.)--Illinois State University, 1999.
Title from title page screen, viewed July 20, 2006. Dissertation Committee: Brian J. Wilkinson (chair), Radheshyam K. Jayaswal, Alan J. Katz, Anthony J. Otsuka, David L. Williams. Includes bibliographical references and abstract. Also available in print.
4

Butler, C. A. „Mechanisms of steroid resistance in therapy resistant asthma“. Thesis, Queen's University Belfast, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.546017.

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5

Maffeis, Laura <1981&gt. „Correlation between insulin resistance and treatment-resistant acne“. Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5654/1/maffeis_laura_tesi.pdf.

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Physiologically during puberty and adolescence, when juvenile acne usually appears, the response to a glucose load is increased if compared to the one observed in adult and at pre-pubertal age, while insulin sensitivity is reduced. Insulin is a hormone that acts at different levels along the axis which controls the sex hormones. It increases the release of LH and FSH by pituitary gland, stimulates the synthesis of androgens in the gonads and stimulates the synthesis of androgenic precursors in adrenal glands. Finally, it acts in the liver by inhibiting the synthesis of Sex Hormone Binding Globulin (SHBG). Insulin is also able to act directly on the production of sebum and amplify the effects of Iinsulin Growth Factor-1 in the skin, inhibiting the synthesis of its binding protein (IGF Binding Protein-1). In female subjects with acne and Polycystic Ovary Syndrome (PCOS) insulin resistance is a well known pathogenetic factor, while the relationship between acne and insulin resistance has been poorly investigated in males so far. The purpose of this study is to investigate the correlation between insulin resistance and acne in young males who do not respond to common therapies. Clinical and biochemical parameters of glucose, lipid metabolism, androgens and IGF-1 were evaluated. Insulin resistance was estimated by Homeostasis Model assessment (HOMA-IR) and Oral Glucose Tolerance Test was also performed. We found that subjects with acne had higher Sistolic and Diastolic Blood Pressure, Waist/Hip Ratio, Waist Circumference, 120' OGTT serum insulin and serum IGF-1 and lower HDL-cholesterol than subjects of comparable age and gender without acne. The results thus obtained confirmed what other authors have recently reported about a metabolic imbalance in young males with acne. Furthermore, these results support the hypothesis that insulin resistance might play an important role in the pathogenesis of treatment-resistant acne in males.
6

Maffeis, Laura <1981&gt. „Correlation between insulin resistance and treatment-resistant acne“. Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5654/.

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Physiologically during puberty and adolescence, when juvenile acne usually appears, the response to a glucose load is increased if compared to the one observed in adult and at pre-pubertal age, while insulin sensitivity is reduced. Insulin is a hormone that acts at different levels along the axis which controls the sex hormones. It increases the release of LH and FSH by pituitary gland, stimulates the synthesis of androgens in the gonads and stimulates the synthesis of androgenic precursors in adrenal glands. Finally, it acts in the liver by inhibiting the synthesis of Sex Hormone Binding Globulin (SHBG). Insulin is also able to act directly on the production of sebum and amplify the effects of Iinsulin Growth Factor-1 in the skin, inhibiting the synthesis of its binding protein (IGF Binding Protein-1). In female subjects with acne and Polycystic Ovary Syndrome (PCOS) insulin resistance is a well known pathogenetic factor, while the relationship between acne and insulin resistance has been poorly investigated in males so far. The purpose of this study is to investigate the correlation between insulin resistance and acne in young males who do not respond to common therapies. Clinical and biochemical parameters of glucose, lipid metabolism, androgens and IGF-1 were evaluated. Insulin resistance was estimated by Homeostasis Model assessment (HOMA-IR) and Oral Glucose Tolerance Test was also performed. We found that subjects with acne had higher Sistolic and Diastolic Blood Pressure, Waist/Hip Ratio, Waist Circumference, 120' OGTT serum insulin and serum IGF-1 and lower HDL-cholesterol than subjects of comparable age and gender without acne. The results thus obtained confirmed what other authors have recently reported about a metabolic imbalance in young males with acne. Furthermore, these results support the hypothesis that insulin resistance might play an important role in the pathogenesis of treatment-resistant acne in males.
7

Barnawi, Ibrahim. „Mechanisms of apoptosis resistance in chemotherapy-resistant cancer cells“. Thesis, University of Reading, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627931.

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Most chemotherapy drugs work through the induction of apoptosis in tumour cells. Mutations in apoptotic pathways, notably p53 and Bcl-2, are common in cancer and associated with increased resistance to apoptosis and therefore to chemotherapy. The sensitivity of two types of cells have been compared, A 172 human glioblastoma cells and MDA-MB-23l human breast cancer cells, for their sensitivity to the chemotherapy drugs cisplatin, doxorubicin, gemcitabine, vincristine and temozolomide. Data shows that there were differences in the sensitivity of breast cancer and glioma cells to a variety of chemotherapy drugs. There were differences in nitric oxide synthase (NOS) expression between the two different types of cancer cells and yet a role was not observed for nitric oxide in changing the expression of the Bcl-2 family of proteins (both pro- or antiapoptotic). Additionally, NOS expression changed following treatment with chemotherapy drugs, suggesting that this may be the mechanism by which the cells become resistant.
8

Pelfrey, Clara Mackenzie. „Genetic resistance to experimental autoimmune encephalomyelitis : analysis of the resistance mechanisms in the Lewis resistant or LeR rat /“. The Ohio State University, 1988. http://rave.ohiolink.edu/etdc/view?acc_num=osu148759742413614.

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9

Cornick, Jen. „Roadmap to resistance : antimicrobial resistance in Malawian pneumococci“. Thesis, University of Liverpool, 2012. http://livrepository.liverpool.ac.uk/11173/.

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Multi-drug resistant (MDR) Streptococcus pneumoniae are a major public health concern worldwide. In Malawi a resource poor country, even the simplest antimicrobials remain a precious commodity. Given the limited number of antimicrobials available for the management of MDR invasive pneumococcal disease (IPD) measures need to be implemented to limit the spread of resistance. In order to design such measures it is essential that we gain a better understanding of the evolution of antimicrobial resistance in this setting. The purpose of this thesis was to assess the molecular basis and mode of dissemination of antimicrobial resistance in S. pneumoniae with the aim of identifying a biomarker of antimicrobial resistance that could be used to design a diagnostic PCR to assist epidemiological surveillance of antimicrobial resistance and inform treatment policy. Malawi introduced the 13-valent pneumococcal conjugate vaccine (PCV13) in 2011. To provide baseline data to assess the impact of PCV13 all invasive pneumococci isolated from children admitted to Queen Elizabeth Central Hospital, Malawi 2004-2006 were serotyped and subjected to antimicrobial susceptibility testing. The data suggested PCV13 will not provide protection against 61% of penicillin resistant pneumococci and if serotype replacement occurs following the introduction of PCV13, the incidence of penicillin resistant IPD could therefore increase Over 130 resistant and susceptible pneumococcal isolates from carriage and invasive disease were subjected to whole genome sequencing. The employment of an in vitro and in silco analytical approach established that S. pneumoniae employs a diverse array of antimicrobial resistance mechanisms, the dissemination of which is likely to be driven by high antimicrobial consumption. A relatively high incidence of antimicrobial resistant was observed in serotype 1 pneumococci, the most common cause of IPD in Malawi. This serotype is not usually associated with resistance in other geographic locations, the short duration of serotype 1 carriage is assumed to limit the chance it has to acquire resistance mechanisms via recombination. Interestingly the resistance mechanisms employed by serotype 1 had been acquired through multiple recombination events. Recombination was evidenced to contribute to >90% of the variation in the serotype 1 genomes. To allow the identification of resistance biomarkers free from any preconceptions about which genes are involved in resistance, multiple antimicrobial resistant lineages were generated in vitro. Isolates were sequenced at several time points as resistance developed. Comparison of the resistant isolates to the wild type isolates identified single nucleotide polymorphisms in 46 genes, 40 of these genes have not previously been implicated in antimicrobial resistance. The role of these genes in resistance warrants further investigation. The analysis suggests that rather than a single biomarker future research needs to identify multiple biomarkers; the dynamic nature of this organism means that it can adopt one of many routes to antimicrobial resistance.
10

Chung, Whasun Oh. „Macrolide resistance and its linkage to tetracycline resistance /“. Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/9279.

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Bücher zum Thema "Resistance":

1

Shreve, Anita. Resistance. Bath: Chivers, 1996.

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Amadahy, Zainab. Resistance. Toronto, Ontario]: [Zainab Amadahy], 2013.

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Shreve, Anita. Resistance. New York: Little, Brown and Company, 2008.

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Graber, Janet. Resistance. New York: Marshall Cavendish, 2005.

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Nielsen, Jennifer A. Resistance. New York: Scholastic, Incorporated, 2018.

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McCay, Bill. Resistance. New York, N.Y: New American Library, 1999.

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1967-, Carter Michael, und National AIDS manual, Hrsg. Resistance. 5. Aufl. London: NAM, 2004.

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Zeidenstein, Sondra. Resistance. Goshen, Conn: Chicory Blue Press, 2003.

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manual, National AIDS. Resistance. London: NAM Publications, 1999.

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Dillard, J. M. Resistance. New York: Pocket Books, 2007.

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Buchteile zum Thema "Resistance":

1

Shkurti, Gentian. „Unframe Your Polis“. In Resistance, 136–47. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.08.

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HAVEIT. „Shaving Patriarchy“. In Resistance, 160–67. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.10.

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Stamenkoviç, Marko. „Editorial Note“. In Resistance, 6–11. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.02.

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Romano, Stefano. „Time As Resistance in Public Space“. In Resistance, 178–89. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.12.

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Budina, Edmond, Ervin Goci und Lindita Komani. „"An Opportunity for Albania to Expand Its Concept of Public Property"“. In Resistance, 108–17. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.05.

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Hoçia, Blerta. „Heterotopias of Resistance“. In Resistance, 30–31. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.03.

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Bedeni, Luçjan. „How Can a Museum Change a City?“ In Resistance, 148–59. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.09.

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Xhafa, Pleurad. „What Will Remain from Resistance“. In Resistance, 168–77. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.11.

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Zaloshnja, Ergin. „Water Sleeps, The Enemy Does Not Sleep“. In Resistance, 118–25. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.06.

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Qylafi, Leonard. „Images in Spite of All“. In Resistance, 126–35. 2. Aufl. Earth, Milky Way: punctum books, 2021. http://dx.doi.org/10.53288/0384.1.07.

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Konferenzberichte zum Thema "Resistance":

1

Grosjean, Luce. „Resistance“. In SA '17: SIGGRAPH Asia 2017. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3145631.3145660.

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Hasan, Nazmul. „Ballast Resistance: Discreet vs. Unit Resistance“. In International Conference on Transportation and Development 2022. Reston, VA: American Society of Civil Engineers, 2022. http://dx.doi.org/10.1061/9780784484371.005.

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Roush, Rick. „Resistance management in insect resistant genetically-engineered crops“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93214.

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Takahashi, S., M. Hatano, Y. Kojima, Y. Harada, A. Kawasaki und F. Ono. „Thermal Cycle Resistance of Oxidation-Resistant Metallic Coatings“. In ITSC2010, herausgegeben von B. R. Marple, A. Agarwal, M. M. Hyland, Y. C. Lau, C. J. Li, R. S. Lima und G. Montavon. DVS Media GmbH, 2010. http://dx.doi.org/10.31399/asm.cp.itsc2010p0695.

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Abstract Thermal cycle resistance of Ni-20Cr, Ni-50Cr and CoNiCrAlY coatings manufactured by air plasma spraying was investigated according to a Japanese Industrial Standard "Testing method for thermal cycle resistance of oxidation resistant metallic coatings (JIS H 8452)” established in 2008. The specimens were exposed at 1000 °C and 1093 °C in air under cyclic heating and cooling condition up to 100 times. The thermal cycle resistance of oxidation-resistant metallic coatings was found to depend strongly on the testing temperature and the chemical composition of the coating materials. In the thermal cycle test at 1000 °C, the remarkable failure was not observed in any specimen. However, in the thermal cycle test at 1093 °C, although the Ni-20Cr coating caused the spalling on the whole surface of coating, the Ni-50Cr and the CoNiCrAlY coatings exhibited the excellent thermal cycle resistance even after applying the thermal cycles of 100 times, The CoNiCrAlY coating showed the mass gain with increasing the numbers of thermal cycle due to the preferential oxidation between the splats of the thermal spray particles. Furthermore, the failure behavior of specimens was investigated in detail by SEM, XRD and EPMA etc.
5

Chiang, K. W. „Resistance extraction and resistance calculation in GOALIE?“ In the 1989 26th ACM/IEEE conference. New York, New York, USA: ACM Press, 1989. http://dx.doi.org/10.1145/74382.74503.

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6

Owen, Micheal D. K. „Herbicide Resistance“. In Proceedings of the First Annual Crop Production and Protection Conference. Iowa State University, Digital Press, 1991. http://dx.doi.org/10.31274/icm-180809-359.

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7

Sen, P., und J. A. Cocks. „Collision Resistance“. In The Safety of RO-RO Passenger Ships. RINA, 1990. http://dx.doi.org/10.3940/rina.roro.1990.03.

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8

Wu, T. H., J. E. Ho und P. S. Wei. „Dynamic electrical resistance effects in resistance spot welding“. In 2010 5th International Microsystems, Packaging, Assembly and Circuits Technology Conference (IMPACT). IEEE, 2010. http://dx.doi.org/10.1109/impact.2010.5699534.

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9

Thevanayagam, S., und N. Ecemis. „Effects of Permeability on Liquefaction Resistance and Cone Resistance“. In Geotechnical Earthquake Engineering and Soil Dynamics Congress IV. Reston, VA: American Society of Civil Engineers, 2008. http://dx.doi.org/10.1061/40975(318)92.

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„Contact Resistance Influence in Numerical Simulation of Resistance Sintering“. In The 34th European Modeling & Simulation Symposium. CAL-TEK srl, 2022. http://dx.doi.org/10.46354/i3m.2022.emss.018.

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Berichte der Organisationen zum Thema "Resistance":

1

Soupir, Michelle L., Matthew J. Helmers und Thomas B. Moorman. Antibiotic Resistant Bacteria and Resistance Genes in Crop Fields. Ames: Iowa State University, Digital Repository, 2013. http://dx.doi.org/10.31274/farmprogressreports-180814-640.

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2

Soupir, Michelle, Matt Helmers und Thomas Moorman. Antibiotic Resistant Bacteria and Resistance Genes in Crop Fields. Ames: Iowa State University, Digital Repository, 2015. http://dx.doi.org/10.31274/farmprogressreports-180814-802.

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3

Levin, Ilan, John Thomas, Moshe Lapidot, Desmond McGrath und Denis Persley. Resistance to Tomato yellow leaf curl virus (TYLCV) in tomato: molecular mapping and introgression of resistance to Australian genotypes. United States Department of Agriculture, Oktober 2010. http://dx.doi.org/10.32747/2010.7613888.bard.

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Tomato yellow leaf curl virus (TYLCV) is one of the most devastating viruses of cultivated tomatoes. Although first identified in the Mediterranean region, it is now distributed world-wide. Sequence analysis of the virus by the Australian group has shown that the virus is now present in Australia. Despite the importance of the disease and extensive research on the virus, very little is known about the resistance genes (loci) that determine host resistance and susceptibility to the virus. A symptom-less resistant line, TY-172, was developed at the Volcani Center which has shown the highest resistance level among all tested varieties. Preliminary results show that TY-172 is a good candidate to confer resistance to both TYLCV and to Tomato leaf curl virus (ToLCV) in Queensland conditions. Furthermore, Segregation analysis has previously indicated that the resistance is determined by 2-3 genes. In this proposal we aimed to substantiate that TY-172 can contribute to resistance breeding against TYLCV in Queensland, to develop DNA markers to advance such resistance breeding in both Israel and Queensland, and to exploit these markers for resistant breeding in Australian and Israeli lines. To map quantitative trait loci (QTLs) controlling TYLCVresistance in TY172, appropriate segregating populations were analyzed using 69 polymorphic DNA markers spanning the entire tomato genome. Results show that TYLCV resistance in TY172 is controlled by a previously unknown major QTL, originating from the resistant line, and four additional minor QTLs. The major QTL, termed Ty-5, maps to chromosome 4 and accounts for 39.7-to-46.6% of the variation in symptom severity among segregating plants (LOD score: 33-to-35). The minor QTLs, originated either from the resistant or susceptible parents, were mapped to chromosomes 1, 7, 9 and 11, and contributed 12% to the variation in symptom severity in addition to Ty-5. Further analysis of parental lines as well as large F₁, BC₁F₁, F₂ and BC₁F₂ populations originating from crosses carried out, in reciprocal manner, between TY172 and the susceptible processing line M-82 (LA3475) during spring-summer 2010, indicated that: (1) the minor QTLs we have previously identified are in effect not reproducible, (2)Ty-5 alone can yield highly resistant plants with practically no extra-chromosomal effects, and (3) the narrow-sense heritability estimate of resistance levels, attributed to additive factors responsive to selection, does not significantly deviate from 1. All of these results point to Ty-5 as the sole resistance locus in TY172 thus significantly increasing the likelihood of its successful molecular dissection. The DNA markers developed during the course of this study were transferred together with the TY172 genotype to Queensland. TY172 was crossed to a panel of Australian genotypes and the resulting populations were subjected to segregation analysis. Results showed that resistant locus, Ty-5, is highly reproducible in the Australian conditions as well. The Australian group was also able to make improvements to the marker assays by re-designing primer pairs to provide more robust PCR fragments. The Ty-5 locus has now been introgressed into elite Australian germplasm and selection for TYLCV resistance has begun. Cumulatively, our results show that Ty-5 can be effectively used, together with the TY172 genotype to expedite TYLCV resistance breeding and improve our understanding of the genetics that underline the response of tomato to TYLCV. Contributions to agriculture include: (1) the development of tools for more efficient resistance breeding, allowing the incorporation of resistance to local tomato varieties in Australia, Israel and elsewhere; and (2) establish a solid framework for a future attempt to clone the genes that encode such resistance. The latter will enable to decipher the resistance mechanisms that could be applied to other geminiviruses in tomato and possibly in other plant species.
4

Spiekhout, J. R9218-3a Designing for Gouge-Resistance and Puncture-Resistance. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), Januar 1993. http://dx.doi.org/10.55274/r0011822.

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5

Ramirez, Abelardo L., William D. Daily und Andrew M. Binley. ELECTRICAL RESISTANCE TOMOGRAPHY. Office of Scientific and Technical Information (OSTI), Juni 2000. http://dx.doi.org/10.2172/15010154.

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6

Oettinger, Frank F., und David L. Blackburn. Thermal resistance measurements. Gaithersburg, MD: National Institute of Standards and Technology, 1990. http://dx.doi.org/10.6028/nist.sp.400-86.

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7

Al-Merjan, Sami M. The Kuwaiti Resistance. Fort Belvoir, VA: Defense Technical Information Center, April 1996. http://dx.doi.org/10.21236/ada309263.

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8

Dhuley, R. C. Thermal contact resistance. Office of Scientific and Technical Information (OSTI), Juli 2019. http://dx.doi.org/10.2172/1556950.

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Thatcher, R. Resistance seam welding. Office of Scientific and Technical Information (OSTI), Juli 1986. http://dx.doi.org/10.2172/5568130.

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Albers, John, und Harry L. Berkowitz. A collection of computer programs for two-probe resistance (spreading resistance) and four-probe resistance calculations, RESPAC. Gaithersburg, MD: National Institute of Standards and Technology, 1993. http://dx.doi.org/10.6028/nist.sp.400-91.

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