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Thematische Bibliographien / Pseudonoja textilis

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Zeitschriftenartikel

Auswahl der wissenschaftlichen Literatur zum Thema „Pseudonoja textilis“

Autor: Grafiati

Veröffentlicht am 18. Mai 2024

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Zeitschriftenartikel zum Thema "Pseudonoja textilis"

1

Tibballs, J., and S. Sutherland. "The Efficacy of Antivenom in Prevention of Cardiovascular Depression and Coagulopathy Induced by Brown Snake (Pseudonaja) Species Venom." Anaesthesia and Intensive Care 19, no. 4 (1991): 530–34. http://dx.doi.org/10.1177/0310057x9101900407.

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The efficacy of antivenom in prevention of cardiovascular depression and coagulopathy induced by Brown Snake species (Pseudonaja textilis, Pseudonaja affinis) was investigated in anaesthetised mechanically ventilated dogs. Venom and antivenom in variable amounts were incubated together at 37°C for 30 minutes prior to intravenous injection. The dose of antivenom required to prevent severe cardiovascular depression and coagulopathy induced by Pseudonaja textilis venom was 25 times the current recommended dose for clinical use. A tenfold dose of antivenom was required to neutralise similar effects induced by Pseudonaja affinis venom. Large amounts of antivenom may be required in clinical use if coagulopathy or cardiovascular depression are present after envenomation by species of the Brown Snake genus.

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2

Tibballs, J., S. K. Sutherland, R. A. Rivera, and P. P. Masci. "The Cardiovascular and Haematological Effects of Purified Prothrombin Activator from the Common Brown Snake (Pseudonaja textilis) and their Antagonism with Heparin." Anaesthesia and Intensive Care 20, no. 1 (1992): 28–32. http://dx.doi.org/10.1177/0310057x9202000105.

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The cardiovascular and haematological effects of purified prothrombin activator derived from the venom of the Australian Common Brown Snake (Pseudonaja textilis) were studied in anaesthetised, mechanically ventilated dogs. Severe depression of systemic blood pressure and cardiac output and a rise in central venous pressure were observed. Thrombocytopenia, prolongation of both prothrombin time and activated partial thromboplastin time and a reduction in serum fibrinogen were also observed. All of these observed effects were prevented by the prior administration of heparin — a naturally occurring anticoagulant. We conclude that the prothrombin activator in Pseudonaja textilis venom may cause cardiovascular depression due to myocardial dysfunction secondary to disseminated intravascular coagulation.

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3

Armugam, Arunmozhiarasi, NanLing Gong, XiaoJie Li, et al. "Group IB phospholipase A2 from Pseudonaja textilis." Archives of Biochemistry and Biophysics 421, no. 1 (2004): 10–20. http://dx.doi.org/10.1016/j.abb.2003.09.045.

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4

Gong, Nanling, Arunmozhiarasi Armugam, and Kandiah Jeyaseelan. "Postsynaptic short-chain neurotoxins from Pseudonaja textilis." European Journal of Biochemistry 265, no. 3 (2001): 982–89. http://dx.doi.org/10.1046/j.1432-1327.1999.00800.x.

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5

Tibballs, J., and S. K. Sutherland. "The Efficacy of Heparin in the Treatment of Common Brown Snake (Pseudonaja textilis) Envenomation." Anaesthesia and Intensive Care 20, no. 1 (1992): 33–37. http://dx.doi.org/10.1177/0310057x9202000106.

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The efficacy of heparin therapy after subcutaneous injection of Common Brown Snake (Pseudonaja textilis) venom was studied in anaesthetised, mechanically ventilated dogs. Intravenous heparin (100 U/kg), administered fifteen minutes after envenomation, neither prevented nor hastened the recovery from cardiovascular depression and coagulopathy observed after venom administration. Heparin therapy is not recommended for the treatment of established human envenomation.

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6

Watson, Gregory S., David W. Green, and Jolanta A. Watson. "Observations supporting parental care by a viviparous reptile: aggressive behaviour against predators demonstrated by Cunningham’s skinks." Australian Journal of Zoology 67, no. 3 (2019): 180. http://dx.doi.org/10.1071/zo20024.

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Most reptiles exhibit no parental care and aggressive behaviour towards heterospecific predators has rarely been recorded in the natural environment. Several species of the subfamily Egerniinae are amongst the most highly social of all squamate reptiles, exhibiting stable social aggregations and high levels of long-term social and genetic monogamy. We have examined Cunningham’s skinks, Egernia cunninghami, over a three-year period during late January and early February (total 32 days) in the alpine region of New South Wales using video and thermal imaging. Four birthing sessions were witnessed during our field studies of social aggregations of skinks. Our observations monitored skink encounters, in the presence of offspring, with an eastern brown snake, Pseudonaja textilis (two separate encounters, one recorded by video/imaging) and 12 encounters with the Australian magpie, Gymnorhina tibicen. All events were associated with aggressive chasing and/or attack by adult skinks. The first snake encounter involved the active targeting of a recently born juvenile with the mother of the juvenile attacking the snake (running towards the snake, biting and remaining attached for several seconds). The second encounter (the following year) comprised two adult skinks attacking and biting a snake, Pseudonaja textilis. All magpie encounters resulted in chases by adult skinks.

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7

Whitaker, P. B., K. Ellis, and R. Shine. "The defensive strike of the Eastern Brownsnake, Pseudonaja textilis (Elapidae)." Functional Ecology 14, no. 1 (2000): 25–31. http://dx.doi.org/10.1046/j.1365-2435.2000.00385.x.

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8

Willmott, N., P. Gaffney, P. Masci, and A. Whitaker. "A novel serine protease inhibitor from the Australian brown snake, Pseudonaja textilis textilis: Inhibition kinetics." Fibrinolysis 9, no. 1 (1995): 1–8. http://dx.doi.org/10.1016/s0268-9499(08)80040-9.

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9

GONG, NanLing, Arunmozhiarasi ARMUGAM, Peter MIRTSCHIN, and Kandiah JEYASEELAN. "Cloning and characterization of the pseudonajatoxin b precursor." Biochemical Journal 358, no. 3 (2001): 647–56. http://dx.doi.org/10.1042/bj3580647.

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An Australian common brown snake, Pseudonaja textilis, is known to contain highly lethal neurotoxins. Among them, a long-chain α-neurotoxin, pseudonajatoxin b, has been identified. In this report, while presenting evidence for the presence of at least four such long-chain α-neurotoxins in the venom of P. textilis, we describe the characteristics of both the mRNA and the gene responsible for the synthesis of these neurotoxins. A precursor toxin synthesized from the gene has been identified as being capable of producing the isoforms possibly by post-translational modifications at its C-terminal end. Recombinant toxins corresponding to the precursor and its product have been found to possess similar binding affinities for muscular acetylcholine receptors (IC50 = 3×10−8 M) and a lethality, LD50, of 0.15μg/g in mice.

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10

Tibballs, J., S. Sutherland, and S. Kerr. "Studies on Australian Snake Venoms. Part 1: The Haemodynamic Effects of Brown Snake (Pseudonaja) Species in the Dog." Anaesthesia and Intensive Care 17, no. 4 (1989): 466–69. http://dx.doi.org/10.1177/0310057x8901700412.

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The haemodynanic effects of Brown Snake (Pseudonaja) species (textilis, nuchalis, affinis) were investigated in anaesthetised, mechanically ventilated dogs. Blood pressure decreased to minimal levels five minutes after intravenous envenomation. Hypotension was accompanied by significant decrements in cardiac output and stroke volume and a rise in peripheral vascular resistance. Heart rate increased transiently during 0.5-2.0 minutes after envenomation but had declined below resting levels five minutes after envenomation. No statistically significant change was recorded in central venous pressure. Depression of myocardial contractility is postulated as the mechanism of venom induced hypotension.

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