Auswahl der wissenschaftlichen Literatur zum Thema „Proximal interactomics“
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Zeitschriftenartikel zum Thema "Proximal interactomics"
Sofianatos, Yorgos, Étienne Coyaud und Caroline Demeret. „Towards a three-dimensional mapping of virus-host proximal protein interactions“. Project Repository Journal 13, Nr. 1 (07.05.2022): 14–17. http://dx.doi.org/10.54050/prj1318790.
Der volle Inhalt der QuelleHarris, C. Jake, Marion Scheibe, Somsakul Pop Wongpalee, Wanlu Liu, Evan M. Cornett, Robert M. Vaughan, Xueqin Li et al. „A DNA methylation reader complex that enhances gene transcription“. Science 362, Nr. 6419 (06.12.2018): 1182–86. http://dx.doi.org/10.1126/science.aar7854.
Der volle Inhalt der QuelleHaider, Nasir A., Joanna Kelly, Duncan Smith und Claus Jorgensen. „Abstract A099: Utilising interactomics to uncover oncogenic KRAS signalling networks in the context of the tumor microenvironment“. Cancer Research 84, Nr. 2_Supplement (16.01.2024): A099. http://dx.doi.org/10.1158/1538-7445.panca2023-a099.
Der volle Inhalt der QuelleChua, Xien Yu, Timothy Aballo, William Elnemer, Melanie Tran und Arthur Salomon. „Quantitative Interactomics of Lck-TurboID in Living Human T Cells Unveils T Cell Receptor Stimulation-Induced Proximal Lck Interactors“. Journal of Proteome Research 20, Nr. 1 (13.11.2020): 715–26. http://dx.doi.org/10.1021/acs.jproteome.0c00616.
Der volle Inhalt der QuelleIbarz, Antoni, Ignasi Sanahuja, Waldo G. Nuez-Ortín, Laura Martínez-Rubio und Laura Fernández-Alacid. „Physiological Benefits of Dietary Lysophospholipid Supplementation in a Marine Fish Model: Deep Analyses of Modes of Action“. Animals 13, Nr. 8 (18.04.2023): 1381. http://dx.doi.org/10.3390/ani13081381.
Der volle Inhalt der QuelleOsagie, Oloruntoba I., Jordann Smakk, Deborah E. Citrin und Travis H. Stracker. „Abstract 4802: Identification of critical hypoxia induced factors in castrate resistant prostate cancer“. Cancer Research 83, Nr. 7_Supplement (04.04.2023): 4802. http://dx.doi.org/10.1158/1538-7445.am2023-4802.
Der volle Inhalt der QuelleCutler, Jevon, Rahia Tahir, Jingnan Han, Raja Sekhar Nirujogi, Tai-Chung Huang, Xianrong Wong, Saradhi Mallampati et al. „Differential Signaling through p190 and p210 Forms of BCR-ABL Fusion Proteins Revealed By Proteomic Analysis“. Blood 126, Nr. 23 (03.12.2015): 3651. http://dx.doi.org/10.1182/blood.v126.23.3651.3651.
Der volle Inhalt der QuelleRuminski, Kilian, Javier Celis-Gutierrez, Nicolas Jarmuzynski, Emilie Maturin, Stephane Audebert, Marie Malissen, Luc Camoin, Guillaume Voisinne, Bernard Malissen und Romain Roncagalli. „Mapping the SLP76 interactome in T cells lacking each of the GRB2-family adaptors reveals molecular plasticity of the TCR signaling pathway“. Frontiers in Immunology 14 (15.03.2023). http://dx.doi.org/10.3389/fimmu.2023.1139123.
Der volle Inhalt der QuelleKulyyassov, Arman, Gulsamal Zhubanova, Erlan Ramanculov und Vasily Ogryzko. „Proximity Utilizing Biotinylation of Nuclear Proteins in vivo“. Central Asian Journal of Global Health 3 (15.06.2015). http://dx.doi.org/10.5195/cajgh.2014.165.
Der volle Inhalt der QuelleChastney, Megan R., Craig Lawless, Jonathan D. Humphries, Stacey Warwood, Matthew C. Jones, David Knight, Claus Jorgensen und Martin J. Humphries. „Topological features of integrin adhesion complexes revealed by multiplexed proximity biotinylation“. Journal of Cell Biology 219, Nr. 8 (25.06.2020). http://dx.doi.org/10.1083/jcb.202003038.
Der volle Inhalt der QuelleDissertationen zum Thema "Proximal interactomics"
Bachiri, Kamel. „Identification des interactions oncogéniques du Polyomavirus à cellules de Merkel“. Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS113.
Der volle Inhalt der QuelleMerkel cell carcinoma (MCC) is an extremely aggressive skin cancer with a high mortality rate. In 80% of cases, this cancer is linked to the presence of the Merkel cell polyomavirus which expresses two viral oncoproteins, small T and a truncated form of large T. The expression of these proteins is sufficient for carcinogenesis, inducing the disruption of cell cycle checkpoints, changes in the epigenetic profile, and immune evasion. The combination of interactomics and proteomics approaches allowed us to identify numerous epigenetic factors related to the T antigens. These studies have also highlighted potential mechanisms involving the regulation of protein stability, gene expression, and genetic stability via an altered DNA damage response pathway. The hypotheses formulated following these analyses were investigated in targeted assays. A link between the peptidyl-prolyl cis/trans isomerase and neddylation was demonstrated. We have thus identified a new potential mechanism for regulating the activity of ubiquitination complexes involving the recruitment of PIN1 to these complexes, thus their isomerization, by neddylation. The study of the EHMT2 functions, an epigenetic regulator of interest, revealed the importance of its role in the DNA damage response in virus positive MCC (VP-MCC). We were able to identify a protective role of tLT and EHMT2 in DNA damages in VP-MCC cells. More specifically, we also report a role of EHMT2 in solving single strand DNA breaks following replication stress. Our works have enabled the discovery of new important mechanisms in VP-MCC oncogenesis, reporting for the first time a central role of tLT in DNA damages management