Bücher zum Thema „Premier moteur“

This chapter presents two excerpts from Ramseur's first letter to his mother from West Point, dated August 15, 1855 that illustrates his transition from upbringing to matriculation. It reports that the U.S. Military Academy was the nation's premier engineering school with a curriculum designed to prepare its graduates to build the river and harbour works, lighthouses, canals, and railroads needed by a burgeoning nation. The U.S. Military Academy also served to prepare topographical and military engineers for times of war. The chapter notes that the institution had been founded early in the nineteenth century, modeled after Sandhurst and St. Cyr, its counterparts in England and France respectively.

Sports on Film takes readers behind the scenes of how movies get made and puts them in the stands for some of the key moments in sports in America. Sports on Film documents key events in American sports history through the films that depict them, starting with the integration of major-league baseball when Jackie Robinson signed with the Brooklyn Dodgers. Other significant events and personalities examined include the college basketball point-shaving incident of the 1950s; journalist George Plimpton's attempt to go through the Detroit Lions' NFL training camp in the early 1960s; the originations and popularity of rodeo; the brief run of women's professional baseball during World War II; the underdog racehorse Seabiscuit during the Great Depression; the rise of African American boxer Muhammad Ali; the unique 1970s "Battle of the Sexes" tennis event between Bobby Riggs and Billie Jean King; and Ford Motor Company's run in the 1960s to take motorsports to Europe's premier event in Le Mans, France.

This chapter looks into Article 30 of the Vienna Convention on Diplomatic Relations which deals with the inviolability of a diplomatic agent’s residence and property. Under Article 30, the private residence of a diplomatic agent shall enjoy the same inviolability and protection as the premises of the mission. The agent’s papers, correspondence, and property shall likewise enjoy inviolability except as provided in Article 31.3. The chapter elaborates the scope of the inviolability in terms of private residences, papers and correspondence, property, and also exchange control. In addition, it also discusses the removal or towing away of motor vehicles of the diplomatic agent.

This chapter examines the development of early Jewish martyrdom from the Bible to late antiquity. The chapter argues that martyrdom does not exist in the Hebrew Bible and that the stories of Eleazar and the mother with her seven sons from 2 Maccabees are not indicative of an existing Hellenistic tradition of martyrdom. The Jewish concept of martyrdom started to develop in Roman times, due to the influence of the popular Roman idea of noble death. The Jewish acceptance of the Roman idea created also moral and theological dilemmas. The idea of noble death needed to be reconciled with a Jewish tradition that emphasized the holiness of life. These martyrological premises and predicaments continued to be developed in rabbinic literature. The end result was the presentation of a rabbinic martyrological genre that set the Jewish lore and law of kiddush ha-Shem.

From 1939 to 1940 Balanchine and Kirstein continued their independent activities. Balanchine and Zorina took on additional projects on the stage and screen, including a film adaptation of On Your Toes, the romantic melodrama I Married an Adventuress, and a new stage musical by Irving Berlin, Louisiana Purchase. Balanchine also created dances for the short-lived revue Keep Off the Grass as well as serving as producer and director of Cabin in the Sky, for which he collaborated with Vernon Duke and choreographer Katherine Dunham and her dance company. Kirstein continued to tour with the company now billed as the American Ballet Caravan, which completed a second transcontinental tour that included the premieres of the ballets City Portrait and Charade. The company secured one final engagement in a ballet called A Thousand Times Neigh, presented at the World’s Fair Pavilion of the Ford Motor Company.

The objective in this book is to show how poetry enables us cognitively to aesthetically access, experience, and identify with the visible and invisible “being” of reality, with art as one cognitive expression of the aesthetic faculty, science another. Just as scientific knowledge of reality is achieved through physically exploring the far reaches of the visible and invisible worlds, so is poetic experience achieved through iconically simulating in semblance the “being” of reality that integrates both self and world in participatory unity. “Being” here should not be understood as the existence of material substance, but as the essence of all that is, both visible and invisible, material and immaterial, a life force in continuous flux and change. The book explores cognition as the sensory-motor-emotive-conceptual processes of “minding” and the aesthetic faculty as the processes of attention, imagination, memory, discrimination, expertise, and judgment that underlie all human cognition, including the arts and the sciences. Drawing from research such as blending and neurocognition in interdisciplinary cognitive literary studies, the book attempts to resolve long-standing questions about the function of poetry. Accepting the premise that poetry is its own artistic reason for being, it introduces the major elements—semblance, metaphor, schema, and affect—that constitute a poem as icon in motivating a poet’s intension and a respondent’s engagement. In so doing the book makes the case that a poem is a potential icon of the felt reality of being and shows that poetic iconicity provides a means for evaluating great poetry and an explanation for its endurance.

Any public debate about air pollution starts with the premise that air pollution cannot be good for you, so we should have less of it. However, it is much more difficult to determine how much is dangerous, and even more difficult to decide how much we are willing to pay for improvements in measured air pollution. Recent UK estimates suggest that fine particulate pollution causes about 6500 deaths per year, although it is not clear how many years of life are lost as a result. Some deaths may just be brought forward by a few days or weeks, while others may be truly premature. Globally, household pollution from cooking fuels may cause up to two million premature deaths per year in the developing world. The hazards of black smoke air pollution have been known since antiquity. The first descriptions of deaths caused by air pollution are those recorded after the eruption of Vesuvius in ad 79. In modern times, the infamous smogs of the early twentieth century in Belgium and London were clearly shown to trigger deaths in people with chronic bronchitis and heart disease. In mechanistic terms, black smoke and sulphur dioxide generated from industrial processes and domestic coal burning cause airway inflammation, exacerbation of chronic bronchitis, and consequent heart failure. Epidemiological analysis has confirmed that the deaths included both those who were likely to have died soon anyway and those who might well have survived for months or years if the pollution event had not occurred. Clean air legislation has dramatically reduced the levels of these traditional pollutants in the West, although these pollutants are still important in China, and smoke from solid cooking fuel continues to take a heavy toll amongst women in less developed parts of the world. New forms of air pollution have emerged, principally due to the increase in motor vehicle traffic since the 1950s. The combination of fine particulates and ground-level ozone causes ‘summer smogs’ which intensify over cities during summer periods of high barometric pressure. In Los Angeles and Mexico City, ozone concentrations commonly reach levels which are associated with adverse respiratory effects in normal and asthmatic subjects. Ozone directly affects the airways, causing reduced inspiratory capacity. This effect is more marked in patients with asthma and is clinically important, since epidemiological studies have found linear associations between ozone concentrations and admission rates for asthma and related respiratory diseases. Ozone induces an acute neutrophilic inflammatory response in both human and animal airways, together with release of chemokines (e.g. interleukin 8 and growth-related oncogene-alpha). Nitrogen oxides have less direct effect on human airways, but they increase the response to allergen challenge in patients with atopic asthma. Nitrogen oxide exposure also increases the risk of becoming ill after exposure to influenza. Alveolar macrophages are less able to inactivate influenza viruses and this leads to an increased probability of infection after experimental exposure to influenza. In the last two decades, major concerns have been raised about the effects of fine particulates. An association between fine particulate levels and cardiovascular and respiratory mortality and morbidity was first reported in 1993 and has since been confirmed in several other countries. Globally, about 90% of airborne particles are formed naturally, from sea spray, dust storms, volcanoes, and burning grass and forests. Human activity accounts for about 10% of aerosols (in terms of mass). This comes from transport, power stations, and various industrial processes. Diesel exhaust is the principal source of fine particulate pollution in Europe, while sea spray is the principal source in California, and agricultural activity is a major contributor in inland areas of the US. Dust storms are important sources in the Sahara, the Middle East, and parts of China. The mechanism of adverse health effects remains unclear but, unlike the case for ozone and nitrogen oxides, there is no safe threshold for the health effects of particulates. Since the 1990s, tax measures aimed at reducing greenhouse gas emissions have led to a rapid rise in the proportion of new cars with diesel engines. In the UK, this rose from 4% in 1990 to one-third of new cars in 2004 while, in France, over half of new vehicles have diesel engines. Diesel exhaust particles may increase the risk of sensitization to airborne allergens and cause airways inflammation both in vitro and in vivo. Extensive epidemiological work has confirmed that there is an association between increased exposure to environmental fine particulates and death from cardiovascular causes. Various mechanisms have been proposed: cardiac rhythm disturbance seems the most likely at present. It has also been proposed that high numbers of ultrafine particles may cause alveolar inflammation which then exacerbates preexisting cardiac and pulmonary disease. In support of this hypothesis, the metal content of ultrafine particles induces oxidative stress when alveolar macrophages are exposed to particles in vitro. While this is a plausible mechanism, in epidemiological studies it is difficult to separate the effects of ultrafine particles from those of other traffic-related pollutants.