Thematische Bibliographien / Placenta – Physiologie

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Placenta – Physiologie“

Autor: Grafiati
Veröffentlicht am 29. März 2025
Zuletzt aktualisiert am 31. Juli 2025

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Zeitschriftenartikel zum Thema "Placenta – Physiologie"

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Taher, Shèdy, Yamilette Borja, Lucía Cabanela, et al. "Cholecystokinin, gastrin, cholecystokinin/gastrin receptors, and bitter taste receptor TAS2R14: trophoblast expression and signaling." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 5 (2019): R628—R639. http://dx.doi.org/10.1152/ajpregu.00153.2018.

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We investigated expression of cholecystokinin (CCK) in humans and mice, and the bitter taste receptor TAS2R14 in the human placenta. Because CCK and gastrin activate the CCKBR receptor, we also explored placental gastrin expression. Finally, we investigated calcium signaling by CCK and TAS2R14. By RT-PCR, we found CCK/Cck and GAST/Gast mRNA expression in both normal human and mouse placentas, as well as in human trophoblast cell lines (TCL). Although both Cckar and – br mRNA were expressed in the mouse placenta, only CCKBR mRNA was detected in the human placenta and TCL. mRNA expression for TA
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Lien, Yu-Chin, Zhe Zhang, Yi Cheng, et al. "Human Placental Transcriptome Reveals Critical Alterations in Inflammation and Energy Metabolism with Fetal Sex Differences in Spontaneous Preterm Birth." International Journal of Molecular Sciences 22, no. 15 (2021): 7899. http://dx.doi.org/10.3390/ijms22157899.

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A well-functioning placenta is crucial for normal gestation and regulates the nutrient, gas, and waste exchanges between the maternal and fetal circulations and is an important endocrine organ producing hormones that regulate both the maternal and fetal physiologies during pregnancy. Placental insufficiency is implicated in spontaneous preterm birth (SPTB). We proposed that deficits in the capacity of the placenta to maintain bioenergetic and metabolic stability during pregnancy may ultimately result in SPTB. To explore our hypothesis, we performed a RNA-seq study in male and female placentas
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Osifo, E. O., and V. C. Ezeuko. "Histological Assessment of Placental Development Following Intrauterine Exposure to Caffeine in Adult Wistar Rats." Journal of Applied Sciences and Environmental Management 28, no. 4 (2024): 1115–20. http://dx.doi.org/10.4314/jasem.v28i4.11.

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In recent years, there have been concerns about human reproductive disorders. Physiological adaptations are crucial for optimal fetal development during pregnancy. The widespread consumption of caffeine by pregnant women raises questions about its impact on maternal physiology and fetal development. Hence, the objective of this study was to evaluate the histological assessment of placenta development following intrauterine exposure to caffeine in adult Wistar rats using appropriate standard techniques. On each gestational day (GD13, GD15, GD17, and GD19), five (5) animals were sampled from eac
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Rampon, Christine, Stéphanie Bouillot, Adriana Climescu-Haulica, et al. "Protocadherin 12 deficiency alters morphogenesis and transcriptional profile of the placenta." Physiological Genomics 34, no. 2 (2008): 193–204. http://dx.doi.org/10.1152/physiolgenomics.00220.2007.

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Protocadherins are transmembrane proteins exhibiting homophilic adhesive activities through their extracellular domain. Protocadherin 12 ( Pcdh12) is expressed in angiogenic endothelial cells, mesangial cells of kidney glomeruli, and glycogen cells of the mouse placenta. To get insight into the role of this protein in vivo, we analyzed PCDH12-deficient mice and investigated their placental phenotype. The mice were alive and fertile; however, placental and embryonic sizes were reduced compared with wild-type mice. We observed defects in placental layer segregation and a decreased vascularizatio
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Selvaratnam, Johanna, Haiyan Guan, James Koropatnick, and Kaiping Yang. "Metallothionein-I- and -II-deficient mice display increased susceptibility to cadmium-induced fetal growth restriction." American Journal of Physiology-Endocrinology and Metabolism 305, no. 6 (2013): E727—E735. http://dx.doi.org/10.1152/ajpendo.00157.2013.

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Maternal cadmium exposure induces fetal growth restriction (FGR), but the underlying mechanisms remain largely unknown. The placenta is the main organ known to protect the fetus from environmental toxins such as cadmium. In this study, we examine the role of the two key placental factors in cadmium-induced FGR. The first is placental enzyme 11β-hydroxysteroid dehydrogenase type 2 (11β-HSD2), which is known to protect the fetus from exposure to high cortisol levels and subsequently FGR, and the second the cadmium binding/sequestering proteins metallotheionein (MT)-I and -II. Using the MT-I/II −
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Flores-Pliego, Arturo, Jael Miranda, Sara Vega-Torreblanca, et al. "Molecular Insights into the Thrombotic and Microvascular Injury in Placental Endothelium of Women with Mild or Severe COVID-19." Cells 10, no. 2 (2021): 364. http://dx.doi.org/10.3390/cells10020364.

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Clinical manifestations of coronavirus disease 2019 (COVID-19) in pregnant women are diverse, and little is known of the impact of the disease on placental physiology. Severe acute respiratory syndrome coronavirus (SARS-CoV-2) has been detected in the human placenta, and its binding receptor ACE2 is present in a variety of placental cells, including endothelium. Here, we analyze the impact of COVID-19 in placental endothelium, studying by immunofluorescence the expression of von Willebrand factor (vWf), claudin-5, and vascular endothelial (VE) cadherin in the decidua and chorionic villi of pla
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Vaughan, Owen R., Fredrick Thompson, Ramón A. Lorca, et al. "Effect of high altitude on human placental amino acid transport." Journal of Applied Physiology 128, no. 1 (2020): 127–33. http://dx.doi.org/10.1152/japplphysiol.00691.2019.

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Women residing at high altitudes deliver infants of lower birth weight than at sea level. Birth weight correlates with placental system A-mediated amino acid transport capacity, and severe environmental hypoxia reduces system A activity in isolated trophoblast and the mouse placenta. However, the effect of high altitude on human placental amino acid transport remains unknown. We hypothesized that microvillous membrane (MVM) system A and system L amino acid transporter activity is lower in placentas of women living at high altitude compared with low-altitude controls. Placentas were collected a
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Assad, R. S., F. Y. Lee, and F. L. Hanley. "Placental compliance during fetal extracorporeal circulation." Journal of Applied Physiology 90, no. 5 (2001): 1882–86. http://dx.doi.org/10.1152/jappl.2001.90.5.1882.

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The fetus requires large amounts of volume when weaning from cardiac bypass. This suggests that placental vasculature can act as a large capacitor in the fetal circulation. To assess placental compliance of fetal lambs, seven isolated in situ lamb placentas were placed on extracorporeal circulation. Umbilical artery blood flow was varied from 0 to 350 ml · min−1· kg fetal wt−1. Because the extracorporeal circuit is a closed system, volume changes in the placenta induced by umbilical artery pressure changes were measured from reciprocal volume changes in the reservoir. There was a wide range of
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Shanes, Elisheva D., Leena B. Mithal, Sebastian Otero, Hooman A. Azad, Emily S. Miller, and Jeffery A. Goldstein. "Placental Pathology in COVID-19." American Journal of Clinical Pathology 154, no. 1 (2020): 23–32. http://dx.doi.org/10.1093/ajcp/aqaa089.

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Abstract Objectives To describe histopathologic findings in the placentas of women with coronavirus disease 2019 (COVID-19) during pregnancy. Methods Pregnant women with COVID-19 delivering between March 18, 2020, and May 5, 2020, were identified. Placentas were examined and compared to historical controls and women with placental evaluation for a history of melanoma. Results Sixteen placentas from patients with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) were examined (15 with live birth in the third trimester, 1 delivered in the second trimester after intrauterine fetal demi
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Tissot van Patot, M. C., J. Bendrick-Peart, V. E. Beckey, N. Serkova, and L. Zwerdlinger. "Greater vascularity, lowered HIF-1/DNA binding, and elevated GSH as markers of adaptation to in vivo chronic hypoxia." American Journal of Physiology-Lung Cellular and Molecular Physiology 287, no. 3 (2004): L525—L532. http://dx.doi.org/10.1152/ajplung.00203.2003.

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Vascularity is increased in placentas from high- compared with low-altitude pregnancies. An angiogenic response to hypoxia may protect an organ from further hypoxic insult by increasing blood flow and oxygen delivery to the tissue. We hypothesized that increased placental vascularity is sufficient to adapt to high altitude. Therefore, indexes of hypoxic stress would not be present in placentas from successful high-altitude pregnancies. Full-thickness placental biopsies were 1) collected and frozen in liquid nitrogen within 5 min of placental delivery and 2) fixed in formalin for stereologic an
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Dissertationen zum Thema "Placenta – Physiologie"

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Ayache, Mohammad. "Application des réseaux de neurones à la classification automatisée des grades placentaires." Tours, 2007. http://www.theses.fr/2007TOUR3315.

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Le placenta est un organe provisoire joignant la mère et le fœtus qui transfère l’oxygène et des nutriments de la mère au fœtus et permet l’évacuation de l’anhydride carbonique et des produits du métabolisme du fœtus. Le but de notre travail était d’étudier la fonction de transfert des tissus placentaires selon son développement en se basant sur les images ultrasonores. Nous avons développé au cours de ce travail une nouvelle approche de la classification du développement placentaire en ultrasons par des techniques de traitement d’images avancées basée sur une représentation par réseau neurona
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Routhier, Catherine. "Étude de la voie des polyols dans le placenta en prééclampsie." Thesis, Université Laval, 2014. http://www.theses.ulaval.ca/2014/30339/30339.pdf.

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La prééclampsie (PE) est une pathologie obstétricale complexe associée à un défaut de placentation. Selon la littérature, le placenta anormalement développé libèrerait des facteurs qui induiraient une dysfonction endothéliale maternelle. Nous avons émis comme hypothèse qu’une accumulation de sorbitol, un sucre hyperosmotique produit par la voie des polyols, pourrait induire la libération par le placenta de sFlt-1, un facteur anti-angiogénique antagoniste du VEGF en PE. Nous avons comparé les niveaux d’expression d’ARNm et de protéines de AKR1B1 et SORD, les deux enzymes impliquées dans la voie
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Beauchemin, David. "Étude de la synthèse des prostanoïdes vasoactifs et de leurs récepteurs dans le placenta en prééclampsie." Thesis, Université Laval, 2011. http://www.theses.ulaval.ca/2011/27970/27970.pdf.

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Le, Duc Kévin. "Physiologie des échanges gazeux et de l'hémodynamique transplacentaire lors d'une réanimation à cordon intact : modèle expérimental d'agneau porteur de hernie diaphragmatique." Electronic Thesis or Diss., Université de Lille (2022-....), 2024. http://www.theses.fr/2024ULILS079.

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La naissance est une période à risque qui met en jeux de multiples mécanismes qui permettent une transition de la vie fœtale et la vie extra-utérine. Chaque année, dans les conséquences d'une mauvaise adaptation à la vie extra-utérine et la persistance de résistances vasculaires pulmonaires trop élevées, 1 million de nouveau-né décède dans les 24 premières heures de vie. Dix pourcents des nouveau-nés requièrent une assistance médicale en salle de naissance. Le clampage du cordon ombilical retardé entre 60 et 180 secondes après la naissance est désormais recommandé pour toutes les situations où
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Bartho, Lucy A. "Mitochondrial Alterations Through Gestation and in Placental Pathologies." Thesis, Griffith University, 2021. http://hdl.handle.net/10072/411262.

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The placenta is a transient organ, essential for the growth and development of the fetus throughout pregnancy. This temporary organ brings maternal and fetal blood circulation into close proximity, which allows for the exchange of oxygen, carbon dioxide, waste, and other essential nutrients. Despite constant influences by internal and external factors, the human placenta has a defined life span of approximately 280 days. From conception, through to term, the placenta undergoes chronological aging, which is regulated by a range of cellular processes. Advanced placental aging and cellular senesc
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Corbel, Tanguy. "Mécanismes toxicocinétiques impliqués dans l'exposition foetale au Bisphénol A." Thesis, Toulouse, INSA, 2013. http://www.theses.fr/2013ISAT0034/document.

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Le Bisphénol A (BPA) est un perturbateur endocrinien dont les effets développementaux observés chez les rongeurs soulèvent la question du risque pour la santé humaine relatif à une exposition fœtale au BPA. L’objectif de cette thèse est de déterminer les mécanismes toxicocinétiques impliqués dans l’exposition fœtale au BPA. La caractérisation in vivo dans un modèle intégratif ovin des expositions maternelles et fœtales au BPA et à ses métabolites ont permis d’identifier le transfert placentaire et le métabolisme fœto-placentaire comme les déterminants majeurs de l’exposition fœtale au BPA. Le
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Rao, M. Rekha. "Functional Differentiation Of The Human Placenta : Insights From The Expression Of Two Developmentally - Regulated Genes." Thesis, Indian Institute of Science, 2000. https://etd.iisc.ac.in/handle/2005/177.

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Placenta is a transient association of the fetal and maternal tissues, that develops during pregnancy, in most viviparous animals. The evolution of placenta ensured the development of the fetus inside the womb of the mother, providing a protected environment for the development of the fetus, and preventing the loss of progeny due to unfavorable environmental conditions. Because it is strategically poised at the maternal and fetal interface, the placenta is ideally suited to carry out alimentary, respiratory and excretory functions for the developing fetus. In addition, it serves as an immunolo
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Rao, M. Rekha. "Functional Differentiation Of The Human Placenta : Insights From The Expression Of Two Developmentally - Regulated Genes." Thesis, Indian Institute of Science, 2000. http://hdl.handle.net/2005/177.

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Placenta is a transient association of the fetal and maternal tissues, that develops during pregnancy, in most viviparous animals. The evolution of placenta ensured the development of the fetus inside the womb of the mother, providing a protected environment for the development of the fetus, and preventing the loss of progeny due to unfavorable environmental conditions. Because it is strategically poised at the maternal and fetal interface, the placenta is ideally suited to carry out alimentary, respiratory and excretory functions for the developing fetus. In addition, it serves as an immunolo
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Griffith, Oliver William. "Mechanisms of placental evolution: the genetics and physiology of pregnancy in lizards." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/13600.

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The placenta is a unique example of a complex organ that has evolved independently more than 115 times in amniotes (birds, reptiles, and mammals). Placentae exchange materials including respiratory gasses, nutrients, and hormones, between the mother and embryo. I aimed to identify the physiological and ecological factors that contribute to the evolution of placentation in vertebrates, focusing on the genetic mechanisms underpinning the evolution of placental functions, and the role of parent-offspring conflict in the evolution of placental traits. Given placentation has evolved independently m
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Jokhi, Percy Parvez. "Cytokines and their receptors in human placental implantation." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338321.

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Bücher zum Thema "Placenta – Physiologie"

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Olga, Genbačev, Klopper Arnold, and Beaconsfield Rebecca, eds. Placenta as a model and a source. Plenum Press, 1989.

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Selʹkov, S. A. Plat︠s︡entarnye makrofagi. Tov-vo nauch. izd. KMK, 2007.

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Lise, Cédard, and Firth Anthony, eds. Placental signals: Autocrine and paracine control of pregnancy. University of Rochester Press, 1992.

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1946-, Miller Richard K., Thiede Henry A, and Rochester Trophoblast Conference. (11th : 1988 : Rochester, N.Y.), eds. Molecular biology and cell regulation of the placenta. Verav Medical Publishers, 1991.

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E, Rice Gregory, and Brennecke S. P, eds. Molecular aspects of placental and fetal membrane autacoids. CRC Press, 1993.

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1932-, Battaglia Frederick C., Nestlé Nutrition Services, and Nestlé Nutrition Workshop (39th : 1996 : East Sussex, England), eds. Placental function & fetal nutrition. Nestlé Nutrition Services, 1997.

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1940-, Hata Toshio, Takayama Masaomi 1938-, Taki Ichiro 1919-, Foidart Jean-Michel 1949-, Japanese Placenta Group Meeting, and European Placenta Group Meeting, eds. Placental molecules in hemodynamics, transport, and cellular regulation. University of Rochester Press, 1997.

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L, Power Michael, and Schulkin Jay, eds. Birth, distress, and disease: Placental-brain interactions. Cambridge University Press, 2005.

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Wooding, F. B. P. Comparative placentation: Structures, functions and evolution. Springer, 2008.

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Nadirashvili, S. A. Iskusstvennoe plat͡s︡entarnoe krovoobrashchenie ploda. "Met͡s︡niereba", 1986.

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Buchteile zum Thema "Placenta – Physiologie"

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Ebbesen, P. "Placenta physiology." In Congenital toxoplasmosis. Springer Paris, 2000. http://dx.doi.org/10.1007/978-2-8178-0847-5_3.

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McNamara, Jennifer M., and Helen H. Kay. "Placental Hormones: Physiology, Disease, and Prenatal Diagnosis." In The Placenta. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444393927.ch8.

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Dancis, Joseph, and Henning Schneider. "Physiology of the Placenta." In Human Growth. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2101-9_12.

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Kam, Peter, Ian Power, Michael J. Cousins, and Philip J. Siddal. "Physiology of the Placenta." In Principles of Physiology for the Anaesthetist. CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-73.

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Karck, U., and M. Breckwoldt. "Functions of the Placenta." In Comprehensive Human Physiology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60946-6_118.

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Mounce, Ginny. "The placenta and developing fetus." In The Anatomy and Physiology Textbook for Midwives. Routledge, 2025. https://doi.org/10.4324/9781003227571-10.

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Butt, W. R., T. Chard, and R. K. Iles. "Hormones of the Placenta: hCG and hPL." In Marshall’s Physiology of Reproduction. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1286-4_5.

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Longo, Lawrence D. "Some Aspects of the Physiology of the Placenta." In The Rise of Fetal and Neonatal Physiology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7921-5_8.

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Longo, Lawrence D. "Some Aspects of the Physiology of the Placenta." In The Rise of Fetal and Neonatal Physiology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7483-2_8.

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Longo, Lawrence D. "Some Aspects of Endocrinology of the Placenta." In The Rise of Fetal and Neonatal Physiology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7483-2_9.

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Konferenzberichte zum Thema "Placenta – Physiologie"

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Thompson, Jaylyn D., Jenna Zappetti, and Clarence Julian II Clark. "Management Strategies for Traumatic Injuries in Pregnant Women: A Comprehensive Literature Review." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.122_2024.

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Introduction: Traumatic injuries in pregnant women are the leading cause of non-obstetric death and affect 6% to 8% of all pregnancies. Therefore, it is imperative to maintain updated guidelines to construct a framework for the optimal management strategies for traumatic injuries in pregnant women, considering the perspectives of obstetrics/gynecology, emergency medicine, and orthopedics. Methods: To conduct this study, research was done via a database search through the Rowan-Virtua School of Osteopathic Medicine’s research library. PubMed was the sole database used to review 8 peer-reviewed
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"Effect of Hydrocortisone On Some Physiological and Morphological Changes in Mice Fetus." In XII. International Scientific Congress of Pure, Applied and Technological Sciences. Rimar Academy, 2024. https://doi.org/10.47832/minarcongress12-25.

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The purpose of the current study was to investigate the effects of hydrocortisone during the prenatal stage and its subsequent effects on various phenotypic abnormalities and morphologies in pregnant mice and their offspring. Rats were given hydrocortisone acetate treatment from the first day of pregnancy to day eighteen, at varying doses of 0.10– 0.15–0.20 mg/day. An experiment using thirty-two pregnant mice was conducted. Three groups of eight pregnant mice each make up each experimental set-up. Eight untreated pregnant mice (control group) were included in the first group; eight pregnant mi
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Kh ALJUBOURY, Maha, and khawla E ALJUBOURY. "EFFECT OF DEPAKINE ON SOME PHYSIOLOGICAL CHANGES OF PREGNANT MICE AND THEIR EMBRYOS USING FOLIC ACID." In VII. INTERNATIONAL SCIENTIFIC CONGRESSOF PURE,APPLIEDANDTECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress7-24.

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The present workconducted to evaluate the preventive role of folic acid in decreasing harmful effect on some phisiological changes & malformation in pregnant mice and their embryos. In this research we used Depakine (anti convulsant) at concentration of (300,500,600). In the present work, administration of Depakine (anti-convolsant) at the concentration of (300,500,600) mg/kg and folic acid 5 mg/kg to pregnant mice from the 1st till the 18th day of gestation. 30 pregnant mice were used and 2 experiments were performed.each experimental have 3 groups with 5 pregnant mice in each group. In t
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Berichte der Organisationen zum Thema "Placenta – Physiologie"

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Bazer, Fuller W., Arieh Gertler, and Elisha Gootwine. Role of Placental Lactogen in Sheep. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574339.bard.

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Central problems in sheep and dairy cattle production are reproductive failure due to embryonic/fetal mortality and low birth weights, especially in prolific breeds, and reduced milk yields which adversely affect neonatal survival and economy of production. The sheep placenta expresses lactogenic (ovine placental lactogen, oPL) and somatogenic (ovine placental growth hormone, oGH) hormones. Our research has focused on the biological roles of oPL and oGH in function of the uterine endometrium during gestation and the mammary gland during pregnancy and lactation. Major conclusions were that: ( 1
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