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Auswahl der wissenschaftlichen Literatur zum Thema „Phlébotomes – Parasites“
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Zeitschriftenartikel zum Thema "Phlébotomes – Parasites"
Haddad, N., N. Léger und R. Sadek. „Les phlébotomes du Liban Inventaire faunistique“. Parasite 10, Nr. 2 (Juni 2003): 99–110. http://dx.doi.org/10.1051/parasite/200310299.
Der volle Inhalt der QuelleDepaquit, J., N. Léger, H. Ferté und V. Robert. „Les Phlébotomes de Madagascar (Diptera : Psychodidae)“. Parasite 11, Nr. 2 (Juni 2004): 201–9. http://dx.doi.org/10.1051/parasite/2004112201.
Der volle Inhalt der QuelleTrouillet, J., und G. Vattier-Bernard. „Les phlébotomes(Diptera, Psychodidae)de la Likouala“. Annales de Parasitologie Humaine et Comparée 63, Nr. 6 (1988): 455–61. http://dx.doi.org/10.1051/parasite/1988636455.
Der volle Inhalt der QuelleRusso, J., J. A. Rioux, M. Lambert, P. Rispail, A. Belmonte und S. Berchi. „Chorologie des Phlébotomes de l’Est algérien. (Diptera, Phlebotomidae)“. Annales de Parasitologie Humaine et Comparée 66, Nr. 6 (1991): 247–51. http://dx.doi.org/10.1051/parasite/1991666247.
Der volle Inhalt der QuelleDepaquit, J., N. Léger, H. Ferté, J. A. Rioux, J. C. Gantier, A. Michaelides und P. Economides. „Les phlébotomes de l'Île de Chypre III - Inventaire faunistique“. Parasite 8, Nr. 1 (März 2001): 11–20. http://dx.doi.org/10.1051/parasite/2001081011.
Der volle Inhalt der QuelleTrouillet, J., und O. Faye. „Phlébotomes du Sénégal. Présence dePhlebotomus (Phlebotomus) bergerotiParrot, 1934 (Diptera, Psychodidae)“. Annales de Parasitologie Humaine et Comparée 68, Nr. 2 (1993): 101–3. http://dx.doi.org/10.1051/parasite/1993682101.
Der volle Inhalt der QuelleLe Pont, F., J. C. Gantier, S. Hue und S. Valle. „Phlébotomes du Nicaragua. II. Description deLutzomyia legeraen. sp. (Diptera : Psychodidae)“. Parasite 2, Nr. 1 (März 1995): 75–79. http://dx.doi.org/10.1051/parasite/1995021075.
Der volle Inhalt der QuelleTrouillet, J., Y. Ba, M. Traore-Lamizana, H. G. Zeller und D. Fontenille. „Phlébotomes (Diptera - Psychodidae) du Sénégal. Peuplements du Ferlo. Isolement d'arbovirus.“ Parasite 2, Nr. 3 (September 1995): 289–96. http://dx.doi.org/10.1051/parasite/1995023289.
Der volle Inhalt der QuelleBerchi, S., J. A. Rioux, A. Belmonte und J. Russo. „Un Phlébotome nouveau pour l’Algérie,Phlebotomus (Paraphlebotomus) kazeruni“. Annales de Parasitologie Humaine et Comparée 61, Nr. 4 (1986): 507–8. http://dx.doi.org/10.1051/parasite/1986614507.
Der volle Inhalt der QuelleTrouillet, J., und G. Vattier-Bernard. „Les Phlébotomes(Diptera, Psychodidae)du plateau Koukouya (République Populaire du Congo)“. Annales de Parasitologie Humaine et Comparée 62, Nr. 4 (1987): 345–53. http://dx.doi.org/10.1051/parasite/1987624345.
Der volle Inhalt der QuelleDissertationen zum Thema "Phlébotomes – Parasites"
Akhoundi, Sheikhahmadlou Mohammad. „Contribution à l'étude des leishmanioses en Iran : Phlébotomes, parasites, réservoirs et Homme“. Thesis, Reims, 2013. http://www.theses.fr/2013REIMP209.
Der volle Inhalt der QuelleThis work focused on the leishmaniases in Iran. It includes three topics: i) the Phlebotomine sand flies, ii) the rodent reservoirs of Leishmania major and iii) the Humans.The entomological studies concern both systematics and epidemiology.The systematic part of the study of Phlebotomine sand flies includes several inventories carrired out in several provinces of Iran (North-West, North-East, East and Center of the country). Our results update the distribution of the subgenus Adlerius. We also recorded two new species for the fauna of Iran: Phlebotomus turanicus and P. salangensis. We also carried out evolutive and comparative systematics including specimens from Iran, neighbouring and Mediterranean countries. We coupled morphology to morphometrics and molecular systematics. In the latter approach, we coupled a ribosomal DNA marker to a mitochondrial one. We studied P. perfiliewi s.l. and the subgenus Adlerius. Our epidemiological works focusing on the epidemiology in several parts of the country showed that the studied foci are classical: P. papatasi, and also the females of the Caucasicus group transmit Leishmania major whereas P. sergenti is the vector of L. tropica.The identification of the Leishmania has been done using PCR then RFLP and/or sequencing of rDNA Internal Transcribed Spacer 1.We studied the rodent reservoirs of L. major. Rhombomys opimus and Meriones libycus play an important role and the percentage of L. major infection is high (> 30%).Lastly, we carried out a study on patients from the province of Fars. In this area, leishmaniases are due to L. major, L. tropica and L. infantum. We have typed strains isolated from 42 ot 44 patients. The majority of the strains have been identified as L. major and a few L. tropica
Holzmuller, Philippe. „Etude des mécanismes cellulaires et moléculaires impliqués dans l'activité microbicide des molécules de l'immunité protectrice (monoxyde d'azote) et chimiothérapeutique (antimoine) chez Leishmania“. Montpellier 2, 2002. http://www.theses.fr/2002MON20149.
Der volle Inhalt der QuelleJaouadi, Kaouthier. „Contribution à l'étude de leishmaniose cutanée à Leismania killicki dans un foyer emergent en Tunisie : Parasites, phlébotomes et réservoirs“. Thesis, Reims, 2013. http://www.theses.fr/2013REIMP208.
Der volle Inhalt der QuelleTunisia is an endemic country for leishmaniasis. In 1980, Rioux, Pratlong & Lanotte described from Tunisia a new species they called Leishmania (L.) killicki based on the typing of 30 strains isolated from an outbreak of cutaneous leishmaniasis occuring in the Tataouine area and exhibiting a zymodeme: MON-8. The vector and the reservoir if exixting were still uncknown. Human cases have been mentioned out of this area. Gafsa is a province were cutaneous leishmanaisis due to L. major is endemic.The recent identification of L. killicki as the causative agent of a cutaneous leishmaniasis occuring patients from Metlaoui incitated us to explore this focus. This is the aim of our Ph. D. thesis: identification of the Phlebotomine sand flies vector(s) and possible role of reservoirs using both parasitological, isoenzymatical and molecular approaches.Three main topics have been developed during this work:i) To characterize the Leishmania strains from cutaneous lesions obtained from patients from Gafsa area using molecular tools. We updated epidemiological, spatial and clinical data related to L. killicki leishmaniasis in Tunisia.ii) To carry out entomological studies:-to do sand flies inventories in two distinct areas,-to study the molecular intraspecific variablility in Phlebotomus sergenti and Sergentomyia. minuta by comparison of Tunisian specimens with other ones coming from several countries,-to show the probable role of P. sergenti in the transmission of L. killicki,-to study blood meal origin of engorged females: many Phlebotomus are opportunistic species and Sergentomyia minuta is not exclusively herpetophilic.iii) Lastly, we carried out a study on the Leishmania infection on wild and urban rodents in Metlaoui and around this city. The isoenzymatic and molecular typing show the role of Ctenodactylus gondii in the cycle of L. killicki in Tunisia
Bron, Magali. „Etude écologique et éthologique de Phlebotomus perniciosus (vecteur de leihmaniose canine et humaine) et de Sergentomya minuta dans le Sud Est de la France (région marseillaise)“. Aix-Marseille 3, 1994. http://www.theses.fr/1994AIX30039.
Der volle Inhalt der QuelleMonier, Maëlle. „Modification du métabolisme énergétique et carboné au cours du cycle de Leishmania infantum“. Electronic Thesis or Diss., Université de Toulouse (2023-....), 2024. http://www.theses.fr/2024TLSES117.
Der volle Inhalt der QuelleLeishmania spp. is a unicellular parasite of the Trypanosomatidae family, responsible for leishmaniasis. Leishmania is transmitted to Vertebrate hosts by a sand-fly vector of the Psychodidae family. In the sand-fly, procyclic promastigotes (PP), the non-infectious form of the parasite, localize in the gut. By migrating to the vector's salivary glands, PP become metacyclic promastigotes (PM), the infectious form of the parasite. PM are then transmitted to the vertebrate host through a blood meal, where once in the host's macrophages, they differentiate into amastigote (A), the pathogenic stage of the parasite. The life cycle starts over by further transmission of A to other sand-flies through a new blood meal, where the parasite become PP in the sand fly gut. These environmental variations lead Leishmania to adapt its metabolism to survive. In a first part, this work aimed at studying central carbon metabolism changes and lipid peroxidation, which were characterized using transcriptomic and metabolomic in Leishmania infantum. The transcriptomic and metabolomic results demonstrated significant diversity in gene expression between PP, PM and A, focusing on lipid metabolism between PP and PM, but carbon metabolism between PM and A. In A, compared to PP and PM, glutaminolysis appeared to remain stable but TCA cycle and glycolysis enzymes were down-regulated while metabolites quantities were equal. Lipid peroxidation was decreased in A. β-oxidation was up-regulated however purine and pyrimidine synthesis were strongly down-regulated. These results demonstrate a reduced use of carbon metabolism except for β-oxidation in A compared to PP and PM, meaning that A seems to have a slower metabolism. In a second part of the present work, the investigation on the parasite life cycle focused on lipid metabolism, as part one of this work showed that hydroxylated lipids quantities in A are reduced compared to PP and PM. This decrease was reproduced in vitro in PM using a nordihydroguaiaretic acid (NDGA) treatment (Paloque et al., 2019). NDGA induced a morphologic transition of treated PM (PM NDGA), reversible and non-lethal for the parasite, suggesting a PM - A transition NDGA induced. Transcriptomic and metabolomic studies have demonstrated (i) PM NDGA clustered with A, (ii) PM NDGA exhibited similar amastigotes-specific genes expression as A and (iii) TCA cycle, glycolysis and purine and pyrimidine synthesis expression in PM NDGA were close to A levels but different from PM results. These findings taken together showed a PM - A transition induced by NDGA, creating a potential target for modulating parasite infectivity