Thematische Bibliographien / Pathology

Auswahl der wissenschaftlichen Literatur zum Thema „Pathology“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 31. Juli 2024

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Zeitschriftenartikel zum Thema "Pathology"

1

Dib, Kariml. „b2 integrin signaling in leukocytes“. Frontiers in Bioscience 5, Nr. 1 (2000): d438. http://dx.doi.org/10.2741/pathology.

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Kulkarni, Sushma P. „Therapeutic Pathology- The Pathology of Tomorrow“. Journal of Experimental Pathology 5, Nr. 1 (März 2024): 1–5. http://dx.doi.org/10.33696/pathology.5.045.

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Fournié, Bernard. „Pathology and clinico-pathologic correlations in spondyloarthropathies“. Joint Bone Spine 71, Nr. 6 (November 2004): 525–29. http://dx.doi.org/10.1016/j.jbspin.2004.02.002.

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Miller, N. „Oral pathology: clinical pathologic correlations, 6th edition“. British Dental Journal 212, Nr. 11 (Juni 2012): 567. http://dx.doi.org/10.1038/sj.bdj.2012.516.

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Rudisch, Ansgar, Ruediger Emshoff, Herbert Maurer, Peter Kovacs und Gerd Bodner. „Pathologic-sonographic correlation in temporomandibular joint pathology“. European Radiology 16, Nr. 8 (01.03.2006): 1750–56. http://dx.doi.org/10.1007/s00330-006-0162-0.

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Burgdorf, Walter H. C. „Oral pathology: Clinical pathologic correlations, 5th ed“. Journal of the American Academy of Dermatology 58, Nr. 6 (Juni 2008): 1086–87. http://dx.doi.org/10.1016/j.jaad.2008.02.029.

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Fedotov, V. V. „Gait – Pathology or Physiology“. Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 3, Nr. 4 (18.05.2018): 124–27. http://dx.doi.org/10.26693/jmbs03.04.124.

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Attwood, H. D. „Pathology and Clinical Pathology“. Pathology 20, Nr. 2 (1988): 206. http://dx.doi.org/10.1016/s0031-3025(16)36647-8.

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Tran, Anthony, und Carlos J. Roldan. „Non-Interventional Treatment of Post-Dural-Puncture Headache; High-Flow Oxygen and Pro-Serotonin Agents a Safe and Effective Alternative“. Journal of Experimental Pathology 3, Nr. 2 (10.11.2022): 35–39. http://dx.doi.org/10.33696/pathology.3.038.

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Shirani, Fatemeh, und Leila Barahimi. „Evaluation of the Relationship between Capillaroscopic Symptoms and the Severity of Systemic Lupus Erythematous“. Journal of Experimental Pathology 3, Nr. 2 (30.08.2022): 29–34. http://dx.doi.org/10.33696/pathology.3.037.

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Objectives: Use of imaging tools can detect some specific pathological changes associated with systemic lupus erythematous (SLE). This study aimed to investigate the relationship between capillaroscopic symptoms and the severity of SLE. Methods: This was a cross-sectional study carried out on 56 patients with SLE undergoing capillaroscopy referred to Rasool-e Akram hospital in Tehran in 2018. Capillaroscopy findings were assessed according to demographic characteristics and duration of disease. Results: All patients had at least one positive finding related to capillaroscopy. Regarding capillaroscopic findings, abnormal microvascular structure in 37.5%, decreased vascular density in 78.6%, enlarged cap loop in 32.1%, microhemorrhage in 16.1% and neoangiogenesis in 25.0% were observed. The results revealed higher vascular density loss in women and higher neoangiogenesis in affected men and higher rate of abnormal microvascular structure at older ages and microhemorrhage at younger ages. Direct relationship was also found between duration of disease and microhemorrhage. Conclusion: Almost all patients with SLE undergoing capillaroscopy had at least one pathophysiological change in the capillary bed. The most common pathophysiological change was decreased vascular density, abnormal microvascular structure and capillary loop enlargement.
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Dissertationen zum Thema "Pathology"

1

Bax, Trent Malcolm. „"Internet addiction" in contemporary China: individual pathology or pathology of normalcy?“ Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B45815021.

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Stuart, Persoons Maria Cornelia Johanna. „Cytomegalovirus and vascular pathology“. Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=8496.

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Singh, Mark. „D6 in cutaneous pathology“. Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4636/.

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Chemokines are central to the migration of leukocytes around the body, during both inflammatory and homeostatic conditions. Chemokines mediate their effects by binding to chemokine receptors found on the migrating cell’s surface. Chemokine binding to the chemokine receptor results in signaling, which allows the cell to migrate towards the epicenter of chemokine production. In addition to ‘classical’ chemokine receptors which are involved in leukocyte migration, a discrete family of chemokine receptors exist which are considered to be ‘atypical’, as binding to their cognate ligands does not result in classical signaling as detected by calcium flux assays. One of these atypical chemokine receptors is the chemokinescavenging receptor D6, which can bind to and internalize at least 14 inflammatory CC chemokines in vitro. In addition, an analysis of D6 function in vivo has shown that D6 is important for the resolution of the inflammatory response. D6 KO mice treated with phorbol ester to the shaved dorsal skin developed an inflammatory skin pathology that resembled the human condition psoriasis in many respects. In contrast, WT mice treated with phorbol ester developed a very mild inflammatory response, which quickly resolved. These data suggested that a loss of D6 expression ‘primed’ the mouse to develop a psoriasisform pathology, requiring only minor irritation/trauma to develop the pathology. Similarly, histologically normal (uninvolved) skin from a psoriatic patient has a propensity to developing inflammatory lesions upon minor trauma, and could also be suggested to be ‘primed’ for lesion development. Collectively, these data led us to the following hypothesis, ‘A loss of D6 expression in uninvolved psoriatic skin is associated with the development of psoriatic lesions’. To test this hypothesis, D6 expression in clinical samples from psoriasis patients was analysed. Full thickness biopsies from psoriasis patients were taken from a histologically normal site (uninvolved psoriatic skin), in addition to an elliptical biopsy covering the skin directly adjacent to the psoriatic lesion (peri-lesional psoriatic skin), in addition to the lesion itself (lesional psoriatic skin). D6 expression was analysed in these biopsies by QPCR and immuno-staining. It was observed that D6 expression was significantly elevated in psoriatic skin compared to healthy control skin. In particular, in uninvolved psoriatic skin D6 was significantly increased compared to healthy control skin, or peri-lesional 3 psoriatic skin or lesional psoriatic skin. The increase in D6 expression in uninvolved psoriatic skin localised to the epidermis and the LVs. A significant increase in PBMC-D6 expression was also noted in psoriatic patients compared to healthy control PBMCs. These data suggest that at sites not directly involved in the pathology, D6 is elevated in an attempt to limit inflammation-induced damage. Further immuno-staining showed the inflammatory CC chemokines CCL2 and CCL5 (both high affinity D6-binding ligands) were detected in uninvolved psoriatic epidermis, but were apparently unable to mediate their function due to the lack of significant leukocyte infiltration into the tissue. These data gave rise to the idea that D6 in uninvolved psoriatic skin was significantly elevated in an attempt to block the release of inflammatory CC chemokines into the dermis, and subsequent migration of inflammatory leukocytes into the tissue, and the onset of lesion formation. Interestingly, D6 expression on the epidermis was strongest towards the lower layers of the epidermis, which suggested a role for epidermal- D6 in ‘barrier function’, preventing the uncontrolled release of inflammatory CC chemokines into the dermis. In addition to inflammatory CC chemokines, a variety of inflammatory cytokines have been previously detected in uninvolved psoriatic skin. Several of these cytokines were shown to increase D6 expression in vitro in this study. Therefore, it is possible the significant increase in D6 expression in uninvolved psoriatic skin is partly mediated by cytokine stimulation. A loss of D6 expression was observed when comparing uninvolved psoriatic skin and perilesional psoriatic skin. These data suggested that a loss of D6 expression occurs directly before the onset of lesion formation. It was also shown in this study that a loss of D6 expression could occur after micro-trauma to uninvolved psoriatic skin, which suggests a possible mechanism of how D6 expression is lost in peri-lesional psoriatic skin. To analyze whether the increase in D6 expression in psoriatic skin was disease specific, or a generic response to cutaneous inflammation, D6 expression in eczema skin was studied. It was found that D6 expression in eczema skin is elevated compared to healthy control skin, but less so compared to psoriatic skin. Collectively these data suggest that increased D6 expression may be a feature of inflammatory skin diseases.
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Dougherty, Janis Ruden Ronald A. „The pathology of addiction /“. Online version of thesis, 2009. http://hdl.handle.net/1850/11763.

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Kubarych, Thomas. „Narcissism, personality and personality pathology“. Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/28380.

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This thesis used the methods of differential, cognitive and theoretical psychology to investigate the relationships between pathology narcissism and maladaptive personality and behaviour in general, and to attempt the beginnings of construct validation of M. Scott Peck's proposed 'evil' subtype of the DSM-IV Narcissistic Personality Disorder in particular. After a review of theoretical and empirical contributions to the psychology of narcissism, two empirical studies were conducted. In the first, joint self-report survey research using 338 subjects investigated the psychometric structure of narcissism, normal and abnormal personality, and constructs theoretically related to narcissism. Item-level exploratory principal components analysis and confirmatory factor analysis resulted in new subscales for the Narcissistic Personality Inventory. Scale-level exploratory principal components analysis of the combined questionnaires found evidence for a five-factor structure of abnormal personality. One of the five factors was related to narcissistic will to power and low agreeableness; another was related to narcissistic self-love and extraversion. Confirmatory factor analysis of a subset of the data found fair fit for the model. The second study investigated the relationships between narcissism, compartmentalisation, splitting, attribution style and response to disconfirming feedback. No evidence was found to support the hypothesis that narcissists have compartmentalised self-concepts. Moderate test-retest and alternative-form reliability data were obtained for the card-sort task used to assess compartmentalisation. No evidence was found that narcissists use splitting to translocate unwanted self-aspects onto others. Narcissistic dominance was associated with claiming personal credit for positive outcomes, while narcissistic vulnerability was associated with self-blame for negative events. Multiple regression with interaction terms indicated that the relationship between narcissism and response to disconfirming feedback is a function of other personality traits such as neuroticism, and may have opposite effects in different personalities and circumstances.
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Wu, Bing. „Pathology of rotator cuff tendonopathy“. University of Western Australia. Centre for Orthopaedic Research, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0032.

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Tendonopathy, resulting in the loss of mechanical strength of a tendon, is a serious health problem affecting many people. The common symptom of tendonopathy is pain – patients' daily activities, their participation in sport and exercise, and their ability to work are greatly compromised. Tendonopathy is considered to be a degenerative disorder caused by repetitive injury of the tendon. The most common tendon lesions are Achilles tendon rupture, lateral epicondylitis (tennis elbow) and rotator cuff tear. However, in spite of its clinical significance, our knowledge about tendonopathy is still very poor. This research was undertaken to investigate the pathology of tendonopathy. It is proposed that apoptosis, autophagic cell death and myofibroblasts play a role in the progression of tendonopathy in the rotator cuff; the aim of this study was therefore to determine if this was indeed the case. Tendon tissues were collected from 30 patients suffering from rotator cuff tears. A terminal deoxynucleotidyl transferase biotin-dUTP nick end labelling (TUNEL assay) was performed to detect apoptosis. Autophagic cell death of the tenocytes in the ruptured rotator cuff tendon was detected by immunohistochemical staining for ubiquitin. Myofibroblasts were identified immunohistochemically with anti-alpha-smooth muscle actin (anti--SMA) antibody. The distribution of apoptosis, autophagic cell death and myofibroblasts, as well as the total cell density, were assessed respectively and were correlated using a four-category (i.e. graded from 0-3) degeneration of collagen matrix. – 6 – The results showed that apoptosis, autophagic cell death and myofibroblasts were observed in all of the samples. The highest percentage of autophagic cell death was evidenced in the Grade 2 matrix, while the percentage of apoptosis increased significantly with the increase of matrix degeneration from Grade 0-3; a similar pattern was found for myofibroblasts. The total cell numbers varied among the matrix grades, with the maximum and minimum percentages occurring in Grades 1 and 3, respectively. It can be concluded that apoptosis, autophagic cell death and myofibroblasts might be closely related to the damage of the extracellular matrix (ECM) structure.
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Goszer, Libby. „Adolescent suicidality and attachment pathology“. Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape15/PQDD_0027/NQ37706.pdf.

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Bruijn, Lambertus Matthias de. „Automatic classification of pathology reports“. [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5936.

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Kee, Francis. „Molecular pathology of hepatocellular carcinoma“. Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/b40203785.

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Roe, Thomas John Kelsey. „TGFβ in protection and pathology“. Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406711.

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Bücher zum Thema "Pathology"

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J, Sciubba James, Hrsg. Oral pathology: Clinical-pathologic correlations. 2. Aufl. Philadelphia: Saunders, 1993.

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J, Sciubba James, und Jordan Richard C. K, Hrsg. Oral pathology: Clinical pathologic correlations. 5. Aufl. St. Louis, Mo: Saunders/Elsevier, 2008.

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J, Sciubba James, und Jordan Richard C. K, Hrsg. Oral pathology: Clinical pathologic correlations. 6. Aufl. St. Louis, Mo: Elsevier/Saunders, 2012.

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Regezi, Joseph A. Oral pathology: Clinical pathologic correlations. 5. Aufl. St. Louis, Mo: Saunders/Elsevier, 2008.

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Regezi, Joseph A. Oral pathology: Clinical pathologic correlations. 4. Aufl. St. Louis, Mo: Saunders, 2003.

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Regezi, Joseph A. Oral pathology: Clinical pathologic correlations. 5. Aufl. St. Louis, Mo: Saunders Elsevier, 2008.

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J, Sciubba James, Hrsg. Oral pathology: Clinical pathologic correlations. 3. Aufl. Philadelphia: Saunders, 1999.

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J, Sciubba James, Hrsg. Oral pathology: Clinical-pathologic correlations. Philadelphia: Saunders, 1989.

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Jones, Thomas Carlyle. Veterinary pathology. 6. Aufl. Baltimore, Md: Williams & Wilkins, 1997.

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Robert, Muir. Muir's textbook of pathology. Sevenoaks: E. Arnold, 1992.

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Buchteile zum Thema "Pathology"

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Häberle, Lena. „Pathology Reporting, Surgical Pathology“. In Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5500-1.

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Häberle, Lena. „Pathology Reporting, Surgical Pathology“. In Pathology of the Pancreas, 182–84. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62416-3_5500.

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Frank, J. Howard, J. Howard Frank, Michael C. Thomas, Allan A. Yousten, F. William Howard, Robin M. Giblin-davis, John B. Heppner et al. „Pathology“. In Encyclopedia of Entomology, 2765. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_2803.

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Leong, A. S. Y., und C. Pairojkul. „Pathology“. In Hilar Cholangiocarcinoma, 25–41. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6473-6_3.

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Kornauth, Christoph, Ana-Iris Schiefer und Ingrid Simonitsch-Klupp. „Pathology“. In Non-Hodgkin's Lymphoma in Childhood and Adolescence, 67–95. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11769-6_7.

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Rother, Klaus, Gerd O. Till und G. Maria Hänsch. „Pathology“. In The Complement System, 343–513. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-58753-5_3.

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Scadding, J. G., und D. N. Mitchell. „Pathology“. In Sarcoidosis, 13–35. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4899-2971-6_2.

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Woodley, Cheryl M., Russell A. Harley, James H. Nicholson und Taylor L. Reynolds. „Pathology“. In Diseases of Coral, 4–15. Hoboken, NJ: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118828502.ch2.

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Ulrich, W., und G. Syré. „Pathology“. In Systemic Lupus Erythematosus, 204–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71642-3_13.

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Cesarman, Ethel, und Amy Chadburn. „Pathology“. In HIV-associated Hematological Malignancies, 1–25. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26857-6_1.

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Konferenzberichte zum Thema "Pathology"

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„Front Matter: Volume 10581“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2323941.

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Murugesan, Balamurali, Sakthivel Selvaraj, Kaushik Sarveswaran, Keerthi Ram, Jayaraj Joseph und Mohanasankar Sivaprakasam. „Deep detection and classification of mitotic figures“. In Digital Pathology, herausgegeben von John E. Tomaszewski und Aaron D. Ward. SPIE, 2019. http://dx.doi.org/10.1117/12.2508770.

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Brieu, Nicolas, Peter Caie, Christos Gavriel, Günter Schmidt und David J. Harrison. „Context-based interpolation of coarse deep learning prediction maps for the segmentation of fine structures in immunofluorescence images“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292794.

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Akbar, Shazia, Anne L. Martel, Mohammad Peikari, Sherine Salama und Sharon Nofech-Mozes. „Determining tumor cellularity in digital slides using ResNet“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292813.

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Gangeh, Mehrdad J., Rene Bidart, Mohammad Peikari, Anne L. Martel, Ali Ghodsi, Sherine Salama und Sharon Nofech-Mozes. „Localization and classification of cell nuclei in post-neoadjuvant breast cancer surgical specimen using fully convolutional networks“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292815.

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Qiu, Yuchen, Yue Du, Roy Zhang, Abolfazl Zargari, Theresa Thai, Camille Gunderson, Katherine Moxley, Hong Liu und Bin Zheng. „A performance comparison of low- and high-level features learned by deep convolutional neural networks in epithelium and stroma classification“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292840.

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Kleczek, Pawel, Martyna Lech, Joanna Jaworek-Korjakowska, Grzegorz Dyduch und Ryszard Tadeusiewicz. „Segmentation of black ink and melanin in skin histopathological images“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292859.

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Bulten, Wouter, Geert J. S. Litjens, Christina A. Hulsbergen-van de Kaa und Jeroen van der Laak. „Automated segmentation of epithelial tissue in prostatectomy slides using deep learning“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292872.

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Sibley, Adam R., Maryellen L. Giger, Vladimir Liarski und Marcus Clark. „Simultaneous segmentation and classification of multichannel immuno-fluorescently labeled confocal microscopy images using deep convolutional neural networks“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2292934.

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Ma, Zhaoxuan, Jiayun Li, Hootan Salemi, Corey Arnold, Beatrice S. Knudsen, Arkadiusz Gertych und Nathan Ing. „Semantic segmentation for prostate cancer grading by convolutional neural networks“. In Digital Pathology, herausgegeben von Metin N. Gurcan und John E. Tomaszewski. SPIE, 2018. http://dx.doi.org/10.1117/12.2293000.

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Berichte der Organisationen zum Thema "Pathology"

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Adeogun, M., J. Bunch, A. Dexter, C. Dondi, T. Murta, C. Nikula, M. Shaw et al. Metrology for Digital Pathology. Digital pathology cross-theme project report. National Physical Laboratory, Oktober 2021. http://dx.doi.org/10.47120/npl.as102.

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Pitlick, Frances A. Atlas of Tumor Pathology. Fort Belvoir, VA: Defense Technical Information Center, Oktober 1990. http://dx.doi.org/10.21236/ada248434.

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Pitlick, Frances A. Atlas of Tumor Pathology. Fort Belvoir, VA: Defense Technical Information Center, Oktober 1990. http://dx.doi.org/10.21236/ada241000.

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Williamson, Sean. Pathology of bladder cancer. BJUI Knowledge, November 2019. http://dx.doi.org/10.18591/bjuik.0135.

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Trock, Bruce J. Prostate Cancer Pathology Resource Network. Fort Belvoir, VA: Defense Technical Information Center, Juli 2014. http://dx.doi.org/10.21236/ada613993.

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Trock, Bruce. Prostate Cancer Pathology Resource Network. Fort Belvoir, VA: Defense Technical Information Center, Juli 2012. http://dx.doi.org/10.21236/ada566504.

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Trock, Bruce. Prostate Cancer Pathology Resource Network. Fort Belvoir, VA: Defense Technical Information Center, Juli 2013. http://dx.doi.org/10.21236/ada586663.

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Ordway, Gregory A., Michelle J. Chandley und Jessica D. Crawford. White Matter Glial Pathology in Autism. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada613167.

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Bhargava, Rohit. Infrared Spectroscopic Imaging for Prostate Pathology. Fort Belvoir, VA: Defense Technical Information Center, März 2008. http://dx.doi.org/10.21236/ada510089.

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Ordway, Gregory A., und Michelle J. Chandley. White Matter Glia Pathology in Autism. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada590212.

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