Auswahl der wissenschaftlichen Literatur zum Thema „Pathologies pulmonaires“
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Zeitschriftenartikel zum Thema "Pathologies pulmonaires"
Bourguignon, Chloé, Charlotte Vernisse, Joffrey Mianné, Mathieu Fieldès, Engi Ahmed, Aurélie Petit, Isabelle Vachier et al. „Les organoïdes pulmonaires“. médecine/sciences 36, Nr. 4 (April 2020): 382–88. http://dx.doi.org/10.1051/medsci/2020056.
Der volle Inhalt der QuelleAder, F., S. Nseir, B. Guery und I. Tillie-Leblond. „Aspergillose pulmonaire aiguë invasive et pathologies pulmonaires chroniques“. Revue des Maladies Respiratoires 23, Nr. 3 (Juni 2006): 11–20. http://dx.doi.org/10.1016/s0761-8425(06)71583-2.
Der volle Inhalt der QuelleBonay, Marcel, Françoise Camus und Catherine Bancal. „Pathologies pulmonaires et sport“. EMC - Pneumologie 1, Nr. 1 (Januar 2004): 1–9. http://dx.doi.org/10.1016/s1155-195x(02)00070-1.
Der volle Inhalt der QuelleBoutros, J., und S. Leroy. „Pathologies pulmonaires infiltratives diffuses“. Revue des Maladies Respiratoires Actualités 12, Nr. 2 (Oktober 2020): 298–303. http://dx.doi.org/10.1016/s1877-1203(20)30127-0.
Der volle Inhalt der QuelleMassin, Nicle, und Marie-Nathalie Kolopp-Sarda. „Pathologies pulmonaires d'origine immuno-allergique en milieu professionnel“. Revue Française des Laboratoires 2004, Nr. 361 (März 2004): 39–46. http://dx.doi.org/10.1016/s0338-9898(04)90093-5.
Der volle Inhalt der QuelleDeslée, G. „Macrolides à faible dose dans les pathologies pulmonaires“. Revue des Maladies Respiratoires Actualités 5, Nr. 1 (2013): 54–59. http://dx.doi.org/10.1016/s1877-1203(13)70359-8.
Der volle Inhalt der QuelleBarras, Guillemette, Yann Michel, Noémie Wagner und Louis Loutan. „Pathologies [b]pulmonaires[/b] au retour de voyage“. Revue Médicale Suisse 8, Nr. 340 (2012): 1000–1005. http://dx.doi.org/10.53738/revmed.2012.8.340.1000.
Der volle Inhalt der QuelleKambouchner, M., und J. F. Bernaudin. „Les lymphatiques dans les pathologies pulmonaires acquises non tumorales“. Revue de Pneumologie Clinique 69, Nr. 3 (Juni 2013): 170–74. http://dx.doi.org/10.1016/j.pneumo.2012.12.008.
Der volle Inhalt der QuelleBoyer, L., L. Savale, J. Boczkowski und S. Adnot. „Sénescence cellulaire et pathologies pulmonaires : exemple de la BPCO“. Revue des Maladies Respiratoires 31, Nr. 10 (Dezember 2014): 893–902. http://dx.doi.org/10.1016/j.rmr.2014.07.015.
Der volle Inhalt der QuelleLacombe, C., L. Monnier-Cholley, M. Lewin, L. Arrivé und J. M. Tubiana. „THO12 Imagerie des pathologies pulmonaires chez le patient HIV“. Journal de Radiologie 87, Nr. 10 (Oktober 2006): 1561. http://dx.doi.org/10.1016/s0221-0363(06)87994-1.
Der volle Inhalt der QuelleDissertationen zum Thema "Pathologies pulmonaires"
Couffin, Rozenn Lagarde André. „Inter-relations pathologie générale et pathologie odonto-stomatologique pathologies pulmonaires /“. [S.l.] : [s.n.], 2004. http://theses.univ-nantes.fr/thesemed/CDcouffin.pdf.
Der volle Inhalt der QuelleFlamein, Florence. „Anomalies génétiques d'ABCA3 dans les détresses respiratoires néonatales sévères et les pathologies alvéolo-interstitielles de l'enfant“. Paris 6, 2011. http://www.theses.fr/2011PA066290.
Der volle Inhalt der QuelleMontigaud, Yoann. „Modèles précliniques ex vivo pour l'étude de la délivrance pulmonaire d'aérosols dans le traitement de pathologies pulmonaires“. Thesis, Lyon, 2020. http://www.theses.fr/2020LYSEM028.
Der volle Inhalt der QuellePulmonary delivery seem to be a preferential choice for the treamt of respiratory diseases. However, optimal targeting should be reached to increase efficacy and decrase the risk of side effects. Thus, research is needed to improve aerosol delivery devices. However, ethical restrictions related to human experiment are not in agreement with the previous statement. Therefore, preclinical model are needed but could lack of relevance or generated date could be hard to extrpolate. The present work aimed to develop a panel of preclinical ex vivo respiratory models to systematise knowledge to facilitate the clinical transfer of aerosol technologies. For each developed ex vivo model, the aerosol deposition pattern was assessed and compared to human and/or animal data to ensure the extrapolability of the results and to position the model among the available preclinical models. Applications, such as the optimal position of a nebuliser during invasive mechanical ventilation or the deposition profile of electronic cigarette aerosol, were performed. The developed ex vivo models showed comparability with patients deposition profile of aersosol, as well as their utility as a new preclinical tool fitting 3R guidelines to complete exisiting preclinical models in aerosol therapy
Merrien, Florence Caroff Nathalie. „Pathologies pulmonaires liées aux endotoxines bactériennes le modèle de la byssinose /“. [S.l.] : [s.n.], 2004. http://theses.univ-nantes.fr/thesemed/.pdf.
Der volle Inhalt der QuelleButeau, Marion. „Analyse d'images radiographiques du thorax pour l'aide au diagnostic des pathologies pulmonaires“. Rennes 1, 1998. http://www.theses.fr/1998REN10011.
Der volle Inhalt der QuelleKryza, Thomas. „Etude de l'implication de la kallicréine 12 dans le remodelage tissulaire associé aux pathologies pulmonaires“. Thesis, Tours, 2013. http://www.theses.fr/2013TOUR3314.
Der volle Inhalt der QuelleIn this thesis, we studied the involvement of the serine protease kallikrein-12 (KLK12), in lung carcinogenesis. The KLK12 is known to be overexpressed in non-small cell lung cancer but its role in carcinogenesis is not clearly established. Our work shows that it possesses a proangiogenic effect on pulmonary endothelial cells. Indeed, KLK12 stimulates lung endothelial cells migration notably by modulating the extracellular matrix architecture. On the other hand, KLK12 regulates the bioavailability of growth factors associated with angiogenesis such as plateletderived growth factor-B. In addition, our work has identified a possible regulation of the expression of KLK12 by hypoxia, which would explain its overexpression in lung tumors and would reinforce its involvement in angiogenesis. The confirmation in vivo of these mechanisms could help consider KLK12 as a therapeutic target for regulating tumor angiogenesis
GOUALIN, POUSSET PASCALE. „Interet du diagnostic echographique dans la prise en charge des pathologies pleuro-pulmonaires du foetus“. Angers, 1993. http://www.theses.fr/1993ANGE1054.
Der volle Inhalt der QuelleAndrault, Pierre-Marie. „Rôle des cathepsines à cystéine dans la régulation du peptide antimicrobien LL-37 lors de pathologies inflammatoire chroniques pulmonaires“. Thesis, Tours, 2015. http://www.theses.fr/2015TOUR4035/document.
Der volle Inhalt der QuelleDuring chronic inflammatory lung diseases like cystic fibrosis or COPD, proteases/antiproteases imbalance leads to pulmonary tissue degradation and compromise antimicrobial barrier. Cysteine cathepsins are involved in the proteolytic inactivation of several lung antimicrobial peptides (AMPs) such as SLPI, lactoferrin and β- defensins -2 and -3 during emphysema or cystic fibrosis. During this thesis, we studied the ability of cathepsins B, K, L and S to degrade LL-37, which is an important AMP in lung immunity. Only cathepsins K and S degrade readily LL-37 and inactivate its antimicrobial property. Conversely, LL-37 is a competitive inhibitor of cathepsin L. Beside, lung expression of human cathepsin S is significantly increased in smokers with or without COPD compared to non-smokers. Cigarette smoke that is a major source of oxidative stress significantly increases the expression and activity of cathepsin S. Despite an unfavorable oxidative environment, cathepsin S retains its proteolytic activity toward LL-37 and thus could participate to COPD exacerbation
Lemjabbar, Alaoui Hassan. „Implication de la gélatinase B (92 kDa) au cours des pathologies pulmonaires humaines : fibrose interstitielle diffuse et etat de mal asthmatique“. Paris 12, 1998. http://www.theses.fr/1998PA120028.
Der volle Inhalt der QuelleGoven, Delphine. „Régulation de l’hème oxygénase-1 dans les macrophages au cours des pathologies pulmonaires liées à l’exposition de la fumée de cigarette“. Thesis, Paris Est, 2009. http://www.theses.fr/2009PEST0051.
Der volle Inhalt der QuelleChronic cigarette smoking, a source of oxidants, is an important risk factor for lung emphysema and primary spontaneous pneumothorax development. Alveolar macrophages are mainly involved in lung inflammation observed in these pathologies through the production of metalloproteases and reactive oxygen species resulting to protease/anti-protease and oxidant/anti-oxidant imbalances. Heme oxygenase-1 (HO-1), mainly expressed in macrophages, is a key enzyme in pulmonary anti-oxidant defences. Therefore, the first aim of our studies was to investigate the expression and cellular localisation of HO-1 and its potential regulators (Nrf2, Keap1, Bach1 and HIF-1a) in alveolar macrophages from smoking related lung emphysema and primary spontaneous pneumothorax. Regulation pathways involved in expression of these proteins were assessed in vitro in macrophage cell line THP-1 exposed or not to cigarette smoke condensate and with or without hypoxia-reoxygenation mimicking parts of events induced by atelectasia-reexpansion during recurrent pneumothorax constitution and treatment. In these studies, we showed an altered expression of Nrf2/Keap1- Bach1 pathway associated with a reduced expression of anti-oxidants enzymes, like HO-1, in alveolar macrophages from smoking related lung emphysema patients, despite an important oxidative stress. These alterations might be related to cigarette smoke condensate activated ERK1/2 and JNK MAPKinases as observed in THP-1 cells. Furthermore, we showed that HO- 1 system induction was mediated by HIF-1a instead of Nrf2 pathway in alveolar macrophages from smoking related recurrent primary spontaneous pneumothorax. These findings may contribute to a better knowledge of the pathophysiology of lung emphysema and could provide new therapeutic approaches based on preservation and/or restoration of Nrf2/Keap1-Bach1 equilibrium. Our results also suggest that the pathophysiology of primary spontaneous pneumothorax could be different in smokers and non smokers. Spontaneous pneumothorax in smokers is associated with lung oxidative stress and the orchestrated induction of HO-1 probably via HIF-1a. These results provide a new link between oxidative stress and hypoxia/reoxygenation
Bücher zum Thema "Pathologies pulmonaires"
Herbert, Spencer. Pathology of the lung. 4. Aufl. Oxford [Oxfordshire]: Pergamon Press, 1985.
Den vollen Inhalt der Quelle findenChanussot, Jean-Claude. Kinésithérapie respiratoire : Pathologie pulmonaire. Editions Masson, 1997.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Pathologies pulmonaires"
Lacombe, P., M. El Hajjam und S. Binsse. „Malformations artérioveineuses pulmonaires: Du diagnostic au traitement“. In Thérapeutiques endovasculaires des pathologies veineuses, 277–84. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0291-6_19.
Der volle Inhalt der Quelle„Pathologies cardio-pulmonaires“. In Pathologies maternelles et grossesse, 223–72. Elsevier, 2014. http://dx.doi.org/10.1016/b978-2-294-71330-9.00009-x.
Der volle Inhalt der QuelleBenachi, Alexandra. „Pathologies pulmonaires et diaphragme“. In Conduites pratiques en médecine foetale, 87–100. Elsevier, 2010. http://dx.doi.org/10.1016/b978-2-294-70626-4.00005-9.
Der volle Inhalt der QuelleLaurent, François, Ilyes Benlala und Gaël Dournes. „Pathologie pulmonaire professionnelle“. In Imagerie des Pneumopathies Interstitielles Diffuses (PID), 171–82. Elsevier, 2023. http://dx.doi.org/10.1016/b978-2-294-78190-2.00015-7.
Der volle Inhalt der Quelle„PATHOLOGIE TUMORALE DU PARENCHYME PULMONAIRE“. In Imagerie Thoracique, 277–451. Elsevier, 2013. http://dx.doi.org/10.1016/b978-2-294-71321-7.50014-8.
Der volle Inhalt der Quelle„TROISIÈME CHAPITRE La pathologie pulmonaire“. In Apprenez à respirer à vos enfants, 23–38. EDP Sciences, 2015. http://dx.doi.org/10.1051/978-2-7598-1897-6.c007.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Pathologies pulmonaires"
Lafont, J., J. H. Catherine, M. Lejeune, U. Ordioni, R. Lan und F. Campana. „Manifestations buccales de la sclérose tubéreuse de Bourneville“. In 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603014.
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