Auswahl der wissenschaftlichen Literatur zum Thema „NFATc [Nuclear Factor of Activated T-cell]“
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Zeitschriftenartikel zum Thema "NFATc [Nuclear Factor of Activated T-cell]"
Luo, C., E. Burgeon und A. Rao. „Mechanisms of transactivation by nuclear factor of activated T cells-1.“ Journal of Experimental Medicine 184, Nr. 1 (01.07.1996): 141–47. http://dx.doi.org/10.1084/jem.184.1.141.
Der volle Inhalt der QuelleMasuda, E. S., Y. Naito, H. Tokumitsu, D. Campbell, F. Saito, C. Hannum, K. Arai und N. Arai. „NFATx, a novel member of the nuclear factor of activated T cells family that is expressed predominantly in the thymus.“ Molecular and Cellular Biology 15, Nr. 5 (Mai 1995): 2697–706. http://dx.doi.org/10.1128/mcb.15.5.2697.
Der volle Inhalt der QuelleLunde, Ida G., Heidi Kvaløy, Bjørg Austbø, Geir Christensen und Cathrine R. Carlson. „Angiotensin II and norepinephrine activate specific calcineurin-dependent NFAT transcription factor isoforms in cardiomyocytes“. Journal of Applied Physiology 111, Nr. 5 (November 2011): 1278–89. http://dx.doi.org/10.1152/japplphysiol.01383.2010.
Der volle Inhalt der QuelleWu, Chia-Cheng, Shu-Ching Hsu, Hsiu-ming Shih und Ming-Zong Lai. „Nuclear Factor of Activated T Cells c Is a Target of p38 Mitogen-Activated Protein Kinase in T Cells“. Molecular and Cellular Biology 23, Nr. 18 (15.09.2003): 6442–54. http://dx.doi.org/10.1128/mcb.23.18.6442-6454.2003.
Der volle Inhalt der QuelleAramburu, J., L. Azzoni, A. Rao und B. Perussia. „Activation and expression of the nuclear factors of activated T cells, NFATp and NFATc, in human natural killer cells: regulation upon CD16 ligand binding.“ Journal of Experimental Medicine 182, Nr. 3 (01.09.1995): 801–10. http://dx.doi.org/10.1084/jem.182.3.801.
Der volle Inhalt der QuelleMartínez-Martínez, S., P. Gómez del Arco, A. L. Armesilla, J. Aramburu, C. Luo, A. Rao und J. M. Redondo. „Blockade of T-cell activation by dithiocarbamates involves novel mechanisms of inhibition of nuclear factor of activated T cells.“ Molecular and Cellular Biology 17, Nr. 11 (November 1997): 6437–47. http://dx.doi.org/10.1128/mcb.17.11.6437.
Der volle Inhalt der QuelleWang, Qingding, Yuning Zhou, Lindsey N. Jackson, Sara M. Johnson, Chi-Wing Chow und B. Mark Evers. „Nuclear factor of activated T cells (NFAT) signaling regulates PTEN expression and intestinal cell differentiation“. Molecular Biology of the Cell 22, Nr. 3 (Februar 2011): 412–20. http://dx.doi.org/10.1091/mbc.e10-07-0598.
Der volle Inhalt der QuelleAmasaki, Yoshiharu, Esteban S. Masuda, Ryu Imamura, Ken-ichi Arai und Naoko Arai. „Distinct NFAT Family Proteins Are Involved in the Nuclear NFAT-DNA Binding Complexes from Human Thymocyte Subsets“. Journal of Immunology 160, Nr. 5 (01.03.1998): 2324–33. http://dx.doi.org/10.4049/jimmunol.160.5.2324.
Der volle Inhalt der QuelleSUGIMOTO, TOSHIRO, MASAKAZU HANEDA, HIROTAKA SAWANO, KEIJI ISSHIKI, SHIRO MAEDA, DAISUKE KOYA, KEN INOKI, HITOSHI YASUDA, ATSUNORI KASHIWAGI und RYUICHI KIKKAWA. „Endothelin-1 Induces Cyclooxygenase-2 Expression Via Nuclear Factor of Activated T-Cell Transcription Factor in Glomerular Mesangial Cells“. Journal of the American Society of Nephrology 12, Nr. 7 (Juli 2001): 1359–68. http://dx.doi.org/10.1681/asn.v1271359.
Der volle Inhalt der QuelleLiu, Yewei, Zoltán Cseresnyés, William R. Randall und Martin F. Schneider. „Activity-dependent nuclear translocation and intranuclear distribution of NFATc in adult skeletal muscle fibers“. Journal of Cell Biology 155, Nr. 1 (01.10.2001): 27–40. http://dx.doi.org/10.1083/jcb.200103020.
Der volle Inhalt der QuelleDissertationen zum Thema "NFATc [Nuclear Factor of Activated T-cell]"
Lejard, Véronique. „Etude de la régulation transcriptionnelle du collagène de type I dans les fibroblastes tendineux“. Paris 6, 2007. http://www.theses.fr/2007PA066465.
Der volle Inhalt der QuelleSolovey, Maria [Verfasser], und Andreas [Akademischer Betreuer] Burchert. „Nuclear factor of activated T-cells, NFATC1, governs FLT3-ITD-driven hematopoietic stem cell transformation and a poor prognosis in AML / Maria Solovey ; Betreuer: Andreas Burchert“. Marburg : Philipps-Universität Marburg, 2019. http://d-nb.info/1202110460/34.
Der volle Inhalt der QuelleCatherinet, Claire. „Etude des effecteurs de la voie Ca2+/Calmoduline dans les leucémies aiguës lymphoblastiques T“. Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCC293/document.
Der volle Inhalt der QuelleT cell acute lymphoblastic leukemia (T-ALL) is an aggressive malignancy of T cell progenitors. Despite initial response to chemotherapy, relapses remain frequent in children and adults. Previous results identify sustained activation of Calcineurin (Cn)/NFAT signaling pathway in human T-ALL and murine T-ALL models. Importantly, they also demonstrated Cn is essential for T-ALL Leukemia Initiating Cells (LIC) activity in a murine model of T-ALL induced by an activated allele of NOTCH1 (ICN1). Since pharmacologic inhibition of Cn induces side effects, we aim to identify downstream effectors involved in T-ALL. NFAT (Nuclear Factor of Activated T cells) factors play crucial roles downstream Cn during development and activation of T cells. To address their role in T-ALL, we generated mouse ICN1-induced T-ALL in which NFAT genes can be inactivated either single or in combination following Cre-mediated gene deletion. We demonstrated that (i) NFAT factors are required downstream Cn for LIC activity in T-ALL in vivo (ii) ex vivo NFAT factors deletion alters survival, proliferation and migration of T-ALL (iii) NFAT1, 2 and 4 have a largely redundant function in T-ALL. Moreover, the NFAT-dependant transcriptome allowed to identify important targets (CDKN1A, MAFB) involved in T-ALL survival and proliferation in vitro. Calmodulin-dependant kinases (CaMK) are kinases activated by calcium signaling in T cells. We showed that pharmacologic inhibition of CaMKs in ICN1-induced T-ALL alters survival and proliferation of T-ALL in vitro. Beside, specific inhibition by RNA interference of CaMKIIg and CaMKIId suggests a putative role of these kinases in T-ALL maintenance
Arabanian, Laleh Sadat. „Role of NFAT (Nuclear Factor of Activated T Cells) Transcription Factors in Hematopoiesis“. Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-99739.
Der volle Inhalt der QuelleUlrich, Jason Daniel. „The regulaton and function of nuclear factor of activated T-cells in neurons“. Diss., University of Iowa, 2011. https://ir.uiowa.edu/etd/2782.
Der volle Inhalt der QuelleArabanian, Laleh Sadat [Verfasser], Gerhard [Akademischer Betreuer] Rödel, Alexander [Akademischer Betreuer] Kiani und Gerhard [Akademischer Betreuer] Ehninger. „Role of NFAT (Nuclear Factor of Activated T Cells) Transcription Factors in Hematopoiesis / Laleh Sadat Arabanian. Gutachter: Gerhard Rödel ; Alexander Kiani ; Gerhard Ehninger. Betreuer: Gerhard Rödel“. Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2012. http://d-nb.info/1068148918/34.
Der volle Inhalt der QuelleBaggott, Rhiannon Rebecca. „Role of the plasma membrane calcium ATPase as a negative regulator of angiogenesis“. Thesis, University of Wolverhampton, 2014. http://hdl.handle.net/2436/332139.
Der volle Inhalt der QuelleZhang, Danfeng [Verfasser], und Benito A. [Akademischer Betreuer] Yard. „The role of nuclear factor of activated T cells 5 (NFAT5) in inflammation and the potential use of bifunctional enzyme triggered carbon monoxide releasing molecule in treatment of systemic inflammation / Danfeng Zhang ; Betreuer: Benito Yard“. Heidelberg : Universitätsbibliothek Heidelberg, 2020. http://nbn-resolving.de/urn:nbn:de:bsz:16-heidok-279386.
Der volle Inhalt der QuelleZhang, Danfeng [Verfasser], und Benito [Akademischer Betreuer] Yard. „The role of nuclear factor of activated T cells 5 (NFAT5) in inflammation and the potential use of bifunctional enzyme triggered carbon monoxide releasing molecule in treatment of systemic inflammation / Danfeng Zhang ; Betreuer: Benito Yard“. Heidelberg : Universitätsbibliothek Heidelberg, 2020. http://d-nb.info/1205807497/34.
Der volle Inhalt der QuelleChebel, Amel. „Influence de la stimulation et de la sénescence réplicative des lymphocytes T sur le métabolisme des télomères“. Thesis, Lyon 1, 2010. http://www.theses.fr/2010LYO10008.
Der volle Inhalt der QuelleLymphocytes are an example of somatic cells capable to induce telomerase activity when stimulated. We showed that lymphocytes, during long-term culture and repeated PHA stimulations, present a progressive drop in telomerase activity interrupted at each stimulation by a transitory increase. These variations are positively correlated with hTERT and telomere length variations. γ-H2AX and 53BP1 foci and their localization on telomeres increase with cell aging. We show a telomere dysfunction during in vitro lymphocyte senescence resulting from an excessive telomere shortening and a decrease in shelterin content. The mechanism involved in early variations of hTERT expression during lymphocyte activation remained to be understood. Consequences of lymphocyte treatment with different immunosuppressors, all acting directly or indirectly on NFAT activation, suggested a role for NFAT in the regulation of hTERT transcription. Five putative responsive elements for NFAT were identified in the hTERT promoter. We showed that NFAT activates in vitro the hTERT promoter mainly via a consensus site localized in the promoter core at position -40 and a functional synergy between NFAT and SP1. Furthermore, NFAT1 binds directly to the endogenous hTERT promoter via this consensus site in vivo. Thus, NFAT positively regulates the hTERT transcription and we propose its implication in telomerase activation during lymphocyte stimulation
Buchteile zum Thema "NFATc [Nuclear Factor of Activated T-cell]"
Horstkorte, Rüdiger, Bettina Büttner, Kaya Bork, Navdeep Sahota, Sarah Sabir, Laura O’Regan, Joelle Blot et al. „NFAT2 (Nuclear Factor of Activated T-Cells 2, NFATc, NFAT Cytosolic, NFATc1, NFAT Cytosolic 1)“. In Encyclopedia of Signaling Molecules, 1215. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100915.
Der volle Inhalt der QuelleHorstkorte, Rüdiger, Bettina Büttner, Kaya Bork, Navdeep Sahota, Sarah Sabir, Laura O’Regan, Joelle Blot et al. „NFAT4 (Nuclear Factor of Activated T-Cells 4, NFATx, NFATc3, NFAT Cytosolic 3)“. In Encyclopedia of Signaling Molecules, 1215. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100917.
Der volle Inhalt der QuelleHorstkorte, Rüdiger, Bettina Büttner, Kaya Bork, Navdeep Sahota, Sarah Sabir, Laura O’Regan, Joelle Blot et al. „NFAT1 (Nuclear Factor of Activated T-Cells 1, NFATp, NFAT Preexisting, NFATc2, NFAT Cytosolic 2)“. In Encyclopedia of Signaling Molecules, 1215. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100914.
Der volle Inhalt der QuelleHorstkorte, Rüdiger, Bettina Büttner, Kaya Bork, Navdeep Sahota, Sarah Sabir, Laura O’Regan, Joelle Blot et al. „NFAT3 (Nuclear Factor of Activated T-Cells 3, NFATc4, NFAT Cytosolic 4)“. In Encyclopedia of Signaling Molecules, 1215. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100916.
Der volle Inhalt der QuelleAramburu, Jose, und Cristina López-Rodriguez. „Nuclear Factor of Activated T Cells (NFAT)“. In Encyclopedia of Medical Immunology, 824–33. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-84828-0_41.
Der volle Inhalt der QuelleHorstkorte, Rüdiger, Bettina Büttner, Kaya Bork, Navdeep Sahota, Sarah Sabir, Laura O’Regan, Joelle Blot et al. „NFAT5 (Nuclear Factor of Activated T-Cells 5, TonEBP, Tonicity-Responsive Enhancer Binding Protein, NFATz, OREBP, Osmotic Response Element-Binding Protein, NFATz, NFAT-L1, NFAT-Related Protein 1)“. In Encyclopedia of Signaling Molecules, 1215. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100918.
Der volle Inhalt der Quelle„NFAT (nuclear factor activated T cell)“. In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1343. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_11384.
Der volle Inhalt der Quelle„Nuclear Factor of Activated T-Cell C3 (NFATc3)“. In Encyclopedia of Metalloproteins, 1594. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_100902.
Der volle Inhalt der QuelleMacian, F. „Nuclear Factor of Activated T Cells and Tolerance“. In Encyclopedia of Cell Biology, 573–79. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-394447-4.30088-8.
Der volle Inhalt der QuelleMacian, Fernando. „Cellular Immunology: Transcriptional Basis of T Cell Lineages – Nuclear Factor of Activated T Cells and Tolerance“. In Reference Module in Life Sciences. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-821618-7.00132-2.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "NFATc [Nuclear Factor of Activated T-cell]"
Lainšček, D., V. Mikolič, Š. Malenšek, A. Verbič und R. Jerala. „P07.02 Regulation of CD19 CAR T- cell activation based on engineered Nuclear factor of activated T cells artificial transcription factors“. In iTOC8 – the 8th Leading International Cancer Immunotherapy Conference in Europe, 8–9 October 2021, Virtual Conference. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/jitc-2021-itoc8.43.
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